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Maxillary Anesthesia I: Pulpal Anesthesia
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252
● Needle orientation and course.
⚪ Tilt the tip of the needle inward (the syringe tilts outward),
theoretically forming an angle of around 30–45° relative
to the sagittal or medial plane to keep it flush against the
posterior wall of the maxilla (Figure14.12). The tilt angle
must be wider, up to 70°, in children (Hayden1965).
⚪ Advance the needle upward, inward, and slightly rear-
ward, trying to keep as close as possible to the bone
without tearing the periosteum. That precaution normally (but not always) protects the pterygoid plexus
vessels and the posterior superior alveolar artery from
puncture (DuBrul1988).
⚪ If resistance is encountered (because the needle hits
bone), draw the needle back slightly, narrow the angle
of the inward tilt, and continue.
● Insertion length (Figure14.13).
⚪ Insert the needle 15–20 mm on average (Loetscher
etal.1988; Pfeil etal.2010). Remember that the deeper
the needle, the greater the risk of injuring a vessel and
causing hematomas. In children with mixed dentition,
insert the needle 10–15 mm (Maljaei etal.2017).
⚪ Remember as well that the posterior superior alveolar
nerve has one to four branches (Jones 1939;
McDaniel 1956; Heasman 1984) that in most
individuals lie less than 30 mm from the lower part of
the maxillary tuberosity and only rarely higher (maximum 40
distance the 3–12
mm) (Heasman1984). Subtracting from that
mm between the roof of the buccal
cavity and the gingival margin in molars
(Jorgensen1948) and given that the anesthetic diffuses
there, an insertion 15–20
3–12
mm for the roof, total 18–32 mm) deep should
mm (15–20 mm plus the
ensure an effective block.
● Aspire before injecting. Aspiration is positive in 5% of
cases (Annex 22).
● Inject 1.8 ml (one full cartridge) slowly, over the course
of 40–60
seconds. Injecting two cartridges (3.6 ml)
yields better results and increases the duration of pulpal anesthesia (Pfeil etal.2010). Inject 1
ml in children
with mixed dentition (Hayden 1965: Maljaei
etal.2017).
● Subjective symptoms of anesthesia. Soft tissue anesthe-
sia, defined by patients as dullness, numbing, tingling,
tickling or “fattening” sensation in the lip (Table13.1,
Chapter13), appears after 2–3
⚪ Tests with a 15–20- minute wait (Loetscher etal.1988;
minutes.
Pfeil etal.2010) report better results than those with a
5- minute wait (Padhye et al. 2011). A compromise
might be a waiting period of 5–10
minutes after injec-
tion to enable the anesthetic solution to take full effect.
⚪ Optionally some clinicians insert a probe in the buccal
mucosa around the molars to determine soft tissue
anesthesia.
⚪ The definitive sign of efficacy is obtained by working
on the molars (i.e. prepping into dentin), initially cautiously and then proceeding normally if the patient
expresses no discomfort.
● Soft tissue anesthesia usually lasts 3–4 hours (200 min-
utes), a fact of which the patient should be notified after
treatment.
Figure14.13 Needle height necessary to block the posterior
superior alveolar nerve.
Efficacy ofthis Technique
When assessed with an electrical pulp tester or dry ice (at
−78°C), pulpal anesthesia is successful in 90% of maxillary
molars, but in only 25–50% of premolars (Table 14.3).
Similar results (around 90%) are reported for children with
mixed dentition for the first permanent and second primary molars, although the success rate drops to 65% in the
first primary molar (Hayden1965) (assessed with the less
rigorous extraction method). As the preceding values are
referred to the standard local anesthetic, 2% lidocaine,
1:100 000 (10 μg/ml) epinephrine (L- 100), use of a more
powerful solution such as articaine (A- 100) would be
expected to improve the results.
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High Tuberosity Approach 253
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Table14.3 Successful (%) pulpal anesthesia withposterior
superior alveolar nerve block (−78
tester assessment andstandard solution L-
Variable
Tooth Second molar (%) 96 97 100
First molar (%) 88 77 84
Second premolar (%) 31 45 68
First premolar (%) 10 10 32
Amount injected (ml) 1.2 1.8 3.6
Stimulus Cold
L- 100 → 2% lidocaine + 1:100 000 (10 μg/ml) epinephrine.
°C dry ice or electrical pulp
Loetscher
etal. (1988)
n=45–56
°C)
(−78
100)
Pfeil
(2010)
n=31
Electrical Electrical
Pfeil
(2010)
n=31
Complications Specific tothis Technique
1) Extraoral hematomas may appear on the face in less
than 0.5% of cases (further to the Kuster and Udin1984;
Loetscher etal.1988; Pfeil etal.2010; Padhye etal.2011
series). They occur when the needle pierces a vein in the
pterygoid plexus or the buccogingival branches of the
posterior superior alveolar artery that course somewhat
erratically near the maxillary tuberosity (Harn
etal.2002; Padhye et al.2011). They may present with
swelling in the malar or superior masseteric region.
