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- •Foreword
- •Preface
- •Contents
- •List of Invited Discussants
- •History
- •Physical Examination
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Procedure
- •Discussion
- •References
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Discussion
- •Reference
- •9: Secondary Aortoduodenal Fistula Following Abdominal Aortic Aneurysm Repair
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •12: Large Symptomatic Abdominal Aortic Aneurysm
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •History
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •History
- •Procedure
- •Discussion
- •References
- •History
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Procedure
- •Discussion
- •Reference
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •Reference
- •34: Infected Dacron Patch Following Carotid Endarterectomy
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •38: Intracerebral Hemorrhage Following Carotid Endarterectomy
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •40: Nonconvulsive Status Epilepticus Following Carotid Endarterectomy
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •45: Redo Aorto-bifemoral Graft
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •48: Infected Aorto-bifemoral Graft
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •50: Aorto-Bifemoral Grafting for Infrarenal Aortic Occlusion
- •Procedure
- •Discussion
- •Reference
- •51: Exposed Femoral Graft Following Multiple Arterial Reconstruction
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Patient A: Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •58: Repeat Femoral Posterior Tibial Bypass Using Spliced Cephalic Vein
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Discussion
- •References
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •Reference
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •The Ruptured Kommerell’s Diverticulum
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Discussion
- •References
- •Discussion
- •References
- •Discussion
- •References
- •History
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •90: Iliac Stenting Complicated by Iliac Artery Rupture
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •96: Superior Mesenteric Artery In-stent Restenosis
- •Physical Examination
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •References
- •Procedure
- •Discussion
- •Reference
- •Procedure
- •Discussion
- •References
- •101: 100 Multiple Choice Questions
- •Part X Carotid Endarterectomy
- •Part XI Aortofemoral Grafting
- •Part XII Aortomesenteric Bypass
- •Part XIII Infrainguinal Arterial Bypass Graft
- •Part XX Thoracic Endovascular Aneurysm Repair
- •Part XXIII Carotid Stenting
- •Part XXIV Iliac Stenting
- •Part XXV Aortoiliac Stenting
- •Part XXVIII Renal Artery Stenting
- •Part XXIX Subclavian Artery Stenting
- •Part XXX Acquired Arteriovenous Fistula
- •Index

References
225
stump of the SFA (endarterectomized) or off the
graft itself as was the case here. Since this was
an in situ conguration, the vein graft hood
should be large enough that when you are suturing at directly onto a prosthetic graft material, it
should not scar down. However, if a reversed
saphenous vein bypass graft is used, it may end
up scaring at the prosthetic vein interface, and
an anastomosis to native artery should be
considered.
Pseudoaneurysms may also be a sign of infection, usually indolent, if late as is the case here
[4]. Oftentimes, the graft is non-incorporated and
may or may not have perigraft uid present. It is
beyond the scope of this commentary, but in situ
options exist for repair, including rifampinsoaked Dacron as well as using ePTFE.Overall,
aortofemoral bypasses are durable procedures
but do certainly have complications. One quite
rare and one common late complication is noted
here, but this procedure should remain an option
for vascular surgeons in their treatment of aortoiliac disease.
References
1. Gloriczki P, Cross A, Stenson AW, Carr M, Bower TC,
et al. Ischemic injury to spinal cord a lumbar-sacral
plexus as after aorto-iliac reconstruction. Am J Surg.
1991;162:131–6.
2. Bredahl K, Jensen LP, Schroeder TV, Sillesen H,
Nielsen H, Eiberg JP. Mortality and complications after aortic bifurcated bypass procedures for
chronic aortoiliac occlusive disease. J Vasc Surg.
2015;62(1):75–82.
3. Rosenthal D. Spinal cord ischemia after abdominal aortic operation: is it preventable? J Vasc Surg.
1999;30(3):391–7.
4. Seabrook GR, Schmitt DD, Bandyk DF, Edmiston CE,
Krepel CJ, Towne JB.Anastomotic femoral pseudoaneurysm: an investigation of occult infection as an etiologic factor. J Vasc Surg. 1990;11(5):629–34.

