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351
93. Jones P, Mempin M, Hu H, et al. The functional
inuence of breast implant outer shell morphology
on bacterial attachment and growth. Plast Reconstr
Surg. 2018;142:837–49.
94. Collett DJ, Rakhorst H, Lennox P, et al. Current
risk estimate of breast implant-associated anaplastic large cell lymphoma in textured breast implants.
Plast Reconstr Surg. 2019;143:30S–40S.
95. Doren EL, Miranda RN, Selber JC, et al. U.S.
epidemiology of breast implant-associated anaplastic large cell lymphoma. Plast Reconstr Surg.
2017;139:1042–50.
96. Brody GS, Deapen D, Taylor CR, et al. Anaplastic
large cell lymphoma occurring in women with breast
implants: analysis of 173 cases. Plast Reconstr Surg.
2015;135:695–705.
97. National Comprehensive Cancer Network. https://
www.nccn.org/guidelines. Accessed 5 Sep 2020.
98. Deva AK, Cuss A, Magnusson M, etal. The “game of
implants”: a perspective on the crisis-prone history
of breast implants. Aesthet Surg J. 2019;39:S55–65.
99. Deva AK. A perspective on the never-ending
cycle of breast implant crises. Aesthet Surg J.
2019;39:NP85–6.
100. (ANSM) Andsdmedpds Le marquage CE des
implants mam-maires texturés de la marque
Allergan (Microcell et Biocell) n’a pas été renouvelé par l’organisme notié GMED - Point
d’information. 2018. https://ansm.sante.fr/S-
informer/Points- d- information- Points- d- information/
Le- marquage- CE- des- implants- mammaires- texturesde- la- marque- Allergan- Microcell- et- Biocell- n- apas- ete- renouvele- par- l- organisme- notifie- GMED-
Point- d- information. Accessed 5 Sep 2020.
101. Santanelli di Pompeo F, Laporta R, Sorotos M, etal.
Breast implant-associated anaplastic large cell lymphoma: proposal for a monitoring protocol. Plast
Reconstr Surg. 2015;136:144e–51e.
102. Swanson E.The textured breast implant crisis: a call
for action. Ann Plast Surg. 2019;82:593–4.
103. FDA. Anaplastic Large Cell Lymphoma (ALCL)
in women with breast implants. https://www.fda.
gov/medical- devices/breast- implants/questionsand- answers- about- breast- implant- associatedanaplastic- large- cell- lymphoma- bia- alcl. Accessed
5 Sep 2020.
104. FDA.USFaDA Statement from FDA Principal Deputy
Commissioner Amy Abernethy, Jeff Shuren, director
of the FDA’s Center for Devices and Radiological
Health on FDA’s new efforts to protect women’s health
and help to ensure the safety of breast implants.
www.fda.gov/ news-
events/press- announcements/
https://
statement- fda- principal- deputy- commissioner- amyabernethy- md- phd- and- jeff- shuren- md- jd- directorfdas. Accessed 5 Sep 2020.
105. FDA. USFaDA FDA. Breast implant- associated
anaplastic large cell lymphoma (BIA-ALCL).
https://www.fda.gov/medical- devices/safety-
communications/fda- requests- allergan- voluntarily-
recall- natrelle- biocell- textured- breast- implants- and-
Accessed 5 Sep 2020.
tissue.
106. Groth AK, Graf R.Breast implant-associated anaplastic large cell lymphoma (BIA-ALCL) and the
textured breast implant crisis [published correction
appears in Aesthetic Plastic Surgery]. Aesthetic Plast
Surg. 2020;44(1):1–12.
107. Sharma A, Schwartz RA, Swan KG. Marjolin’s
warty ulcer. J Surg Oncol. 2011;103:193–5.
108. Copcu E. Marjolin’s ulcer: a preventable complication of burns? Plast Reconstr Surg. 2009;124:156–64.
109. Pekarek B, Buck S, Osher L. A comprehensive
review on Marjolin’s ulcers: diagnosis and treatment. J Am Col Certif Wound Spec. 2011;3(3):60–4.
110. Saaiq M, Ashraf B. Marjolin’s ulcers in the postburned lesions and scars. World J Clin Cases.
2014;2(10):507–14.
111. Bostwick J 3rd, Pendergrast WJ Jr, Vasconez
LO.Marjolin’s ulcer: an immunologically privileged
tumor? Plast Reconstr Surg. 1976;57:66–9.
112. Kerr-Valentic M, Samimi K, Rohlen B, et al.
Marjolin’s ulcer: modern analysis of an ancient
problem. Plast Reconstr Surg. 2009;123:184–91.
113. Treves N, Pack GT.The development of cancer in
burn scar: an analysis and report of thirty-four cases.
Surg Gynecol Obstet. 1930;58:749–51.
114. Bazaliński D, Przybek-Mita J, Barańska B, Więch
P. Marjolin’s ulcer in chronic wounds—review
of available literature. Contemp Oncol (Pozn).
2017;21(3):197–202.
115. Koval-Vern A, Criswell BK. Burn scar neoplasm:
a literature review and statistical analysis. Burns.
2005;31:403–13.
116. Zieliński T, Lewandowska M. Owrzodzenie
Marjolina– nowo- twór złośliwy rozwijający się na
podłożu przewlekłych owrzodzeń i blizn. Analiza 8
przypadków [Marjolin’s ulcer—malignancy developing in chronic ulcers and scars. Analysis of 8
cases]. Przegl Dermatol. 2010;97:38–42.
117. Weedon D. Skin pathology. 3rd ed. London:
Churchill Livingstone; 2009.
118. Byrom L, Barksdale S, Weedon D, Muir J.Unstable
solar lentigo: a dened separate entity. Australas J
Dermatol. 2016;57:229–34.
119. Kasprzak JM, Xu YG. Diagnosis and management of lentigo maligna: a review. Drugs Context.
2015;4:212281.
120. Holm-Schou AS, Philipsen PA, Idorn LW, et al.
Lifetime UVR dose and skin cancer risk, determined by their common relation to solar lentigines.
Anticancer Res. 2020;40(1):557–64.
121. Kinsler V, Shaw AC, Merks JH, etal. The face in
congenital melanocytic nevus syndrome. Am J Med
Genet A. 2012;158A:1014–9.
122. Kinsler VA, Thomas AC, Ishida M, etal. Multiple
congenital melanocytic nevi and neurocutaneous melanosis are caused by postzygotic mutations in codon 61 of NRAS. J Invest Dermatol.
2013;133:2229–36.

352
Ö. F. Dilek et al.
123. Krengel S, Hauschild A, Schafer T. Melanoma
risk in congenital melanocytic naevi: a systematic
review. Br J Dermatol. 2006;155:1–8.
