- •Contents
- •Preface
- •Contributors
- •1 Vessels
- •1.1 Aorta, Vena Cava, and Peripheral Vessels
- •Aorta, Arteries
- •Anomalies and Variant Positions
- •Dilatation
- •Stenosis
- •Wall Thickening
- •Intraluminal Mass
- •Perivascular Mass
- •Vena Cava, Veins
- •Anomalies
- •Dilatation
- •Intraluminal Mass
- •Compression, Infiltration
- •1.2 Portal Vein and Its Tributaries
- •Enlarged Lumen Diameter
- •Portal Hypertension
- •Intraluminal Mass
- •Thrombosis
- •Tumor
- •2 Liver
- •Enlarged Liver
- •Small Liver
- •Homogeneous Hypoechoic Texture
- •Homogeneous Hyperechoic Texture
- •Regionally Inhomogeneous Texture
- •Diffuse Inhomogeneous Texture
- •Anechoic Masses
- •Hypoechoic Masses
- •Isoechoic Masses
- •Hyperechoic Masses
- •Echogenic Masses
- •Irregular Masses
- •Differential Diagnosis of Focal Lesions
- •Diagnostic Methods
- •Suspected Diagnosis
- •3 Biliary Tree and Gallbladder
- •3.1 Biliary Tree
- •Thickening of the Bile Duct Wall
- •Localized and Diffuse
- •Bile Duct Rarefaction
- •Localized and Diffuse
- •Bile Duct Dilatation and Intraductal Pressure
- •Intrahepatic
- •Hilar and Prepancreatic
- •Intrapancreatic
- •Papillary
- •Abnormal Intraluminal Bile Duct Findings
- •Foreign Body
- •The Seven Most Important Questions
- •3.2 Gallbladder
- •Changes in Size
- •Large Gallbladder
- •Small/Missing Gallbladder
- •Wall Changes
- •General Hypoechogenicity
- •General Hyperechogenicity
- •General Tumor
- •Focal Tumor
- •Intraluminal Changes
- •Hyperechoic
- •Hypoechoic
- •Nonvisualized Gallbladder
- •Missing Gallbladder
- •Obscured Gallbladder
- •4 Pancreas
- •Diffuse Pancreatic Change
- •Large Pancreas
- •Small Pancreas
- •Hypoechoic Texture
- •Hyperechoic Texture
- •Focal Changes
- •Anechoic Lesion
- •Hypoechoic Lesion
- •Isoechoic Lesion
- •Hyperechoic Lesion
- •Irregular (Complex Structured) Lesion
- •Dilatation of the Pancreatic Duct
- •Marginal/Mild Dilatation
- •Marked Dilatation
- •5 Spleen
- •Nonfocal Changes of the Spleen
- •Diffuse Parenchymal Changes
- •Large Spleen
- •Small Spleen
- •Focal Changes of the Spleen
- •Anechoic Mass
- •Hypoechoic Mass
- •Hyperechoic Mass
- •Splenic Calcification
- •6 Lymph Nodes
- •Peripheral Lymph Nodes
- •Head/Neck
- •Extremities (Axilla, Groin)
- •Abdominal Lymph Nodes
- •Porta Hepatis
- •Splenic Hilum
- •Mesentery (Celiac, Upper and Lower Mesenteric Station)
- •Stomach
- •Focal Wall Changes
- •Extended Wall Changes
- •Dilated Lumen
- •Narrowed Lumen
- •Small/Large Intestine
- •Focal Wall Changes
- •Extended Wall Changes
- •Dilated Lumen
- •Narrowed Lumen
- •8 Peritoneal Cavity
- •Anechoic Structure
- •Hypoechoic Structure
- •Hyperechoic Structure
- •Anechoic Structure
- •Hypoechoic Structure
- •Hyperechoic Structure
- •Wall Structures
- •Smooth Margin
- •Irregular Margin
- •Intragastric Processes
- •Intraintestinal Processes
- •9 Kidneys
- •Anomalies, Malformations
- •Aplasia, Hypoplasia
- •Cystic Malformation
- •Anomalies of Number, Position, or Rotation
- •Fusion Anomaly
- •Anomalies of the Renal Calices
- •Vascular Anomaly
- •Diffuse Changes
- •Large Kidneys
- •Small Kidneys
- •Hypoechoic Structure
- •Hyperechoic Structure
- •Irregular Structure
- •Circumscribed Changes
- •Anechoic Structure
- •Hypoechoic or Isoechoic Structure
- •Complex Structure