Swelling appears earlier if an artery rather than a venous
plexus vein is injured. In both cases the symptom is a
dark brown spot on the skin due to blood extravasation
(ecchymosis) that in the next few days spreads forward
and downward along the muscular planes of the cheek,
while changing to a purplepainless, causing no discomfort through the 10–15
yellowish hue. The process is
days
it takes to reabsorb the spot.
2) Acute trismus, with the inability to open the mouth par-
tially or wholly, normally a painful process sensitive to
exploration at the insertion site. The cause is needle
penetration in the lateral (external) pterygoid muscle,
inducing irritation and spasms (Kramer and
Mitton 1973; Stone and Kaban 1979). This complication, which may appear in less than 0.5% of cases (further to the Kuster and Udin1984; Loetscher etal.1988;
Pfeil etal.2010; Padhye etal.2011 series), can be treated
as described in Chapter22.
● It is identical to buccal infiltration at the second maxillary
molar behind the zygomatic osseous crest of the malar
bone except that the needle runs a distance of around
10
mm instead of 2–3 mm to pass over the buccinator
muscle insertion.
● A 1.8- ml cartridge is injected, forming a sort of subcuta-
neous blister immediately over the buccinator muscle in
the molar buccal cavity. The blister should be pushed
backward, upward, and inward with the fingertip. This
operation is easier if the patient’s mouth is completely
closed (Figure14.5).
This procedure facilitates diffusion of the anesthetic
solution toward the molar apices as well as toward the
upper posterior part of the maxilla, i.e. toward the posterior
superior alveolar nerve branches before they course into
the foramina in the tuberosity.
The success rate, assessed in extractions only (rather
than with the more rigorous electrical pulp tester), comes
to nearly 100% in molars and nearly 90% in premolars with
the standard lidocaine solution (L-
80) (Adatia1976).
High Tuberosity Approach
This technique aims to block the maxillary nerve (V2), the
second division of the trigeminal nerve, within the pterygopalatine fossa, numbing a wide area by anesthetizing all
its branches at once.
It is known as high tuberosity because the tuberosity
approach (without “high”) is a synonym for PSA nerve
block. Here the needle must travel further (higher) into the
pterygopalatine fossa, hence the name. Other synonyms
for this approach are posterior infraorbital approach
because it is like infraorbital block but posterior, or Smith’s
technique because it was developed by Dr. Arthur Ervin
Smith of Chicago around 1913 (Smith1920).
This is a difficult technique because it is highly arbitrary
because of the lack of osseous references to guide the needle during injection. It calls for introducing the needle
higher and at a wider angle to avoid the posterior wall of
the maxilla (tuberosity) and reach the pterygopalatine
fossa (Figure14.1). That raises the risk of hematomas due
to puncture of pterygoid plexus veins or branches of the
posterior superior alveolar artery. It is also one of the most
painful techniques (Annex 23).
Modified Adatia Technique
In 1968, Adatia (1968) introduced a simple method that
prevents the risk of hematomas and swelling
(Adatia1974,1976).
Uses
As noted earlier, the most common technique in the upper
arch is buccal infiltration anesthesia, although on occasion
a more powerful anesthetic effect is required, as in the following examples.
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1) Lengthy oral surgery in the maxillary zone because the
high tuberosity approach covers a larger area with less
anesthetic and fewer injections.
2) After buccal infiltration failure, particularly in the
molar zone.
Zone Anesthetized
Theoretically the technique would affect the entire area
innervated by the second division of the trigeminal nerve,
the maxillary nerve (V
● Teeth, including pulp and periodontium of molars and pre-
), including the following.
2
molars on the side injected. Whilst incisors and canines may
be anesthetized, as they are contralaterally cross-
innervated,
they may require reinforcement via buccal infiltration.
● Buccal cavity, including fibromucosa (alveolar mucosa,
gingiva, and interdental papillae), bone, and periosteum
around molars and premolars. As discussed earlier, the
anterior teeth are often contralaterally cross-
innervated.
On occasion the upper area in the roof of the buccal cavity at the first maxillary molar may require supplementary infiltration, as it may be innervated by a branch of
the ophthalmic nerve (first division of the trigeminal
nerve) (Saborido1977).
● Palate, including the fibromucosa, bone, and periosteum
of the entire hemicontralaterally cross-
arch except the anterior part, which is
innervated by the nasopalatine
nerve (Saborido1977).
● Maxillary sinus on the side injected.
● Terminal branches of the infraorbital nerve that inner-
vate the upper half-
lip, the nasi ala, and the lower eyelid
on the side injected.
Technique
● Long (42 mm), 27G or (preferably) 25G needle. In
patients with small mouths or where a high angle is
needed (see below), the needle should be bent at a 30–45°
angle several millimeters ahead of the cone or hub to
facilitate needle progress along the posterior wall of the
maxilla. Note that the needle is not bent at the hub itself
because as this is the weakest part it might readily break.
● Dentist’s and patient’s positions.
