When discussing headaches attributed to preeclampsia and eclampsia, it refers to one of the neurological symptoms that can result from both increased blood pres­sure and endothelial dysfunction [4]. This specic type of headache can vary signi­cantly in presentation, duration, and characteristics [4]. Therefore, the presence of headaches during pregnancy should be a warning sign, especially when accompa­nied by other clinical and laboratory signs and symptoms [4].

Preeclampsia is a progressive multisystem disorder characterized by the develop­ment of hypertension and proteinuria or organ failure after 20weeks of gestation, during pregnancy, labor, or postpartum [6]. It is believed that a combination of maternal and fetal/placental factors is the cause [6]. Abnormalities in placental vas­culature can lead to relative hypoxia, ischemia, or placental hyperperfusion, result­ing in the release of antiangiogenic factors into the maternal circulation, altering the mother’s systemic endothelial function and causing hypertension and other mani­festations of the disease (hematological, neurological, cardiac, pulmonary, renal, and hepatic) [6].

• Aura accompanied by numbness in hands and feet, and blurred vision, lasting up

The patient has a history of migraine with and without aura, with signicant wors­ening during the second half of the rst pregnancy and the rst half of the second pregnancy. She experienced attacks with focal neurological phenomena, including transient hemiplegia and aphasia. Preventive treatment was not maintained consis­tently after the rst delivery, leading to an increased frequency of attacks.

In the second pregnancy, late-onset preeclampsia was observed, requiring inter­vention. The patient requires neurological monitoring and tailored interventions. She remains under observation for the frequency of migraine attacks.

When patients present with dyspnea, auscultation and percussion of the lungs should be performed to examine pulmonary disturbances [8]. An assessment of edema should also be completed, specically evaluating areas of dependent edema (gravity-related), such as the lower extremities, or independent edema, such as in the face or hands [8].

Signs or symptoms of target organ damage may include one or more of the fol­lowing [10]:

Eclampsia is dened as new-onset tonic–clonic, focal, or multifocal seizures with­out other known causes (e.g., epilepsy, cerebral ischemia or arterial infarction, intra­cranial hemorrhage, or drug use) [8].

Researchers investigated the relationship between gestational hypertension, pre­eclampsia, and eclampsia with neurological disorders following childbirth [6]. Utilizing data from the National Patient Registry, the study included 648,385 women with no prior history of chronic hypertension or neurological disorders [6].

The ndings revealed an increased risk of migraine, headache, epilepsy, sleep dis­orders, and mental fatigue among women with hypertensive complications during pregnancy [6]. Compared to normotensive pregnancies, the risk of developing such conditions was 27% higher for gestational hypertension, 32% for preeclampsia, and 70% for eclampsia [6]. The most pronounced association was between eclampsia and epilepsy, with a vefold increased risk. Women with preeclampsia and preterm delivery showed a higher risk of migraine and headache than those with term deliv­ery [6]. The researchers emphasize the need for postpartum follow-up for these women to identify neurological symptoms early [6]. Given the impact on quality of life, medical attention is crucial [6]. Future studies are necessary to understand the mechanisms involved and develop preventive strategies [6]. These ndings justify additional investigations and underscore the importance of continuous monitoring of these women for post-gestational hypertension neurological complications [6].

The treatment of preeclampsia begins with early diagnosis and intervention, focus­ing on adequate blood pressure control and seizure prevention [8]. The denitive treatment for preeclampsia is delivery [8]. However, the risk of premature delivery is weighed against gestational age, fetal growth restriction, fetal distress, and the severity of preeclampsia. Immediate delivery after maternal stabilization (e.g., sei­zure control, initiation of blood pressure management) is indicated for [8]:

• Initial Support Measures: Call for assistance, manage seizures, prevent injuries,

• Magnesium Sulfate: Preferred over other anticonvulsant treatments for prevent-

larly every 4 hours, can be used as an alternate regimen [14]. In patients with

contraindications to magnesium sulfate (e.g., myasthenia gravis, hypocalcemia,

renal insufciency, cardiac ischemia, atrioventricular block, or myocarditis),