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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_4421_Библиотеки_им_академика_М_И_Перельмана.pdf
Present condition
Surgical treatment objection
Maxillary Surgical Procedures forCorrection ofObstructive Sleep Apnea
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25
15
. Fig. 25.1 Tracing of lateral cephalometric radiograph of the
patient prior to surgery with Wolford Analysis [42]
sist of a clinical exam, radiographs, models, and prediction tracing of lateral cephalogram. The clinical exam
will consist of measuring facial and occlusal landmarks
on a patient and comparing them against a standard
which accounts for a patient’s age, race, and sex.
Radiographs should include a panoramic lm, lateral
cephalometric lm, or a 3D cone beam CT.A prediction
tracing should be constructed for the patient’s current
condition which when taken along with clinical impression help determine the diagnosis (. Fig. 25.1). The
surgical treatment objective (. Fig.25.2) tracing should
demonstrate the planned movement during surgery.
More recently, virtual surgical planning (VSP) based on
advanced imaging, has in some cases, replaced traditional haptic methods (. Fig. 25.3). Maxillary and
mandibular models should be related to each other in
centric relation and mounted in an articulator to allow
spatial orientation of the planned advancement and surgical splint fabrication (. Fig.25.4).
37
43
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2
19
4
1
. Fig. 25.2 Surgical treatment objective tracing demonstrates
planned movement of maxilla and mandible during surgery using
Wolford Analysis [42]
4
4
12
2
cesses. The frontal process of the maxilla extends superiorly to form the lateral aspect of the nose and the
medial aspect of the orbit. The inferior-medial aspect of
the orbital rim is formed from the maxilla. Approximately,
5–7mm below the orbital rim is where the infraorbital
nerve exits its canal. The zygomatic process extends laterally to meet the zygomatic bone. The palatine processes extend medially as a horizontal shelf of bone
which fuses midline into the median palatine suture.
This process extends posteriorly to join the palatine
bone to form the hard palate. Distal to the maxillary
tuberosity is the infratemporal surface of the maxilla
which is the site of lateral pterygoid plate separation
during LeFort I and SARPE surgery (. Fig.25.5).
25.2.2 Vascular Anatomy
25.2.1 Bone Anatomy
The body of the maxilla is composed of two halves
which fuse midline and contain four projections called
the frontal, zygomatic, dentoalveolar, and palatine pro-
The LeFort I osteotomy was performed before the vascular anatomy was fully understood. During surgery, the
nasopalatine artery and descending palatine artery can
be separated and direct perfusion from the maxillary soft
tissue is disrupted. This led to the belief that collateral
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ab
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W. R. Allen and M. J. Madsen
. Fig. 25.3 Virtual surgical planning (VSP) is performed using 3D cone beam CT and digital scan of dentition. This planning can supple-
ment or take the place of conventional lateral cephalometric tracing
. Fig. 25.4 Dental models of maxillary and mandibular arch are
related to each other in centric relation on an articulator using
mounted facebow transfer. Model surgery is performed on these
circulation from the soft palate was sufcient to provide
vascular support to the maxilla. Bell demonstrated in a
models to fabricate acrylic splints used to control movements of
each arch during surgery
25.3 Lefort IOsteotomy Including
Modications
classic study that blood supply to the maxilla came from
the ascending pharyngeal artery and the ascending palatine artery [43]. The ascending palatine artery is a branch
of the facial artery and the ascending pharyngeal artery
is a branch of the external carotid artery (. Fig.25.6).
