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Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3860_Библиотеки_им_академика_М_И_Перельмана

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284 J.J. Trambert
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nosis. Renal cell carcinoma, Wilms’ tumor, and even an­giomyolipoma and benign renal adenomas can present similar angiographic appearances. In addition, renal cell carcinoma, although usually hypervascular, occasionally is avascular, making angiographic distinction from a sim­ple cyst difficult (Fig. 23-15). Inflammatory infiltrative disease such as xanthogranulomatous pyelonephritis sometimes results in a neovascularity similar to a moder­ately vascular renal cell carcinoma. Frequently, the cor­rect diagnosis is made by correlating clinical history, non­invasive imaging studies, and angiographic findings; however, the definitive diagnosis is not available until histologic evaluation of the resected surgical specimen is complete.
REFERENCES
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2. Kadir, Saadoon. Diagnostic angiography. Philadelphia: WB Saunders, 1986:457–458.
3. Bookstein JJ, Maxwell MH, Abrams HL, et al. Cooperative study of the radiologic aspects of renovascular hypertension. JAMA 1977; 237:1706–1709.
4. Knutson DW, Abt AB. Pathophysiology, pathology, and clinical fea­tures of renovascular hypertension. In: Strandness DE, van Breda A, eds. Vascular diseases: surgical and interventional therapy. New York: Churchill Livingstone, 1994.
5. Thornbury JR, Stanley JC, Fryback DG. Hypertensive urogram: a nondiscriminatory test for renovascular hypertension. AJR Am J Roentgenol 1982;138:43–49.
6. Nally JV Jr. Provocative captopril testing in the diagnosis of renovas­cular hypertension. Urol Clin North Am 1994;21:227–234.
7. Sfakianakis GN, Bourgoignie JJ, Jaffe D, et al. Single dose captopril scintigraphy in the diagnosis of renovascular hypertension. J Nucl Med 1987;28:1383–1392.
8. Berland LL, Koslin DB, Routh WD, et al. Renal artery stenosis: prospective evaluation of diagnosis with color duplex ultrasound compared with angiography. Radiology 1990;174:421–423.
9. Schwerk WB, Restrepo IK, Stellway M, et al. Renal artery stenosis: grading with image-directed Doppler ultrasound evaluation of re­nal resistive index. Radiology 1994;190:785–790.
10. Grist TM, Kennell TW, Sprivat IA, et al. Prospective evaluation of renal MR angiography: comparison with conventional angiography in 35 patients. Radiology 1993;189(P):190.
11. Kim D, Edelman RR, Kent KC, et al. Abdominal aorta and renal artery stenosis: evaluation with MR angiography. Radiology 1990; 174:727–731.
12. Galanski M, Prokop M, Chavan A, et al. Renal arterial stenosis: spiral CT angiography. Radiology 1993;189:185–192.
13. Holly KE, Hunt JC, Brown AL, et al. A clinical pathologic study in normotensive and hypertensive patients. Am J Med 1964;37:14–22.
14. Pickering TG, Sos TA, Vaughan ED, et al. Predictive value and changes of renin secretion in hypertensive patients with unilateral renovascular disease undergoing successful renal angioplasty. Am J Med 1984;76:398–404.
15. Atnip RG, Thiele BL. Surgical management of renovascular hyper­tension. In: Strandness DE, van Breda A, eds. Vascular diseases: surgical and inter ventional therapy. New York: Churchill Livingstone, 1994:703–720.
16. Weibull H, Bergqvist D, Bergentz SE, et al. Percutaneous translumi­nal renal angioplasty versus surgical reconstruction of atheroscle­rotic renal artery stenosis: a prospective randomized study. J Vasc Surg 1993;18:841–852.
17. Sos TA, Pickering TG, Saddekni S, et al. The current role of renal angioplasty in the treatment of renovascular hypertension. Urol Clin North Am 1984;11:503–512.
18. Tegtmeyer CJ, Sos TA. Techniques of renal angioplasty: state of the art. Radiology 1986;161:577–586.
19. Martin LG, Rees CR, O’Bryant T. Percutaneous angioplasty of the renal arteries. In: Strandness DE, van Breda A, eds. Vascular diseases: surgical and interventional therapy. New York: Churchill Livingstone, 1994:721–741.
