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- •Foreword
- •Preface
- •Second Edition Clinical Decision Making
- •Acknowledgments
- •Contents
- •Editors and Contributors
- •Editors
- •Contributors
- •Refer to Algorithm in Fig. 1.1
- •Conclusion
- •Suggested Reading
- •1: Anorectal Examination
- •Suggested Reading
- •3: Physiologic Testing
- •Refer to Algorithm in Fig. 3.3
- •Suggested Reading
- •Refer to Algorithm in Fig. 4.1
- •Single Center Studies
- •Special Considerations
- •Low Rectal or Coloanal Anastomosis
- •Multi-center Studies
- •Suggested Reading
- •Summary
- •Suggested Reading
- •Introduction
- •Refer to Algorithm in Fig. 6.1
- •Minimally Invasive Colorectal Surgery
- •Intraoperative Fluid Administration
- •Analgesia
- •Venous Thromboembolism Prophylaxis
- •Surgical Site Infection Prevention
- •Postoperative Analgesia
- •Intravenous Fluid Management
- •Early Oral Feeding
- •Early Ambulation
- •Conclusion
- •Suggested Reading
- •Refer to Algorithm in Fig. 7.1
- •Refer to Algorithm in Fig. 7.2
- •Melena Caused by Upper Gastrointestinal Bleeding
- •Hematochezia Caused by Anorectal Bleeding
- •Severe Hematochezia Causing Hemodynamic Instability
- •Suggested Reading
- •Suggested Reading
- •Suggested Reading
- •10: Anal Conditions: Anal Fissure/Recurrent Anal Fissure
- •Suggested Reading
- •Suggested Reading
- •12: Anorectal Abscess
- •Suggested Reading
- •13: Anal Conditions: Fistula-in-Ano
- •Suggested Reading
- •14: Anal Conditions: Rectovaginal Fistula
- •Refer to Algorithm in Fig. 14.1
- •Background
- •Etiology
- •Evaluation
- •Treatment
- •Ileoanal Pouch-Vaginal Fistulas
- •Vaginal Approaches
- •Conclusion
- •Suggested Reading
- •15: Anal Conditions: Anorectal Crohn’s Disease—Fistula
- •Introduction
- •Conclusion
- •Suggested Reading
- •Suggested Reading
- •Suggested Reading
- •18: Anal Conditions: External Hemorrhoids
- •Introduction
- •Refer to Algorithm in Fig. 18.4
- •Suggested Reading
- •Refer to Algorithm in Fig. 19.1
- •D. Hair Removal
- •Suggested Reading
- •20: Anal Conditions: Pruritus Ani
- •Suggested Reading
- •21: Anal Conditions: Hidradenitis Suppurativa
- •Suggested Reading
- •22: Anal Conditions: Anorectal Trauma
- •Suggested Reading
- •23: Anal Conditions: STDs
- •Refer to Algorithm in Fig. 23.1
- •Anal Conditions: Sexually Transmitted Diseases
- •Suggested Reading
- •24: Anal Considerations: Fournier’s Gangrene
- •Refer to Algorithm in Fig. 24.1
- •Suggested Reading
- •25: Non-healing Perineal Wounds
- •Suggested Reading
- •26: Anal Intraepithelial Neoplasms
- •Diagnoses
- •Suggested Reading
- •27: Anal Conditions: Anal Margin Tumors
- •Suggested Reading
- •28: Invasive Anal Canal Neoplasia
- •Suggested Reading
- •29: Pelvic Floor Conditions: Rectal Prolapse/Recurrence
- •Suggested Reading
- •30: Pelvic Floor Conditions: Rectal Intussusception
- •Suggested Reading
- •31: Pelvic Outlet Obstruction
- •Suggested Reading
- •32: Pelvic Floor Conditions: Biofeedback
- •Background
- •Pelvic Floor Dysfunction
- •Biofeedback Therapy
- •Suggested Reading
- •33: Pelvic Floor Conditions: Fecal Incontinence
- •Fiber Supplementation
- •Medications
- •Biofeedback
- •End-to-End Sphincteroplasty
- •Tibial Nerve Stimulation
- •Graciloplasty
- •Gluteoplasty
- •∗Other Therapies
- •Injectables
- •RF Remodeling
- •Conclusion
- •Suggested Reading
- •34: Pelvic Floor Conditions: Diarrhea
- •Refer to Algorithm in Fig. 34.1
- •Suggested Reading
- •35: Chronic Constipation
- •Introduction
- •Diagnosis
- •Management
- •Suggested Reading
- •36: Retrorectal Tumors
- •Evaluation
- •Risk Assessment
- •Pathology: Four Tissue Types
- •Treatment
- •Suggested Reading
- •37: Rectal Cancer: Local Therapy
- •Suggested Reading
- •38: Rectal Conditions: Rectal Cancer—Proctectomy
- •Suggested Reading
- •39: Rectal Conditions: Rectal Cancer—Adjuvant and Neoadjuvant Therapy
- •Refer to Algorithm in Fig. 39.1
- •Suggested Reading
- •40: Rectal Conditions: Stage IV Rectal Cancer
- •Introduction
- •Refer to Algorithm in Fig. 40.1
- •Suggested Reading
- •Refer to Algorithm in Fig. 41.1
- •Suggested Reading
- •42: Rectal Conditions: Rectal Cancer—Postoperative Surveillance
- •Suggested Reading
- •43: Recurrent Rectal Cancer
- •Introduction
- •Refer to Algorithm in Fig. 43.2
- •A–C.
