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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_1365_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Contents
- •List of Contributors
- •1.1 Introduction
- •1.2 Risk Factors
- •1.6.1 Esophagitis
- •1.6.2 Barrett Esophagus
- •1.6.3 Esophageal Neoplasia
- •1.6.4 Esophageal Peptic Stricture
- •1.7.1 Perforation
- •1.7.2 Fundoplication Construction
- •1.8.1 Perforation
- •1.8.2 Tight Fundoplication
- •1.8.3 Disrupted/Loose Fundoplication
- •1.8.4 Slipped Fundoplication
- •1.8.5 Recurrent Hiatal Hernia
- •1.8.6 Twisted or Malconstructed Fundoplication
- •1.9 Conclusions
- •References
- •2.1 Summary
- •2.4 Summary
- •References
- •3.1 Introduction
- •3.2 EGJ Anatomy
- •3.3 EGJ Function
- •3.4.3 Hiatus Hernia
- •3.5 Hiatus Hernia: Diagnosis
- •3.6 EGJ Measurement
- •3.10 Summary
- •References
- •4.1.2.1 Mucosal Breaks
- •4.1.2.2 Barrett’s Esophagus
- •4.1.2.3 Contrast Esophagrams
- •4.1.3 Catheter-Based pH Monitoring
- •4.1.4 Wireless pH Monitoring
- •4.1.6 pH Electrode Placement
- •4.1.8 Symptoms Association
- •4.1.9 pH testing On- versus Off-Acid Suppressive Medication
- •4.1.11 Proximal Esophageal pH Assessment
- •4.1.12 Multichannel Intraluminal Impedance
- •4.1.14 Other Preoperative Tests
- •References
- •5.1 Introduction
- •5.3 Clinical Presentation
- •5.3.1 Atypical Symptoms
- •5.3.2 Dysphagia
- •5.4 Preoperative Work-Up
- •5.4.1 pH Monitoring
- •5.4.2 Esophageal Manometry
- •5.4.3 Esophagogastroduodenoscopy
- •5.4.4 Barium Esophagram
- •5.4.5 Impedance Testing
- •5.5 Additional Preoperative Considerations
- •5.5.1 Obesity
- •5.5.2 Partial Versus Complete Fundoplication
- •5.5.3 Barrett’s Esophagus
- •5.6 Surgical Management
- •5.7 Operative Technique
- •5.8 Postoperative Care
- •5.9.1 Pneumothorax
- •5.9.3 Splenic Injury or Bleeding
- •5.9.4 Bloating
- •5.9.5 Dysphagia
- •5.10 Conclusion
- •References
- •6.1 Introduction
- •6.2 Preoperative Evaluation
- •6.3 Partial Anterior Fundoplication Technique
- •6.4 Posterior Partial Fundoplication Technique
- •6.5 Posterior Complete Fundoplication Technique
- •6.6 Medical Management Versus Surgery
- •6.8 Dysphagia Side Effects
- •6.11 Conclusions
- •References
- •7.1 Introduction
- •7.2 Precision GERD Management
- •7.2.1 GERD Validation
- •7.2.2 Hiatal Hernia Assessment
- •7.2.4 Prior Therapies
- •7.2.5 Obesity
- •7.2.6 Extra-Esophageal Symptoms
- •7.3.2 Transoral Fundoplication (TF)
- •7.3.3 MUSE
- •7.4 Conclusions
- •References
- •8.5 Conclusions
- •References
- •9.1 Introduction
- •9.2 Epidemiology
- •9.4 Diagnostic Evaluation
- •9.7 Mesh Complications
- •9.7.1 Mesh Erosion
- •9.7.2 Mesh Related Fibrosis
- •9.7.3 Recurrence
- •9.7.4 Reoperation
- •9.7.5 Dysphagia
- •9.8 Conclusions
- •References
- •10.1 Introduction
- •11.1 Introduction
- •11.2.1 Indications
- •11.2.2 Preoperative Workup
- •11.2.3 Is One Fundoplication Better than Another?
