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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_4507_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Preface
- •Contents
- •1.3.4 The Eustachian Tube
- •1.3.5 Muscles
- •1.3.6 Innervation
- •1.3.7 Vascular Supply
- •1.4 The Inner Ear (Labyrinthine Cavity)
- •1.4.1 The Vestibule
- •1.4.2 Semicircular Canals
- •1.4.4 The Cochlea
- •1.4.5 Innervation
- •1.1 Introduction
- •1.2 The External Ear
- •1.2.1 The Auricle
- •1.2.3 The Eternal Auditory Canal/External Acoustic Meatus
- •1.3 The Middle Ear (Tympanic Cavity)
- •1.3.1 The Tympanic Membrane
- •1.3.3 Ossicles
- •1.4.6 Cochlea Nerve Anatomy
- •1.4.7 Vestibular Nerves
- •1.4.8 The Vestibulocochlear Nerve
- •1.5 The Central Hearing System
- •1.5.3 Auditory Input
- •1.5.4 The Auditory Nerve’s Descending Routes
- •References
- •2: Outer–Middle–Inner Ear Embryology
- •2.1 Introduction
- •2.2 Embryology
- •2.3.1 First Week
- •2.3.3 Third Week
- •2.3.4 Fourth Week
- •2.3.5 Sixth Week
- •References
- •3.1 Introduction
- •3.3 The Outer Ear
- •3.3.1 Anatomy
- •3.3.3 Localization
- •3.4 The Middle Ear
- •3.4.3 Middle Ear Muscles
- •3.4.4 The Eustachian Tube
- •3.4.5 Impedance Matching
- •3.5 The Inner Ear
- •3.5.1.1 Lateral Wall
- •3.5.1.2 Reissner’s Membrane
- •3.5.1.3 The Basilar Membrane
- •3.5.2.1 Hair Cells
- •Inner Hair Cells
- •Outer Hair Cells
- •3.5.3 The Tectorial Membrane
- •3.5.4 The Osseous Spiral Lamina
- •3.5.5 Cochlear Mechanics
- •3.5.5.1 Passive Mechanics
- •3.5.5.2 Active Mechanics
- •3.6.1 Auditory Nerve Fibers
- •3.6.2 The Subcortical Auditory Nuclei
- •3.6.2.1 The Cochlear Nucleus
- •3.6.2.2 The Superior Olivary Complex
- •3.6.2.3 The Lateral Lemniscus
- •3.6.2.4 Inferior Colliculus
- •3.6.2.5 The Medial Geniculate Body
- •3.6.3 The Auditory Cortex
- •3.7 Conclusion
- •References
- •4.1 Introduction
- •4.2 Eustachian Tube Anatomy
- •4.4 Eustachian Tube Dysfunction
- •References
- •5: Temporal Bone Radiology
- •5.1.1 Introduction
- •5.1.2 Computed Tomography (CT)
- •5.1.3 Temporal Bone CT Angiography
- •5.1.4 Magnetic Resonance Imaging (MRI)
- •5.1.5 Diffusion-Weighted Imaging (DWI)
- •5.1.6 Conclusion
- •5.2.1 Introduction
- •5.2.2.1 The External Auditory Canal (EAC)
- •5.2.3 Temporal Bone Fractures
- •5.2.4 Conclusion
- •5.3.1 Introduction
- •5.3.2 Necrotizing Otitis Externa
- •5.3.3 Middle Ear
- •5.3.3.2 Chronic Otitis Media
- •5.3.3.3 Cholesteatomas
- •5.3.3.4 Cholesterol Granulomas
- •5.3.4 Inner Ear
- •5.3.4.1 Labyrinthitis
- •5.3.4.2 Petrous Apicitis
- •5.3.5 Conclusion
- •5.4.1 Introduction
- •5.4.2.1 Cerebellopontine Angle Tumors
- •Vestibular Schwannomas
- •Arachnoid Cysts
- •Meningiomas
- •5.5.2 External Auditory Canal Aplasia
- •5.5.4 Inner Ear Malformations
- •5.5.4.1 Complete Labyrinthine Aplasia/Michel Anomaly
- •5.5.4.2 Rudimentary Otocysts
- •5.5.4.3 Common Cavity Malformation
- •5.5.4.4 Incomplete Partition (IP) Type I
- •5.5.4.5 Incomplete Partition Type II/Mondini Malformation
- •5.5.4.6 Incomplete Partition Type III
- •5.5.4.7 Cochlear Anomalies
- •5.5.4.8 Semicircular Canal Anomalies
- •5.5.6 Conclusion
- •5.6.1 Introduction
- •5.6.2 Otospongiosis/Otosclerosis
- •Epidermoids
- •5.4.2.2 The Middle Ear
- •5.4.2.4 Petrous Bone
- •5.4.2.5 Metastatic Tumors
- •5.4.3 Conclusion
- •5.5.1 Introduction
- •5.6.3 Third Window Lesions
- •5.6.4 Conclusion
- •References
- •6.1 Introduction
- •6.3.1 What Is Sound?