⚪ Dentist at 9:00–10:00 o’clock.
⚪ Patient in supine position with head slightly hyperex-
tended to enable the dentist to turn it slightly to the left
to anesthetize the right side or to the right to anesthetize the left side.
● Advise the patient that during injection they may experi-
ence (i) heart palpitations (up to 30% of individuals in
some series; Forloine etal.2010) due to the use of epinephrine solutions and (ii) temporary ocular and visual
alterations (up to 10–15% of individuals) (Table14.4).
● Determine the depth of the needle (Jorgensen 1948;
Hayden1965).
⚪ The height of the posterior maxilla or distance the nee-
dle should travel may vary from 30 to 50
mm
(Jorgensen 1948), and although the normal range is
30–40 mm with a mean of around 35 mm (Broering
etal. 2009; Forloine etal. 2010), the distance must be
individually estimated. One fairly reliable procedure is
to measure the height of the anterior maxilla from the
first maxillary premolar gingival margin to the edge of
the inferior orbit because in 90% of individuals this is
practically the same distance as the height of the posterior maxilla. The latter is 4–6 mm higher in only 5% of
Table14.4 Successful (%) pulpal anesthesia after thehigh tuberosity approach (electrical pulp tester assessment and3.6 ml
ofstandard solution L- 100): percentage ofocular complications andlower lip anesthesia
Variable
Tooth Second molar (%) 100 100 98
Lower lip anesthesia (%) — 2 32
Ocular complications Diplopia (%) — 10 12–16
Broering etal. (1991)
n=40
First molar (%) 78 95 92
Second premolar (%) 52 73 76
First premolar (%) 30 52 58
Canine (%) 22 32 54
Lateral incisor (%) 4 18 8
Central incisor (%) 4 5 10
Blurred vision (%) — 5 0
Mydriasis (%) — 2 0
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Broering etal. (2009)
n=40
Forloine etal. (2010)
n=50

High Tuberosity Approach 255
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individuals, in which case the needle may not be inserted
high enough, and 1–3
mm lower in the other 5%, leading
to an overly high insertion site (Jorgensen1948). Only in
the presence of open anterior bite is the height of the
posterior maxilla 2–9
mm lower than the anterior (90%
of individuals affected), raising the risk of overestimating the desired height (Jorgensen1948).
⚪ The height of the anterior maxilla is measured along
the face with a dental caliper from the first premolar
gingival margin to immediately below the infraorbital
bone margin, taking care not to accidentally injure the
eye and marking the distance with a rubber stop on the
needle to prevent over-
● Examine the entire roof of the buccal cavity over the maxil-
lary molars with the non-
insertion (Hayden1965).
injection index finger to locate
the osseous bridge that protrudes from the zygomatic
crest on the malar bone (normally alongside the first
maxillary molar).
● Pull the labial commissure outward and upward with the
non-
injection hand to firmly stretch the jugal mucosa
(Figure4.12).
⚪ Stretch the tissue intraorally with the index finger on
the right side and the thumb on the left.
⚪ The aim is to (i) bring the concavity of the buccal cav-
ity around the molars into view and (ii) reduce any
injection- induced pain by inserting the needle in a
tense mucosa.
● Increase the space for maneuvering in the mouth.
⚪ Ask the patient to partially close their mouth to enlarge
the workspace, which (i) broadens the labial commissure and (ii) shifts the coronoid apophysis of the mandible rearward to prevent it from taking up space and
getting in the way (Figure14.5).
⚪ If despite the foregoing the space is insufficient, ask
the patient to shift their mandible toward the side to be
injected to increase the separation between the ascending ramus of the mandible and the posterior maxilla
(Sicher1950).
● Insert the needle in the roof of the buccal cavity.
⚪ Behind the zygomatic crest of the malar bone, which is
usually the same as saying behind the second or
third molar.
⚪ Point the needle upward and as perpendicularly as
possible to the molar occlusal plane. As a full 90° angle
is not possible due to the presence of the jugal mucosa,
the needle should be rearwardly oblique, which is also
desirable.
⚪ Avoid the osseous wall to prevent the needle from hit-
ting or catching in the surface of the maxillary tuberosity (outer and posterior wall of the maxilla).
⚪ Push the needle a few millimeters into the buc-
cal cavity.
● Needle re- orientation and course.
⚪ Tilt the tip of the needle inward (the syringe tilts out-
ward), theoretically forming an angle of around 30–45°
relative to the sagittal or medial plane to keep it flush
against the posterior wall of the maxilla (Figure14.12).
⚪ Push the needle upward, inward, and slightly rearward,
trying to keep as close as possible to the bone without
tearing the periosteum. That protects the pterygoid
plexus veins, the posterior superior alveolar artery,
Bichat’s fat pads, and the lateral (external) pterygoid
muscle from puncture. Note that it is not always possible to avoid impacting the vessels in this zone
(Sicher1950).
⚪ If resistance is met (because the needle hits bone),
withdraw the needle slightly, re-
enter at a less inward
angle, and continue.
