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9.2 Auricular Hematoma, “Cauliower Ear”
87
tection, and immune mediators, cartilage is prone to infection, erosive chondritis, and subsequent necrosis. It is therefore of great importance, when repairing injuries to the auricle, to adequately cover exposed cartilage (Belleza 2006).
When repairing lacerations to the ear, one aims to appropriately and expediently cover car­tilage avoiding wound hematoma (Fig. 9.3) or abscess formation, but also to restore cosmesis. A potential hematoma or abscess will prevent adherence of the nutrient-rich perichondrium to the auricular cartilage and might result in its destruction, loss of the skeletal support of the ear, and eventually abnormal cartilage production, which leads to the development of a brotic, cal­cied mass referred to as the “cauliower ear” (Jones and Mahendran 2004; Ingvaldsen and Tønseth 2017) or “wrestler’s ear,” because this type of injury is very common among wrestlers since early Olympic competition times (Fig.9.4)
(Gifn 1992). Auricular hematoma mοst com­monly affects the helical rim or pinna, but some­times can affect the conchal bowl (Macdonald etal. 2005). There is no clearly dened best treat- ment for acute auricular hematoma (Jones and Mahendran 2004). In the early stages after injury, simple aspiration is an option (Fig.9.3). However, reaccumulation occurs, complicating matters, unless adequate pressure can be maintained with packing (Gernon 1980). Incision over the most dependent part of the swelling, raising of skin aps, drainage of the collection, and scraping of the undersurface of the aps followed by inser­tion of a small drain proved to be effective methods to treat primary as well as recurrent auricular hematomas (Ghate etal. 2022).
Fig. 9.3 An auricular hematoma of the left ear being aspirated Fig. 9.4 Example of a “cauliower” or “wrestler’s” ear
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Conventional packing materials occasionally fail. Splinting the pinna by two molds held in place by light compression bandaging offers a possible solution in such cases (Macdonald etal.
2005). A more denitive result can be obtained
by incision and drainage. A suction drain is left in situ or a “button” is sutured over the area in order to force apposition of the skin, perichondrium, and cartilage (Bull and Lancer 1984; Tallat etal.
1985). If left untreated, an auricular hematoma
can result in complications such as perichondri­tis, infection, and necrosis. If treatment is delayed, a plate of broneocartilage can form resulting in ear deformity. Surgical excision of the broneocartilage, done up to 1month follow­ing injury, will restore the pinna form. If there is delay in the excision, a mature cauliower ear will have formed, and reconstructive plastic sur­gery is necessary to restore the pinna form (Yotsuyanagi etal. 2002). The zone and degree of deformity dene the type of surgical interven­tion. Cartilage deformity without change in the outline of the ear can be treated by shaving the deformed cartilage through a suitable skin inci­sion. For deformities accompanied by a skin de­cit, a postauricular skin ap should be used (Yotsuyanagi et al. 2002). Deformities having caused changes in the outline of the ear require conchal or costal cartilage grafts, depending on the structural integrity of the ear, in order to be restored (Yotsuyanagi etal. 2002).
9.3 Lacerations
Prompt treatment of ear lacerations (Fig. 9.5) will help avoid functional and aesthetic complications.
In children, general anesthesia is often required for more severe lacerations, whereas in adults, almost every wound can be treated under local anesthesia, which can be obtained by a great auricular (a branch of the cervical plexus) and an auriculotemporal (a branch of V3) nerve block. Initial wound care of ear lacerations consists of irrigation. Special attention is paid to full­thickness wounds so that cartilage is not further dissected from its adherent soft tissues (Fig.9.6).
