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166 Theory and Practice of Squint and Orthoptics
functional development. General debility, psychological instability, over work and worry may be the precipitating factors.
2. Refractive errors. Convergence insufficiency may be associated with uncorrected high hypermetropia and myopia. Diseases of accommodative convergence mechanism result in convergence insufficiency in such patients as follows:
High hypermetropes (more than 5D) usually
make no effort to accommodate and thus there is deficient accommodative convergence as well.
Myopes may not need accommodation and
thus lack accommodative convergence.
Patients who have worn too full a plus
spherical correction may also exert less accommodation and thus less accommodative convergence.
3. Presbyopia. With the advent of presbyopia,
near point of eye recedes and so there is less use of convergence. Neglect of presbyopia may lead to fixation of this anomaly.
On the other hand, patients may also develop convergence insufficiency with the first time use of presbyopic correction. This has been explained by the fact that the relief of sustained accommodative effort afforded by the use of presbyopic correction causes a decrease of accommodative convergence.
4. Muscular imbalances. Extraocular muscular imbalances in the form of exophoria, intermittent exotropia and vertical muscle imbalances, if neglected for a long time may be associated with convergence insufficiency.
5. Consecutive convergence insufficiency may occur following either recession of medial recti or resection of lateral recti muscles.
Clinical features
Convergence insufficiency becomes a clinical problem in children with increased school work, prolonged periods of reading, desk workers and percision workers. It is usually not a problem in farm and manual labour workers.
Symptoms of convergence insufficiency are similar to that of heterophoria and in general the term asthenopia is used to denote the symptom complex. Unsuitability of the glasses
is the most frequent complaint of patients using glasses and having asthenopic symptoms. Such patients change their refractionist and glasses frequently without any satisfaction. Asthenopic symptoms may be grouped as given below.
1. Symptoms of muscular fatigue
These result due to continuous use of the neuromuscular power and are usually marked with near work. These include:
Eye strain and a sensation of tension in and
around the globes is a common complaint of such patients.
Headache and eye ache after prolonged use of
eyes especially for near work, which are relieved, when the eyes are closed for a while. Some patients may show even migrainous tendencies.
Difficulty in changing the focus from distant to
near objects.
Itching, burning and soreness of eyes and even
hyperaemia of the nasal half of the conjunctiva may occur after prolonged close work.
2. Symptoms due to failure to maintain binocular vision
Blurred near vision and crowding of words
while reading.
Intermittent crossed diplopia for near vision
under conditions of fatigue is not uncommon.
Characteristically, one eye will be closed or
covered while reading to obtain relief from visual fatigue.
Diagnosis
Diagnosis of convergence insufficiency is confirmed by:
1. Remote near point of convergence (NPC).
Convergence insufficiency is said to exist, if NPC is more than 10 cm from the baseline.
2. Decreased fusional convergence for near.
When measured on synoptophore, the conver­gence insufficiency is said to exist, if there is difficulty in attaining 30° of convergence.
3. Prism convergence is low but prism diver- gence is normal.
4. Exophoria at near with orthophoria at distance may occur. However, convergence insufficiency may be associated with ortho­phoria and even exophoria.
167Anomalies of Convergence, Divergence and Accommodation
5. Near point of accommodation (NPA) is normal and corresponds to the age of the patient. However, measurement of NPA is essential in each case to diagnose and manage patients suffering from a combined insufficiency of convergence and accommodation. Further, rarely accommodative spasm may occur, if voluntary accommodation and convergence are stimulated in an effort to overcome the convergence insufficiency.
Differential diagnosis
Convergence insufficiency needs to be differen­tiated from the following conditions presenting with almost similar symptoms.
1. Convergence insufficiency versus convergence paralysis
In convergence paralysis, there is total lack of
ability to overcome any amount of base-out prism; while in convergence insufficiency several dioptres of convergence amplitude can be demonstrated.
On receiving a convergence impulse, a patient
with convergence paralysis will show pupillary constriction but inability to converge; in a patient with convergence insufficiency, pupillary constriction will occur while converging on an approaching target, followed by dilation of the pupil, when convergence can no longer be maintained.
2. Convergence insufficiency versus accommoda­tive effort syndrome
Usually an exophoria at near is associated
with convergence insufficiency, while patients with accommodative effort syndrome have esophoria.