- •Carbon-Ion Radiation (CIRT)
- •Conclusion
- •Suggested Reading
- •44: Locally Advanced Rectal Cancer
- •Suggested Reading
- •45: Colonic: Diverticulitis
- •Refer to Algorithm in Fig. 45.1
- •Suggested Reading
- •46: Colonic Conditions: Large Bowel Obstruction
- •Suggested Reading
- •47: Colonic Conditions: Volvulus
- •Refer to Algorithm in Fig. 47.1
- •Introduction
- •Suggested Reading
- •48: Colonic Stricture
- •Suggested Reading
- •49: Acute Colonic Pseudo-Obstruction (ACPO): Ogilvie’s Syndrome
- •Suggested Reading
- •50: Colonic Conditions: Irritable Bowel Syndrome (IBS)
- •Introduction
- •Suggested Reading
- •51: Colorectal Trauma
- •Suggested Reading
- •52: Endometriosis
- •Suggested Reading
- •53: Colonic Conditions: Ulcerative Colitis
- •Conclusions
- •Suggested Reading
- •54: Colonic Conditions: Indeterminate Colitis
- •Suggested Reading
- •55: Colonic Conditions: Toxic Colitis
- •Medical Management
- •Risk Assessment
- •Surgical Management
- •Suggested Reading
- •56: Crohn’s Colitis
- •Suggested Reading
- •57: Ischemic Colitis
- •Suggested Reading
- •58: Colonic Conditions: Infectious Colitis
- •Suggested Reading
- •59: Colonic Conditions: Benign Colonic Neoplasia
- •Suggested Reading
- •60: Familial Adenomatous Polyposis
- •Suggested Reading
- •61: Colonic Conditions: Lynch Syndrome
- •Suspected Lynch Syndrome
- •Lynch Syndrome Diagnosis Without Clinical Symptoms or Phenotype
- •Suggested Reading
- •62: Malignant Colon Polyps
- •Suggested Reading
- •63: Colonic Conditions: Adenomatous Polyps
- •Suggested Reading
- •64: Colon Cancer Surgical Therapy
- •Suggested Reading
- •65: Colonic Conditions: Locally Advanced Colon Cancer
- •Conclusion
- •Suggested Reading
- •66: Recurrent Colon Cancer
- •Suggested Reading
- •67: Appendiceal Neoplasms

45 Colonic: Diverticulitis
359
Ambrosetti P, Becker C, Terrier F. Colonic diverticuli-
tis: impact of imaging on surgical management – a
prospective study of 542 patients. Eur Radiol.
2002;12(5):1145–9.
Ambrosetti P, Robert JH, Witzig JA, Mirescu D, Mathey
P, Borst F, etal. Acute left colonic diverticulitis: a prospective analysis of 226 consecutive cases. Surgery.
1994;115(5):546–50.
Angenete E, Thornell A, Burcharth J, Pommergaard H,
Skullman S, Bisgaard T, et al. Laparoscopic lavage
is feasible and safe for the treatment of perforated
diverticulitis with purulent peritonitis. Ann Surg.
2016;263(1):117–22.
Beard JD, Nicholson ML, Sayers RD, Lloyd D, Everson
NW. Intraoperative air testing of colorectal anastomoses: a prospective, randomized trial. Br J Surg.
1990;77(10):1095–7.
Binda GA, Karas JR, Serventi A, Sokmen S, Amato A,
Hydo L, etal. Primary anastomosis vs nonrestorative
resection for perforated diverticulitis with peritonitis:
a prematurely terminated randomized controlled trial.
Colorectal Dis. 2012;14(11):1403–10.
Broderick-Villa G, Burchette RJ, Collins JC, Abbas MA,
Haigh PI.Hospitalization for acute diverticulitis does
not mandate routine elective colectomy. Arch Surg.
2005;140(6):576–81.
Brook I, Frazier EH.Aerobic and anaerobic microbiology
in intra-abdominal infections associated with diverticulitis. J Med Microbiol. 2000;49(9):827–30.
Chabok A, Pahlman L, Hjern F, Haapaniemi S,
Smedh K. Randomized clinical trial of antibiotics in acute uncomplicated diverticulitis. Br J Surg.
2012;99(4):532–9.
Chautems RC, Ambrosetti P, Ludwig A, Mermillod B,
Morel P, Soravia C. Long-term follow-up after rst
acute episode of sigmoid diverticulitis: is surgery
mandatory?: a prospective study of 118 patients. Dis
Colon Rectum. 2002;45(7):962–6.