9 Injuries oftheEars
Fig. 9.5 Patient with lacerations to the right ear
A cotton plug should be inserted into the external acoustic meatus to prevent the entrance of uid during irrigation and blood during repair, which causes discomfort to the patient. Meticulous and minimal debridement of nonviable tissue is the next step. Jagged or devitalized cartilage and skin should be trimmed. Up to 5mm of cartilage can be removed without risk of evident deformity or asymmetry of the ear (Brown et al. 2007). Extensive skin debridement may result in the need to overstretch the skin and may disrupt the natural contour of the underlying cartilage and thus the ear. The rst step in repairing the wound is the reapproximation of the cartilage. Either clear Prolene 4.0 or an absorbable 4.0 suture material is usually used. The initial sutures should be placed at critical grooves and elds in the car­tilage. The stitch through the cartilage should include the anterior as well as the posterior peri­chondrium to ensure maximal stability of the suture. Tearing through the delicate cartilage is thus avoided and nutrient supply to the damaged cartilage is provided. Subsequently, the posterior
9.4 Avulsions
Fig. 9.6 Patient with full-thickness laceration of the left ear (From: The Atlas of Emergency Medicine, 4th Edition, Knoop KJ, Stack LB, Storrow AB, Thurman RJ (Eds), McGraw-Hill Education, China, 2016, with permission)
skin, followed by the anterior skin, is repaired. Interrupted Prolene 5.0 or preferably 6.0 is used for this purpose. If signicant cartilage and/or soft-tissue loss precludes an adequate and cos­metically satisfying closure, immediate recon­struction is considered, as will be analyzed in the following paragraph. In children, the majority of the wounds can be repaired using steristrips. In a study of 111 children from Shefeld Children’s Hospital, 65% of lacerations were treated with steristrips (Steele and Brennan 2002). Finishing the repair, the cotton plug is removed from the external meatus and the meatus is thoroughly cleaned. Finally, a pressure dressing must be applied to the ear to prevent postoperative bleed­ing, hematoma formation, and resulting defor­mity. Petrolatum jelly-coated gauze should be placed in the ssures and folds of the ear until the gauze is level with the helix. The same gauze is also placed behind the ear. This padding prevents pain and pressure necrosis caused by the ear
89
pressing against the skull. Another gauze covers the anterior ear surface. A circumferential head bandage secures in place the entire assembly. The dressing is rst changed 2–3 days postopera­tively. A lighter dressing can then be applied.
Lacerations to the lobule do not involve carti­lage and thus are more easily repaired by simple suturing or a Z-plasty. Other methods of earlobe repair have been reviewed by Vujevich et al. (2007).
9.4 Avulsions
Avulsions can be classied into partial or com­plete. A partial avulsion or extended laceration (Steffen et al. 2006) can be then classied depending on the width of the pedicle (wide or narrow pedicle) (Lavasani etal. 2010).
Complete avulsions or otherwise traumatic ear amputations, of a part (Fig.9.7) or of the total auricular tissue, are rare. Only 74 cases had been reported in the literature till 2009 (Ihrai et al.
2009). Three-quarters of all patients were
between 11 and 40years of age; the median age was 28.0 years. There were 56 men and 18 women (male-to-female 3:1). The most frequent causes were trafc accidents (25/74 cases) and ghts (21/74 cases). An astonishing one-third of all injuries resulted from bites, mostly human (17 cases) or dogs (9 cases).
Treatment of these injuries is complex. A good cosmetic result is the surgeon’s objective; how­ever, demolishing of the auricular area should be avoided so that future ear reconstruction in case of replantation failure is possible. The main tech­niques used for ear replantation are microsurgical replantation, pocket techniques, and reattach­ment techniques. Microsurgical replantation should be the method of choice, whenever this is possible, because a superior outcome can be achieved without jeopardizing a subsequent ear reconstruction with rib cartilage in case of a failure.
The rst successful microsurgical ear replan­tation was reported in 1980 (Pennington et al.
1980), and only 25 cases were reported till 2002
(Kind 2002) and 47 until 2013 (Jung etal. 2013).
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ab c
9 Injuries oftheEars
Fig. 9.7 (a and b) Patient with complete avulsion (ampu- tation) of the lower part of the right auricle and earlobe. Unfortunately, the avulsed part was lost. The patient opted
It is a lengthy operation; it requires technical expertise and a lengthy hospital stay, results in multiple blood transfusions, and has quite a high failure rate. The main problem is that no suitable veins can be found; therefore, medicinal leech therapy is necessary for venous drainage result­ing in considerable blood loss. Occasionally, this leads to a successful outcome (Otto etal. 1999; Hussey and Kelly 2010), whereas other times partial or total failure of development of venous channels leads to ear necrosis when leeching is stopped (Akyürek etal. 2001; Lin et al. 2010). Lin et al. (2010) reported on their experience with six patients who underwent microvascular ear replantation. In three of them, only arterial anastomosis was feasible. These patients required intra-replant heparin injection (chemical leech) to resolve venous congestion. They all sustained partial loss of the replanted ear, and secondary procedures were necessary to repair the reconstructions.