- •References
- •11.3 Conclusions
- •References
- •12.1 Introduction
- •12.3 High-Resolution Impedance Manometry (HRIM)
- •12.3.1 HRIM Study Protocol
- •12.3.2 HRIM Interpretation
- •12.3.2.1 Individual High-Resolution Manometry Metrics
- •12.3.2.3 Deglutitive LES Relaxation
- •12.3.2.4 Distal Latency
- •12.3.2.5 Peristaltic Vigor
- •12.3.2.6 Peristaltic Integrity
- •12.3.2.7 Pressurization Pattern
- •12.3.2.8 Individual Impedance Based Metrics
- •Bolus Flow Time
- •12.4 Functional Lumen Imaging Probe (Flip)
- •12.4.1 FLIP: Protocol
- •12.4.2 FLIP Analysis
- •12.5 Conclusions
- •References
- •13.2 Pathophysiology
- •13.3 Clinical Presentation
- •13.4 Radiologic Studies
- •13.5 Upper Gastrointestinal Endoscopy
- •13.6 High Resolution Manometry
- •13.7 Esophageal pH Monitoring
- •13.8 Assessment Under Urgent Conditions
- •13.9 Decision Making
- •References
- •14.2.1 Patient History
- •14.2.2 Diagnostic Tests
- •14.4.1 Poor Patient Selection
- •14.4.2 Improper Surgical Technique
- •14.4.3 Inadequate Patient Counseling
- •14.4.4 Fundoplication/Hiatus Disruption
- •14.4.5 Patient Body Habitus
- •14.5 Conclusions
- •References
- •15.1 Introduction
- •15.2 Clinical Presentation
- •15.3 Evaluation
- •15.4 Surgical Planning
- •15.5 Technical Considerations
- •15.6 Post Operative Care
- •15.7 Conclusion
- •Appendix
- •References
- •16.1 Introduction
- •16.2 Causes of Failure
- •16.2.2 Technical Issues
- •16.2.3 Patient Factors
- •16.3 Identifying Recurrence After Hiatal Hernia Repair
- •16.4 Surgical Strategies
- •16.4.1 Preparation
- •16.4.2 Exposure/Dissect3ion
- •16.4.3 Crural Closure
- •16.4.4 Intra-operative Endoscopy
- •16.4.5 Short Esophagus
- •16.4.6 Fundoplication
- •16.4.7 Gastropexy/Gastrostomy Tube
- •16.4.10 Post-operative Considerations
- •16.4.11 Long-Term Post-operative Care
- •16.5 Summary
- •References
- •17.1 Introduction
- •17.3 Reoperation Techniques
- •17.5 Long-Term Outcomes
- •17.6 Conclusions
- •References
- •18.1 Introduction
- •18.4 Da Vinci Surgical System
- •18.7 Redo Paraesophageal Hernia Repair
- •18.9 Conclusion
- •References
- •19.1 Introduction
- •19.9 Conclusion
- •References
- •Index

214
b
c
d
e
S. Mittal
29
27
24
21
18
15
12
9
5
3
0
-3
-6
14
28
21
24
27
30
33
35
39
42
45
49
29
27
24
21
18
15
12
9
6
3
0
-3
-5
29
27
24
21
18
15
12
9
5
3
0
-3
-5
IRP
IRP
Disrupted fundoplication
Basal LESP
Twisted fundoplication
Basal LESP
Intact intra-thoracic fundoplication
Basal LESP
Diaphragmatic Hiatus
Disrupted intra-thoracic fundoplication
Basal LESP
Diaphragmatic Hiatus
Fig. 14.2 (continued)

f
g
14 Recurrent GERD After aFundoplication: Failure or Wrong Procedure
215
12
15
18
21
24
27
30
33
36
39
42
45
47
29
27
24
25
18
15
12
9
6
3
0
-3
-6
Fig. 14.2 (continued)
Slipped fundoplication
Basal LESP
Fundoplication
Secondary achalasia
Aperistaltic body contraction
IRP Basal LESP
(d) Prolonged pH monitoring
Either 24h catheter based or 48h Bravo pH monitoring off acid suppression
therapy is the gold standard to assess for extent of gastro-esophageal reux. In
our experience impedance reux testing on therapy has not been very useful.