⚪ Needle progress is interrupted when the rubber stop
(used to mark the insertion length) reaches the second
and third molar CEJ or gingival margin. Remember
that the mucosa at the roof of the buccal cavity is not a
good reference because its depth may vary from 3 to
12 mm (Jorgensen1948).
● Aspire before injecting. Aspiration is positive in 13% of
cases (Goldman and Gray1963).
● Inject the full 1.8- ml cartridge over the course of
40–60
seconds, after which the entire operation is
repeated to inject a second cartridge, for a total of 3.6
ml.
Many authors deem a single cartridge to be insufficient
because as the tip of the needle does not reach but merely
comes close to the pterygopalatine fossa (where the maxillary nerve lies), the anesthetic solution must diffuse
across a longer distance to bathe the nerve (Smith1920;
Collon1946; Jorgensen1948; Forloine etal.2010).
● Subjective symptoms of soft tissue anesthesia appear in
3–5
minutes with patients describing tickling, tingling,
or numbing (Table13.1, Chapter13) at the injection site
and (i) the lower eyelid, nasi ala and half of the upper lip
and (ii) the half of the palate on the side injected.
⚪ Wait 5–10 minutes after injection to allow the anesthetic
to take effect and anesthetize the pulp of maxillary
molars and premolars. Note that this approach takes a
little more time.
⚪ Optionally some clinicians insert a probe in the buccal
mucosa around the molars to determine soft tissue
anesthesia.
⚪ The definitive sign of efficacy is obtained by working
on the molars (i.e. prepping into dentin), initially cautiously and then proceeding normally if the patient
expresses no discomfort.
● Soft tissue anesthesia usually lasts 3–4 hours (200 minutes),
a fact which the patient should be notified of after
treatment.
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Efficacy ofthis Technique
As Table 14.4 shows, according to several clinical tests
pulpal anesthesia is successful in nearly 95% of molars, but
just 60% of premolars. The success rate drops even lower in
the anterior teeth. Surprisingly, although this is an upper
arch approach, it anesthetizes the lower lip in a small percentage of cases. As these findings are referred to a standard 2% lidocaine, 1:100 000 (10 μg/ml) epinephrine solution
(L-
100), a more powerful solution such as 4% articaine,
1:100
000 (10 μg/ml) epinephrine (A- 100) would be
expected to deliver somewhat better results.
Complications Specific tothis Technique
1) Anesthetic failure is fairly common due to the difficulty
and arbitrary nature of this approach.
⚪ If the needle is not close enough to the pterygopalatine
fossa, rather than the entire maxillary nerve (V
), its
2
posterior superior alveolar nerve branches are blocked,
anesthetizing the molars only.
⚪ If the solution is injected too outwardly, in the
infratemporal fossa (formerly the zygomatic fossa), it
is too far away from the pterygopalatine fossa to anesthetize the whole nerve. This can be corrected by tilting the needle more medially, closer to the posterior
side of the maxilla (Collon1946).
⚪ If only one instead of two 1.8- ml cartridges (3.6 ml) is
injected, the amount of the solution diffusing is insufficient to anesthetize the maxillary nerve (V
2
(Collon1946).
2) An extraoral hematoma on the face is the most common
complication. This dark-
brown purplish, normally swollen spot in the malar or superior masseteric area is generally painless. Its cause is blood extravasation
(ecchymosis) due to puncture of a vein in the pterygoid
plexus or the buccogingival branches of the posterior
superior alveolar artery that course somewhat erratically near the maxillary tuberosity (Harn etal.2002). In
either case in the next few days the lesion spreads forward and downward along the muscular planes of the
cheek, while changing to a purple- yellowish hue. It is
wholly reabsorbed in 10–15 days.
3) Ocular and visual alterations in the eye on the side
injected appear in 10–15% of cases. The most common
symptom is double vision (dipoplia), although others
such as blurred vision, dilated pupil (mydriasis), and
drooping eyelid (ptosis) may also appear. When the
anesthetic solution is deposited too high in the pterygopalatine fossa (rather uncommon in this approach), the
solution diffuses across the inferior orbital fissure to the
interior of the orbit, anesthetizing ocular nerves, and
muscles with the concomitant loss of motor coordination (Collon 1946; Forloine et al. 2010). In such situations, (i) reassure the patient that the effect will disappear
spontaneously with the effect of the anesthesia, (ii) protect the eye with a patch until the effect subsides, and
(iii) advise the patient that they may neither drive nor
operate hazardous machinery while the effect lasts.
4) Acute trismus, with the inability to open the mouth par-
tially or wholly, is normally a painful process sensitive
to exploration at the insertion site. The cause is needle
penetration in the lateral (external) pterygoid muscle,
inducing irritation and spasms (Kramer and
Mitton1973; Stone and Kaban 1979). Permanent trismus may appear more rarely (Stone and Kaban 1979)
(see Chapter22).
5) Pain is inherent in this technique, one of the most
painful.