124. Kinsler VA, Chong WK, Aylett SE, et al.
Complications of congenital melanocytic naevi in
children: analysis of 16 years’ experience and clinical practice. Br J Dermatol. 2008;159:907–14.
125. Ka VS, Dusza SW, Halpern AC, etal. The association between large congenital melanocytic naevi and
cutaneous melanoma: preliminary ndings from an
Internet-based registry of 379 patients. Melanoma
Res. 2005;15:61–7.
126. Vourc’h-Jourdain M, Martin L, Barbarot S, et al.
Large congenital melanocytic nevi: therapeutic management and melanoma risk: a systematic review. J
Am Acad Dermatol. 2013;68:493–8.e1-14.
127. Price HN. Congenital melanocytic nevi: update
in genetics and management. Curr Opin Pediatr.
2016;28(4):476–82.
128. Marghoob AA, Agero ALC, Benvenuto-Andrade
C, etal. Large congenital melanocytic nevi, risk of
cutaneous melanoma, and prophylactic surgery. J
Am Acad Dermatol. 2006;54:868–70.
129. Allen AC, Spitz S.Malignant melanoma: a clinicopathological analysis or the criteria for diagnosis and
prognosis. Cancer. 1953;6:1–45.
130. Kachare SD, Agle SC, Englert ZP, et al. Malignant
blue nevus: clinicopathologically similar to melanoma. Am Surg. 2013;79:651–6.
131. Martin RCW, Murali R, Scolyer RA, etal. So-called
malignant blue nevus. Cancer. 2009;115:2949–55.
132. Sugianto JZ, Ralston JS, Metcalf JS, et al. Blue
nevus & “malignant blue nevus:” a concise review.
Semin Diagn Pathol. 2016;33(4):219–24.
133. Zembowicz A. Blue nevi and related tumors. Clin
Lab Med. 2017;37(3):401–15.
134. Sainz-Gaspar L, Sánchez-Bernal J, Noguera-Morel
L, et al. Spitz nevus and other spitzoid tumors in
children. Part 2: cytogenetic and molecular features.
Prognosis and treatment. Actas Dermosiliogr.
2020;111(1):20–5.
135. Menezes FD, Mooi WJ.Spitz tumors of the skin.
Surg Pathol Clin. 2017;10(2):281–98.
136. Crowson AN, Magro CM, Mihm MC.The melanocytic proliferations: a comprehensive textbook of
pigmented lesions. New York: Wiley-Liss; 2001.
p.348.
137. Massi G, LeBoit P.Spitzoid melanoma. Histological
diagnosis of nevi and melanoma. Heidelberg:
Steinkopff-Verlag Darmstadt; 2004. p.463–86.
138. Gelbard SN, Tripp JM, Marghoob AA, et al.
Management of Spitz nevi: a survey of dermatologists in the United States. J Am Acad Dermatol.
2002;47:224–30.
139. Argenziano G, Agozzino M, Bonifazi E, et al.
Natural evolution of Spitz nevi. Dermatology.
2011;222:256–60.
140. Lallas A, Apalla Z, Ioannides D, et al. Update on
dermoscopy of Spitz/Reed naevi and management
guidelines by the International Dermoscopy Society.
Br J Dermatol. 2017;177:645–55.
141. Nino M, Brunetti B, Delno S, et al. Spitz nevus:
follow-up study of 8 cases of childhood starburst
type and proposal for management. Dermatology.
2009;218:48–51.
142. Kelley SW, Cockerell CJ. Sentinel lymph node
biopsy as an adjunct to management of histologically difcult to diagnose melanocytic lesions: a
proposal. J Am Acad Dermatol. 2000;42:527–30.
143. Kopf AW, Morrill SD, Silberberg I. Broad spectrum of leukoderma acquisitum centrifugum. Arch
Dermatol. 1965;92:14–33; discussion 33–5.
144. Aouthmany M, Weinstein M, Zirwas MJ, etal. The
natural history of halo nevi: a retrospective case
series. J Am Acad Dermatol. 2012;67:582–6.
145. Toussaint S, Kamino H.Dysplastic changes in different types of melanocytic nevi. A unifying concept. J Cutan Pathol. 1999;26:84–90.
146. Weyant GW, Chung CG, Helm KF. Halo nevus:
review of the literature and clinicopathologic ndings. Int J Dermatol. 2015;54(10):e433–5.
147. Epstein WL, Sagebeil R, Spitler L, etal. Halo nevi
and melanoma. JAMA. 1973;225:373–7.
148. Patel P, Malik K, Khachemoune A.Sebaceus and
Becker’s nevus: overview of their presentation,
pathogenesis, associations, and treatment. Am J Clin
Dermatol. 2015;16(3):197–204.
149. Domingo J, Helwig EB.Malignant neoplasm associated with nevus sebaceus of Jadassohn. J Am Acad
Dermatol. 1979;1:54556.
150. Santibanez-Gallerani A, Marshall D, Duarte AM,
etal. Should nevus sebaceus of Jadassohn in children be excised? A study of 757 cases, and literature
review. J Craniofac Surg. 2003;14:658–60.
151. Aslam A, Salam A, Grifths CE, McGrath
JA.Naevus sebaceous: a mosaic RASopathy. Clin
Exp Dermatol. 2014;39(1):1–6.
152. Rook A, Burns T.Rook’s textbook of dermatology.
8th ed. Wiley-Blackwell: Chichester; 2010.
153. Cribier B, Scrivener Y, Grosshans E.Tumors arising
in nevus sebaceus: a study of 596 cases. J Am Acad
Dermatol. 2000;42(2 Pt 1):263–8.
154. Costa C, Scalvenzi M, Ayala F, etal. How to treat
actinic keratosis? An update. J Dermatol Case Rep.
2015;9(2):29–35.
155. Quist SR, Gollnick HP. Imiquimod 3.75% cream
(Zyclara) for the treatment of actinic keratoses.
Expert Opin Pharmacother. 2011;12(3):451–61.
156. Berman B, Cockerell CJ. Pathobiology of actinic
keratosis: ultraviolet-dependent keratinocyte proliferation. J Am Acad Dermatol. 2013;68:S10–9.
157. de Oliveira ECV, da Motta VRV, Pantoja PC, etal.
Actinic keratosis—review for clinical practice. Int J
Dermatol. 2019;58(4):400–7.
158. Stocketh E.From a new vision of actinic keratosis
to imiquimod 3.75%, the new treatment standard.
J Eur Acad Dermatol Venereol. 2015;29(Suppl
1):1–2.

