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- •Preface
- •Contents
- •1.3.4 The Eustachian Tube
- •1.3.5 Muscles
- •1.3.6 Innervation
- •1.3.7 Vascular Supply
- •1.4 The Inner Ear (Labyrinthine Cavity)
- •1.4.1 The Vestibule
- •1.4.2 Semicircular Canals
- •1.4.4 The Cochlea
- •1.4.5 Innervation
- •1.1 Introduction
- •1.2 The External Ear
- •1.2.1 The Auricle
- •1.2.3 The Eternal Auditory Canal/External Acoustic Meatus
- •1.3 The Middle Ear (Tympanic Cavity)
- •1.3.1 The Tympanic Membrane
- •1.3.3 Ossicles
- •1.4.6 Cochlea Nerve Anatomy
- •1.4.7 Vestibular Nerves
- •1.4.8 The Vestibulocochlear Nerve
- •1.5 The Central Hearing System
- •1.5.3 Auditory Input
- •1.5.4 The Auditory Nerve’s Descending Routes
- •References
- •2: Outer–Middle–Inner Ear Embryology
- •2.1 Introduction
- •2.2 Embryology
- •2.3.1 First Week
- •2.3.3 Third Week
- •2.3.4 Fourth Week
- •2.3.5 Sixth Week
- •References
- •3.1 Introduction
- •3.3 The Outer Ear
- •3.3.1 Anatomy
- •3.3.3 Localization
- •3.4 The Middle Ear
- •3.4.3 Middle Ear Muscles
- •3.4.4 The Eustachian Tube
- •3.4.5 Impedance Matching
- •3.5 The Inner Ear
- •3.5.1.1 Lateral Wall
- •3.5.1.2 Reissner’s Membrane
- •3.5.1.3 The Basilar Membrane
- •3.5.2.1 Hair Cells
- •Inner Hair Cells
- •Outer Hair Cells
- •3.5.3 The Tectorial Membrane
- •3.5.4 The Osseous Spiral Lamina
- •3.5.5 Cochlear Mechanics
- •3.5.5.1 Passive Mechanics
- •3.5.5.2 Active Mechanics
- •3.6.1 Auditory Nerve Fibers
- •3.6.2 The Subcortical Auditory Nuclei
- •3.6.2.1 The Cochlear Nucleus
- •3.6.2.2 The Superior Olivary Complex
- •3.6.2.3 The Lateral Lemniscus
- •3.6.2.4 Inferior Colliculus
- •3.6.2.5 The Medial Geniculate Body
- •3.6.3 The Auditory Cortex
- •3.7 Conclusion
- •References
- •4.1 Introduction
- •4.2 Eustachian Tube Anatomy
- •4.4 Eustachian Tube Dysfunction
- •References
- •5: Temporal Bone Radiology
- •5.1.1 Introduction
- •5.1.2 Computed Tomography (CT)
- •5.1.3 Temporal Bone CT Angiography
- •5.1.4 Magnetic Resonance Imaging (MRI)
- •5.1.5 Diffusion-Weighted Imaging (DWI)
- •5.1.6 Conclusion
- •5.2.1 Introduction
- •5.2.2.1 The External Auditory Canal (EAC)
- •5.2.3 Temporal Bone Fractures
- •5.2.4 Conclusion
- •5.3.1 Introduction
- •5.3.2 Necrotizing Otitis Externa
- •5.3.3 Middle Ear
- •5.3.3.2 Chronic Otitis Media
- •5.3.3.3 Cholesteatomas
- •5.3.3.4 Cholesterol Granulomas
- •5.3.4 Inner Ear
- •5.3.4.1 Labyrinthitis
- •5.3.4.2 Petrous Apicitis
- •5.3.5 Conclusion
- •5.4.1 Introduction
- •5.4.2.1 Cerebellopontine Angle Tumors
- •Vestibular Schwannomas
- •Arachnoid Cysts
- •Meningiomas
- •5.5.2 External Auditory Canal Aplasia
- •5.5.4 Inner Ear Malformations
- •5.5.4.1 Complete Labyrinthine Aplasia/Michel Anomaly
- •5.5.4.2 Rudimentary Otocysts
- •5.5.4.3 Common Cavity Malformation
- •5.5.4.4 Incomplete Partition (IP) Type I
- •5.5.4.5 Incomplete Partition Type II/Mondini Malformation
- •5.5.4.6 Incomplete Partition Type III
- •5.5.4.7 Cochlear Anomalies
- •5.5.4.8 Semicircular Canal Anomalies
- •5.5.6 Conclusion
- •5.6.1 Introduction
- •5.6.2 Otospongiosis/Otosclerosis
- •Epidermoids
- •5.4.2.2 The Middle Ear
- •5.4.2.4 Petrous Bone
- •5.4.2.5 Metastatic Tumors
- •5.4.3 Conclusion
- •5.5.1 Introduction
- •5.6.3 Third Window Lesions
- •5.6.4 Conclusion
- •References
- •6.1 Introduction
- •6.3.1 What Is Sound?
