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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_874_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Contents
- •1.1 Introduction
- •1.2 Ancient Past
- •1.3 Modern Period
- •1.4 Robot
- •1.5 Contemporary Period
- •1.6 Healthcare Robotics
- •1.9 Robotic-Assisted Surgery Logistics
- •1.10 Future Directions
- •1.7 Twenty-First Century
- •1.8 Hernia Repair
- •References
- •2.1 Introduction
- •2.2 Advantages
- •2.3 Disadvantages/Barriers
- •2.4 Training Requirements
- •2.6 Conclusion
- •References
- •3: Enhanced Recovery After Hernia Repair
- •3.1 Introduction
- •3.2 Pre-Operative Measurements
- •3.2.1 Smoking Cessation
- •3.2.2 Weight Loss
- •3.2.3 Diabetes Optimization
- •3.2.4 Nutritional Optimization
- •3.2.5 Prehabilitation
- •3.3 Intra-operative Measures
- •3.3.2 Perioperative Antibiotics
- •3.3.3 Surgical-Site Infections (SSI)
- •3.3.4 Improving Postoperative Intestinal Function
- •3.4 Post-operative Measures
- •3.4.2 Multimodal Pain Control
- •3.4.3 Early Enteral Feeding
- •3.5 Discussion
- •References
- •4.1 Introduction
- •4.3 Prosthetic Materials: History
- •4.4 Absorbable Synthetic Biomaterials
- •4.5 Biologic Products
- •4.5.1 Bovine Products
- •4.5.2 Cadaveric Products
- •4.5.3 Porcine Products
- •4.6 Hybrid Products
- •4.7 Flat Prosthetic Products
- •4.8 Miscellaneous Flat Products
- •4.9 Combination Flat Synthetic Prosthetics
- •4.14 Hiatal Hernia Repair Products
- •4.15 Fixation Devices
- •4.16 Conclusion
- •References
- •5.1 Inguinal Hernia
- •5.1.2 Inguinal Preoperative Imaging
- •5.1.3 Operative Approach
- •5.1.4 Laparoscopic Inguinal Hernia Repairs
- •5.1.5 Bilateral Hernias
- •5.1.6 Obesity
- •5.1.7 Anticoagulated Patients
- •5.1.8 Medical Comorbidities
- •5.1.9 Women
- •5.1.10 Femoral Hernias
- •5.1.11 Preperitoneal Mesh/Lower Midline Surgery
- •5.1.12 Scrotal/Nonreducible Hernia
- •5.1.13 Summary
- •5.1.14 Ventral/Incisional Hernia
- •5.1.16 Preoperative Imaging
- •5.1.17 Prehabilitation
- •5.1.18 Operative Approach
- •5.1.19 Mesh Utilization
- •5.2 Conclusion
- •References
- •6.1 Background
- •6.2 Pain Classification
- •6.3 Anatomic Considerations
- •6.7 Chronic Pain After Ventral Hernia Repair
- •6.8 Chronic Pain After Inguinal Hernia Repair
- •6.10 Open Extended Triple Neurectomy
- •6.11 Laparoscopic Retroperitoneal Triple Neurectomy
- •6.12 Chronic Orchialgia
- •6.14 Conclusion
- •References
- •7.1 Introduction
- •7.3 The Robotic Equipment
- •7.4.1 Patient Positioning
- •7.4.2 Cannulas
- •7.4.3 Robot Docking
- •7.5 Conclusion
- •References
- •8.6 Controversies
- •8.6.1 Direct Hernia Defect Closure
- •8.6.2 Mesh Fixation
- •8.6.3 Non-Mesh Robotic TAPP Repairs
- •8.7 Conclusion
- •References
- •8: Routine Robotic Inguinal Hernia Repair
- •8.1 Introduction
- •8.2 Patient Selection
- •8.3 Surgical Technique
- •8.3.2 Dissection
- •8.3.3 Mesh Placement
- •8.3.4 Peritoneal Closure
- •8.4 Recovery
