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- •Preface
- •Contents
- •Contributors
- •Endovascular Aneurysm Repair
- •Clinical Applications
- •Aortic Procedures Planning
- •Performance Assessment
- •Future Prospects
- •References
- •References
- •Introduction
- •Medical Error
- •Traditional Training
- •Animal Simulation Labs
- •Virtual Reality Simulation
- •3: Radiation Safety
- •Introduction
- •Basic Radiation Physics Units
- •Personnel Dose Limits
- •Pregnant Personnel
- •References
- •4: Tools of the Trade
- •Needles, Catheters, and Wires
- •Vascular Access
- •Double Wall
- •Single Wall
- •Advantages/Disadvantages
- •Nonvascular Needles (Table 4.1)
- •Guidewires
- •Curved
- •Straight/Angled
- •Stiffness
- •Flexibility
- •Coating
- •Torqueability
- •Opacity
- •Catheters
- •Flush Catheters
- •Visceral Catheters
- •Multipurpose Catheters
- •Cerebral Catheters
- •Guiding Catheters
- •Microcatheters
- •Vascular Sheaths
- •Vessel Dilators
- •Accessories
- •Embolic Agents
- •Temporary Agents
- •Permanent Agents
- •Pushable Coils
- •Detachable Coils
- •Coiling Techniques (Fig. 4.48)
- •Vascular Plugs
- •Particulates
- •Liquid Embolics
- •Fogarty Balloons
- •Angioplasty Balloons
- •Drug-Coated Balloons
- •Vascular Stents
- •Balloon Expandable Stents
- •Self-Expandable Stents
- •Specialty Stents
- •References
- •Consults
- •Pre-procedure Evaluation
- •Consent
- •Code Status
- •Laboratory Testing
- •Antibiotic Prophylaxis
- •Anticoagulation
- •Antihypertensives
- •Contrast Allergy Prophylaxis
- •Procedure Plan
- •Post-procedure Management
- •Hospital Admission
- •Discharge
- •Follow-up Visits
- •IR Clinic
- •Conclusion
- •References
- •6: The IR Road Map: Vascular Anatomy Overview
- •Introduction
- •Imaging Modalities
- •Ultrasound
- •Computed Tomography
- •Magnetic Resonance Imaging
- •Cross-Sectional Anatomy
- •Chest
- •Segmental Lung Anatomy
- •Mediastinum
- •Pulmonary Arteries
- •Pulmonary Veins
- •Bronchial Arteries
- •Liver
- •Arterial Access
- •Double-Wall Technique
- •Common Femoral Artery Access
- •Kidneys
- •Ureters
- •Bladder
- •Uterus
- •References
- •Alternative Arterial Access Sites
- •Venous Access
- •Manual Compression
- •Closure Devices
- •Compression Devices
- •Topical Agents
- •Invasive Devices
- •References
- •9: Central Venous Access
- •Pathophysiology
- •Non-tunneled Central Catheters (NTCCs)
- •Tunneled Central Catheters (TCCs)
- •Implantable Ports
- •Peripherally Inserted Central Catheters (PICCs)
- •Clinical Indication
- •Conventional Therapy
- •Non-tunneled Central Catheters
- •Tunneled Central Catheters
- •Ports
- •PICCs
- •Interventional Therapy
- •Ports
- •PICCs
- •Pre-procedural Prep
- •History
- •Physical Exam
- •Imaging
- •Complex Venous Access
- •Post-procedural Management
- •Complications
- •Acute Complications
- •Long-Term Complications
- •Device Removal
- •Tunneled Catheter Removal
- •Port Removal
- •References
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •References
- •11: IVC Filters
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •IVC Filter Placement
- •VTE Prevention
- •Preprocedural Preparation
- •Complication
- •Access Site
- •Device-Related
- •Postprocedural Management
- •IVC Filter Retrieval
- •Advanced IVC Filter Retrieval Techniques
- •Conclusion
- •References
- •Pathophysiology
- •Arteriovenous Fistula
- •Arteriovenous Graft
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •AVG Angioplasty
- •AVF Angioplasty
- •References
- •13: Pelvic Congestion Syndrome
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •References
- •14: Varicocele
- •Pathophysiology
- •Conventional Therapy
- •Interventional Therapy
- •References
- •15: Varicose Veins
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •References
- •16: Vascular Malformations
- •Pathophysiology
- •Hemangiomas
- •Vascular Malformations
- •Arteriovenous Malformations (High Flow)
- •Venous Malformations (Low Flow)
- •Lymphatic Malformations
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •High-Flow AVMs
- •Low-Flow Venous Malformations
- •Klippel-Trenaunay Syndrome
- •Lymphatic Malformations
- •References
- •Pathophysiology
- •Abdominal Aortic Aneurysm (AAA)
- •Thoracic Aortic Aneurysm (TAA)
- •Clinical Indication
- •Abdominal Aortic Aneurysm
- •Thoracic Aortic Aneurysm
- •Conventional Therapy
- •Abdominal Aortic Aneurysm
- •Thoracic Aortic Aneurysm
- •Interventional Therapy
- •Abdominal Aortic Aneurysm
- •Thoracic Aortic Aneurysm
- •Common Complications
- •Access
- •Contrast Nephropathy
- •Spinal Cord Ischemia
- •Postoperative Monitoring
- •References
- •18: Aortic Dissection
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Preprocedure Work-Up
- •Post-procedural Management
- •References
- •19: Endoleak
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Type II Endoleaks
- •Type III Endoleaks
- •Type IV Endoleaks
- •Type V Endoleaks
- •References
- •20: Traumatic Aortic Injury
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Pre-procedural Prep
- •Pre-procedural Imaging
- •Post-procedural Management
- •Post-procedural Imaging
- •References
- •21: Bronchial Artery Embolization
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Outcomes