Convergence insufficiency is helped by the
–3D test, which compensates for the lack of good fusional convergence; while a patient with accommodative effort syndrome breaks into a tropia during this test.
Plus lenses will worsen the convergence
insufficiency due to relaxation of accommo­dative convergence, while they will improve the symptoms in the accommodative effort syndrome for the same reason.
Treatment
Convergence insufficiency has an excellent prognosis in the majority of cases. Children are treated, when fusional vergences are poor and the patient is showing signs of becoming exotropic. Adults with this condition receive treatment only in the presence of symptoms. Treatment of convergence insufficiency includes: optical treatment, orthoptic treatment, prismo­therapy and surgery.
1. Optical treatment
Proper refraction should be carried out and the correct glasses should be prescribed for any associated refractive error. Myopes are given full correction and hypermetropes undercorrection to stimulate their accommodation which will simultaneously stimulate convergence. In adults above the age of 40 years, proper presbyopic correction should also be done.
2. Orthoptic treatment
Aim of orthoptic exercises is to improve the binocular convergence and to increase the amplitude of fusional convergence. ‘Vision therapy’. The term used for orthoptic treatment includes: A. Conventional or non-computerised exercises for:
In-office vision therapy, and
Home vision therapy
B. Computerised orthoptic exercises for:
In-office vision therapy, and
Home vision therapy
Note. Conventional, i.e. non-computerised excercises are described here. For computerised exercises see page 154.
Conventional or non-computerised exercises
Orthoptic treatment for convergence insuffi­ciency is by and large same as for exophoria and includes the following exercises:
(a) Exercises to improve near point of convergence
i. Advancement exercises (see page 228).
ii. Jump convergence exercises (see page 228).
iii. Pencil dips: This exercise involves dipping the
target (pencil) in and out of your field of vision to challenge your convergence ability.
168 Theory and Practice of Squint and Orthoptics
iv. Pencil push-ups: This is one of the most
commonly prescribed exercises for conver­gence insufficiency. Here's how it's done:
Hold a small target (like a pen or pencil)
vertically at arm's length in front of nose and focus on the target.
Slowly bring the target closer to your nose
while maintaining clear and single vision.
Stop the moment you notice double vision
or the target splitting into two.
Hold the target at that distance for a few
seconds, and then move it back to arm's length.
Repeat this process for about 10–15
repetitions, a few times a day.
iv. Brock string exercise: The Brock string is a
simple tool that can help train convergence and depth perception (Fig. 7.4).
Attach a colored string (around 3–4 feet
long) to a stationary object at eye level (Fig. 7.4A).
A
B
Fig. 7.4: Brock String exercise (A), and its three steps (B)
Place the other end of the string on the tip
of your nose (Fig. 7.4A).
Focus on the string, and you should see a
series of colored beads along the string.
As you focus on the string, the beads
should appear to merge into a single bead. This indicates that your eyes are converging properly.
Gradually move the end of the string closer
to your nose and repeat the process.
(b) Exercises to increase amplitude of fusional convergence
i. Convergence exercises with prisms (see page
227).
ii. Convergence exercises using synoptophore (see
page 227).
iii. Exercises using convergence card. Convergence
card consists of dots on either side; so also known as physiologic dot card. On one side of the card, dots are coloured red and on the other side blue. Dots identical in size are in the same place on each side of the card and these are of three sizes—large, medium and small.
To perform the exercise, the card is put in front
of the patient's eyes with one end of the card resting on his/her nose, with the large dots farthest away, so that he/she will see the red dots with one eye and the blue with the other eye. The patient is instructed to look at the large dots and to see them fused or blended together, then the middle dots and finally the smallest ones. The patient must be aware of heteronymous physiologic diplopia on those dots between his/ her eyes and the fused ones; and homonymous physiologic diplopia on those beyond the fused one.
With the use of convergence card, there is a great deal of retinal rivalry. If the patient is unable to do the exercises as instructed above, it may be easier for him/her, if the three dots are connected with a black line on both sides of the card (Fig. 7.5). Now, when fixating on the large size dots, he/she should see the lines as . When he/she is able to fuse the large dots easily, he/ she may then fixate on the middle size dots and see the black lines as X. Lastly, he/she should fixate the small size dots and see the black lines as .