● Needle progression may induce moderate to severe
pain in 50% of patients (Annex 23).
● Moderate to severe post- operative pain around the
injection site may last 1–3
days in 25% of individuals
(Broering etal.2009; Forloine etal.2010), subsiding
spontaneously with no sequelae.
Remarks
The high tuberosity approach is not recommended for routine dentistry practice because it is difficult, entails more
complications, and delivers results only marginally better
)
than simple buccal infiltration anesthesia. The most important factors for success with this approach are as follows:
1) The tip of the needle must be placed at the right height.
2) The tip of the needle must be oriented at the angle that
brings it closest to the pterygopalatine fossa.
3) Two 1.8- ml (3.6- ml) cartridges must be injected to pro-
vide sufficient volume for the solution to diffuse to the
pterygopalatine fossa and reach the maxillary nerve (V
Transpalatal Technique
As in the high tuberosity approach, this technique aims to
block the maxillary nerve (V
trigeminal nerve, within the pterygopalatine fossa, rendering
a wide area numb by anesthetizing all its branches at once.
It is also known as the greater palatine canal technique.
The term “transpalatal,” coined by Dr. Gerardo Saborido
(1977), seems to be much more illustrative of what the technique actually involves. It was first described in 1921 by Juan
Ubaldo Carrea of Buenos Aires, who recommended it in
molars and premolars (Carrea1921a,b), and subsequently
), the second division of the
2
).
2
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applied in 1922 by Otto Hofer of Vienna (Hofer 1922b). It
was also described in 1922 by Mendel Nevin of New York,
who claimed to have developed it in 1917, but did not recommend it due to the high frequency of obstructions in the
canal and the risk of injury to the vascular nervous package
(Nevin 1922). The following year Samuel Silverman of
Atlanta (Silverman 1923), apparently unaware of Carrea’s
work, described it as his own.
Note: In the 1921 paper the surname “Carrea” is mis-
spelled as “Carrba.”
In this relatively simple technique (an enormous advantage over the high tuberosity approach) the needle penetrates the greater palatine foramen through the greater
palatine canal to reach the pterygopalatine fossa, where the
second division of the trigeminal nerve (maxillary nerve,
) transmits before entering the infraorbital canal.
V
2
Uses
As noted earlier, buccal infiltration is the most common
technique to anesthetize the upper arch, although on occasion a more powerful anesthetic is required, such as in the
following examples.
1) Lengthy oral surgery in the maxillary zone, as the
transpalatal approach covers a larger area with less
anesthetic and fewer injections.
2) After buccal infiltration failure, particularly in the
molar zone.
3) In acute buccal cavity infection around molars and pre-
molars. Under such circumstances the buccal infiltration, superior alveolar nerve block, and high tuberosity
procedures are contraindicated since the needle must
be inserted in this area (Szerlip and Morristown1950;
Sved etal.1992). Today, in 95% of the cases where the
transpalatal technique is chosen it is for this reason
(Sved etal.1992).
Zone Anesthetized
Theoretically the technique would affect the entire area
innervated by the second division of the trigeminal nerve,
the maxillary nerve (V
● Teeth, including pulp and periodontium of the molars
).
2
and premolars on the side injected. Whilst incisors and
canines may be anesthetized, as they are contralaterally
cross- innervated, they may require reinforcement via
buccal infiltration.
● Buccal cavity, including fibromucosa (alveolar mucosa,
gingiva, and interdental papillae), bone, and periosteum
around molars and premolars. As discussed earlier, the
anterior teeth are often contralaterally cross- innervated.
● On occasion the upper area in the roof of the buccal cav-
ity at the first maxillary molar may require supplementary infiltration, as it may be innervated by a branch of
the ophthalmic nerve (first division of the trigeminal
nerve) (Saborido1977).
● Palate, including the fibromucosa, bone, and periosteum
of the entire hemicontralaterally cross-
arch except the anterior part, which is
innervated by the nasopalatine
nerve (Saborido1977).
● Maxillary sinus on the side injected.
● Terminal branches of the infraorbital nerve that inner-
vate the upper half-
lip, the nasi ala, and the lower eyelid
on the side injected.
Technique
● Very long (42 mm), 27G needle (preferred over 25G as it is
more flexible and less prone to jamming as it progresses;
Cohn1986). In patients with small mouths or where a
sharp angle is needed (see below), the needle should be
bent at a 30–45° angle, several millimeters ahead of the
cone or hub to facilitate needle progress along the greater
palatine canal (Cook1950b; Mercuri1979; Lepere1993;
Douglas and Wormald2006). Note that the needle is not
bent at the hub itself because as this is the weakest part it
could break.
● Dentist’s and patient’s positions.
⚪ Dentist at 9:00–10:00 o’clock.
⚪ Patient in supine position with head slightly hyperex-
tended to enable the dentist to turn it slightly to the left
to anesthetize the right side or to the right to anesthetize the left side.