- •6.3.2 Sound Intensity
- •6.4 Psychoacoustics
- •6.4.1 Signal Detection Theory
- •References
- •7.1 Introduction
- •7.1.1 What Is Sound?
- •7.2 Fundamental Acoustic Concepts
- •7.2.3 Period
- •7.2.4 Frequency
- •7.2.5 Wavelength
- •7.3 Psychoacoustics
- •7.3.1 Loudness
- •7.3.2 Auditory Masking
- •7.3.2.1 Simultaneous Masking
- •7.3.2.2 Temporal Masking
- •7.4.2 Spatial Hearing
- •References
- •8.1 Introduction
- •8.2 Case History
- •8.3 The Audiology Test Room
- •8.4.1 Pure-Tone Audiometry
- •8.4.1.1 Masking
- •8.4.2 Speech Audiometry
- •8.4.3 Pediatric Assessment
- •8.5.1 Acoustic Immittance Audiometry
- •8.5.1.1 Tympanometry
- •Tympanogram Interpretation
- •8.5.1.2 Multifrequency Tympanometry
- •8.5.1.3 Wideband Tympanometry
- •8.5.1.4 Acoustic Reflex Test
- •8.5.1.5 The Reflex Decay Test
- •8.5.1.6 Eustachian Tube Evaluation
- •8.5.2 Otoacoustic Emissions
- •8.5.2.2 Performing Otoacoustic Emission Tests
- •8.5.3 Auditory Evoked Potentials
- •8.5.3.2 Auditory Evoked Brainstem Response
- •Stimulus Types
- •Stimulus Polarity
- •Stimulus Presentation Rate
- •Stimulus Intensity
- •Analysis Time (Recording Epoch)
- •Filters
- •Artifact Rejection Level
- •Electrodes
- •8.5.3.3 Auditory Steady-State Responses
- •8.5.3.4 Electrocochleography
- •Electrocochleography Analysis
- •8.5.3.5 Cortical Auditory Evoked Potentials
- •8.5.3.6 Event-Related Auditory Potentials
- •P300
- •Mismatch Negativity
- •Acoustic Change Complex
- •8.6 Conclusion
- •References
- •9.1 Introduction
- •9.2.3 Conductive Hearing Loss
- •9.2.4 Sensorineural Hearing Loss
- •9.2.4.1 Internal Acoustic Canal Tumors
- •9.2.4.2 Auditory Neuropathy Spectrum Disorder
- •9.2.4.3 Third Window Syndrome
- •9.2.4.4 Dead Region
- •9.2.5 Mixed Hearing Loss
- •9.3 Hearing Loss Configuration
- •9.3.3 Unilateral or Bilateral Hearing Loss
- •9.3.4 Symmetric or Asymmetric Hearing Loss
- •9.3.5 Fluctuating or Stable Hearing Loss
- •9.4 Diagnostic Tests
- •9.4.1 Pure Tone Threshold Testing
- •9.4.2 Speech Recognition Tests
- •9.4.3 Tympanometric Tests
- •9.4.4 Stapedial Reflex
- •9.4.5 Otoacoustic Emission Test
- •9.4.6 Auditory Brainstem Responses
- •9.6 Reporting Audiological Findings
- •9.7 Conclusion
- •References
- •10.1 Introduction
- •10.2.1 Anamnesis
- •10.2.2 Hearing Loss
- •10.2.3 Ear Pain (Otalgia)
- •10.2.4 Ear Discharge (Otorrhea)
- •10.2.5 Itchy Ear
- •10.2.8 Physical Examination
- •10.2.8.1 Inspection
- •10.2.8.2 Palpation
- •10.2.8.3 Otoscopy
- •10.2.12 Hearing Examination
- •10.2.13 Hearing Assessment
- •10.2.13.1 Whisper Test
- •10.2.13.2 Tuning Fork Tests
- •Rinne Test
- •Weber Test
- •Schwabach Test
- •Gelle Test
- •10.3 Conclusion
- •References
- •11.1 Introduction
- •11.2.1 Microphone
- •11.2.2 Amplifier
- •11.2.3 Receiver
- •11.2.4 Batteries
- •11.2.5 Earmolds/Domes
- •11.4 Hearing Aid Types
- •11.5.1 Directional Microphone Technologies
- •11.5.2 Digital Noise Reduction
- •11.5.3 Frequency Lowering
- •11.5.4 Feedback Canceller
- •11.5.5 Bluetooth
- •11.6 Other Hearing Aid Technologies
- •11.7 Pediatric Hearing Aid Application
- •11.7.3.7 Hearing Aid Fitting
- •Prescription Formula Preference
- •Objective Verification Tools
- •Subjective Verification Tools
- •Fine-Tuning
- •11.8 Adult Hearing Aid Application
- •11.8.1.1 Medical Evaluation
- •11.8.1.2 Audiological Evaluation
- •11.8.1.3 Physical Evaluation
- •11.8.1.4 Psychological Evaluation