- •Hyperechoic Structure
- •10 Adrenal Glands
- •Enlargement
- •Anechoic Structure
- •Hypoechoic Structure
- •Complex Echo Structure
- •Hyperechoic Structure
- •11 Urinary Tract
- •Malformations
- •Duplication Anomalies
- •Dilatations and Stenoses
- •Dilated Renal Pelvis and Ureter
- •Anechoic
- •Hypoechoic
- •Hypoechoic
- •Hyperechoic
- •Large Bladder
- •Small Bladder
- •Altered Bladder Shape
- •Intracavitary Mass
- •Hypoechoic
- •Hyperechoic
- •Echogenic
- •Wall Changes
- •Diffuse Wall Thickening
- •Circumscribed Wall Thickening
- •Concavities and Convexities
- •12.1 The Prostate
- •Enlarged Prostate
- •Regular
- •Irregular
- •Small Prostate
- •Regular
- •Echogenic
- •Circumscribed Lesion
- •Anechoic
- •Hypoechoic
- •Echogenic
- •12.2 Seminal Vesicles
- •Diffuse Change
- •Hypoechoic
- •Circumscribed Change
- •Anechoic
- •Echogenic
- •Irregular
- •12.3 Testis, Epididymis
- •Diffuse Change
- •Enlargement
- •Decreased Size
- •Circumscribed Lesion
- •Anechoic or Hypoechoic
- •Irregular/Echogenic
- •Epididymal Lesion
- •Anechoic
- •Hypoechoic
- •Intrascrotal Mass
- •Anechoic or Hypoechoic
- •Echogenic
- •13 Female Genital Tract
- •Masses
- •Abnormalities of Size or Shape
- •Uterus
- •Abnormalities of Size or Shape
- •Myometrial Changes
- •Intracavitary Changes
- •Endometrial Changes
- •Fallopian Tubes
- •Hypoechoic Mass
- •Anechoic Cystic Mass
- •Solid Echogenic or Nonhomogeneous Mass
- •14 Thyroid Gland
- •Diffuse Changes
- •Enlarged Thyroid Gland
- •Small Thyroid Gland
- •Hypoechoic Structure
- •Hyperechoic Structure
- •Circumscribed Changes
- •Anechoic
- •Hypoechoic
- •Isoechoic
- •Hyperechoic
- •Irregular
- •Differential Diagnosis of Hyperthyroidism
- •Types of Autonomy
- •15 Pleura and Chest Wall
- •Chest Wall
- •Masses
- •Parietal Pleura
- •Nodular Masses
- •Diffuse Pleural Thickening
- •Pleural Effusion
- •Anechoic Effusion
- •Echogenic Effusion
- •Complex Effusion
- •16 Lung
- •Masses
- •Anechoic Masses
- •Hypoechoic Masses
- •Complex Masses
- •Index
8
Peritoneal Cavity
LocalizedPerforation
If the gas leak is small and the perforation in the GI tract is well localized, there will not be any frank perforation. The hyperechoic collection of gas with its characteristic reverberation echoes is truly local and will not shift on patient repositioning. Most of the localized hyperechoic collections of gas will be seen in the immediate vicinity of the GI tract and will be difficult to differentiate from the intraluminal gas collections (Fig. 8.41). Apart from the intracavitary hyperechoic structures of the perforation, there may be fluid as well (mixed with hypoechoic regions), resulting in a mixed image.
Fig. 8.41
a Localized hyperechoic structure with reverberation echoes and posterior shadowing around the hepatic portal (subhepatic left) in a localized perforated duodenal ulcer.
b Hyperechoic structures in localized perforation.
Gas Abscess
In a gas abscess the hypoechoic regions of the liquid components may be predominant. The gas produced by the bacteria is characterized by individual marked echo complexes increasing over the course of the abscess (Fig. 8.42). In most cases it displays an irregular margin. Differential diagnosis is based on the clinical picture supplemented by fine-needle puncture.