Deans GT, Krukowski ZH, Irwin ST. Malignant obstruc-
tion of the left colon. Br J Surg. 1994;81(9):1270–6.
Dharmarajan S, Hunt SR, Birnbaum EH, Fleshman JW,
Mutch MG. The efcacy of nonoperative management of acute complicated diverticulitis. Dis Colon
Rectum. 2011;54(6):663–71.
Di Saverio S, Birindelli A, Catena F, Sartelli M, Segalini
E, Masetti M, etal. The Ladies Trial: premature termination of the LOLA arm and increased adverse events
incidence after laparoscopic lavage may be inuenced by inter-hospital and inter- operator variability?
Take-home messages from a center with laparoscopic
colorectal expertise. Int J Surg. 2016;36:118–20.
Eglinton T, Nguyen T, Raniga S, Dixon L, Dobbs B,
Frizelle FA.Patterns of recurrence in patients with
acute diverticulitis. Br J Surg. 2010;97(6):952–7.
Etzioni DA, Chiu VY, Cannom RR, Burchette RJ, Haigh
PI, Abbas MA.Outpatient treatment of acute diverticulitis: rates and predictors of failure. Dis Colon
Rectum. 2010;53(6):861–5.
Feingold D, Steele SR, Lee S, Kaiser A, Boushey R,
Buie WD, et al. Practice parameters for the treat-
ment of sigmoid diverticulitis. Dis Colon Rectum.
2014;57(3):284–94.
Floch MH, Bina I. The natural history of diverticulitis:
fact and theory. J Clin Gastroenterol. 2004;38(5 Suppl
1):S2–7.
Francis NK, Sylla P, Abou-Khalil M, Arolfo S, Berler
D, Curtis NJ, et al. EAES and SAGES 2018 consensus conference on acute diverticulitis management:
evidence-based recommendations for clinical practice. Surg Endosc. 2019;33(9):2726–41. https://doi.
org/10.1007/s00464-019-06882-z. Epub 2019 Jun 27.
Gear JS, Ware A, Fursdon P, Mann JI, Nolan DJ, Brodribb
AJ, etal. Symptomless diverticular disease and intake
of dietary bre. Lancet. 1979;1(8115):511–4.
Gervaz P, Mugnier-Konrad B, Morel P, Huber O, Inan
I.Laparoscopic versus open sigmoid resection for
diverticulitis: long-term results of a prospective, randomized trial. Surg Endosc. 2011;25(10):3373–8.
Greenlee HB, Pienkos EJ, Vanderbilt PC, Byrne MP,
Mason JH, Banich FE, etal. Proceedings: acute large
bowel obstruction. Comparison of county, Veterans
Administration, and community hospital populations.
Arch Surg. 1974;108(4):470–6.
Guzzo J, Hyman N. Diverticulitis in young patients: is
resection after a single attack always warranted? Dis
Colon Rectum. 2004;47(7):1187–90.
Hall JF, Roberts PL, Ricciardi R, Read T, Scheirey C,
Wald C, et al. Long-term follow-up after an initial episode of diverticulitis: what are the predictors of recurrence? Dis Colon Rectum. 2011;54(3):
283–38.
Hinchey EJ, Schaal PG, Richards GK.Treatment of per-
forated diverticular disease of the colon. Adv Surg.
1978;12:85–109.
Hughes LE. Postmortem survey of diverticular disease
of the colon: I.Diverticulosis and diverticulitis. Gut.
1969;10(5):336–44.
Hughes LE.Postmortem survey of diverticular disease of
the colon: II. The muscular abnormality of the sigmoid colon. Gut. 1969;10(5):344–51.
Hwang SS, Cannom RR, Abbas MA, Etzioni
D.Diverticulitis in transplant patients and patients on
chronic corticosteroid therapy: a systematic review.
Dis Colon Rectum. 2010;53(12):1699–707.
Jacobs DO. Clinical practice. Diverticulitis. N Engl J
Med. 2007;357(20):2057–66.
Janes S, Meagher A, Frizelle FA. Elective surgery after
acute diverticulitis. Br J Surg. 2005;92(2):133–42.
Jones OM, Stevenson AR, Clark D, Stitz RW, Lumley
JW. Laparoscopic resection for diverticular disease:
follow-up of 500 consecutive patients. Ann Surg.
2008;248(6):1092–7.
Kaiser AM, Jiang JK, Lake JP, Ault G, Artinyan A,
Gonzalez-Ruiz C, etal. The management of complicated diverticulitis and the role of computed tomography. Am J Gastroenterol. 2005;100(4):910–7.
Keranen I, Lepisto A, Udd M, Halttunen J, Kylanpaa
L.Outcome of patients after endoluminal stent placement for benign colorectal obstruction. Scand J
Gastroenterol. 2010;45(6):725–31.