- •6.3.2 Sound Intensity
- •6.4 Psychoacoustics
- •6.4.1 Signal Detection Theory
- •References
- •7.1 Introduction
- •7.1.1 What Is Sound?
- •7.2 Fundamental Acoustic Concepts
- •7.2.3 Period
- •7.2.4 Frequency
- •7.2.5 Wavelength
- •7.3 Psychoacoustics
- •7.3.1 Loudness
- •7.3.2 Auditory Masking
- •7.3.2.1 Simultaneous Masking
- •7.3.2.2 Temporal Masking
- •7.4.2 Spatial Hearing
- •References
- •8.1 Introduction
- •8.2 Case History
- •8.3 The Audiology Test Room
- •8.4.1 Pure-Tone Audiometry
- •8.4.1.1 Masking
- •8.4.2 Speech Audiometry
- •8.4.3 Pediatric Assessment
- •8.5.1 Acoustic Immittance Audiometry
- •8.5.1.1 Tympanometry
- •Tympanogram Interpretation
- •8.5.1.2 Multifrequency Tympanometry
- •8.5.1.3 Wideband Tympanometry
- •8.5.1.4 Acoustic Reflex Test
- •8.5.1.5 The Reflex Decay Test
- •8.5.1.6 Eustachian Tube Evaluation
- •8.5.2 Otoacoustic Emissions
- •8.5.2.2 Performing Otoacoustic Emission Tests
- •8.5.3 Auditory Evoked Potentials
- •8.5.3.2 Auditory Evoked Brainstem Response
- •Stimulus Types
- •Stimulus Polarity
- •Stimulus Presentation Rate
- •Stimulus Intensity
- •Analysis Time (Recording Epoch)
- •Filters
- •Artifact Rejection Level
- •Electrodes
- •8.5.3.3 Auditory Steady-State Responses
- •8.5.3.4 Electrocochleography
- •Electrocochleography Analysis
- •8.5.3.5 Cortical Auditory Evoked Potentials
- •8.5.3.6 Event-Related Auditory Potentials
- •P300
- •Mismatch Negativity
- •Acoustic Change Complex
- •8.6 Conclusion
- •References
- •9.1 Introduction
- •9.2.3 Conductive Hearing Loss
- •9.2.4 Sensorineural Hearing Loss
- •9.2.4.1 Internal Acoustic Canal Tumors
- •9.2.4.2 Auditory Neuropathy Spectrum Disorder
- •9.2.4.3 Third Window Syndrome
- •9.2.4.4 Dead Region
- •9.2.5 Mixed Hearing Loss
- •9.3 Hearing Loss Configuration
- •9.3.3 Unilateral or Bilateral Hearing Loss
- •9.3.4 Symmetric or Asymmetric Hearing Loss
- •9.3.5 Fluctuating or Stable Hearing Loss
- •9.4 Diagnostic Tests
- •9.4.1 Pure Tone Threshold Testing
- •9.4.2 Speech Recognition Tests
- •9.4.3 Tympanometric Tests
- •9.4.4 Stapedial Reflex
- •9.4.5 Otoacoustic Emission Test
- •9.4.6 Auditory Brainstem Responses
- •9.6 Reporting Audiological Findings
- •9.7 Conclusion
- •References
- •10.1 Introduction
- •10.2.1 Anamnesis
- •10.2.2 Hearing Loss
- •10.2.3 Ear Pain (Otalgia)
- •10.2.4 Ear Discharge (Otorrhea)
- •10.2.5 Itchy Ear
- •10.2.8 Physical Examination
- •10.2.8.1 Inspection
- •10.2.8.2 Palpation
- •10.2.8.3 Otoscopy
- •10.2.12 Hearing Examination
- •10.2.13 Hearing Assessment
- •10.2.13.1 Whisper Test
- •10.2.13.2 Tuning Fork Tests
- •Rinne Test
- •Weber Test
- •Schwabach Test
- •Gelle Test
- •10.3 Conclusion
- •References
- •11.1 Introduction
- •11.2.1 Microphone
- •11.2.2 Amplifier
- •11.2.3 Receiver
- •11.2.4 Batteries
- •11.2.5 Earmolds/Domes
- •11.4 Hearing Aid Types
- •11.5.1 Directional Microphone Technologies
- •11.5.2 Digital Noise Reduction
- •11.5.3 Frequency Lowering
- •11.5.4 Feedback Canceller
- •11.5.5 Bluetooth
- •11.6 Other Hearing Aid Technologies
- •11.7 Pediatric Hearing Aid Application
- •11.7.3.7 Hearing Aid Fitting
- •Prescription Formula Preference
- •Objective Verification Tools
- •Subjective Verification Tools
- •Fine-Tuning
