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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3591_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Preface
- •Acknowledgments
- •Contents
- •About the Editor
- •1.6 Acute Gastrointestinal Bleeding
- •1.7 Transjugular Intrahepatic Portosystemic Shunt
- •1.8 Conclusion
- •References
- •2.1 Introduction
- •2.2 History-Taking
- •2.4 Pre-procedure Imaging
- •2.5 Pre-procedure Investigations
- •2.8 Informed Consent
- •2.10 Part Preparation
- •2.11 Antibiotic Prophylaxis
- •References
- •3.1 Introduction
- •3.2 Ultrasonography
- •3.2.1 Advantages
- •1.1 Introduction
- •1.2 Early Beginnings
- •1.3 Catheter-Directed Thrombolysis
- •1.4 Stents
- •1.5 Coronary Angiography
- •3.2.3 Limitations
- •3.3 Fluoroscopy
- •3.3.1 Indications
- •3.3.2 Interventional Fluoroscopy
- •3.3.2.1 Digital Subtraction Angiography
- •3.4 Computed Tomography
- •3.4.2 Indications
- •3.4.3 Limitations
- •3.5 Magnetic Resonance Imaging
- •3.5.3 Limitations
- •3.6.3 Remove Anti-scatter Grid
- •3.6.4 Pulsed Fluoroscopy
- •3.6.5 Checklist
- •3.9 Conclusion
- •References
- •4.1 Introduction
- •4.2 Pharmaceutical Agents
- •4.2.1 Contrast Agents
- •4.2.2 Iodinated Contrast Media (ICM)
- •Pathogenesis
- •Risk Factors
- •Diagnosis
- •Risk Threshold
- •4.5 Conclusion
- •References
- •4.3 Carbon Dioxide
- •4.3.1 Indication
- •4.3.2 Contraindications
- •4.3.3 Equipment
- •4.3.4 Preparation
- •4.3.5 Technique
- •4.3.6 Complications
- •4.3.7 Advantages
- •4.3.8 Disadvantages
- •4.4.1 Thrombolytics
- •First-Generation Thrombolytic Agents
- •Second-Generation Thrombolytic Agents
- •Third-Generation Thrombolytic Agents
- •4.4.1.2 Contraindications
- •4.4.2 Anticoagulants
- •4.4.2.1 Unfractionated Heparin (UFH)
- •Mechanism
- •Indications
- •4.4.2.2 Low-Molecular-Weight Heparin (LMWH)
- •Mechanism
- •Indications
- •Special Considerations
- •4.4.2.3 Warfarin
- •Mechanism
- •Indications
- •Special Considerations
- •4.4.3 Antiplatelet Drugs
- •4.4.3.1 Aspirin
- •Mechanism
- •Recommendations
- •4.4.3.2 Clopidogrel
- •Mechanism
- •Recommendations
- •4.4.3.3 Glycoprotein IIb/IIIa Inhibitors (GPI)
- •Mechanism
- •Recommendations
- •4.4.4 Vasodilators
- •4.4.4.1 Nitroglycerine
- •Mechanism
- •Indications
- •4.4.4.2 Verapamil
- •Mechanism
- •Indications
- •Contraindications
- •Complications
- •4.4.5 Vasoconstrictors
- •4.4.5.1 Mechanism
- •4.4.5.3 Indication
- •4.4.6 Prothrombotics
- •4.4.6.1 Mechanism
- •4.4.6.3 Indications
- •4.4.6.5 Special Considerations
- •5.1 Introduction
- •5.2 Pre-procedure Tasks
- •5.2.4 Pre-anesthetic Evaluation
- •5.3 Anesthesia Techniques
- •5.3.1 Local Anesthesia
- •5.4 Pediatric IR Procedures
- •5.5 Anesthesia Considerations
- •5.11 Conclusion
- •References
- •6.1 Introduction
- •6.3 Pre-procedural Workup
- •6.3.1 Prothrombin Time (PT-INR)
- •6.3.3 Viscoelastic Tests
- •6.4.1 Procedure-Related Bleeding Risk
- •6.5.1 Chronic Liver Disease
- •6.5.2 Chronic Kidney Disease
- •6.5.3 Thrombocytopenia
- •6.5.4 Disseminated Intravascular Coagulation (DIC)
- •6.5.5 Malignancy
- •6.6 Bridge Therapy
- •6.7 Deep Vein Thrombosis (DVT)
- •6.8 Atrial Fibrillation (AF)
- •6.9 Coronary Stents
- •6.10 Conclusion
- •References
- •7.1 Introduction
- •7.4 Embolic Agent Selection
- •7.5.1 Gelfoam Embolization
- •7.5.2 Coil Embolization
- •7.5.3 Amplatzer Vascular Plug Embolization
- •7.5.4 Glue Embolization
- •7.6 Clinical Applications
- •7.6.1.1 Endovascular Approach
- •Parent Artery Preservation [16, 17]
- •Stent or Balloon-Assisted Coiling [20, 21]
- •Multi-Layered Flow-Diverting Stents [22]
- •7.6.1.2 Parent Artery Occlusion
- •Sandwich Technique [19–22]
- •7.6.1.3 Percutaneous Approach [16, 17, 24, 25]
- •7.6.2 Tumoral Embolization
- •7.6.3 AVM Embolization
- •7.6.5 Special Scenario
- •7.6.5.1 Provocative Angiography
- •7.6.5.2 Lower GI Bleeding
- •7.6.5.3 Hepatic Artery Aneurysm
- •7.6.5.4 Renal Artery Aneurysm (RAA)
- •7.7 Newer Embolizations
- •7.7.1 Genicular Artery Embolization
- •7.8 Conclusion
- •References
- •8.1 Introduction
- •8.2 Puncture Needles
- •8.3 Guidewires
- •8.4 Sheath System
- •8.5 Catheters
- •8.6 Microcatheters
- •8.7 Embolizing Agents
- •8.7.1 Gelatin Foam
- •8.7.2 Autologous Blood Clot
- •8.7.3 Thrombin
- •8.7.4 Coils
- •8.7.5 Vascular Plugs
- •8.7.6 Particulate Agents
- •8.7.7 Liquid Embolic Agents
- •8.8 Detachable Balloons
- •References
- •9.1 Introduction
- •9.2 Balloons
- •9.2.1 Basics
- •9.2.7 Balloon Ratings
- •9.2.11 Balloon Catheter Design
- •9.2.13 Complications
- •9.2.14 Occlusion Balloons
- •9.2.15 Angioplasty–Pearls
- •9.3 Stents
- •9.3.4 Raw Material Form
- •9.3.5 Fabrication
- •9.3.6 Geometry
- •9.3.7 Additions
- •9.3.8 Drug-Eluting Stents
- •9.3.10.1 Arterial Indications
- •9.3.10.2 Venous Indications
- •9.3.10.3 Non-vascular Indications
- •9.3.11 Uncovered vs. Covered (PTFE) SEMS
- •9.3.12 Stent Grafts
- •References
- •10: Vascular Access
- •10.1 Introduction
- •10.2 Indications
- •10.3 Hardware
- •10.3.1 Intravenous Cannula
- •10.3.2 Puncture Needle
- •10.3.3 Arterial Access Sheath
- •10.4 Local Anesthesia
- •10.6 General Considerations Before Procedure
- •10.7 Arterial Access
- •10.7.2 Arterial Access Sites
- •10.7.2.1 Common Femoral Artery
- •10.7.2.2 High Brachial or Axillary Artery
- •10.7.2.3 Radial Artery
- •10.7.2.4 Pedal Access [3]
- •10.7.2.5 Others
- •10.8 Post-procedure Care