- •References
- •Pathophysiology
- •Pulmonary Arteriovenous Malformation
- •Pulmonary Artery Pseudoaneurysm
- •Clinical Indication
- •Pulmonary Arteriovenous Malformation
- •Pulmonary Artery Pseudoaneurysm
- •Conventional Therapy
- •Pulmonary Arteriovenous Malformation
- •Pulmonary Artery Pseudoaneurysm
- •Interventional Therapy
- •Pulmonary Arteriovenous Malformation
- •Pulmonary Artery Pseudoaneurysm
- •References
- •23: Lymphatic Interventions
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Pedal Lymphangiography (PL)
- •Intranodal Lymphangiography (IL)
- •Dynamic Contrast Enhanced MR Lymphangiography (DCMRL)
- •Thoracic Duct Embolization
- •Plastic Bronchitis
- •References
- •24: Mesenteric Ischemia
- •Pathophysiology
- •Acute Mesenteric Ischemia
- •Chronic Mesenteric Ischemia
- •Clinical Indication
- •Acute Mesenteric Ischemia
- •Arterial Occlusive Disease
- •Nonocclusive Mesenteric Ischemia (NOMI)
- •Portomesenteric Vein Thrombosis
- •Chronic Mesenteric Ischemia
- •Conventional Therapy
- •Acute Mesenteric Ischemia
- •Arterial Occlusive Disease
- •Nonocclusive Mesenteric Ischemia (NOMI)
- •Portomesenteric Vein Thrombosis
- •Chronic Mesenteric Ischemia
- •Interventional Therapy
- •Acute Mesenteric Ischemia
- •Chronic Mesenteric Ischemia
- •References
- •25: Visceral Aneurysms
- •Pathophysiology
- •Visceral Artery True Aneurysms (VATAs)
- •Visceral Artery Pseudoaneurysm (VAPA)
- •Clinical Indication
- •VATA
- •VAPA
- •Conventional Therapy
- •Interventional Therapy
- •Splenic Artery Aneurysms
- •Renal Artery Aneurysms
- •Hepatic Artery Aneurysms
- •Celiac Artery Aneurysms
- •Complications
- •Splenic Aneurysm
- •Renal Aneurysm
- •Hepatic Aneurysm
- •References
- •26: Renal Artery Stenosis
- •Pathophysiology
- •Clinical Indications
- •Conventional Therapy
- •Interventional Therapy
- •Post-procedural Care
- •Conclusion
- •References
- •27: GI Bleeding
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Complications
- •References
- •28: Uterine Artery Embolization
- •Pathophysiology
- •Fibroids
- •Adenomyosis
- •Postpartum Hemorrhage
- •Clinical Indication
- •Conventional Therapy
- •Fibroids
- •Adenomyosis
- •Postpartum Hemorrhage
- •Interventional Therapy
- •Fibroids
- •Adenomyosis
- •Postpartum Hemorrhage
- •AV Fistula
- •References
- •29: Prostate Artery Embolization
- •Pathophysiology
- •Benign Prostatic Hyperplasia
- •Prostate Cancer/Hematuria
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •References
- •30: Aortoiliac Disease
- •Pathophysiology
- •Blue Toe Syndrome
- •Leriche Syndrome
- •Fibromuscular Dysplasia
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Abdominal Aorta
- •Aortic Bifurcation
- •Common Iliac Artery
- •External Iliac Artery
- •Internal Iliac Artery
- •Blue Toe Syndrome
- •References
- •31: Infrainguinal Disease
- •Pathophysiology
- •Claudication (Rutherford Categories 1–3)
- •Critical Limb Ischemia: Rest Pain (Rutherford Category 4)
- •Critical Limb Ischemia: Skin Lesions (Rutherford Categories 5–6)
- •Acute Limb Ischemia
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Percutaneous Transluminal Angioplasty
- •Stents
- •Acute Limb Ischemia
- •References
- •Pathophysiology
- •Spleen
- •Liver
- •Kidney
- •Clinical Indication
- •Spleen
- •Liver
- •Kidney
- •Conventional Therapy
- •Spleen
- •Liver
- •Kidney
- •Interventional Therapy
- •Spleen
- •Pre-procedure
- •Post-procedure
- •Liver
- •Pre-procedure
- •Post-procedure
- •Kidney
- •Pre-procedure
- •Post-procedure
- •References
- •Pathophysiology
- •Pelvic Fractures
- •Extremity Fractures
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •References
- •34: Transarterial Chemoembolization
- •Pathophysiology
- •Clinical Indications
- •Conventional Therapy
- •Medical Management
- •Surgical Management
- •Interventional Therapy
- •Post-procedure
- •References
- •35: Transarterial Radioembolization (TARE)
- •Introduction
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Primary Liver Cancers
- •Hepatic Metastatic Disease
- •References
- •36: Liver Ablation
- •Pathophysiology
- •Liver Cancer
- •Liver Metastases
- •Liver Cysts
- •Clinical Indication
- •Conventional Therapy
- •Liver Cancer
- •Liver Metastases
- •Liver Cysts
- •Interventional Therapy
- •References
- •Pathophysiology
- •Lung Cancer
- •Renal Cell Carcinoma
- •Bone Lesions
- •Clinical Indication
- •Lung Cancer
- •Kidney Cancer
- •Bone Lesions
- •Conventional Therapy
- •Lung Cancer
- •Kidney Cancer
- •Bone Lesions
- •Interventional Therapy
- •Radiofrequency Ablation (RFA)
- •Microwave Ablation (MWA)
- •Cryoablation
- •Irreversible Electroporation (IRE)
- •Lung Cancer
- •Kidney Cancer
- •Bone Lesions
- •References
- •Pathophysiology
- •Conventional Therapy
- •Ascites
- •Varices
- •Interventional Therapy
- •References
- •Pathophysiology
- •Etiology
- •Clinical Indication
- •Conventional Therapy
- •Medical Management
- •Surgical Management
- •Interventional Therapy
- •Post-procedural Management
- •Complications
- •References
- •40: Biliary Drainage
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Conclusion
- •References
- •41: Biopsy Techniques
- •Introduction
- •Clinical Indication
- •Interventional Therapy
- •Needle Selection