(e) Gastric Emptying Study (GES)
Nuclear medicine GES is useful to assess presence and degree of delayed gastric emptying and will help direct the need for gastric drainage procedure. A 4h
study is preferable and moderate to severe delayed GE should be addressed
with either a pyloroplasty or anterectomy at the time of redo surgery.
14.3 How toClassify Failed Fundoplication?
Failed fundoplication have most commonly classied anatomically based on recurrence of hiatus hernia, relative location of the fundoplication to lower esophagus and
geometry of the fundoplication. Jobe etal. [6] described in detail endoscopic characteristics of technically sound fundoplication and laid out the criteria for endoscopic

216
S. Mittal
assessment. However, majority of endoscopic assessment done in the community do
not describe the fundoplication adequately. An audit of reported endoscopic ndings
by community gastroenterologists by Juhasz etal. [3] revealed a shockingly low concordance with subsequent ndings of an experienced foregut surgeon. Surprisingly
they found that nearly a 1/3rd of the endoscopic reports did not even mention a previous fundoplication while majority of the reports simply stated, “fundoplication changes
noted” without any specic description. Proper and uniform endoscopic assessment of
fundoplication is an import aspect in assessing causes of failure and devising a management strategy. Various terminologies have been used to describe failed fundoplication anatomy. Most commonly terms such as recurrent hiatus hernia, slipped,
intra-thoracic, disrupted, twisted and telescopic fundoplication have been used. They
have also been used interchangeably without accepted denitions. For example one
may classify an intra-thoracic fundoplication is “slipped into the chest” or “telescoping
in to the chest” while a fundoplication at the hiatus with GEJ above the hiatus may be
called “ slipped below the GJ” or recurrent HH” or “misplaced fundoplication”.
Horagan etal. [7] were the rst to propose an anatomic classication for failed
fundoplication. They classied failed fundoplication into type IA (both EGJ and
Fundoplication above the hiatus), type 1B (GEJ above hiatus with fundoplication
below the hiatus, type II (only a portion of fundus/ greater curvature herniated above
the hiatus) and type III ( body of the stomach used to create the wrap rather than the
fundus) (Table14.1). More recently a standardized system based on location of the
GEJ relative to the hiatus, the fundoplication relative to the GEJ and geometry of the
fundoplication has been proposed by Mittal etal. [8] (Table14.2). Each of the component E, S, F and P are given a ‘sufx’ and nal description is given as E
xSxFxPx
.
While not discussed in literature till now a failed fundoplication may be better clas-
sied based on the underlying physiological derangements. Normal gastrointestinal
function requires the ante-grade esophageal clearance thru the gastroesophageal junction (GEJ) and gastric clearance thru the pylorus along with a competent lower esophageal junction (LES) to prevent backow. Furthermore, some degree of GEJ complex
incompetence (permissiveness) is desirable to allow for gastric venting i.e. belching.