● Anesthetize the greater palatine nerve where it exits
from the greater palatine foramen and wait 2
minutes
before continuing (Chapter 15). This measure aims to
minimize possible discomfort during the rest of the
procedure.
● Advise the patient that during the injection they may feel
(i) pressure below the maxillary facial tissues on the side
injected and (ii) temporary ocular and visual alterations
such as double vision (diplopia), drooping eyelid (ptosis),
or blurred vision (10% of cases) (Table14.5).
● Determine the depth (length) of the insertion.
⚪ In dry skulls, the mean length along the greater palatine
canal between the greater palatine foramen and the roof
of the pterygopalatine fossa is around 35 mm, normally
ranging from 20 to 45 mm (Table A 42.6, Annex 42). As
the 4–6 mm (3–7 mm) thickness of the fibromucosa
over the greater palatine foramen (Table14.6) must be
added to that dimension, and take away 1–2 mm of soft
tissues overlaying the alveolar crest in bicuspids
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Table14.5 Percentage ofvisual/ocular alterations andtheir
relationship toneedle length andamount (ml) ofsolution
injected withthe transpalatal technique
Percentage of
Sample
Reference
Dickson and
Coates (1945)
Malamed and
Trieger (1983)
Sved etal. (1992) 101 39 42 4.4
Schwartz- Arad
etal. (2004)
Broering etal.
(2009)
Torres etal. (2011) 82 7 41 1.2–1.8
Mean 11, 8
Rounded mean 10
a
Computer- controlled device (the wand).
Table14.6 Depth ofsoft tissue overlaying thegrater palatine
foramen
Reference Sample Depth (mm)
Dickson and Coates (1945) Patients 3–4
Cook (1950b) Patients 4–7
Canter etal. (1964) Patients 2–7
Viegas and Hemphill (1961) Patients 2–5
Malamed and Trieger (1983) Patients 3–4
Methathrathip etal. (2005) Cadavers 6.7 ± 2.3 (2–13)
Douglas and Wormald (2006) Cadavers 6.9
Nimigean etal. (2013) Patients 6 (4–8)
Shalaby etal. (2015) Cadavers 4, 9 ± 1, 9
a
size
80 6 42 2.0
150 1 35 1.8
76 0 30 3, 2
40 18 —
visual/ocular
alterations (%)
Rounded mean 4–6
Needle
length
(mm)
3.6
Mean 4.7
Amount
injected
(ml)
(Malamed and Trieger 1983), the total may come to
around 37–39 mm on average. Clinical trials yield lower
heights, however, around 33 mm (29–35 mm), because
the needle need not reach the roof of the pterygopalatine fossa (Broering etal.2009).
⚪ Two methods for a more personalized estimate, par-
ticularly for children or adults with very small or large
faces, are in place and closely correlated to the posterior part of the maxilla.
1) Measurement of the low anterior height. This con-
sists of measuring the distance between the infraorbital foramen and the alveolar crest of the second
maxillary premolar.The mean is 30–35
mm (Table
A 2.5, Annex 2).
2) Measurement of the high anterior or orbit height.
This consists of measuring the distance between
the upper rim of the orbit and the lower rim at the
infraorbital foramen. The mean is 33–34
mm
(Canter etal.1964; Slavkin etal.1966).
⚪ The measurement as estimated with one method or
the other should be increased by 5
mm to accommodate the fibromucosa over the greater palatine foramen. A stop is placed on the needle to prevent
over-
insertion (silicone or rubber stop) (Figure14.14).
● Ask the patient to open their mouth as widely as possible
for full vision and to insert the needle in the greater palatine canal. A mouthmay be optionally placed on the non-
opener (bite block or mouth prop)
injection side
(Mercuri1979; Malamed and Trieger1983).
● Locate the greater palatine foramen.
⚪ This formation lies on the horizontal hard palate at the
abutment with the vertical alveolar process: in nearly
90% of individuals it is located around the third maxillary molar (Annex 42) (Figure14.15). The third molar
zone refers to the area ranging from a point slightly
forward of the third molar (between it and the second
molar) to a point slightly backward of the third molar
and including the third molar zone.
⚪ It also helps to know that it lies around 15 mm, later-
ally, from the midline of the palate (Szerlip and
Morristown1950; Annex 42).
⚪ It is around 4 mm forward of the posterior edge of the
hard palate (Annex 42). This is important when the
maxillary arch is edentulous (Mercuri1979; Wong and
Sved1991). One way of distinguishing the hard and soft
palate is by locating the line where the color of the
palatal mucosa changes.
● Insert the needle in the greater palatine foramen.
⚪ The approach is from the opposite side of the mouth to
more readily locate and insert the needle into the
greater palatine canal (Figure14.14).
⚪ Insert the needle slightly forward of where the fora-
men is presumed to be (Figure14.15). Remember that
the foramen is covered by around 4–6 mm of fibromucosa (Cook etal.1950b) (Table14.6).