- •Biopsy Techniques
- •References
- •Introduction
- •Pathophysiology
- •Ascites
- •Clinical Indication
- •Ascites
- •Conventional Therapy
- •Ascites
- •Interventional Therapy
- •Ascites
- •References
- •43: Obstructive Uropathy
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Urolithiasis
- •Infection
- •Urothelial Carcinoma
- •Neurogenic Bladder
- •Interventional Therapy
- •References
- •Pathophysiology
- •Clinical Indications
- •Percutaneous Radiologic Gastrojejunostomy (PRGJ) Tube
- •Percutaneous Jejunostomy (PJ) Tube
- •Conventional Therapy
- •Interventional Therapy
- •Percutaneous Radiologic Gastrostomy (PRG)
- •Post-procedural Management
- •Percutaneous Radiologic Gastrojejunostomy (PRGJ)
- •Percutaneous Jejunostomy (PJ)
- •References
- •45: Stroke
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Complications
- •Post-procedure Management
- •References
- •46: Cerebral Angiography: Aneurysms
- •Pathophysiology
- •Clinical Indication
- •Conventional Therapy
- •Interventional Therapy
- •Pre-procedural Preparation
- •Post-procedural Management
- •Complications
- •References

32 Visceral andSolid Organ Trauma
367
Fig. 32.7 A 23-year-old female with a grade V renal laceration as seen
on coronal (a) and axial (b) imaging. Angiography demonstrates renal
artery pseudoaneurysm, active extravasation, and extensive parenchy-
4. Renal artery catheterization is typically performed
with a 5 Fr catheter followed by digital subtraction
angiography, often in AP and ipsilateral projections.
5.
fully crossed with a guidewire and catheter or
microcatheter. Angiography distal to the injury is
performed to ensure location within the true lumen.
occlude life-threatening bleeding and is followed
by placement of a stent or stent graft across the arterial injury.
mal injury (c and d). Digital subtraction angiography following coil
embolization for hemodynamic instability demonstrates complete
occlusion beyond the mid-right renal artery (e)
6.
is then further selected with either the 5 Fr catheter
or a coaxial microcatheter. The microcatheter system is advanced super-selectively to the area of
injury, and embolization is performed with an
embolic agent of choice.
7. Completion renal angiography is performed from
the renal artery through the 5 Fr catheter or sheath
to ensure endpoint has been obtained and to verify
absence of additional angiographic abnormalities
warranting treatment.

368
K. J. Nelson and M. Daun
Despite the high success rate of RAE, complications do
occur. Repeat embolization may be required and has been
associated with higher trauma grade [29]. Nontarget embolization is rare. Post-embolization syndrome is self-limited
and occurs in a small minority of patients; it is characterized
by fever, leukocytosis, and back pain. Potential complications also include arterial hypertension, decreased renal
function, and abscess. Late-term complications following
blunt or penetrating renal trauma include the development of
pseudoaneurysm or arteriovenous stulas, which may occur
even in patients who demonstrate spontaneous control of
bleeding with expectant management only. Presentation with
delayed symptoms such as pain or hematuria may occur up
to months or even years following the traumatic insult [32].
Key Point
Complications of renal artery embolization include
arterial hypertension, decreased renal function,
abscess, pseudoaneurysm, and AV stula.
Post-procedure
Patients are closely monitored as inpatients following blunt
renal trauma. Duration of hospital stay varies by severity of
renal injury and management, with median hospital stays
following grade V injury ranging from 13days for NOM to
22days for operative management [29].
References
1. Miller P, Chang M, Hoth J, Mowery N, Hildreth A, Martin R, etal.
Prospective trial of angiography and embolization for all grade III
to V blunt splenic injuries: nonoperative management success rate
is signicantly improved. JAm Coll Surg. 2014;218(4):644–8.
2. Olthof D, van der Vlies C, Joosse P, van Delden O, Jurkovich G,
Goslings J.Consensus strategies for the nonoperative management
of patients with blunt splenic injury. JTrauma Acute Care Surg.
2013;74(6):1567–74.
3. Cales R, Trunkey D.Preventable trauma deaths. A review of trauma
care systems development. JAMA. 1985;254(8):1059–63.
4. Soto J, Anderson S.Multidetector CT of blunt abdominal trauma.
Radiology. 2012;265(3):678–93.
5. Moore EE, Cogbill TH, Jurkovich GJ, Shackford SR, Malangoni
MA, Champion HR.Organ injury scaling: spleen and liver (1994
revision). JTrauma Acute Care Surg. 1995;38(3):323–4.
6. Hagiwara A, Fukushima H, Murata A, Matsuda H, Shimazaki
S.Blunt splenic injury: usefulness of transcatheter arterial embolization in patients with a transient response to uid resuscitation 1.
Radiology. 2005;235(1):57–64.
7. Beuran M, Gheju I, Venter MD, Marian RC, Smarandache R.Nonoperative management of splenic trauma. JMed Life. 2012;5(1):47.
8. Uranüs S, Pfeifer J.Nonoperative treatment of blunt splenic injury.
World JSurg. 2001;25(11):1405–7.
9. Schroeppel TJ, Croce MA.Diagnosis and management of blunt abdominal solid organ injury. Curr Opin Crit Care. 2007;13(4):399–404.
