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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_4507_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Preface
- •Contents
- •1.3.4 The Eustachian Tube
- •1.3.5 Muscles
- •1.3.6 Innervation
- •1.3.7 Vascular Supply
- •1.4 The Inner Ear (Labyrinthine Cavity)
- •1.4.1 The Vestibule
- •1.4.2 Semicircular Canals
- •1.4.4 The Cochlea
- •1.4.5 Innervation
- •1.1 Introduction
- •1.2 The External Ear
- •1.2.1 The Auricle
- •1.2.3 The Eternal Auditory Canal/External Acoustic Meatus
- •1.3 The Middle Ear (Tympanic Cavity)
- •1.3.1 The Tympanic Membrane
- •1.3.3 Ossicles
- •1.4.6 Cochlea Nerve Anatomy
- •1.4.7 Vestibular Nerves
- •1.4.8 The Vestibulocochlear Nerve
- •1.5 The Central Hearing System
- •1.5.3 Auditory Input
- •1.5.4 The Auditory Nerve’s Descending Routes
- •References
- •2: Outer–Middle–Inner Ear Embryology
- •2.1 Introduction
- •2.2 Embryology
- •2.3.1 First Week
- •2.3.3 Third Week
- •2.3.4 Fourth Week
- •2.3.5 Sixth Week
- •References
- •3.1 Introduction
- •3.3 The Outer Ear
- •3.3.1 Anatomy
- •3.3.3 Localization
- •3.4 The Middle Ear
- •3.4.3 Middle Ear Muscles
- •3.4.4 The Eustachian Tube
- •3.4.5 Impedance Matching
- •3.5 The Inner Ear
- •3.5.1.1 Lateral Wall
- •3.5.1.2 Reissner’s Membrane
- •3.5.1.3 The Basilar Membrane
- •3.5.2.1 Hair Cells
- •Inner Hair Cells
- •Outer Hair Cells
- •3.5.3 The Tectorial Membrane
- •3.5.4 The Osseous Spiral Lamina
- •3.5.5 Cochlear Mechanics
- •3.5.5.1 Passive Mechanics
- •3.5.5.2 Active Mechanics
- •3.6.1 Auditory Nerve Fibers
- •3.6.2 The Subcortical Auditory Nuclei
- •3.6.2.1 The Cochlear Nucleus
- •3.6.2.2 The Superior Olivary Complex
- •3.6.2.3 The Lateral Lemniscus
- •3.6.2.4 Inferior Colliculus
- •3.6.2.5 The Medial Geniculate Body
- •3.6.3 The Auditory Cortex
- •3.7 Conclusion
- •References
- •4.1 Introduction
- •4.2 Eustachian Tube Anatomy
- •4.4 Eustachian Tube Dysfunction
- •References
- •5: Temporal Bone Radiology
- •5.1.1 Introduction
- •5.1.2 Computed Tomography (CT)
- •5.1.3 Temporal Bone CT Angiography
- •5.1.4 Magnetic Resonance Imaging (MRI)
- •5.1.5 Diffusion-Weighted Imaging (DWI)
- •5.1.6 Conclusion
- •5.2.1 Introduction
- •5.2.2.1 The External Auditory Canal (EAC)
- •5.2.3 Temporal Bone Fractures
- •5.2.4 Conclusion
- •5.3.1 Introduction
- •5.3.2 Necrotizing Otitis Externa
- •5.3.3 Middle Ear
- •5.3.3.2 Chronic Otitis Media
- •5.3.3.3 Cholesteatomas
- •5.3.3.4 Cholesterol Granulomas
- •5.3.4 Inner Ear
- •5.3.4.1 Labyrinthitis
- •5.3.4.2 Petrous Apicitis
- •5.3.5 Conclusion
- •5.4.1 Introduction
- •5.4.2.1 Cerebellopontine Angle Tumors
- •Vestibular Schwannomas
- •Arachnoid Cysts
- •Meningiomas
- •5.5.2 External Auditory Canal Aplasia
- •5.5.4 Inner Ear Malformations
- •5.5.4.1 Complete Labyrinthine Aplasia/Michel Anomaly
- •5.5.4.2 Rudimentary Otocysts
- •5.5.4.3 Common Cavity Malformation
- •5.5.4.4 Incomplete Partition (IP) Type I
- •5.5.4.5 Incomplete Partition Type II/Mondini Malformation
- •5.5.4.6 Incomplete Partition Type III
- •5.5.4.7 Cochlear Anomalies
- •5.5.4.8 Semicircular Canal Anomalies
- •5.5.6 Conclusion
- •5.6.1 Introduction
- •5.6.2 Otospongiosis/Otosclerosis
- •Epidermoids
- •5.4.2.2 The Middle Ear
- •5.4.2.4 Petrous Bone
- •5.4.2.5 Metastatic Tumors
- •5.4.3 Conclusion
- •5.5.1 Introduction
- •5.6.3 Third Window Lesions
- •5.6.4 Conclusion
- •References
- •6.1 Introduction
- •6.3.1 What Is Sound?
- •6.3.2 Sound Intensity
- •6.4 Psychoacoustics
- •6.4.1 Signal Detection Theory
- •References
- •7.1 Introduction
- •7.1.1 What Is Sound?