- •8.5 Adverse Events
- •8.5.1 Small Bowel Obstruction
- •8.5.2 Recurrence
- •8.5.3 Chronic Pain
- •9.1 Introduction
- •9.2 History
- •9.3 Pre-operative Preparation
- •9.4 Operative Techniques
- •9.6 Summary
- •References
- •10: Pelvic Hernias
- •10.1 Introduction
- •10.2 Technique
- •10.5 Docking
- •10.6 Surgical Technique
- •10.7 Dissection/Adhesiolysis
- •10.8 Defect Closure
- •10.10 Complications
- •10.12 Summary
- •10.13 Concluding Remarks
- •References
- •Glossary
- •11.1 Introduction
- •11.4 Other
- •11.5 Conclusion
- •References
- •12: Re-operation After Robotic Inguinal Hernia Repair
- •12.1 Introduction
- •12.6.1 Open Repair
- •12.6.2 Laparoscopic Repair
- •12.6.3 Robotic Repair
- •12.7 Special Considerations
- •12.8 Conclusions
- •References
- •13: Botulinum Toxin Aided Hernia Repair
- •13.1 Introduction
- •13.3 Existing Clinical Applications
- •13.5.1 Anatomy
- •13.5.2 Our Technique
- •13.6.4 Other Uses
- •13.7 Conclusion
- •References
- •14: Pneumoperitoneum Aided Hernia Repair
- •14.1 Introduction
- •14.1.1 Preoperation Treatment Options
- •14.2 Progressive Preoperative Pneumoperitoneum (PPP)
- •14.2.4 PPP Protocol
- •14.3 Surgical Repair: Minimally Invasive
- •14.5 Conclusion
- •References
- •15.1 Introduction
- •15.2 Patient Selection
- •15.5 Port Placement
- •15.6 Intraoperative Considerations
- •15.7 Conclusion
- •References
- •16.2 Operative Technique
- •16.2.2 Access
- •16.2.3 Port Placement
- •16.2.5 Upper Midline Defects (Lower Dock Setup)
- •16.2.6 Lower Midline Defects (Upper Dock Setup)
- •16.2.7 Side Dock Setup
- •16.2.8 Conclusion
- •17: Robotic IPOM-Plus Repair
- •17.1 Introduction
- •17.2 Definition
- •17.3 Surgical Technique
- •17.3.1 Preoperative Care
- •17.3.2 Patient Positioning
- •17.3.3 Trocar Placement
- •17.3.4 Docking
- •17.3.5 Instrumentation
- •17.3.6 Adhesiolysis
- •17.4 Postoperative Care
- •17.5 Conclusions
- •References
- •18: Transabdominal Preperitoneal (rTAPP) Repair
- •18.1 Introduction
- •18.2 Surgical Anatomy
- •18.4 Patient Selection
- •18.5 Preoperative Evaluation
- •18.6 Equipment
- •18.7 Surgical Technique
- •18.7.2 Trocar Placement, Adhesiolysis, Preperitoneal Dissection
- •18.8 Postoperative Care
- •18.9 Complications
- •18.9.1 Bleeding-Hematoma
- •18.9.2 Seroma
- •18.9.3 Intestinal Injury
- •18.9.4 Chronic Pain
- •18.9.5 Recurrence
- •18.10 Limitations
- •18.11 Conclusion
- •References
- •19.1 Introduction
- •19.2 Background
- •19.3 History
- •19.4 Pre-Operative Workup
- •19.6 Surgical Technique
- •19.6.1 Access
- •19.6.2 Port Placement
- •19.6.3 Dissection/Adhesiolysis
- •19.6.5 Midline Reconstruction
- •19.7 Complications
- •19.9 Discussion
- •19.10 Concluding Remarks
- •References
- •Glossary
- •20: Endoscopic Component Separation Techniques
- •20.1 Endoscopic Component Separation Techniques
- •20.4 Operative Steps
- •20.4.1 Preoperative Preparation
- •20.5 Operative Technique
- •20.5.1 Transfascial Approach