20. Miller RL, Fontaine AB, Nahman NS Jr, et al. Balloon angioplasty improves and stabilizes glomerular filtration rate in patients with bilateral renal artery stenosis or single kidney with renal artery stenosis. SCVIR 21st Annual Scientific Meeting. J Vase Interv Radiol 1996;7(suppl):140.
21. Henry M, Amor M, Henry I, et al. Renal artery stent placement with the Palmaz stent: six year single center experience. SCVIR 21st Annual Scientific Meeting. J 140–141.
22. Flechner SM. Current status of renal transplantation. Urol Clin North Am 1994;21:265–282.
23. Orrons PD, Zaiko AB. Angiography and interventional aspects of renal transplantation. Radial Clin North Am 1995;33:461–471.
24. McAninch JW, Carroll PR. Renal exploration after trauma: indica­tions and reconstructive techniques. Urol Clin North Am 1989; 16:203–212.
25. Peterson NE. Complications of renal trauma. Urol Clin North Am 1989;16:224.
26. Kantor A, Sclafani SJA, Scalea T, et al. The role of interventional radiology in the management of genitourinary trauma. Urol Clin North Am 1989;16:255–264.
27. Huppert PE, Duda SH, Erley CM, et al. Embolization of renal vascular lesions: clinical experience with microcoils and tracker catheters. Cardiovasc Interv Radiol 1993;16:361–367.
28. Eastham JA, Wilson TG, Larsen DW, et al. Angiographic emboliza­tion of renal stab wounds. J Urol 1992;148:268–270.
29. Longstreth PL, Korobkin M, Palubinskas AJ. Renal microaneurysms in a patient with systemic lupus erythematosus. Radiology 1974;113: 65–66.
30. Kopchick JH, Boume NK, Fine SW, et al. Congenital renal arte­riovenous malformations. Urology 1981;17:13–17.
31. Beinart C, Sniderman KW, Saddekni S, et al. Left renal vein hyper­tension: a cause of occult hematurial. Radiology 1982;145:647–650.
32. Trambert JJ, Rabin AM, Weiss KL, et al. Pericaliceal varices due to the nutcraker phenomenon. AJR Am J Roentgenol 1990;154:305–
306.
33. Mitty HA, Goldman H. Angiography in unilateral renal bleeding with a negative urogram. AJR Am J Roentgenol 1974;121:508–517.
34. Elkin ME. Radiology of the urinary system. Boston: Little, Brown and Company, 1980:297.
35. Bruenton JN, Ballanger P, Ballanger R, et al. Renal adenomas. Clin Radiol 1979;30:343–352.
36. Weiner SN, Bernstein RG. Renal oncocytoma: angiographic fea­tures of two cases. Radiology 1977;125:633–635.
37. Amis ES, Newhouse JH. Essentials of uroradiology. Boston: Little, Brown and Company, 1991:127.
38. Elkin ME. Radiology of the urinary system. Boston: Little, Brown and Company, 1980:344.
39. Soulen MC, Faykus MH, Shlansky-Goldberg RD, et al. Elective em­bolization for prevention of hemorrhage from renal angiomyolipo­mas. J Vasc Interv Radiol 1994;5:587–591.
40. Kadir S. Diagnostic angiography. Philadelphia: WB Saunders, 1986:
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41. Amis ES, Newhouse JH. Essentials of uroradiology. Boston: Little, Brown and Company, 1991:128.
42. Newsam JE, Tulloch WS. Metastatic tumors in the kidney. Br J Urol 1966;38:1–6.
43. Elkin ME. Radiology of the Urinary System. Boston: Little, Brown and Company, 1980:296.
44. Amis ES, Newhouse JH. Essentials of uroradiology. Boston: Little, Brown and Company, 1991:142.
45. Abrams HL. Abram’s angiography, 3rd ed. Boston: Little, Brown and Company, 1983:1136.
46. Bakal CW, Cynamon J, Lakritz PS, et al. Value of preoperative renal
artery embolization in reducing blood transfusion requirements during nephrectomy for renal cell carcinoma. J Vasc Interv Radiol 1993;4:727–731.