- •7.2 Fundamental Acoustic Concepts
- •7.2.3 Period
- •7.2.4 Frequency
- •7.2.5 Wavelength
- •7.3 Psychoacoustics
- •7.3.1 Loudness
- •7.3.2 Auditory Masking
- •7.3.2.1 Simultaneous Masking
- •7.3.2.2 Temporal Masking
- •7.4.2 Spatial Hearing
- •References
- •8.1 Introduction
- •8.2 Case History
- •8.3 The Audiology Test Room
- •8.4.1 Pure-Tone Audiometry
- •8.4.1.1 Masking
- •8.4.2 Speech Audiometry
- •8.4.3 Pediatric Assessment
- •8.5.1 Acoustic Immittance Audiometry
- •8.5.1.1 Tympanometry
- •Tympanogram Interpretation
- •8.5.1.2 Multifrequency Tympanometry
- •8.5.1.3 Wideband Tympanometry
- •8.5.1.4 Acoustic Reflex Test
- •8.5.1.5 The Reflex Decay Test
- •8.5.1.6 Eustachian Tube Evaluation
- •8.5.2 Otoacoustic Emissions
- •8.5.2.2 Performing Otoacoustic Emission Tests
- •8.5.3 Auditory Evoked Potentials
- •8.5.3.2 Auditory Evoked Brainstem Response
- •Stimulus Types
- •Stimulus Polarity
- •Stimulus Presentation Rate
- •Stimulus Intensity
- •Analysis Time (Recording Epoch)
- •Filters
- •Artifact Rejection Level
- •Electrodes
- •8.5.3.3 Auditory Steady-State Responses
- •8.5.3.4 Electrocochleography
- •Electrocochleography Analysis
- •8.5.3.5 Cortical Auditory Evoked Potentials
- •8.5.3.6 Event-Related Auditory Potentials
- •P300
- •Mismatch Negativity
- •Acoustic Change Complex
- •8.6 Conclusion
- •References
- •9.1 Introduction
- •9.2.3 Conductive Hearing Loss
- •9.2.4 Sensorineural Hearing Loss
- •9.2.4.1 Internal Acoustic Canal Tumors
- •9.2.4.2 Auditory Neuropathy Spectrum Disorder
- •9.2.4.3 Third Window Syndrome
- •9.2.4.4 Dead Region
- •9.2.5 Mixed Hearing Loss
- •9.3 Hearing Loss Configuration
- •9.3.3 Unilateral or Bilateral Hearing Loss
- •9.3.4 Symmetric or Asymmetric Hearing Loss
- •9.3.5 Fluctuating or Stable Hearing Loss
- •9.4 Diagnostic Tests
- •9.4.1 Pure Tone Threshold Testing
- •9.4.2 Speech Recognition Tests
- •9.4.3 Tympanometric Tests
- •9.4.4 Stapedial Reflex
- •9.4.5 Otoacoustic Emission Test
- •9.4.6 Auditory Brainstem Responses
- •9.6 Reporting Audiological Findings
- •9.7 Conclusion
- •References
- •10.1 Introduction
- •10.2.1 Anamnesis
- •10.2.2 Hearing Loss
- •10.2.3 Ear Pain (Otalgia)
- •10.2.4 Ear Discharge (Otorrhea)
- •10.2.5 Itchy Ear
- •10.2.8 Physical Examination
- •10.2.8.1 Inspection
- •10.2.8.2 Palpation
- •10.2.8.3 Otoscopy
- •10.2.12 Hearing Examination
- •10.2.13 Hearing Assessment
- •10.2.13.1 Whisper Test
- •10.2.13.2 Tuning Fork Tests
- •Rinne Test
- •Weber Test
- •Schwabach Test
- •Gelle Test
- •10.3 Conclusion
- •References
- •11.1 Introduction
- •11.2.1 Microphone
- •11.2.2 Amplifier
- •11.2.3 Receiver
- •11.2.4 Batteries
- •11.2.5 Earmolds/Domes
- •11.4 Hearing Aid Types
- •11.5.1 Directional Microphone Technologies
- •11.5.2 Digital Noise Reduction
- •11.5.3 Frequency Lowering
- •11.5.4 Feedback Canceller
- •11.5.5 Bluetooth
- •11.6 Other Hearing Aid Technologies
- •11.7 Pediatric Hearing Aid Application
- •11.7.3.7 Hearing Aid Fitting
- •Prescription Formula Preference
- •Objective Verification Tools
- •Subjective Verification Tools
- •Fine-Tuning
- •11.8 Adult Hearing Aid Application
- •11.8.1.1 Medical Evaluation
- •11.8.1.2 Audiological Evaluation
- •11.8.1.3 Physical Evaluation
- •11.8.1.4 Psychological Evaluation
- •11.8.2 Hearing Aid Application Process
- •11.8.2.1 Anamnesis
- •11.8.2.6 Hearing Aid Fitting
- •Fine-Tuning
- •11.9 Conclusion
- •11.10 Case Studies
- •11.10.1 Case 1
- •11.10.2 Case 2
- •11.10.3 Case 3
- •11.10.4 Case 4
- •References
- •12.1 Introduction
- •12.3.1 Pathophysiology
- •12.3.2 Management
- •12.3.3 Etiology
- •12.3.4 Epidemiology
- •12.3.5 Assessing
- •12.3.6 Treatment
- •References
- •13: Otoplasty
- •13.1 Introduction
- •13.2 General Information
- •13.2.1 Auricular Anthropometry
- •13.3 History
- •13.8.1 Conservative Treatment
- •13.8.2 Surgical Treatment
- •13.11 Patient Follow-Up
- •13.12 Case Examples
- •13.13 Complications
- •13.13.1 Early Complications
- •13.13.2 Late Complications
- •13.13.3.1 Telephone Ear Deformity
- •13.13.3.2 Reverse Telephone Ear Deformity
- •13.13.3.5 Antihelical Malposition