Part XII
Aorto-Mesenteric Bypass

Mesenteric Revascularization
inPatients withAcute onChronic
Bowel Ischemia
53
Patient A: History andPhysical
Examination
A 70-year-old female was seen in the outpatient
clinic in June 2002 with symptoms of postprandial abdominal pain, anorexia, gas pains, loss of
weight, and diarrhea of 6–9 months duration.
Medical comorbidities included Type II diabetes
mellitus, coronary artery disease with remote
coronary artery bypass graft, hyperlipidemia,
hypothyroidism, and stage III chronic kidney disease. She underwent CTA of the abdomen and
pelvis which showed 70% stenosis of the celiac
artery and occlusion of the rst two centimeters
of the superior mesenteric artery. She was admitted to the hospital as an emergency because of
right lower quadrant pain in July 2002.
Patient A: Procedure
She was started on IV heparin and underwent
antegrade aorto-superior mesenteric bypass graft
with 8mm PTFE interring prosthesis via midline
transperitoneal approach in July 2002. After the
division of the left triangular ligament, left lobe
of the liver was retracted toward the right.
Dissection was done by opening the gastrohepatic omentum, the esophagus with nasogastric
tube was retracted toward the left, and deep
blades of the retractor system (Bookwalter III
retractor system, Symmetry Surgical, Antioch,
TN) were inserted to expose the right crus of the
diaphragm. The muscular bers of the right crus
were separated with long Metzenbaum scissors
so that a 5–6cm long opening was made in the
crus. The preaortic fascia over the aorta was
divided, and supraceliac aorta was exposed and
mobilized in its anterior aspect and on each side
for a length of 3–4cm.
Superior mesenteric artery was exposed at the
root of the small bowel mesentery after mobilizing the ligament of Treitz. It was exposed on the
left side of the superior mesenteric vein, and it
had no pulse. A tunnel was made posterior to the
posterior wall of the body of the stomach but
anterior to the body of the pancreas through the
right side of the transverse mesocolon.
Intravenous heparin (8000units) was administered by the anesthesia team; a Satinsky clamp
was applied to supraceliac aorta near its occluding lumen. Aortotomy incision 5.5–2 cm long
was made, and edges of the aorta were divided
by curved scissors to open the aortotomy. An
8 mm INTERING PTFE (W.L. Gore Inc.,
Newark, DE) was anastomosed end to side to the
supraceliac aorta and end to end to the divided
superior mesenteric artery. Patient had excellent
superior mesenteric artery pulse distal to the
bypass. The pale caecal wall at the time of laparotomy became pink in color. In the ensuing few
months, patient had weight gain and was relieved
of her symptoms of intestinal angina. In 2010
patient presented with ischemic ulceration with
© Springer Nature Switzerland AG 2020
S. S. Hans, Challenging Arterial Reconstructions, https://doi.org/10.1007/978-3-030-44135-7_53
229

230
53 Mesenteric Revascularization inPatients withAcute onChronic Bowel Ischemia
osteomyelitis of the left big toe with gangrenous
changes. She underwent CO2 with low-dose contrast arteriography of the abdominal aorta and
lower extremities (Fig.53.1). CO2 was preferred
as patient’s chronic kidney disease has progressed to stage IV. She underwent left femoral
endarterectomy and a left femoral popliteal
bypass graft on June 2010. An 8mm PTFE prosthesis was used as greater saphenous vein in the
left leg has been used for coronary bypass graft.
In July 2010, patient had below-knee amputation
following failure of the left femoral popliteal
bypass graft. Patient subsequently expired in
October 2010.
Patient B: History andProcedure
A 60-year-old female in August 1989 underwent
aorto-bifemoral graft and proximal anastomosis
end to end with a 14 × 7 mm knitted Dacron
graft. Patient’s main symptoms were intermittent
Fig. 53.1 Postoperative (abdominal aortogram) showing patent aorto-superior mesenteric artery bypass and few
enlarged visceral collaterals

Discussion
231
Fig. 53.2 Aortogram showing juxtarenal aortic occlusion
with occlusion of proximal superior mesenteric artery and
small IMA
claudication; she was found to have juxtarenal
aortic occlusion and occlusion of proximal 3cm
segment of superior mesenteric artery, a patent
inferior mesenteric artery, and a right common
femoral artery occlusion (Fig. 53.2). After the
proximal anastomosis was completed (proximal
anastomosis end to end) and distal aorta was
sutured shut above the inferior mesenteric artery,
the terminal 6 inches of the small intestine
became ischemic. Greater saphenous vein was
harvested from the right upper thigh, and a
5- centimeter length of the greater saphenous
vein in a reversed fashion was sutured to the
proximal right limb of the Dacron graft and to
the superior mesenteric artery (retrograde
approach) (Fig.53.3). The small bowel regained
its normal color. Patient was seen 8 years later
with symptoms of abdominal pain and some
weight loss. Aortogram was performed which
showed occlusion of the graft. Patient was
offered antegrade bypass, but she refused any
further surgical intervention.
Fig. 53.3 Retrograde bypass from the Dacron graft to the
superior mesenteric artery
Discussion
The diagnosis of chronic mesenteric ischemia is
increasing because of better awareness of its
symptoms and readily availability of duplex
ultrasound scan for evaluating patients with suspected mesenteric artery occlusive disease. The
optimal method of treatment and type of revascularization (open versus endovascular) and type of
surgical repair (transaortic endarterectomy versus aortomesenteric bypass grafting; antegrade
versus retrograde bypass) remain controversial
[1]. In most series, long-term patency after revascularization with objective means is lacking.
Endovascular therapy is signicantly less invasive but is only successful in patients with stenotic lesions of the visceral arteries. Antegrade
synthetic aortomesenteric bypass is associated
with long-term graft patency and satisfactory
functional outcome as reported by Jimenez etal.
in 47 patients with in-hospital mortality of 11%
and with primary assisted and secondary 5-year
graft patency of 69% [2].