47. Wallace S, Charnsangavej C, Carrasco CH, et al. Interventional radiology in renal neoplasms. Semin Roentgenol 1987;22:303–315.
48. Lanigan D, Jurriaans E, Hammonds JC, et al. The current status of embolization in renal cell carcinoma—survey of local and national practice. Clin Radiol 1992;46:176–178.
49. Kadir S. Diagnostic angiography. Philadelphia: WB Saunders, 1986:
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M.E. RacksonObstructiveUropathy and Renal Calculus Disease
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24
■■■
Obstructive Uropathy and
Renal Calculus Disease
MARLENE E. RACKSON
Ureteral obstruction leads to dilation of the proximal ure­ter and hydronephrosis. This chapter deals with percu­taneous nephrostomy (PCN) and ureteral stenting pro­cedures, which are used to relieve obstruction and help in the management of renal calculus disease.
■ Clinical Presentation
Many patients presenting with obstructive uropathy have malignancies; the etiology and the obstruction can be bilateral or unilateral. When both kidneys are obstructed, the patient will present in renal failure with elevated blood urea nitrogen (BUN) and creatinine levels. This clinical picture often is seen in patients who have prostate cancer, bladder cancer, or gynecologic malignancies. Be­cause malignancies grow slowly, they cause the kidney to dilate slowly. Thus, malignant obstruction is usually pain­less. Unilateral obstruction caused by malignancy may be difficult to diagnose clinically if the other kidney is func­tioning normally, because the obstruction is painless and the BUN and creatinine levels may be normal. Unsus­pected unilateral obstruction often is detected during the course of an imaging study such as ultrasound or com­puted tomography (CT) of the abdomen. The decision to treat an asymptomatic unilateral obstruction caused by malignancy is a clinical one that may be influenced by many factors, such as the need to optimize renal function before administration of chemotherapy with known ne­phrotoxicity (Fig. 24-1).
Patients undergoing PCN for benign causes of obstruc­tion usually have calculi or strictures. Only a small frac­tion of patients presenting with renal stones require per-
4,5
1–3
cutaneous decompression. In the acute setting, a patient with pyonephrosis may have severe flank pain, fever, ele­vated white blood cell (WBC) count, and other signs of infection along with a stone and hydronephrosis identi­fied on an imaging study. performed to provide preoperative access to the renal collecting system in patients about to undergo percutane­ous nephrolithotomy (PNL).
Ureteral strictures also may be treated by percutaneous methods. Causes of strictures include radiation, retroperi­toneal fibrosis, prior instrumentation, and fistulae. These patients may present with one or many signs and symp­toms: fever, flank pain, renal failure, hydronephrosis, or urinoma formation on imaging studies.
Patients with renal allografts (transplants) may present with urine leaks, ischemic ureter, and obstruction and may be treated with PCN. Percutaneous procedures also are used to perform Whitaker tests (see Chapter 9), which measure the pressure gradient across a ureteral stenosis, to infuse antibiotics for fungal infections, to infuse agents for stone dissolution, and to provide access for removal of encrusted stents.
■ Differential Diagnosis
The need fora PCN is generally straightforward. The pres­ence or absence of hydronephrosis can be determined quickly, safely, and with certainty by ultrasound or CT scan­ning. The proper clinical setting will confirm the need for the procedure. Hydronephrosis can be due to reflux,how­ever, and does not always mean obstruction or translate into a need for a PCN. Furthermore, it is important to
8–10
6
In the elective setting, PCN is
7
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FIGURE 24-1. Percutaneous nephrostomy (PCN) placed to relieve ureteral obstruction resulting from cervical carcinoma. Note that the tube enters the collecting system below the costal margin through a posterior mid–lower pole calyx, which pro­vides safe avascular access and a good “pushing angle” for future attempts at ureteral stent placement. Irregularity of up­per ureter is due to ureteritis cystica.
inine levels are rising but there is minimal hydroneph­rosis, also can occur. Although the renal failure is most likely due to medical renal disease, there is also the possi­bility of obstruction without dilation, which occurs when the kidney shuts down before it dilates. This type of ob­struction has been reported in patients with prostatic car­cinoma.