Fig. 7.5 Convergence card.
169Anomalies of Convergence, Divergence and Accommodation
3. Prismotherapy
When all the exhaustive orthoptic exercises fail, then prismotherapy may be tried to relieve symptoms.
Base in prism reading glasses or bifocals with
prism in the lower segment are useful as relieving prisms.
Relieving prisms and bifocals in young age
should be avoided.
iv. Physiologic diplopia exercise using stereogram in the uncrossed position (see page 227).
v. Convergence exercise using diploscope (see page 149).
(c) Training of voluntary convergence
It is very helpful, if the patient is intelligent and co-operative. It aims at developing the control of the position of eyes. Patient is made to understand physiological diplopia which he/she practices. If a finger is brought in the field of vision while the patient is fixing a distant light, there will appear two fingers. Now, if the patient fixes at the finger, then there will appear two lights at the distance. While the finger is moved to and fro, the distance between the lights increases or decreases. Patient is asked to maintain the two lights apart as long as possible. The finger may again be brought in, if the two lights become single as soon as the finger is removed. This exercise is completed, when the patient is able to double the lights without the aid of the finger. Development of voluntary convergence goes a long way in relieving symptoms.
(d) Relaxation exercises
Relaxation using relative negative convergence may be carried out after the treatment by any of the following methods:
i. Stereogram in crossed position (see page 225).
ii. Divergence with prisms (see page 225).
iii. Synoptophore exercises (see page 225).
Criteria for good orthoptic management
Patient should be symptom free.
There should be good binocular convergence.
Voluntary convergence should be possible
easily.
Patient should have good fusional reserves.
4. Surgical treatment
As a last resort, when all other measures fail, especially when convergence insufficiency is associated with a large exophoria at near vision, medial rectus muscle resection can be performed in one or both eyes. In some cases, exophoria at near fixation tends to recur.
Convergence insufficiency treatment trial
Convergence insufficiency treatment trial (CITT) was conducted to compare three forms of vision therapy with office-based placebo therapy:
Office-based vision therapy with a trained therapist
along with home reinforcement. Children in this group came to the office once per week for a 60-minute therapy session with a trained therapist. During these sessions, the children worked on 4–6 procedures designed to improve the ability to converge the eyes. The children in this group also did home therapy for 15 minutes, 5 days per week to practice the procedures learned during the office visits.
Home-based pencil push-ups therapy. In this
group, the child had to follow a small letter on a pencil as the pencil was moved toward the bridge of his nose. His goal was to keep the letter clear and single, but to stop if the letter became double. The child was told to try and get the pencil closer and closer to the bridge of his nose each day. This was practiced for 15 minutes, 5 days per week.
Home-based computer vision therapy and pencil
push-ups. In this group, the child was given complex exercises using a computer program plus pencil push-ups.
Office-based placebo therapy. This group was
given placebo vision activities designed to simulate office-based therapy.
170 Theory and Practice of Squint and Orthoptics
CITT concluded that office-based vision therapy with a trained therapist plus at-home reinforce­ment was most effective in treating CI in children 9 to 17 years old.
CONVERGENCE INSUFFICIENCY ASSOCIATED WITH ACCOMMODATIVE INSUFFICIENCY
Convergence insufficiency in some patients may be secondary to accommodation insufficiency. Therefore, before treating the patient for a functional convergence insufficiency, it is important to rule out associated accommodation insufficiency.
Etiology
Secondary convergence insufficiency associated with primary accommodation insufficiency has been reported to occur in following conditions:
1. Early Adie's syndrome
2.Sequelae to head trauma, particularly posterior occipital or whiplash injury.
3. Subclinical viral encephalopathies.
4. Infectious mononucleosis
5. Diphtheria
6. As a conversion reaction.
Clinical features
1. Symptoms of the patients are similar to those of functional convergence insufficiency.
2. Near point of convergence (NPC) is reduced.
3.Near point of accommodation (NPA) is reduced drastically.
4.Accommodative convergence/accommo­dation (AC/A) ratio may be low or even absent.
Treatment
1. Orthoptic exercises alone are usually not
much effective. Exercises need to be combined with reading glasses.