- •11.8 Adult Hearing Aid Application
- •11.8.1.1 Medical Evaluation
- •11.8.1.2 Audiological Evaluation
- •11.8.1.3 Physical Evaluation
- •11.8.1.4 Psychological Evaluation
- •11.8.2 Hearing Aid Application Process
- •11.8.2.1 Anamnesis
- •11.8.2.6 Hearing Aid Fitting
- •Fine-Tuning
- •11.9 Conclusion
- •11.10 Case Studies
- •11.10.1 Case 1
- •11.10.2 Case 2
- •11.10.3 Case 3
- •11.10.4 Case 4
- •References
- •12.1 Introduction
- •12.3.1 Pathophysiology
- •12.3.2 Management
- •12.3.3 Etiology
- •12.3.4 Epidemiology
- •12.3.5 Assessing
- •12.3.6 Treatment
- •References
- •13: Otoplasty
- •13.1 Introduction
- •13.2 General Information
- •13.2.1 Auricular Anthropometry
- •13.3 History
- •13.8.1 Conservative Treatment
- •13.8.2 Surgical Treatment
- •13.11 Patient Follow-Up
- •13.12 Case Examples
- •13.13 Complications
- •13.13.1 Early Complications
- •13.13.2 Late Complications
- •13.13.3.1 Telephone Ear Deformity
- •13.13.3.2 Reverse Telephone Ear Deformity
- •13.13.3.5 Antihelical Malposition
- •13.13.3.6 Tragal Prominence
- •13.13.3.7 Auricular Lines
- •13.14 Revision Otoplasty
- •References
- •14: External Ear Tract Diseases
- •14.1 Introduction
- •14.2.1 Atopic Dermatitis
- •14.2.2 Allergic Contact Dermatitis
- •14.2.3 Photoallergic Dermatitis
- •14.2.4 Psoriasis
- •14.2.5 Relapsing Polychondritis
- •14.2.6 Gout
- •14.3 Traumatic Disorders
- •14.3.1 Irritant Contact Dermatitis
- •14.3.2 Phototoxic Dermatitis
- •14.3.3 Phototrauma
- •14.4 Infectious Diseases
- •14.4.1 Otitis Externa
- •14.4.1.1 Background
- •14.4.1.2 Anatomy
- •14.4.1.3 Classification
- •14.4.1.5 Diagnosis
- •14.4.1.6 Management
- •References
- •15: Auricula Tumors
- •15.1 Introduction
- •15.2 Benign Tumors
- •15.2.1 Chondrodermatitis Nodularis Chronica Helicis
- •15.2.2 Cystic Chondromalacia
- •15.2.3 Ceruminous Gland Adenoma
- •15.3 Malign Tumors
- •15.3.1 Basal Cell Carcinoma (BCC)
- •15.3.2 Squamous Cell Carcinoma
- •15.3.3 Ceruminous Gland Adenocarcinoma
- •15.4 Conclusion
- •References
- •16: Acute Suppurative Otitis Media
- •16.1 Introduction
- •16.2 Pathophysiology
- •16.3 Etiology
- •16.3.1 Host Factors
- •16.3.1.1 Immune System
- •16.3.1.2 Hereditary Susceptibility
- •16.3.1.3 Mucins
- •16.3.1.4 Anatomic Abnormalities
- •16.3.1.5 Physiologic Dysfunction
- •16.3.2 Infectious Factors
- •16.3.2.1 Bacterial Pathogens
- •16.3.2.2 Viral Pathogens
- •16.3.3 Environmental Factors
- •16.3.3.1 Infant Feeding Methods
- •16.4 Classification
- •16.6 Diagnosis
- •16.7 Treatment
- •16.7.1 Antibiotic Therapy Versus Observation
- •16.7.2 Initial Antibiotic Therapy
- •16.7.3 Supplemental Programs
- •References
- •17.1 Introduction
- •17.2 Definition
- •17.4 Pathophysiology
- •17.5 Diagnosis
- •17.5.1 Clinical Evaluation
- •17.6 Treatment
- •17.6.1 Medical Treatment
- •17.6.2 Surgical Treatment
- •17.7 Conclusion
- •References
- •18: Chronic Suppurative Otitis Media
- •18.1 Introduction
- •18.2 Epidemiology
- •18.3 Pathophysiology
- •18.4 Microbiology
- •18.5 Histopathology
- •18.6 Clinical Manifestations
- •18.6.1 Tubotympanic Type
- •18.6.2 Atticoantral Type
- •18.7 Diagnosis
- •18.7.1 Anamnesis
- •18.7.2 Otoscopic Examination
- •18.7.3 Audiological Evaluation
- •18.7.4 Imaging
- •18.8 Treatment