Fig. 8.42 Localized hypoechoic, rather smoothly defined structure with a hyperechoic region corresponding with gas; the “curtain-like” appearance and the posterior shadowing are indicative. Large paracolic gas abscess on the right complicating necrotizing pancreatitis.
■ Wall Structures
Smooth Margin
Cavity |
Diffuse Changes |
||||
|
|
|
|||
|
|
|
Localized Changes |
||
|
|
|
|||
|
|
|
Wall Structures |
||
Peritoneal |
|||||
|
Smooth Margin |
||||
|
|
|
|
||
|
|
|
|
Irregular Margin |
|
|
|
|
Differentiating Intraand Extraluminal |
||
|
|
|
|||
|
|
|
GI Tract Fluid |
Right-sided Heart Failure
Cirrhosis
Peritoneal Carcinomatosis
The peritoneum can only be visualized indirectly (see above). Usually the peritoneum is smoothly defined.
Right-sided Heart Failure
In right ventricular failure the intra-abdominal collection of fluid is a transudate. The outline of the peritoneum is always smooth and lacks any juxtaposition, while the ascites is always anechoic (Fig. 8.15).
312
Cirrhosis
Decompensated cirrhosis of the liver with ascites is characterized by a smoothly delineated peritoneum. The ascites is mainly anechoic. The liver displays the changes in shape and texture, among others, characteristic of cirrhosis (Fig. 8.43a,b).
Fig. 8.43 |
b In caput medusae, the tortuous course of the blood |
a Smoothly delineated visceral and parietal peritoneum |
vessels on the inside of the abdominal wall is a sure sign |
in decompensated alcoholic cirrhosis of the liver. |
of portal hypertension in cirrhosis. |
Peritoneal Carcinomatosis
In peritoneal carcinomatosis the peritoneum may still be smoothly delineated and appear absolutely normal (Figs. 8.17, Fig. 8.18). However, much more common are irregularities, septation, and tumor nodes on the peritoneum itself. The findings are considered as cardinal signs of malignancy.
Irregular Margin
Cavity |
Diffuse Changes |
||||
|
|
|
|||
|
|
|
Localized Changes |
||
|
|
|
Wall Structures |
||
Peritoneal |
|||||
|
Smooth Margin |
||||
|
|
|
|
||
|
|
|
|
Irregular Margin |
|
|
|
|
|
||
|
|
|
Differentiating Intraand Extraluminal |
||
|
|
|
|||
|
|
|
GI Tract Fluid |
Pancreatitis
Peritonitis
Peritoneal Carcinomatosis
Pancreatitis
The intra-abdominal collection of fluid in pancreatitis is characterized not only by its internal echoes but, as has already been stated above, quite often by septation and strands of fibrin. These originate from the peritoneum and will lead to an irregular outline. “Fluttering fibrin strands” originating from the peritoneum may be found in pancreatitis as well as peritonitis or after a bleeding has occurred (Fig. 8.44,
Fig. 8.45).
Fig. 8.44 “Flags” of fibrin coating the peritoneum and dominating the picture in necrotizing pancreatitis (exudate).
Fig. 8.45 The septation may become almost complete and can mask all of the peritoneum. Honeycomb texture due to strands of fibrin in necrotizing pancreatitis.
8
Wall Structures
313
8
Peritoneal Cavity
Peritonitis
Bacterial peritonitis is not necessarily accompanied by ascites. In the classic case, the peritoneum is lined by inflammatory accretions and will display an irregular outline on ultrasonography. In addition, there may be strandlike extensions and irregular thickening (Fig. 8.46a,b).
Fig. 8.46 |
b Subphrenic fluid collections with gas accumulations as |
a Diffuse purulent peritonitis with fluid collections inter- |
a sign of bacterial colonization. |
spersed with echoes and septations. |
|
Peritoneal Carcinomatosis
In peritoneal carcinomatosis the peritoneum may be thickened and display an irregular outline. These irregularities in the contour, including tumor masses on the peritoneum, are indicative of advanced peritoneal carcinomatosis. The more ascites the better the irregularities will be visualized. Usually, these changes are easier to demonstrate on the visceral peritoneum (Figs. 8.25, 8.47, 8.48, 8.3).