The OSA application of the Lefort I Osteotomy is similar to that described by Obwegeser in 1965 [44]. The goal
is complete mobilization of the maxilla to allow for signicant advancement. An external reference is recom-
Greater palatine
ry
Maxillary Surgical Procedures forCorrection ofObstructive Sleep Apnea
Nasopalatine
artery
artery
385
Descending
palatine artery
Maxillary artery
Lesser
palatine
artery
Ascending
pharyngeal
artery
Facial arte
25
. Fig. 25.5 The maxilla has four projections called the frontal,
zygomatic, dentoalveolar, and palatine processes which articulate
with nine bones of the midface and cranium
mended to allow proper orientation. This is accomplished
with Kirschner wire (0.035 inch) insertion into the
nasion region (. Fig. 25.7). A caliper is then used to
measure from the central incisor brackets to the K-wire
and recorded for reference (. Fig.25.8). Surgical exposure is accomplished using a maxillary vestibular incision that extends from rst molar to rst molar. It is
imperative to leave an adequate margin of nonkeratinized mucosa for closure. The incision should be made
5mm above the mucogingival junction (. Fig.25.9). A
full thickness mucoperiosteal ap is elevated with dissection and elevation of periosteum around the piriform
rim and anterior nasal spine. Dissection is then carried
superiorly identifying the infraorbital nerve and infraorbital foramen. Next, the lateral maxilla is the dissected
to the zycomaticomaxillary junction and dissection is
carried to the pterygomaxillary junction. The nasal
mucosa is elevated from the nasal oor and lateral nasal
walls with a curved Freer elevator or Molt 9 elevator.
Ascending
palatine artery
. Fig. 25.6 Ascending palatine artery and ascending pharyngeal
artery anastomose with the greater palatine artery to provide blood
supply to the maxilla
. Fig. 25.7 External reference is performed by insertion of a
K-wire into the intersection of frontal and nasal bones
External
carotid
Once surgical exposure is complete, a curved tip
Obwegeser retractor is placed into the pterygomaxillary
junction using care to make sure the instrument is subperiosteal and is resting on the lateral pterygoid plate. A
Seldin retractor is placed submucosally along the lateral
nasal wall. A horizontal osteotomy is then completed
using a reciprocating saw starting from the pterygomaxillary junction medially to the piriform rim or lateral
nasal wall depending on where the cut is desired. Care
should be exercised to maintain at least 5 mm of distance from the tooth root apices and horizontal osteotomy. Prior to horizontal osteotomy, internal reference
25
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W. R. Allen and M. J. Madsen
. Fig. 25.8 The vertical height of the maxilla is conrmed using a
caliper which measures from the K-wire to the orthodontic bracket
on the central incisor
. Fig. 25.10 Bony reduction of nasal septum, tuberosity, pyrami-
dal process, and zygomatic buttress is performed to allow proper
condylar positioning prior to xation
. Fig. 25.9 Incision is made in alveolar mucosa 5 mm above the
keratinized gingiva. This cuff of alveolar mucosa allows for proper
closure following surgery
points may be scored on the bony surface but are not
always necessary. Next, separation of the nasal septum,
lateral nasal walls, and pterygoid plates is accomplished
in that order. A spatula or straight edge osteotome is
used to begin separation of the nasal septum from the
anterior nasal spine. The septal osteotomy is completed
by the use of a double-guarded septal osteotome. It is
important to maintain contact with the bony nasal oor
to prevent tearing of the nasal oor mucosa. Drive the
osteotome inferiorly and posteriorly with a mallet while
keeping one nger on the posterior aspect of the palatine bone for spatial reference. Lateral nasal wall osteotomies are completed using single- guarded osteotomes.
A mallet is used to drive the osteotome posteriorly and
parallel to the nasal oor until resistance is met at the
pyramidal process of the palatine bone. The nal osteotomy is of the pterygoid plates, performed with a
curved osteotome placed at the bony junction of the
pterygoid plate and posterior maxilla. A mallet is used
to drive the osteotome in an inferior and medial direction. A nger can be placed intraorally near the tuberosity for spatial orientation. Hypotensive anesthesia can
be utilized to minimize intraoperative blood loss. At this
point, maxillary down fracture is initiated using manual
pressure by distraction of the anterior maxilla inferiorly.
If resistance is encountered, rene the osteotomies in
the areas where resistance is met. Once the down fracture is complete, Rowe disimpaction forceps or Tessier
mobilizers may be utilized to pull the maxilla down and
forward. The maxilla must be mobilized freely from all
of its bony attachments. The soft tissues of the nasal
oor and posterior maxilla should be protected from
trauma during this mobilization using Seldin retractors
or Obwegeser retractors. Following down fracture and
mobilization (.