- •18.8.1 Medical Treatment
- •18.8.2 Surgical Treatment
- •18.9 Complications
- •18.10 Future Directions
- •18.11 Conclusion
- •References
- •19: Cholesteatoma
- •19.1 Introduction
- •19.2 Definition
- •19.3 Epidemiology
- •19.4 Histopathology
- •19.7 Cholesteatoma Types
- •19.7.1 Congenital Cholesteatoma
- •19.7.2 Acquired Cholesteatoma
- •19.7.2.2 Epithelial Migration Theory
- •19.7.2.3 Basal Cell Hyperplasia Theory
- •Tos Staging
- •Sade Staging
- •19.7.3 Unclassified Cholesteatomas
- •19.7.4 Petrous Bone Cholesteatomas
- •19.8 Practical Classification
- •19.8.1 Attic Cholesteatomas
- •19.8.2 Sinus Cholesteatomas
- •19.8.3 Pars Tensa Cholesteatomas
- •19.9 Clinical Presentations
- •19.9.1 Cholesteatoma Microbiology
- •19.10 Diagnosis
- •19.10.2 Computed Tomography
- •19.10.3 Magnetic Resonance Imaging
- •19.10.4 Audiometric Evaluation
- •19.11.1 Closed Techniques
- •19.11.2 Open Techniques
- •19.12 Conclusion
- •References
- •20.1 Introduction
- •20.2 Physiology
- •20.2.4 Tympanic Isthmus
- •20.4 Pathophysiology
- •20.5 Clinical Picture
- •20.6 Management
- •20.6.1 Surgical Management
- •20.6.1.2 Tympanoplasty
- •20.6.1.3 Mastoid Surgery
- •20.7 Adhesive Otitis Media
- •20.7.1 Pathogenesis
- •20.7.2 Clinical Findings
- •20.7.3 Imaging
- •20.7.4 Treatment
- •20.8 Conclusion
- •References
- •21.1 Introduction
- •21.2 Intratemporal Complications
- •21.2.1 Acute Mastoiditis
- •21.2.2 Facial Nerve Paralysis
- •21.2.3 Labyrinthitis
- •21.2.4 Labyrinthine Fistula
- •21.2.5 Petrositis
- •21.3 Intracranial Complications
- •21.3.1 Meningitis
- •21.3.2 Lateral Sinus Thrombosis
- •21.3.3 Brain Abscess
- •21.3.4 Otitic Hydrocephalus
- •21.3.5 Epidural Abscess
- •21.3.6 Subdural Empyema
- •21.4 Conclusion
- •References
- •22: Basic Otological Surgical Techniques
- •22.1 Introduction
- •22.3 Atticotomy
- •22.4 Mastoidectomy
- •22.4.1 Simple (Cortical) Mastoidectomy
- •22.4.2 Canal Wall-Up Mastoidectomy
- •22.4.3 Canal Wall-Down Mastoidectomy
- •22.4.4 Retrograde Mastoidectomy
- •22.4.5 Modified Radical Mastoidectomy
- •22.4.6 Radical Mastoidectomy
- •22.4.7 Mastoid Obliteration
- •22.5 Petrosectomy
- •22.6 Conclusion
- •References
- •23: Tympanoplasty
- •23.1 Introduction
- •23.2.1 Chronic Otitis Media
- •23.2.2 Traumatic Perforations
- •23.5 Tympanoplasty Types
- •23.7 Graft Materials
- •23.8 Graft Techniques
- •23.8.1 The Perichondrium/Cartilage Island Graft
- •23.8.2 The Palisade Graft
- •23.8.3 The Temporalis Fascia Graft
- •23.9 Surgical Approaches
- •23.9.1 Microscopic Approach
- •23.9.2 Endoscopic Approach
- •23.10.1 Transmeatal Incisions
- •23.10.1.1 The Rosen Incision
- •23.10.1.3 Anterior Tympanomeatal Flap
- •23.10.2 Endaural Incision
- •23.10.3 Postauricular Incision
- •23.11 Pediatric Tympanoplasty
- •23.12 Prognostic Factors
- •23.14 Conclusion
- •References
- •24: Ossiculoplasty
- •24.1 Introduction
- •24.4 Indications/Contraindications
- •24.5 Reconstruction Materials
- •24.7 Surgical Preparation
- •24.8 Surgical Technique
- •24.9 Ossiculoplasty Results
- •24.10 Complications
- •24.11 Postoperative Care
- •24.12 Follow-Up
- •24.13 Conclusion
- •References
- •25: Tympanomastoidectomy
- •25.1 Introduction
- •25.2 Surgical Anatomy
- •25.4 Indications