- •11.8.2 Hearing Aid Application Process
- •11.8.2.1 Anamnesis
- •11.8.2.6 Hearing Aid Fitting
- •Fine-Tuning
- •11.9 Conclusion
- •11.10 Case Studies
- •11.10.1 Case 1
- •11.10.2 Case 2
- •11.10.3 Case 3
- •11.10.4 Case 4
- •References
- •12.1 Introduction
- •12.3.1 Pathophysiology
- •12.3.2 Management
- •12.3.3 Etiology
- •12.3.4 Epidemiology
- •12.3.5 Assessing
- •12.3.6 Treatment
- •References
- •13: Otoplasty
- •13.1 Introduction
- •13.2 General Information
- •13.2.1 Auricular Anthropometry
- •13.3 History
- •13.8.1 Conservative Treatment
- •13.8.2 Surgical Treatment
- •13.11 Patient Follow-Up
- •13.12 Case Examples
- •13.13 Complications
- •13.13.1 Early Complications
- •13.13.2 Late Complications
- •13.13.3.1 Telephone Ear Deformity
- •13.13.3.2 Reverse Telephone Ear Deformity
- •13.13.3.5 Antihelical Malposition
- •13.13.3.6 Tragal Prominence
- •13.13.3.7 Auricular Lines
- •13.14 Revision Otoplasty
- •References
- •14: External Ear Tract Diseases
- •14.1 Introduction
- •14.2.1 Atopic Dermatitis
- •14.2.2 Allergic Contact Dermatitis
- •14.2.3 Photoallergic Dermatitis
- •14.2.4 Psoriasis
- •14.2.5 Relapsing Polychondritis
- •14.2.6 Gout
- •14.3 Traumatic Disorders
- •14.3.1 Irritant Contact Dermatitis
- •14.3.2 Phototoxic Dermatitis
- •14.3.3 Phototrauma
- •14.4 Infectious Diseases
- •14.4.1 Otitis Externa
- •14.4.1.1 Background
- •14.4.1.2 Anatomy
- •14.4.1.3 Classification
- •14.4.1.5 Diagnosis
- •14.4.1.6 Management
- •References
- •15: Auricula Tumors
- •15.1 Introduction
- •15.2 Benign Tumors
- •15.2.1 Chondrodermatitis Nodularis Chronica Helicis
- •15.2.2 Cystic Chondromalacia
- •15.2.3 Ceruminous Gland Adenoma
- •15.3 Malign Tumors
- •15.3.1 Basal Cell Carcinoma (BCC)
- •15.3.2 Squamous Cell Carcinoma
- •15.3.3 Ceruminous Gland Adenocarcinoma
- •15.4 Conclusion
- •References
- •16: Acute Suppurative Otitis Media
- •16.1 Introduction
- •16.2 Pathophysiology
- •16.3 Etiology
- •16.3.1 Host Factors
- •16.3.1.1 Immune System
- •16.3.1.2 Hereditary Susceptibility
- •16.3.1.3 Mucins
- •16.3.1.4 Anatomic Abnormalities
- •16.3.1.5 Physiologic Dysfunction
- •16.3.2 Infectious Factors
- •16.3.2.1 Bacterial Pathogens
- •16.3.2.2 Viral Pathogens
- •16.3.3 Environmental Factors
- •16.3.3.1 Infant Feeding Methods
- •16.4 Classification
- •16.6 Diagnosis
- •16.7 Treatment
- •16.7.1 Antibiotic Therapy Versus Observation
- •16.7.2 Initial Antibiotic Therapy
- •16.7.3 Supplemental Programs
- •References
- •17.1 Introduction
- •17.2 Definition
- •17.4 Pathophysiology
- •17.5 Diagnosis
- •17.5.1 Clinical Evaluation
- •17.6 Treatment
- •17.6.1 Medical Treatment
- •17.6.2 Surgical Treatment
- •17.7 Conclusion
- •References
- •18: Chronic Suppurative Otitis Media
- •18.1 Introduction
- •18.2 Epidemiology
- •18.3 Pathophysiology
- •18.4 Microbiology
- •18.5 Histopathology
- •18.6 Clinical Manifestations
- •18.6.1 Tubotympanic Type
- •18.6.2 Atticoantral Type
- •18.7 Diagnosis
- •18.7.1 Anamnesis
- •18.7.2 Otoscopic Examination
- •18.7.3 Audiological Evaluation
- •18.7.4 Imaging
- •18.8 Treatment
- •18.8.1 Medical Treatment
- •18.8.2 Surgical Treatment
- •18.9 Complications
- •18.10 Future Directions
- •18.11 Conclusion
- •References
- •19: Cholesteatoma
- •19.1 Introduction
- •19.2 Definition
- •19.3 Epidemiology
- •19.4 Histopathology
- •19.7 Cholesteatoma Types
- •19.7.1 Congenital Cholesteatoma
- •19.7.2 Acquired Cholesteatoma