Fig.25.10), all bony interferences are
removed that inhibit the forward positioning of the
maxilla. This usually involves reduction ostectomy of
the maxillary bony septum, posterior tuberosity regions,
pyramidal process and lateral maxillary wall, or zygomatic buttress areas. Once mobilization of the maxilla
and interferences have been removed, the segmental surgery of the maxilla is completed. Depending on the surgical plan, widening of the maxilla is accomplished
using a 2 or 3 piece modication of the traditional
Lefort I osteotomy. Interdental osteotomies are made
with a ne-tapered ssure bur, oscillating saw or piezoelectric unit (.
Fig.25.11). Spatula osteotomes are used
for renements (. Fig.25.12). Sagittal or paramedian
cuts in the posterior maxilla connect to anteriorly based
interdental vertical cuts in the premaxilla to allow mobilization of the segments (. Fig. 25.13a–c). The inter-
Maxillary Surgical Procedures forCorrection ofObstructive Sleep Apnea
387
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dental osteotomies should be outlined prior to
downfracture to allow a precise osteotomy in the maxilla that is stable rather than mobile. Palatal incisions
may be implemented to allow more denitive mobilization of the maxillary segments. (. Fig.25.14a, b). This
. Fig. 25.11 Maxillary segmentation is performed using monocor-
tical interdental osteotomies
incision is paramedian and made over supported bone,
not the osteotomy to prevent postoperative oronasal stula. Final renements are carried out after the surgical
splint has been placed. Acrylic surgical splints which are
. Fig. 25.12 The initial monocortical osteotomy is completed
using spatula osteotome. The index nger is placed on the palate.
Care should be taken to avoid perforation of palatal soft tissue
a
c
b
. Fig. 25.13 Sagittal osteotomies are performed midline or paramedian where the bone is slightly thinner. These osteotomies are connected
with the interdental osteotomies. The maxilla can be widened with Turvey maxillary expander
25
ab
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W. R. Allen and M. J. Madsen
. Fig. 25.14 Widening of the maxilla greater than 5 mm will
require palatal releasing incisions to undermine the soft tissue prior
to bony expansion. These incisions are made over supported bone.
fabricated preoperatively on the articulated models are
ligated against the maxillary arch with 26-gauge wire.
With segmental surgery, care must be taken to ensure
that all pieces are securely ligated to the splint passively.
The mandible is then wired into the splint. This allows
the manipulation of the maxilla and mandible as a single unit. While attempting to seat the mandibular condyle upward, posterior pressure is applied in a posterior
and superior vector and nal interferences can be identied and removed. Vertical reference is veried using a
caliper (. Fig.25.8). Rigid xation is performed using
mini-plates at the buttress regions of the zygoma and
piriform (. Fig. 25.15). Incision closure is performed
using V-Y suturing to optimize aesthetic outcome
(. Fig.25.16). A single skin hook is used to retract the
superior margin of the incision upward. The vertical
midportion of the incision is closed rst, followed by
standard vestibular closure of the remainder of the incision. This closure seeks to prevent foreshortening of the
upper lip.
The soft tissue is undermined to midline. Passive maxillary expansion without stretching palatal soft tissue is important to prevent
tearing which will result in oronasal perforation
. Fig. 25.15 Passive mini-plate adaptation and drilling holes cen-
ter mass prevents plate or segment shifting during xation
25.4 Surgically Assisted Rapid Palatal
Expansion (SARPE)
Transverse maxillary expansion by growth modication or surgical separation of the midpalatal suture is
often used as a reliable correction for dentofacial deformities. The benets in treating OSA using SARPE
applications involve increasing the transverse width of
the nasal and upper airway. The decision to perform
SARPE versus rapid maxillary expansion (RME) is
solely a question of growth potential. Surgical options
are usually recommended beyond the age of 16 [45].