- •20.5.2 Modified Subfascial Approach
- •20.5.3 Endoscopic Subcutaneous CS Approach
- •20.8 Conclusions
- •References
- •21: Robotic Retro-Rectus Repairs
- •21.1 Introduction
- •21.2 Robotic Rives: Retromuscular Repairs
- •21.2.1 Patient Selection
- •21.2.2 General Measures
- •21.2.3 Single Docking: Cranial Approach
- •21.2.4 Double Docking: Lateral Approach
- •21.2.5 Single Docking: Lateral Approach
- •21.3 e-TEP
- •21.3.3 Upper Midline Defect
- •21.3.4 Lower Midline Defects
- •21.3.5 Side-Docking
- •21.4 Conclusion
- •References
- •22: Robotic Transversus Abdominus Release
- •22.1 Introduction
- •22.2 Historical Context
- •22.2.3 The Rives-Stoppa Repair
- •22.2.4 Posterior Component Separation
- •22.2.6 Minimally Invasive Approaches
- •22.2.7 Operative Considerations
- •22.2.8 Patient Selection
- •22.3 Pre-Operative Planning
- •22.4 Technique
- •22.4.3 Trocar Placement
- •22.4.4 Docking
- •22.4.5 Retromuscular Dissection
- •22.4.6 Transversus Abdominis Release
- •22.4.8 Contralateral Dissection
- •22.4.9 Fascial Closure
- •22.4.11 Post-Operative Care
- •22.5 Conclusions
- •References
- •23.1 Introduction
- •23.2 Operating Room Set Up
- •23.3 Surgical Technique
- •23.4 Postoperative Care
- •23.5 Conclusion
- •References
- •24: Lumbar Hernia
- •24.1 Introduction
- •24.1.1 Historical Background
- •24.1.2 Classifications
- •24.1.3 Surgical Anatomy
- •24.1.4 Pathogenesis
- •24.1.5 Clinical Presentation
- •24.2 Preoperative Planning
- •24.3 Operative Technique
- •24.3.1 Open Approach
- •24.3.2 Mimimally Invasive Approach
- •24.3.2.1 Conventional Laparoscopy
- •24.3.2.2 Robotic Assisted
- •24.3.3 Hybrid Approach
- •24.4 Conclusion
- •References
- •25.1 Background
- •25.3 Preoperative Considerations
- •25.4 Operating Room Set Up
- •25.5.2 Transversus Abdominis Release (TAR)
- •25.5.4 Mesh Placement
- •25.6 Postoperative Management of Modified Sugarbaker with TAR
- •25.7 Complications
- •25.8 Traditional Sugarbaker Repair
- •25.8.1 Operating Room Set Up
- •25.9 Postoperative Management
- •25.10 Conclusion
- •References
- •References
- •27.2 Obesity
- •27.3 Malnutrition
- •27.4 Immunosuppression
- •27.5 Age
- •27.6 Special Considerations: Cytoreductive Surgery
- •27.7 Future Thoughts
- •References
- •28.1 Morgagni Hernia
- •28.1.1 Si
- •28.1.2 Xi
- •28.2 Bochdalek Hernia
- •28.2.1 Si
- •28.2.2 Xi
- •28.3 Traumatic Diaphragmatic Hernia
- •28.4 Summary
- •References
- •29: Robotic Assisted Morgagni Hernia Repair
- •29.1 Introduction
- •29.2 Preoperative Evaluation
- •29.3 Patient Selection
- •29.6 Intraoperative Considerations
- •29.7 Recommended Instruments
- •29.8 Postoperative Care
- •29.9 Conclusion
- •References
- •30: Robotic Paraesophageal Hernia Repair
- •30.1 Introduction
- •30.2 Preoperative Evaluation
- •30.2.1 Upper Endoscopy
- •30.2.2 Barium Swallow
- •30.2.3 High Resolution Esophageal Manometry
- •30.2.4 pH Monitoring
- •30.3 Operative Technique
- •30.3.1 Operating Room (OR) Setup