- •13.13.3.6 Tragal Prominence
- •13.13.3.7 Auricular Lines
- •13.14 Revision Otoplasty
- •References
- •14: External Ear Tract Diseases
- •14.1 Introduction
- •14.2.1 Atopic Dermatitis
- •14.2.2 Allergic Contact Dermatitis
- •14.2.3 Photoallergic Dermatitis
- •14.2.4 Psoriasis
- •14.2.5 Relapsing Polychondritis
- •14.2.6 Gout
- •14.3 Traumatic Disorders
- •14.3.1 Irritant Contact Dermatitis
- •14.3.2 Phototoxic Dermatitis
- •14.3.3 Phototrauma
- •14.4 Infectious Diseases
- •14.4.1 Otitis Externa
- •14.4.1.1 Background
- •14.4.1.2 Anatomy
- •14.4.1.3 Classification
- •14.4.1.5 Diagnosis
- •14.4.1.6 Management
- •References
- •15: Auricula Tumors
- •15.1 Introduction
- •15.2 Benign Tumors
- •15.2.1 Chondrodermatitis Nodularis Chronica Helicis
- •15.2.2 Cystic Chondromalacia
- •15.2.3 Ceruminous Gland Adenoma
- •15.3 Malign Tumors
- •15.3.1 Basal Cell Carcinoma (BCC)
- •15.3.2 Squamous Cell Carcinoma
- •15.3.3 Ceruminous Gland Adenocarcinoma
- •15.4 Conclusion
- •References
- •16: Acute Suppurative Otitis Media
- •16.1 Introduction
- •16.2 Pathophysiology
- •16.3 Etiology
- •16.3.1 Host Factors
- •16.3.1.1 Immune System
- •16.3.1.2 Hereditary Susceptibility
- •16.3.1.3 Mucins
- •16.3.1.4 Anatomic Abnormalities
- •16.3.1.5 Physiologic Dysfunction
- •16.3.2 Infectious Factors
- •16.3.2.1 Bacterial Pathogens
- •16.3.2.2 Viral Pathogens
- •16.3.3 Environmental Factors
- •16.3.3.1 Infant Feeding Methods
- •16.4 Classification
- •16.6 Diagnosis
- •16.7 Treatment
- •16.7.1 Antibiotic Therapy Versus Observation
- •16.7.2 Initial Antibiotic Therapy
- •16.7.3 Supplemental Programs
- •References
- •17.1 Introduction
- •17.2 Definition
- •17.4 Pathophysiology
- •17.5 Diagnosis
- •17.5.1 Clinical Evaluation
- •17.6 Treatment
- •17.6.1 Medical Treatment
- •17.6.2 Surgical Treatment
- •17.7 Conclusion
- •References
- •18: Chronic Suppurative Otitis Media
- •18.1 Introduction
- •18.2 Epidemiology
- •18.3 Pathophysiology
- •18.4 Microbiology
- •18.5 Histopathology
- •18.6 Clinical Manifestations
- •18.6.1 Tubotympanic Type
- •18.6.2 Atticoantral Type
- •18.7 Diagnosis
- •18.7.1 Anamnesis
- •18.7.2 Otoscopic Examination
- •18.7.3 Audiological Evaluation
- •18.7.4 Imaging
- •18.8 Treatment
- •18.8.1 Medical Treatment
- •18.8.2 Surgical Treatment
- •18.9 Complications
- •18.10 Future Directions
- •18.11 Conclusion
- •References
- •19: Cholesteatoma
- •19.1 Introduction
- •19.2 Definition
- •19.3 Epidemiology
- •19.4 Histopathology
- •19.7 Cholesteatoma Types
- •19.7.1 Congenital Cholesteatoma
- •19.7.2 Acquired Cholesteatoma
- •19.7.2.2 Epithelial Migration Theory
- •19.7.2.3 Basal Cell Hyperplasia Theory
- •Tos Staging
- •Sade Staging
- •19.7.3 Unclassified Cholesteatomas
- •19.7.4 Petrous Bone Cholesteatomas
- •19.8 Practical Classification
- •19.8.1 Attic Cholesteatomas
- •19.8.2 Sinus Cholesteatomas
- •19.8.3 Pars Tensa Cholesteatomas
- •19.9 Clinical Presentations
- •19.9.1 Cholesteatoma Microbiology
- •19.10 Diagnosis
- •19.10.2 Computed Tomography
- •19.10.3 Magnetic Resonance Imaging
- •19.10.4 Audiometric Evaluation
- •19.11.1 Closed Techniques
- •19.11.2 Open Techniques
- •19.12 Conclusion
- •References
- •20.1 Introduction
- •20.2 Physiology
- •20.2.4 Tympanic Isthmus
- •20.4 Pathophysiology
- •20.5 Clinical Picture
- •20.6 Management
- •20.6.1 Surgical Management
- •20.6.1.2 Tympanoplasty
- •20.6.1.3 Mastoid Surgery
- •20.7 Adhesive Otitis Media
- •20.7.1 Pathogenesis
- •20.7.2 Clinical Findings
- •20.7.3 Imaging
- •20.7.4 Treatment
- •20.8 Conclusion
- •References
- •21.1 Introduction
- •21.2 Intratemporal Complications
- •21.2.1 Acute Mastoiditis
- •21.2.2 Facial Nerve Paralysis
- •21.2.3 Labyrinthitis
- •21.2.4 Labyrinthine Fistula
- •21.2.5 Petrositis
- •21.3 Intracranial Complications
- •21.3.1 Meningitis
- •21.3.2 Lateral Sinus Thrombosis
- •21.3.3 Brain Abscess
- •21.3.4 Otitic Hydrocephalus
- •21.3.5 Epidural Abscess
- •21.3.6 Subdural Empyema
- •21.4 Conclusion
- •References
- •22: Basic Otological Surgical Techniques
- •22.1 Introduction
- •22.3 Atticotomy
- •22.4 Mastoidectomy