11
Although this situation is unusual, it should be kept in mind because percutaneous decompression will improve renal function.
PCN should be performed as an emergency proce-
12
dure
in the following situations:
1. In the setting of high fever and elevated WBC count or frank sepsis, when the patient is suspected of having pus in the kidney: This is usually seen in a patient with hydronephrosis and an identifiable ob­structing calculus causing urosepsis.
2. Iatrogenic trauma to the ureter that is recognized when it happens: If the urologist calls from the operating room and says that the ureter has been dissected, the patient should have a PCN to divert the urine and to avoid urinoma formation. Stent placement to preserve the integrity of the ureter and avoid stricture formation should be attempted.
3. Severe unmanageable flank pain resulting from an obstructing calculus or steinstrasse from a preced­ing extracorporeal shock-wave lithotripsy (ESWL) treatment: Decompression via a PCN (antegrade approach) or a ureteral stent placed by the urologist (retrograde approach) will provide pain relief.
4. Acute renal failure resulting from bilateral obstruc­tion or obstruction in a solitary kidney: This proce­dure should be done urgently to preserve renal parenchyma and to induce a diuresis to correct fluid and electrolyte imbalances.
determine whena PCNis elective and whenthe procedure needs to be performed as an emergency.
Some patients have chronic hydronephrosis (deter­mined from serial imaging studies) with a new elevation of the BUN and creatinine levels. In the patient with a solitary kidney, PCN is certainly indicated to preserve the remaining renal parenchyma. The same is true when one kidney demonstrates cortical atrophy and the other dem­onstrates relatively normal cortex: The more normal kid­ney should be decompressed. When bilateral cortical at­rophy and hydronephrosis are present, progressive renal failure may not be stabilized or improved by decompres­sion. Sometimes the only way to prove or disprove the value of decompression in preserving renal function is to perform the PCN and then remove the tube if the kidney does not produce urine or if the function on a nuclear medicine scan does not improve.
The opposite situation, in which the BUN and creat-
■ Treatment Alternatives
Whenever possible, the retrograde approach (i.e., oppo­site the direction of flow of urine) to ureteral stent place­ment is preferable to the antegrade approach. The retro­grade approach, usually performed by the urologist, has the advantage of avoiding creation of an 8 or 10 Fr hole extending from the flank to the kidney, which can be complicated by bleeding and may produce pain. Retro­grade ureteral stent placement is also a one-step proce­dure, whereas antegrade stent placement may require at least two visits to the radiology department: first for the initial nephrostomy and then for placement of the stent. Until the antegrade stent has been placed and the urine is free of blood, the patient must wear an external neph­rostomy bag (so that the stent will not occlude from blood clots). Patients who have small ureteral calculi and require stents with normal bladders and visible ureteral
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A B
FIGURE 24-2. A: “Upside down” percutaneous nephrostomy (PCN) to relieve hydronephrosis in a patient with an ileal conduit.
After standard PCN access is achieved, a guidewire is maneuvered down the ureter into the conduit. The guidewire is retrieved from the conduit, and an “upside down” PCN is inserted. The external portion of the PCN lies in the ostomy bag rather than exiting the flank. Completely internalized stents occlude rapidly because of mucus production in the conduit. B: Tubes can be changed easily over a guidewire from the ostomy site.
orifices and patients undergoing precautionary ureteral stenting prior to bowel or pelvic surgery are examples of two situations in which retrograde ureteral stenting is preferable to the antegrade approach and is usually suc­cessful. In patients with malignant obstructions at the level of the ureterovesicle junction, the ureteral orifice often cannot be identified and attempts at retrograde
vention usually are treated by ESWL rather than by PNL; however, when ESWL is not applicable or has failed, per­cutaneous approaches areused. Situations in which ESWL is not applicable are stones larger than 3 cm, cysteine stones, lucent or infected stones, large staghorn calculi, and stones trapped in an infected calyx. Open surgery is
rarely necessary for the treatment of stone disease. stent placement will usually fail. In fact, many patients undergo percutaneous procedures after failure of the retrograde approach.