- •30.3.2 Patient Positioning
- •30.3.3 Trocar Placement
- •30.3.4 Docking
- •30.3.5 Visualization
- •30.3.7 Esophageal Lengthening
- •30.3.8 Crural Closure
- •30.3.9 Relaxing Incisions
- •30.3.10 Fundoplication
- •30.3.11 Mesh Reinforcement
- •30.4 Peri-Operative Complications
- •30.4.1 Pneumothorax
- •30.4.2 Vagal Injury
- •30.4.3 Esophageal Perforation
- •30.4.4 Gastric Perforation
- •30.4.5 Bleeding
- •30.4.6 Dysphagia
- •30.4.7 Reflux
- •30.5 Outcomes
- •30.6 Reoperative Considerations
- •30.9 Conclusion
- •References
- •31.1 Introduction
- •31.2 Surgical Indications
- •31.3 Preoperative Evaluation
- •31.4 Surgical Technique
- •31.5 Postoperative Care
- •31.6 Outcomes
- •31.7 Conclusion
- •References
- •32.4 Organ Perforation
- •32.6 Postoperative In-hospital Complications
- •32.7 Late Complications
- •32.8 Conclusion
- •References
- •33: Reoperation After Robotic Diaphragmatic Hernia Repair
- •33.1 Introduction
- •33.6 Open Repair
- •33.7 Laparoscopic Repair
- •33.8 Robotic Repair
- •33.9 Conclusions
- •References
- •Index

146
A. Ramaswamy
repair [69]. Similarly, a single institution retrospective review also noted a decrease
in hospital stay following robotic TAR compared with open TAR [70].
Robotic technology has been anticipated to increase the adoption of MIS
techniques in hernia- both for inguinal hernia, with the improved visualization
and ergonomics, and for ventral hernia, with the ability to performed complex
abdominal wall reconstruction previously performed with open methods. The
limited published data has noted that, in the time period between 2008 and
2015, growth in robotic surgery paralleled a reduction in laparoscopic surgery
for 5 general surgical procedures, including inguinal and ventral hernia repair.
The conclusion from this data was that laparoscopic procedures were being converted to robotic procedures rather than conversion of open procedures to
robotic procedures [71]. Another consideration is that the robotic methodology
was approved for hernia repair in mid-2014, making this a technology that is
still in clinical development.
5.2 Conclusion
It is difcult to create an algorithm for ventral hernia repair as expertise and opinion
vary signicantly, even among experts. In a study of ve surgeons presented with
case scenarios, there was consensus on decision to operate for 56% of the scenarios,
consensus on operation type in 40%, and on mesh position in 40% [72]. Even situations where an open strategy seems like the most obvious choice (signicant excess
skin, bowel compromise, enterocutaneous stula, previous skin graft, excision of a
large piece of mesh), there still exist options for a minimally invasive approach, or
with a hybrid approach. There are several options for each patient, (see Fig.5.3)
which highlights the importance of shared decision making. Future research will
determine the algorithm for each surgeon and the patient.
