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Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3676_Библиотеки_им_академика_М_И_Перельмана

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C. Brown and C.S. White
a
b
Fig. 11.26 (a) Sagittal. Widespread metastatic bone dis- ease from breast carcinoma (arrowheads). Status post ver­tebroplasty with injection of methylmethacrylate into a vertebral body with a small amount of leakage of the cement into the disc space (long arrows). (b) Sagittal. B-cell lymphoma involving the thoracic spine (arrows).
(c) Sagittal. Widespread bony blastic metastases from prostate carcinoma (arrowheads). Status post CABG. (d) Sagittal. Expansile lytic lesion in the lower sternum (arrows) incidentally found in a 91-year-old female with intractable chest pain. Diagnosis: myeloma—Contributed J. Lee and C. Smuclovisky
11 Extracardiac Findings on Cardiac CTA
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11.25 Case 11.24 Contributed by Lee and C. Smuclovisky
J.
11.25.1 Extracoronary Disease
During the interpretation of cardiac CT, extra­coronary findings are frequently identified.
a
It is important to evaluate the entire anatomy in the field of view in order not to miss a clinically relevant finding (Fig. 11.27a–h).
b
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Fig. 11.27 (a) Axial. Left renal upper pole carcinoma (arrows). (b) Axial. Gastric carcinoma with ulceration of the tumor (long arrow) and adjacent adenopathy (arrow- heads). (c and d) Axial and coronal maximum intensity projection. PFO closure device (arrow). (e and f) Coronal and axial. Foreign body in the right pulmonary artery
from a wire lost during placement of an ICD device (arrows). ICD wires in the SVC (f, long arrow). Status post CABG. (g) Sluggish filling in the LA appendage; questionable for clot (arrow). (h) Immediate delay acqui­sition demonstrates complete filling with no evidence of thrombus (arrow)
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C. Brown and C.S. White
ef
g
Fig. 11.27 (continued)
h
11 Extracardiac Findings on Cardiac CTA
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References
1. Hunold P, Schmermund A, Seibel RM, Gronemeyer
DH, Erbel R. of accidental findings in electron beam tomographic scans for coronary artery calcification. Eur Heart J. 2001;22:1748–58.
2. Macmahon H, Austin JH, Gamsu G, et al. Guidelines
for management of small pulmonary nodules detected on CT scans: a statement from the Fleischner society. Radiology. 2005;237:395–400.
Prevalence and clinical significance
Suggested Reading
Abbara S, Kalan MM, Lewicki AM. Intrathoracic stomach
revisited. AJR Am J Roentgenol. 2003;181:403–14.
Baughman RP, Teirstein AS, Judson MA, et al.
Clinical characteristics of patients in a case control study of sarcoidosis. Am J Respir Crit Care Med. 2001;164:1885–9.
Bonder A, Afdhal N.
Liver Dis. 2012;16:271–83.
Bosanko CM, Korobkin M, Fantone JC, et
mary pulmonary lymphoma: CT findings. J Comput Assist Tomogr. 1991;15(4):679–82.
Chakko S. Pleural effusion in congestive heart failure.
Chest. 1990;98:521–2.
Delmas PD, van de Langerijt L, Watts NB, Eastell R,
Genant H, Grauer A, Cahall DL. Underdiagnosis of vertebral fractures is a worldwide problem: the IMPACT study. J Bone Miner Res. 2005;20:557–63.
Descombes E, Gardiol D, Leuenberger P. Transbronchial
lung biopsy: an analysis of 530 cases with reference to the number of samples. Monaldi Arch Chest Dis. 1997;52:324–9.
Franquet T.
Gharib AM, Stern EJ. Radiology of pneumonia. Med Clin
Haniuda M, Kondo R, Numanami H, et al. Recurrence of
Jang HJ, Kim TK, Lim HK, et al. Hepatic hemangioma:
Kahrilas PJ, Kim HC, Pandolfino JE. Approaches to the
Konstantinides SV, Torbicki A, Agnelli G, et
Imaging of pneumonia: trends and algorithms.
Eur Respir J. 2001;18:196–208.
North Am. 2001;85:1461–91. x
thymoma: clinicopathological features, re-operation, and outcome. J Surg Oncol. 2001;78:183–8.
atypical appearances on CT, MR imaging, and sonog­raphy. AJR Am J Roentgenol. 2003;180:135–41.
diagnosis and grading of hiatal hernia. Best Pract Res Clin Gastroenterol. 2008;22:601–16.
ESC guidelines on the diagnosis and manage­ment of acute pulmonary embolism. Eur Heart J. 2014;35:3033–69.