29 Prophylactic Surgical Procedures inPlastic Surgery
Данная книга находится в списке для перевода на русский язык сайта https://meduniver.com/
353
159. Ferrandiz C. Update on actinic keratosis in clinical trial experience with imiquimod. Br J Dermatol.
2007;157(Suppl 2):32–3.
160. Fleming P, Zhou S, Bobotsis R, Lynde C.Comparison
of the treatment guidelines for actinic keratosis:
a critical appraisal and review. J Cutan Med Surg.
2017;21(5):408–17.
161. Dianzani C, Conforti C, Giuffrida R, etal. Current
therapies for actinic keratosis. Int J Dermatol.
2020;59(6):677–84. https://doi.org/10.1111/
ijd.14767
.
162. Schwartz RA. Keratoacanthoma. J Am Acad
Dermatol. 1994;30:1–19; quiz 20–22.
163. Mandrell JC, Santa Cruz D.Keratoacanthoma: hyperplasia, benign neoplasm, or a type of squamous cell
carcinoma? Semin Diagn Pathol. 2009;26:150–63.
164. Kwiek B, Schwartz RA. Keratoacanthoma (KA):
an update and review. J Am Acad Dermatol.
2016;74(6):1220–33.
165. Kiss N, Avci P, Bánvölgyi A, etal. Intralesional therapy for the treatment of keratoacanthoma. Dermatol
Ther. 2019;32(3):e12872.
166. Morton CA, Birnie AJ, Eedy DJ.British Association
of Dermatologists’ guidelines for the management
of squamous cell carcinoma in situ (Bowen’s disease) 2014. Br J Dermatol. 2014;170(2):245–60.
167. McGregor JM, Proby CM.The role of papillomaviruses in human non-melanoma skin cancer. Cancer
Surv. 1996;26:219–36.
168. Mitsuishi T, Kawana S, Kato T, Kawashima
M. Human papilloma virus infection in actinic
keratosis and bowen’s disease: comparative study
with expression of cell-cycle regulatory proteins p21 (Waf1/Cip1), p53, PCNA, Ki-67, and
Bcl-2in positive and negative lesions. Hum Pathol.
2003;34:886–92.
169. Peterka ES, Lynch FW, Goltz RW. An association
between Bowen’s disease and internal cancer. Arch
Dermatol. 1961;84:623–9.
170. Kao GF. Carcinoma arising in Bowen’s disease.
Arch Dermatol. 1986;122:1124–6.
171. Porter WM, Francis N, Hawkins D, et al. Penile
intraepithelial neo plasia: clinical spectrum and treatment of 35 cases. Br J Dermatol. 2002;147:1159–65.
172. Papadopoulos AJ, Schwartz RA, Lefkowitz A, etal.
Extragenital bowenoid papulosis associated with
atypical human papillomavirus genotypes. J Cutan
Med Surg. 2002;6:117–21.
173. Cox NH, Eedy DJ, Morton CA. Guidelines for
management of Bowen’s disease. Br J Dermatol.
1999;141:633–4.
174. Bunker CB.Topics in penile dermatology. Clin Exp
Dermatol. 2001;26:469–79.
175. Stamm AW, Kobashi KC, Stefanovic KB.Urologic
dermatology: a review. Curr Urol Rep. 2017;18(8):62.
176. Henquet CJ. Anogenital malignancies and premalignancies. J Eur Acad Dermatol Venereol.
2011;25(8):885–95.
177. Schiffman M, Kjaer SK.Natural history of anogenital human papillomavirus infection and neoplasia. J
Natl Cancer Inst Monogr. 2003;31:14–9.
178. Dupin N. Genital warts. Clin Dermatol.
2004;22(6):481–6.
179. Shabbir M, Minhas S, Muneer A. Diagnosis and
management of premalignant penile lesions. Ther
Adv Urol. 2011;3:151–8.
180. Majewski S, Jablonska S. Human papillomavirusassociated tumors of the skin and mucosa. J Am
Acad Dermatol. 1997;36:659–85.
181. Spinu D, Rădulescu A, Bratu O, etal. Giant condyloma acuminatum - Buschke-Lowenstein disease – a literature review. Chirurgia (Bucur).
2014;109(4):445–50.
182. Fathi R, Tsoukas MM.Genital warts and other HPV
infections: established and novel therapies. Clin
Dermatol. 2014;32(2):299–306.
183. Castellsagué X, Bosch FX, Munoz N, etal. Male
circumcision, penile human papillomavirus infection, and cervical cancer in female partners. N Engl
J Med. 2002;346:1105–12.
184. Villa A, Sonis S.Oral leukoplakia remains a challenging condition. Oral Dis. 2018;24(1–2):179–83.
185. Villa A, Woo SB. Leukoplakia—a diagnostic and
management algorithm. J Oral Maxillofac Surg.
2017;75(4):723–34.
186. Wong L, Spence RJ. Escharotomy and fasciotomy of the burned upper extremity. Hand Clin.
2000;16(2):165–vii.
187. Zhang L, Hughes PG.Escharotomy. In: StatPearls.
Treasure Island: StatPearls Publishing; 2019.
188. Orgill DP, Piccolo N. Escharotomy and decompressive therapies in burns. J Burn Care Res.
2009;30(5):759–68.
189. Kupas DF, Miller DD.Out-of-hospital chest escharotomy: a case series and procedure review. Prehosp
Emerg Care. 2010;14(3):349–54.
190. Ipaktchi K, Wingeld J, Colakoglu S.Fasciotomy:
upper extremity. In: Mauffrey C, Hak DJ, Martin
III MP, editors. Compartment syndrome: a guide to
diagnosis and management. Cham: Springer; 2019.
p.59–66.
191. Higgins JP. Ectopic banking of amputated
parts: a clinical review. J Hand Surg Am.
2011;36(11):1868–76.
192. Godina M, Bajec J, Baraga A. Salvage of the
mutilated upper extremity with temporary ectopic
implantation of the undamaged part. Plast Reconstr
Surg. 1986;78(3):295–9.
193. Tu Y, Lineaweaver WC, Culnan DM, et al.
Temporary ectopic implantation for salvaging
amputated parts: a systematic review. J Trauma
Acute Care Surg. 2018;84(6):985–93.
194. DiSipio T, Rye S, Newman B, Hayes S.Incidence of
unilateral arm lymphoedema after breast cancer: a
systematic review and meta-analysis. Lancet Oncol.
2013;14:500–15.

354
Ö. F. Dilek et al.
195. Erickson VS, Pearson ML, Ganz PA, et al. Arm
edema in breast cancer patients. J Natl Cancer Inst.
2001;93:96–111.