360
M. T. Brady and J. F. Hall
Kiguli-Malwadde E, Kasozi H. Diverticular disease
of the colon in Kampala, Uganda. Afr Health Sci.
2002;2(1):29–32.
Kumar RR, Kim JT, Haukoos JS, Macias LH, Dixon MR,
Stamos MJ, et al. Factors affecting the successful
management of intra-abdominal abscesses with antibiotics and the need for percutaneous drainage. Dis
Colon Rectum. 2006;49(2):183–9.
Lahat A, Yanai H, Menachem Y, Avidan B, Bar-Meir
S. The feasibility and risk of early colonoscopy in
acute diverticulitis: a prospective controlled study.
Endoscopy. 2007;39(6):521–4.
Lahat A, Yanai H, Sakhnini E, Menachem Y, Bar- Meir
S. Role of colonoscopy in patients with persistent acute diverticulitis. World J Gastroenterol.
2008;14(17):2763–6.
Lau KC, Spilsbury K, Farooque Y, Kariyawasam SB,
Owen RG, Wallace MH, et al. Is colonoscopy still
mandatory after a CT diagnosis of left-sided diverticulitis: can colorectal cancer be condently excluded?
Dis Colon Rectum. 2011;54(10):1265–70.
Manousos O, Day NE, Tzonou A, Papadimitriou C,
Kapetanakis A, Polychronopoulou-Trichopoulou A,
etal. Diet and other factors in the aetiology of diverticulosis: an epidemiological study in Greece. Gut.
1985;26(6):544–9.
Marshall J, Buchwald P, Gandhi J, Schultz J, Hider P,
Frizelle F, etal. Laparoscopic lavage in the management of Hinchey grade III diverticulitis: a systematic
review. Ann Surg. 2017;265:670–6.
Nakaji S, Danjo K, Munakaata A, Sugawara K, MacAuley
D, Kernohan G.Comparison of etiology of right-sided
diverticula in Japan with that of left-sided diverticula
in the west. Int J Color Dis. 2002;17:365–73.
Nielsen K, Richir MC, Stolk TT, van der Ploeg T,
Moormann GR, Wiarda BM, etal. The limited role of
ultrasound in the diagnostic process of colonic diverticulitis. World J Surg. 2014;38(7):1814–8.
Oberkoer CE, Rickenbacher A, Raptis DA, Lehmann K,
Villiger P, Buchli C, et al. A multicenter randomized
clinical trial of primary anastomosis or Hartmann’s procedure for perforated left colonic diverticulitis with purulent or fecal peritonitis. Ann Surg. 2012;256(5):819–26.
Painter NS, Burkitt DP.Diverticular disease of the colon:
a deciency disease of Western civilization. Br Med J.
1971;2(5759):450–4.
Painter NS, Truelove SC, Ardran GM, Tuckey M.Effect
of morphine, prostigmine, pethidine, and probanthine
on the human colon in diverticulosis studied by intraluminal pressure recording and cineradiography. Gut.
1965;6:57–63.
Poletti PA, Platon A, Rutschmann O, Kinkel K, Nyikus V,
Ghiorghiu S, etal. Acute left colonic diverticulitis: can
CT ndings be used to predict recurrence? AJR Am J
Roentgenol. 2004;182(5):1159–65.
Ricciardi R, Baxter NN, Read TE, Marcello PW, Hall
J, Roberts PL. Is the decline in the surgical treatment for diverticulitis associated with an increase
in complicated diverticulitis? Dis Colon Rectum.
2009;52(9):1558–63.
Ricciardi R, Roberts PL, Marcello PW, Hall JF, Read
TE, Schoetz DJ. Anastomotic leak testing after
colorectal resection: what are the data? Arch Surg.
2009;144(5):407–11.
Scheidbach H, Schneider C, Rose J, Konradt J, Gross
E, Barlehner E, et al. Laparoscopic approach to
treatment of sigmoid diverticulitis: changes in the
spectrum of indications and results of a prospective, multicenter study on 1,545 patients. Dis Colon
Rectum. 2004;47(11):1883–8.
Schultz J, Yaqub S, Wallon C, Blecic L, Forsmo H,
Folkesson J, et al. Laparoscopic lavage vs primary
resection for acute perforated diverticulitis. JAMA.
2015;314(13):1364.
Schussel AT, Lustik MB, Cherng NB, Maykel JA,
Quinton MH, Steele SR. Right-sided diverticulitis requiring colectomy: an evolving demographic?
A review of surgical outcomes from the national
inpatient sample database. J Gastrointest Surg.
2016;20:1874–85.
Shabanzadeh DM, Wille-Jorgensen P. Antibiotics for
uncomplicated diverticulitis. Cochrane Database Syst
Rev. 2012;(11):CD009092.
Siewert B, Tye G, Kruskal J, Sosna J, Opelka F,
Raptopoulos V, etal. Impact of CT-guided drainage in
the treatment of diverticular abscesses: size matters.