Ventral Hernia
Asymptomatic
Watchful
waiting,
elective repair
Primary hernia-defect
<1 cm
Open suture repair
Low risk
patient
Surgical intervention
Primary or incisional-
defect 1-5 cm
Mesh repair
• open, lap or robotic
• preperitoneal, retrorectus, IPOM/
IPOM with defect closure
Symptomatic
Defect 5-10 cm
High risk patient (especially if associated
with large defect size, multiply recurrent
hernia, EC fistula…)
smoking cessation, weight loss to BMI<40,
Same options as for
1-5 cm defects, but
consider possible
need for component
release
Fig. 5.3 Algorithm for the approach to ventral/incisional hernias
Prehabilitation (nutritional support,
improved blood sugar control)
Defect >10 cm
Mesh repair
• open, lap or robotic
• retrorectus
• will require component release
(surgical or chemical)
Hostile abdomen
Onlay repair

5 Algorithm ofOpen/Laparoscopic/Robotic Repair
147
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A. Ramaswamy

Etiology andManagement ofHerniaRelated Chronic Pain: Implications
ofRobotics
IanT.MacQueen andDavidC.Chen
6.1 Background
Many techniques using open, laparoscopic, robotic, and hybrid procedures exist
in the treatment of hernia with a broad variety of disease requiring many different
options to address hernia type, location, patient factors, available tools, and technical expertise. The adoption of the routine use of mesh in ventral and inguinal hernia
repair has improved recurrence rates. This is especially true for inguinal hernias,
since tension-free, mesh-based repair has become the standard of care. High level
evidence in ventral hernias supports the use of prosthetic reinforcement with signicant reduction in recurrence rates [1, 2]. Postherniorrhaphy chronic pain, however,
remains a prevalent complication representing a substantial burden of morbidity for
patients after both ventral and inguinal hernia repair. Exact rates of postherniorrhaphy chronic pain depend on the denition used, but generally range widely in the literature from 0% to upwards of 60% [3, 4]. The Swedish Hernia Registry reports that
severe or debilitating postherniorrhaphy chronic pain after inguinal hernia repair
occurs at a rate of between 5% and 7% [5].
Development of postherniorrhaphy chronic pain may develop regardless of the
repair technique [6–8], and as such must remain a consideration for any surgeon performing robotic-assisted ventral or inguinal hernia repair. A thorough understanding of the causes of pain, groin and abdominal wall neuroanatomy, and technical
aspects of the initial operation are necessary to successfully manage this complication [8–10]. These factors determine the medical and operative interventions available to address chronic pain after hernia repair. In addition to the implications of
robotic repairs on the development of chronic pain and hernia repair, the technical
6
I. T. MacQueen (*) · D. C. Chen
Lichtenstein Amid Hernia Clinic, David Geffen School of Medicine at UCLA,
Los Angeles, CA, USA
e-mail: dcchen@mednet.ucla.edu
© Springer Nature Switzerland AG 2019
K. A. LeBlanc (ed.), Robotic Assisted Hernia Repair,
https://doi.org/10.1007/978-3-030-23025-8_6
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I. T. MacQueen and D. C. Chen
advantages of robotic assisted surgery including superior optics, range of motion,
precision, and dexterity also play a role in the surgical treatment of this challenging
problem. Effective management is crucial given the personal and societal consequences of postherniorrhaphy chronic pain on quality of life, disability, and healthcare utilization.
6.2 Pain Classification
Postherniorrhaphy pain may be divided into two broad categories: nociceptive pain
and neuropathic pain. Nociceptive pain is the result of direct injury to tissue and
the subsequent local inammatory processes. Endogenous nociceptive molecules
mediate the production of this pain by acting on nociceptors. Neuropathic pain, in
contrast, results from nerve injury. After hernia repair, neuropathic pain symptoms
may include pain with radiation (especially inguinodynia radiating to the scrotum or
femoral triangle, in the case of inguinal hernia repair), hyperalgesia, hyperesthesia,
hypoesthesia, paresthesia, allodynia, or hyperpathia. A positive Tinel sign may be
present. Nerves may be injured by indirect or direct structural damage during the
operation, or by entrapment by suture, contact with mesh (Fig.6.1), folded mesh
(Fig.6.2), meshoma, or xating devices. In the postherniorrhaphy patient, nociceptive and neuropathic pain exist on a spectrum with signicant overlap between the
categories. Somatic and visceral etiologies of pain including soft tissue and bony
injury and inammation as well as adhesions and obstruction add to the complexity of presentation, As is common in establishing the cause of any chronic pain, the
diagnosis of postherniorrhaphy chronic pain is confounded by individual, social,
genetic, and psychological factors.