- •19.7.2.2 Epithelial Migration Theory
- •19.7.2.3 Basal Cell Hyperplasia Theory
- •Tos Staging
- •Sade Staging
- •19.7.3 Unclassified Cholesteatomas
- •19.7.4 Petrous Bone Cholesteatomas
- •19.8 Practical Classification
- •19.8.1 Attic Cholesteatomas
- •19.8.2 Sinus Cholesteatomas
- •19.8.3 Pars Tensa Cholesteatomas
- •19.9 Clinical Presentations
- •19.9.1 Cholesteatoma Microbiology
- •19.10 Diagnosis
- •19.10.2 Computed Tomography
- •19.10.3 Magnetic Resonance Imaging
- •19.10.4 Audiometric Evaluation
- •19.11.1 Closed Techniques
- •19.11.2 Open Techniques
- •19.12 Conclusion
- •References
- •20.1 Introduction
- •20.2 Physiology
- •20.2.4 Tympanic Isthmus
- •20.4 Pathophysiology
- •20.5 Clinical Picture
- •20.6 Management
- •20.6.1 Surgical Management
- •20.6.1.2 Tympanoplasty
- •20.6.1.3 Mastoid Surgery
- •20.7 Adhesive Otitis Media
- •20.7.1 Pathogenesis
- •20.7.2 Clinical Findings
- •20.7.3 Imaging
- •20.7.4 Treatment
- •20.8 Conclusion
- •References
- •21.1 Introduction
- •21.2 Intratemporal Complications
- •21.2.1 Acute Mastoiditis
- •21.2.2 Facial Nerve Paralysis
- •21.2.3 Labyrinthitis
- •21.2.4 Labyrinthine Fistula
- •21.2.5 Petrositis
- •21.3 Intracranial Complications
- •21.3.1 Meningitis
- •21.3.2 Lateral Sinus Thrombosis
- •21.3.3 Brain Abscess
- •21.3.4 Otitic Hydrocephalus
- •21.3.5 Epidural Abscess
- •21.3.6 Subdural Empyema
- •21.4 Conclusion
- •References
- •22: Basic Otological Surgical Techniques
- •22.1 Introduction
- •22.3 Atticotomy
- •22.4 Mastoidectomy
- •22.4.1 Simple (Cortical) Mastoidectomy
- •22.4.2 Canal Wall-Up Mastoidectomy
- •22.4.3 Canal Wall-Down Mastoidectomy
- •22.4.4 Retrograde Mastoidectomy
- •22.4.5 Modified Radical Mastoidectomy
- •22.4.6 Radical Mastoidectomy
- •22.4.7 Mastoid Obliteration
- •22.5 Petrosectomy
- •22.6 Conclusion
- •References
- •23: Tympanoplasty
- •23.1 Introduction
- •23.2.1 Chronic Otitis Media
- •23.2.2 Traumatic Perforations
- •23.5 Tympanoplasty Types
- •23.7 Graft Materials
- •23.8 Graft Techniques
- •23.8.1 The Perichondrium/Cartilage Island Graft
- •23.8.2 The Palisade Graft
- •23.8.3 The Temporalis Fascia Graft
- •23.9 Surgical Approaches
- •23.9.1 Microscopic Approach
- •23.9.2 Endoscopic Approach
- •23.10.1 Transmeatal Incisions
- •23.10.1.1 The Rosen Incision
- •23.10.1.3 Anterior Tympanomeatal Flap
- •23.10.2 Endaural Incision
- •23.10.3 Postauricular Incision
- •23.11 Pediatric Tympanoplasty
- •23.12 Prognostic Factors
- •23.14 Conclusion
- •References
- •24: Ossiculoplasty
- •24.1 Introduction
- •24.4 Indications/Contraindications
- •24.5 Reconstruction Materials
- •24.7 Surgical Preparation
- •24.8 Surgical Technique
- •24.9 Ossiculoplasty Results
- •24.10 Complications
- •24.11 Postoperative Care
- •24.12 Follow-Up
- •24.13 Conclusion
- •References
- •25: Tympanomastoidectomy
- •25.1 Introduction
- •25.2 Surgical Anatomy
- •25.4 Indications
- •25.5 Technique
- •25.5.1 Patient’s Preparation
- •25.5.3 Simple Mastoidectomy
- •25.5.4 Posterior Tympanostomy or Facial Recess Approach
- •25.5.5 Epitympanectomy
- •25.5.6 Endolymphatic Sac Procedures
- •25.5.8 Atticotomy-Atticoantrotomy

64
Z. Öztürk et al.
are two examples of medical therapies. If indicated, treatment for rhinosinusitis
should be initiated. Immunosuppressive drugs may be needed for granulomatous
diseases such as sarcoidosis or granulomatosis associated with polyangiitis
(Wegener’s disease) [1].