The peritoneum is a frequent site for metastases. Eighty percent of all malignancies can result in peritoneal carcinomatosis; the most
common origin is GI and gynecological tumors (Table 8.5). However, diagnostic ultrasound in peritoneal carcinomatosis is not without pitfalls. Its sensitivity is only 50–60%, one of the prime reasons being the fact that only about 50% of all cases with peritoneal carcinomatosis will present with ascites. The optimum diagnostic modality is laparoscopy, but some criteria for ultrasound morphology can be quite useful for differentiating benign from malignant ascites (Table 8.6, Table 8.7, Fig. 8.49, 8.6).
The importance of ultrasound-guided fineneedle puncture in the precise assessment of the fluid has already been discussed. In malignant ascites this is an exudate that quite often has high cholesterol levels. Malignant cells will be found in 50–70% of cases. Repeat paracentesis and cytological evaluation will increase the sensitivity to 70–80%.7
Fig. 8.47 Inhomogeneous solid tumor masses lining the |
Fig. 8.48 Juxtapositions and strands of fibrin on the vis- |
Fig. 8.49 Schematic representation of the characteristic |
parietal (anterior) peritoneum, which on the whole dis- |
ceral peritoneum (liver and bowel) destroying an other- |
signs in benign (left) and malignant (right) ascites.11 |
plays an irregular outline. |
wise smooth contour: peritoneal carcinomatosis in ad- |
|
|
vanced gastric cancer. |
|
Table 8.5 Primary malignancies in peritoneal carcinomatosis
GI tract (stomach, pancreas, colon) |
34% |
Ovaries |
27% |
Breast |
14% |
Lymphoma |
4% |
Sarcoma, kidney, uterus, unknown |
18% |
primary |
|
Table 8.6 Signs of peritoneal carcinomatosis2
Thickened omentum |
97% |
Peritoneal mass |
19% |
Interrupted peritoneum |
16% |
Mesenteric adhesions |
16% |
Hepatic metastases |
38% |
Lymph node metastases |
24% |
Ascites |
49% |
314
Table 8.7 Ultrasound in the differential diagnosis of benign and malignant ascites |
|
|
Ultrasound morphology |
Benign |
Malignant |
Echogenicity of the ascites |
|
|
● Anechoic |
++ |
+ |
● Internal echoes (echogenic) |
+ |
++ |
● Septation |
+ |
++ |
Fluid shift on repositioning |
Unrestricted |
Limited |
Septation |
Unrestricted |
Walled o , encapsulated |
Peritoneal margin |
Smooth |
Irregular, mass |
Greater omentum |
Thin |
Thickened, rigid |
Mesentery |
Unrestricted |
Retracted |
Small-bowel loops |
“Sea anemone,” “creepers” |
Conglomerate |
Abdominal wall |
Thin, mobile |
Thickened, rigid |
Bowel–abdominal wall |
Unrestricted |
Adherence |
Other signs of disease |
Cirrhosis, pancreatitis, right ventricular failure |
Metastases; malignancy of the bowel, pancreas, |
|
|
uterus, ovaries |
Lymph node and hepatic metastases |
None |
++ |
8.6 Differential Diagnosis of Benign and Malignant Ascites
Benign ascites
a Smooth peritoneum and freely floating |
b “Angle of ascites” and mesenteric col- |
c The “heart” in the lower abdomen, re- |
loops of bowel (“sea anemone” phenom- |
laterals (shunts) in portal hypertension |
sulting from the tortuous collaterals in |
enon) in benign ascites: decompensated |
point to benign ascites: portal hyperten- |
portal hypertension with shunts, points |
cirrhosis. |
sion. |
to benign ascites. |
Malignant ascites
d Internal echoes and a clearly retracted |
e The presence of hepatic metastasis usu- |
f Marked septation and strand-like struc- |
mesentery signifying malignant ascites, |
ally proves the malignant origin of the |
tures originating from the peritoneum |
compared with the “sea anemone phe- |
ascites: small hepatic metastasis in pan- |
(here, ovarian malignancy) tend to be |
nomenon” in benign ascites. |
creatic cancer. |
indicative of malignant rather than be- |
|
|
nign ascites. |
8
Wall Structures
315