2. Plus lenses for reading and base-in prism is
the treatment of choice. The reading spectacle prescription should be titrated according to patients need. The minimal power necessary to achieve comfortable vision should be pres­cribed. Fresnel membrane prisms glued on glass lenses in the lower segment of bifocals may be useful, since a frequent change may be necessary
before the final adjustment is made. Alter­natively, two piece executive bifocals with decentered plus lenses for prism power in the lower half may be prescribed.
3. Surgery is usually not indicated for this condition. However, it has been reported that resection of both medial rectus muscles followed by prescription of bifocal adds may be helpful in untreatable cases.
CONVERGENCE PARALYSIS
Convergence paralysis refers to a total lack of ability to overcome any amount of base-out prisms. It is an uncommon entity and should not be confused with functional convergence insufficiency which is very common.
Etiology
Convergence paralysis occurs secondary to some organic diseases of the brain in the region of corpora quadrigemina or the nucleus of third cranial nerve. The organic brain lesions reported to be associated with convergence paralysis are as follows:
Head injury
Encephalitis
Disseminated sclerosis
Tabes dorsalis
Narcolepsy
Tumours
Clinical features
Convergence paralysis is characterized by following features:
1. Convergence is completely absent. There is
acute onset of convergence failure.
2. Exotropia and crossed diplopia occur on
attempted near fixation only.
3. Adduction is normal.
4. Accommodation is usually normal. But in
some cases it may be reduced or even absent.
Parinaud's syndrome refers to convergence paralysis associated with vertical gaze paralysis.
Pretectum-posterior commissure syndrome
(dorsal midbrain syndrome) which is commonly caused by tumour in the pineal region includes:
Convergence paralysis
171Anomalies of Convergence, Divergence and Accommodation
Vertical gaze paralysis
Pupillary areflexia to light with light near
dissociation.
Bilateral fourth nerve paralysis may be present
sometimes.
Lid retraction may also occur in some patients.
Diagnosis
Bielschowsky's criteria for diagnosis of the convergence paralysis include: (1) evidence of intracranial disease, (2) history of sudden onset of crossed horizontal diplopia at near fixation, (3) reproducible findings on sub­sequent examinations and (4) preservation of accommodation and pupillary reaction on attempts to converge.
Differential diagnosis
Convergence paralysis can be differentiated from functional convergence insufficiency by use of base-out prisms. A patient with convergence paralysis will immediately have diplopia, while in convergence insufficiency several dioptres of convergence amplitude can be demonstrated.
Treatment
1. Base-in prisms are prescribed at near to
alleviate the diplopia at near.
2. Plus lenses with base-in prisms may be required
in patients having weakness of accommo­dation.
3. Occlusion of one eye at near may be indicated
in patients where it is not possible to restore comfortable single binocular vision.
4. Eye muscle surgery is contraindicated in this
condition.
CONVERGENCE SPASM
Convergence spasm refers to a condition characterized by intermittent episodes of maxi­mal convergence usually associated with spasm of accommodation.
Etiology
1. Functional causes. It has been reported that
in most of the cases, convergence spasm is functional in origin. It occurs in patients with hysteria or neurosis.
2. Organic causes. Rarely convergence spasm may be secondary to some underlying organic lesion. It has been reported to occur after head trauma, encephalitis, tabes, pituitary adenomas, posterior fossa neurofibroma and Arnold-Chiari malformation.
Clinical features
In most of the cases, the condition is episodic. In between the attacks, patients are normal. During the episode of convergence spasm, a patient may exhibit following clinical features:
1. Extreme convergence. Eyes may be fixed in a position of extreme convergence resembling bilateral abducens palsy.
2. Homonymous diplopia may be experienced. Patient may give history of intermittent diplopia.
3. Blurring of vision. During the attack, patient may have blurred vision for near due to associated spasm of accommodation. Patient may come with a complaint of difficulty in reading.
4. Miosis. Pupils usually become miotic, as a part of near reflex.
5. Induced myopia to the tune of 6D (confirmed by retinoscopy) has been reported due to associated spasm of accommodation. Thus, during the attack, patient may also have a reduced distance visual acuity.
Psychiatric examination may reveal the underlying hysteria and neurosis in many cases.
Management
Neurological evaluation. Organic lesions are a rare cause of spasm of convergence; however, each patient should undergo detailed neuro­logical evaluation to rule out associated condition, if any.
