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32
Step1
Section II • Esophagus, Stomach, and Duodenum
Ligamentum teres (round ligament) plasty
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Complicated reux disease (esophagitisIV)
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Fundoplication+dilation (of orid esophagitis)
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Fundoplication+parietal cell vagotomy (in gastric hyperacidity)+if necessary dilation
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(of orid esophagitis)
Fundoplication+parietal cell vagotomy+stricturoplasty (of scarred strictures)
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Limited resection of the gastroesophageal junction
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Mobilization of the distal esophagus and fundus
e distal esophagus is completely dissected and encircled with a vessel loop being careful to avoid injury to the vagus nerves. e gastric fundus is completely mobilized by division of the short gastric vessels in order to form a loose, “oppy” fundoplication.
If ligamentum teres plasty is planned, there is no need for fundic mobilization. Special atten­tion has to be paid to thoroughly preserving the ligament at laparotomy.
In the presence of a hiatal hernia, a posterior hiatoplasty is performed using nonabsorbable suture material (
. Fig. 32.1).
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. Fig.32.1
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Chapter  • Operation for GERD: Conventional Approach
Total (“Nissen”) Fundoplication


Step2
Passage of the fundus
e mobilized fundus is passed behind the esophagus to the right side so far that it can be easily sewn to the remaining front wall of the wrapped fund is in front of the esophagus (
. Fig.32.2
. Fig. 32.2).
Step3
Formation of the wrap
e two cu folds are xed with three, maximally four, nonabsorbable sutures. At least, one or more sutures should include the esophageal wall to prevent proximal or distal "slippage" of the fundoplication wrap (
. Fig. 32.3).
. Fig.32.3
Section II • Esophagus, Stomach, and Duodenum
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Step4
Anchoring of the wrap
Finally the fundic cu is again tested. Two ngers should easily pass under the loose wrap around the distal esophagus (“oppy Nissen”).
One or two additional sutures can x the le cu of the anterior gastric wall to the right crus in order to prevent slippage (
. Fig. 32.4).
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Step1
Step2
. Fig.32.4
Ligamentum Teres (Round Ligament) Plasty
See above.
Dissection of the round ligament
e round ligament is carefully dissected from the abdominal wall and from the liver, respectively. e free end of the ligament is transposed posteriorly around the esophagus coming from the right side (
. Fig. 32.5).
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. Fig.32.5
Chapter  • Operation for GERD: Conventional Approach


Step3
Fixation to the anterior gastric wall
e round ligament is then attached to the anterior gastric wall under relative tension using three or four nonabsorbable sutures. Fixation to the anterior aspect of the gastric corpus is performed (
. Fig. 32.6).
. Fig.32.6
Standard Postoperative Investigations
See Chap. 31 “Operation for GERD: Laparoscopic Approach.”
Postoperative Complications
z Short term
Esophageal perforation
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Dysphagia
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z Long term
Dysphagia
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Gas bloat
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Recurrent disease
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Tricks of the Senior Surgeon
Perform Nissen fundoplication around a large 45 to 60 Fr bougie to facilitate formation of a
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loose “oppy,” fundic wrap.
Use of a self-retaining retractor system will facilitate exposure of the esophagogastric junction.
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Dissection of the short gastrics is not mandatory but will ensure a loose fundoplication, thus
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preventing postoperative dysphagia.
Do not dissect the round ligament at laparotomy.
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Consider partial posterior fundoplication (Toupét technique) especially in patients with poor
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esophageal motility – the only dierence is that in step4 (as described above) suture both
fundus cu folds to the right and left esophageal aspect respectively, thus forming a poste-
rior 270°cu instead of a 360°cu. Anchor the posterior wall of the cu to the right and left
crus with two sutures each.

Operation for Paraesophageal Hernia

Jean-Marie Michel, Lucas Krähenbühl
In 1889, Postempski rst reported the repair of a wound of the diaphragm. Ackerlund described dierent types of paraesophageal hernia in 1926, and the rst hiatal hernia repair (fundoplication) was reported by Nissen in 1955. Since then, Nissen fundoplication has gained wide acceptance and is now recognized as the operation of choice for antireux surgery and, although technically challenging, the laparoscopic approach has become the “gold standard” of paraesophageal hernia repair.
e goal of a paraesophageal hernia repair is to bring the stomach (with other organs such as colon, omentum, spleen) and the lower esophagus back into the abdominal cavity, to excise the hernia sac, to approximate crura, to perform a fundoplication in order to prevent gastroesophageal reux, and nally to perform a gastropexy in order to prevent gastric volvulus.
Indications and Contraindications for Laparoscopy