Studies have shown that the ETDQ-7 score improves at 12months after surgical
dilatation of the ET with a balloon catheter. In most cases, a tympanostomy tube is
inserted to control ET dysfunction caused by otitis media with effusion [1].
If adenoid hypertrophy is suspected as the primary cause of ET dysfunction, the
patient should be considered for adenoidectomy. Middle ear effusion is a common
symptom in the younger generation. Self-inating devices that increase nasal pressure have shown promising results in reopening the ET and facilitating uid clearance [20, 21].
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Eustachii. Aktueller Stand [Function tests for the Eustachian tube. Current knowledge].
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Srp Arh Celok Lek. 2004;132(5–6):148–51.
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25, 2022. https://emedicine.medscape.com/article/874348- overview#a5 (Accessed online on
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with effusion: pepsinogen study of adenoid tissue and middle ear uid. J Otolaryngol Head
Neck Surg. 2008;37(4):565–71.
7. Sedlmaier B, Pomorzev A, Haisch A, Halleck P, Scherer H, Goktas O.The improvement of
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study. Lasers Med Sci. 2009;24(5):793–800.
8. Poe DS, Silvola J, Pyykkö I.Balloon dilation of the cartilaginous Eustachian tube. Otolaryngol
Head Neck Surg. 2011;144(4):563–9.
9. Tisch M, Maier H, Sudhoff H.Balloon dilation of the Eustachian tube: clinical experience in
the management of 126 children. Acta Otorhinolaryngol Ital. 2017;37(6):509–12.
10. Satmis MC, van der Torn M.Balloon dilatation of the Eustachian tube in adult patients with
chronic dilatory tube dysfunction: a retrospective cohort study. Eur Arch Otorrinolaringol.
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11. Mehta NK, Ma C, Nguyen SA, McRackan TR, Meyer TA, Lambert PR.Medical management for Eustachian tube dysfunction in adults: a systematic review and meta-analysis.
Laryngoscope. 2022;132(4):849–56.
12. Kanemaru SI, Umeda H, Yamashita M, etal. Improvement of eustachian tube function by
tissue-engineered regeneration of mastoid air cells. Laryngoscope. 2013;123(2):472–6.
13. Alper CM, Teixeira MS, Swarts JD, etal. Quantitative description of Eustachian tube movements during swallowing as visualized by transnasal videoendoscopy. JAMA Otolaryngol
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4 Eustachian Tube: AnOverview
14. Kalcioglu MT, Sallavaci S, Hrncic N, etal. Prevalence of and factors affecting otitis media
with effusion in children in the region from Balkans to Caspian basin: a multicentric crosssectional study. Int J Pediatr Otorhinolaryngol. 2021;143:110647. https://doi.org/10.1016/j.
ijporl.2021.110647.
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65

Temporal Bone Radiology
MehmetBilginEser, UmutPerçemOrhan Söylemez,
andGazanferEkici
5.1 Imaging Modalities andTechniques forTemporal
Bone Evaluation
5.1.1 Introduction
In the evaluation of the temporal bone, the complex anatomical structure and limitations of otoscopic examination make radiologic imaging indispensable [1]. This
chapter explores the role of various imaging modalities and techniques in the evaluation of the temporal bone and aims to explain which radiologic modalities should
be used in temporal bone pathology.
5
5.1.2 Computed Tomography (CT)
Currently, the most recent method of temporal bone imaging offering the highest
resolution is photon-counting CT. It will probably replace the current CT technology in the next few decades. Cone beam CT also offers better resolution than
conventional CT, but it is not widely available. So high-resolution computed
tomography (HRCT) is the reference method for assessing the bony anatomy of
the temporal bone. It provides detailed images of the ossicles, cochlea, semicircular canals (SCCs), and mastoid air cells. HRCT’s thin slices allow for multiplanar
reconstructions, enabling precise visualization of intricate structures and
M. B. Eser (*)
Bornova Türkan Ozilhan State Hospital, Radiology Clinic, Izmir, Turkey
U. P. Orhan Söylemez
Goztepe Prof. Dr. Süleyman Yalcin City Hospital, Department of Radiology, Istanbul, Turkey
G. Ekici
Yeditepe University, Medical Faculty, Department of Radiology, Istanbul, Turkey
© The Author(s), under exclusive license to Springer Nature
Switzerland AG 2024
M. T. Kalcioglu et al. (eds.), Otology Updates, Comprehensive ENT,
https://doi.org/10.1007/978-3-031-76173-7_5
67

68
pathologies such as fractures, congenital malformations, and otosclerosis.