Poor esophageal emptying due to either poor motility and/or non-compliant
lower esophageal sphincter- fundoplication (LES-F) complex will manifest with
dysphagia and bland regurgitation. Not infrequently, in patients with poor motility,
there may be aspiration associated cough or throat clearance. The patient or an inexperienced physician may confuse these symptoms as recurrent reux, especially if
regurgitation and cough are more prominent. On the other hand a non-functioning
LES-F complex with poor competence allowing abnormal backow of gastric contents will present with true recurrent GERD.Underlying delayed gastric emptying
either pre-existing or due to vagal injury will present with bloating, nausea and
Table 14.1 Classication of failed fundoplication (by Horgan et al. [7])
Type of failure Description
Type Ia Both GEJ and fundoplication herniated above the hiatus
Type Ib GEJ above the hiatus and fundoplication at the hiatus
Type II Herniated stomach/greater curvature with GEJ and fundoplication
Type III Wrong part (body) of the stomach used for fundoplication
below the hiatus

14 Recurrent GERD After aFundoplication: Failure or Wrong Procedure
Table 14.2 Endoscopic classication of failed fundoplication (by Mittal et al. [8])
Classication of the endoscopic ndings of fundoplication
Type of failure Description
“E” component Distance of GEJ to the level of crura component
E
0
E
1
E
2
GEJ is located intra-abdominally, at or under the level of crura
GEJ is located less than 2 cm above the level of crura
GEJ is located more than 2 cm above the level of crura
“S” component Amount of gastric tissue above the fundoplication and below the GEJ
S
0
S
1
S
2
Fundoplication is around the distal esophagus
Less than 2 cm gastric tissue above the fundoplication
More than 2 cm gastric tissue above the fundoplication
“F” component Description of the fundoplication
F
0
F
1a
F
1b
F
2a
F
2b
Intact fundoplication (competent, symmetrical)
Partially disrupted fundoplication
Completely disrupted fundoplication
Twisted fundoplication
Two-compartment stomach
“P” component Present of paraesophageal hernia (veried by endoscopy
oresophagogram)
P
0
P
1
No PEH
Recurrent PEH
217
epigastric pain, which may again be interpreted as recurrent GERD by patients and
physicians alike. Not infrequently these symptoms become more prominent once
reux symptoms are addressed specially in patients with underlying Irritable Bowel
Syndrome (IBS). There is a signicant overlap in the clinical spectrum of GERD
and IBS. Careful assessment of each dissatised post-fundoplication patient is
essential to identify the underlying physiological derangement and one must be
aware that more than one derangement may co-exist.
In the dissatised post-fundoplication patient, the anatomical and physiological
ndings should be concordant with symptomatology before surgical re-intervention
is attempted. In our experience, patients with functional complaints without
supportive objective ndings may present with “failed fundoplication” and as
“dissatised patients” and are best addressed with reassurance and counseling rather
than surgical re-intervention.
14.4 Causes ofFailed Fundoplication
14.4.1 Poor Patient Selection
Objective evidence of the presence of pathological reux is important before a surgical fundoplication. Campos etal. showed that a good response to acid- suppression
therapy, abnormal preoperative 24-h pH score and typical symptoms were good
predictors of success of fundoplication [9]. While logical, it is surprising that it is

218
S. Mittal
not uncommon for patients with failed fundoplication to not have had an adequate
documentation of disease prior to surgery. In these patients, it is hazardous to guess
whether the surgery has failed or the disease never existed.
In our opinion, ‘objective’ evidence of GERD are: a classical history of heart-
burn or regurgitation with a very good response to acid suppressive therapy or grade
B or greater reux esophagitis noted on endoscopy. In absence of these, an elevated
24-h pH score as measured by prolonged monitoring off acid suppression is considered gold standard test.
In addition to above clinical scenario, esophago-gastric motility assessment
should be done prior to surgery. High-resolution manometry and video esophagram
are used to assess esophageal function and a gastric emptying study is used as indicated by patient symptoms to assess gastric motility. Additionally we strongly advocate that the operating surgeon performs an endoscopic assessment preferably
pre-operatively and if not possible in the operative room prior to surgery.
Common foregut symptoms of heartburn, regurgitation and dysphagia span the
gamut of underlying esophageal disorders from GERD to Achalasia and not infrequently are simply functional. Included in these are scleroderma patients who often
present with severe reux. Precise diagnosis is needed to direct appropriate surgical
procedure. Patients with scleroderma and primary aperistalsis (non– Achalasia)
may be better managed with primary laparoscopic Roux-en-Y gastric bypass
(LRYGB) rather than a partial fundoplication.