Treatment of functional spasm of convergence includes:
1.Prolonged atropinization with plus lenses in lower
segment of bifocals for near work may be required to break the cycle.
2.Alternate monocular occlusion may be
considered as an alternative to atropinization.
3.Psychiatric work-up and therapy is useful as a
long-term measure.
172 Theory and Practice of Squint and Orthoptics

DIVERGENCE

As mentioned earlier, divergence refers to disjugate but symmetrical and simultaneous outward rotation of the two eyes from a given position. It is unknown whether voluntary divergence exists. Certainly, it is possible to relax one's convergence voluntarily and to produce in this way a more divergent position of the eyes. Thus, from clinical point of view, the only significant form of divergence is fusional divergence.
Fusional divergence
Fusional divergence, also called negative fusional convergence, is the divergence that is produced (like the positive fusional convergence) to ensure that similar retinal images are projected onto corresponding retinal points of two eyes. It occurs without a change in refractive state of eye and is initiated by a binasal retinal image disparity. In other words, fusional divergence implies a responsiveness to disparate stimuli lying outside of Panum's fusional area. Similar to fusional convergence, it is a kind of optomotor reflex and includes:
1. Positive fasional divergence: It is the ability
of the eyes to move outward to maintain binocular single vision when the eyes focus from near to distant object. It is measured using base out prism test.
Base-out prism test (BO Test): In this test, base­out prisms are placed in front of one eye while the patient fixates on a near target. The examiner gradually increases the prism power until the patient reports seeing double or experiences a break in fusion. The amount of prism required to break fusion provides information about positive fusional divergence.
When fusional divergence amplitudes are measured with a prism bar, the normal range is 4D to 6D at distance and 8D to 12D at near. As discussed earlier, in the case of fusional convergence, the end point of the amplitude (i.e. the point of maximal convergence) is marked by an identical phenomenon, that is, blurring of vision both for distance and near. However, in fusional divergence, the end point of amplitude differs in character for near
(blurring of vision) and distance (double vision) as follows:
End point of amplitude of fusional divergence for
distance: Beyond the maximal limit of fusional divergence, there occurs diplopia (end point).
End point of amplitude of fusional divergence for
near: In near vision, as we know, the eyes are accommodated and also converged. Under these circumstances, when measuring the amplitude of fusional divergence beyond the limits of maximal fusional divergence the accommodative convergence is relaxed to prevent retinal disparity. But along with the relaxation of accommodative convergence, the accommodation is also relaxed resulting in blurring of vision (end point).
2. Negative fusional divergence (base-in
divergence): Negative fusional divergence is the
ability of the eyes to move inward (converge) to maintain single binocular vision when the eyes are initially diverged for a distant object, and then the target moves closer. This type of divergence is less commonly assessed but is still important for binocular vision.
Base-in prism test (BI test): Similar to the BO
test, the BI test involves the use of base-in prisms. Prisms are introduced in front of one eye, and the patient fixates on a distant target. The prism power is gradually increased until the patient reports seeing double or experiences a break in fusion. The amount of prism required to break fusion indicates negative fusional divergence.
Convergence to divergence ratio (Con/D): This ratio represents the balance between convergence and divergence abilities. It is calculated by comparing the amount of convergence (measured in prism diopters) that a person can achieve to the amount of divergence. A Con/D ratio of 3:1 is often considered normal. Divergence facility test: Divergence facility refers to the speed and accuracy with which the eyes can move outward to diverge. This test typically involves a flipper with base-out prisms. The patient alternates between different divergence targets with varying prism powers while maintaining fusion. The speed and ease with which the patient can maintain fusion provide information about their divergence facility.
173Anomalies of Convergence, Divergence and Accommodation
Measurement of amplitude of divergence
See page 124.
ANOMALIES OF DIVERGENCE
DIVERGENCE INSUFFICIENCY
Divergence insufficiency is not synonymous with divergence paralysis, but is a separate clinical entity.
Etiology
Exact etiology is not known. It has been assumed that perhaps in divergence insufficiency, a pre­existing esophoria at distance fixation caused by an anomalous position of rest becomes manifest.