Computed tomography (CT) must be chosen for bone destruction, tegmen defects,
ossicular chain dislocations, dehiscences, and fractures [2]. Cone beam CT is
increasingly used for post-implant imaging to conrm the position of the electrode array within the cochlea [3]. Due to its limited radiation exposure, temporal
bone HRCT is preferred for evaluation of pediatric patients with suspected congenital or developmental anomalies. In pre-cochlear implant evaluation, CT is
used both to classify congenital–developmental anomalies and to identify variations to minimize complications prior to surgery [4].
M. B. Eser et al.
5.1.3 Temporal Bone CT Angiography
CT angiography has a limited role in temporal bone imaging. In visualizing vascular anomalies such as glomus tumors or vascular malformations, it provides valuable information for surgical planning and assessment of tumor vascularity [5].
5.1.4 Magnetic Resonance Imaging (MRI)
Magnetic resonance imaging (MRI) sequences, such as T2- (T2W) and T1-weighted
images, provide excellent soft tissue contrast and are particularly useful for evaluation of the internal auditory canal (IAC), cochlea, and vestibule. These sequences aid
in the detection of pathologies such as vestibular schwannomas and labyrinthitis.
Contrast-enhanced imaging should be used to look for tumors, neural pathologies
(vestibular neuritis, perineural extension, facial nerve hemangiomas, etc.), and complications such as abscess, meningitis, or cerebritis. MRI in pre-cochlear implant
evaluation is extremely important to conrm the presence of the eighth nerve [1, 6].
5.1.5 Diffusion-Weighted Imaging (DWI)
This MRI sequence is an extremely important weapon in the diagnosis of cholesteatomas. With the diffusion sequences developed in recent years, it is possible to distinguish a cholesteatoma larger than 2–3mm from other secretions [7, 8]. Non-echo
planar imaging diffusion-weighted sequence (non-EPI DWI) is the recommended
standard imaging modality for cholesteatomas [8].
5.1.6 Conclusion
Evaluation of the temporal bone benets from a comprehensive understanding of
different imaging modalities and techniques. CT and MRI, along with specialized
approaches, offer distinct advantages for evaluating the various structures and
pathologies within the temporal bone. Sometimes combined radiologic modalities
are used. Choosing the right imaging modality would help achieve the correct diagnosis by avoiding wasted time and unnecessary imaging. All radiologic evaluations

5 Temporal Bone Radiology
Table 5.1 Imaging modalities for suspected pathologies
The rst imaging
Pathology
Chronic otitis media CT MRI for complications such as
Congenital inner ear
anomalies
Cholesteatoma MRI Diffusion-weighted images,
Trauma (fracture, ossicular
chain evaluation)
Neuritis Contrast-enhanced MRI
Labyrinthitis Contrast-enhanced MRI
Otosclerosis CT
Large vestibular aqueduct CT
Superior semicircular canal
dehiscence
CT computed tomography, MRI magnetic resonance imaging, EPI echoplanar imaging
modality to choose
CT MRI for cochlear nerve evaluation
CT
CT
Additional information
meningitis, abscess, etc.
non-EPI diffusion
69
should be conducted in the light of clinical knowledge, and there should be radiologic and clinical solidarity. The radiologic modalities that should be requested
according to the clinically suspected pathology are summarized in Table5.1.
5.2 Radiologic Anatomy andFractures
oftheTemporal Bone
5.2.1 Introduction
The temporal bone is a complex and vital structure within the skull, housing intricate anatomical components related to hearing, balance, and critical neurovascular
pathways. This chapter will explore the radiologic imaging anatomy of the temporal
bone, focusing on images from both CT and MRI.
5.2.2 Parts oftheTemporal Bone
The temporal bone, which participates in the formation of the lateral skull base,
consists of ve bony anatomical regions: the squamous, tympanic, mastoid, petrous,
and styloid processes. The squamous part contains the zygomatic arch, and the
petrous bone houses the labyrinthine and cochlear structures (Fig.5.1) [1, 5, 9].