14.4.2 Improper Surgical Technique
Multitude of fundoplication congurations both via thoracic and abdominal cavity
have been described ranging from partial anterior or posterior to a complete fundoplication. All types of fundoplications require obtaining an adequate intra- abdominal
length of the esophagus around which the fundus is wrapped such that the entire
complex lays tension free below a closed hiatus.
The surgical technique needs to sufciently mobilize the mediastinal esophagus
to achieve adequate infra-diaphragmatic esophageal length. The crus pillars need to
be approximated to close the hiatus with or without mesh reinforcement. Finally, the
fundus (part of the stomach above an imaginary horizontal plane from the GEJ) is
wrapped and anchored around the distal esophagus. Inadequate esophageal length
will result in the positioning of fundoplication around the proximal stomach resulting in a “slipped wrap”. Inadequate integrity of crus closure will result in reherniation of the GEJ above the hiatus with (herniated) or without (slipped)
fundoplication. Using the body of the stomach rather than fundus to create the ‘fundoplication’ usually results in a twisted fundoplication. We routinely perform and
strongly recommend that an intra-operative endoscopy should be done to assess
fundoplication prior to nishing the procedure.
Inadequate attention to these essential surgical steps will increase the likelihood
of “failed fundoplication”.

14 Recurrent GERD After aFundoplication: Failure or Wrong Procedure
219
14.4.3 Inadequate Patient Counseling
A correctly created fundoplication increases barrier pressure and decreases the
compliance (i.e., permissiveness) of the GEJ compared to the non-operative state.
This requires patients to eat slowly and to chew the food bolus thoroughly, otherwise they risk experiencing dysphagia. Additionally, given the inherent loss of transient lower esophageal sphincter relaxations (TLESRs) after the fundoplication,
patients may not be able to belch easily, and must avoid carbonated beverages especially in the immediate post-operative period. It is imperative that the patients be
counseled to be cautious and expect these symptoms in the early post-operative
period. These tend to resolve to a great extent in the rst few weeks following
fundoplication.
Retching, gagging and vomiting increase the likelihood of wrap herniation or
disruption, resulting in recurrent GERD.This is a life-time caution that the patients
must be aware of and take precautions against and, have easy and ready access to
anti-emetics. We have also encountered some patients who gag themselves during
vigorous tongue cleansing and this increases failure. Simple counselling to avoid
this has helped tremendously [10].
Additionally, it has been proposed that heavy lifting should be avoided in the
early post-operative period to allow for healing of apposed tissues. Comprehensive
patient counseling should improve patient compliance with post-operative care and
help reduce failure rates.
14.4.4 Fundoplication/Hiatus Disruption
Crus closure and fundoplication are secured with sutures. Natural forces and tissue
characteristics may slowly or abruptly disrupt these anchoring sutures and the scarring, resulting in recurrent hiatus hernia and or disrupted fundoplication. Use of
synthetic mesh has been shown to decrease recurrence of hiatus hernia but is also
associated with increased incidence of severe complications [11, 12]. On the other
hand, bio-prosthesis, though safer, have been shown to decrease only short term
recurrence without affecting long term recurrence rate [13]. Improved understanding of factors leading to recurrent hiatus hernia and development of better surgical
techniques/tools including mesh is needed to decrease these causes of failure.