Although not considered rst-line therapy of OSA
treatment, it has been shown to be an effective alterna-
. Fig. 25.16 V-Y closure minimizes lip shortening postoperatively.
Superior retraction allows closure of the vertical midportion of incision rst. This is followed by standard vestibular closure of remaining incision
Maxillary Surgical Procedures forCorrection ofObstructive Sleep Apnea
tive to traditional Lefort I surgery in patient with a
mild AHI index and can be employed when medical
management is not an option. In addition, a benet to
SARPE treatment is that it can often be employed as
an outpatient setting.
The surgical technique of SARPE shares many of
the same principles with Lefort I osteotomy. The surgery begins with injection of local anesthetic with a
vasoconstrictor. A vestibular incision is made from rst
molar to the canine region. The incision is not carried
across the midline. The same principles apply where a
5mm cuff of mucosal tissue preserved superior to the
mucogingival junction to allow for proper closure. Full
thickness mucoperiosteal elevation is accomplished
around the piriform, zygomaticomaxillary, and pterygomaxillary regions. The mucosa of the lateral nasal
wall and oor of the nose is also elevated and protected. A horizontal osteotomy is then made using a
reciprocating saw starting at the zygomaticomaxillary
buttress area and extending medially through the lateral maxilla to the piriform region. A back cut through
the tuberosity can also be made while protecting the
pterygomaxillary area with an Obwegeser retractor.
The same principle of keeping a safe distance of 5mm
from the teeth root apices applies during this horizontal osteotomy (.
Fig.25.17). Next, a midline vertical
incision is made over the anterior nasal spine and
extended inferiorly into the keratinized tissue of the
gingiva between the central incisors. Subperiosteal
reection identies the piriform, anterior nasal spine,
and tooth root eminences of the central incisors. A thin
oscillating saw is used to make an osteotomy into the
midpalatal suture using care not to perforate the pala-
389
. Fig. 25.18 SARPE: Sagittal osteotomy is performed through a
vertical incision. The osteotomy is performed using an oscillating or
reciprocating saw. An osteotome is placed in the osteotomy and
directed posteriorly to complete division of maxilla into separate
halves
tal mucosa. A thin spatula osteotome is then used to
complete the osteotomy driving the osteotome posteriorly through the palatal bone with a mallet using a nger intraorally for orientation (. Fig. 25.18).
Separation of the suture is veried by opening the
Hyrax expander. If the segments are not mobilized, the
decision may be made to separate the pterygomaxillary
attachment using a curved osteotome. Following mobilization of the maxilla, closure is achieved with a 4.0
chromic suture.
25
. Fig. 25.17 SARPE: Horizontal osteotomy maxilla is performed
5mm superior to apex of tooth roots
25.5 Adjunct Procedures
In order to decrease the airway resistance, a number of
modications to the maxilla and surrounding structures may also be implemented. These include easy
access for inferior turbinectomy, septoplasty, and nasal
oor contouring. With the maxilla downfractured, the
inferior turbinate is directly visualized and can be
removed with a large hemostat and Dean scissors. In
cases where septal deviation is encountered septoplasty
may also easily be accomplished. Also as noted, OSA
can be related to a constricted maxilla. In these cases,
attention should be directed to the alar base and corresponding bony piriform which are likely also to be constricted. With surgical exposure of the piriform, bony
contouring may be accomplished using a round bur or
pineapple bur for osteoplasty or ostectomy with the
objective of widening of the piriform to improve nasal
airow (. Figs.25.19a, b).
25
390
W. R. Allen and M. J. Madsen
a b
. Fig. 25.19 Nasal aperture before and after widening using bur to contour bone at piriform aperture
. Fig. 25.20 Mild vascular insufciency postoperatively results in
necrosis of gingival soft tissue. These patients will have bony union
but soft tissue defect which will require additional surgery for soft
tissue grafting
25.6 Complications
Immediate complications may include bleeding, unfavorable fractures in the segmented maxilla, trauma to
teeth or root apices, malocclusion from unidentied
interferences, or septal deviation. With nal positioning
and plating of the maxilla, it is important to be mindful
of the nal position of the septum, as the nal position
of the maxilla may cause septal deviation. Septal position may even be altered during extubation.