Steffen etal. (2006) reviewed the trauma cases of the auricle between 1980 and 2004. Thirty­seven case reports described microsurgical repairs, mostly used in total amputations. Two­thirds (67%) showed a complete recovery. In 14 microsurgical ear replantations, no venous anas-
for primary closure and would consider secondary recon­struction. (c) The patient’s left normal ear
tomosis was performed. In total amputations, the rate of total recovery without venous anastomosis was slightly below (ve of eight cases) the rate in cases with venous anastomosis (13 of 18 cases), whereas in partial amputations, the reverse was seen (three of six cases without venous repair versus one of three cases with) (Steffen et al.
2006).
As already mentioned before, venous conges-
tion is a very regular occurrence in microsurgical ear replantation. Therefore, most surgeons con­sider it a characteristic accompanying symptom rather than a complication (Steffen etal. 2006). Anemia demanding blood transfusion has been reported by many authors. Other typical compli­cations for the microsurgical replantation were arterial spasm, thrombosis of arteries, inamma­tions, hematoma requiring relief, or meatal steno­sis (Steffen etal. 2006).
Most microsurgically replanted cases were
amputations of the auricle. However, a few cases of replantation of the earlobe have also been reported. Jung etal. (2009) reported the rst suc­cessful replantation of an avulsed earlobe by sin­gle arterial anastomosis.
When microsurgical replantation is not possi-
ble, one can choose between ear reattachment
9.5 Reconstruction
91
and a pocket technique according to two clinical features: (1) the size of the amputated part and (2) the involvement of the earlobe (Ihrai et al.
2009). Ear reattachment is feasible when the
amputated part is smaller than 15 mm or when amputation involves the earlobe. Pocket tech­niques, which are appropriate for the replantation of the auricular cartilage, can be used when the amputated part is bigger than 15 mm and does not involve the earlobe (Ihrai etal. 2009).
The reported pocket methods generally dealt with partial amputations. All pocket reattach­ments but one were reported to have survived, and in 67%, complete success was achieved. Local aps were used in all degrees of trauma. The classic composite graft method was mainly used in cases of extended lacerations, with a high rate of complete intake (Steffen etal. 2006). In composite graft techniques, several authors noted the use of heparin or leeches and stitch incisions. Some surgeons emphasized the importance of cooling the repaired auricle (Fuleihan et al.
1987).
Despite the fact that the pocket method proved a success in partial avulsions, it should be remem­bered that it is a two-stage technique and, in case of failure, leaves an anatomically altered area. Especially in complete amputations, indications for the pocket method should be very carefully considered. It rarely achieves as good and consis­tent an aesthetic result as a secondary reconstruc­tion with rib or conchal cartilage, and it almost always results in a shrunken cartilage and scar contracture (Steffen etal. 2006).
If a successful revascularization of the ampu­tated auricle seems to be unlikely or if blood transfusions in the peri- and postoperative course are denied by the patient, then he/she is best served with a primary wound closure and the prospect of a subsequent reconstruction with rib cartilage (Steffen etal. 2006; Ihrai etal. 2009).
9.5 Reconstruction
Although primary repair is feasible in most cases of ear trauma, there are instances when delayed and staged reconstruction is necessary to restore
functional anatomy and aesthetic appearance. It should be noted that the auricle plays a central role in facial aesthetics and that auricular defor­mities can have a signicant impact on psychoso­cial functioning and self-esteem (Wong et al.
2021).
Understanding the anatomy and recognizing
the circumstances regarding the mechanism of injury are crucial for successful reconstruction, though unique and individual structural charac­teristics should be taken into account (Wong etal. 2021; Nojoumi and Woo 2021).
A satisfactory primary reconstruction of a
severe auricular trauma is not always possible to obtain due to either local conditions or failure of the primary attempt. The resulting defect, which can produce major psychological distress, ought to be reconstructed secondarily.
The localization of the defect, its extent, and
the condition of the surrounding tissues are essential criteria for treatment planning. Advances in autologous reconstruction using costal cartilage have resulted in the production of high-quality auricles, if the procedures are per­formed in specialist centers with exposure to a high volume of cases.