■ Imaging Workup
Although retrograde approaches usually areperformed by the urologist, they also can be performed by the inter­ventional radiologist. This approach is easier in female patients because the urethra is shorter than in male pa­tients. On occasion, the urologist will be able to advance a small catheter fromthe urethra into the ureter butwill not be able to place a true stent. If the patient is transferred to the radiology suite with the catheter still in place, the interventionalist often will succeed in retrograde place­ment of the stent, thereby avoiding nephrostomy. Retro­grade approaches also can be useful in patients with ileal conduits and other diversionary pouches (Fig. 24-2).
13
Actually, most cases of stone disease that require inter-
When a patient is referred for a PCN to relieve obstruc­tion, an imaging study is needed to prove hydronephrosis. This may be done quickly and easily by using ultrasound, which also will demonstrate cortical thickness and the level of obstruction, as well as the presence of stones and potential technical difficulties such as large cysts, uri­nomas, and duplicated collecting systems. The ultrasound machine also can be wheeled into the interventional suite and can be very helpful in localizing the kidney. The more costly CT scan gives the same information as the ultra­sound, but it also shows the level of the kidney, which, if it is located unexpectedly high or low, could make fluoro-
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scopic localization difficult. Sometimes an excretory uro­gram is performed just before the PCN (usually in stone cases); if there is contrast in the collecting system of the obstructed kidney, access to the collecting system is facili­tated.
PCNs often are performed to provide access to the
renal collecting system prior to PNL.
14,15
Excretory uro­grams and retrograde pyelograms are best for locating the number and size of stones in the intrarenal collecting system before access for removal. When outpatients ar­rive for procedures, often only a clinical history has been provided but no studies are available. In this situation, good prone scout films with 30-degree obliques before and after contrast is injected should be obtained, either intravenously or through an antegrade pyelogram (direct skinny-needle puncture into the renal pelvis).
■ Interventions: Percutaneous
Nephrostomy and Ureteral Stenting
The approach to the PCN could be titled, “Why are we doing this case?” The goal of the procedure will deter­mine how general or specific the puncture into the col­lecting system needs to be. The main goals of PCN are to relieve obstruction (the simplest approach), to stent the ureter (the more specific approach), and to provide ac­cess for stone removal (the approach that requires the most planning and is the most specific).
PCN to relieve obstruction
When the goal of the PCN is to relieve obstruction by providing external drainage without planning any further interventions, a nonspecific approach will suffice. Essen­tially any access from the flank to the intrarenal collecting system will be sufficient as long as the tube traverses the renal cortex (to anchor the tube) and enters through a calyx (an avascular area that does not have large crossing vessels) and direct pelvic punctures are avoided (see Fig. 24-1). The initial puncture can be made into the renal pelvis from a direct posterior translumbar approach in order to opacify the collecting system; a skinny (21- or 22-gauge) needle should be used. The final entry site into the kidney should be roughly along the posterior axillary line, below the 12th rib. An entry site that is too medial will be too posterior and will be uncomfortable for the patient. An entry site that is too lateral runs the risk of puncturing the colon. A subcostal approach will avoid the risk of pneumothorax and the potential for periosteal pain from a tube placed between the ribs as well as avoid­ing the liver and spleen. as the Neff set (Cook, Inc., Bloomington, IN) or the Accu­stick set (Medi-tech, Boston, MA) have been more fully described previously. Establishment of percutaneous ac-
16,17
Percutaneous access kits such
cess is followed by tract dilatation and placement of the PCN tube.
When frank pus is aspirated from the intrarenal collect­ing system, only external drainage should be performed. Contrast injections will pressurize the collecting system and increase the risk of sepsis resulting from pyelovenous backflow. These injections should be limited to a few milliliters, just enough to confirm needle placement in the collecting system. Catheter and guidewire manipula­tions in a grossly infected collecting system also increase the risk of sepsis resulting from pyelovenous backflow and must be minimized. The aspirated material should be sent for Gram’s stain and culture and sensitivity.