- •10.8.1 Manual Compression
- •10.8.2 Vascular Closure Devices (VCD)
- •10.9 Complications [5–8]
- •10.10 Venous Access
- •10.10.1 Common Femoral Vein
- •10.10.2 Internal Jugular Vein
- •10.10.3 Subclavian Vein Access
- •10.10.4 Upper Extremity Vein Access
- •10.11 Conclusion
- •References
- •11: Neurointerventions Including Aneurysm Interventions
- •11.1 Introduction
- •11.2 Neurovascular Interventions
- •11.3.1 Introduction
- •11.3.3 Clinical Presentation
- •11.3.8 Endovascular Techniques
- •11.3.9 General Technical Guidelines
- •11.3.10 Simple Coiling
- •11.3.11 Balloon-Assisted Coiling
- •11.3.12 Stent-Assisted Coiling
- •11.3.13 Flow Diverter/Braided Stents
- •11.3.14 Stent Graft
- •11.3.15 Parent Artery Occlusion
- •11.3.16 Endosaccular Devices
- •11.4.1 Pathophysiology
- •References
- •12.1 Introduction
- •12.4 Thrombectomy Techniques
- •12.5 Aspiration Technique
- •12.5.1 Stentriever Technique
- •12.6 Combination Technique
- •12.7 Balloon Guide Catheters (BGC)
- •12.8 Special Situations
- •12.8.1 Posterior Circulation Stroke
- •12.8.2 Tandem Occlusions
- •12.8.3 Intracranial Atherosclerotic Disease (ICAD)
- •12.8.4 Medium Vessel Occlusions (MeVO)
- •12.9 Complications
- •12.10 Immediate Post-procedure Care
- •References
- •13.1 Introduction
- •13.2 Brain AVMs
- •13.2.1 Introduction
- •13.2.2 Epidemiology
- •Embolic Agents
- •Embolization Techniques
- •Embolization by Copolymers: Technical Aspects
- •13.3 Intracranial Dural Arterio-Venous Fistulas (DAVFs)
- •13.4 VGAM
- •13.5 Spinal Vascular Malformations
- •13.5.1 Introduction
- •13.5.2 Anatomy
- •13.5.4.3 Spinal Epidural AVMs
- •13.5.4.4 Metameric AVMs
- •References
- •14: Other Neurointerventions
- •14.1 Introduction
- •14.2 Direct CCF
- •14.2.2 Treatment
- •14.3 Epistaxis
- •14.4 Neck Vessel Pathologies
- •14.4.2 Etiopathogenesis
- •14.4.3 Clinical Presentation
- •14.4.4 Management
- •References
- •15.1 Introduction
- •15.4.2 Embolization Agents
- •15.5 Conclusion
- •References
- •16: Carotid Artery Interventions
- •16.1 Introduction
- •16.2.1 Imaging
- •16.2.2.1 Symptomatic Carotid Artery Stenosis
- •16.2.2.2 Asymptomatic Carotid Artery Stenosis
- •16.2.3 Best Medical Therapy
- •16.3 Carotid Artery Stenting
- •16.3.1 Preprocedure
- •16.3.2 Hardware
- •16.3.2.1 Procedure
- •16.5 Complications
- •16.5.1 Early Complications
- •16.5.2 Late Complications
- •16.6 Internal Carotid Artery Stenting
- •16.7 Conclusion
- •References
- •18.1 Introduction
- •18.2 Anatomical Considerations
- •18.3 Aortic Dissection
- •18.3.1 Introduction
- •18.3.2.3 Natural History
- •18.3.2.4 Imaging Options
- •18.3.2.6 Conventional Management Plan
- •18.4 Aortic Aneurysms
- •18.4.1 Overview
- •18.4.4 Thoraco-Abdominal Aortic Aneurysm
- •18.4.5 Abdominal Aortic Aneurysm (AAA)
- •18.4.5.3 Endovascular Intervention
- •18.4.6.1 Epidemiology
- •18.4.6.3 Pathophysiology
- •18.4.7 PAU
- •18.4.7.1 Imaging
- •18.4.7.2 Prognosis
- •18.4.7.3 Management
- •18.4.8 PAU
- •18.5.1 Access Site
- •18.5.2 Procedure
- •18.5.3 Post-Procedure Care
- •18.5.4 Follow-Up Imaging
- •18.6.1 Device-Related Complications
- •18.6.2 Systemic Complications
- •18.7 Endoleaks
- •References
- •19: Vascular Thoracic Interventions
- •19.1 Introduction
- •19.2 Bronchial Artery Embolization (BAE)
- •19.2.1.3 Pre-Procedure Evaluation
- •19.2.2 Bronchoscopy
- •19.2.2.1 Technique
- •19.2.2.2 Post-Procedure Care
- •19.2.2.3 Complications
- •19.3 Pulmonary Artery Pseudoaneurysm (PAPA)/Pulmonary AVM (PAVM) Embolization
- •19.3.1 Pre-Procedure Evaluation
- •19.3.1.2 Technique
- •19.3.2 Post-Procedure Care
- •19.3.3 Complications
- •19.4.1 Pre-Procedure Evaluation
- •19.4.2 Technique
- •19.4.3 Complications
- •19.4.4 Post-Procedure Follow-Up
- •19.5 Thoracic Duct Interventions
- •19.5.1 Pre-Procedure Evaluation
- •19.5.2 Lymphangiography Technique
- •19.5.3 Thoracic Duct Embolization Technique
- •19.5.4 Complications
- •19.5.4.2 Central Lymphatic Access Complications
- •References
- •20.1 Introduction
- •20.2 Pulmonary Embolism
- •20.2.3 Pulmonary Angiography
- •20.2.3.1 Technique
- •20.2.3.2 Post-Procedure Care
- •20.2.3.3 Complications
- •20.2.4.1 Intravenous Thrombolysis
- •20.2.4.2 Catheter-Directed Thrombolysis
- •20.2.4.3 Mechanical Thrombectomy
- •Rheolytic Thrombectomy Devices
- •Aspiration Thrombectomy Devices
- •20.3.1 Clinical Manifestations [16, 17]
- •20.3.3 Radiological Findings
- •20.3.4 Endovascular Management
- •20.3.4.1 Pre-Procedure Evaluation
- •20.3.4.2 Technique
- •20.3.4.3 Post-Procedure Care
- •20.3.4.5 Current Evidence
- •20.4 Pulmonary Artery Aneurysm
- •20.4.1 Endovascular Management
- •20.4.1.1 Technique
- •References
- •21: Hepatic Arterial Interventions
- •21.1 Introduction
- •21.2 Hepatic Arterial Anatomy
- •21.2.1 Normal Celiac Anatomy
- •21.2.2 Normal Hepatic Artery Anatomy
- •21.2.3 Variant Anatomies
- •21.3.1.2 Contraindications
- •21.3.1.3 Patient Selection
- •21.3.1.4 Patient Preparation
- •21.3.1.6 Response Evaluation
- •21.3.1.7 TACE Failure
- •21.3.1.8 TACE Discontinuation
- •21.3.1.9 Complications
- •21.3.2 Transarterial Radioembolization (TARE)
- •21.3.2.2 Contraindications [24]
- •21.3.2.3 Agents Used
- •21.3.2.4 First Visit (Planning)
- •21.3.2.5 Second Visit (Microsphere Injection)
- •21.3.2.6 Complications
- •21.3.3 Transarterial Bland Embolization (TAE)
- •21.3.4 Hepatic Artery Infusion Chemotherapy (HAIC)