AJR Am J Roentgenol. 2006;186(3):680–6.
Slack WW. The anatomy, pathology, and some clini-
cal features of diverticulitis of the colon. Br J Surg.
1962;50:185–90.
Strate LL, Liu YL, Syngal S, Aldoori WH, Giovannucci
EL. Nut, corn, and popcorn consumption and
the incidence of diverticular disease. JAMA.
2008;300(8):907–14.
Tamim WZ, Ghellai A, Counihan TC, Swanson RS, Colby
JM, Sweeney WB. Experience with endoluminal
colonic wall stents for the management of large bowel
obstruction for benign and malignant disease. Arch
Surg. 2000;135(4):434–8.
Tan KK, Wong J, Sim R. Non-operative treatment of
right-sided colonic diverticulitis has good long-term
outcome: a review of 226 patients. Int J Color Dis.
2013;28:849–54.
Tyau ES, Prystowsky JB, Joehl RJ, Nahrwold DL.Acute
diverticulitis. A complicated problem in the immunocompromised patient. Arch Surg. 1991;126(7):855–8.
van de Wall BJ, Reuling EM, Consten EC, van Grinsven
JH, Schwartz MP, Broeders IA, et al. Endoscopic
evaluation of the colon after an episode of diverticulitis: a call for a more selective approach. Int J Color
Dis. 2012;27(9):1145–50.
Vennix S, Musters GD, Mulder IM, Swank HA, Consten
EC, Belgers EH, et al. Laparoscopic peritoneal
lavage or sigmoidectomy for perforated diverticulitis with purulent peritonitis: a multicentre, parallel-group, randomised, open-label trial. Lancet.
2015;386(10000):1269–77.
Vignati PV, Welch JP, Cohen JL.Long-term management
of diverticulitis in young patients. Dis Colon Rectum.
1995;38(6):627–9.

45 Colonic: Diverticulitis
361
Wasvary H, Turfah F, Kadro O, Beauregard W. Same
hospitalization resection for acute diverticulitis. Am
Surg. 1999;65(7):632–5.
Wess L, Eastwood MA, Wess TJ, Busuttil A, Miller A.
Cross linking of collagen is increased in colonic
diverticulosis. Gut. 1995;37(1):91–4.
Westwood DA, Eglinton TW, Frizelle FA.Routine colo-
noscopy following acute uncomplicated diverticulitis.
Br J Surg. 2011;98(11):1630–4.
Wexner SD, Talamini MA. EAES/SAGES consensus
conference on acute diverticulitis: a paradigm shift in
the management of acute diverticulitis. Surg Endosc.
2019;33(9):2724–5. https://doi.org/10.1007/s00464-
019-06998-2. No abstract available.
Whiteway J, Morson BC.Elastosis in diverticular disease
of the sigmoid colon. Gut. 1985;26(3):258–66.
Woods RJ, Lavery IC, Fazio VW, Jagelman DG, Weakley
FL.Internal stulas in diverticular disease. Dis Colon
Rectum. 1988;31(8):591–6.
Yang HR, Huang HH, Wang YC, et al. Management of
right colon diverticulitis: a 10-year experience. World
J Surg. 2006;30:1929–30.

Colonic Conditions: Large Bowel Obstruction
SarahB.Stringeld andBardC.Cosman
46
Refer toAlgorithm inFig. 46.1
A. Although large bowel obstruction (LBO) may
have a long prodromal period, the clinical
presentation is usually acute. A thorough history is important, though focus should be on
bowel movements, abdominal pain, and atus. Common associated symptoms include
abdominal distention, crampy pain, nausea
and vomiting, obstipation or constipation,
and bloating relieved by defecation.
Signicant prodromal symptoms may include
chronic constipation, stool caliber change,
and intermittent left lower quadrant pain over
months to years. A physical examination
should be performed, focusing on the abdomen to assess for tenderness, peritoneal signs,
and any mass or fullness. Additionally, the
patient should be checked for incarcerated
inguinal and femoral hernias, and a rectal
examination should be performed to assess
stool consistency and the contents of the rectal vault.
B. Due to potential morbidity and mortality, sus-
picion of LBO should lead to rapid evaluation
S. B. Stringeld
Department ofSurgery, University ofCalifornia San
Diego Health System, San Diego, CA, USA
B. C. Cosman (*)
Surgical Services, VA San Diego Healthcare System,
University ofCalifornia School ofMedicine,
San Diego, CA, USA
and surgical consultation. Pain relief, control
of vomiting with antiemetics and/or nasogastric decompression, and correction of uid
and electrolyte abnormalities should occur
simultaneously with diagnostic evaluation. It
is unproven, but reasonable, to give antibiotics with gram-negative aerobic and anaerobic
coverage to patients with suspected or conrmed LBO if the patient appears septic and
there is concern for perforation. Laboratory
studies can assess degree of dehydration and
electrolyte imbalance, and evaluate for infection, anemia, and ischemia.