- •25.5 Technique
- •25.5.1 Patient’s Preparation
- •25.5.3 Simple Mastoidectomy
- •25.5.4 Posterior Tympanostomy or Facial Recess Approach
- •25.5.5 Epitympanectomy
- •25.5.6 Endolymphatic Sac Procedures
- •25.5.8 Atticotomy-Atticoantrotomy

440
Fig. 21.5 Subdural empyema in temporal region
S. Şerier et al.
treatment is started to treat the main cause of the condition. If the empyema is due
to AOM, myringotomy is included in the treatment plan. In cases associated with
COM, mastoidectomy is performed. In appropriate clinical circumstances, mastoidectomy and craniotomy may be performed simultaneously [59].
21.4 Conclusion
The use of antibiotics has reduced the incidence of complications. Early diagnosis
is a critical element in preventing these potential complications and requires sound
clinical knowledge. Although some fatal complications are unavoidable, timely
diagnosis and appropriate treatment can reduce or even prevent the damage that can
result from such complications.
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443

Basic Otological Surgical Techniques
22
HakanBayraktar, MeteIseri, andMarcoMandala
22.1 Introduction
Chronic suppurative otitis media (CSOM) is a chronic infectious inammation of
the middle ear and its associated spaces that lasts longer than 3months. Its main
features are tympanic membrane perforation, ear discharge, and hearing loss. In
addition to the CSOM clinic, the presence of subepithelial squamous cell keratinized epithelium accumulation in the middle ear and mastoid cavity and cholesteatoma accompanied by an inammatory layer around it is dened as CSOM with
cholesteatoma. Treatment of both CSOM and CSOM with cholesteatoma is surgical
intervention. Tympanoplasty and/or mastoidectomy techniques have been dened
as two main surgical approaches.
22.2 Tympanoplasty, Ossiculoplasty, andMyringoplasty
Myringoplasty, tympanoplasty, and ossiculoplasty are techniques used in the surgical management of acute and chronic middle ear disease. The goal of tympanoplasty is to treat existing middle ear disease, restore functional hearing, restore
middle ear ventilation, and create a self-cleaning external auditory canal (EAC).
Myringoplasty only treats a perforated eardrum. It does not involve the middle
ear and mastoid interventions. Myringoplasty is usually used for small and mediumsized perforations with no ossicular chain involvement. Fat, temporal muscle fascia,
H. Bayraktar (*)
Department of Otorhinolaryngology, Kocaeli City Hospital, Kocaeli, Turkey
M. Iseri
Private ENT Clinic, Kocaeli, Turkey
M. Mandala
Department of Otolaryngology, University of Siena, Siena, Italy
© The Author(s), under exclusive license to Springer Nature
Switzerland AG 2024
M. T. Kalcioglu et al. (eds.), Otology Updates, Comprehensive ENT,
https://doi.org/10.1007/978-3-031-76173-7_22
445

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perichondrium, and cartilage with composite perichondrium are used as autografts.
It is often used in the clinic as a fat and buttery myringoplasty. Allografts such as
bovine pericardium collagen or dehydrated derma from donors can be used in case
of repeated procedures with limited availability of autograft.