The initial drainage procedure in a renal transplant patient should be kept simple. Because the allograft is placed anterior to the iliac wing in the pelvis, the proce­dure is done with the patient in a supine position, not prone (Fig. 24-3). The hydronephrotic renal pelvis is anterior to the calyces and is sometimes palpable under the anterior abdominal wall. Therefore, the initial drain­age procedure is often through a puncture directly into the renal pelvis.
8
Ultrasound guidance not only localizes the transplant kidney, it also eliminates the possibility of transgressing an interposed bowel loop.
Ureteral stenting and specific techniques of stent insertion
Internal ureteral drainage through a stent is preferable to external drainage with a leg bag for the urine. Whenever possible, access for the PCN should be planned in antici­pation of ureteral stent placement, which entails creating a favorable angle for pushing a catheter across a ureteral lesion. In a normally located kidney, middle-pole calyces are preferable to lower-pole calyces, which require push­ing a catheter upward to the renal pelvis and then down­ward into the ureter. If the kidney is high above the ribs and the patient cannot inspire deeply enough to depress the kidney, only lower-pole calyces will be accessible for puncture. Conversely, if the kidney is ptotic, superior pole calyces may be low enough to puncture and will provide a straight downward line of pushing force into the ureter.
Ureteral stenting is indicated in the presence of be­nign and malignant strictures, fistulae, some stones, trauma, and iatrogenic complications (Fig. 24-4). The length of time a ureteral stent remains in place depends on why the stent was placed. Stents for strictures and fistulae can be in place anywhere from 3 weeks to 3 months. Stents for malignant strictures are usually per­manent. All patients who require long-term stenting should have the stents exchanged every 3 to 6 months to prevent encrustation and occlusion of the stent by uri­nary sediment. The stent can be inserted at the time of the original PCN or after the kidney has been decom­pressed at a separate session.
18–20
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A
C
There are two basic types of stent: the completely inter­nalized ureteral stent and the nephroureteral stent (NU) (Fig. 24-5).
The internalized stent
The internalized ureteral stent lies completely within the body and extends the length of the ureter from the renal pelvis to the bladder.This stentis commonlycalled adouble pigtail or doubleJ stent, which refers to the shape of the tips of the stent. The proximal pigtail or J-shaped tip anchors the stent in the renal pelvis and prevents it from slipping down into the bladder. The distal pigtail prevents the tip
B
FIGURE 24-3. Distal ureteral stricture in a transplant kidney. A: Antegrade nephrostogram demonstrates marked hy-
dronephrosis with a stricture of the distal ureter. Note the position of allograft over the iliac bone and the presence of a J-tip “safety wire” in the renal pelvis. B: The stricture was dilated with a 12-mm-high pressure balloon. C: An internal–ex­ternal stent was placed across the stricture for 6 weeks follow­ing the procedure.
from wedging into the bladder wall and causing discom­fort or exiting the urethra. For a patient who requires long-term stenting, a completely internalized stent is pref­erable because the patient does not have to wear a leg bag with its attendant discomfort and lifestyle inconveniences. Interventional radiologists usually place 8 or 10 Fr stents from the antegrade approach, whereas urologists usually place 5 or 6 Fr stents from the retrograde approach.
The nephroureteral stent
The NU is a one-piece combination of a nephrostomy tube and an internal ureteral stent that extends from the
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A B
FIGURE 24-4. Bilateral percutaneous nephrostomy (PCN) tubes placed for urinary diversion after attempt at suprapubic
cystostomy resulted in an intraperitoneal bladder perforation. A: Cystogram demonstrates contrast extravasation superior to the bladder. B: Bilateral PCN tubes alongside ureteral occlusion balloons placed to prevent any urine from flowing into the bladder. (
arrows
)
skin into the renal pelvis and descends through the ure­ter to terminate in the bladder. The NU can be left to external drainage with a leg bag, or it can be capped off to promote internal drainage.
Internalized ureteral stents usually are replaced by us-
ing cystoscopy.
21
If the exchange is anticipated to be difficult, an NU tube that is capped off to external drain­age is a good solution, because the tube can be simply replaced over a guidewire.