- •21.4.1 Neuroendocrine Liver Metastasis (NELM)
- •21.4.2 Other Liver Metastases
- •21.4.3 Intrahepatic Cholangiocarcinoma (IHCC)
- •21.4.4 Benign Liver Tumors
- •21.4.4.1 Hemangiomas
- •21.4.4.2 Focal Nodular Hyperplasia
- •21.4.4.3 Hepatocellular Adenoma
- •21.4.4.4 Polycystic Liver Disease
- •21.8 Hepatic Parenchyma Repopulation
- •References
- •22.1 Introduction
- •22.2 Transjugular Liver Biopsy (TJLB)
- •22.2.1 Patient Preparation
- •22.2.2 Procedure
- •22.2.3 Post-Procedural Care
- •22.2.4 Complications
- •22.3 Transjugular Intrahepatic Portosystemic Shunt (TIPS)
- •22.3.3 Pre-Procedural Evaluation [12–14]
- •22.3.4 Technique
- •22.3.5 Post-Procedural Care
- •22.3.6.1 Extrahepatic Portal Vein Puncture [18, 19]
- •22.3.6.2 Hepatic Artery Injury [18–20]
- •22.3.7 Discussion
- •22.4 Balloon-Occluded Retrograde Transvenous Obliteration (BRTO)
- •22.4.1 Indications and Contraindications of BRTO
- •22.4.2 Pre-Procedural Evaluation
- •22.4.3 Requirements
- •22.4.4 Sclerosants
- •22.4.5 Relevant Anatomy
- •22.4.6 Techniques
- •22.4.8 Discussion
- •22.5 Portal Vein Thrombosis (PVT)
- •22.6.2 HV/IVC Stenting
- •22.6.3 Tips/Dips
- •22.6.4 HV/IVC Thrombolysis
- •22.6.5 Discussion
- •22.7 Portal Vein Embolization
- •22.7.5 Pre-Procedural Evaluation
- •22.7.6 Techniques
- •22.7.7 Embolizing Materials
- •22.7.8 Hypertrophy Response
- •22.7.11 Discussion
- •22.8 Transjugular Kidney Biopsy (TJKB)
- •22.8.1 Indications
- •22.8.2 Rationale
- •22.8.3 Pre-Procedural Workup
- •22.8.4 Techniques
- •22.9 IVC Filter
- •22.9.2 Patient Preparation
- •22.9.4 Procedure
- •22.9.5 Complications [98, 102]
- •References
- •23.1 Introduction
- •23.2 Anatomy
- •23.2.1 Arterial Anatomy
- •23.2.2 Venous Anatomy
- •23.3 Arterial Interventions
- •23.3.1 Renovascular Hypertension
- •23.3.2 Atherosclerotic Renal Artery Stenosis
- •23.3.3 Non-atherosclerotic RAS
- •23.3.4 Takayasu Arteritis (TA)
- •23.3.5 Fibromuscular Dysplasia (FMD)
- •23.5.2.1 Preprocedural Evaluation
- •23.5.2.2 Preprocedural Instructions
- •23.5.2.3 Procedure
- •23.5.2.4 Angiography
- •23.5.2.5 Balloon Angioplasty
- •23.5.2.6 Cutting Balloon Angioplasty
- •23.5.2.7 Stenting
- •23.5.2.9 Post-Procedural Care
- •23.5.2.10 Complications
- •23.6.1 Procedure
- •23.7 Renal Artery Aneurysms (RAAs)
- •23.8.1 Etiology
- •23.8.2 Clinical Presentation
- •23.8.3 Endovascular Management
- •23.9.1 Angiomyolipoma
- •23.9.2 Renal Cell Carcinoma (RCC)
- •23.10 Venous Interventions
- •23.10.1 Nutcracker Syndrome (NCS)
- •23.10.1.1 Diagnosis
- •23.10.1.2 Management
- •23.10.1.3 Endovascular Management
- •23.10.1.4 Procedure
- •23.10.1.5 Complications
- •23.10.2 Renal Vein Thrombosis
- •23.10.2.1 Clinical Presentation
- •23.10.2.2 Management
- •23.10.2.4 Procedure
- •References
- •24.1 Introduction
- •24.2 Relevant Anatomy
- •24.3 Mesenteric Ischemia
- •24.3.1 Clinical Features
- •24.3.2 Imaging
- •24.3.3 Treatment
- •24.3.3.2 Intra-Arterial Thrombolysis
- •24.3.3.3 Mechanical Thrombectomy
- •24.4 Gastrointestinal Hemorrhage
- •24.4.1 Clinical Features
- •24.4.2 Endoscopy
- •24.4.3 Imaging
- •24.4.4.2 Complications
- •24.5 Bariatric Embolization
- •24.5.1 Technique
- •References
- •25.1 Introduction
- •25.2 Uterine Artery Embolization (UAE)
- •25.2.1 Indications [1, 2]
- •25.2.2 Contraindications [1, 2]
- •25.2.3 Relevant Vascular Anatomy [2]
- •25.2.4 Preprocedural Evaluation
- •25.2.5 Technique
- •25.2.6 Post-Procedural Care
- •25.2.7 Complications
- •25.2.8 Outcome
- •25.3 Prostatic Artery Embolization (PAE)
- •25.3.1 Rationale Behind PAE
- •25.3.2 Indications [17, 18]
- •25.3.3 Contraindications [18]
- •25.3.4 Preprocedural Evaluation [17]
- •25.3.5 Clinical Assessment
- •25.3.7 Imaging
- •25.3.7.1 Ultrasonography (USG)
- •25.3.7.2 Computed Tomography (CT)
- •25.3.7.3 Magnetic Resonance Imaging (MRI)
- •25.3.9 Relevant Vascular Anatomy
- •25.3.10 Technique
- •25.3.11 Complications
- •25.3.12 Post-Procedural Follow-Up
- •25.3.13 Outcome
- •25.4 Varicocele Embolization
- •25.4.1 Indications [22, 26]
- •25.4.4 Relevant Vascular Anatomy
- •25.4.5 Preprocedural Evaluation [23, 25]
- •25.4.6 Technique
- •25.4.6.2 Venous Access [25, 27, 28]
- •25.4.6.3 Venography [25, 27]
- •25.4.6.4 Embolization [25, 27, 29–31]
- •25.4.7 Post-Procedural Care
- •25.4.8 Complications
- •25.4.9 Outcome
- •25.5 Pelvic Congestion Syndrome
- •25.5.1 Indication [38]
- •25.5.2 Contraindications [38]
- •25.5.3 Preprocedural Evaluation
- •25.5.4 Relevant Vascular Anatomy
- •25.5.5 Technique
- •25.5.6 Post-Procedural Care
- •25.5.7 Complications
- •25.5.8 Outcome
- •25.6 Penile Angiography
- •25.6.1 Penile Vascular Anatomy
- •25.6.2 Technique
- •25.6.3 Complications
- •25.6.4 Outcome
- •References
- •26.2 Peripheral Arterial Disease (PAD)
- •26.2.1 Introduction
- •26.2.5.1 Non-imaging/Functional Modalities
- •26.2.5.2 Imaging Evaluation
- •26.2.6 Management Strategies
- •26.2.6.3 Revascularization Strategies
- •26.3.2 Preprocedural Evaluation
- •26.3.5 Complications
- •26.3.6 Stenting
- •26.3.7 Post-Procedure
- •26.5 Recent Advances
- •26.5.2 Drug-Eluting Technology
- •26.5.3 Bioresorbable Stents
- •26.5.6 Pedal Arch Revascularization
- •26.5.7 Percutaneous Deep Vein Arterialization (DVA)
- •26.6 Acute Limb Ischemia
- •26.7 Popliteal Artery Entrapment Syndrome (PAES)
- •26.8 Genicular Artery Embolization (GAE)
- •References
- •27.1 Introduction
- •27.2 Relevant Anatomy