Delayed complications may include bleeding from
Pseudoaneurysm which can be severe requiring emboli-
zation via interventional radiology. Devitalization of
teeth occurs when an osteotomy is performed too close
to a tooth. It will often demarcate within weeks and
require endodontic therapy. Hardware exposure can
occur and will usually re-mucosalize with oral antibiotics, daily irrigation, and vigilant oral hygiene.
The two most difcult complications are infection and
vascular insult. Mild infection can be treated as an outpatient with oral antibiotics and daily irrigation. More severe
infection is an indication to return to the operating room
for surgical irrigation or hardware exchange. Mild vascular
insufciency will result in gingival necrosis and may been
seen in smoking patients or after ligation of descending
palatine arteries in segmental osteotomy (.
Fig.25.20).
Maxillary hypoperfusion from arterial injury or impingement which is prolonged may result in maxillary segment
loss. It can also result from vascular congestion due to
injury to the venous drainage. Prompt identication is
imperative. Reentry with hardware removal can allow for
reperfusion. Hyperbaric oxygen therapy may also be considered if vascular insufciency is identied early. Failure
to identify this or inability to correct it may eventually
result in bony necrosis (. Fig.25.21). Necrotic bone must
be removed and grafting procedures are often required to
correct the resulting defect.
Lastly, this patient population often presents with a
constellation of medical issues from OSA.A sick patient
with OSA and failed phase I therapy may not be a medically optimized patient for major surgery. Care should
be exercised postoperatively where myocardial infarction, stroke, renal insufciency, or other medical emergencies may be confronted.
Maxillary Surgical Procedures forCorrection ofObstructive Sleep Apnea
. Fig. 25.21 Severe vascular insult will result in loss of teeth and
or segment
References
1. Stierer T, Punjabi NM.Demographics and diagnosis of obstructive sleep apnea. Anesthes Clin North Am. 2005;23:405–20.
2. Peppard NR, Young T, Palta M, Skatrud J.Prospective study of
the association between sleep-disordered breathing and hypertension. N Engl J Med. 2000;342(19):1378–84.
3. Pedrotti E, Demasi CL, Bruni E, etal. Prevalence and risk factors of eye diseases in adult patients with obstructive sleep
apnea: results from the SLE.E.P.Y cohort study. BMJ Open.
2017;7:e016142. https://doi.org/10.1136/bmjopen-2017-016142.
4. Elshaug AG, Moss JR, Southcott AM, Hiller JE. Redening
success in airway surgery for obstructive sleep apnea: a meta
analysis and synthesis of the evidence. Sleep. 2007;30(4):461–7.
5. Somers VK, White DP, Amin R, etal. Sleep apnea and cardiovascular disease: An American Heart Association/American
College of Cardiology Foundation Scientic Statement from
American Heart Association Council for High Blood pressure
Research Professional Education Committee, Council of
Clinical Cardiology, Stroke Council and Council on
Cardiovascular Nursing. J Am Coll Cardiol. 2008;52:686–717.
6. Epstein LJ, Kristo D, Strollo PJ, etal. Clinical guideline for evaluation, management and long-term care of obstructive sleep
apnea in adults. J Clin Sleep Med. 2009;5(3):263–76.
7. Holty J, Cuilleminault C.Maxillomandibular advancement for
the treatment of obstructive sleep apnea; a systemic review and
meta analysis. Sleep Med Rev. 2010;14:287–97.
8. Dentino K, Ganjawalla K, Inverso G, etal. Upper airway length
is predictive of obstructive sleep apnea is syndromic craniosynostosis. J Oral Maxillofac Surg. 2015;73:S20–5.
9. Susarla SM, Abrahamson ZR, Dodson, et al. Cephalometric
measurement of upper airway length correlates with presence of
severity of obstructive sleep apnea. J Oral Maxillofac Surg.
2010;68:2846–55.