⚪ If the needle does not go in after the first puncture,
repeat the operation several times until the foramen is
reached (Hofer 1922b; Peckham 1938; Wong and
Sved1991). Contrary to what some authors contend,
the foramen is not readily found by touch and must be
located by trial and error (Wong and Sved1991). It is
nearly impossible to detect the foramen if the diameter
is too small (it varies from 3 to 4 mm; Annex 42) and/
or the fibromucosa is thick.
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Transpalatal Technique 259
op
e
ea
Foramen
Occlusal
Pt
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Table14.7 Angulation toocclusal plane andmid- sagittal
plane (°) inthe transpalatal technique
Silicone st
Figure14.14 Silicone stop or rubber stop to mark the needle
insertion limit; maneuver facilitated by non- injection side
approach.
Reference Sample
Angle
occlusal
plane (°)
Angle
sagittal
plane (°)
Carrea (1921a) Patients — 5–10
Dickson and Coates
Patients 45 5–10
(1945)
Cook (1950b) Patients 60–70
Ries Centeno (1979) Dry skulls 60 —
Malamed and Trieger
Dry skulls 46 (20–70) —
(1983)
Chentanez etal.
Dry skulls 60 ± 9 —
(1985)
Austin (1987) Dry skulls 60 ± 8
(51–76)
7 ± 5 (−3 to
20)
Wong and Sved (1991) Patients 60 5–8
Methathrathip etal.
Dry skulls 58 ± 6 7 ± 5
(2005)
Douglas and Wormald
Cadavers 60 —
(2006)
Howard- Swirzinski
CBCT 63 —
etal. (2010)
Shalaby etal. (2015) Dry skulls 50 —
Mean 57.7 7.1
Rounded mean 50–60 5–10
Boundary between
horizontal hard palat
and vertical alveolar
process
Needle insertion ar
Greater palatine
foramen
Figure14.15 Location of greater palatine foramen and needle
insertion area.
● Push the needle slowly and gently across the greater pal-
atine canal.
⚪ Direct the needle rearward and upward at an angle of
rotundum
erygopalatine
fossa
Greater
palatine canal
3rd molar
Infraorbital
canal
Greater
palatine
foramen
plane
60°
Figure14.16 Needle angle relative to the occlusal plane of the
maxillary molars. Schematic representation in saggital section.
around 50–60° relative to the occlusal plane of the
upper molars (Table 14.7 and Figure 14.16) (sagittal
section: antero- posterior direction). It is frequent that
the needle has to be bent in the shaft but not in the
hub, the weakest part.
⚪ Needle movement relative to the medial plane should
be 5–10° outward (Table 14.7 and Figure 14.17)
(coronal section: medial- lateral direction).
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Maxillary Anesthesia I: Pulpal Anesthesia
Medial plane
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260
● Subjective symptoms of anesthesia appear in 3–5 min-
erygopalatine
fossa
Nasal cavity
utes, with patients describing soft tissue anesthesia as
tickling, tingling, or numbing (Table13.1, Chapter13) at
the injection site and (i) the lower eyelid, nasi ala and
Maxillary sinus
Greater
palatine canal
Greater
palatine
foramen
5–10°
half of the upper lip; and (ii) the half of the palate on the
side injected.
⚪ Wait 5–10 minutes after injection to allow the anes-
thetic to take effect and anesthetize the pulp of maxillary molars and premolars. Note that this approach
takes a little more time.
⚪ Optionally some clinicians insert a probe in the buccal
mucosa around the molars to determine soft tissue
anesthesia.
⚪ The definitive sign of efficacy is obtained by working
on the molars, initially cautiously and then proceeding
Figure14.17 Needle angle relative to the medial plane
(coronal section).
normally if the patient expresses no discomfort.
● Soft tissue anesthesia usually lasts 2–3 hours (Sved
et al. 1992; Lepere 1993), a fact of which the patient
⚪ Never force the needle. Any resistance is an indication
should be notified after treatment.
that it is not running parallel to the canal walls. This
minor angle deviation can be corrected by drawing the
needle back by 1–3
mm to free it of any contact with
the bone and readjusting the orientation with minor
movements (Peckham1938; Dickson and Coates1945;
Saborido1977; Malamed and Trieger1983; Wong and
Sved 1991). Note that the scant space available in a
greater palatine canal with a narrow diameter (1.5 mm)
severely limits any possible redirection of the needle
(Cook 1950b). This circumstance is fortunately not
very common.
⚪ The clinical impression is that maneuvering inside the
canal causes patients little discomfort (Silverman1923;
Malamed and Trieger 1983) and clinical tests show
Efficacy ofthis Technique
As Table 14.8 shows, according to several clinical tests
pulpal anesthesia is successful in nearly 95% of molars,
but just 65% of premolars. The success rate drops even
lower in the anterior teeth. Surprisingly, although this is
an upper arch approach, it anesthetizes the lower lip in a
small percentage of cases. The clinical success rate in maxillary molars and premolars determined via extractions,
oral surgery, and dental treatment (less rigorous method)
is in the order of 80% (Table14.9). As these findings are
referred to a standard 2% lidocaine, 1:100 000 (10 μg/ml)
epinephrine solution (L-
100), a more powerful anesthetic
that this technique is less painful than the tuberosity
approach (Annex 23).