C. Radiographic studies can conrm obstruction
and identify its cause or other pathology
causing the patient’s symptoms. Computed
Tomography (CT) is the imaging of choice
for suspected LBO, as it can conrm the diagnosis, identify intraluminal, mural, and extramural causes, and detect inammation and
bowel ischemia. The presence of a transition
point in the colon can make the diagnosis of
LBO, though it does not always distinguish
between mechanical obstruction and pseudoobstruction. Intravenous and oral contrast can
often help delineate between partial and complete obstruction, ileus, and small bowel
obstruction. Rectal contrast may be useful
when the suspected obstruction is in the rectum or sigmoid, and it allows a clearer distinction between mechanical and functional
obstruction. Water-soluble contrast enema
© Springer Nature Switzerland AG 2020
S. R. Steele etal. (eds.), Clinical Decision Making in Colorectal Surgery,
https://doi.org/10.1007/978-3-319-65942-8_46
363

364
Fig. 46.1 Algorithm for evaluation and management of large bowel obstruction
S. B. Stringeld and B. C. Cosman
with plain lms has been replaced by CT with
rectal contrast, but the enema itself has therapeutic value in intussusception, regardless of
what imaging modality is used to monitor it.
Plain lms are of little value when compared
to CT, although upright chest radiographs
may be useful as an initial screen for pneumoperitoneum, pneumatosis intestinalis, and
portal venous gas, suggesting a concomitant
perforation or ischemia that might necessitate
prompt surgical intervention. Sigmoid or
cecal volvulus (Fig.46.2) may have a kidneybean or giant-loop appearance on abdominal
radiographs. Abdominal radiographs may
also help distinguish constipation from LBO
and may localize the site of obstruction,
showing colonic dilation proximal to the
obstruction and a paucity or absence of gas
distal to the obstruction.
D. All patients with peritoneal signs on abdomi-
nal examination or signs of perforation or
ischemia on radiographs should undergo
prompt surgical exploration. Cecal diameter
greater than 12cm, unless it is known to be
chronic, should raise concern for impending
perforation, and urgent decompression should
Fig. 46.2 Cecal volvulus on plain lm
be considered, either via endoscopic or surgical intervention. The patient without signs of
an acute abdomen can undergo further
evaluation and consideration of whether surgical or endoscopic intervention is indicated,

46 Colonic Conditions: Large Bowel Obstruction
365
or whether non-operative measures, e.g. for
pseudo-obstruction, may be appropriate.
E. Colonic malignancy is the most common
cause of LBO.The most common locations
are the rectum, sigmoid colon, and the splenic
exure; while lesions in the right colon,
where stool is liquid, require the lumen to be
almost completely closed to cause clinical
LBO. In the absence of perforation or ischemia, endoscopic dilation and stenting of
masses and strictures may be helpful in
selected cases, and may provide an alternative to multistage surgery. Stenting or dilation
permits relief of acute obstruction, resuscitation of the patient, and mechanical bowel
preparation prior to resection and reanastomosis, thus potentially avoiding ileostomy or colostomy. Stenting is most often
successful for left-sided lesions. Right-sided
lesions and distal rectal lesions tend to be
much more difcult technically and not great
candidates. Patients with left-sided obstruction may be treated with a decompression
tube as a bridge to surgery. Palliative stenting
may be an option in patients who are poor
surgical candidates or who have advanced
cancer.
F. Acute colonic volvulus may account for
10–15% of LBO. In the United States, sigmoid volvulus is 3–4 times more common
than cecal volvulus; however, their relative
frequency varies greatly internationally. In a
stable patient, sigmoid volvulus can be treated
with endoscopic reduction and decompression. Yet, if mucosal ischemia is found on
colonoscopy, the procedure should be aborted
and the patient should undergo urgent surgical exploration. Recurrence of volvulus after
decompression is common and expected,
therefore surgical resection is indicated in all
but the sickest patients. Elective resection
should be performed in all patients with cecal
volvulus and in patients with sigmoid volvulus following successful endoscopic reduction, if they are surgical candidates from a
co-morbidity standpoint. Emergent operation
is indicated in patients for whom endoscopic
reduction is not successful.
G. Acute diverticulitis can rarely present as par-
tial or complete LBO due to bowel wall edema
and/or pericolonic inammation. Obstruction
usually occurs after multiple episodes, which
causes narrowing and stricture formation.
Diverticulitis is seen on CT as segmental,
symmetric bowel wall thickening with hyperemia, in a longer segment than a typical
malignancy (Fig. 46.3). However, it can be
difcult to distinguish radiographically
between diverticulitis and cancer, so colonoscopy and biopsy is valuable if there is the
opportunity. Principles of treatment are the
same as with malignant obstruction. Elective
resection should be offered to patients with
recurrent diverticulitis with LBO, following
resolution of an acute episode.
H. Intussusception can successfully be treated with
a contrast enema in 60–80% of cases. This is
more successful in children, in whom a pathologic lead point is unlikely. In adults, a pathologic lead point is usually present, and patients
are more likely to require elective surgery after
successful reduction by contrast enema. The
lead point should be investigated by colonoscopy after reduction of the intussusception.