The NU often is placed during the initial PCN pro­cedure when an internal stent is eventually planned. Frequently, the ureteral obstruction can be transversed during the initial PCN, but the procedure is bloody.
the trial of internal drainage fails, the internal portion of the NU may be occluded, or there may also be a bladder outlet obstruction. If an internal stent is placed in the presence of a bladder outlet obstruction, hydronephrosis would persist because the urine in the bladder would reflux up the stent and the patient would still require a Foley catheter to drain the bladder.
22
An NU tube left to external drainage will decompress the kidney and the urinary bladder, which drains the other kidney, without the need for the Foley. Even if the patient has only one kidney and a bladder outlet obstruction, a long NU is less likely than a short PCN tube to fall out inadvertently because it is more firmly anchored.
Placement of a completely internalized ureteral stent in a bloody system will not allow adequate flushing and the stent may clot; therefore, the NU is placed until the urine is clear. Once the urine has cleared, the NU is capped off to external drainage overnight to determine whether the patient can void normally without developing fever, flank pain, or urine leaking around the tube at the skin. If these events occur, the tube should be reopened to exter­nal drainage. An NU tube always should be left to exter­nal drainage whenever the urine is bloody, the patient is febrile, or the urine is infected. If the patient tolerates capping the tube, the NU can be replaced easily by an internal stent using a stiff guidewire for the exchange. If
Specific techniques of stent insertion
The preexisting PCN tube or percutaneous access cathe­ter is removed from the renal pelvis over a stiff guidewire, such as an Amplatz Superstiff (Meditech, Boston, MA) wire, and is replaced fora short directional catheter witha distal curve such as a cobra or hockey stick shape, and the system is opacified with dilute contrast injections. The ureteropelvic junction is engaged with the catheter tip and a floppy-tipped wire (Bentson) or hydrophilic guidewire (Glidewire Meditech, Boston, MA), and the catheter–wire combination is advanced down the ureter to the point of obstruction. With contrast injections and gentle torquing
23
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FIGURE 24-5. Examples of percutaneous urinary drainage catheters: Top: Percutaneous (PCN) tube. The pigtail lies in the renal pelvis and external portion of the tube connects to a drainage bag. Middle: Internal ureteral stent. One pigtail lies in the renal pelvis, and the other pigtail lies in the bladder. The entire tube is inside the patient’s body. Bottom: Nephroureteral (NU) tube. The pigtail at the end of the tube lies in the bladder, the central pigtail lies in the renal pelvis, and the external portion of the tube can be capped off or connected to a drainage bag.
of the catheter, the guidewire usually can be advanced beyond the obstruction without perforating the ureter, even though the ureter appears to be completely oc­cluded. Once the catheter and guidewire are in the blad­der, the initial guidewire is replaced for a stiff guidewire, which will support advancement of the stent. The stiff guidewire must be advanced far into the bladder to allow good “purchase.” If the bladder is not full, this maneuver will be painful to the patient. A Foley catheter, if present, should be clamped. The nephrostomy tract is dilated, and the NU or ureteral stent can be inserted over the wire with the distal pigtail positioned in the bladder and the proxi­mal pigtail positioned in the renal pelvis.
When it is difficult to advance the stent over the stiff guidewire, a few techniques will help “inch” the process along. First, the bladder needs to be full, and as much guidewire as possible needs to be in the bladder. The stent will tend to creep downward when there is countertension on the wire, that is, pulling back the wire as the stent is
being pushed forward. Second, introducing the stent through a “peel-away” sheath usually corrects the difficul­ties resulting from a steep pushing angle and tightly stric­tured distal ureters. The peel-away sheath requires a sepa­rate step, a slightly larger hole in the kidney, and an additional pieceof equipment. Third,the guidewire tip in the bladder sometimes can be steered out the urethra, more easily in women, or when there is a Foley in the bladder. When the wire extends from the flank, through the kidney, down the ureter, and out the bladder, both wire ends can be held taut, thus easing the stent’s ad­vancement down the ureter.
A “safety” wire is also useful to have in position when performing complex manipulations or in less experi­enced hands. This safety wire is simply a second guidewire placed alongside the first wire, known as the working wire. If the working wire is inadvertently removed from the collecting system, access will be preserved by having the safety wire in place.