- •27.3 Varicose Veins
- •27.3.1 Clinical Evaluation
- •27.3.2 Physical Examination
- •27.3.3 Sonological Evaluation
- •27.3.3.1 Duplex Sonographical Evaluation
- •27.3.5.1 Thermal Ablation
- •Endovenous Laser Ablation
- •Radiofrequency Ablation
- •Endovenous Steam Ablation
- •27.3.5.2 Non-thermal Ablative Methods
- •Foam Sclerotherapy
- •Cyanoacrylate Closure (CAC)
- •Mechanochemical Ablation (MOCA)
- •Cryosclerosis
- •27.4 Deep Vein Thrombosis
- •27.4.1 Diagnosis
- •27.4.1.1 Pre-Test Probability
- •27.4.1.2 D-Dimer Assessment
- •27.4.1.3 Radiological Evaluation
- •27.4.5.1 Catheter-Directed Thrombolysis
- •Single-Session (Second-Generation) Pharmacomechanical Catheter-Directed Thrombolysis
- •27.5.1 Pulmonary Embolism (PE)
- •27.5.2 Clinical Features
- •27.5.3 Imaging Evaluation
- •27.5.4 Management
- •27.5.5 Endovascular Techniques
- •References
- •28.1 Introduction
- •28.3.2 Imaging Evaluation
- •28.4 Endovascular Management
- •28.4.1 Nonmature Fistulas
- •28.4.3 Acute Thrombosis
- •28.5 Central Venous Stenosis
- •28.7 Pseudoaneurysm
- •References
- •29.1 Introduction
- •29.2 Low-Flow Vascular Malformations
- •29.2.1.1 Pre-procedural Requirements
- •29.2.1.2 Procedure
- •29.2.1.3 Post-procedure Care
- •Ethanol
- •Detergent Sclerosant
- •Bleomycin
- •Doxycycline
- •OK-432 (Picibanil)
- •29.2.1.5 Complications
- •29.3 High-Flow Vascular Malformations
- •29.3.1.1 Pre-procedure Evaluation
- •29.3.1.2 Technique
- •29.3.1.3 Complications
- •29.4 Fibro-Adipose Vascular Anomaly (FAVA)
- •29.5 Conclusion
- •References
- •30.1 Introduction
- •30.2 Splenic Injuries
- •30.2.2 Technique
- •30.2.3 Patient Preparation
- •30.2.4 Procedure
- •30.2.5 Embolizing Agent
- •30.2.6 Post-procedural Care
- •30.2.7 Complication
- •30.3 Hepatic Injury
- •30.3.1 Background
- •30.3.2 Indication
- •30.3.3 Technique
- •30.3.4 Patient Preparation
- •30.3.6 Embolization Agent
- •30.3.7 Post-procedural Care
- •30.3.8 Complications
- •30.4 Peripheral Vascular Injuries (PVI)
- •30.4.1 Background
- •30.4.2 Indication
- •30.4.3 Patient Preparation
- •30.4.5 Embolization Agent
- •30.4.6 Post-procedural Care
- •30.4.7 Complications
- •30.5 Pelvic Trauma
- •30.5.1 Background
- •30.5.2 Indication
- •30.5.3 Technique
- •30.5.4 Patient Preparation
- •30.5.6 Embolization Agent
- •30.5.7 Complications
- •30.6 Maxillofacial Injury (MFI)
- •30.6.1 Background
- •30.6.2 Indication
- •30.6.3 Technique
- •30.6.4 Patient Preparation
- •30.6.6 Embolization Agent
- •30.6.7 Complications
- •30.7 Conclusion
- •References
- •31.1 Introduction
- •31.2 Inferior Petrosal Sinus Sampling
- •31.2.2 Cushing Syndrome (CS)
- •31.2.4 Pre-Procedure Instructions
- •31.2.5 Minimum Hardware Required
- •31.2.7 Result Interpretation
- •31.3 Adrenal Venous Sampling
- •31.3.1 Adrenal Gland Anatomy
- •31.3.2 Vascular Anatomy
- •31.3.3 Primary Hyperaldosteronism
- •31.3.5 Pre-Procedure Instructions
- •31.3.6 Minimum Hardware Required
- •31.3.8 Result Interpretation
- •31.4 Pancreatic Venous Sampling
- •31.4.1 Pancreatic Venous Anatomy
- •31.4.3 Pancreatic Neuroendocrine Tumors
- •31.4.4 Pre-Procedure Instructions
- •31.4.5 Minimum Hardware Required
- •31.4.7 Result Interpretation
- •31.5 Ovarian Venous Sampling
- •31.5.1 Anatomy
- •31.5.2 Hyperandrogenism
- •31.5.3 Pre-Procedure Instructions
- •31.5.4 Minimum Hardware Required
- •31.5.6 Result Interpretation
- •31.6.1 Anatomy
- •31.6.2 Pre-Procedure Instructions
- •31.6.3 Minimum Hardware Required
- •31.6.4 Procedure
- •31.6.5 Result Interpretation
- •31.7.1 Pre-Procedure Instructions
- •31.7.2 Minimum Hardware Required
- •31.7.3 Procedure
- •31.7.4 Result Interpretation
- •31.8 Conclusion
- •References
- •32.1 Introduction
- •32.2 Priapism
- •32.3 Erectile Dysfunction
- •32.3.2 Epidemiology
- •32.3.3 Aetiology
- •32.3.4 Pathophysiology
- •32.3.6 Imaging
- •32.3.7 Relevant Anatomy
- •32.3.7.2 Venous Anatomy
- •32.3.8 Penile Doppler
- •32.4.1 Indications
- •32.4.2 Contraindications
- •32.4.3 Equipment
- •32.4.4 Technical Aspects
- •32.4.5 Procedure Timing
- •32.4.6 Outcomes
- •32.4.7 Complications
- •32.4.8 Post-Procedural Care
- •32.5.1 Indications
- •32.5.2 Contraindications
- •32.5.3 Equipment
- •32.5.4 Technical Aspects
- •32.5.5 Outcomes
- •32.5.6 Complications
- •32.6.1 Indications
- •32.6.2 Contraindications
- •32.6.3 Equipment
- •32.6.4 Procedural Planning
- •32.6.5 Technical Aspects
- •32.6.6 Outcomes
- •32.6.7 Complications
- •32.7 Conclusion
- •References
- •33: Image-Guided Biopsy
- •33.1 Introduction
- •33.2 Biopsy Devices
- •33.3 Pre-Procedural Evaluation
- •33.4 Ultrasound-Guided Biopsy
- •33.5 CT-Guided Biopsy
- •33.6 MRI-Guided Biopsy
- •33.7.2 Breast
- •33.7.3 Lung
- •33.7.4 Mediastinum
- •33.7.5 Liver
- •33.7.6 Gallbladder
- •33.7.7 Spleen
- •33.7.8 Bowel
- •33.7.9 Retroperitoneum
- •33.7.11 Pelvis
- •33.7.12 Spine
- •33.7.13 Extremities
- •33.8 Conclusion
- •References
- •34: Image-Guided Drainage Procedures
- •34.1 Introduction
- •34.2 Etiology
- •34.4 Contraindications
- •34.5 Imaging Modalities
- •34.6 Pre-Procedure Evaluation
- •34.8 Post-Procedure Care
- •34.9 Complications
- •34.10.1 Postoperative Fluid Collection
- •34.10.2 Ascites
- •34.10.3 Liver Abscess
- •34.10.4 Peripancreatic Fluid Collection
- •34.10.5 Splenic Abscess
- •34.10.6 Appendicitis
- •34.10.8 Renal Abscess
- •34.10.9 Pelvic Abscess
- •34.10.10 Prostatic Abscess