10. Schnüriger B, Inaba K, Konstantinidis A, Lustenberger T, Chan LS,
Demetriades D.Outcomes of proximal versus distal splenic artery
embolization after trauma: a systematic review and meta-analysis.
JTrauma. 2011;70(1):252–60.
11. Ekeh AP, Khalaf S, Ilyas S, Kauffman S, Walusimbi M, McCarthy
MC. Complications arising from splenic artery embolization: a
review of an 11-year experience. Am JSurg. 2013;205(3):250–4.
12. Frandon J, Rodière M, Arvieux C, Michoud M, Vendrell A, Broux
C, etal. Blunt splenic injury: outcomes of proximal versus distal
and combined splenic artery embolization. Diagn Interv Imaging.
2014;95(9):825–31.
13. McIntyre LK, Schiff M, Jurkovich GJ. Failure of nonoperative
management of splenic injuries: causes and consequences. Arch
Surg. 2005;140(6):563–9.
14. Leeper WR, Leeper TJ, Ouellette D, Moffat B, Sivakumaran T,
Charyk-Stewart T, etal. Delayed hemorrhagic complications in the
nonoperative management of blunt splenic trauma: early screening leads to a decrease in failure rate. JTrauma Acute Care Surg.
2014;76(6):1349–53.
15. Jahromi AH, Migliaro M, Romano M, Sangster G.Delayed splenic
rupture; normal appearing spleen on the initial multidetector computed tomography (MDCT) can sometimes be misleading. Trauma
Mon. 2016;21(5):e24465.
16. Wahl WL, Ahrns KS, Chen S, Hemmila MR, Rowe SA, Arbabi
S. Blunt splenic injury: operation versus angiographic embolization. Surgery. 2004;136(4):891–9.
17. Dake M, Geschwind J, editors. Abrams’ angiography: interventional radiology. 3rd ed. Philadelphia: Lippincott Williams &
Wilkins; 2014.
18. Hiatt JR, Gabbay J, Busuttil RW.Surgical anatomy of the hepatic
arteries in 1000 cases. Ann Surg. 1994;220(1):50.
19. Stassen NA, Bhullar I, Cheng JD, Crandall M, Friese R,
Guillamondegui O, et al. Nonoperative management of blunt
hepatic injury: an Eastern Association for the Surgery of Trauma
practice management guideline. J Trauma Acute Care Surg.
2012;73(5):S288–93.
20. Arvieux C, Letoublon C. Abbreviated laparotomy. J Chirurg.
2000;137(3):133–41.
21. Tinkoff G, Esposito TJ, Reed J, Kilgo P, Fildes J, Pasquale
M, Meredith JW. American Association for the Surgery of
Trauma Organ Injury Scale I: spleen, liver, and kidney, validation based on the National Trauma Data Bank. JAm Coll Surg.
2008;207(5):646–55.
22. Green CS, Bulger EM, Kwan SW.Outcomes and complications of
angioembolization for hepatic trauma: a systematic review of the
literature. JTrauma Acute Care Surg. 2016;80(3):529–37.
23. Melloul E, Denys A, Demartines N. Management of severe blunt
hepatic injury in the era of computed tomography and transarterial embolization: a systematic review and critical appraisal of the
literature. JTrauma Acute Care Surg. 2015;79(3):468–74.
24. Kozar RA, Moore FA, Cothren CC, Moore EE, Sena M,
Bulger EM, et al. Risk factors for hepatic morbidity following nonoperative management: multicenter study. Arch Surg.
2006;141(5):451–9.
25. Venkatesan AM, Kundu S, Sacks D, Wallace MJ, Wojak JC, Rose
SC, et al. Practice guideline for adult antibiotic prophylaxis during vascular and interventional radiology procedures. JVasc Interv
Radiol. 2010;21(11):1611–30.
26. Richardson JD, Franklin GA, Lukan JK, Carrillo EH, Spain DA,
Miller FB, etal. Evolution in the management of hepatic trauma: a
25-year perspective. Ann Surg. 2000;232(3):324–30.
27. Maarouf AM, Ahmed AF, Shalaby E, Badran Y, Salem E, Zaiton
F.Factors predicting the outcome of non-operative management of
high-grade blunt renal trauma. Afr JUrol. 2015;21(1):44–51.
28. Moore EE, Shackford SR, Pachter HL, McAninch JW, Browner
BD, Champion HR, etal. Organ injury scaling: spleen, liver, and
kidney. JTrauma Acute Care Surg. 1989;29(12):1664–6.
29. Lanchon C, Fiard G, Arnoux V, Descotes JL, Rambeaud JJ, Terrier
N, etal. High grade blunt renal trauma: predictors of surgery and

32 Visceral andSolid Organ Trauma
369
long-term outcomes of conservative management. A prospective
single center study. JUrol. 2016;195(1):106–11.
30. Miller KS, McAninch JW. Radiographic assessment of renal
trauma: our 15-year experience. JUrol. 1995;154(2):352–5.
31. Vozianov S, Sabadash M, Shulyak A.Experience of renal artery
embolization in patients with blunt kidney trauma. Cen Eur JUrol.
2015;68(4):471–7.
32. Dinkel HP, Danuser H, Triller J.Blunt renal trauma: minimally
invasive management with microcatheter embolization—experience in nine patients 1. Radiology. 2002;223(3):723–30.
33. Beyer C, Zakaluzny S, Humphries M, Shatz D.Multidisciplinary
management of blunt renal artery injury with endovascular therapy
in the setting of polytrauma: a case report and review of the literature. Ann Vasc Surg. 2017;38:318–e11.

