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12.2 Sonographic Findings
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
Fig. 470 Multiple rounded, echogenic splenic foci (S) due to diffuse metastasis from a carcinoid
– Usually round with smooth margins – Occasional intralesional calcifications – CDS: usually sparse vascularity
x
Clinical findings: usually asymptomatic, often detected incidentally on ultra­sound
n
Splenic calcification (Fig. 471):
x
Sonographic criteria:
– Predominantly echogenic focus – Variable size, smooth margins; variable shape – Posterior acoustic shadowing – CDS: absence of vascularity
x
Clinical findings: usually asymptomatic. May result from or accompany inflam­mations, abscesses, infarctions, cysts, and metabolic diseases (calcification of the splenic artery)
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Spleen
Spleen
Spleen
Fig. 471 Multiple intrasplenic calcifications in an asymptomatic patient. S = acoustic shadows
Further Testing
..............................................................................................................
n
Splenomegaly: A complete workup may require laboratory tests, lymph node
histology, bone marrow histology, and liver histology.
n
Small spleen: laboratory parameters, Howell–Jolly bodies in stained blood,
99m
Tc colloid scintigraphy
n
Splenic cyst : see Fig. 472).
n
Splenic abscess :
x
Diagnostic fine-needle aspiration (see p. 53), repetitive percutaneous drainage, catheter drainage (see p. 58), surgical options
319
12.2 Sonographic Findings
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
12
12
12
Spleen
Spleen
Spleen
Fig. 472 Therapeutic algorithm for a splenic cyst
x
With microabscesses (Fig. 462, p. 316): antimycotic therapy with sonographic follow-up
n
Splenic lymphoma: sonographic follow-up. Isolated splenic lymphoma can be
evaluated by percutaneous biopsy, but the sonographic course is usually enough to make a benign–malignant differentiation.
n
Splenic infarction: requires sonographic follow-up. Most cases undergo complete
resolution. Surgery may be necessary if complications arise (e.g., increasing lique­faction of the infarction, lack of regression, splenic rupture, infection) (Fig.
n
Splenic trauma :
x
CT is more reliable than ultrasound for grading the splenic injury.
x
Sonographic follow-up or surgery, depending on imaging findings and espe­cially on clinical status
!
Caution: Look out for two-stage splenic rupture with subcapsular hemorrhage.
n
Splenic metastases:
x
Sonographic follow-up is usually sufficient, because of the presence of an inop­erable tumor or end-stage disease.
x
With isolated splenic infiltration : fine-needle aspiration (see p. 53) or surgical diagnosis
n
Hemangioma, splenoma: CT may be required. Sonographic follow-ups or surgical
diagnosis may be advised, depending on clinical findings. Often a definitive diag­nosis is not made if the lesion remains unchanged over time.
n
Splenic calcification: Sonographic follow-up may be advised. If ultrasound con-
firms the diagnosis, there is no need for additional tests.
473).
320
12.2 Sonographic Findings
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
Fig. 473 Sonographic manifestations, course, and complications of splenic infarction (after Görg)
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12
Spleen
Spleen
Spleen
321
13.1 Examination
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
13
13
13
13 Bile Ducts