- •34.11 Summary
- •References
- •35: Ablation Techniques
- •35.1 Introduction
- •35.2 Chemical Ablation
- •35.3 Thermal Ablation Techniques
- •35.3.1 Radiofrequency Ablation
- •35.3.2 Microwave Ablation
- •35.3.3 Cryoablation
- •35.3.4 High-Intensity Focused Ultrasound
- •35.3.5 Laser Ablation
- •35.4 Non-thermal Ablation Techniques
- •35.4.1 Irreversible Electroporation
- •35.6.1 Liver
- •35.6.2 Kidney
- •35.6.3 Lung
- •35.6.4 Breast
- •35.6.5 Thyroid
- •35.6.6 Musculoskeletal System
- •35.6.7 Nerve Ablation
- •35.7 Conclusion
- •References
- •36.1 Introduction
- •36.3 CT-Guided Biopsy
- •36.3.2 Complications
- •36.3.3 Technical Approaches
- •36.3.4.3 Paramaxillary Approach
- •36.3.4.4 Submastoid Approach
- •36.3.4.5 Transoral Approach
- •36.3.5.1 Anterolateral Approach
- •36.3.5.2 Posterolateral Approach
- •36.3.5.3 Posterior Approach
- •36.4 Ultrasound-Guided Biopsy/FNAC
- •36.4.2.5 Carotid Space
- •36.7 Conclusion
- •References
- •37: Nonvascular Thoracic Interventions
- •37.1 Introduction
- •37.2 Thoracic Drainage Procedures
- •37.2.1 Pre-Procedure Evaluation
- •37.2.2 Imaging
- •37.3 Thoracocentesis
- •37.3.1 Indications
- •37.3.2 Relative Contraindications
- •37.3.4 Technique
- •37.4.1 Indications
- •37.4.2 Contraindications
- •37.4.3 Drain Size
- •37.4.5 Post-Insertion Care
- •37.5 Intrapleural Fibrinolytic Therapy
- •37.5.1 Catheter Removal
- •37.6 Practice Points
- •37.7 Indwelling Pleural Catheter Insertion
- •37.7.1 Duration
- •37.8.1 Procedure
- •37.8.2 Trocar Drainage
- •37.8.3 Seldinger Technique
- •37.8.4 Post-Procedure Follow-Up
- •37.8.5 Removal
- •37.9 Complications
- •37.9.1 Thoracentesis Related
- •37.9.2 Drainage Chest Tube Related
- •37.9.3 IPC Related
- •37.11.1 Pre-Procedure Evaluation
- •37.11.3 Technique
- •37.11.4 Post-Procedure Care
- •37.11.5 Complications [11, 14, 15]
- •37.12.1 Indications [11, 19]
- •37.12.2 Contraindications [11, 19]
- •37.12.3 Technique
- •37.12.4 Post-Procedure Care
- •37.12.5 Complications
- •References
- •38.1 Introduction
- •38.2 Biliary Anatomy
- •38.3 Biliary Obstruction
- •38.4 Percutaneous Transhepatic Biliary Drainage
- •38.4.2 Indications
- •38.4.3 Contraindications
- •38.4.4 Technique
- •38.4.5 Post-Procedure Care
- •38.4.6 Complications
- •38.5 Biliary Stenting
- •38.5.1 Indications
- •38.5.2 Technique
- •38.7 Intraluminal Procedures Through Percutaneous Biliary Access
- •38.7.1 Endobiliary Biopsy
- •38.7.2 Intraluminal Brachytherapy
- •38.7.3 Gallstone Extraction
- •38.8 Percutaneous Cholecystostomy
- •38.8.1 Indications
- •38.8.2 Technique
- •References
- •39.1 Introduction
- •39.2 Percutaneous Gastrostomy
- •39.2.1 Indications
- •39.2.2 Contraindications
- •39.2.3 Pre-procedural Work-Up
- •39.2.4 Technique
- •39.2.6 Pull-Type PRG
- •39.3 Percutaneous Jejunostomy
- •39.3.1 Indications
- •39.3.2 Technique
- •39.4 Percutaneous Cecostomy
- •39.4.1 Indications
- •39.4.2 Technique
- •39.4.3 Post-procedure Care
- •39.5.1 Technique
- •39.6.1 Technique
- •39.6.3 Malignant Tracheoesophageal Fistula Stenting
- •39.6.5 Complications
- •References
- •40.1 Introduction
- •40.2 Percutaneous Nephrostomy (PCN)
- •40.2.5 Post-procedure Care
- •40.4.3 Ureteroarterial Fistula
- •References
- •41.1 Introduction
- •41.2 Fallopian Tube Recanalization (FTR)
- •41.2.1 Technique
- •41.2.2 Complications
- •41.2.3 Results
- •41.3 Amniocentesis
- •41.3.1 Indications
- •41.3.2 Contraindications [7]
- •41.3.4 Technique
- •41.3.5 Complications
- •41.4 Chorionic Villous Sampling (CVS)
- •41.4.1 Indications
- •41.4.2 Contraindications
- •41.4.4 Technique
- •41.4.5 Complications
- •41.5.1 Indications [15–19]
- •41.5.2 Contraindications [20–23]
- •41.5.3 Technique [15, 23]
- •41.5.4 Complications [15, 23, 24]
- •41.5.5 Outcome [23]
- •41.6.1 Technique
- •41.6.2 Complications
- •41.7.1 Contraindications
- •41.7.2 Technique
- •41.7.3 Complications
- •41.8.1 Technique
- •41.8.2 Complications
- •41.9.1 Technique
- •41.9.2 Complications [28, 44, 45]
- •References
- •42.1 Introduction
- •42.2 Breast Biopsy
- •42.2.2 USG-Guided Biopsy [1, 2]
- •42.2.3 MG-Guided Biopsy [3, 4]
- •42.2.4 MRI-Guided Biopsy [1, 11–13]
- •42.2.5 Vacuum-Assisted Biopsy [1, 4, 14, 15, 16]

30 Vascular Interventions inTrauma
379
Table 30.2 The hard and soft
signs of peripheral arterial injury
HARD SIGN:
H : Expanding hematoma
A : Arterial pulsatile bleeding
R : Bruit and Thrill
D : Do not feel distal pulse
Evidence of regional ischemia: “6P”
(Pallor, paresthesia, palsy, pain, pulseless
ness, poikilothermia)
Table: Clinical sign of Arterial Injury :
ab
SOFT SIGN:
Non Expanding hematoma
Nonpulsatile bleeding
Proximity Injury
Unexplained hypotension
Injury to anatomically
related nerve
c
d
Fig. 30.5 Stent graft vascular reconstruction in a patient with a right
lower limb hemorrhagic injury involving a non-sacricable artery (popliteal artery) following blunt trauma
The coronal reformatted and volume rendering (VRT) CT angiography
images of the bilateral lower limbs (a, b) depict a large, well-dened
pseudoaneurysm arising from the distal supercial femoral artery. The
selective angiogram of the left common femoral artery (c, d) shows a
large contrast blob (pseudoaneurysm) in the distal part of the supercial
femoral artery. Post-vascular reconstruction (stent graft) images (d)
demonstrate complete occlusion of the pseudoaneurysm with preserved
forward ow