10. Abrahamson Z, Susarla SM, Lawler M, etal. Three-dimensional
computed tomographic airway analysis of patients with obstructive sleep apnea treated by maxillomandibular advancement. J
Oral Maxillofac Surg. 2011;69:677–86.
11. Sursarla SM, Abramson ZR, Dodson TB, et al. Upper airway
length decreases after maxillomandibular advancement in
patients with obstructive sleep apnea. J Oral Maxillofac Surg.
2011;69:2872–8.
391
12. Zinser MJ, Zachow S, Sailer HF, et al. Bimaxilary ‘rotational
advancement’ procedures in patients with obstructive sleep
apnea: a 3-dimensional airway analysis of morphological
changes. Int J Oral Maxillofac Surg. 2013;42:569–78.
13. Buttereld KJ, Marks PL, McLean L, etal. Pharyngeal airway
morphology in healthy individuals and in obstructive sleep
apnea in patients treated with maxillomandibular advancement:
a comparative study. Oral Surg Oral Med Oral Pathol Oral
Radiol. 2015;119(3):285–92.
14. Vinha PP, Faria AC, Xavier SP, etal. Enlargement of the pharynx resulting from surgically assisted rapid maxillary expansion.
J Oral Maxillofac Surg. 2016;74:369–79.
15. Yu CC, Hsiao HD, Lee LC.Computational uid dynamic study
on obstructive sleep apnea syndrome treated with maxillomandibular advancement. J Craniofacial Surg. 2009;20(2):426–30.
16. Liu SYC, Huon LK, Powell NB, etal. Lateral pharyngeal wall
tension after maxillomandibular advancement for obstructive
sleep apnea is a marker for surgical success: observations from
drug induced sleep endoscopy. J Oral Maxillofac Surg.
2015;73:1575–82.
17. Goodday RH, Bourque SE, Edwards PB.Objective and subjective outcomes following maxillomandibular advancement surgery for treatment of patients with extremely severe obstructive
sleep apnea. J Oral Maxillofac Surg. 2016;74:583–9.
18. Zaghi S, Holty JE, Certal V, etal. Maxillomandibular advancement for treatment of obstructive sleep apnea, a meta analysis.
JAMA Otolaryngol Head Neck Surg. 2016;142(1):58–66.
19. Yu W, Wang M, He J, etal. Combined counterclockwise maxillomandibular advancement and uvulopalatopharyngoplasty
surgeries for severe obstructive sleep apnea. J Craniofacial Surg.
2017;28(2):366–71.
20. Islam S, Uwadiae N, Ormiston IW.Othognathic surgery in the
management of obstructive sleep apnea: experience from maxillofacial surgery unit in the United Kingdom. Br J Oral
Maxillofac Surg. 2014;52:496–500.
21. Boyd SB, Walters AD, Waite P, et al. Long term effectiveness
and safety of maxillomandibular advancement for treatment of
obstructive sleep apnea. J Clin Sleep Med. 2015;11(7):699–708.
22. Won CH, Li KK, Guilleminault C. Surgical treatment of
obstructive sleep apnea. Proc Am Thorac Soc. 2008;5:193–9.
23. Varghese R, Adams NG, Slocumb NL, etal. Maxillomandibular
advancement in the management of obstructive sleep apnea. Int
J Otolaryngol. 2012;2012:373025.
24. Buttereld KJ, Marks PL, McLean L, et al. Quality of life
assessment after maxillomandibular advancement surgery for
obstructive sleep apnea. J Oral Maxillofac Surg. 2016;74:1228–
37.
25. Lee KW. Effect of orthognathic surgery on posterior airway
space. Ann Acad Med. 2008;37(8):p677–82.
26. Fairburn SC, Waite PD, Vilos G, et al. Three-dimensional
changes in upper airways of patients with obstructive sleep
apnea following maxillomandibular advancement. J Oral
Maxillofac Surg. 2007;65:6–12.