232
53 Mesenteric Revascularization inPatients withAcute onChronic Bowel Ischemia
Both patients in this report had a follow-up of
8 years. The rst patient had an abdominal aortography and study of the lower extremities for
ischemic lesions of the left foot. The second
patient in this report had subsequent occlusion of
the graft (retrograde bypass graft), but the exact
time of the occlusion cannot be determined.
Invited Commentary from
Frank M.Davis, MD, andPeter
K.Henke, MD
The diagnosis and management of chronic mesenteric ischemia (CMI) remains a challenge and
has a considerable morbidity and risk of mortality for patients aficted with this condition.
Although the exact incidence of CMI remains
unknown, current estimates indicate that this
condition accounts for less than 1 per 100,000
hospital admissions in the United States; however, recent investigations suggest that CMI
admissions are increasing [3, 4]. For patients
aficted with CMI, classic symptoms include
abdominal pain, weight loss, and “food fear.”
The abdominal pain is often postprandial and
begins within a few minutes to 30minutes after
meals, persisting for as long as 5–6 hours.
Unintentional weight loss can progress to malnutrition and cachexia, which is often present at
the time of intervention. In some patients, clinical presentation of CMI can be less specic with
vague abdominal pain, nausea, vomiting, or
change in bowel habits, without the classic postprandial component. Lastly, a minority of
patients can remain completely asymptomatic as
prior autopsy studies have shown that 6–10% of
people have a greater than 50% stenosis in at
least one mesenteric artery; in those with peripheral vascular disease, the incidence may be as
high as 27% [5]. CMI patients often have traditional atherosclerotic risk factors with tobacco
history, hypertension, and hyperlipidemia documented in more than 60% to 70% [6, 7]. Given
the variable nature of symptoms combined with
the potential overlap with other disease processes, the diagnosis of CMI remains elusive and
can delay treatment.
One of the most devastating complications of
CMI is mesenteric arterial thrombosis resulting
in acute mesenteric ischemia (AMI). Acute arterial thrombosis superimposed on preexisting
severe atherosclerotic disease represents that second most common cause of AMI as up to 20% of
AMI patients have demonstrated a history of
CMI, with symptoms of abdominal pain, food
avoidance, or weight loss [8, 9]. For patients who
progress from CMI to AMI, the classic symptom
includes “abdominal pain of out proportion to
exam” for early mesenteric ischemia. Yet this
may be absent in 20–25% of cases, depending on
the cause and timing of presentation [10]. Until
transmural ischemia has developed, there is relatively little peritoneal irritation, thus minimizing
tenderness to palpation. Further, in contrast to
patients who develop AMI secondary to an
embolic event, patients who develop acute on
chronic mesenteric ischemia often have bowel
infarction that is more insidious in onset because
extensive collaterals are able to maintain viability
until there is nal closure of a critically stenotic
vessel or collateral. As such, a high index of suspicion in the setting of a compatible history and
physical examination serves as a cornerstone of
prompt treatment. Regarding the diagnostic algorithm for AMI, there has been a paradigm shift.
Older models advocated early and aggressive use
of diagnostic arteriography; however, this has
now largely been supplanted with computed
tomographic angiography (CTA) when AMI is
suspected. The use of ultrafast multidetector CTA
(MDCTA) for the evaluation of both acute and
chronic mesenteric ischemia is well described
[11]. The widespread availability of the newest
generation of CT scanners has advanced the diagnostic algorithm for AMI, providing a signicant
amount of information about the central arterial
and venous circulations.
Once a diagnosis of acute on chronic mesenteric ischemia is achieved, the goal of therapy for
patients is the prompt restoration of blood ow to
the visceral organs. Patients presenting with signs
and symptoms of AMI require urgent abdominal
exploration, assessment of bowel viability, and
revascularization. Several techniques for the restoration of intestinal perfusion are available