- •22.4.1 Simple (Cortical) Mastoidectomy
- •22.4.2 Canal Wall-Up Mastoidectomy
- •22.4.3 Canal Wall-Down Mastoidectomy
- •22.4.4 Retrograde Mastoidectomy
- •22.4.5 Modified Radical Mastoidectomy
- •22.4.6 Radical Mastoidectomy
- •22.4.7 Mastoid Obliteration
- •22.5 Petrosectomy
- •22.6 Conclusion
- •References
- •23: Tympanoplasty
- •23.1 Introduction
- •23.2.1 Chronic Otitis Media
- •23.2.2 Traumatic Perforations
- •23.5 Tympanoplasty Types
- •23.7 Graft Materials
- •23.8 Graft Techniques
- •23.8.1 The Perichondrium/Cartilage Island Graft
- •23.8.2 The Palisade Graft
- •23.8.3 The Temporalis Fascia Graft
- •23.9 Surgical Approaches
- •23.9.1 Microscopic Approach
- •23.9.2 Endoscopic Approach
- •23.10.1 Transmeatal Incisions
- •23.10.1.1 The Rosen Incision
- •23.10.1.3 Anterior Tympanomeatal Flap
- •23.10.2 Endaural Incision
- •23.10.3 Postauricular Incision
- •23.11 Pediatric Tympanoplasty
- •23.12 Prognostic Factors
- •23.14 Conclusion
- •References
- •24: Ossiculoplasty
- •24.1 Introduction
- •24.4 Indications/Contraindications
- •24.5 Reconstruction Materials
- •24.7 Surgical Preparation
- •24.8 Surgical Technique
- •24.9 Ossiculoplasty Results
- •24.10 Complications
- •24.11 Postoperative Care
- •24.12 Follow-Up
- •24.13 Conclusion
- •References
- •25: Tympanomastoidectomy
- •25.1 Introduction
- •25.2 Surgical Anatomy
- •25.4 Indications
- •25.5 Technique
- •25.5.1 Patient’s Preparation
- •25.5.3 Simple Mastoidectomy
- •25.5.4 Posterior Tympanostomy or Facial Recess Approach
- •25.5.5 Epitympanectomy
- •25.5.6 Endolymphatic Sac Procedures
- •25.5.8 Atticotomy-Atticoantrotomy

Tympanomastoidectomy
25
YükselOlgun, CemUzun, andSimonIgnacioAngeli
25.1 Introduction
Tympanomastoidectomy is the workhorse of the otology-neurotology surgeon. It
encompasses a wide range of different techniques, from the simple mastoidectomy
to extended procedures to access different areas of the temporal bone. The usual
indications are disease eradication, implantation of auditory devices, and approaches
for skull base surgery. This chapter will discuss the relevant surgical anatomy, currently accepted denitions, indications, and surgical techniques.
From a historical perspective [1], in 1873, Schwartz rst described the technique
of cortical mastoidectomy. VonTrölch stated for the rst time that the simple mastoidectomy techniques should be modied to prevent recurrent otorrhea and residual cholesteatoma. VonBergmann, on the other hand, used the term “radical” in
approaches where the posterior and superior parts of the bone canal as well as the
ossicular chain are removed and an open cavity is created. In 1890, Zaufal described
the radical mastoidectomy technique in detail for the rst time. In 1910, Bondy
proposed the modied radical mastoidectomy with preservation of the ossicular
chain technique to preserve the hearing of the patients. In 1938, Lempert used a
modied canal-wall-down mastoidectomy with fenestration of the lateral
semicircular canal for the treatment of otosclerotic conductive hearing. In the 1950s
Y. Olgun (*)
Faculty of Medicine, Department of Otorhinolaryngology, Dokuz Eylul University,
Izmir, Turkey
e-mail: yuksel.olgun@deu.edu.tr
C. Uzun
Faculty of Medicine, Department of Otorhinolaryngology, Koc University, Istanbul, Turkey
S. I. Angeli
Department of Otorhinolaryngology-Head and Neck Surgery, University of Miami Miller
School of Medicine, Miami, FL, USA
e-mail: SAngeli@med.miami.edu
© The Author(s), under exclusive license to Springer Nature
Switzerland AG 2024
M. T. Kalcioglu et al. (eds.), Otology Updates, Comprehensive ENT,
https://doi.org/10.1007/978-3-031-76173-7_25
503

504
Y. Olgun et al.
Zollner and Wullstein described tympanum and ossicular reconstruction techniques,
so-called tympanoplasty techniques, and in the 1960s Jansen, Sheehy, and other
authors introduced the facial recess approach in combination with intact-canal-wall
mastoidectomy to gain access to the posterior recesses of the middle ear and thus
facilitate removal of cholesteatoma disease and preserve hearing function.