196. Harris SR, Hugi MR, Olivotto IA, Levine M.Steering
Committee for Clinical Practice Guidelines for the
care and treatment of breast cancer. Clinical practice
guidelines for the care and treatment of breast cancer: 11. Lymphedema. CMAJ. 2001;164:191–9.
197. Lee TS, Kilbreath SL, Refshauge KM, et al.
Prognosis of the upper limb following surgery and
radiation for breast cancer. Breast Cancer Res Treat.
2008;110:19–37.
198. Shah C, Vicini FA.Breast cancer-related arm lymphedema: incidence rates, diagnostic techniques, optimal management and risk reduction strategies. Int J
Radiat Oncol Biol Phys. 2011;81:907–14.
199. Warren AG, Brorson H, Borud LJ, Slavin
SA. Lymphedema: a comprehensive review. Ann
Plast Surg. 2007;59:464–72.
200. Maunsell E, Brisson J, Deschenes L.Arm problems
and psychological distress after surgery for breast
cancer. Can J Surg. 1993;36:315–20.
201. McWayne J, Heiney SP. Psychologic and social
sequelae of secondary lymphedema: a review.
Cancer. 2005;104:457–66.
202. Passik SD, McDonald MV. Psychosocial aspects
of upper extremity lymphedema in women
treated for breast carcinoma. Cancer. 1998;83(12
Suppl):2817–20.
203. Box RC, Reul-Hirche HM, Bullock-Saxton JE,
Furnival CM. Physiotherapy after breast cancer
surgery: results of a randomized controlled study to
minimize lymphoedema. Breast Cancer Res Treat.
2002;75:51–64.
204. Torres Lacomba M, Yuste Sanchez MJ, Zapico Goni
A, et al. Effectiveness of early physiotherapy to
prevent lymphoedema after surgery for breast cancer: randomised, single blinded, clinical trial. BMJ.
2010;340:b5396.
205. Zimmermann A, Wozniewski M, Szklarska A, etal.
Efcacy of manual lymphatic drainage in preventing
secondary lymphedema after breast cancer surgery.
Lymphology. 2012;45:103–12.
206. Feldman S, Bansil H, Ascherman J, et al. Single
institution experience with lymphatic microsurgical
preventive healing approach (LYMPHA) for the primary prevention of lymphedema. Ann Surg Oncol.
2015;22(10):3296–301.
207. Giuliano AE, Hunt KK, Ballman KV, etal. Axillary
dissection vs no axillary dissection in women
with invasive breast cancer and sentinel node
metastasis: a randomized clinical trial. JAMA.
2011;305(6):569–75.
208. Ochoa D, Klimberg VS.Surgical strategies for prevention and treatment of lymphedema in breast cancer patients. Curr Breast Cancer Rep. 2015;7(1):1–7.
209. Mamounas EP, Kuehn T, Rutgers EJT, von
Minckwitz G. Current approach of the axilla in
patients with early-stage breast cancer. Lancet. 2017
Aug 14:S0140–6736(17)31451–4.
210. Thompson M, Korourian S, Henry-Tillman R, et al.
Axillary reverse mapping (ARM): a new concept to
identify and enhance lymphatic preservation. Ann
Surg Oncol 2007;14:1890–5.
211. Tummel E, Ochoa D, Korourian S, etal. Does axillary reverse mapping prevent lymphedema after
lymphadenectomy. Ann Surg. 2017;265(5):987–92.
212. Boneti C, Korourian S, Bland K, et al. Axillary
reverse mapping: mapping and preserving arm lymphatics may be important in preventing lymphedema
during sentinel lymph node biopsy. J Am Coll Surg.
2008;206:1038–42.
213. Boneti C, Korourian S, Diaz Z, et al. Scientic
Impact Award: axillary reverse mapping (ARM)
to identify and protect lym- phatics draining the
arm during axillary lymphadenectomy. Am J Surg.
2009;198:482–7.
214. Thompson M, Korourian S, Henry-Tillman R, etal.
Axillary reverse mapping (ARM): a new concept to
identify and enhance lymphatic preservation. Ann
Surg Oncol. 2007;14:1890–5.
215. Gennaro M, Maccauro M, Sigari C, et al. Selective
axillary dissection after axillary reverse mapping to
prevent breast-cancer-related lymphoedema. Eur J
Surg Oncol. 2013;39(12):1341–5.
216. Nos C, Kaufmann G, Clough KB, etal. Combined
axillary reverse mapping (ARM) technique for
breast cancer patients requiring axillary dissection.
Ann Surg Oncol. 2008;15(9):2550–5.
217. Yue T, Zhuang D, Zhou P, etal. A prospective study
to assess the feasibility of axillary reverse mapping and evaluate its effect on preventing lymphedema in breast cancer patients. Clin Breast Cancer.
2015;15(4):301–6.
218. Nos C, Clough KB, Bonnier P, et al. Upper outer
boundaries of the axillary dissection. Result of the
SENTIBRAS protocol: multicentric protocol using
axillary reverse mapping in breast cancer patients
requiring axillary dissection. Eur J Surg Oncol.
2016;42(12):1827–33.
219. Bedrosian I, Babiera GV, Mittendorf EA, et al. A
phase I study to assess the feasibility and oncologic
safety of axillary reverse mapping in breast cancer
patients. Cancer. 2010;116(11):2543–8.
220. Connor C, McGinness M, Mammen J, etal. Axillary
reverse mapping: a prospective study in women with
clinically node negative and node positive breast
cancer. Ann Surg Oncol. 2013;20(10):3303–7.
221. Rubio IT, Cebrecos I, Peg V, et al. Extensive
nodal involvement increases the positivity of
blue nodes in the axillary reverse mapping procedure in patients with breast cancer. J Surg Oncol.
2012;106(1):89–93.
222. Schunemann E Jr, Dória MT, Silvestre JB, et al.
Prospective study evaluating oncological safety
of axillary reverse mapping. Ann Surg Oncol.
2014;21(7):2197–202.
223. Gallagher KK, Lopez M, Iles K, Kugar M.Surgical
approach to lymphedema reduction. Curr Oncol
Rep. 2020;22(10):97.

29 Prophylactic Surgical Procedures inPlastic Surgery
Данная книга находится в списке для перевода на русский язык сайта https://meduniver.com/
355
224. Boccardo F, Casabona F, De Cian F, et al.
Lymphedema microsurgical preventive healing
approach: a new technique for primary prevention
of arm lymphedema after mastectomy. Ann Surg
Oncol. 2009;16(3):703–8.
225. Boccardo F, Casabona F, De Cian F, etal. Lymphatic
microsurgical preventing healing approach
(LYMPHA) for primary surgical prevention of breast
cancer-related lymphedema: over 4 years follow up [published correction appears in Microsurgery.