380
abc
R. Yadav et al.
Fig. 30.6 Coil embolization of a patient with a right lower limb hemorrhagic injury involving a sacricable artery following a road trafc
accident. Selective angiogram of the left common femoral artery (a, b)
reveals a large contrast blob (pseudoaneurysm) in the distal part of the
artery-related injuries. USG guidance when available is preferable, especially in cases with hemodynamic instability.
Heparin and heparin saline should be withheld in the setting
of acute trauma. A selective angiogram is performed using a
catheter like Picard or vertebral catheter. Cross-over sheath
or long sheath is used when stent grafting is used, to stabilize
the hardware if the injury is located distally. The long sheath
is used for upper limb arterial injury, to stabilize the hardware and place stent graft.
Superselective catheterization of the targeted artery is
usually done with a microcatheter. Depending upon the
imaging and angiographic injury pattern, embolization or
vascular reconstruction is done. In case of focal active bleeding or pseudoaneurysm or large AV stula, with an appropriate landing zone in the sacricable artery, proximal
embolization is done using permanent embolizing agents
like coils or coils with glue or plug, aimed to complete
blockage of the forward ow of the targeted artery. In case of
focal active bleeding or pseudoaneurysm or AV stula, with
an appropriate landing zone in the main artery or nonsacricable artery, vascular reconstruction is done using a
stent graft, aimed to salvage the limb and vessels, to achieve
hemostasis.
deep femoral artery. Post-coil embolization images (c) demonstrate
complete coil occlusion as close as possible to the culprit vessels without any further extravasation
30.4.5 Embolization Agent
The endovascular intervention aims to achieve hemostasis by
occluding the injured artery or performing vascular reconstruction. The choice of endovascular treatment depends on
the type of vessel, the nature of the injury, and the size of the
artery. In case of injury to the main artery, vascular reconstruction using a covered stent graft is indicated for hemodynamically stable patients, to preserve patency for forward
ow [23]. Conversely, in case of injury to the small to
medium-sized or an end artery, feasible to sacrice or
occlude the vessel, the preferred treatment is using an embolization agent or vascular plug [24]. Embolizing agents such
as coil, glue, or a combination thereof, as well as, vascular
plugs are selected depending on the specic characteristics
of the landing zone.
30.4.6 Post-procedural Care
The immediate post-procedural care includes close observation of vitals and access site care at least for 24hours to
prevent complications. A routine post-procedural ultraso-

30 Vascular Interventions inTrauma
381
nography (USG) is recommended on the next day to look for
residual pseudoaneurysm and to assess the puncture site
complication. Low molecular weight heparin is advised for
the next 24hours then tapered and shifted to oral antiplatelets for at least 6months in a trauma setting to prevent stent
thombosis.
30.4.7 Complications
Local site complications or rebleeding after embolization,
reperfusion injury, embolization-related complications (coil
migration, vessel perforation), and infection [25]. However,
these complications are rare, and the benets of the embolization procedure often outweigh the risks [26].
30.5 Pelvic Trauma
30.5.1 Background
Traumatic pelvic injuries are associated with signicant
morbidity and mortality, with a mortality rate of ~16%,
which is the highest mortality rate of any skeletal injury [27].
Hemorrhage is the most common treatable cause of death in
pelvic trauma and the source of the massive pelvic bleed are
rupture of the vein or venous plexus, bleeding from cancellous bone, rupture of arteries, or a combination [28]. The
majority are the result of venous injury, which accounts for
80–90% of bleeding, while arterial injury accounts for
10–20% [29]. Hence timely identication and control of
bleeding is pivotal in reducing mortality.
The rst line of management is the pelvic xation with
binder or pelvic packing, and after that further assessment
depends on the hemodynamic status of the patient.
Intervention radiology serves at the forefront of pelvic
trauma management and has several advantages over surgical intervention. First, the relatively conned space makes
surgical access difcult; second, surgery may release the
tamponade effect, which can be catastrophic; and nally, the
rich vasculature and collaterals network may make arterial
ligation difcult [30].
30.5.2 Indication
screening to exclude pelvic hemorrhage, predict the type
(arterial vs venous), and site of injury, and tailor the embolization accordingly [31]. The ideal candidates for angioembolization are pelvic trauma with the following:
1. CT evidence of focal arterial or diffuse arterial microbleeds, in patients with stable hemodynamics.
2. In transient responders, where vitals remain unstable
even after >1.5L of IV uid or after pelvic packing, and
represents slow and continuous bleeding, TAE is warranted even without preceding CT.
3. Ongoing bleeding despite the surgical intervention or pelvic packing, even though the patient is hemodynamically
unstable.
4. Delayed vascular complications like pseudoaneurysm
formation in follow-up cases, managed conservatively
initially.
30.5.3 Technique
Includes patient preparation, procedure steps, approaches,
the embolizing agent used, and post-procedural care. The
planning is based on ndings on CECT ndings, which not
only delineate the characteristics of the injury but also furnish a roadmap for localizing the site of hemorrhage and any
concomitant anatomical variations of the artery, particularly
“corona mortis,” to circumvent unnecessary time expenditure in identifying or localizing vessel origins.
30.5.4 Patient Preparation
Patient preparation is an essential aspect of trauma care and
encompasses ongoing resuscitation, securing the airway,
ensuring effective breathing, and maintaining circulation.
Establishing and maintaining adequate IV access with a
large bore IV line is crucial to facilitate the administration of
life-saving uids and medications. In cases of trauma, basic
laboratory parameters are not essential prerequisites, as the
primary goal is to achieve hemostasis and stabilize the
patient rapidly. In instances of pelvic trauma, it is important
to ensure that the urinary bladder is empty, as a full bladder
may obscure the eld of view and complicate assessment.