27. Faria AC, Xavier SP, Silva SN, etal. Cephalometric analysis of
modications of the pharynx due to maxilla-mandibular
advancement surgery in patients with obstructive sleep apnea.
Int J Oral Maxillofac Surg. 2013;42:579–84.
28. Schendel SA, Broujerdi JA, Jacobson RL. Three dimensional
upper airway changes with maxillomandibular advancement for
obstructive sleep apnea treatment. Am J Orthod Dentofac
Orthop. 2009;135:468–79.
29. Comacho M, Chang ET, Song SA, etal. Rapid maxillary expansion for pediatric obstructive sleep apnea: a systematic review
and meta-analysis. Laryngoscope. 2017;127:1712–9.
25
392
W. R. Allen and M. J. Madsen
25
30. Villa MP, Rizzoli A, Miano S, etal. Efcacy of rapid maxillary
expansion in children with obstructive sleep apnea syndrome: 36
months of follow-up. Sleep Breath. 2011;15:179–84.
31. McNamara JA, Lione R, Franchi L, etal. The role of rapid maxillary expansion in the promotion of oral and general health.
Prog Orthod. 2015;16:33.
32. Galeotti A, Pavone M, De Vincentiis DE.Effects of simultaneous palatal expansion and mandibular advancement in child suffering from OSA.Acta Otorhinolaryngol Ital. 2016;36:328–32.
33. Villa MP, Malagola C, Pagani J, etal. Rapid maxillary expansion
in children with obstructive sleep apnea syndrome: 12 month
follow-up. Sleep Med. 2007;8:128–34.
34. Pirelli P, Saponara M, Guilleminault C.Rapid maxillary expansion (RME) for pediatric obstructive sleep apnea: a 12 year follow- up. Sleep Med. 2015;16:993–35.
35. Iwasaki T, Saitoh I, Takemoto Y, etal. Tongue posture improvement and pharyngeal airway enlargement as secondary effects of
rapid maxillary expansion: a cone beam computed tomography
study. Am J Orthod Dentofac Orthop. 2013;143(2):235–45.
36. Bach N, Tuomilehto H, Gauthier C, etal. The effect of surgically
assisted rapid maxillary expansion on sleep architecture: an
exploratory risk study in healthy young adults. J Oral Rehabil.
2014;40:818–25.
37. Conley SR, Legan HL. Correction of severe obstructive sleep
apnea with bimaxillary transverse distraction osteogenesis and
maxillomandibular advancement. Am J Orthod Dentofac
Orthop. 2006;129:283–92.
38. Foltan R, Hoffmannova J, Pavlikova G, etal. The inuence of
orthognathic surgery on ventilation during sleep. Int J Oral
Maxillofac Surg. 2011;40:146–9.
39. Liu SYC, Guilleminault C, Huon LK, etal. Distraction osteogenesis maxillary expansion (DOME) for adult obstructive sleep
apnea patients with high arched palate. Otolaryngol Head Neck
Surg. 2017;157(2):345–8.
40. Posnick JC, Agnihotri N. Consequences and management of
nasal airway obstruction in the dentofacial deformity patient.
Curr Opin Otolaryngol Head Neck Surg. 2010;18(4):323–31.
41. Abdullatif J, Certal V, Zaghi S.Maxillary expansion and maxillomandibular expansion for adult OSA: a systematic review and
meta-analysis. J Craniomaxillofac Surg. 2016;44(5):574–8.
42. Wolford LM, Hilliard FW, Dugan DJ.Surgical treatment objective. A systematic approach to the prediction tracing. St Louis:
Mosby Year Book; 1985. p.54–74.
43. Bell WH. Revascularization and bone healing after anterior
maxillary osteotomy: a study using adult rhesus monkeys. J Oral
Surg. 1969;27:249–55.
44. Obwegeser HL. Surgical correction of the small or retrodisplaced maxilla: the “dish-face” deformity. Plast Reconstr Surg.
1969;43:351.
45. Epker BN, Wolford LM.Transverse maxillary deciency dentofacial deformities: integrated orthodontic and surgical correction. St. Louis: Mosby; 1980.