13.1 Examination

Bile Ducts

Bile Ducts
Bile Ducts
Scan Planes
..............................................................................................................
n
Intercostal scan, extended right intercostal scan, porta hepatis scan (see p. 33)
n
Subcostal oblique scan on the right side (see p. 22).
Sonographic Anatomy and Normal Findings
..............................................................................................................
n
With careful scrutiny, the intrahepatic bile ducts can be identified as fine anechoic bands running anterior to the portal venous branches (except the left anterior seg­mental branch). The hepatic artery runs between or posterior to the intrahepatic ducts.
n
The right and left hepatic ducts unite at the porta hepatis to form the common hepatic duct.
n
The cystic duct is very difficult to visualize because of its tortuous course.
n
In most cases the common bile duct and pancreatic duct empty into the duode­num by a common opening. (Since the termination of the cystic duct generally cannot be identified with ultrasound, the common hepatic duct and common bile duct are usually referred to collectively as “the bile duct,” Figs
n
Normal dimensions : The extrahepatic duct is I 7 mm in diameter, depending on age.
474 and 475.)
Fig. 474a Topographic anatomy of the gallbladder and biliary tract
322
13.1 Examination
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
Fig. 474b Topographic anatomy of the gallbladder and biliary tract
Fig. 475 ERCP demonstrates the de­scending, prepapillary (common) course of the bile duct and pancreatic duct in the head of the pancreas
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Bile Ducts
Bile Ducts
Bile Ducts
Scanning Protocol
..............................................................................................................
n
Transducer: 3.5–5.0 MHz.
n
The patient is scanned early in the morning in a fasting state.
n
Use the supine position for scanning the prepapillary bile duct; use left lateral decubitus and deep inspiration for scanning the upper two-thirds of the bile duct.
n
A parasagittal upper abdominal longitudinal scan is best for evaluating the prepa­pillary segment of the bile duct (see Fig.
n
A semiupright position is occasionally helpful in the detection of prepapillary stones.
n
Scanning tip: Carefully controlled compression of the abdominal fat and bowel
gas with a small-footprint transducer will generally permit good imaging of the distal bile duct and its termination in the duodenum (Fig.
84, p. 64).
476).
323
13.1 Examination
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
13
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a b
Bile Ducts
Bile Ducts
Bile Ducts
c
Fig. 476a–c Intra- and extrahepatic course of the bile duct (BD) anterior to the proper hepatic artery (A, arrow, cut transversely by the scan), the portal vein (PV, cut obliquely by the scan), and the inferior vena cava (VC, cut longitudinally by the scan). Paramedian upper ab­dominal longitudinal scan on the right side. a B-mode image. b Correlative diagram. c CDS. L = liver, P = pancreas
Overview and Classification of Findings
..............................................................................................................
n
Incidental findings in the biliary tract are rare because clinical manifestations will usually prompt a specific ultrasound examination of that region. Thus, the sono­grams in this chapter do not illustrate findings that correlate with a clinical pre­sentation of “jaundice” or “cholestasis” (jaundice is reviewed on pp. 139–149).
n
Most abnormal findings are characterized by circumscribed or diffuse biliary sta­sis. It is rare to encounter a nonobstructive biliary tract lesion.
n
Echogenic structures projected into the liver or biliary tract may represent stones, calcifications, vascular calcification, abscesses, clots, or an echogenic ligamentum teres. Biliary sludge is apt to be mistaken for an intraluminal tumor.
n
Classification and overview:
x
Intrahepatic duct changes: see Table 55, p. 325.
x
Extrahepatic duct changes: see Table 56, p. 330.
n
Typical duct changes in:
x
Biliary cysts, liver cysts: Biliary cysts communicate with the biliary system. They contain biliary fluid and are therefore susceptible to stone formation (see Fig.
478a–c, p. 326). “Liver cysts” usually originate from the biliary tract but
do not communicate with the bile ducts and therefore contain serous fluid rather than bile. Sclerosing cholangitis, biliary cirrhosis : Despite cholestasis, primary biliary cirrhosis is not associated with bile duct changes (unlike primary sclerosing cholangitis, it affects only canaliculi and does not affect larger bile ducts).
x
Duct stones : Intraductal stones are not necessarily associated with cholestasis or ductal dilatation.
324
13.2 Intrahepatic Ductal Changes
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.