Pelvic andExtremity Trauma
HowardM.Richard
33
Pathophysiology
An anonymous Czech surgeon is quoted as saying “bloody
vascular trauma– it’s either bleeding too much or it’s not
bleeding enough.” “Bleeding too much” will result in hemorrhagic shock, multi-system organ failure, exsanguination,
and death. “Not bleeding enough” can result in ischemia,
limb loss, and stroke. Pelvic and extremity trauma is comprised of blunt and penetrating trauma to the pelvis and
extremities. Blunt trauma is typically the result of a motor
vehicle collision, crush injury, or fall from signicant height.
Penetrating trauma is typically the result of a missile injury
such as a gunshot wound or stab wound. Vascular injury can
result in either bleeding or ischemia.
Initial management of the trauma patient usually follows
the standard ABCs: airway, breathing, and circulation [1, 2].
Once the initial trauma survey is complete, the history and
physical examination will direct the ongoing management
of the patients’ injuries. Upon arrival in the hospital, plain
lms, ultrasound (FAST exam), and CT are utilized to further characterize the extent of injuries and direct further
management.
• Pericardium (subxiphoid)
• Morrison’s pouch (RUQ)
• Left perisplenic space (LUQ)
• Pouch of Douglas (suprapubic)
Pelvic Fractures
Pelvic fractures are categorized into lateral compression,
anterior posterior compression, and vertical shear types of
injuries [3]. Acetabular fractures can be classied as posterior
wall/column, anterior wall/column, or transverse [4].
Of pelvic ring fractures, 58% are not associated with an
acetabular fracture, while 32% of fractures are isolated acetabular fractures [5, 6]. A pelvic fracture in combination with
an acetabular fracture is identied in 10% of pelvic fractures.
Lateral compression fractures are typically seen in motor
vehicle collisions in which the driver or passenger are hit from
the side, aka “T-boned.” Anterior posterior compression fractures can also be described as open-book fractures as there is
typically a large diastasis of the pubic symphysis. Vertical
shear injuries are typically identied after a fall from a height
or a vertical stress applied to one side of the pelvis.
Key Point
Focused Assessment with Sonography for Trauma
(FAST) scan aims to look quickly for free uid,
assumed hemoperitoneum in the trauma patient. Four
regions are scanned:
H. M. Richard (*)
University of Maryland School of Medicine, University of
Maryland Medical Center, Division of Interventional Radiology,
Department of Diagnostic Radiology and Nuclear Medicine,
Baltimore, MD, USA
e-mail: hrichard@umm.edu
© Springer International Publishing AG, part of Springer Nature 2018
N. A. Keefe et al. (eds.), IR Playbook, https://doi.org/10.1007/978-3-319-71300-7_33
Key Point
Pelvic ring fracture=58%.
Acetabular fracture=32%.
Pelvic ring + acetabulum fracture=10%.
Pelvic fracture patterns can predict vascular arterial injury
patterns [7] (Table 33.1). The superior gluteal and internal
pudendal arteries are most commonly injured arteries, while
the deep circumex iliac and inferior epigastric arteries
are more rarely injured. Anterior posterior compression or
open- book fractures are often associated with arterial vascular
371

372
H. M. Richard
injury [8]. Lateral compression and vertical shear fractures
are less likely to be associated with signicant vascular injury.
Patients older than 60 can have calcied arteries that may not
be as effective in vasoconstricting. Therefore, they are prone
to active bleeding at a higher rate and inlocations that are
harder to predict. In fact, it is prudent to study patients who
are older than 60 with pelvic angiography regardless of
presenting hemodynamic stability [9].
Extremity Fractures
Extremity fractures can be classied as open or closed
depending on the presence of skin laceration secondary to
the bone fragment. Open fractures can be classied based on
the severity ranging from a grade 1 to grade 3 injury which
can be associated with signicant arterial injuries based on
the Gustilo classication system; of these, Gustilo class 3C
involves arterial injury (Table33.2) [10].
Risk factors for amputation include Gustilo 3C, sciatic or
tibial nerve injury in the lower extremity, transection of two
of three upper extremity nerves, prolonged ischemia greater
than for 6h, extensive crush or destructive soft tissue injury,
or signicant wound contamination.
Clinical Indication
Initial management of pelvic and extremity trauma outside
of the hospital setting focuses on control of bleeding and preliminary splinting of obvious fractures. Upon transfer to a
trauma center, the patient’s ABCs are evaluated and stabilized. A comprehensive physical examination and directed
imaging ndings will dictate further treatment.
The history of the trauma should focus on mechanism of
injury and include time of injury, presenting Glasgow Coma
Table 33.1 Site of possible vessel injury based on fracture location
[7, 8]
Site of fracture Likely site of vessel injury
Anterior posterior compression
fracture
Pubic rami and open-book fractures Internal or external pudendal
Acetabular fracture Superior gluteal artery
Superior gluteal artery
Internal pudendal artery
artery
Score (GCS) and initial resuscitation efforts including the
amount of uids administered and pressor and transfusion
requirements. Physical exam should document ongoing swelling or hematoma formation as well as a detailed neurological
and distal pulse examination with arterial brachial indices
(blood pressure of the injured limb/uninjured limb) if feasible.
A value of less than 0.9 is abnormal and requires further attention [11]. Serial monitoring of the ABI is usually warranted.
Following stabilization of the airways, breathing, and central
circulation, attention can be directed toward management of
pelvic and extremity trauma [11]. The key tenants are control of
bleeding and restoration of normal circulation. Control of bleeding begins with manual compression, with direct handheld pressure over the arterial injury. An aortic balloon catheter can be
placed at the bedside before surgery to tamponade bleeding.
Volume resuscitation with 3:1:1 packed RBCs, platelets, and
FFP is used for large transfusion requirements. Prior to control
of bleeding, minimal IV uids are administered as raising the
blood pressure is counterproductive. After control of bleeding,
aggressive volume resuscitation is undertaken with warmed uids, blood, and clotting factors [2, 11].
