- •Contents
- •Contributors and consultants
- •Not another boring foreword
- •A look at cardiac anatomy
- •A look at cardiac physiology
- •A look at ECG recordings
- •All about leads
- •Observing the cardiac rhythm
- •Monitor problems
- •A look at an ECG complex
- •8-step method
- •Recognizing normal sinus rhythm
- •A look at sinus node arrhythmias
- •Sinus arrhythmia
- •Sinus bradycardia
- •Sinus tachycardia
- •Sinus arrest
- •Sick sinus syndrome
- •A look at atrial arrhythmias
- •Premature atrial contractions
- •Atrial tachycardia
- •Atrial flutter
- •Atrial fibrillation
- •Wandering pacemaker
- •A look at junctional arrhythmias
- •Premature junctional contraction
- •Junctional escape rhythm
- •Accelerated junctional rhythm
- •Junctional tachycardia
- •A look at ventricular arrhythmias
- •Premature ventricular contraction
- •Idioventricular rhythms
- •Ventricular tachycardia
- •Ventricular fibrillation
- •Asystole
- •A look at AV block
- •First-degree AV block
- •Type I second-degree AV block
- •Type II second-degree AV block
- •Third-degree AV block
- •A look at pacemakers
- •Working with pacemakers
- •Evaluating pacemakers
- •A look at biventricular pacemakers
- •A look at radiofrequency ablation
- •A look at ICDs
- •A look at antiarrhythmics
- •Antiarrhythmics by class
- •Teaching about antiarrhythmics
- •A look at the 12-lead ECG
- •Signal-averaged ECG
- •A look at 12-lead ECG interpretation
- •Disorders affecting a 12-lead ECG
- •Identifying types of MI
- •Appendices and index
- •Practice makes perfect
- •ACLS algorithms
- •Brushing up on interpretation skills
- •Look-alike ECG challenge
- •Quick guide to arrhythmias
- •Glossary
- •Selected references
- •Index
- •Notes
INTERPRETING A 12-LEAD ECG
262
Causes of axis deviation
This list covers common causes of right and left axis deviation.
Left |
Right |
• Normal variation |
• Normal variation |
• Inferior wall myocardial infarc- |
• Lateral wall MI |
tion (MI) |
• Left posterior hemiblock |
• Left anterior hemiblock |
• Right bundle-branch block |
• Wolff-Parkinson-White |
• Emphysema |
syndrome |
• Right ventricular hypertrophy |
• Mechanical shifts (ascites, |
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pregnancy, tumors) |
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• Left bundle-branch block |
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• Left ventricular hypertrophy |
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• Aging |
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No worries
Axis deviation isn’t always cause for alarm, and it isn’t always cardiac in origin. For example, infants and children normally have right axis deviation. Pregnant women normally have left axis deviation.
Disorders affecting a 12-lead ECG
A 12-lead ECG is used to diagnose such conditions as angina, bundle-branch block, and myocardial infarction (MI). By reviewing sample ECGs, you’ll know what classic signs to look for. Here’s a rundown on these three common cardiac conditions and what 12-lead ECG signs to look for.
Angina
During an episode of angina, the myocardium demands more oxygen than the coronary arteries can deliver. The arteries can’t deliver enough blood, commonly as a result of a narrowing of the arteries from coronary artery disease (CAD), a condition that may be complicated by platelet clumping, thrombus formation, or vasospasm.
An episode of angina usually lasts between 2 and 10 minutes. The closer to 30 minutes the pain lasts, the more likely the pain is from an MI rather than angina.
Angina pain that lasts close to 30 minutes most likely signals an MI.
DISORDERS AFFECTING A 12-LEAD ECG |
263 |
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Stable or unstable?
You may hear the term stable angina applied to certain conditions and unstable angina applied to others. In stable angina, pain is triggered by exertion or stress and is usually relieved by rest. Each episode follows the same pattern.
Unstable angina, which is one of the components of acute coronary syndrome, is more easily provoked, usually waking the patient. It’s also unpredictable and worsens over time. The patient with unstable angina is treated as a medical emergency. The onset of unstable angina commonly indicates an MI.
In addition to ECG changes, the patient with unstable angina will complain of chest pain that may radiate. The pain is generally more intense and lasts longer than the pain of stable angina. The patient may also be pale, clammy, nauseous, and anxious.
Fleeting change of heart
A patient with either form of angina typically shows ischemic changes on the ECG only during the angina attack. (See ECG changes associated with angina.) Because these changes may be fleeting, always obtain an order for, and perform, a 12-lead ECG as soon as the patient reports chest pain.
The ECG will allow you to analyze all parts of the heart and pinpoint which area and coronary artery are involved. By recognizing danger early, you may be able to prevent an MI or even death.