13.2 Intrahepatic Ductal Changes

Overview (Table 55):
..............................................................................................................
Table 55.Intrahepatic duct changes
yyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyy
Anechoic or hypoechoic Echogenic or sonodense
yyyyyyyyyyyyyyyyyyyyyyyy yyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyy yyyyyyyyy
Biliary cysts, Caroli syndrome (p. 326)
Sclerosing cholangitis (p. 327) Pneumobilia (p. 330)
Parasites (p. 328)
Papillomatosis, papillomatous carcinoma (p. 328)
Intrahepatic bile duct carcinoma (p. 328)
Compression or infiltration by metastases (p. 329)
Anechoic or Hypoechoic Changes
..............................................................................................................
n
Ductal dilatation: The dilated duct and associated portal vein branch appear as
anechoic tramlines. Generally the bile duct lies anterior and the portal vein branch is posterior.
x
Exception: With the anterior and posterior branches of the left main branch of the portal vein, the bile duct is posterior to the anterior branch and anterior to the posterior branch. Doubts can be resolved by CDS (Fig.
yyyyyyyyyyyyyyyyyyyyyyyyyy yyyyyyyyyyyyyy
Duct stones (p. 330)
477).
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Bile Ducts
Bile Ducts
Bile Ducts
ab
Fig. 477a, b Intrahepatic bile duct dilatation secondary to a long-standing elevation of biliary pressure. a B-mode image: dilated intrahepatic duct branch in segment II and an accompanying anterior portal vessel, also an expanded bile duct branch in segment III. b CDS: The left main branch of the portal vein is encoded in red. CDS (with spectral analysis if necessary) can positively distinguish between a portal venous branch, an artery, and a bile duct (BD)
325
13.2 Intrahepatic Ductal Changes
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
13
13
13
n
Biliary cysts (Caroli syndrome, Fig. 478):
x
Circumscribed anechoic ductal dilatation
x
Possible hyperechoic stone with a distal acoustic shadow
Bile Ducts
Bile Ducts
Bile Ducts
ab
Fig. 478a–c Caroli syndrome (detected incidentally in a patient with iritis). a Cystic dilatation of intrahepatic bile duct with a peripheral echogenic zone and distal acoustic shadowing. b Scan after operative treatment shows regression of cholestasis with a persis­tent echogenic stone casting a partial shadow. c ERC image: cystic dilatation of bile ducts including the cystic duct, with partial filling defects caused by
c
intraductal stones
326
13.2 Intrahepatic Ductal Changes
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
n
Sclerosing cholangitis (Fig. 479):
x
Irregularly dilated bile ducts with caliber variations
x
Stenoses
x
Broadened, fragmented portal tracts extending toward the periphery
x
Accentuated arteries (between the portal branch and bile duct)
b
a
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Bile Ducts
Bile Ducts
Bile Ducts
Fig. 479a–c Chronic sclerosing cholangitis. a Wall-thickened common bile duct (1). b Caliber irregularities in intrahepatic bile ducts (2) and sludge (3). c Corresponding retrograde cholangio­gram: segmental stenoses and preste­notic dilatation
c
327
13.2 Intrahepatic Ductal Changes
Schmidt, Ultrasound © 2007 Thieme
All rights reserved. Usage subject to terms and conditions of license.
13
13
13
n
Parasites (in order of frequency) : ascarids, Echinococcus alveolaris, liver fluke
n
Note: The overall incidence of biliary parasites is very low.
n
Biliary papillomatosis, papillomatous carcinoma (Fig. 480):
x
Lobular, polypoid intraluminal mass
x
Dilated intrahepatic (and extrahepatic) bile ducts
Bile Ducts
Bile Ducts
Bile Ducts
n
Note: Papillomatosis is rare. The prognosis is poor because of the high potential
for recurrence and malignant change. Liver transplantation may be required.
ab
Fig. 480a, b Papillomatous bile duct carcinoma. a Tumor mass in the left hepatic duct (LHD), appearing isoechoic to liver parenchyma. Arrow: biliary drain. b CDS: tumor ingrowth into the bile duct (BD). There are practically no intratu­moral vessels. PV = portal vein, GB = gallbladder
n
Intrahepatic bile duct carcinoma (Fig. 481):
x
Circumscribed, anechoic area of ductal dilatation
x
Contour cutoff
x
Often a tumor is not directly visualized but is evidenced by the hypoechoic transformation of proximal liver segments
x
Pericanalicular tumor spread
328