Ossiculoplasty is an additional surgical procedure that completes the tympanoplasty. The goal of ossiculoplasty is to transfer mechanical energy from the tympanic membrane to the base of the stapes via the ossicular chain. Ossiculoplasty
uses autograft, homograft, and allograft materials for ossicular chain defects.
Autograft materials are divided into bone and cartilage. Incus bone is the most
commonly used material, although cortical mastoid bone may be used in rare cases.
Concha, cymba, and tragus are used as cartilage materials. Autograft is inexpensive
and easy to obtain, biocompatible, and non-extruded, which are its main advantages. However, there are also disadvantages, such as nutritional problems of the
bone and cartilage tissue and the associated demineralization process, xation to
surrounding tissues, and cholesteatoma recurrence.
Homograft materials are taken from the same site, so they adapt to the anatomy.
Graft rejection is rare. There is also a risk of infection, especially with blood-borne
and tissue-borne diseases.
Allograft materials are easy to use, but their disadvantages are cost and biocompatibility (extruded prosthesis). Cartilage grafts are used between the prosthesis
head and the eardrum to prevent extrusion of the allograft prosthesis. Prosthetic
materials can be polymer, ceramic, or metal. Titanium prostheses are more commonly used today, while polymer and ceramic prostheses were more commonly
used in the past. The main advantages of titanium prostheses are their stiffness, tissue compatibility, lightweight, and durability. However, some studies have found no
signicant difference in postoperative hearing outcomes between titanium and nontitanium prostheses [1]. Prostheses are divided into partial ossicular replacement
prosthesis (PORP) for stapes suprastructure cases and total ossicular replacement
prosthesis (TORP) for stapes base cases. In addition, the shape of the prosthesis
may change depending on the tissue (tympanic membrane or malleus) in contact
with the head of the prosthesis. The presence of a stapes suprastructure is extremely
important for successful postoperative hearing outcomes. This is supported by studies reporting better hearing outcomes in cases with PORP than TORP [2–6].
22.2.1 Transcanal, Endaural, andRetroauricular Approaches
The transcanal approach is often used for acute traumatic and/or posterior quadrant
perforations. It is used in cases where the perforation margins are clearly visible on
direct microscopic examination with the ear speculum and in large EACs. The procedure refreshes the acute perforation edges, turns the epithelium outward, and lls
the middle ear with spongostan for support. Myringoplasty in acute perforations is
completed by placing the irregular edges of the perforation on the spongostan and
placing it on the gel lm, silastic sheet, or paper membrane. Repaired tympanic
membrane perforations and tympanomeatal aps are supported by silastik sheets

22 Basic Otological Surgical Techniques
447
and spongels at the end of the operation. The transcanal approach is commonly used
in surgery for chronic otitis media and has recently been increasingly used in endoscopic ear surgery. A rectangular tympanomeatal ap with the medial base of the
EAC is usually raised between the 12 o’clock and 6 o’clock positions to allow the
endoscope to enter the middle ear.
The endaural approach views the tympanic membrane more posteriorly than the
transcanal approach in microscopic ear surgery. This allows both posterior and limited anterior quadrant perforations to be treated with the transcanal approach. An
endaural incision combined with canalplasty can treat all anterior quadrant perforations. In addition to tympanoplasty/myringoplasty, ossiculoplastic procedures such
as exploratory tympanotomy and otosclerosis surgery can be performed using this
approach. In the endaural approach, a mini-incision is made in the incisura terminalis between the tragus and the helix, which does not involve the cartilage and extends
to the inferior border of the temporal muscle and the EAC bone. This approach
allows simultaneous removal of the temporal muscle fascia for grafting purposes.
The incision is then continued along the tympanosquamous suture, 6–8mm close to
the tympanic annulus, and joined to the superior border of the horizontal incision
between the 12 o’clock and 6 o’clock positions. A posteroinferior eshy skin ap is
xed posteriorly by elevation with self-retrain retractors. The middle ear is entered
under the annulus brosus by elevating the tympanomeatal ap while preserving the
chorda tympani.