2015; 35(1):83. DeCian, Franco [corrected to De
Cian, Franco]]. Microsurgery. 2014;34(6):421–4.
226. Ozmen T, Lazaro M, Zhou Y, etal. Evaluation of
simplied lymphatic microsurgical preventing
healing approach (S-LYMPHA) for the prevention of breast cancer-related clinical lymphedema
after axillary lymph node dissection. Ann Surg.
2019;270(6):1156–60.
227. Kruger E, Thomson WM, Konthasinghe P. Third
molar outcomes from age 18 to 26: ndings from
a population-based New Zealand longitudinal study.
Oral Surg Oral Med Oral Pathol Oral Radiol Endod.
2001;92(2):150–5.
228. Venta I, Turtola L, Ylipaavalniemi P. Change in
clinical status of third molars in adults during
12 years of observation. J Oral Maxillofac Surg.
1999;57(4):386–91.
229. Carter K, Worthington S. Morphologic and demographic predictors of third molar agenesis: a systematic review and meta-analysis. J Dent Res.
2015;94(7):886–94.
230. Dodson TB. How many patients have third molars
and how many have one or more asymptomatic,
disease-free third molars? J Oral Maxillofac Surg.
2012;70(9):4–7.
231. Friedman JW. Containing the costs of third molar
surgery: a dilemma for health insurance. Public
Health Rep. 1983;98:379–84.
232. Friedman JW.The prophylactic extraction of third
molars: a public health hazard. Am J Public Health.
2007;97:1554–9.
233. Song F, Landes DP, Glenny AM, etal. Prophylactic
removal of impacted third molars: an assessment of
published reviews. Br Dent J. 1997;182:339–46.
234. Costa MG, Pazzini CA, Pantuzo MC, et al. Is
there justication for prophylactic extraction of
third molars? A systematic review. Braz Oral Res.
2013;27:183–8.
235. Song F, O’Meara S, Wilson P, et al. The effectiveness
and cost-effectiveness of prophylactic removal of wisdom teeth. Health Technol Assess. 2000;4(15):1–55.
236. Stordeur S, Eyssen M. Prophylactic removal of
pathology-free wisdom teeth: rapid assessment.
Belgian Health Care Knowledge Centre: Brussels;
2012.
237. Canadian Agency for Drugs and Technologies in
Health (CADTH). Prophylactic removal of wisdom
teeth: a review of the clinical benet and guidelines. Ottawa: CADTH; 2010. https://www.cadth.
ca/prophylactic- removal- wisdom- teeth- review-
clinical- benet- and- guidelines- 0. Accessed 5 Aug
2020.
238. Suska F, Kjeller G, Molander A, et al. Removal of
impacted wisdom teeth. Gothenburg: The Regional
Health Technology Assessment Centre (HTAcentrum); 2010.
239. Bouloux GF, Busaidy KF, Beirne OR, etal. What is
the risk of future extraction of asymptomatic third
molars? A systematic review. J Oral Maxillofac
Surg. 2015;73:806–11.
240. Mettes TD, Ghaeminia H, Nienhuijs ME, et al.
Surgical removal versus retention for the management of asymptomatic impacted wisdom teeth.
Cochrane Database Syst Rev. 2012;6:CD003879.
241. Hounsome J, Pilkington G, Mahon J, et al.
Prophylactic removal of impacted mandibular third
molars: a systematic review and economic evaluation. Health Technol Assess. 2020;24(30):1–116.
242. Ghaeminia H, Nienhuijs ME, Toedtling V, et al.
Surgical removal versus retention for the management of asymptomatic disease-free impacted
wisdom teeth. Cochrane Database Syst Rev.
2020;5(5):CD003879.
243. Finnish Current Care Guidelines 2014. Working
group set up by the Finnish Medical Society
Duodecim and the Finnish Dental Society Apollonia.
Third Molar. https://www.kaypahoito./hoi50074.
Accessed 20 Aug 2020.
244. Dutch Clinical Care Guidelines 2020. Third Molar
[Derde molaar]. www.hetkimo.nl/richtlijnen/derde- -
molaar/introductie/. Accessed 20 Aug 2020.
245. American Association of Oral and Maxillofacial
Surgeons. www.aaoms.org/images/uploads/pdfs/
evidence_based_management_third_molars.pdf
(Cited August 20, 2020).
246. Zhou H, Lv K, Yang R, et al. Mechanics in the
production of mandibular fractures: a clinical retrospective case-control study. PLoS One.
2016;11:e0149553.
247. Sawazaki R, Junior SM, Asprino L, etal. Incidence
and patterns of mandibular condyle fractures. J Oral
Maxillofac Surg. 2010;68:1252.
248. Armond ACV, Martins CC, Gloria JCR, et al.
Inuence of third molars in mandibular fractures.
Part 1: mandibular angle—a meta-analysis. Int J
Oral Maxillofac Surg. 2017;46:716.
249. Armond ACV, Martins CC, Gloria JCR, et al.
Inuence of third molars in mandibular fractures.
Part 2: mandibular condyle—a meta-analysis. Int J
Oral Maxillofac Surg. 2017;46:730.
250. Meisami T, Sojat A, Sandor GK, et al. Impacted
third molars and risk of angle fracture. Int J Oral
Maxillofac Surg. 2002;31:140–4.
251. Schwimmer A, Stern R, Kritchman D.Impacted third
molars: a contributing factor in mandibular fractures
in contact sports. Am J Sports Med. 1983;11:262–6.
252. Tevepaugh DB, Dodson TB.Are mandibular third
molars a risk factor for angle fractures? A retrospective cohort study. J Oral Maxillofac Surg.
1995;53:646–9.

356
Ö. F. Dilek et al.
253. Xu S, Huang JJ, Xiong Y, Tan YH.How is third molar
status associated with the occurrence of mandibular
angle and condyle fractures? J Oral Maxillofac Surg.
2017;75:1476.
254. Mehra A, Anehosur V, Kumar N. Impacted mandibular third molars and their inuence on mandibular angle and condyle fractures. Craniomaxillofac
Trauma Reconstr. 2019;12:291.
255. Tiwari A, Lata J, Mishra M. Inuence of the
impacted mandibular third molars on fractures of the
mandibular angle and condyle—a prospective clinical study. J Oral Biol Craniofac. 2016;6:227.
256. Antic S, Saveljic I, Nikolic D, etal. Does the presence of anv unerupted lower third molar inuence
the risk of mandibular angle and condylar fractures?
Int J Oral Maxillofac Surg. 2016;45:588.
257. Anderl H.Reconstruction of the face through crossface nerve transplantation in facial paralysis. Chir
Plast. 1973;2:17.