5.2.2.1 The External Auditory Canal (EAC)
The distal one-third is surrounded by cartilage and the medial part by the tympanic
portion of the temporal bone (Figs.5.1 and 5.2). The anterior wall of the EAC forms
the posterior surface of the glenoid fossa; therefore, the extension of the pathology
of the external auditory canal to the temporomandibular joint is not surprising. The

70
Fig. 5.1 Axial computed tomographic anatomy of the temporal bone. 1: Mastoid part; 2: External
auditory canal; 3: Tympanic membrane; 4: Tympanic part; 5: Petrous part; 6: Squamous part; 7:
Cochlea; 8: Lateral semicircular canal; 9: Posterior semicircular canal; 10: Malleus; 11: Incus; and
12: Internal auditory canal
M. B. Eser et al.
Fig. 5.2 Coronal computed tomographic anatomy of the temporal bone. 1: Internal auditory
canal; 2: Oval window; 3: Scutum; 4: Tegmen tympani; 5: Ossicles, 6: Incus; 7: Cochlea; 8: Facial
nerve; 9: Lateral semicircular canal; 10: Superior semicircular canal; and 11: Vestibule
tympanic membrane (TM) separates the external auditory canal from the tympanic
cavity. The TM is more visible on CT when it is thickened and has two parts: the
accida and the tensa [5].
5.2.2.2 The Middle Ear (ME) andtheOssicular Chain
The tympanic cavity is an air-lled structure containing the ossicular chain and
muscles and is separated from the inner ear by the otic capsule. The ossicles, the
malleus, incus, and stapes, can be clearly visualized on thin-section CT (Figs.5.1
and 5.2). The tegmen tympani is the bony roof of the tympanic cavity. Defects in the
tegmen tympani can lead to spread of infection to the middle cranial fossa. The roof
of the mastoid bone is called the tegmen mastoideum. The tympanic cavity is

5 Temporal Bone Radiology
71
divided into three parts: the epitympanum, mesotympanum, and hypotympanum.
Prussak’s space is the specic name of the part of the lateral epitympanic recess,
which is the area between the pars accida and the scutum (Fig.5.3). The ossicular
chain is a highly complex anatomical structure [5].
The malleus consists of the head, neck, manubrium, and anterior and lateral processes. The manubrium is the part that attaches the TM and the head, which is
articulated to the body of the incus. The incus consists of a body that articulates with
the malleus, short, long, and lenticular processes. The lenticular process articulates
with the stapes. The stapes consists of a head, a footplate, and anterior and posterior
crus. The head of the stapes articulates with the incus, and the footplate attaches to
the oval window (Fig.5.2) [5].
The jugular vein, sigmoid sinus, and internal carotid artery neighborhoods are
highly important in radiologic evaluation. If variations and dehiscences are not
known prior to surgery, they can lead to signicant complications. A high-riding
jugular bulb is quite common. If the sigmoid plate is not intact, it is called dehiscence (Fig.5.4). An aberrant internal carotid artery and a persistent stapedial artery
are less common anomalies [5].
5.2.2.3 The Cochlea andtheVestibular System
The otic capsule surrounds the bony labyrinth. The cochlea appears as a spiralshaped structure that rotates about 2.5 turns and is exquisitely visualized on highresolution MRI sequences (Fig.5.5). It is divided into the basal, middle, and apical
turns by interscalar septa. MRI ndings allow assessment of cochlear nerve integrity and detection of cochlear abnormalities such as cochlear nerve aplasia.
Vestibular and SCCs: the vestibular system is essential for maintaining balance and
is assessed by MRI’s ability to delineate the labyrinthine structures, aiding in the
detection of vestibular schwannoma and other vestibular disorders. CT scans show
its bony labyrinthine canal [5].
Other anatomical structures to be aware of include the endolymphatic duct,
cochlear aqueduct, and vestibular aqueduct (VA). The endolymphatic duct is
Fig. 5.3 Coronal
computed tomographic
anatomy of the tympanic
cavity. 1: Medial
epitympanic recess; 2:
Superior attic; 3: Lateral
epitympanic recess; 4:
Prussak’s space; 5:
Mesotympanum; and 6:
Hypotympanum

72
ab
Fig. 5.4 (a) A high-riding jugular bulb (black arrow) and sigmoid sinus (black arrow). (b) A high-
riding jugular bulb and dehiscence (white arrow); no bony structure is seen in the sigmoid plate.
The dotted arrow represents dilated emissary veins passing through the mastoid bone, which
should be noted before mastoidectomy
M. B. Eser et al.
ab c
Fig. 5.5 Axial (a) and coronal (b and c) thin-slice MR images. 1: Lateral semicircular canal; 2:
Cochlea; 3: Cochlear nerve; 4: Facial nerve; 5: Vestibule; 6: Posterior semicircular canal; 7: Pons;
8: Fourth ventricle; 9: Superior semicircular canal; 10: Lateral semicircular canal; 11: Posterior
semicircular canal; 12: Cochlea; 13: Pons; and 14: Vertebral artery
surrounded by the VA.The size of the VA is 1–2mm. The cochlear aqueduct is an
even thinner structure (0.1–0.2mm) [10, 11].