According to ATLS guidelines, the assessment starts with the
pelvic binder, pelvic packing, and after fracture stabilization
in case of pelvic trauma, and then is followed by computed
tomography angiography (CECT) torso, which is also an
independent predictor for survival. CT is an excellent tool for
30.5.5 Procedure (Fig.30.7)
The arterial access is gained via puncturing the common
femoral artery. The ipsilateral or contralateral site can be
used for a puncture, depending upon the choice of IR radi-

382
Fig. 30.7 Bilateral internal
iliac artery angioembolization
in a transient responder
patient with multiple pelvic
fractures following a road
trafc accident
Selective angiogram of the right
internal iliac artery (a, b, c)
demonstrates multifocal contrast
blush consistent with extravasation from the superior, inferior
gluteal, and internal pudendal
artery branches of the anterior
division of the internal iliac
artery on either side. Post-coil
embolization images (d) show
complete occlusion of the
anterior division of the bilateral
internal iliac artery using Nester
coils without further
extravasations
R. Yadav et al.
a
b
cd
ologist. However, the contralateral site or site opposite of
pelvic fracture is used for puncture, for ease, and to allow
more secure seating of the catheter. USG guidance when
available is preferable, especially in cases with hemodynamic instability and given pelvic binder, that can be cut
away to create a sufcient window. Heparin and hep saline
should be withheld in the setting of acute trauma. A selective
external iliac and common femoral artery angiogram is performed using appropriately shaped catheters like Picard or
Simmons reverse curve (SIM1) depending on the angle of
aortic bifurcation, and usually contralateral angiogram is
preferred (if suspected), followed by ipsilateral.
Superselective catheterization of the targeted artery is done
with a microcatheter. The angiographic evidence of arterial
injury is spasm, truncation of the artery (cut-off sign), extravasation, pseudoaneurysm, and AV stula formation. Oblique
view and delayed images are taken until venous return.
Always look for multiple bleeder sites and the primary aim is
to manage the massive bleeder site rst and embolize as
selectively as time permits. A tailored approach is essential
for optimizing patient outcomes and minimizing potential
complications. Selective embolization is the preferred
method if patient hemodynamics are stable as it minimizes
damage to the noninjured vessel, but this can be technically
challenging in hemodynamic instability, vasoconstriction,
and vascular spasm. Nonselective embolization where gelfoam is delivered to the main internal iliac artery or its divisions, either unilaterally or bilaterally depending on the
severity of fracture and bleeding. Nonselective embolization
is the preferred method when the patient is unstable and time
is of the essence and in the presence of multiple arterial
bleeding points and when active extravasation is not demonstrated, but there is a high index of suspicion that arterial
bleeding is the cause of ongoing instability.
30.5.6 Embolization Agent
The selection of an embolization material is meticulously
tailored to the specic requirements of each individual case,
taking into consideration the imaging characteristics of the
injury and the preferences of the interventional radiologist
[32]. Embolization can be performed in a selective or nonselective manner. In the context of pelvic trauma, embolization
should be as selective as time permits. However, bilateral
nonselective embolization of the internal iliac artery is justied in cases of transient responders. The embolizing agent of
choice for nonselective embolization or in the presence of

30 Vascular Interventions inTrauma
383
diffuse bleeding is gelfoam (in the form of a slurry), whereas
coils are utilized for selective focal arterial bleeds [33].
Furthermore, other embolization agents that can be utilized
include glue and onyx [34].
30.5.7 Complications
Complications are rare and associated with access site complications or embolization-related issues, with an incidence
of 5–6% [35]. Short-term complications commonly observed
with nonselective embolization encompass infection, gluteal
necrosis, skin breakdown, bladder necrosis, urinary retention, and rhabdomyolysis. Long-term complications, while
very rare, may include sciatic or sacral plexus palsies, gluteal
claudication, or impotence [36].
30.6 Maxillofacial Injury (MFI)
30.6.1 Background
Blunt trauma is the predominant mechanism responsible for
maxillofacial injuries, often resulting in copious oronasal
bleeding. Among traumatic maxillofacial injuries, the internal maxillary artery emerges as the most frequently implicated artery, followed by the facial and lingual arteries [37].
The primary objective in managing maxillofacial injuries
lies in ensuring the patency of the airway, as bleeding into
the oral cavity, particularly posterior epistaxis, can swiftly
compromise respiratory function. Therefore, securing the
airway constitutes the foremost priority in such cases. The
second is to halt the hemorrhage through various methods
such as nasal packing with gauze, Foley’s catheter insertion,
fracture reduction, arterial ligation, and selective angioembolization. Conventional approaches like surgical arterial
ligation may be ineffective in cases involving injury to multiple vessels and a well-established collateral vascular network in complex facial trauma scenarios. Furthermore, the
nonselective nature and associated high morbidity of surgical ligation of external carotid arteries underscore the necessity for alternative strategies to achieve effective and safe
hemostasis [38].
Transarterial embolization (TAE) emerges as a highly
efcient and clinically signicant treatment modality for
cases of intractable refractory hemorrhage associated with
maxillofacial injuries [39]. This minimally invasive technique offers precise and targeted occlusion of bleeding vessels using embolic agents, thereby overcoming the limitations
of conventional surgical approaches. The conservative treatment consisting of packing of the nares, compression, and
blood transfusion should always precede TAE as the primary
protocol.
30.6.2 Indication
The decision to pursue IR angioembolization in the maxillofacial injury is made in collaboration with a multidisciplinary team, including interventional radiologists, trauma
surgeons, and maxillofacial specialists, taking into account
the specic clinical presentation and imaging ndings of the
individual patient. The indications are:
1. Refractory or intractable bleeding (microhemorrhages)
from Lefort or non-Lefort maxillofacial injury evident on
CTA.
2. In transient responders, where bleeding persists or vitals
remain unstable even after conservative treatment or the
need for continued blood product replacement exceeding
1500mL and a systolic blood pressure<90mm Hg, TAE
intervention is warranted even without CT or before other
surgical interventions.
3. Presence of CT evidence of direct focal vascular injury
(pseudoaneurysm, AV stula, or focal contrast extravasation in MFI with stable hemodynamic status.
4. Delayed vascular injury like pseudoaneurysm in the follow- up cases of MFI, managed earlier nonoperatively.
30.6.3 Technique
Includes patient preparation, procedure steps, approaches,
the embolizing agent used, and post-procedural care. The
planning is based on CT angiography ndings, which not
only delineate the characteristics of the injury but also furnish a roadmap for localizing the site of hemorrhage and any
concomitant injury.
30.6.4 Patient Preparation
Patient preparation is an essential aspect of trauma care and
encompasses ongoing resuscitation, securing the airway,
ensuring effective breathing, and maintaining circulation.
Establishing and maintaining adequate IV access with a
large bore IV line is crucial to facilitate the administration of
life-saving uids and medications. In cases of trauma, basic
laboratory parameters are not essential prerequisites, as the
primary goal is to achieve hemostasis and stabilize the
patient rapidly.
30.6.5 Procedure (Figs.30.8 and30.9)
The arterial access is gained via puncturing the common
femoral artery. After getting arterial access the carotid arteries are cannulated using a 4F or 5F diagnostic catheter

384
R. Yadav et al.
a
c
Fig. 30.8 Gelfoam angioembolization in a patient with multiple maxillofacial injuries following a road trafc accident, experiencing uctuating blood pressure and persistent tachycardia (transient responder),
despite nasal packing, Foley catheter placement, and blood transfusion
Axial (a) with sagittal reformatted (b) contrast- enhanced CT images of
the face reveal extensive maxillofacial injuries with Foley bulbs in the
nasopharynx
b
d
Selective angiogram of the right maxillary artery (c) shows contrast
blush (extravasation) from the facial branch of the right internal maxillary artery in the distalmost part, which could not be reached due to the
small caliber of the vessel. Therefore, gelfoam embolization was performed. Post-coil embolization images (d) demonstrate complete
occlusion without further extravasation
(VERT slip-cath or Picard), and selective angiograms of
common carotid arteries (CCAs), internal carotid arteries
(ICAs), and external carotid artery (ECAs) are obtained. If
the bleeding is from a single nostril or predominantly from
one side, the ipsilateral arteries should be interrogated rst.