258. Anderl H. Cross-face nerve grafting: up to 12
months of seventh nerve disruption. In: Rubin LR,
editor. Reanimation of the paralyzed face. St. Louis:
Mosby; 1977. p.241.
259. Anderl H. Cross-facial nerve transplant. Clin Plast
Surg. 1979;6:433.
260. Terzis JK, Tzafetta K. The “babysitter” procedure: minihypoglossal to facial nerve transfer and
cross-facial nerve grafting. Plast Reconstr Surg.
2009;123(3):865–76.
261. Terzis JK. ‘Babysitters’: an exciting new concept in
facial reanimation. The facial nerve. In: Castro D,
editor. Proceedings of the sixth international symposium on the facial nerve, Rio de Janeiro, Brazil,
October 2–5, 1988. Amsterdam: Kugler & Ghedini;
1990. p.525.
262. Mersa B, Tiangco DA, Terzis JK. Efcacy of the
“babysitter” procedure after prolonged denervation.
J Reconstr Microsurg. 2000;16:27–35.
263. May M, Schaitkain BM.History of facial nerve surgery. Facial Plast Surg. 2000;16:301–7.
264. Manni JJ, Beurskens CHG, van de Velde C, Stokroos
RJ. Reanimation of the paralyzed face by indirect
hypoglossal-facial nerve anastomosis. Am J Surg.
2001;182:268–73.
265. Koh KS, Kim J, Kim CJ, Kwun BD, Kim
S. Hypoglossal-facial crossover in facial nerve
palsy: pure end-to-side anastomosis technique. Br J
Plast Surg. 2002;55:25–31.
266. Spira M.Anastomosis of masseteric nerve to lower
division of facial nerve for correction of lower facial
paralysis. Preliminary report. Plast Reconstr Surg.
1978;61:330–4.
267. Biglioli F, Frigerio A, Colombo V, etal. Massetericfacial nerve anastomosis for early facial reanimation. J Craniomaxillofac Surg. 2012;40:149–55.
268. Endo T, Hata J, Nakayama Y. Variations on the
“babysitter” procedure for reconstruction of facial
paralysis. J Reconstr Microsurg. 2000;16:37–43.
269. Liu HF, Chen ZG, Lineaweaver WC, Zhang F.Can
the babysitter procedure improve nerve regenera-
tion and denervated muscle atrophy in the treatment
of peripheral nerve injury? Plast Reconstr Surg.
2016;138(1):122–31.
270. Wang Y, Meng D, Zhang J, et al. Efcacy and
safety of the babysitter procedure with different
percentages of partial neurectomy. Ann Plast Surg.
2017;79(3):286–92.
271. Beck-Broichsitter BE, Becker ST, Lamia A, et al.
Sensoric protection after median nerve injury:
babysitter-procedure prevents muscular atrophy
and improves neuronal recovery. Biomed Res Int.
2014;2014:724197.
272. Post R, de Boer KS, Malessy MJ.Outcome following nerve repair of high isolated clean sharp injuries
of the ulnar nerve. PLoS One. 2012;7(10):e47928.
273. Li Q, Zhang P, Yin X, Jiang B.Early nerve protection with anterior interosseous nerve in modied end-to-side neurorrhaphy repairs high ulnar
nerve injury: a hypothesis of a novel surgical
technique. Artif Cells Nanomed Biotechnol.
2015;3(2):103–5.
274. Giuffre JL. Anterior Interosseous-to-Ulnar Motor
Nerve Transfers: A Single Center’s Experience in
Restoring Intrinsic Hand Function. Hand (NY).
2020 Jul 22:1558944720928482.
275. Spector TD, Hart DJ, Powell RJ.Prevalence of rheumatoid arthritis and rheumatoid factor in women:
evidence for a secular decline. Ann Rheum Dis.
1993;52:254–7.
276. Abe A, Ishikawa H, Murasawa A, et al. Extensor
tendon rupture and three- dimensional computed
tomography imaging of the rheumatoid wrist.
Skeletal Radiol. 2010;39:325–31.
277. McQueen F, Beckley V, Crabbe J, et al. Magnetic
resonance imaging evidence of tendinopathy in early
rheumatoid arthritis predicts tendon rupture at six
years. Arthritis Rheum. 2005;52:744–51.
278. Ishikawa H, Abe A, Murasawa A, etal. Rheumatoid
wrist deformity and risk of extensor tendon rupture evaluated by 3DCT imaging. Skeletal Radiol.
2010;39:467–72.
279. Seki E, Ishikawa H, Murasawa A, etal. Dislocation of
the extensor carpi ulnaris tendon in rheumatoid wrists
using three-dimensional computed tomographic imaging. Clin Rheumatol. 2013;32:1627–32.
280. Ryu J, Saito S, Honda T, et al. Risk factors and
prophylactic tenosynovectomy for extensor tendon
ruptures in the rheumatoid hand. J Hand Surg Br.
1998;23:658–61.
281. Hsueh JH, Liu WC, Yang KC, etal. Spontaneous
extensor tendon rupture in the rheumatoid wrist: risk
factors and preventive role of extended tenosynovectomy. Ann Plast Surg. 2016;76(Suppl 1):S41–7.
282. Björkman A, Jörgsholm P. Rupture of the extensor pollicis longus tendon: a study of aetiological
factors. Scand J Plast Reconstr Surg Hand Surg.
2004;38(1):32e35.
283. Rada EM, Shridharani SM, Lifchez SD.Spontaneous
atraumatic extensor pollicis longus rupture in the
nonrheumatoid population. Eplasty. 2013;13:e11.

29 Prophylactic Surgical Procedures inPlastic Surgery
Данная книга находится в списке для перевода на русский язык сайта https://meduniver.com/
357
284. Barnes CK.Spontaneous rupture of the extensor pollicis longus. JAMA. 1926;87(9):663.
285. Kim CH.Spontaneous rupture of the extensor pollicis
longus tendon. Arch Plast Surg. 2002;39(6):680–2.
286. Choi JC, Kim WS, Na HY, etal. Spontaneous rupture of the extensor pollicis longus tendon in a tailor.
Clin Orthop Surg. 2011;3(2):167–9.
287. Dawson J. Sports-induced spontaneous rupture of
the extensor pollicis longus tendon. J Hand Surg
Am. 1992;17(3):457–8.
288. Fujita N, Doita M, Yoshikawa M, etal. Spontaneous
rupture of the extensor pollicis longus tendon in a
professional skier. Knee Surg Sports Traumatol
Arthrosc. 2005;13(6):489–91.
289. Perrugia D, Ciurluini M, Ferretti A. Spontaneous
rupture of the extensor pollicis tendon in a young
goalkeeper: a case report. Scand J Med Sci Sports.
2009;19(2):257e259.