14.4.5 Patient Body Habitus
Obesity is a risk factor for GERD and prevalence of obesity has dramatically
increased in the Western world especially in the US.Perez etal. [14] were the rst
to report that morbid obesity is associated with poor outcomes after primary

220
S. Mittal
anti- reux surgery. However, others [5] have argued that obesity does not affect
outcomes. However, most surgeons agree that fundoplication may not be the best
procedure for reux control in morbidly obese patients. Laparoscopic Roux en Y
Gastric Bypass (LRYGB) is an excellent anti-reux operation that additionally provides for signicant weight loss and it’s associated salutatory effects on health. Most
2
surgeons would steer morbidly obese patients (BMI>35kg/m
2
as primary intervention. While BMI>35kg/m
has been used as a cut of to direct
) towards a LRYGB
towards bariatric procedure a study by Akimoto etal. [15] showed that patients with
2
BMI>30kg/m
have similar patterns of failure after fundoplication as those with
BMI > 35 kg/m2 and these are distinct from failure patterns in patients with
BMI<30kg/m2. Performing a fundoplication in a morbidly obese patient can be
judged partly a wrong patient choice and partly a wrong procedure choice, but occasionally it has to be considered in severely symptomatic patients (especially with
volume regurgitation and pulmonary disease) if insurance denies coverage for bariatric procedure.
14.5 Conclusions
Recurrent GERD after fundoplication is a complex situation and requires exclusion
of other diagnoses which may mimic GERD symptoms (delayed gastric emptying
or post fundoplication achalasia) as well as separating the real symptoms from functional ones. Not infrequently, an anatomically distorted fundoplication will not have
any associated symptoms and these should not undergo redo surgery. In cases with
objective recurrence of the disease, many underlying factors—both patient and
technical, may be responsible and must be explored to avoid failure of redo surgery.
In certain situations such as missed short esophagus or failure to create a symmetrical fundoplication, there is technical inadequacy or surgical inexperience. However,
non-identication of severe dysmotility or even achalasia and delayed gastric emptying, and proceeding with fundoplication should be diligently avoided.
What Is the Current Knowledge and What Future Direction
Is Required
• Laparoscopic anti-reux surgery in form of hiatus hernia repair and fundoplication is the gold standard for denitive management of pathological
GERD. Excellent patient centered outcomes have been reported with
greater than 90% patient satisfaction at 10 years.
• A subset of patients require re-operative intervention for failed fundoplication which is technically more difcult and associated with increased
peri-operative morbidity along with lesser patient oriented outcomes
compared to primary fundoplication.

14 Recurrent GERD After aFundoplication: Failure or Wrong Procedure
221
• A uniformly accepted and widely endorsed denition of what constitutes a
failed fundoplication is needed which may help tailor the subsequent treatment strategies.
• Understanding the causes of failure need to be elucidated and best strategies to minimize these undertaken.
• Additionally, surgical outcomes of community centers (where majority of
the procedures are done) are less than stellar and have scared patients and
referring physicians alike resulting in signicant decline in use of the
procedure.
• Future undertakings need to critically assess learning curve for laparoscopic anti-reux surgery and incorporating appropriate curricula for training surgeons to work-up patients prior to surgical intervention.
References
1. Zaninotto G, Portale G, Costantini M, Rizzetto C, Guirroli E, Ceolin M, etal. Long-term
results (6–10 years) of laparoscopic fundoplication. J Gastrointest Surg. 2007;11(9):1138–45.
2. Robinson B, Dunst CM, Cassera MA, Reavis KM, Sharata A, Swanstrom LL. 20 years later:
laparoscopic fundoplication durability. Surg Endosc. 2015;29(9):2520–4.
3. Juhasz A, Sundaram A, Hoshino M, Lee TH, Filipi CJ, Mittal SK.Endoscopic assessment of
failed fundoplication: a case for standardization. Surg Endosc. 2011;25(12):3761–6.
4. Masato H, Srinivasan A, Mittal SK.High resolution manometry patterns of lower esophageal sphincter complex in symptomatic post-fundoplication patients. J Gastrointest Surg.
2012;16(4):705–14.
5. D’Alessio MJ, Arnaoutakis D, Giarelli N, Villadolid DVm Rosemurgy AS.Obesity is not a
contra-indication to laparoscopic Nissen fundoplication. J Gastrointest Surg. 2005;9:949–54.
6. Jobe BA, Kahrilas PJ, Vernon AH, Sandone C, Gopal DV, Swanstrom LL, etal. Endoscopic
appraisal of the gastroesophageal valve after antireux surgery. Am J Gastroenterol. 2004
Feb;99(2):233–43.