Indications
Contraindications
Contraindications
Symptomatic or asymptomatic typeII, typeIII, and typeIV hiatal hernia
TypeII: Pure paraesophageal hernias; the gastroesophageal junction remains in its normal
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anatomical position but a portion of the fundus and proximal stomach herniates through
the diaphragmatic hiatus adjacent to the esophagus.
TypeIII: e gastroesophageal junction and the fundus herniating through the hiatus. e
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fundus lies above the gastroesophageal junction.
TypeIV: ese hernias are characterized by the presence of a structure other than the stom-
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ach, like the omentum, colon, small bowel, or spleen, within the hernia sac.
z Absolute
Intrathoracic gastric perforation with typeII, typeIII, or typeIV hiatal hernia
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z Relative
Gastric incarceration
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Partially xed paraesophageal hernia
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Short esophagus
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Indications and Contraindications for Laparotomy
Intrathoracic gastric perforation with typeII, typeIII, or typeIV hiatal hernia due to the
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peritonitis/mediastinatis no plastic reparation in the acute phase.
z Relative
None
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P.-A. Clavien, M. G. Sarr, Y. Fong, M. Miyazaki (Eds.), Atlas of Upper Gastrointestinal and Hepato-Pancreato-Biliary Surger y, DOI 10.1007/978-3-662-46546-2_33, © Springer-Verlag Berlin Heidelberg 2016
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10 to 12 cm
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Section II • Esophagus, Stomach, and Duodenum
Preoperative Investigations/Preparation for the Procedure
History: Long-term history of gastroesophageal reux disease (GERD), symptoms of upper gas­trointestinal occlusion
Upright radiograph of the thorax: Search for a retrocardiac air-uid level Contrast radiographic studies (barium swallow): Preoperative localization of the gastroesoph-
ageal junction, assessment of the type of hernia
Esophageal manometry: To exclude a motility disorder of the esophagus Upper endoscopy: To conrm ndings of GERD and/or exclusion of gastric ulcer disease
24-h pH monitoring and stationary manometry: To document GERD and esophageal dys­motility. In typeII hernias, 70 % of patients have pathologic pH-metry, with up to 100 % of patients with typeIII hernias.
Actively treat dehydration
Empty the stomach: Nasogastric tube or immediate preoperative endoscopy
One-shot prophylactic antibiotic with second-generation cephalosporine
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Procedure
e patient is placed in a modied lithotomy position. e table is placed in a steep reverse Tren­delenburg position (French position), with the surgeon standing between the patient’s legs, the rst assistant on the patient’s le, and the camera assistant on the patient’s right.
Port Placement
A 10-mm port is placed 5 to 8 cm cranial to the umbilicus in the midline (open Hasson tech­nique). A carbon dioxide pneumoperitoneum is established (12 mmHg). A 30°laparoscope is mandatory. Aer exploratory laparoscopy, the next four trocar sleeves are placed under direct vision. A subxiphoid 5-mm port for liver retraction; two working ports: one 5-mm one in the right upper quadrant (UQ), another 10-mm one in the le UQ; and a 5-mm le subcostal port (
. Fig. 33.1).
5 mm ports
12 mm
optical port
12 mm
working port
about
from xiphoid
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. Fig.33.1
Chapter  • Operation for Paraesophageal Hernia
Exposure
To allow free access to the enlarged esophageal hiatus, the le lobe of the liver has to be elevated with a liver retractor to expose the esophago-gastric junction (
. Fig. 33.2).


Step1
. Fig.33.2
Reduction of herniated stomach
e herniated stomach and the greater omentum are reduced into the abdominal cavity with two Babcock graspers. A nasogastric tube is then introduced to decompress the stomach.
is maneuver is a dangerous step of the procedure with risks of stomach perforation, particu­larly in case of mechanical obstruction of the stomach (volvulus) with incarceration and gastric wall ischemia.
e spleen, colon, and omentum should also be reduced if they have herniated into the thorax (. Fig. 33.3).
. Fig.33.3
Section II • Esophagus, Stomach, and Duodenum
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Step2
Exposure of the hiatal hernia
Open the gastrohepatic ligament aer reduction of the hernia content and expose the right crus of the diaphragm. e hepatic trunk of the vagus nerve and aberrant le hepatic artery should be preserved if possible. e hiatus and the hernia sac are now visible (
. Fig. 33.4).
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Step3
. Fig.33.4
Circular incision of the hernia sac
Start the procedure on the right side and dissect the hernia sac o the right crural edge using the harmonic scalpel (Ethicon Endo-Surgery, Cincinnati, OH). Complete the dissection inferiorly and obtain a good exposure of the junction between the right and the le crura, then cranially with the incision of the phrenoesophageal membrane, nally to the le over the le crus. e dissection over the inferoposterior edge of the le crus is dicult at this moment and is best achieved when the hernia sac is completely reduced from the mediastinum.
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Chapter  • Operation for Paraesophageal Hernia


Step4
Blunt dissection of the hernia sac
e hernia sac now should be bluntly removed from the mediastinum with complete exposure of the right and le crura (see STEP3) (
During this step anterior and posterior vagal nerves have to be identied and protected; this can be dicult to perform if there is associated inammation.
It is not unusual for the le and/or right pleura to be opened within the mediastinum during blunt dissection, but most of the time pleural drainage is not mandatory.
Complete the dissection of the inferoposterior edge of the le crus. Pay particular attention to nding the plane between the esophagus and the body of the le crus, which may sometimes be extraordinarily dicult. It is not necessary to excise the hernia sac.
. Fig. 33.5).
Step5
. Fig.33.5
Intra-abdominal reduction of the gastroesophageal junction
e distal esophagus is now completely freed and a Penrose drain is placed around the gastro­esophageal junction to permit a better retraction in the abdominal cavity.
It is reported that as many as 15 % of giant typeIII paraesophageal hernias will present with a shortened esophagus and have an irreducible gastroesophageal junction. Adequate mobilization of the esophagus then should be performed as high as possible into the mediastinum. If the reduc­tion remains impossible aer this maneuver, the patient will most benet from a Collis-Nissen gastroplasty, which can be done via a laparoscopic and/or a thoracoscopic approach.
Section II • Esophagus, Stomach, and Duodenum
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Step6
Closure of the hiatal defect (posterior cruroplasty)
e hiatal defect is closed with ve to six nonabsorbable 2-0Ethibond mattress sutures placed posteriorly and anteriorly from the esophagus to return the gastroesophageal junction into the abdomen (. around the esophagus.
e axis of the hiatal hernia has an inferosuperior direction with an angle of about 10°clock­wise in the perpendicular plane, and an inferosuperior direction with an angle of about 70°clock­wise in the sagittal plane. us closure of the hiatal defect must follow the schema represented in
. Fig. 33.6b.
Fig. 33.6a
). e sutures are placed from caudad to cephalad so the hiatus is snug
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. Fig.33.6