Urgent operation is indicated in all patients with
signs of peritonitis or bowel perforation, or if
reduction with contrast enema is unsuccessful.
I. Colonic ileus may not exist as a distinct entity:
certainly, the colon shares in generalized ileus,
and arguably isolated colonic ileus may be the
Fig. 46.3 Diverticulitis on CT

366
S. B. Stringeld and B. C. Cosman
same as acute colonic pseudo- obstruction. In
principle, adynamic ileus of the colon is treated
with conservative measures, including correction of uid and electrolyte abnormalities and
treatment of the underlying disorder. Nasogastric
tube decompression may be helpful if the
patient is vomiting. Medications that slow
colonic motility should be stopped or avoided.
J. Acute colonic pseudo-obstruction, also called
Ogilvie’s syndrome, is treated the same as
colonic ileus if there are no signs of colonic
perforation. Additionally, IV neostigmine and/
or colonoscopic decompression may be effective. Surgery may be required in refractory
cases and those complicated by perforation.
Suggested Reading
Frago R, Ramirez E, Millan M, Kreisler E, del Valle E,
Biondo S. Current management of acute malignant
large bowel obstruction: a systematic review. Am J
Surg. 2014;207:127–38.
Jaffe T, Thompson W. Large-bowel obstruction in the
adult: classic radiographic and CT ndings, etiology,
and mimics. Radiology. 2015;275:651–63.
Lopez-Kostner F, Hool GR, Lavery IC.Management and
causes of acute large-bowel obstruction. Surg Clin
North Am. 1997;77:1265–90.
Yeo HL, Lee SW. Colorectal emergencies: review and
controversies in the management of large bowel
obstruction. J Gastrointest Surg. 2013;17:2007–12.
Zahid A, Young CJ.How to decide on stent insertion or
surgery in colorectal obstruction? World J Gastrointest
Surg. 2016;8:84–9.

Colonic Conditions: Volvulus
RichardGarnkle andMaryliseBoutros
47
Refer to Algorithm in Fig. 47.1
Introduction
Colonic volvulus accounts for 10–15% of all
large bowel obstructions in Western countries,
and is the third leading cause behind colorectal
cancer and diverticular disease. Volvulus refers
to torsion of a segment of bowel on its mesentery; colonic volvulus involves the sigmoid
colon and cecum in over 95% of cases, with the
transverse colon and splenic exure more seldom affected. With rotation of the redundant and
mobile segment of colon, closed-loop luminal
obstruction and mesenteric malperfusion ensue,
leading to the symptoms of bowel obstruction
and possible ischemia. If not treated urgently,
colonic volvulus can be fatal, and carries a variable mortality rate that can exceed 50% when
gangrenous colon is present.
The sigmoid colon accounts for the majority
of colonic volvulus, with a variable incidence.
The anatomic abnormality that predisposes to
volvulus is a long redundant sigmoid colon with
a narrow mesenteric attachment.
R. Garnkle · M. Boutros (*)
Division ofColon and Rectal Surgery, Jewish
General Hospital, Montreal, QC, Canada
The incidence of cecal volvulus has increased
steadily over the past decade. Like its sigmoidal
counterpart, cecal volvulus occurs secondary to
organoaxial rotation of the colon. The basic anatomic requirement is a sufciently mobile cecum
and ascending colon, which is present in 11–22%
of adults according to autopsy studies. Cecal bascule, a distinct and less commonly observed
entity, involves anterosuperior folding of the
cecum over a xed ascending colon. Though it
presents similarly to cecal volvulus, it does not
involve axial rotation of the bowel and thus does
not result in mesenteric vascular obstruction.
A. Sigmoid volvulus usually occurs in the
elderly with a mean age of presentation of
70years old, and affects males in a 2:1 ratio.
Patients typically have many medical comorbidities, and often suffer from chronic constipation, cathartic/laxative abuse, or colonic
motility disorders. They are also disproportionately institutionalized with neuropsychiatric conditions. In many third-world
countries, where sigmoid volvulus represents
the leading cause of large bowel obstruction,
Chagas disease and high-ber diets have been
implicated in the pathogenesis of the redundant sigmoid, and affected adults are typically younger, between 40–50 years old.
Patients with a cecal volvulus are usually
female (3:1) and 10–20 years younger than
those with sigmoid volvulus. Precipitating
© Springer Nature Switzerland AG 2020
S. R. Steele etal. (eds.), Clinical Decision Making in Colorectal Surgery,
https://doi.org/10.1007/978-3-319-65942-8_47
367

368
Fig. 47.1 Sigmoid volvulus algorithm
R. Garnkle and M. Boutros
factors include surgical adhesions, congenital
bands, pregnancy, and colonic atony.
B. Colonic volvulus may be difcult to distin-
guish from other forms of bowel obstruction
on history and physical examination alone.