Clinical features
Characteristic features are as follows:
1. Esodeviation. Intermittent or constant eso-
deviation is much larger at distance than near. Usually, patients maintain the fusion at near. Esodeviation is comitant in all fields of gaze.
2. Ductions and versions are normal in all the
directions.
3. Fusional divergence is markedly reduced both
at distance and near fixation.
4. Absence of any neurologic disease (e.g.
divergence paralysis).
Treatment
1. Orthoptic exercises for divergence insufficiency
i. Pencil/pen divergence: This exercise helps
strengthen the eye muscles responsible for diverging. It is the opposite of the pencil push-up exercise used for convergence insufficiency.
Hold a pencil or pen vertically at arm's
length, with the tip at your eye level.
Focus on the tip of the pencil.
Slowly move the pencil away from your
nose while maintaining clear and single vision.
Stop if you experience double vision and
then bring the pencil back closer to your nose.
Perform this exercise for about 10–15
repetitions, several times a day.
ii. Brock string exercise: The Brock string (Fig. 7.4)
can be adapted to train divergence abilities by using colored beads that appear to move apart.
Attach a colored string (around 3–4 feet
long) to a stationary object at eye level.
Hold the other end of the string to your
nose.
Focus on the string, and you should see a
series of colored beads along the string.
As you focus on the string, the beads
should appear to move further apart, indicating that your eyes are diverging properly.
Gradually move the end of the string
closer to your nose and repeat the process.
iii. Near-far target switching: This exercise helps
improve the coordination between near and far vision.
Hold a target (e.g. a finger or small object)
at arm's length, focusing on it.
Without moving your head, shift your
focus to a distant object in the background.
Alternate between the near and far targets,
working on the smooth transition of your gaze.
2.Computer based exercises for divergence insufficiency
Some computer-based vision therapy programs offer exercises specifically designed to improve divergence abilities. These programs often involve tracking moving objects that require your eyes to diverge correctly. Computer based exercises and tools that may help with divergence insufficiency include:
i. Interactive vision therapy software: Several
vision therapy software programs are designed to improve eye coordination and alignment. These programs typically offer exercises that challenge the eyes to diverge correctly. Some examples include:
Vectograms: These computer-generated
images create an illusion of depth, and the patient is asked to identify specific patterns within the images while maintaining proper eye alignment.
Brock string simulation: Some software
programs simulate the brock string exercise, which involves tracking colored
174 Theory and Practice of Squint and Orthoptics
beads on a string and working to keep them single or separated at different distances.
ii. 3D and virtual reality (VR) exercises: Three-
dimensional and VR exercises can provide an immersive experience to improve divergence abilities. These exercises often involve interacting with 3D objects or virtual environments that require proper eye coordination.
iii. Online vision therapy platforms: Some online
platforms offer comprehensive vision therapy programs, including computer­based exercises tailored to address specific vision problems like divergence insufficiency. These programs are typically supervised by eye care professionals.
iv. Interactive games: There are video games and
apps designed to improve eye coordination and divergence skills. These games may involve tracking moving objects or completing visual tasks that require precise eye movements.
3. Prismotherapy. Divergence insufficiency is a self-limiting condition. However, to alleviate the patient of symptoms, prism base-out is prescribed to provide comfortable single vision at distance. Prism power is decreased stepwise in months and ultimately discarded.
4. Surgical treatment. In the form of resection of both lateral rectus muscles is indicated, rarely when the prismotherapy fails.
DIVERGENCE PARALYSIS
Divergence paralysis is a rare entity charac­terized by a sudden onset esotropia at distance fixation and a homonymous diplopia.
Etiology
Divergence paralysis may be associated with a head trauma or other neurologic diseases such as encephalitis, tabes, disseminated sclerosis, vascular disorder, neoplasm and raised intracranial pressure.
Clinical features
Clinical features of divergence paralysis are similar to divergence insufficiency except that it is of sudden onset and usually associated with some organic neurologic diseases.
Differential diagnosis
1. Unilateral or bilateral sixth nerve palsy.
Clinically, a paralysis of fusional divergence is differentiated from the bilateral sixth cranial nerve paralysis by the fact that in the former, at a given distance, the deviation is comitant and that the distance between the double images remains the same irrespective of the position of the object of regard, as it is moved laterally in the radius of a circle with the patient at the centre. This observation indicates that perhaps a separate divergent centre exists. However, still no specific midbrain or central nervous system centre has been proved to be divergence centre that abduction is completely normal bilaterally.