Initially, angiography of ICAs is done to rule out an ICA
source of bleed, to see the supply of ophthalmic artery (choroidal blush), and to rule out communication between
branches of ICA and ECAs. In such cases, surgery is preferred over embolization due to the higher risk of complications from accidental nontarget embolization. Selective
angiography of ECAs is then done to look for the source of
the bleed and to rule out potentially dangerous collaterals,
followed by super selective cannulation of ECA branches
using microcatheters in selective cases with active bleeding
in distal branches or with tortuous anatomy of the parent vessel. The most commonly seen angiographic patterns of arterial injury in MFI are active contrast extravasation or micro
blushes from branches of the internal maxillary artery. The
most commonly injured artery is the internal maxillary artery
followed by the facial, lingual, and supercial temporal
artery. Gelfoam pledgets and bered coils are used as embolizing agents [40]. Gelfoam pledget/slurry is the most commonly used agent. In cases when angiography does not
reveal any active contrast leak, the internal maxillary arteries
on either side can be empirically embolized using gelfoam
injection. To avoid nontarget embolization the catheter tip is
placed distal to meningeal and deep temporal branches.
30.6.6 Embolization Agent
The selection of an embolization material is meticulously
tailored to the specic requirements of each case, considering the imaging characteristics of the injury. Gelfoam and
coil are commonly used embolizing agents for oronasal
bleeding, with gelfoam being the most frequently used.
Gelfoam is preferred in cases where there is diffuse blush
from small distal vessels on angiography or when superselective catheterization or reaching the site of active contrast
extravasation by microcatheter is not feasible [41]. In
selected cases, gelfoam embolization may be performed
prophylactically to reduce the perfusion pressure to manage
venous bleeding [42]. Before gelfoam injection, it is essential to carefully assess and rule out the communicating
channels between the branches of the external and internal
carotid arteries. In cases where communicating channels are
evident on angiography, coils are preferred over gelfoam to

bc
30 Vascular Interventions inTrauma
a
385
de
Fig. 30.9 Coil angioembolization in a patient with maxillofacial injuries following a road trafc accident
The sagittal reformatted (a) and volume- rendering (VRT) (b) contrastenhanced CT images of the face reveal a left hemimandible fracture
with a pseudoaneurysm arising from the supercial temporal artery.
The selective angiogram of the right external carotid artery (c) shows a
contrast blob (pseudoaneurysm) from a branch of the left supercial
temporal artery. Post-coil embolization images (d, e, f) demonstrate
complete proximal coil occlusion without further extravasation
f
mitigate the risk of nontarget embolization. Coils are also a
preferred choice for the presence of focal active contrast
extravasation observed on angiography and when superselective catheterization allows reaching near the site using a
microcatheter. A combined embolization approach using
both gelfoam and coil, known as the sandwich technique,
can be done to reinforce the embolization and achieve
immediate hemostasis [43].
30.6.7 Complications
Complications are rare and primarily associated with access
sites, as well as complications secondary to embolizationrelated issues, such as nontarget embolization and local ischemic changes or soft tissue swelling [44]. Other uncommon
complications include cerebrovascular accidents, blindness,
lip-tongue necrosis, facial nerve palsy, and trismus [45].

386
R. Yadav et al.
30.7 Conclusion
Hemodynamically stable patients with splenic and hepatic
injuries are the candidates for embolization. The choice of
proximal vs distal coil embolization relies on the imaging
pattern of injury and the aim to achieve. Endovascular management is used only in hemorrhagic type of peripheral arterial injury. A multidisciplinary approach is preferred for
managing pelvic, head, and neck trauma patients with arterial injury.
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Vascular Interventions
inEndocrinopathies
DevasenathipathyKandasamy andKavirajanKabilan
31
Key Messages
1. Venous sampling plays an important role in management
wherever the ndings on diagnostic modalities are
equivocal.
2. In certain situations, they are considered the gold
standard.
3. Bilateral inferior petrosal sinus sampling and adrenal
vein sampling are the most commonly performed
procedures.
4. Inferior petrosal sinus sampling can reliably differentiate
the central source from the ectopic source in patients with
ACTH-dependent Cushing’s disease.
5. Adrenal vein sampling can reliably differentiate unilateral from bilateral secretion in patients with primary
hyperaldosteronism.
6. Selective arterial calcium stimulation test (SACST) can
help in localizing the source of insulin in patients with
hypoglycemic hyperinsulinoma where diagnostic imaging is either not helpful or equivocal.
7. Whole-body venous sampling is generally performed in
patients with tumor-induced osteomalacia where diagnostic imaging fails to localize the source with certainty.
8. The venous sampling procedures are safe and well tolerated by the patients.
9. Meticulous planning of these procedures is equally
important to the procedure itself.
The interventional radiologist should be well versed in
the basic hormonal pathways, actions, and the appropriate methods to transport the samples for accurate
analysis.
31.1 Introduction
Endocrinopathies are a group of disorders resulting from
pathological sources of hormone production. Cross-sectional
imaging techniques such as CT and MRI are good morphological modalities but lack functional information which is
important for deciding the management of patients with various endocrinopathies. Venous sampling should be considered in patients with high suspicion of endocrine
abnormalities with normal or doubtful cross-sectional imaging ndings; in some situations, it has a role in patients with
unequivocal imaging ndings. Venous sampling plays an
important role in endocrinopathies such as endogenous adrenocorticotropic hormone (ACTH)-dependent Cushing syndrome (CS) to differentiate pituitary versus ectopic source;
in primary hyperaldosteronism to differentiate unilateral disease from bilateral disease; in small pancreatic neuroendocrine tumors (panNETs) presenting with hyper insulinemic
hypoglycemia and Zollinger-Ellison syndrome; in hyperandrogenism to identify the androgen-producing ovarian or
adrenal tumors; renal vein renin sampling in renovascular
hypertension to identify unilateral or bilateral secretion; and
tumor-induced osteomalacia (TIO) to help in preoperative
localization.
31.2 Inferior Petrosal Sinus Sampling
31.2.1 The Pituitary Gland andInferior
Petrosal Sinus Anatomy
The pituitary gland is situated in the sella and consists of the
anterior (adenohypophysis) and posterior lobes (neurohypophysis). The anterior lobe comprises pars tuberalis, pars
intermedia, and pars distalis. The majority of the pituitary
hormone production occurs in pars distalis.
D. Kandasamy (*) · K. Kabilan
Department of Radiodiagnosis and Interventional Radiology, All
India Institute of Medical Sciences, Delhi, India
© The Author(s), under exclusive license to Springer Nature Singapore Pte Ltd. 2024
S. H. Chandrashekhara (ed.), Textbook of Interventional Radiology, https://doi.org/10.1007/978-981-97-9601-4_31
Adrenocorticotropic hormone (ACTH), prolactin (PRL),
luteinizing hormone (LH), follicle stimulating hormone
(FSH), growth hormone (GH), and thyroid-stimulating hor-
389
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