25.2 Surgical Anatomy
The temporal bone consists of four parts: petrous, mastoid, tympanic, and squamous. Tympanomastoidectomy is usually performed from the retroauricular
approach, therefore the surgical anatomy will be described from this vantage point.
In the lateral aspect of the temporal bone, the most important surface landmarkes
are the following: The mastoid process is located laterally just behind the external
auditory canal, and inferior to the temporalis line; this latter represents the prominence that the insertion of the temporalis muscle creates on the bone and extends
anteriorly towards the zygomatic root. The temporalis line approximates the position of the middle cranial fossa just superior to the mastoid process and separates
this process from the squamous part of the temporal bone. The spine of Henle, also
called the suprameatal spine, is a small bony prominence in the lateral, posterior,
and superior region of the bony external auditory canal. Immediately posterior to
the Spine of Henle lies the cribriform area, within MacEwen’s triangle. The
MacEwen’s triangle is delineated by the temporalis line superiorly, and anteriorly
by a perpendicular line that travels tangentially to the posterior margins of the external bony meatus. The mastoid antrum lies deep to the surface of MacEwen’s triangle. An embryological remnant, Körner’s septum, can be encountered at the junction
of the petrous and squamous parts of the temporal bone forming the antrum’s lateral
roof. The prominence of the lateral semicircular canal and the bony labyrinth is in
the medial side of the antrum. The antrum communicates with the posterior epitympanum and middle ear via the aditus ad antrum. The body of the incus and the head
of the malleus are suspended in the posterior epitympanic space by ligaments from
the tegmen tympani. In front of the malleus’s head, one can recognize a bone spiculum that originates from the tegmen, Sheehy’s cog. The anterior epitympanic space
is found in front of the cog and leads to the petrous apex anterior-medially and to
the supratubal recess lateral to the tensor tympani muscle and its semi-canal anterior-inferiorly. The tensor tympani muscle ends in a tendon that curves laterally at
the cochleariform process and attaches to the malleus neck. The long process of the
incus joins with the stapes which lies in the oval window inferior to the tympanic
segment of the facial nerve. The sinus tympani, a recess of variable depth, is found
in the posterior wall of the tympanic cavity medial to the facial nerve. The opening
of the sinus tympani is bordered by two osseous folds called the ponticulum and
subiculum, The subiculum can be followed inferiorly to the area of the round window niche.
In the exposed mastoid cavity, Trautmann’s triangle, which serves as a pathway
to the posterior cranial fossa from the mastoid, is bordered by the bony labyrinth,

25 Tympanomastoidectomy
sigmoid sinus, and middle cranial fossa dura. In the lower part of the mastoid cavity,
there is a groove called “digastric ridge” which represents the impression that the
posterior belly of the digastric muscle creates in the medial surface of the mastoid
process. This is an important landmark for the facial nerve, as the stylomastoid foramen is located just medial of the anterior end of the digastric ridge.
505
25.3 Definitions andClassification
Recently, a classication of tympanomastoid surgery has been proposed by the
International Otology Outcome Group (IOOG) and adopted by several of the
world’s otology societies [2]. The IOOG classication uses the acronym SAMEO to
categorize mastoid bone operations, representing the stage of surgery, approach,
mastoid bone extirpation, external bony wall repair, and mastoid cavity obliteration.
The table depicts the accepted nomenclature to describe the different tympanomastoidectomy procedures. (Table25.1).
The type of tympanomastoidectomy is determined by many factors, among these
are the goal of surgery (i.e., surgery for disease eradication, implantation of auditory
prosthesis, or for exposure), extent of the pathology, the patient’s functional hearing, the degree of mastoid bone pneumatization, the type of surgery (i.e., primary or
revision), the position of anatomical structures (such as the sigmoid sinus, facial
nerve, and jugular bulb), the patient’s age, the anticipated patient’s compliance with
post-operative follow-up, the presence of complications, and the surgeon’s skill and
preference. To determine the relevant anatomical relationships, degree of pneumatization of the temporal bone, and extent of the pathology, imaging studies such as
temporal computed tomography and/or magnetic resonance scans should be carefully evaluated, and a detailed otomicroscopic and/or otoendoscopic examination
has also crucial importance.
25.4 Indications
Tympanomastoidectomy is the initial step of a variety of otologic procedures such
as chronic otitis media surgery, cochlear implantation, active middle ear implant
surgery, facial nerve decompression, endolymphatic sac surgery, labyrinthectomy,
and is used as an access in otoneurologic procedures to reach the lateral skull base,
cerebellopontine angle, and petrous apex. However, in this chapter, this procedure
will be addressed in relation to chronic ear disease and other indications will be
briey discussed when necessary.