For partial ear defects, the two-stage tech-
nique with autologous cartilage previously described by Brent (1974) and adopted by Nagata (1993) can be used. In the rst stage, the costal cartilage of the oating (eight) rib can be utilized for helical rim defects. For larger defects, a block of cartilage from the synchondrosis of the sixth and seventh rib can be harvested. The eighth rib can serve as a provider of the helical rim if the block is not of sufcient volume. The rib is carved into the shape of the missing segment of the disgured ear, using the uninjured side as a template. After having been sculpted, the carti­lage framework is held together by ne stainless steel wire. The cartilage construct is attached to the remaining native ear cartilage and covered with, usually, a posteriorly based advancement skin ap. Suction drains are placed under the construct to allow the overlying skin ap. Occasionally, a bipedicled skin ap (for middle helical rim defects) or a postauricular skin ap (for small upper pole defects) is used (Pearl and
92
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9 Injuries oftheEars
Sabbagh 2011). The bipedicled skin ap can be used if there is concern about the blood supply; however, surgical access is more difcult by this approach (Pearl and Sabbagh 2011).
In cases of insufcient or poor-quality skin, preoperative tissue expansion or a temporopari­etal fascial ap plus split skin graft is used to cover the cartilage framework. The latter did not give a good denition as native or tissue­expanded skin (Pearl and Sabbagh 2011).
In a second stage, 3–6 months later, the ear construct is released from the side of the head and the posterior sulcus is created with a split skin graft. If there is no reasonable projection, Pearl and Sabbagh (2011) have suggested the use of a small piece of cartilage behind the construct covered with either a posterior fascial ap or a temporoparietal fascial ap covered with a split skin graft. This provides adequate projection.
Reconstruction of total traumatic defects is based on the same principles. In such cases, a very large temporoparietal fascial ap is needed to cover the cartilage construct (Yamada and Ueda 2012). Other authors have used a free radial forearm ap to cover the cartilage and consider it an excellent choice (Lauwers and van der Hulst 2009).
Simple defects conned to the earlobe can be reconstructed with conchal cartilage graft, an anterior skin ap, and a V-Y advancement ap on the posterior auricular skin (Pearl and Sabbagh
2011). A variety of other, one- or two-stage, pro-
cedures have also been used. The technique described by Gavello is one of the earliest and simplest. The bilobed skin ap is designed and raised, by subcutaneous dissection, at the caudal end of the ear stump based on the occipital branch of the posterior auricular artery. Caution not to injure the latter during ap dissection is essential. The procedure has several advantages and yields excellent cosmetic results with preservation of the earlobe shape and volume. An intact postau­ricular mastoid region is a sine qua non for the use of the procedure (Chattopadhyay etal. 2012).
Complications after autologous post- traumatic ear reconstruction are low. Partial skin necrosis (4%) and infection (2%) are the commonest. Conservative management with minimal loss of denition is feasible in cases of small areas of skin necrosis. Infection can result in signicant loss of
denition of the construct necessitating further car­tilage grafting procedures with additional coastal cartilage to correct (Pearl and Sabbagh 2011).
Autologous reconstruction is not always wish­ful or feasible. In such cases, a prosthesis secured to the remaining ear stump with adhesive (silastic medical grade adhesive) is a valuable technique. Silicone elastomers are frequently used for the fabrication of such auricular prostheses, and the achieved results are satisfactory (Adeyemo etal.
2008). For virtually complete amputation, osseo-
integrated implants provide a more rigid alterna­tive to adhesive prosthetics. Autologous reconstruction presents a technical challenge to the surgeon; however, it is the prosthetic recon­struction which requires lifelong attention and may be associated with late complications (Thorne etal. 2001).
References
Adeyemo WL, Jokomba LA, Somefun OA, et al.
Experience with prosthetic reconstruction of ear
defects at LUTH, Lagos, Nigeria. Niger Postgrad Med
J. 2008;15:238–42. Akyürek M, Safak T, Keҫik A.Microsurgical ear replanta-
tion without venous repair: failure of development of
venous channels despite patency of arterial anastomo-
sis for 14 days. Ann Plast Surg. 2001;46:439–42. Belleza WG.Otolaryngologic emergencies in the outpa-
tient setting. Med Clin North Am. 2006;90:329–33. Brent B. Ear reconstruction with an expansible frame-
work of autogenous rib cartilage. Plast Reconstr Surg.