Hard signs of vascular injury include active bleeding,
expanding pulsatile hematoma, bruit or thrill over wound,
absent distal pulses, or distal ischemic changes and mandate
immediate operative exploration and repair. Soft signs of vascular injury include small non-expanding hematoma, peripheral nerve decit, history of severe bleeding at the time of
injury, unexplained hypotension, and signicant bone injury,
i.e., fracture with dislocation or a proximal penetrating
wound. These soft signs warrant evaluation of the arterial
tree. See Table33.3.
Key Point
Six P’s of distal ischemia (same for acute aortic
occlusion and peripheral arterial disease):
• Pain
• Pallor
• Paresthesia
• Poikilothermia
• Pulselessness
• Paralysis
Table 33.2 Gustilo classication system of extremity fractures
Type Wound length Soft tissue damage Fracture pattern Soft tissue coverage Vasculature
1 <1cm Minimal soft tissue damage Simple Adequate soft tissue coverage Intact
2 >1cm Moderate soft tissue damage,
3A >10cm Extensive soft tissue damage,
3B Inadequate soft tissue coverage
3C Arterial damage
contamination, or comminution
contamination, or comminution;
segmental fracture
Moderate comminution Adequate soft tissue coverage Intact
Severe comminution or
segmental fractures
Adequate soft tissue coverage Intact
requiring ap coverage

33 Pelvic andExtremity Trauma
373
Further evaluation including plain lms, bedside ultrasound,
and CT scan can help identify internal injuries (Fig. 33.1a).
Ultrasound and Doppler evaluations including the calculation
of the ABI are very useful bedside evaluations. Multi-slice CT
evaluation through the pelvis should evaluate for hematoma,
fractures, and concurrent injuries to the bowel and bladder
which can guide selective angiography [12]. CT evaluation of
the extremities can better characterize a fracture as well as
neurovascular involvement to aid in orthopedic and angiographic planning. Contrast extravasation indicates an area of
active bleeding, while failure of distal vasculature to enhance
is evidence of distal ischemia. Patients with coagulopathy
should be reversed prior to performing angiography.
Key Point
Contrast extravasation on CT corresponds to an area of
active bleeding.
Conventional Therapy
Once the decision is made to take the patient to the operating
room, the entire limb is sterilely prepared. Prior to investigating
the site of the injury, proximal and distal control of the
extremity arteries is obtained. Separate incisions proximal
Table 33.3 Signs of vascular injury
Hard signs Soft signs
Active bleeding Non-expanding hematoma
Expanding pulsatile
hematoma
Bruit/thrill over wound History of severe bleeding at time of
Absent distal pulses Unexplained hypotension
Distal ischemic changes Signicant bone injury
Peripheral neurological decit
incident
and distal are utilized to isolate the vessel. Vessel slings are
passed around the vessel twice to allow for control of the
blood ow. Identication of the injured vessel can then be
undertaken. The inow (aortoiliac arteries) and outow
(infrainguinal arteries) can be evaluated. In cases where
there is limited inow or outow due to thrombus, a Fogarty
catheter can be passed to remove thrombi adjacent to the
vessel injury. If a small, clean partial circumference laceration is identied, it can be repaired by directly suturing or by
the application of a small vein patch. If the vessel is transected, the cut ends tend to retract. If possible, the arterial
ends are mobilized, and an end-to-end repair is attempted. If
vessel approximation is not possible, a synthetic graft or
reverse vein graft can be employed.
Key Point
Surgical treatment options for vessel injury:
• Fogarty catheter to remove thrombus
• Primary surgical repair
• Small vein patch
• Re-anastomosis of transection
• Synthetic or reverse vein graft
• Ligation or shunting with delayed denitive repair
In some cases, the concept of damage control surgery is
more appropriate [13, 14]. Damage control surgery posits
that a minimal surgery now can temporize the patient in a
more expedient way and allow for faster time to complete
resuscitation. This can then be followed by more denitive
surgical repair later when the patient is stabilized and can
tolerate a longer procedure. When this concept is applied to
extremity vascular trauma, ligation of active bleeding arteries and shunting is performed immediately with formal
repair performed at a later date. Ligation of the common and
external carotid artery, subclavian axillary, and internal iliac
Fig. 33.1 (a) Right pelvic trauma with active extravasation from the
superior gluteal (arrowhead) and inferior gluteal (arrow) artery injuries.
(b) Selective catheterization of the inferior gluteal artery followed by
coil embolization (not shown). (c) Selective catheterization with coil
embolization of the superior gluteal artery. (d) Completion angiogram
following coil embolization of the inferior and superior gluteal artery
injuries shows no evidence of active extravasation

374
H. M. Richard
arteries is considered safe. Ligation of the internal carotid
artery is associated with a 10–20% stroke rate. Ligation of
the external iliac, common femoral, or supercial femoral
arteries can result in critical limb ischemia. For these reasons,
shunting is required prior to ligation of these vessels to
prevent limb loss or stroke. Extra anatomic shunts allow
elective fracture repair and delayed vascular reconstruction.
Another concern of operative management is compartment syndrome. Ischemia followed by local inammatory
response can lead to edema and reperfusion injury during
revascularization which can increase the compartment pressure. When compartment pressures rises to greater than
30mmHg, this can result in venous and capillary compression. This may result in vascular stasis, cellular ischemia, and
ultimately cell death. Whenever the possibility of compartment syndrome is identied, it can be treated with surgical
fasciotomy. If a fasciotomy is not performed, then compartment pressures should be monitored.