506
Y. Olgun et al.
Table 25.1 The IOOG SAMEO scheme of the SAMEO-ATO framework
S stage of the surgery
S1 primary (rst surgery)
S2p (planned second look or staged procedure)
S2r Revison (unplanned)
*
2 represents non- primary surgery not the number of previous surgery
*
*
A approach
A1 endoscopic Transcanal
A2 microscopic Transcanal
A3 Endaural
**
A4 Retroauricular
**
A1 and A2 become A3 when a (external/widening) incision is made
M Mastoidectomy
Mx no mastoidectomy
M1a Mastoidectomy with canal wall preserved (cortical mastoidectomy)
M1b Mastoidectomy canal wall preserved (cortical mastoidectomy)+posterior tympantomy
M2a Mastoidectomy with only the upper scutum removed (Atticotomy)
M2b Mastoidectomy with removal of the postero-superior canal wall (Attico-antrostomy)
M2c Mastoidectomy with removal of the whole canal (modied radial or radical
mastoidectomy)
M1a + 2a Mastoidectomy with canal preserved + atticotomy
M1b+2a Mastoidectomy with canal preservation + posterior tympanotomy + atticotomy
M3a subtotal petrosectomy with preservation of the otic capsule
*
Exenteration of all mastoid cells
M3b subtotal petrosectomy with otic capsule removed
Includes labyrinthinthectomy and/or removal of the cochlea
*
M3a differs from M2c in that eardrum is removed intentionally prior to obliteration of cavity
and blind pit closure as well as blocking up of the tympanic opening of the Eustachian tube
E external ear canal reconstruction
Ex no reconstruction of the external auditory canal
E1 reconstruction using soft materials
E2 reconstruction using rigid materials
*
Space behind graft not obliterated
*
*
O obliteration of mastoid cavity
Ox no obliteration
O1 partial obliteration
O2 Total obliteration
*
Total obliteration is the obliteration of the entire mastoid and attic cavities. Partial obliteration
*
*
spares the attic cavity ± part of the mastoid cavity (i.e. only reduces the size of the cavity).
Adapted from Ref. [2]
25.5 Technique
25.5.1 Patient’s Preparation
Tympanomastoidectomy may be performed under general or local anesthesia, with
the patient is supine position with the head slightly turned to the opposite side.

25 Tympanomastoidectomy
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Long-acting muscle relaxants are avoided to enable facial nerve electromyography
monitoring. We favor the use of the otologic microscope which is then sterily draped
in preparation to the surgery. The otologic drill and suction-irrigation system are
assembled. The postauricular skin, auricle, ear canal, and adjacent areas are prepped
and draped sterilely with care to avoid placing thick drapes over the anticipated
surgical site.
Local inltration of a solution of 1–2% lidocaine with 1:50,000–100,000 epinephrine is applied to the incision area and external ear canal to facilitate hemostasis. In the ear canal the anesthetic agent is inltrated at the bony-cartilaginous
junction posteriorly, anteriorly, superiorly, and inferiorly. Sufcient time should be
allowed before performing the incision to allow for hemostatic vasoconstriction.
During the operation, a retroauricular incision is made 4–10mm posterior to retroauricular sulcus. Schwartz incision should be limited between the superior margin
of auricular attachment and the inferior level of external meatus.
25.5.2 Incision andSoft Tissue Flap
The postauricular incision is more commonly used than the endaural approach.
When there is a need to expose the ear canal, epitympanum and the middle ear, such
as in cases of cholesteatomatous chronic suppurative otitis media, the incision is
carried anteriorly over the auricle up to the root of the zygoma. The inferior limb of
the incision should be designed to allow exposure of the mastoid tip and area of the
sino-dural angle, but it should not extend medial to the mastoid tip. This is of particular importance in infants with underdeveloped mastoid to avoid injury to the
facial nerve as it exists through the styloid–mastoid foramen and found supercially
just medial to the ear lobule. If an extensive surgery is planned, the incision should
be placed more posteriorly [1, 2]. Tympanomastoidectomy can also be performed
via endaural incisions (Lempert, Rosen, vertical, and intercartilaginous incisions),
depending on the surgeon’s experience and preference [1, 2]. The most common
endaural incision is a 1–1.5cm intercartilaginous incision made between the helix
and the tragus. This incision starts from the tympanosquamous suture and continues
upward parallel to the helix cartilage and reaches the bone by passing through the
skin and subcutaneous tissue. Afterward, this incision is combined with the intracanal and parallel incisions dened by Rosen to reach the middle ear and mastoid
bone, and Heermann A and B incisions to harvest a temporalis muscle fascia graft
[1]. Although the endaural incision is mostly used with inside-out tympanomastoidectomy in limited pathologies, it is used for all types of mastoidectomy in some
centers.
A muscle periosteum ap is created and elevated to expose the mastoid process
[1, 3]. There are many variations of the muscle-periosteum ap. For most indications, we favor the anteriorly based ap also known as the Palva ap, starting with
a horizontal incision just superior to the temporalis line, and with the posterior
extension dictated by the need to expose the sino-dural angle and sigmoid sinus.
Some surgeons favor a posteriorly based ap, creating a perpendicular incision to

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the incision at the temporalis line just posterior to the external ear meatus.