Sigmoid volvulus tends to have an insidious
onset of progressive abdominal pain, followed by nausea, vomiting, abdominal distension and obstipation. Patients tend to
present after 3–4 days of symptoms, and perhaps longer in institutionalized patients. The
symptoms of cecal volvulus are similar, but
affected individuals are more likely to present
earlier with steady abdominal pain. In both
cases, physical examination may reveal varying degrees of abdominal tenderness, distension and tympany. The presence of
hemodynamic abnormalities (tachycardia or
hypotension), fever, rigidity, severe guarding,
and rebound tenderness are indicative of
colonic ischemia and/or perforation.
C. The diagnosis of colonic volvulus usually
relies on a combination of imaging modalities
(Fig. 47.2). Plain abdominal radiographs are
diagnostic of sigmoid volvulus in roughly
50% of cases, and classically feature a mark-
edly distended, gas-lled anhaustral colon
extending from the pelvis to as high as the diaphragm, referred to as the “bent inner tube”,
“omega”, or “coffee-bean” sign. The addition
of a water-soluble contrast enema can improve
the diagnostic accuracy of plain radiograph up
to 90%, giving the characteristic “bird’s beak”
sign as the contrast tapers off to the point of
obstruction. Abdominal radiographs are less
diagnostic of cecal volvulus, with only 15% of
cases readily recognizable. Unless the clinical
exam or radiograph ndings warrant urgent
exploratory laparotomy, contrast-enhanced
CT scan has become the diagnostic test of
choice for both sigmoid and cecal volvulus.
The “whirl sign” is specic for volvulus, representing the swirling appearance of the collapsed bowel and its mesentery.
D. The initial assessment of a patient with sus-
pected colonic volvulus should focus on identifying signs of colonic ischemia or
perforation. Vital sign abnormalities and/or
peritonitis on exam, free air on plain radiograph or CT scan, and leukocytosis or elevated serum lactate are all indicative of
ischemia or perforation, and the presence of

47 Colonic Conditions: Volvulus
abc
369
Fig. 47.2 Characteristic imaging ndings for sigmoid
volvulus. (a) “Coffee-bean” sign on plain radiograph.
Note the thick inner wall, representing a double-layer of
apposed bowel wall (red arrows). (Reused with permission © 2015 Springer) (b) “Bird’s beak” sign with rectal
any one of these should prompt the consideration for urgent operation. Management
begins with the general principles of bowel
obstruction and/or intra-abdominal sepsis:
uid resuscitation, correction of electrolyte
abnormalities, bladder catheterization for
urine output monitoring, nasogastric tube
insertion (to decompress the stomach and
small bowel, and to prevent aspiration), and
intravenous antibiotics in cases of ischemia
or perforation.
E. In the absence of suspected ischemia or per-
foration, the initial treatment of sigmoid volvulus is typically endoscopic detorsion with
exible sigmoidoscopy (Fig.47.1). If signs of
colonic ischemia or gangrene are observed
endoscopically (blood or dusky mucosa),
detorsion and colonic manipulation should be
immediately aborted to avoid perforation of
the bowel or bacterial translocation, and an
urgent operation should be planned. In the
absence of these signs, detorsion is successful
in 60–95% of cases. Following detorsion, a
rectal tube should be left in place to allow for
continued colonic decompression and to prevent re-torsion of the colon. To ensure proper
placement, the rectal tube can be inserted
over a guide wire through the sigmoidoscope,
reaching just past the point of the volvulus. If
detorsion is unsuccessful, an urgent operation
is warranted.
contrast. Note the smooth tapering of contrast as the colon
narrows at the point of obstruction (red arrow). (Reused
with permission © 2015 Springer) (c) “Whirl” sign seen
on CT scan (white circle). (Reused with permission ©
2013 John Wiley and Sons Inc.)
Unlike with sigmoid volvulus, endoscopic
detorsion is not recommended as part of the
management strategy of cecal volvulus.
Various small studies have reported low success rates in achieving cecal reduction with
endoscopy, and have described it as technically challenging. As such, surgery is always
performed to relieve the obstruction, detorse
the bowel, and address the colon which is
prone to volvulize again.
F. Many operative interventions have been
described for the treatment of sigmoid volvulus with varying success rates: simple
detorsion, sigmoidopexy (intra- or extraperitoneal), mesosigmoidoplasty (Fig.47.3),
and colectomy with or without primary
anastomosis. Of all, sigmoid colectomy is
the best option to prevent recurrences. The
decision to perform a primary anastomosis
must take into account the timing of the
operation (urgent vs. semi-elective), intraoperative ndings (bowel viability, contamination), the clinical status of the patient
(hemodynamics, metabolic derangements),
and the patient’s baseline risk (nutritional
status, comorbidities).
All urgent operations should be performed
via midline laparotomy. In general, colectomy with end colostomy (with mucous stula or Hartmann’s closure) should be
performed in the presence of gangrenous
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