2. Convergence spasm may also be confused with
divergence paralysis because in both the conditions patients have uncrossed diplopia at distance fixation. However, in convergence spasm visual acuity is reduced due to induced myopia and the divergence fusional range is normal.
Management
A thorough neurologic work-up is required for any associated disease. However, divergence paralysis has no localizing significance for the neurologist. Further, despite the association of neurologic diseases, mostly divergence paralysis is self-limiting and disappears within 5 to 6 months. During this period, prismotherapy as described for divergence insufficiency is required. Bilateral lateral rectus resection may be indicated in some cases where divergence paralysis persists beyond 6 months.
ACQUIRED MOTOR FUSION DEFICIENCY
Acquired motor fusion deficiency refers to a combined deficiency of the fusional convergence and fusional divergence.
Etiology
It has been assumed that, perhaps, a midbrain lesion accounts for the acquired motor fusion deficiency. The lesions reported to be associated with this rare entity are as follows:
Closed head trauma
Cerebrovascular accidents
Intracranial tumours
Brain surgery
Clinical features
1.Asthenopia. Most patients complain of severe
asthenopic symptoms.
2.Diplopia is usually intractable and variable, i.e.
may be crossed at one moment and uncrossed at another.
3.Fusional amplitudes (convergence as well as
divergence) are either markedly decreased or completely absent.
4.Sensory fusion and stereopsis are intact during
the brief moments and that such patients may be able to superimpose the double images.
5.Accommodation range may be decreased.
Differential diagnosis
Acquired motor fusion deficiency needs to be differentiated from following conditions:
1.Combined convergence and accommodation deficiency (see page 170).
2.Motor fusion deficiency in adults due to prolonged unilateral use of the eye as seen with unilateral cataract or untreated unilateral aphakia.
3.Malingering: Some patients may malinger for diplopia-like acquired motor fusion deficiency following work-related trauma to get compen­sation. Measurement of fusional amplitudes with a rotatory prism helps in differentiating the two conditions.
Treatment
Treatment of this condition is usually frustrating. Prisms cannot be employed because of frequent adjustments for near and distance. The only way to get relief from symptoms is unilateral occlusion. Infrequently, a spontaneous recovery may occur.

ACCOMMODATION

ACCOMMODATION AND RELATED TERMS
Accommodation
As we know that in an emmetropic eye, parallel rays of light coming from infinity are brought to focus on the retina, with accommodation at rest. Our eyes have been provided with a unique mechanism by which we can even focus the diverging rays coming from a near object on the
175Anomalies of Convergence, Divergence and Accommodation
Fig. 7.6 Effect of accommodation on divergent rays entering
the eye.
retina in a bid to see clearly (Fig. 7.6). This mechanism is called accommodation. In it, there occurs increase in the power of the crystalline lens.
Far point, near point, range and amplitude of accommodation
The nearest point at which small objects can be seen clearly is called near point or punctum proximum and the distant (farthest) point is called far point or punctum remotum. The distance between the near point and the far point is called range of accommodation. The difference between the dioptric power needed to focus at near point (P) and to focus at far point (R) is called amplitude of accommodation (A). Thus, A = P – R.
Far point and near point of the eye vary with the static refraction of the eye. In hypermetropic eye, far point is virtual and lies behind the eye, while in myopic eye, it is real and lies in front of the eye (Fig. 7.7). In an emmetropic eye, far point is at infinity and near point varies with age; being about 7 cm at age of 10 years, 25 cm at the age of 40 years and 33 cm at the age of 45 years. Thus the amount that the eye can alter its refraction is greatest in childhood and slowly decreases until it is lost in middle age. Amplitude of accommodation in dioptres as function of the age, as studied by Duane is depicted in Fig. 7.8.
ASSESSMENT OF ACCOMMODATION
See page 125.
AGE-RELATED CHANGES IN ACCOMMODATION
As discussed earlier, in an emmetropic eye, far point is infinity and near point changes with age, being about 7 cm at the age of 10 years, 25 cm at the age of 40 years, 33 cm at the age of 45 years