1974;53:619–28. Brown DJ, Jaffe JE, Henson JK, et al. Advanced lac-
eration management. Emerg Med Clin North Am.
2007;25:83–92. Bull PD, Lancer JM. Surgeon’s workshop: treatment
of auricular haematomas by suction drainage. Clin
Otorarlyngol Allied Sci. 1984;9:355–60. Chattopadhyay D, Gupta S, Murmu MB, etal. Revisiting
Gavello’s procedure for single-stage reconstruction of
the earlobe: the vascular basis, technique and clinical
uses. Can J Plast Surg. 2012;20:e22–4. Fuleihan NS, Natout MAY, Webster RC, etal. Successful
replantation of amputated nose and auricle.
Otolaryngol Head Neck Surg. 1987;97:18–23. Gernon WH.The care and management of acute hematoma
of the external ear. Laryngoscope. 1980;90:881–5. Ghate SK, Kalambe A, Maldhure S.Auricular haematoma
an avoidable cosmetic deformity: a chance or negli-
gence. Am J Otolaryngol. 2022;43:103232. Gifn CS.Wrestler’s ear: pathophysiology and treatment.
Ann Plast Surg. 1992;28:131–9.
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Hussey AJ, Kelly JI.Microsurgical replantation of an ear
with no venous repair. Scand J Plast Reconstr Surg Hand Surg. 2010;44:64–5.
Ihrai T, Balaquer T, Monteil MC, etal. Surgical manage-
ment of traumatic ear amputations: literature review. Ann Chir Plast Esthet. 2009;54:146–51.
Ingvaldsen CA, Tønseth KA. Auricular haematoma.
Tidsskr Nor Laegeforen. 2017;137:105–7.
Jones SEM, Mahendran S. Interventions for acute
auricular hematoma. Cochrane Database Syst Rev. 2004;2004(2):CD004166.
Jung SN, Yoon S, Kwon H, etal. Successful replantation
of an amputated earlobe by microvascular anastomo­sis. J Craniofac Surg. 2009;20:822–4.
Jung SW, Lee J, Oh SJ, etal. A review of microvascular ear
replantation. J Reconstr Microsurg. 2013;29:181–8.
Kind GM. Microvascular ear replantation. Clin Plast
Surg. 2002;29:233–48.
Lauwers T, van der Hulst RR.Microvascular ear recon-
struction using a free radial forearm ap after dog bite. J Plast Reconstr Aesthet Surg. 2009;62:535–8.
Lavasani L, Leventhal D, Constantinides M, et al.
Management of acute soft tissue injury to the auricle. Facial Plast Surg. 2010;26:445–50.
Lin PY, Chiang YC, Hsieh SL, etal. Microsurgical replan-
tation and salvage procedures in traumatic ear amputa­tion. J Trauma. 2010;69:E15–9.
Macdonald DJM, Calder N, Perrett G, etal. A novel way
of treating acute cauliower ear in a professional rugby player. Br J Sports Med. 2005;39:e29.
Nagata S. A new method of total reconstruction of
the auricle for microtia. Plast Reconstr Surg. 1993;92:187–201.
Nojoumi A, Woo BM.Management of ear trauma. Oral
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ear replantation without venous anastomosis by
using leeches. Handchir Microchir Plast Chir.
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Injuries oftheNeck
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10
10.1 Anatomy
The neck lies between the head and the trunk pro­viding passage for the many structures communi­cating between the above two body parts. It extends from the inferior mandibular margin and superior nuchal line superiorly to the suprasternal notch, superior margin of the clavicle, and level of the seventh cervical vertebra inferiorly.
The neck may be divided into three general regions, the anterior, the lateral, and the posterior. The anterior comprises the structures between the sternocleidomastoid muscles. It is divided by the hyoid bone into supra- and infrahyoid areas. The suprahyoid area is further subdivided into the submental and submaxillary space (Fig.10.1). The infrahyoid has several subdivisions: super­cial infrahyoid, laryngotracheal, thyroid, cervical esophageal, and prevertebral. The lateral region is differentiated into the sternocleidomastoid (carotid) and the supraclavicular areas. The pos­terior part of the neck is the area lying beneath the trapezius muscle as far caudally as the rst rib.