7. Horgan S, Pohl D, Bogetti D, Eubanks T, Peligrini C.Failed anti-reux surgery: what have be
we learned from reoperations? Arch Surg. 1999;134(8):809–15.
8. Mittal SK, Juhasz A, Ramanan B, Hoshino M, Lee TH, Filipi CJ.A proposed classication for
uniform endoscopic description of surgical fundoplication. Surg Endosc. 2014;28(4):1103–9.
9. Campos G, Peters JH, TR DM, Öberg S, Crookes PF, Tan S, etal. Multivariate analysis of
factors predicting outcome after laparoscopic Nissen fundoplication. J Gastrointest Surg.
1999;3(3):292–300.
10. Iqbal A, Kakarlapudi GV, Awad ZT, Haynatzki G, Turaga KK, Karu A, etal. Assessment of
diaphragmatic stressors as risk factors for symptomatic failure of laparoscopic Nissen fundoplication. J Gastrointest Surg. 2006;10(1):12–21.
11. Nandipati K, Bye M, Yamamoto SR, Pallati P, Lee T, Mittal SK.Reoperative intervention in
patients with mesh at the hiatus is associated with high incidence of esophageal resection—a
single-center experience. J Gastrointest Surg. 2013;17(12):2039–44.
12. Stadlhuber RJ, Sherif AE, Mittal SK, Fitzgibbons RJ, Brunt LM, Hunter JG, et al. Mesh
complications after prosthetic reinforcement of hiatal closure: a 28-case series. Surg Endosc.
2009;23(6):1219–26.
13. Frantzides CT, Carlson MA, Loizides S, Papali A, Luu M, Roberts J, et al. Hiatal hernia
repair with mesh: a survey of SAGES members. Surg Endosc. 2010;24(5):1017–24.

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14. Perez AR, Moincure AC, Rattner DW.Obesity adversely affects the outcome of anti-reux
operations. Surg Endosc. 2001;15:986–9.
15. Akimoto A, Nandipati KC, Kapoor H, Yamamoto SR, Pallati PK, Mittal SK.Association of
Body Mass Index (BMI) with patterns of fundoplication failure: insights gained. J Gastrointest
Surg. 2015;19(11):1943–8.
S. Mittal

Chapter 15
Management ofRecurrent
Paraesophageal Hernia
JohnH.Rodriguez andJeffreyPonsky
15.1 Introduction
Laparoscopy has become the standard approach for management of most benign
foregut disease including antireux and hiatal hernia repair. Anatomic recurrence
after repair of paraesophageal hernias is high, however, the clinical signicance of
such ndings seems to be less relevant [1]. Reoperative foregut surgery has a signicantly higher morbidity and mortality than their primary counterparts.
Fortunately, recurrence leading to reintervention is far less common, with a 10 year
cumulative risk close to 7% [2].
The risk of recurrence is likely multifactorial, and many different factors have
been investigated. The best results are typically achieved during initial intervention.
Despite some controversies, complete mediastinal dissection and sac reduction,
adequate esophageal length, crural closure, and a well constructed fundoplication,
constitute some of the surgical principles to be followed at the time of paraesophageal hernia repair [1]. Recently, the role of preoperative body mass index has been
taken into consideration as one of the most important factors that predict recurrence
after repair [3].
The goal of this chapter is to review the clinical presentation, preoperative evaluation, and surgical planning relevant to repair of recurrent paraesophageal
hernias.
J.H. Rodriguez, M.D. • J. Ponsky, M.D. (*)
Section of Surgical Endoscopy, Cleveland Clinic, Cleveland, OH, USA
Lerner College of Medicine, Cleveland Clinic, Cleveland, OH, USA
e-mail: JPonsky@yahoo.com
M.A. Memon (ed.), Hiatal Hernia Surgery,
https://doi.org/10.1007/978-3-319-64003-7_15
223© Springer International Publishing AG 2018
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