Evaluation of liver lesions. Clin
al. Lobar pri-
al. 2014
Lee SS, Park SH.
holic fatty liver disease. World J Gastroenterol. 2014;20:7392–402.
Light RW, MacGregor MI, Luchsinger PC, Ball
WC.
Pleural effusions: the diagnostic separation of transudates and exudates. Ann Intern Med. 1972;77:507–13.
Lillington GA, Caskey CI.
solitary and multiple pulmonary nodules. Clin Chest Med. 1993;14:111–9.
Lombardi G, Zustovich F, Nicoletto MO, et
and treatment of malignant pleural effusion: a system­atic literature review and new approaches. Am J Clin Oncol. 2010;33:420–3.
Midthun DE, Swensen SJ, Jett JR.
pulmonary nodule. Mayo Clin Proc. 1993;68:378–85.
Mueller J, Jeudy J, White CS.
phy after coronary artery bypass surgery: preva­lence of incidental findings. AJR Am J Roentgenol. 2007;189:414–9.
Onuma Y, Tanube K, Hatori M, et
in coronary imaging with multi-detector computed tomography. J Am Coll Cardiol. 2006;48:402–6.
Patel U, Skingle S, Campbell GA, Crisp AJ, Boyle
IT.
Clinical profile of acute vertebral compres­sion fractures in osteoporosis. Br J Rheumatol. 1991;30:418–21.
Pickhardt PJ, Park SH, Hahn L, et
hanced CT for non-invasive diagnosis of hepatic steatosis: implications for the investigation of the natural history of incidental steatosis. Eur Radiol. 2012;22:1075–82.
Rybicki BA, Major M, Popovich J Jr, et
ferences in sarcoidosis incidence: a 5-year study in a health maintenance organization. Am J Epidemiol. 1997;145:234–41.
Savage JW, Schroeder GD, Anderson PA.
and kyphoplasty for the treatment of osteoporotic ver­tebral compression fractures. J Am Acad Orthop Surg. 2014;22:653–64.
Smith SB, Geske JB, Maguire JM, et al. Early anticoagu-
lation is associated with reduced mortality for acute pulmonary embolism. Chest. 2010;137:1382–90.
Statement on sarcoidosis. Joint Statement of the
American Thoracic Society (ATS), the European Respiratory Society (ERS) and the World Association of Sarcoidosis and Other Granulomatous Disorders (WASOG) adopted by the ATS Board of Directors and by the ERS Executive Committee. Am J Respir Crit Care Med. 1999;160:736–55.
Stein PD, Beemath A, Matta F, et al. Clinical characteris-
tics of patients with acute pulmonary embolism: data from PIOPED II.
Ströbel P, Bauer A, Puppe B, et al. Tumor recurrence and
survival in patients treated for thymomas and thymic
Radiologic evaluation of nonalco-
Evaluation and management of
al. Diagnosis
Approach to the solitary
Cardiac CT angiogra-
al. Non-cardiac findings
al. Specificity of unen-
al. Racial dif-
Vertebroplasty
Am J Med. 2007;120:871–9.
350
https://t.me/med1917
C. Brown and C.S. White
squamous cell carcinomas: a retrospective analysis. J Clin Oncol. 2004;22:1501–9.
Thomas KW, Hunninghake GW. Sarcoidosis. JAMA.
2003;289:3300–3.
Van Belle A, Büller HR, Huisman MV, et al. Effectiveness
of managing suspected pulmonary embolism using an
algorithm combining clinical probability, D-dimer test­ing, and computed tomography. JAMA. 2006;295:172–9.
Yun EJ, Choi BI, Han JK, et al. Hepatic hemangioma:
contrast-enhancement pattern during the arterial and portal venous phases of spiral CT. 1999;24:262–6.
Abdom Imaging.
Structural Intervention:
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A Cardiologist’s Perspective
John J. Lee, Igor F. Palacios, and Alexander Llanos
12
12.1 Case 12.1
12.1.1 History
A 76-year-old male presenting with chest dis­comfort and shortness of breath.
12.1.2 Findings
Cardiac CT Angiogram (CCTA) demonstrated multi-vessel coronary disease with a critical lesion that was very short in length of the ostial LAD (Fig. 12.1a–c).
Coronary angiography demonstrated a criti­cal stenosis involving the ostial LAD involving the bifurcation of a large first diagonal artery. Mild non-obstructive disease involving the cir­cumflex artery and the right coronary artery (Fig. 12.1d, e).
12.1.3 Diagnosis
Critical ostial LAD disease (short lesion length ~3 mm).