Drugs are a key component of anginal treatment and may include nitrates, beta-adrenergic blockers, calcium channel
ECG changes associated with angina
Here are some classic ECG changes involving the T wave and ST segment that you may see when monitoring a patient
with angina. |
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Peaked T wave |
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Flattened T wave |
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T-wave inversion |
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ST-segment |
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depression with |
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T-wave inversion |
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ST-segment depression without T-wave inversion
INTERPRETING A 12-LEAD ECG
264
blockers, and aspirin or glycoprotein IIb/IIIa inhibitors to reduce platelet aggregation.
Bundle-branch block
One potential complication of an MI is a bundle-branch block. In this disorder, either the left or the right bundle branch fails to conduct impulses. A bundle-branch block that occurs farther down the left bundle, in the posterior or anterior fasciculus, is called a hemiblock.
Some blocks require treatment with a temporary pacemaker. Others are monitored only to detect whether they progress to a more complete block.
Impulsive behavior
In a bundle-branch block, the impulse travels down the unaffected bundle branch and then from one myocardial cell to the next to depolarize the ventricle.
Because this cell-to-cell conduction progresses much more slowly than the conduction along the specialized cells of the conduction system, ventricular depolarization is prolonged.
Wide world of complexes
Prolonged ventricular depolarization means that the QRS complex will be widened. The normal width of the complex is 0.06 to 0.10 second. If the width increases to greater than 0.12 second, a bundle-branch block is present.
After you identify a bundle-branch block, examine lead V1, which lies to the right of the heart, and lead V6, which lies to the left of the heart. You’ll use these leads to determine whether the block is in the right or left bundle.
Right bundle-branch block
RBBB occurs with such conditions as anterior wall MI, CAD, cardiomyopathy, cor pulmonale, and pulmonary embolism. It may also occur without cardiac disease. If this block develops as the heart rate increases, it’s called rate-related RBBB. (See How RBBB occurs.)
In this disorder, the QRS complex is greater than 0.12 second and has a different configuration, sometimes resembling rabbit ears or the letter “M.” (See Recognizing RBBB, page 266.) Septal depolarization isn’t affected in lead V1, so the initial small R wave remains.
The R wave is followed by an S wave, which represents left ventricular depolarization, and a tall R wave (called R prime,
or R ), which represents late right ventricular depolarization. The
DISORDERS AFFECTING A 12-LEAD ECG |
265 |
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How RBBB occurs
In right bundle- |
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branch block |
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(RBBB), the initial |
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impulse activates |
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the interventricu- |
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lar septum from |
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left to right, just |
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as in normal |
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activation. Next, |
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the left bundle |
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branch activates |
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the left ventricle. |
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The impulse then |
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crosses the |
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interventricular |
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septum to activate |
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the right ventricle. |
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T wave is negative in this lead. However, that deflection is called a secondary T-wave change and is of no clinical significance.
Opposing moves
The opposite occurs in lead V6. A small Q wave is followed by depolarization of the left ventricle, which produces a tall R wave. Depolarization of the right ventricle then causes a broad S wave. In lead V6, the T wave should be positive.
Left bundle-branch block
Left bundle-branch block (LBBB) never occurs normally. This block is usually caused by hypertensive heart disease, aortic stenosis, degenerative changes of the conduction system, or CAD. (See How LBBB occurs, page 267.) When it occurs along with an anterior wall MI, it usually signals complete heart block, which requires insertion of a pacemaker.
INTERPRETING A 12-LEAD ECG
266
Recognizing RBBB
This 12-lead ECG shows the characteristic changes of right bundle-branch block (RBBB). In lead V1, note the rsR pattern and T-wave inversion. In lead V6, see the widened S wave and the upright T wave. Also note the prolonged QRS complexes.
Lead I |
Lead aVR |
Lead V1 |
Lead V4 |
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Lead II |
Lead aVL |
Lead V2 |
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Lead V5 |
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Lead III |
Lead aVF |
Lead V3 |
|
Lead V6 |
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One ventricle after another
In LBBB, the QRS complex will be greater than 0.12 second because the ventricles are activated sequentially, not simultaneously. (See Recognizing LBBB, page 268.) As the wave
DISORDERS AFFECTING A 12-LEAD ECG |
267 |
|
How LBBB occurs
In left bundle- |
|
branch block |
|
(LBBB), the impulse |
|
first travels down |
|
the right bundle |
|
branch. Then the |
|
impulse activates |
|
the interventricular |
|
septum from right |
|
to left, the opposite |
|
of normal activa- |
Block |
tion. Finally, the |
|
impulse activates |
|
the left ventricle. |
1 |
3
2
of depolarization spreads from the right ventricle to the left, a wide S wave is produced in lead V1, with a positive T wave. The S wave may be preceded by a Q wave or a small R wave.
