There is a well-established relationship between nasal congestion and poor sleep quality. Several large epide­miologic studies have shown that nasal obstruction, either due to anatomic constraints or inammatory dis­ease, is an independent risk factor for snoring and sleep apnea [24, 25, 26]. The link between these two entities is complex and multifactorial. The nose is the primary breathing pathway under normal conditions. Nasal breathing accounts for 92% of inhaled air while awake and 96% of inhaled air while asleep [27]. In healthy indi­viduals, nasal resistance accounts for 55–60% of total airway resistance [28]. This proportion can be signi­cantly impacted by nasal obstructive issues. Pharyngeal airway collapse has been approximated by many authors using a Starling resistor model. The pharynx is modeled as a homogenous hollow tube suspended between rigid upstream and downstream segments. Nasal airway resis­tance represents a partial obstruction at the aperture of the tube and the pharynx represents a collapsible down­stream segment connected to the rigid trachea. Increasing nasal resistance causes negative intraluminal pressure downstream, increasing the tendency of the pharynx to collapse [29, 30]. If nasal obstruction is severe enough, many individuals convert to oral respiration (i.e., mouth breathing). Experiments have demonstrated that oral breathing is physiologically disadvantageous, with upper airway resistance increasing 2.5-fold as compared to nasal breathing during sleep [31].