The fasciae of the neck are the supercial and the deep. The former, carrying the supercial ves­sels and nerves, is separated from the latter by the thin platysma muscle (Fig.10.2). The deep fascia invests and supports the muscles, the pharynx, the trachea, the esophagus, the lymph nodes, the
large vessels, and the nerves (Anson and McVay
1971). The suprahyoid region is divided into the
median submental and the two lateral submaxil­lary divisions. The submental area contains the submental lymph nodes. The submaxillary area contains the submaxillary (submandibular) sali­vary gland, the facial artery and vein, the lingual artery, the lymph nodes, the hypoglossal nerve, and the lingual nerve.
The supercial infrahyoid region contains the anterior jugular vein and the infrahyoid muscles. Spreading aside of the musculoaponeurotic lay­ers of the infrahyoid region reveals the laryngo­tracheal tube, the thyroid (Fig. 10.3), and the esophagus in their common fascial casing, the “visceral sheath” (Anson and McVay 1971). These structures are designated “the visceral mass.” This is attached to the vertebral column by sagittal septa (Anson and McVay 1971). Important nerves and vessels of the region are the superior and inferior (recurrent) laryngeal nerve and the superior and inferior thyroid artery and vein.
The lateral regions of the neck are the sternomas­toid (carotid) and the supraclavicular. The former comprises the larger area of the lateral region of the neck. The sternocleidomastoid muscle covers a wide area of it (Fig.10.1). Upon its surface, the supercial vessels (external jugular vein) and nerves (greater auricular nerve) run in duplications of the super-
© The Author(s), under exclusive license to Springer Nature Switzerland AG 2023 C. A. Ioannidis, Soft Tissue Injuries of the Head and Neck,
https://doi.org/10.1007/978-3-031-14915-3_10
95
96
)
muscle fibers
a
Fig. 10.1 Schematic representation of the neck regions (and triangles). The sternocleidomastoid muscle (SCM) is clearly recognizable
10 Injuries oftheNeck
Fig. 10.2 Schematic representation of the platysma muscle
SCM
Post cervical
triangle
Accessory
nerve
Omohyoid
(Inf. belly)
Submand triangle
Carotid triangle
Omohyoid (Sup. belly
Platysma
Deep fasci of the neck
In
Thyroid gland
10.1 Anatomy
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97
cial cervical fascia. The common, internal, and external carotid artery with all its branches, the internal jugular vein, and the vagus nerve run deep to the muscle enclosed in the carotid sheath, a tubu­lar investment of deep cervical fascia (Fig.10.2). The phrenic, spinal accessory, hypoglossal nerve, and cervical plexus are important nerve structures which run at the lateral region of the neck.
The supraclavicular fossa, or posterior cervi­cal triangle, is a depressible space above the mid­dle third of the clavicle, between the posterior margin of the sternocleidomastoid muscle and the anterior edge of the trapezius. Although well protected in the medial portion of its course by the overlying sternocleidomastoid muscle and the inner end of the clavicle, the subclavian artery and vein are liable to injury by penetrating wounds in the lower neck region (Fig.10.3). The trunks of the brachial plexus, because of their proximity to the vessels, may be injured at the same time (Fig. 10.3). Further cranially lie the two branches of the subclavian artery in the
supraclavicular fossa, the transverse scapular and the transverse cervical artery.
The right lymphatic duct drains the lymph from the right half of the supradiaphragmatic area of the body. The thoracic duct collects the lymph from the entire subdiaphragmatic portion of the body as well as from the left half of the supradiaphragmatic area. The individual nerves which form the brachial plexus are the anterior roots of the fth, sixth, sev­enth, and eighth cervical and rst thoracic spinal nerves. These roots emerge through the narrow interval between the anterior and middle scalene muscles and appear in the lower part of the posterior triangle of the neck. On the middle scalene muscle, the anterior roots unite to form trunks (upper, mid­dle, lower trunk). Trauma in the supraclavicular region may contuse, compress, or lacerate different portions of the plexus. The subclavian artery and vein located distally and close to the clavicle usually escape. Injuries in the region of the scaleni involve the roots of the plexus. Trauma within the connes of the supraclavicular fossa involves the trunks.
Fig. 10.3 Schematic representation of the great vessels of the neck, the cervical and brachial plexuses, and the thyroid gland
t. carotid artery
Cervical plexus
Int. jugular vein
Brachial plexus
Ext. carotid artery
Trachea