In LBBB, the S wave may be preceded by a Q wave or R wave.
Cowabunga!
Slurring your R waves
In lead V6, no initial Q wave occurs. A tall, notched R wave, or a slurred one, is produced as the impulse spreads from right to left. This initial positive deflection is a sign of LBBB. The T wave is negative.
Myocardial infarction
Unlike angina, pain from an MI lasts for at least 20 minutes, may persist for several hours, and is unrelieved by rest. MI usually occurs in the left ventricle, although the location may vary depending on the coronary artery affected.
INTERPRETING A 12-LEAD ECG
268
Recognizing LBBB
This 12-lead ECG shows characteristic changes of left bundle-branch block (LBBB). All leads have prolonged QRS complexes. In lead V1, note the QS wave pattern. In lead V6, you’ll see the slurred R wave and T-wave inversion. The elevated ST segments and upright T waves in leads V1 to V4 are also common in LBBB.
Lead I |
Lead aVR |
|
Lead V1 |
|
Lead V4 |
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Lead II |
Lead aVL |
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Lead V2 |
Lead V5 |
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Lead III |
Lead aVF |
|
Lead V3 |
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Lead V6 |
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For as long as the myocardium is deprived of an oxygen-rich blood supply, an ECG will reflect the three pathologic changes of an MI: ischemia, injury, and infarction. (See Reciprocal changes in an MI.)
DISORDERS AFFECTING A 12-LEAD ECG |
269 |
|
Reciprocal changes in an MI
Ischemia, injury, and infarction — the three I’s of a myocardial infarction (MI) — produce characteristic ECG changes. Changes shown by the leads that reflect electrical activity in damaged areas are shown on the right of the illustration.
Reciprocal leads, those opposite the damaged area, will show opposite ECG changes, as shown to the left of the illustration.
Changes on damaged side
Injury
Infarction
Reciprocal changes
Ischemia
Injury
Infarction
Ischemia
Zone of infarction
The area of myocardial necrosis is called the zone of infarction. Scar tissue eventually replaces the dead tissue, and the damage caused is irreversible.
INTERPRETING A 12-LEAD ECG
270
The ECG change associated with a necrotic area is a pathologic Q wave, which results from lack of depolarization. Such Q waves are permanent. MIs that don’t produce Q waves are called non–Q-wave MIs. (See Q waves in children.)
Zone of injury
The zone of infarction is surrounded by the zone of injury, which shows up on an ECG as an elevated ST segment. ST-segment elevation results from a prolonged lack of blood supply.
Zone of ischemia
The outermost area is called the zone of ischemia and results from an interrupted blood supply. This zone is represented on an ECG by T-wave inversion. Changes in the zones of ischemia or injury are reversible.
From ischemia to injury
Generally, as an MI occurs, the patient experiences chest pain and an ECG shows changes, such as ST-segment elevation, which indicates that myocardial injury is occurring. You’ll also typically see T waves flatten and become inverted.
Rapid treatment can prevent myocardial necrosis. However, if symptoms persist for more than 6 hours, little can be done to prevent necrosis. That’s one of the reasons patients are advised to seek medical attention as soon as symptoms begin.
The telltale Q wave
Q waves can appear hours to days after an MI and signify that an entire thickness of the myocardium has become necrotic. Tall
R waves in reciprocal leads can also develop. This type of MI is called a transmural, or Q-wave, MI.
Back to baseline
Knowing how long such changes last can help you determine how long ago an MI occurred. ST segments return to baseline within
a few days to 2 weeks. Inverted T waves may persist for several months. Although not every patient who has had an MI develops Q waves, those who do may have them on their ECGs indefinitely.
What to do for an MI
The most important step you can take for a patient with an MI is to remain vigilant about detecting changes in his condition and in his ECG. (See Monitoring MI patients.)
The primary goal of treatment for an MI is to limit the size of the infarction by decreasing cardiac workload and increasing oxygen supply to the myocardium. (See Improving blood flow.)
Ages
and stages
Q waves in children
Q waves in leads II, III,
aVF, V5, and V6 are normal in children. Q waves
in other leads suggest cardiac disease, such as an abnormal left coronary artery.
Mixed signals
Monitoring MI patients
Remember that specific leads monitor specific walls of the heart. Here’s a quick overview of those leads:
• For an anterior wall myocardial infarction (MI), monitor lead V1 or MCL1.
•For a septal wall MI,
monitor lead V1 or MCL1 to pick up hallmark
changes.
•For a lateral wall MI, monitor lead V6 or MCL6.
•For an inferior wall MI, monitor lead II.