Key Point
It is critical to monitor compartment pressures or per-
form fasciotomies in those at risk for compartment
syndrome following revascularization. There are four
compartments of the calf, three of the thigh, and two of
the upper arm and forearm.
Interventional Therapy
Angiography is frequently employed for treatment of bleeding following pelvic or extremity trauma. The development
of catheter-based angiography dates back to the 1950s [15, 16].
Not long after the development of angiography for the
treatment of peripheral arterial disease, Dr. Ring experimented with the rst treatment of pelvic hemorrhage in
1972. Initially, infusions of epinephrine or autologous blood
clot were utilized for active bleeding [17]. Treatment for vascular occlusion came about later when angioplasty balloons
and stents were developed for the treatment of arterial dissections. IR techniques have developed in tandem with the
development of the tools of the trades (refer to Chap. 4 for a
thorough discussion of IR instruments). Numerous different
catheters and wires have been developed to aid the interventional radiologist in accessing various vessels.
Embolization agents vary from temporary to permanent
[18]. An example of a temporary agent is Gelfoam, which
can be administered as a slurry or as pledgets. Permanent
agent includes coils, vascular plugs, or even covered stents.
Gelfoam is most appropriate in embolizing the pelvis where
there may be multiple foci of bleeding. As Gelfoam acts as a
liquid embolic, it can diffuse into multiple areas of bleeding.
Gelfoam is metabolized by the body and allows for recanalization of the embolized vessels in approximately 2weeks.
Permanent agents such as coils or vascular plugs are appropriate for a focal injury or vessel truncation. For dominant
vessels that require distal perfusion, a covered stent can be
used to cover the area of injury. Stent grafts can be placed to
cover a “hole” in a vessel and maintain a patent lumen
(Fig.33.2) [19].
Indication and timing of pelvic angiography is based on
hemodynamic instability and failure to respond to uid
resuscitation. Patients with a very large transfusion requirement or persistent hypotension, who have evidence of ongoing pelvic arterial bleeding, will benet from arteriography
and embolization. Pelvic arterial bleeding may be suspected
following exploratory laparotomy for abdominal organ injuries; if suspected, the pelvis is surgically packed. The retro-
Fig. 33.2 (a) Left femoral artery to femoral vein stula (arrow). (b) Fluency stent graft (Bard Peripheral Vascular) was deployed to repair the
stula

33 Pelvic andExtremity Trauma
375
peritoneum should not be opened surgically as doing so may
release the tamponade and the patient could decompensate. Signs of active bleeding include ongoing hypotension, expanding pelvic hematoma, or a large amount of
bright red blood returned from operative placed pelvic surgical drains.
Indication and timing for extremity angiography is usually based on evidence of active bleeding or loss of distal
perfusion [20, 21]. Hard signs of vascular injury dictate the
need for immediate operative repair, while soft signs, evidence of bleeding following operative repair, or worsening
exam may necessitate an extremity angiogram. Pelvic angiography and embolization has 85–97% success in controlling pelvic bleeding [1, 22, 23]; 5–23% of patients may
require repeat angiography. Angiography and embolization
is 84–97% successful in controlling extremity bleeding
[11, 24].
Following angiographic treatment of an area of injury,
serial hematocrit can be used to conrm resolution of bleeding. If the H&H continues to downtrend, further angiographic
evaluation may be warranted. Frequent evaluation of distal
pulses should be performed to ensure adequate perfusion. The
arterial index should return to greater than 1.0 in patients
without chronic peripheral artery disease. Vigilant examination for compartment syndrome should be performed, particularly in patients with signicant ischemic time. If there is
clinical suspicion for compartment syndrome, then compartment pressure should be measured with potential fasciotomy
creation (Fig.33.3).
Preoperative evaluation of patients prior to pelvic and
extremity angiography will be based on the review of available imaging. The location of the suspected sight of bleeding will weigh heavily on the plan for accessing the vascular
anatomy.
Fig. 33.3 (a) CT of the left leg demonstrates pseudoaneurysm (arrow-
head) secondary to gunshot wound. The bleed is arising from the profunda femoral artery (arrow). Note the extensive femoral fracture that
was repaired. (b) Coronal reformatted images demonstrate profunda
femoral artery (arrow) and pseudoaneurysm (arrowhead). (c)
Angiography demonstrates active bleeding from the profunda femoral
artery. The catheter tip (arrow) is at the distal aspect of the pseudoaneurysm. Note the bullet fragment (arrowhead). (d) Angiography after distal embolization. Note the coil nest (arrow). (e) Angiography after
proximal coil embolization

376
The How To
1. Review imaging and determine areas to be studied
based on expected location of arterial injury.
2. For pelvic and extremity vascular injury, the femoral artery should be accessed via the Seldinger technique. Bilateral femoral access may be needed in
patients with several suspected sites of bleeding.
Radial artery access can be considered on a caseby-case basis.
3.
be performed to identify arterial injuries such as
active extravasation, vessel truncation, or arteriove-
4.
made how to treat the lesion. This may include
embolization using temporary or permanent agents
or stent placement depending on the type of injury.
(a) When there are multiple foci of active bleeding
or vessel injury, this may require a diffuse agent
such as Gelfoam slurry.
(b)
coils may be employed.
(c) As there are multiple collateral pathways in the
pelvis, it is also possible that a collateral artery
is supplying the injured vessel and continuing
to allow the patient to bleed.
(d) Caution is warranted when using Gelfoam in
the extremity to prevent distal embolization.
(e)
important to embolize distal and proximal to
the lesion for control.
5.
adequacy of the treatment.
Angiography and embolization in the pelvis and extremities is safe and effective for pelvic and extremity trauma. CT
angiography is the diagnostic modality of choice in evaluating
patients prior to angiography. Angiography and embolization
can be performed as an adjunct to patients with ongoing bleeding after surgery for pelvic and extremity trauma.