12.1.4 Discussion
The ability for the interventional cardiologist to know the distribution of coronary stenosis prior to arrival to the cath lab facilitates better defining lesion severity with invasive angiography. In par­ticular, ostial coronary lesions of short length can potentially be overlooked at the time of cardiac catheterization due to overlapping of vessel seg­ments. This patient underwent a minimally invasive off-pump coronary bypass surgery consisting of a LIMA to the LAD. Description of the coronary anatomy by CCTA allows for more detailed discus­sion with our patients regarding options for therapy prior to their arrival to the cath lab for invasive angi­ography where they may receive sedation agents.
J.J. Lee, MD Department of Medicine, University of Miami at Holy Cross Hospital, Fort Lauderdale, FL, USA
I.F. Palacios, MD Department of Medicine and Cardiology, Massachusetts General Hospital, Boston, MA, USA
© Springer International Publishing AG 2018 C. Smuclovisky (ed.), Coronary Artery CTA, https://doi.org/10.1007/978-3-319-66988-5_12
A. Llanos, MD (*) Department of Cardiology, Holy Cross Hospital, Jim Morgan Heart and Vascular Research Institute, Fort Lauderdale, FL, USA e-mail: alexander.llanos@holy-cross.com
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a
c
b
Fig. 12.1 (a) cMPR of LAD (b) stretched MPR of LAD (c) coronary tree view (d and e) coronary angiograms
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12.2 Case 12.2
12.2.1 History
A 67-year-old male with previous history of cor­onary artery disease status post percutaneous coronary intervention to the right coronary artery and the left anterior descending, now presenting with recurrent chest pain.
12.2.2 Findings
CCTA demonstrated hard and soft plaque in the mid LAD past the first diagonal that appeared to
a
be hemodynamically significant with a stenosis of at least 70% (Fig. 12.2a, b). There was an occluded proximal RCA stent (Fig. 12.2c).
Coronary angiography demonstrated a well­collateralized right coronary system from the left coronary system. Moderate disease in the left anterior descending system at the distal edge of a previously placed stent: non-flow limiting lesion as evaluated by both angiography and FFR (FFR
0.88) (Fig. 12.2d, e).
12.2.3 Diagnosis
Non-flow limiting lesion in the LAD.
b
c
Fig. 12.2 (a) cMPR of LAD (b) stretched MPR of LAD (c) stretched MPR of RCA (d and e) LAD and LCX coro­nary angiogram (f) RCA Coronary angiogram demon-
strating proximal total occlusion (g) Coronary angiogram demonstrating left to right collaterals
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d
f
e
g
Fig. 12.2 (continued)
12.2.4 Discussion
Being able to understand the coronary anatomy prior to arriving to the cath lab is helpful. The planned addition of physiologic coronary assess­ment by means of flow evaluation is an exciting future advancement that will help to further enhance the utility of CT angiography to the cardiologist.
This patient was found to have an occluded proximal RCA and a 70% lesion in the LAD by coronary CTA. He was further evaluated with invasive angiography which verified the above findings. FFR evaluation of the LAD determined that the lesion was not significantly flow limiting and the decision was made for medical therapy. As detailed in previous chapter, the addition of fractional flow reserve to CCTA imaging will better allow us to determine care options.
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12.3 Case 12.3
12.3.1 History
A 52-year-old male with past medical history of hyperlipidemia and hypertension who presented with exertional chest discomfort.
12.3.2 Findings
CCTA demonstrated critical two vessel disease involving the LAD and LCX. There was a chronic total occlusion of the mid LAD with collateral filling of the distal LAD. There was suspicion of high-grade obstruction in the proximal to mid circumflex (Fig. 12.3a, b).
Invasive coronary angiography was performed and demonstrated a 50% proximal LAD lesion followed by a total occlusion in the distal segment of the LAD with a well-collateralized apical LAD segment (anterograde bridging collateral). The left circumflex had a mid 85% lesion and the right coronary had a mid 70% lesion.
12.3.3 Diagnosis
Severe two vessel coronary artery disease of the LAD and LCX.
12.3.4 Discussion
We performed three vessel fractional flow reserve and found the RCA lesion to be non-flow limiting (FFR = 0.90). Evaluation of the proximal LAD 50% lesion also demonstrated non-flow limiting value (FFR = 0.88). FFR of the mid LCX 85% lesion was found to be flow limiting (FFR = 0.69). It was decided to proceed with percutaneous intervention of the mid left circumflex. A bioab­sorbable stent was placed without complication (Fig. 12.3c). The remaining coronary disease was medically managed.
Current advances in coronary CT angiography are likely to involve the ability to evaluate a noninvasive fractional flow reserve which will help us to better evaluate patients and help guide therapy.