Fig. 6.1 Neuropathic pain is
frequently related to nerve
entrapment by suture, xation
devices, or mesh, as seen in
this explanted mesh with
associated injured nerve
resected en bloc

6 Etiology andManagement ofHernia-Related Chronic Pain: Implications ofRobotics
Fig. 6.2 Folded mesh (shown here), mesh migration, and meshoma may contribute to development of chronic pain.
153
6.3 Anatomic Considerations
Understanding the neuroanatomy of the abdominal wall and groin is crucial to
preventing chronic pain, and to understanding the likely cause in the patient suffering from this complication. The anterior abdominal wall is innervated by the
nerve roots from T7 at the subxiphoid position through L1 at the groin. These
nerve branches travel from spinal cord to anterior midline, traveling initially along
the inferior costal surface (for T7–T12), then traversing between muscle layers of
the abdominal wall and giving off perforating supercial branches along the course
of the nerve. Postherniorrhaphy neuropathic chronic pain in the abdominal wall
above the groin is frequently caused by nerve entrapment or injury by suture, xation devices, or by the mesh itself. While the nerves can be injured anywhere along
their variable and redundant course, the proximal retroperitoneal trunks are most
susceptible in ank hernia repairs while the coalescence of the perforating nerves
at the semilunar line are at risk in ventral repairs. While the precision of robotic
suturing may decrease the traditional risks associated with transfascial sutures and
penetrating xation with tacks or staples during intraperitoneal onlay mesh (IPOM)
repair, robotic- assisted repair of ventral and ank hernias may risk nerve entrapment through other mechanisms. Robotic-assisted surgery facilitates visualization
and placement of sutures with primary closure of ventral and ank defects. The use
of suture through the abdominal wall especially in off-midline and ank hernias

154
I. T. MacQueen and D. C. Chen
may predispose to pain related to nerve entrapment, though this is not currently
well studied. During robotic-assisted ventral hernia repair, risk of nerve injury or
entrapment may be minimized through careful suture placement, avoiding excessively deep suture bites, and sparing use of xation devices, especially in the lateral
areas of the abdominal wall and ank where the nerves course between the deeper
muscle layers.
The neuroanatomy relevant to inguinal hernia repair extends from the retroperitoneal lumbar plexus to the terminal branches exiting through the inguinal canal.
The anatomy of these nerves and nerve plexuses is complex and highly variable
[11, 12]. However, meticulous identication of the relevant nerves and preventing
their injury or direct contact with mesh has been demonstrated to reduce rates of
post- operative chronic pain to less than 1% after open repair [7]. The three nerves
most commonly contributing to chronic postherniorrhaphy inguinal pain (CPIP)
are the ilioinguinal nerve, the genital branch of the genitofemoral nerve, and the
iliohypogastric nerve.
The ilioinguinal nerve (IIN) originates from the L1 nerve root traversing anterior
to the quadratus muscle before exiting through the transversus abdominis muscle
above the level of the iliac crest. It then travels between the transversus abdominis and internal oblique muscles until it enters into the inguinal canal medial to
the anterior superior iliac spine. The IIN then courses over the anterior surface of
the spermatic cord, covered by the investing fascia of the internal oblique muscle.
Care should be taken not to disrupt this protective fascia during open inguinal hernia repair. The iliohypogastric nerve (IHN) also originates from the L1 nerve root
cephalad to the ilioinguinal nerve traversing anterior to the quadratus muscle before
exiting through the transversus abdominis muscle above the level of the iliac crest.