⚪ Advance the needle until the rubber stop used to mark
the insertion depth is reached. Allow at least 2–3 mm
of the needle to protrude outside the mucosa over the
greater palatine foramen so it can be withdrawn in the
event of breakage (Dickson and Coates 1945;
Mercuri1979; Wong and Sved1991).
● Aspire before injecting. Aspiration is positive in 5% of
Table14.8 Successful (%) pulpal anesthesia withthe
transpalatal technique (electrical pulp tester assessment
and3.6 ml ofstandard solution L- 100) andpercentage oflower
lip anesthesia
Variable
Broering etal.
(1991)
n=40
Broering etal.
(2009)
n=40
cases (Annex 22).
● Slowly inject a 1.8- ml cartridge of solution over the
course of 40–60 seconds and then a second cartridge for
a total of 3.6 ml. Some authors deem 1.8 ml to be too little
and therefore recommend two (Wong and Sved 1991;
Sved etal. 1992). Either repeat the entire operation or,
where the patient is highly cooperative, unscrew the needle from the syringe while still in the mouth, load
another cartridge and re- screw the syringe onto the needle for the second injection (Wong and Sved1991).
Tooth Second molar (%) 96 100
First molar (%) 83 95
Second premolar (%) 65 80
First premolar (%) 35 68
Canine (%) 26 60
Lateral incisor (%) 17 43
Central incisor (%) 4 23
Lower lip anesthesia (%) — 12
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Transpalatal Technique 261
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Table14.9 Clinical successful (%) inmolars andpremolars
withthe transpalatal technique
Reference Sample size Success rate (%)
Dickson and Coates (1945) 80 80
Corbett and Helmore (1948) —
Malamed and Trieger (1983) 150 90
Sved etal. (1992) 101 89
SchwartzTorres etal. (2011) 82 60
Arad etal. (2002) 66 91
78
Mean 81.3
Rounded mean 80
such as 4% articaine, 1:100 000 (10 μg/ml) epinephrine
100) would be expected to deliver somewhat better
(Aresults.
Complications Specific toThis Technique
1) Full or partial anesthetic failure for the following
reasons.
● The greater palatine foramen cannot be located
(Mercuri1979; Schwartz-
Arad etal.2004), especially
in edentulous patients. In such cases, review the anatomy (Mercuri1979).
● The needle continuously catches on the posterior wall
of the canal, a common occurrence in canals highly
angled relative to the occlusal plane. Bending the
needle is very useful in such cases, as noted earlier
(Mercuri1979; Cohn1986).
● The needle is not inserted far enough and the tip fails
to reach the pterygopalatine fossa, so the gravitydriven anesthetic solution flows downward (Wong
and Sved1991). In such cases, repeat the technique
with a longer needle (Mercuri1979).
● The amount of anesthetic administered is insuffi-
cient. The solution is to inject another cartridge
(Wong and Sved1991).
2) Anatomical variations may induce failure.
● Canal obstructed because it is contorted or irregular,
with osseous protrusions that hinder passage of the
needle, a situation found in 5% of individuals
(Table14.10). In such cases desist and seek alternative techniques such as the high tuberosity approach
(Dickson and Coates1945; Mercuri1979; Cohn1986;
Wong and Sved1991). Nonetheless, when the obstacle is less than 15
mm from the total length envisaged
the injection may often be successful (Sved etal.1992).
● Excessive rearward slant on the greater palatine canal
at a very small angle relative to the occlusal plane of
the maxillary molars. This anatomic variation may
result in the dose being delivered anterior to the
pterygoid process, preventing the needle from reaching the pterygopalatine fossa (Cook1950b). This circumstance is uncommon.
● Excessive lateral slant on the greater palatine canal,
which empties into the lateral side the pterygoid process without reaching the pterygopalatine fossa
(Cook1950b). This circumstance is also uncommon.
Table14.10 Percentage ofobstructed greater palatine canal indry skulls andclinical trials
Reference n Obstructed (%) Reference n Obstructed (%)
Canter etal. (1964) 205 39 Dickson and Coates (1945) 80 2.5
Slavkin etal. (1966) 58 37 Mercuri (1979) — 5
Jorgensen and Hayden (1970) 200 15 Cohn (1986) —
Ries Centeno (1979) 50 6 Sved etal. (1992) 101 6
Malamed and Trieger (1983) 204 3 Schwartz- Arad etal. (2002 and2004) 66–76 0
Chentanez etal. (1985) 120 54 Broering etal. (2009) 40 8
Austin (1987) 42 5
Ferreira etal. (1990) 100 10
Sharma and Garud (2013) 100 4
The values used are from clinical trials; dry skull values are not representative of clinical reality.
Dry skulls Clinical trials
10
Mean 19.2 5.2
Rounded mean 20 5
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