290. Navaratnam AV, Ball S, Eckersley
R. Prophylactic decompression of extensor pollicis longus to prevent rupture. BMJ Case Rep.
2013;2013:bcr2013010196.
291. Zinger G, Dalu KA, Bregman A, Yudkevich
G.Spontaneous rupture of the extensor pollicis longus tendon with repair and contralateral prophylactic
decompression: a case report and review of the literature. J Hand Surg Am. 2019;44(8):702.e1–5.
292. Kostrubala JG, Greeley PW. The problem of decubitus ulcers in paraplegics. Plast Reconstr Surg.
1947;2:403–12.
293. Arregui J, Cannon B, Murray JE, O’Leary JJ Jr.
Long-term evaluation of ischiectomy in the treatment of pressure ulcers. Plast Reconstr Surg.
1965;36:583–90.
294. Hackler RH, Zampieri TA. Urethral complications
following ischiectomy in spinal cord injury patients:
a urethral pressure study. J Urol. 1987;137:253–5.
295. Karaca AR, Binns JH, Blumenthal
FS.Complications of total ischiectomy for the treatment of ischial pressure sores. Plast Reconstr Surg.
1978;62:96–9.
296. Levi R, Hultling C, Seiger A.The stockholm spinal
cord injury study: 2. Associations between clinical
patient characteristics and post-acute medical problems. Paraplegia. 1995;33:585–94.
297. Foerster. Resection of the posterior spinal nerveroots in the treatment of gastric crises and spastic
paralysis. Proc R Soc Med. 1911;4:254.
298. Munro D. Anterior-rootlet rhizotomy; a method
of controlling spasm with retention of voluntary
motion. N Engl J Med. 1952;246:161–6.
299. Putty TK, Shapiro SA.Efcacy of dorsal longitudinal myelotomy in treating spinal spasticity: a review
of 20 cases. J Neurosurg. 1991;75:397–401.
300. Tonnis W, Bischof W. Results of lumbar myelotomy by the Bischof technic. Zentralbl Neurochir.
1962;23:29–36.
301. Boulton AJ, Vileikyte I, Ragnarson-Tennvall G,
Apelqvist J.The global burden of diabetic foot disease. Lancet. 2005;366:1719–24.
302. Singh N, Armstrong DG, Lipsky BA. Preventing
foot ulcers in patients with diabetes. JAMA.
2005;293:217–28.
303. Apelqvist J, Larsson J, Agardh CD.Long-term prognosis for diabetic patients with foot ulcers. J Intern
Med. 1993;233:485–91.
304. Allan J, Munro W, Figgins E. Foot deformities
within the diabetic foot and their inuence on biomechanics: a review of the literature. Prosthet Orthot
Int. 2016;40(2):182–92.
305. Ahmad J.The diabetic foot. Diabetes Metab Syndr.
2016;10(1):48–60.
306. Delbridge L, Perry P, Marr S, et al. Limited joint
mobility in the diabetic foot: relationship to neuropathic ulceration. Diabet Med. 1988;5(4):333–7.
307. van Schie CH. A review of the biomechanics
of the diabetic foot. Int J Low Extrem Wounds.
2005;4(3):160–70.
308. van Netten JJ, Price PE, Lavery LA, etal. Prevention
of foot ulcers in the at-risk patient with diabetes: a systematic review. Diabetes Metab Res Rev.
2016;32(Suppl 1):84–98.
309. Dellon AL.A cause for optimism in diabetic neuropathy. Ann Plast Surg. 1988;20(2):103–5.
310. Dellon AL. Treatment of symptomatic diabetic
neuro-pathy by surgical decompression of multiple peripheral nerves. Plast Reconstr Surg.
1992;89(4):689–97; discussion 689–698.
311. Aszmann OC, Kress KM, Dellon AL. Results of
decompression of peripheral nerves in diabetics:
a prospective, blinded study. Plast Reconstr Surg.
2000;106(4):816–22.
312. Aszmann O, Tassler PL, Dellon AL.Changing the
natural history of diabetic neuropathy: incidence of
ulcer/amputation in the contralateral limb of patients
with a unilateral nerve decompression procedure.
Ann Plast Surg. 2004;53(6):517–22.
313. Wood WA, Wood MA, Werter SA, et al. Testing
for loss of protective sensation in patients with foot
ulceration: a cross-sectional study. J Am Podiatr
Med Assoc. 2005;95(5):469–74.
314. Dellon AL, Muse VL, Nickerson DS, et al.
Prevention of ulceration, amputation, and reduction of hospitalization: outcomes of a prospective
multicenter trial of tibial neurolysis in patients
with diabetic neuropathy. J Reconstr Microsurg.
2012;28(4):241–6.
315. Dellon AL, Muse VL, Scott ND, et al. A positive
Tinel sign as predictor of pain relief or sensory
recovery after decompression of chronic tibial nerve
compression in patients with diabetic neuropathy. J
Reconstr Microsurg. 2012;28(4):235–40.
316. Siemionow M, Alghoul M, Molski M, etal. Clinical
outcome of peripheral nerve decompression in diabetic and nondiabetic peripheral neuropathy. Ann
Plast Surg. 2006;57:385–90.
317. Nickerson DS, Rader AJ.Low long-term risk of foot
ulcer recurrence after nerve decompression in a diabetes neuropathy cohort. J Am Podiatr Med Assoc.
2013;103(5):380–6.

358
Ö. F. Dilek et al.
318. Karagoz H, Yuksel F, Ulkur E, Celikoz B. Early
and late results of nerve decompression procedures
in diabetic neuropathy: a series from Turkiye. J
Reconstr Microsurg. 2008;24(2):95–101.
319. Dellon AL, Mackinnon SE, Seiler WA.Susceptibility
of the diabetic nerve to chronic compression. Ann
Plast Surg. 1988;20:117–9.
320. Dellon AL, Dellon ES, Seiler WA. Effect of tarsal
tunnel decompression in the streptozosin induced
diabetic rat. Microsurgery. 1994;15:265–8.
321. Kale B, Yuksel F, Celikoz B, et al. Effect of various nerve decompression procedures on the function of distal limbs in streptozocin-induced diabetic
rats: further optimism in diabetic neuropathy. Plast
Reconstr Surg. 2003;111:2265–72.
322. Siemionow M, Sari A, Demir Y.Effect of early nerve
release on the progression of neuropathy in diabetic
rats. Ann Plast Surg. 2007;59(1):102–8.
323. Zhong W, Yang M, Zhang W, Visocchi M, Chen
X, Liao C. Improved neural microcirculation and
regeneration after peripheral nerve decompression
in DPN rats. Neurol Res. 2017;39(4):285–91.