It then travels between the transversus abdominis and internal oblique muscles until
it enters into the inguinal canal typically cephalad and medial to the ilioinguinal
nerve. The IHN then travels between the internal and external oblique muscle layers
in the medial inguinal canal exiting at the conjoined tendon. The investing fascia of
the internal oblique protects the nerve from contacting an anteriorly placed mesh.
Neither ilioinguinal nor iliohypogastric nerves are commonly visualized during
robotic-assisted or laparoscopic inguinal hernia repair. The distal retroperitoneal
course of the IIN and IHN above the iliac crest may be occasionally visualized
with wide lateral dissection during an extended view total extraperitoneal (eTEP)
inguinal approach or with lateral ank repairs. An understanding of their course is
critical though, and care should be taken to avoid passing suture or xation material
through the anticipated course of these nerves, risking injury or entrapment of the
underlying nerve.
The genitofemoral nerve typically originates from the L1 nerve root continuing its retroperitoneal course over the psoas muscle. In the preperitoneal space,
the genital branch continues inferiorly and anteriorly to enter the deep inguinal
ring, traversing the inguinal canal within the spermatic cord. While identication
in the inguinal canal is difcult due to its diminutive size and location within the

6 Etiology andManagement ofHernia-Related Chronic Pain: Implications ofRobotics
155
cremasteric sheath, the genital and femoral branches may be encountered with dissection lateral to the spermatic vessels. In the case of robotic-assisted or laparoscopic pre- peritoneal repair, one must additionally consider that the main trunk or
femoral branch of the genitofemoral nerve, the anterior or lateral femoral cutaneous
nerve, or even the femoral nerve may contribute to the development of chronic pain.
During a pre-peritoneal repair, these structures typically lie within a predictable
area in the surgical eld, termed the “triangle of pain.” The boundaries of this triangle are the iliopubic tract, the gonadal vessels, and the reected border of peritoneum. After reection of the peritoneum away from this area of the abdominal wall,
dissection in this region should be minimized to avoid disrupting the transversalis
fascia and connective tissue layer protecting these nerves from the overlying mesh.
Suture and xation material should be avoided in this area, as placement carries
risk of nerve injury and resulting chronic pain. Avoidance of xation or use of glue
or self-gripping mesh in preperitoneal inguinal hernia repair is recommended to
minimize the risk of developing chronic post-operative inguinal pain. Specic to
robotic inguinal hernia repair is the recommendation to avoid direct suture closure
of the direct and indirect hernia defect. This technique was abandoned from laparoscopic hernia surgery as it increased the risk of pain without benet with regards
to recurrence. While robotic suturing simplies this task, it risks entrapment of the
iliohypogastric nerve along the inguinal oor with direct defects. Damage to the
spermatic cord, genital nerve, and ilioinguinal nerve from entrapment has also been
seen from suturing. Alternative strategies such as imbrication of the transversalis
fascia or suturing this to Cooper’s ligament may obliterate the dead space without
risking nerve entrapment.
6.4 Conservative Management ofNeuropathic Pain
Most pain that develops after hernia repair is self-limited, though it may take
upwards of a year to resolve. For this reason, pain is managed expectantly during
this time period. Pharmacologic therapies including NSAIDs and agents for neuropathic pain, such as gabapentin and pregabalin, may be benecial. Chronic use
of narcotics should be avoided. Behavioral therapies including physical therapy,
stretching, icing, and avoidance of exacerbating activities and positions should be
encouraged. If pain persists for greater than 3months, it is reasonable to consider
procedural intervention. For pain that seems localized to one or more individual
nerve distributions, nerve blocks using local anesthetic or steroids are a typical rst
step to establishing that pain is neuropathic in nature and related to injury or irritation of a particular nerve or nerves. If transient relief is gained from nerve block but
symptoms remain severe, nerve ablation may be pursued for a more durable effect,
or the patient may proceed to operative intervention. It is preferred that any nerve
blocks and/or ablations should be performed by specialists with experience in management of neuropathic pain.
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