5.2.2.4 The Internal Auditory Canal andCranial Nerves
The IAC, a conduit for the seventh and eighth cranial nerves, is optimally visualized
by high-resolution MRI (Fig.5.6). This imaging helps detect tumors such as vestibular schwannomas and to differentiate between facial and cochlear nerve involvement. Accurate identication of the cranial nerves within the IAC is critical for
surgical planning and management decisions. The facial nerve can be followed
along its course using CT (Fig.5.6) [12]. After leaving the pons, the facial nerve
travels in its cisternal segment at the level of the cerebellopontine angle. It then
enters the IAC, and its canalicular segment lies anterosuperior to the auditory canal.
The labyrinthine segment then enters the petrous bone and reaches the geniculate
ganglion. From here, it turns and enters the middle ear (ME) cavity as the tympanic
segment. After the tympanic segment, the facial nerve passes through the mastoid
bone (mastoid segment) and enters the parotid gland through the stylomastoid foramen (Fig.5.6) [5].

5 Temporal Bone Radiology
Fig. 5.6 The internal auditory canal and facial nerve anatomy. 1: Facial nerve; 2: Cochlear nerve;
3: Superior vestibular nerve; 4: Inferior vestibular nerve; 5: Cerebellopontine angle; 6: Internal
auditory canal; 7: Mastoid segment of the facial nerve; 8: Tympanic segment of the facial nerve; 9:
Labyrinthine segment of the facial nerve; 10: Tympanic segment of the facial nerve, and 11:
Geniculate ganglion
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5.2.3 Temporal Bone Fractures
Fractures parallel to the petrous ridge are called longitudinal fractures, and those
perpendicular to the long axis of the petrous ridge are called transverse fractures
[13–15]. In longitudinal fractures, the axis of trauma is lateral, whereas, in transverse fractures, it is occipital. In situations such as high-speed motor vehicle accidents, the impact involves both lateral and occipital components and the injury may
be of a mixed type [15]. The most common type of trauma is longitudinal fractures
with a rate of 70–90%, followed by transverse fractures with a rate of 10–30%. The
main drawback of this classication is that it does not give an idea of the prognosis,
so there are new classications based on whether the otic capsule is preserved or
not. In the classication system proposed by Ishman and Friedland, the main criterion is whether the petrous bone is involved or not [16]. Otic capsule involvement
(5–20%) is an important prognostic criterion and is associated with an increased
incidence of facial paralysis, cerebrospinal uid (CSF) leakage, and profound hearing loss. Otic capsule-sparing fractures involving the ossicular chain usually result
in conductive hearing loss (CHL) or mixed hearing loss [13]. In addition to impact
trauma, foreign bodies introduced into the external ear canal may also cause trauma.
The most commonly injured structures in this type of trauma are the TM, oval window, and ossicular chain. Before evaluating the fracture on imaging, it is important
to know that many anatomic structures are parallel to the fracture lines and will
mimic the fracture on imaging. Some of these structures are listed in Table5.2.
The rst-line imaging modality is multidetector CT (MDCT) with coronal and
sagittal planes in addition to the axial plane [13, 14]. This examination should be

74
M. B. Eser et al.
Table 5.2 Anatomical
structures that mimic
fractures
The cochlear aqueduct
The vestibular aqueduct
The subarcuate canaliculus
The singular canal
The canal for the chorda tympani
The groove of the greater supercial petrosal nerve
The mastoid canaliculus
The inferior tympanic canaliculus
The petrosphenoidal ssure
The occipitomastoid suture
The tympanosquamous ssure
The petro-occipital ssure
The cochlear cleft
abc
def
Fig. 5.7 Examples of temporal bone fractures. (a) Fracture of the mastoid part of the temporal
bone without extension of the otic capsule and secretions compatible with the hemotympanum in
the mastoid cells; this is a longitudinal fracture extending into the mastoid air cells. (b) Mixed
fracture of the mastoid part of the temporal bone without extension of the otic capsule. © Isolated
fracture of the tympanic plate of the temporal bone. (d) Longitudinal fracture of the mastoid bone
with involvement of the tympanic cavity. (e, f) Transverse fracture of the mastoid bone (e, white
arrow) with otic capsule involvement (e, black arrow). It should not be overlooked that there are air
bubbles in the vestibule, and this condition is called pneumolabyrinth
performed with a sharp reconstruction algorithm to nd abnormal uid or air
between fractures between various small anatomical structures in the temporal
bone. While CT can show fractures in detail, MRI can show subtle hemorrhages,
CSF or perilymph leakage, and pneumolabyrinth (Fig.5.7). However, the presence
of hemorrhage in the ME cavity, labyrinth, and endolymphatic sac without a
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