References
233
including endarterectomy, antegrade arterial
bypass, retrograde arterial bypass, or hybrid
revascularization. For arterial bypass in an emergent setting, a single bypass to the SMA is all that
is required, and graft orientation is inuenced
mainly by the degree of atherosclerosis and
occlusive disease present in the inow vessels
and by the overall lie of the graft. As such, a retrograde graft orientation is common with its origin from the right common iliac artery in a “lazy
C” conguration. This avoids any aortic clamping with the increased hemodynamic stress and
usually provides a good lie to prevent kinking.
Alternatively, a hybrid open retrograde mesenteric stenting can be performed for acute atherosclerotic SMA thrombosis. This technique
combines a less invasive mesenteric revascularization without compromising important general
surgical principles. Recent small single center
studies have demonstrated acceptable outcomes
with a hybrid approach [7]. A solely endovascular treatment modality is not generally applied
due to the potential need for bowel resection. As
described previously, operative bypass to the
SMA is traditionally required owing to the extent
of disease. In recent years, mortality after revascularization for AMI has declined from 50% in
the 1990s to 30% in the 2000s; however, mortality remains high in patients with AMI even after
successful surgical revascularization [3]. Factors
associated with increased mortality were renal
insufciency, age older than 70years, metabolic
acidosis, symptom duration, and need for bowel
resection during second-look operations [12].
Lastly, owing to high recurrence rates, both shortand long-term follow-up is critical for maintaining the patency of open and especially
endovascular revascularized vessels [13].
Within this current chapter, the authors detail
two cases of acute on chronic mesenteric ischemia and associated operative repair. These cases
highlight the variable nature of patient presentation for AMI as well as the diversity of surgical
procedures available to treat this potentially
lethal condition. The technical issues are well
delineated and outcomes overall good.
References
1. Scali ST, Ayo D, Giles K, Gray S, etal. Outcomes of
antegrade and retrograde open mesenteric bypass for
acute ischemia. J Vasc Surg. 2019;69:129–40.
2. Jimenez JZ, Huber TS, Ozaki CK, Flynn TC, etal.
Durability of antegrade synthetic aortomesenteric
bypass for chronic mesenteric ischemia. J Vasc Surg.
2002;35:1078–84.
3. Schermerhorn ML, Giles KA, Hamdan AD, Wyers
MC, Pomposelli FB. Mesenteric revascularization:
management and outcomes in the United States,
1988–2006. J Vasc Surg. 2009;50(2):341–8.. e341
4. Mitchell EL, Moneta GL. Mesenteric duplex
scanning. Perspect Vasc Surg Endovasc Ther.
2006;18(2):175–83.
5. Valentine RJ, Martin JD, Myers SI, Rossi MB, Clagett
GP. Asymptomatic celiac and superior mesenteric
artery stenoses are more prevalent among patients
with unsuspected renal artery stenoses. J Vasc Surg.
1991;14(2):195–9.
6. Pecoraro F, Rancic Z, Lachat M, etal. Chronic mesenteric ischemia: critical review and guidelines for
management. Ann Vasc Surg. 2013;27(1):113–22.
7. Kasirajan K, O’Hara PJ, Gray BH, et al. Chronic
mesenteric ischemia: open surgery versus percutaneous angioplasty and stenting. J Vasc Surg.
2001;33(1):63–71.
8. Mansour MA.Management of acute mesenteric ischemia. Arch Surg. 1999;134(3):328–30;discussion 331
9. Endean ED, Barnes SL, Kwolek CJ, Minion DJ,
Schwarcz TH, Mentzer RM Jr. Surgical management
of thrombotic acute intestinal ischemia. Ann Surg.
2001;233(6):801–8.
10. Howard TJ, Plaskon LA, Wiebke EA, Wilcox
MG, Madura JA. Nonocclusive mesenteric ischemia remains a diagnostic dilemma. Am J Surg.
1996;171(4):405–8.
11. Horton KM, Fishman EK. Multidetector CT angiography in the diagnosis of mesenteric ischemia. Radiol
Clin N Am. 2007;45(2):275–88.
12. Kougias P, Lau D, El Sayed HF, Zhou W, Huynh TT,
Lin PH.Determinants of mortality and treatment outcome following surgical interventions for acute mesenteric ischemia. J Vasc Surg. 2007;46(3):467–74.
13. Kanamori KS, Oderich GS, Fatima J, etal. Outcomes
of reoperative open or endovascular interventions
to treat patients with failing open mesenteric reconstructions for mesenteric ischemia. J Vasc Surg.
2014;60(6):1612–9.. e1611–2