324. Cornblath DR, Vinik A, Feldman E, et al. Surgical
decompression for diabetic sensorimotor polyneuropathy. Diabetes Care. 2007;30(2):421–2.
325. Chaudhry V, Russell J, Belzberg A.Decompressive
surgery of lower limbs for symmetrical diabetic
peripheral neuropathy. Cochrane Database Syst Rev.
2008;3:CD006152.
326. Nickerson DS. Nerve decompression and neuropathy complications in diabetes: are attitudes
discordant with evidence? Diabet Foot Ankle.
2017;8(1):1367209.
327. Nickerson DS.Low recurrence rate of diabetic foot
ulcer after nerve decompression. J Am Podiatr Med
Assoc. 2010;100(2):111–5.
328. Nickerson DS, Rader AJ.Nerve decompression after
diabetic foot ulceration may protect against recurrence: a 3-year controlled, prospective analysis. J
Am Podiatr Med Assoc. 2014;104(1):66.
329. Zhang W, Zhong W, Yang M, etal. Evaluation of the
clinical efcacy of multiple lower-extremity nerve
decompression in diabetic peripheral neuropathy. Br
J Neurosurg. 2013;27(6):795–9.
330. Trignano E, Fallico N, Chen HC, etal. Evaluation
of peripheral microcirculation improvement of foot
after tarsal tunnel release in diabetic patients by transcutaneous oximetry. Microsurgery. 2016;36(1):37.

Prophylactic Cardiac andVascular
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Surgery Procedures
TahirYağdı , MustafaÖzbaran ,
andÇağatayEngin
30
30.1 Introduction
Cardiovascular diseases are the leading causes of
mortality, being responsible for approximately
one-third of all deaths globally. Diseases concerning the eld of cardiovascular surgery are
mostly the coronary artery diseases, carotid
artery diseases, valvular heart diseases, cardiac
tumors, aortic aneurysms and peripheral arterial
diseases. Patients may remain asymptomatic for
a long period of time before the diagnosis. Thus,
especially in the high-risk group of patients, periodical laboratory tests and further relevant interventions should be made in time, since a delay
can lead to gradual deterioration of the patients.
The two principal purposes of surgical therapy
for this special patient population are: increase
survival rates and improvement of the symptoms.
This chapter provides a brief summary about the
aforementioned diseases and the preventive surgical strategies, particularly in patients without
symptoms.
T. Yağdı (*) · M. Özbaran · Ç. Engin
Department of Cardiovascular Surgery, School of
Medicine, Ege University, Izmir, Turkey
e-mail: tahir.yagdi@ege.edu.tr;
mustafa.ozbaran@ege.edu.tr;
cagatay.engin@ege.edu.tr
30.2 Coronary Artery Disease
Coronary artery disease, besides being one of the
most important causes of mortality, is also one of
the most frequently seen pathology in patients
who undergo cardiac surgery. Risk factors such
as older age, male gender, hypertension, diabetes
mellitus, hypercholesterolemia, smoking, alcohol, obesity, unhealthy diet and nutrition, insufcient physical activity, family history and
genetic predisposition and stress are commonly
seen in patients with coronary artery disease.
Coronary artery disease is a serious condition
which requires immediate treatment either by
medically or surgically. In surgical point of view,
the approach is to revascularization of the ischemic area. The principal goals of surgical revascularization for patients with coronary artery
disease are to increase survival and to reduce
symptoms [1, 2].
Coronary artery bypass grafting to improve
survival is recommended for patients with more
than 50% diameter stenosis of left main coronary artery [3]. Coronary artery bypass grafting
to improve survival is also useful in patients having no symptom and with more than 70% diameter stenosis in three major coronary arteries or
in the proximal left anterior descending artery
with one of the other major coronary arteries
(Fig.30.1) [4]. Coronary artery bypass grafting
also could be useful to increase survival in
asymptomatic patients with signicant stenosis
in two major coronary arteries with severe or
© The Author(s), under exclusive license to Springer Nature Switzerland AG 2021
O. N. Dilek et al. (eds.), Prophylactic Surgery, https://doi.org/10.1007/978-3-030-66853-2_30
359

360
Fig. 30.1 Completed coronary artery bypass grafting
operation performed with cardiopulmonary bypass via
median sternotomy.*: LIMA graft to LAD; **: saphenous
graft to D
nous graft to OM
left internal mammary artery, LAD left anterior descending artery, D
ginal branch, OM
right coronary artery)
; ***: saphenous graft to RCA; ****: saphe-
1
; *****: saphenous graft to OM2. (LIMA
1
rst diagonal branch, OM1 rst obtuse mar-
1
second obtuse marginal branch, RCA
2
extensive myocardial ischemia or target coronary arteries supplying a sizeable region of viable myocardium [5].
T. Yağdı et al.
Fig. 30.2 3D reconstruction image of preocclusive
carotid stenosis of internal carotid artery on CT angiographic examination (CCA common carotid artery, ICA
internal carotid artery, ECA external carotid artery)
subtraction angiography (DSA). Precise estimation of the stenosis is usually made with CTA
(Fig.30.2).
Following the diagnosis, surgical removal of
the atherosclerotic plaque is essential. Additionally,
concomitant carotid artery endarterectomy in
patients who undergo coronary artery bypass procedure is strongly recommended. Coronary artery
bypass and carotid endarterectomy can be done
either simultaneously or staged [7].
30.3.2 Carotid Body Tumors
30.3 Carotid Artery Diseases
30.3.1 Carotid Artery Stenosis
Carotid artery stenosis is well-known atherosclerotic process and is one of the main reasons
of cerebrovascular accident. The asymptomatic
carotid atherosclerosis study (ACAS) has conrmed that carotid endarterectomy is useful for
the decrease of neurologic sequalae in patients
with signicant carotid stenosis from 18% to
7% over 5 years [6]. Numerous diagnostic tools
exist for assessment of the disease, such as color
Doppler ultrasonography, computed tomography angiography (CTA), magnetic resonance
angiography (MRA) and intra-arterial digital
Carotid body tumors are rarely seen clinical entities, which are generally located at the bifurcation of the common carotid artery as
neuroendocrine neoplasms [8]. They are frequently located unilaterally and mostly have a
benign nature. They can be seen both in men and
women with equal proportions. Clinical presentation of the disease is variable. Approximately,
two-third of the patients present with an asymptomatic mass on the neck, located alongside the
sternocleidomastoid muscle. Since the process of
progression is quite slow, patients often remain
asymptomatic for a long period of time.
Vascular tumors originating from the chemoreceptor cells located at the carotid artery bifurcation are dense. Thus, surgical excision is
considered as a potentially dangerous procedure.
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