Sometimes, the muscle-periosteum ap is elevated in continuity with the deep layer
of the temporalis fascia or with the temporalis muscle periosteum to create a large
pedicled ap that can be used to obliterate a large mastoid cavity [4].
During elevation of the muscle-periosteum ap, it is important not to tear the
skin of the posterior ear canal. When exposure of the ear canal or middle ear is
needed, incisions are created in the skin of the bony ear canal either before or after
the muscle-periosteum ap is elevated. The retroauricular soft tissues and ap are
then retracted for ample exposure of the mastoid process and linea temporalis. The
surface anatomical landmarks of the lateral temporal bone, such as linea temporalis,
spin of Henle, posterior wall of the EAC, area cribrosa, zygoma root, and mastoid
apex should be identied.
25.5.3 Simple Mastoidectomy
The drilling usually begins with a large cutting burr (i.e., 6–7mm), abundant irrigation, and suction. Irrigation is important to wash out the bone dust from the surgical
eld and to avoid burning bone. The rst burr cut is along the temporalis line, and
the second burr cut is along a tangetial line just posterior to the external ear meatus
(Fig.25.1). The drilling proceeds medially through the lateral cortex of the mastoid
process, keeping the area of deepest mastoid penetration at the conuence of the
two initial burr cuts. The mastoid antrum at the level of the lateral semicircular canal
is then identied as the drilling progresses medially through Körner’s septum
(Fig.25.2). The sigmoid sinus lies in a variable distance posterior and medially, and
knowledge of its anatomic location in reference to the mastoid antrum should be
ascertained preoperative by evaluating the imaging studies. As drilling progresses,
the middle fossa tegmen and the posterior wall of the external ear canal are outlined
to avoid bony overhangs at the edges of the mastoidectomy cavity that could affect
the exposure of the most medial planes. Once the lateral semicircular canal is in
view, the drilling proceeds to expose the body of the incus and the aditus ad antrum.
The tip of the short process of the incus should be visible now, and this is a reliable
Fig. 25.1 Lateral surface
of the mastoid bone of the
right ear

25 Tympanomastoidectomy
Fig. 25.2 Right cortical
mastoidectomy performed
as a rst step in cochlear
implantation
Fig. 25.3 Drainage of
subperiosteal abscess due
to acute otitis media
509
landmark for the identication of the second genu and descending portions of the
facial nerve, which lie approximately 1mm medial to the tip and the fossa incudis.
Exenteration of remaining air cells is carried out to skeletonize the sigmoid sinus
and identify the conuence of the sigmoid sinus with the middle fossa dura (i.e.,
sinodural or Citelli’s angle) superiorly, and to identify the digastric ridge inferiorly.
The bluish reex of the sigmoid sinus and the pinkish reex of the dura should be
recognized. The anterior extension of the digastric ridge is a helpful landmark to
estimate the position of the distal decending facial nerve as the nerve approximates
the styloid–mastoid foramen, just medial to the digastric ridge. At this point, with
visualization of the fossa incudi and digastric ridge, the direction and approximate
location of the descending facial nerve can be estimated.
The most common indication for simple mastoidectomy in the context of suppurative otitis media is coalescent mastoiditis and related complications [1, 2, 5].
(Fig.25.3) In addition, the simple mastoidectomy is the rst step of many surgical
access procedures, such as cochlear implantation, middle ear implants,

510
Y. Olgun et al.
endolymphatic sac surgery, labyrinthectomy, internal auditory canal dissection, and
pre-sigmoid posterior cranial fossa approaches.
25.5.4 Posterior Tympanostomy or Facial Recess Approach
The facial recess approach, better known as posterior tympanostomy, allows access
to the posterior recesses of the middle ear, including the areas of the oval and round
windows and the sinus tympani. Again, the tip of the incus is a reliable landmark
that points to the facial recess as the tip is 1mm lateral to the facial nerve. Other
approaches to the facial recess have been proposed [6]. Small diamond burrs of
decreasing sizes are sequentially used to thin the posterior wall of the external auditory canal until the chorda tympani nerve and the descending portion of the facial
nerve are visible through a thin layer of bone. Often, brisk bleeding is encountered
from one of the vessels in the fallopian canal alerting the surgeon to the proximity
of the facial nerve. Sometimes there is a prominent pyramidal eminence blocking
the view into the middle ear, and careful drilling of this structure and the bone anterior to the facial nerve is necessary to enhance the exposure to the round window
area and sinus tympani. In cases of surgery for cochlear or some active middle ear
implants, simple mastoidectomy and posterior tympanotomy approach are the basic
steps (Fig. 25.4). In addition, in cochlear implantation, it is often necessary to
remove the bony overhang of the round window niche to expose the round window
membrane. In cases of tumors involving the hypotympanum, the exposure is
enhanced by extending the facial recess area caudally by sacricing the chorda tympani nerve. While creating the posterior tympanotomy, the surgeon should always
keep in view the position of the bur and drill shaft and use sufcient irrigation to
avoid mechanical or thermal injury to the facial nerve. Similarly, avoid drilling
injury to the tympanic annulus and the bony external auditory canal when outlining
the chorda tympani.