Part XIII
Infrainguinal Arterial Bypass Graft

Femoral-Peroneal Bypass
forCritical Limb Ischemia
inaPatient withUnstable Angina
54
History andPhysical Examination
A 76-year-old male with history of signicant
coronary artery disease (prior coronary artery
bypass graft, coronary stenting, pacemaker) was
admitted to the emergency room with ischemic
rest pain with associated paresthesias involving
the right foot. Other medical comorbidities
included hypertension, chronic renal failure (creatinine 2.1% mg), and nicotine abuse.
Examination of the right lower extremity pulses
revealed that all the pulses were absent below the
right femoral artery level. Doppler arterial study
showed absent ow in the right dorsalis pedis and
posterior tibial artery with unrecordable ankle
brachial index and toe brachial index. Patient
developed increasing symptoms of cardiac ischemia (angina) at rest and was started on IV heparin and underwent urgent coronary vascularization
with stenting of circumex branch of coronary
artery.
Procedure
After the patient was stabilized from cardiac
standpoint, he underwent right lower extremity
arteriography which showed occlusion of the
right supercial femoral artery from the midsegment to involve the popliteal artery. Posterior
tibial artery and anterior tibial arteries were also
occluded. The runoff was by a patent peroneal
artery which showed 60% stenosis in its midsegment (Fig. 54.1). Right greater saphenous vein
had been used for coronary bypass graft in the
past. Patient underwent right proximal supercial
femoral to peroneal artery bypass using contralateral non-reversed greater saphenous vein. The
valve lysis was performed with retrograde valvulotome following proximal anastomosis. Peroneal
artery was exposed in its middle to distal segment
via a medial approach using tourniquet occlusion, and distal anastomosis to the peroneal artery
inferior to the stenotic area was performed
(Fig.54.2). Peroneal artery was slightly less than
2 millimeters in diameter. Patient had a satisfactory postoperative course. Patient was seen in
October 2019 with patent right femoral-peroneal
bypass with normal velocities on duplex imaging, and a follow-up CTA showed patent right
femoral-peroneal bypass (Fig.54.3).
Discussion
Greater saphenous vein is a proven conduit of
choice for infrainguinal arterial bypass because
of its length, size compatibility, and durability.
Ipsilateral greater saphenous vein is inadequate
in 20% of patients [1]. The optimal alternative
conduit for lower extremity revascularization in
such circumstances remains an issue of ongoing
debate. Prosthetic grafts are clearly inferior to
autogenous conduits for infragenicular
© Springer Nature Switzerland AG 2020
S. S. Hans, Challenging Arterial Reconstructions, https://doi.org/10.1007/978-3-030-44135-7_54
237

238
54 Femoral-Peroneal Bypass forCritical Limb Ischemia inaPatient withUnstable Angina
Fig. 54.1 Preoperative arteriogram showing occlusion of
the distal two third of right supercial femoral artery and
popliteal artery. Occlusion of posterior tibial and anterior
Fig. 54.2 Exposure of the distal peroneal artery arteriotomy with blue silastic loops and anastomosed with distal anas-
tomosis with not reversed contralateral saphenous vein
reconstructions. Chew etal. reported 226 autogenous infrainguinal reconstructions in 203
patients using contralateral greater saphenous
vein (31%), single segment lesser saphenous vein
(5%), single segment arm vein (19%), and autogenous composite vein (45%) [1]. They observed
that the 5-year patency rates were signicantly
better for contralateral greater saphenous vein.
Conte et al. noted that patients requiring lower
extremity arterial bypass present increasingly
complex medical and surgical challenge in recent
years characterized by increasing age, increased
proportion of women, and higher prevalence of
tibial artery with runoff with peroneal artery with midsegment stenosis
the National Surgical Quality Improvement
Program database identied 6978 patients from
2012 to 2015 who underwent infrainguinal arterial bypass; of those, 327 (4.7%) had severe
chronic kidney disease and 550 (7.9%) were on
hemodialysis [3]. They concluded that chronic
kidney disease is a signicant predictor of perioperative morbidity and mortality following
lower extremity arterial bypass [3]. Patients with
severe chronic kidney disease have worse postoperative outcomes with increased mortality. Those
on hemodialysis have worse survival and postoperative outcomes [3].
diabetes and renal disease [2]. Ambur etal. from
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