The retroauricular approach allows the eardrum to be viewed from the widest
angle. All quadrants of the tympanic membrane can be seen, often without the need
for canaloplasty. It is used for anterior quadrant perforations, especially in microscopic ear surgery, as it provides the most posterior view possible. In this approach,
incisions are made approximately 1 cm posterior to the retroauricular sulcus,
between the temporal line and the mastoid tip, following the curve of the sulcus. It
passes through the subcutaneous tissue marking and cutting the postauricular muscle. It is dissected to the EAC in the supraperiosteal plane. Periosteal incisions are
made parallel and perpendicular to the temporal line, with a T-shaped incision
extending to the mastoid type. The lifted subperiosteal ap reveals the spine of
Henle, the EAC, the tympanomastoid, and the squamous sutures. The EAC is then
accessed with a medial sharp dissection and the tympanomeatal ap is elevated with
a triangular or rectangular incision. The retroauricular approach allows access to
graft material such as temporal muscle, conchal cartilage, and perichondrium
through the same incision.
22.2.2 Canalplasty andMeatoplasty
Both the endaural and retroauricular approaches in microscopic ear surgery should
be mastered in each quadrant of the tympanic membrane and all perforation margins
for intraoperative and postoperative follow-up. For this reason, widening of the
bony EAC is called canalplasty and widening of the cartilaginous EAC is called
meatoplasty. In canalplasty, all walls, usually the anterior and inferior walls, are

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drilled to expose the tympanic annulus. Canalplasty is used to treat cases such as
EAC cholesteatoma, exostosis, and osteoma.
Conversely, in cases of canal wall-down (CWD) tympanoplasty, meatoplasty is
used to remove the posterior and superior walls of the EAC.Removal of skin and/or
conchal cartilage widens the entrance to the EAC.
22.2.3 Graft Selection andGrafting Technique
Temporal muscle fascia, perichondrium, and cartilage are commonly used as grafts.
Tragal and conchal cartilage are the cartilage sources. Tragal cartilage is preferred
for near-total perforations because it is atter and thinner than conchal cartilage. It
is harder than the fascia and the structure of the cartilage increases its resistance to
retraction. It is used in patients with chronic Eustachian tube dysfunction, retraction
pouch, and tympanic membrane adhesion. It is used between the hearing prosthesis
and the tympanic membrane to prevent prosthesis extrusion. It is also used to close
surgical defects such as atticotomy and cavity obliteration [7]. The cartilage graft
can be used as a palisade, en-bloc, perichondrium composite, malleus notched, or
bilobular cartilage island (pac-man grafth), depending on the surgeon’s preference
and the replacement tissue. The main disadvantages of the cartilage graft compared
to the fascia and perichondrium are its reduced elasticity (lower hearing performance) and the impossibility of looking through. In this case, a palisade-shaped
cartilage can be used in appropriate cases so that chronic otitis media (COM) cases
with cholesteatoma do not present limitations in the postoperative follow-up.
Graft placement is divided into lateral (overlay) and medial (underlay). A graft
placed laterally to the tympanic membrane and bone annulus is dened as overlay,
while those placed medially are dened as underlay. A total overlay technique is
performed by drilling a secondary bony annulus and grafting. In the underlay technique, the graft is placed under the mallei of the manubrium, covered by the skin of
the EAC and/or the residual tympanic membrane.
The fascial graft material, often supported by spongostan in the middle ear, is
placed laterally of the manubrium and under the anterior–inferior bony annulus.
22.3 Atticotomy
Removal of the lateral wall of the epitympanum by drilling allows visualization of
the incudomalleolar joint. This technique is used in cases of attic retraction pouch
and cholesteatoma, as well as in repair of ossicular chain xation, including the
malleus and incus. In particular, it is used in cholesteatoma surgery to remove disease that remains lateral to the head of the malleus and body of the incus. It can be
used as an adjunct to tympanoplasty and in conductive hearing loss to visualize the
ossicular chain during second-look surgery.

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22.4 Mastoidectomy
Cortical mastoidectomy was rst performed by Schwartze [8, 9] in 1873 and radical
mastoidectomy (RM) by Zaufal [10] in 1890. Bondy [8] described modied RM in
the following years, while mastoidectomy was the primary treatment in the preantibiotic era. Jansen [11] and Sheehy and Patterson [12] described intact canal
mastoidectomy with a facial recess approach that preserved hearing and normal
anatomy. In addition to COM surgery, mastoidectomy is used for cochlear implantation, facial nerve decompression, labyrinthine surgery, endolymphatic sac surgery,
and petrosectomy.
22.4.1 Simple (Cortical) Mastoidectomy
The antrum is often reached by partial opening of the mastoid air cells. It is used to
treat acute complications of otitis media such as subperiosteal abscesses and/or
coalescent mastoiditis.