Fig. 25.4 Posterior
tympanotomy and cochlear
implantation in the left ear

25 Tympanomastoidectomy
511
25.5.5 Epitympanectomy
An epitympanectomy is indicated when performing a tympanomastoidectomy for
attic cholesteatoma. The bone lateral to the epitympanum between the middle cranial fossa dura and the superior wall of the external auditory canal is removed to
expose the body of the incus and the head of the malleus. In underdeveloped bones,
there is only a small separation between the dura and the bone of the ear canal, and
to preserve these structures, the surgeon should use small diamond burrs. The incus
and head of the malleus are removed if involved with cholesteatoma with care not
to inadvertendly injure a deshicent tympanic facial nerve. The tensor tymani tendon
and cochleariform process should now be visible. If the incudo-stapedial joint is
still intact, this joint should be severed before manipulating the incus, either through
the ear canal or the posterior tympanostomy. After removing the incus, the fossa
incudi is drilled extending the posterior tympanostomy cephalad. The superior wall
of the external auditory canal is now outlined and the aircells and bone (i.e., Sheehy’s
cog) remaining in the area anterior to the head of the malleus are exenterated for
enhanced visibility of the epitympanum and supratubal recess, respectively.
Similarly, the area of the stapes, oval window and tympanic facial nerve should be
visible.
25.5.6 Endolymphatic Sac Procedures
In endolymphatic sac procedures, after completing a simple mastoidectomy, the
sigmoid sinus is skeletonized and followed caudally towards the jugular bulb. The
position of the descending facial nerve is estimated by joining in one line the second
genu (1mm medial to the tip of the short process of the incus) and the expected area
of the styloid-mastoid foramen (just medial to the anterior extension of the digastric
ridge). Another line is drawn parallel to the lateral semicircular canal; this is
Donaldson’s line. The endolymphatic sac is found attached to the dura of the posterior cranial fossa inferior to this line. Drilling continues to outline the posterior
semicircular canal, which is perpendicular and found just medial to the level of the
lateral semicircular canal. Retrofacial air cells are now removed exposing the dura
of the posterior cranial fossa. The sac is differentiated from the dura by its distinct
color and by a vessel than runs parallel to its longitudinal axis. By carefully depressing the dura, one can see the sac being tethered as it enters the bony opening of the
endolymphatic aqueduct.
25.5.7 Tympanomastoidectomy withRemoval ofExternal
Canal Wall
Canal wall down tympanomastoidectomy describes surgical procedures in which
the posterior wall of the external auditory canal is partially or totally removed [1, 2,
7]. These procedures can be classied as atticotomy, attico-antrotomy, retrograde

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(inside-out) mastoidectomy, Bondy procedure, modied radical mastoidectomy,
and radical mastoidectomy or subtotal petrosectomy. The main purpose and advantage of the canal wall down procedures is that they afford enhanced visualization of
recesses for disease exenteration. In cholesteatoma cases, the incidence of recidivistic disease is usually lower than in intact-canal-wall tympanmastoidectomy.
Moreover, detection of cholesteatoma recurrence (if occurs) is much earlier and
easier in comparison with the canal wall up procedures [1, 2, 7, 8]. The obvious
disadvantage of removing the external auditory canal is the creation of an open
mastoidectomy cavity that is not self-cleaning and requires life-long regular
debridements.
25.5.8 Atticotomy-Atticoantrotomy
This technique may be performed by microscopy or endoscopy, and by retroauricular or transcanal approach. It is indicated for small cholesteatomas conned to the
attic region. During atticotomy, a wide tympanomeatal ap is created and the
exposed attic region is drilled through the meatus [1, 2]. A prominent tympanosquamous suture line is often reduced for maximal transcanal exposure. The body
of the incus, the head of the malleus, and the tympanic segment of the facial nerve
are seen. If the ossicles are involved with cholesteatoma, they are removed. Sheehy’s
cog, which may sometimes prevent cleaning of diseased air cells in the anterior
epitympanum is drilled to expose the supratubal recess [1]. If the pathology extends
posteriorly and reaches the antrum, drilling of the external auditory canal must proceed posteriorly to expose the antrum (atticoantrotomy). The resulting external
auditory canal bony defect should be repaired to prevent recurrent retraction cholesteatoma. This procedure can be combined with a simple mastoidectomy and epitympanectomy, or performed without a previous mastoidectomy. In this latter case,
drilling of the posterior external auditory canal continues in a retrograde fashion
into the antrum using the outline of tegmen tympani as guide. The cholesteatoma or
disease is removed by turning it from the inside to the outside, towards the ear canal
(i.e., retrograde “inside-out mastoidectomy”). A modication of the standard atticoantrostomy was described by Bondy in 1910. In the Bondy procedure, only the
disease in the attic region is resected without touching the middle ear and the ossicular chain [1, 2]. The Bondy procedure is only suitable for cholesteatomas limited
to the attic that do not extend medial to the ossicular chain and into the middle ear.
25.5.9 Mastoidectomy withWhole Canal Removed
The posterior wall of the external auditory canal is removed completely down to the
fallopian canal; the resulting prominence of the fallopian canal is called the facial
ridge. All mastoid air cells are exenterated, and disease is cleaned. The resulting
mastoid cavity is now open to the external auditory canal. The cavity should have
wide and smooth edges. In the modifed radical mastoidectomy technique, the
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