References
1. Coccolini F, Stahel PF, Montori G, Bif W, Horer TM, Catena F,
et al. Pelvic trauma: WSES classication and guidelines. World
JEmerg Surg. 2017;12:5.
2. Cullinane DC, Schiller HJ, Zielinski MD, Bilaniuk JW, Collier
BR, Como J, etal. Eastern Association for the Surgery of Trauma
H. M. Richard
practice management guidelines for hemorrhage in pelvic fracture–
update and systematic review. JTrauma. 2011;71(6):1850–68.
3. Alton TB, Gee AO. Classications in brief: young and burgess classication of pelvic ring injuries. Clin Orthop Relat Res.
2014;472(8):2338–42.
4. Alton TB, Gee AO. Classications in brief: letournel classication
for acetabular fractures. Clin Orthop Relat Res. 2014;472(1):35–8.
5. Halawi MJ.Pelvic ring injuries: emergency assessment and management. JClin Orthop Trauma. 2015;6(4):252–8.
6. Hussami M, Grabherr S, Meuli RA, Schmidt S. Severe pelvic
injury: vascular lesions detected by ante- and post-mortem contrast
medium-enhanced CT and associations with pelvic fractures. Int
JLegal Med. 2017;131(3):731–8.
7. Karadimas EJ, Nicolson T, Kakagia DD, Matthews SJ, Richards
PJ, Giannoudis PV.Angiographic embolisation of pelvic ring injuries. Treatment algorithm and review of the literature. Int Orthop.
2011;35(9):1381–90.
8. Ben-Menachem Y, Coldwell DM, Young JW, Burgess AR.
Hemorrhage associated with pelvic fractures: causes, diagnosis,
and emergent management. AJR Am J Roentgenol. 1991;157(5):
1005–14.
9. Kimbrell BJ, Velmahos GC, Chan LS, Demetriades D.Angiographic
embolization for pelvic fractures in older patients. Arch Surg.
2004;139(7):728–32; discussion 32–3.
10. Kim PH, Leopold SS. In brief: Gustilo-Anderson classication.
[Corrected]. Clin Orthop Relat Res. 2012;470(11):3270–4.
11. Fox N, Rajani RR, Bokhari F, Chiu WC, Kerwin A, Seamon MJ,
etal. Evaluation and management of penetrating lower extremity
arterial trauma: an Eastern Association for the Surgery of Trauma
practice management guideline. J Trauma Acute Care Surg.
2012;73(5 Suppl 4):S315–20.
12. Juern JS, Milia D, Codner P, Beckman M, Somberg L, Webb T, etal.
Clinical signicance of computed tomography contrast extravasation in blunt trauma patients with a pelvic fracture. JTrauma Acute
Care Surg. 2017;82(1):138–40.
13. Roberts DJ, Bobrovitz N, Zygun DA, Ball CG, Kirkpatrick AW,
Faris PD, etal. Indications for use of thoracic, abdominal, pelvic,
and vascular damage control interventions in trauma patients: a
content analysis and expert appropriateness rating study. JTrauma
Acute Care Surg. 2015;79(4):568–79.
14. Walker ML.The damage control laparotomy. J Natl Med Assoc.
1995;87(2):119–22.
15. Kerr WS Jr, Margolies MN, Ring EJ, Waltman AC, Baum
SN. Arteriography in pelvic fractures with massive hemorrhage.
Trans Am Assoc Genitourin Surg. 1972;64:14–7.
16. Margolies MN, Ring EJ, Waltman AC, Kerr WS Jr, Baum
S. Arteriography in the management of hemorrhage from pelvic
fractures. N Engl JMed. 1972;287(7):317–21.
17. Ayella RJ, RW DP Jr, Khaneja SC, Maekawa K, Soderstrom CA,
Rodriguez A, etal. Transcatheter embolization of autologous clot in
the management of bleeding associated with fractures of the pelvis.
Surg Gynecol Obstet. 1978;147(6):849–52.
18. Scemama U, Dabadie A, Varoquaux A, Soussan J, Gaudon C, Louis
G, et al. Pelvic trauma and vascular emergencies. Diagn Interv
Imaging. 2015;96(7–8):717–29.
19. Kufner S, Cassese S, Groha P, Byrne RA, Schunkert H, Kastrati
A, et al. Covered stents for endovascular repair of iatrogenic
injuries of iliac and femoral arteries. Cardiovasc Revasc Med.
2015;16(3):156–62.
20. Abrassart S, Stern R, Peter R. Unstable pelvic ring injury with
hemodynamic instability: what seems the best procedure choice
and sequence in the initial management? Orthop Traumatol Surg
Res. 2013;99(2):175–82.
21. Marzi I, Lustenberger T.Management of bleeding pelvic fractures.
Scand JSurg. 2014;103(2):104–11.

33 Pelvic andExtremity Trauma
377
22. Ierardi AM, Duka E, Lucchina N, Floridi C, De Martino A, Donat
D, et al. The role of interventional radiology in abdominopelvic
trauma. Br JRadiol. 2016;89(1061):20150866.
23. Shi J, Gomes A, Lee E, Kee S, Moriarty J, Cryer H, et al.
Complications after transcatheter arterial embolization for pelvic
trauma: relationship to level and laterality of embolization. Eur
JOrthop Surg Traumatol. 2016;26(8):877–83.
24. DuBose JJ, Savage SA, Fabian TC, Menaker J, Scalea T,
Holcomb JB, etal. The American Association for the Surgery of
Trauma PROspective Observational Vascular Injury Treatment
(PROOVIT) registry: multicenter data on modern vascular injury
diagnosis, management, and outcomes. JTrauma Acute Care Surg.
2015;78(2):215–22; discussion 22–3.
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