22.4.2 Canal Wall-Up Mastoidectomy
The canal wall-up (CWU) approach requires more mastoid air cells to be opened
than cortical mastoidectomy. The facial recess approach is added to the CWU technique to control the disease in the hypo-retrotympanum, especially in cases of COM
with cholesteatoma. It can be associated with atticotomy to explore the epitympanum. Cochlear implant surgery is performed with a combination of cortical mastoidectomy and posterior tympanotomy.
Surgical Technique A C-shaped incision is made 1cm posterior to the retroau-
ricular sulcus. The temporal muscle is identied and muscle fascia graft material is
harvested from the superior temporal line at the superior border of the incision.
Composite grafts with perichondrium are harvested from tragus or conchal cartilage, depending on the surgeon’s preference. Preparation for tympanoplasty is
described in the retroauricular approach section. The posterior ap of the T-shaped
periosteal incision is elevated posteriorly.
The mastoid cortex is exposed with self-retrain retractors. Bone incisions parallel and perpendicular to the temporal line and extending to the mastoid tip, such as
the periosteal incision, are made with the burr. Drilling is performed with the largest
burr available and accompanied by irrigation. Attention must be paid to highlight
the middle fossa dura leaving some bony tissue for its protection. The cortical mastoidectomy is then performed on cells and the osseous septum (Körner) is reached
by drilling parallel to the sigmoid sinus. The antrum is reached at the projection of
the area cribrosa, following the mastoid tegmen and drilling further medially. Bony
structures on the EAC, sigmoid sinus, and tegmen are thinned and the digastric

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ridge is exposed inferiorly. The antrum is drilled anteriorly to dominate the aditus
and epitympanum. The course of the facial nerve should be determined during the
mastoidectomy. In this way, the facial nerve will be preserved in any surgical
maneuver. The short process of the incus and the lateral semicircular canal (LSC)
on the oor of the antrum are recognized as the dominant facial nerve localizations.
The second elbow of the facial nerve and proximal to the mastoid segment lie medial
and anterior to the LSC. The mastoid segment of the facial nerve is located at the
medial part of the EAC and 1–2mm medial to the short process of the incus.
The CWU mastoidectomy and the facial recess approach (posterior tympanotomy) can be combined in some cases, depending on the disease. The limits of the
facial recess approach are the triangular bone formed by the chorda tympani laterally, the mastoid segment of the facial nerve medially, and the fossa incudis superiorly, after the EAC has been thoroughly thinned and the middle ear entered from the
facial recess. In the middle ear, the pyramidal eminence, the stapes and the long
process of the incus, the promontorium, the round window niche, the handle of the
malleus, and the tympanic membrane can be seen. Even the entire tympanic sinus
and hypotympanum can be seen in cases with appropriate anatomy. Tympanic
membrane repair in COM surgery is performed with tympanoplasty after the disease has been cleared from the mastoid cavity, antrum, and middle ear.
22.4.3 Canal Wall-Down Mastoidectomy
The posterior and superior walls of the EAC are removed by drilling in the CWD
approach as opposed to the CWU.The wall of the EAC is lowered to the facial ridge
after the level and location of the facial nerve is revealed, and the tympanic sinus
and facial recess are dominated. Therefore, the mastoid cavity opens into the
EAC.The superior wall of the attic and the cog are removed by drilling, exposing
the supratubal recess and the Eustachian tube orice can be seen. After complete
cleaning of the cholesteatoma or related disease, all surfaces are drilled with a diamond burr, and a at surface is prepared for epithelialization. The cartilaginous
portion of the EAC is enlarged with meatoplasty and postoperative cavity and disease management is followed. The tympanic membrane is repaired with grafts while
ossiculoplasty can be performed in one single procedure or postponed for a second
look placing removed ossicles in the mastoid cavity, depending on the severity of
the disease.
This technique is preferred in patients with contralateral severe-profound hearing loss, high anesthesia risk, and difculties in postoperative clinical follow-up. In
addition, a lowered canal wall facilitates the management of COM complications
such as LSC dehiscence. Depending on the extent of cholesteatoma in the perioperative assessment, CWD may be preferred in the presence of a low tegmen tympani
level, anterior location of the sigmoid sinus, and deep retrotympanic sinuses.
However, an endoscope may be used in both CWU and CWD tympanomastoidectomies if the boundaries of the cholesteatoma are not fully visualized.
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