Chiari (1878) described the anal glands, branched glandular structures with a stratified columnar epithelium lining, approximately six to ten in number, usually more numerous in men than in women. These glands are equally allocated around the circumference of the anal canal, with their ducts opening into the base of the crypts of Morgagni, and are situated above the anal valves at the level of the dentate line (de Miguel Criado et al. 2012). The majority of the glands are subepithelial, with some located in the longitudinal layer of the internal sphincter, although others may terminate in the inter-sphincteric space, next to the external sphincter (de Miguel Criado et al. 2012). Infection caused by the obstruction of the anal crypts is the most plausible pathogenic mechanism in cryptogenic perianal fistulas (Abou-Zeid 2011; Sneider and Maykel 2013; Balcı et al. 2019). The infection begins in the inter­sphincteric space, where it may spread relatively rapidly into adjacent anatomical compartments (Abou-Zeid 2011; Sneider and Maykel 2013; Balcı et al. 2019). In particular, an abscess growing in a superficial gland could spontaneousl y discharge into the anal canal (de Miguel Criado et al. 2012). However, the rupture of an abscess located deep to the internal sphincter, which can serve as a barrier, results in pus traveling along the inter-sphincteric space, which represents the path with the least resistance; from this site it reaches the skin, forming an inter-sphincteric fistula (Parks 1961). Alternatively, a trans-sphincteric fistula forms when the infection passes through both layers of the external sphincter, and a supra-sphincteric fistula forms when the infection spreads around the entire external sphincter and part of the levator ani to reach the ischiorectal fossa (Parks 1961). The infection can reach the perianal skin where it can drain itself (Abou-Zeid 2011; Balcı et al. 2019).