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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_788_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Preface
- •Contributors
- •Contents
- •1. Introduction
- •1.1 Introduction
- •1.2 Minimally Invasive Surgery and Videolaparoscopic Surgery
- •2. Instruments and Methods
- •2.1 Three-Dimensional Optics in Clinical Practice
- •2.2 Access and Exposure Techniques in Laparoscopic Surgery
- •2.3 Joining and Sealing Tissues and Hollow Organs
- •2.4 Gasless Laparoscopy
- •2.5 Anesthesia in Videolaparoscopic Surgery
- •3. Laparoscopic Exploration, Diagnosis, and Staging
- •3.1 Visual Exploration of the Peritoneal Cavity
- •3.2 Diagnostic Laparoscopy for Trauma
- •3.3 Laparoscopy for the Acute Abdomen
- •3.4 Laparoscopy for Peritonitis
- •3.5 Comments on Laparoscopy for the Acute Abdomen
- •3.6 Diagnostic Laparoscopy for Tumors
- •3.7 Staging of Neoplastic Disease with Ultrasound
- •3.8 Comments on Laparoscopic Ultrasonography for Staging
- •3.9 Visual Exploration of the Pelvic Organs in Women
- •4. Laparoscopic Cholecystectomy
- •4.1 Retrograde Cholecystectomy
- •4.2 Anterograde Cholecystectomy
- •4.3 Alternate Method of Gallbladder Retrieval
- •4.4 Comments on Laparoscopic Cholecystectomy
- •5. Extrahepatic Bile Ducts: Diagnosis and Treatment
- •5.1 Ultrasonography of the Bile Ducts
- •5.2 Intraoperative Cholangiography
- •5.3 Comments on Intraopertive Cholangiography
- •5.5 Common Bile Duct Exploration and Stone Removal
- •5.6 Laparoscopic Cholecystojejunostomy
- •5.7 Comments on Laparoscopic Biliary Operations
- •6. Laparoscopic Approach to the Spleen and Liver
- •6.1 Splenectomy
- •6.2 Comments on Laparoscopic Splenectomy
- •6.3 Comments on Laparoscopic Splenectomy
- •6.4 Fenestration of Large Splenic Cysts
- •6.5 Fenestration of Hepatic Cysts
- •7. Intra-abdominal and Endoluminal Gastric Operations
- •7.1 Closure of Peptic Ulcer Perforation
- •7.2 Laparoscopically-Assisted Gastric Resection
- •7.3 Combined Laparoscopic and Endoscopic Gastric Wedge Resections
- •7.4 Gastrostomy
- •7.5 Endoscopic Intraluminal Gastroduodeno-Pancreatic Cystostomy
- •7.6 Combined Endoluminal and Open Gastric Operation
- •8. Vagotomy and Drainage Procedures
- •8.1 Indications for Vagotomy
- •8.2 Posterior Truncal Vagotomy and Denervating Anterior Linear Strip Gastrectomy
- •8.3 Selective Proximal Vagotomy
- •8.4 Posterior Truncal Vagotomy and Anterior Gastric Seromyotomy (Taylor 1985)
- •8.5 Anterior and Posterior Truncal Vagotomy and Pyloroplasty
- •8.6 Laparoscopically Guided Truncal Vagotomy and Assisted Pyloroplasty Using a Circular Stapler
- •8.7 Gastrojejunostomy
- •8.8 Current Status of Laparoscopic Management of Duodenal Ulcers
- •8.9 Thoracoscopic Truncal Vagotomy
- •9. Operations on the G.-E. Junction
- •9.1 Nissen Fundoplication
- •9.2 Fundoplication and Partial Fundoplication Techniques
- •9.3 Comments on Nissen Fundoplication
- •9.4 Gastropexy in Paraesophageal Hiatus Hernia Repair
- •9.5 Cardiomyotomy and Fundoplasty for Achalasia
- •9.7 Laparoscopically Guided Gastric Banding for Morbid Obesity
- •9.8 Comments on Gastric Banding for Morbid Obesity
- •9.9 Alternative Operative Techniques for Gastro-Jejunal Bypass in Morbid Obesity
- •10. Appendectomy and Small Bowel Procedures
- •10.1 Appendectomy
- •10.2 Comments on Laparoscopic Appendectomy
- •10.3 Comments on Laparoscopic Appendectomy
- •10.4 Meckel’s Diverticulectomy
- •10.5 Small-Bowel Resection
- •10.6 Laparoscopic Lysis of Adhesions
- •10.7 Creation of a Loop Ileostomy
- •11. Laparoscopically-Assisted Large Bowel Procedures
- •11.1 Creation of an Intestinal Stoma
- •11.2 Laparoscopically-Assisted Right Hemicolectomy
- •11.3 Resection of Sigmoid Colon
- •11.4 Laparoscopically Assisted Left Hemicolectomy
- •11.5 Combined Endoluminal and Open Colon Procedure
- •12. Laparoscopically-Guided/Assisted Colo-Rectal Procedures
- •12.1 Repair of Perforations of the Colon and Rectum
- •12.2 Repair of Rectal Prolapse
- •12.3 Laparoscopic Second Stage Hartmann Procedure
- •12.4 Laparoscopically Assisted Anterior Resection and Recto-Sigmoidostomy
- •12.5 Abdominoperineal Excision or Amputation of the Rectum (with High Ligation of the Inferior Mesenteric Artery)
- •12.6 Comments on Laparoscopic Colorectal Surgery
- •12.7 Comments on Laparoscopic Colorectal Surgery
- •13. Inguinal Hernia Repair
- •13.1 Videoendoscopic Preperitoneal Hernia Repair
- •13.2 Laparoscopic Transabdominal Preperitoneal Inguinal Hernia Repair
- •13.3 Complicated Laparoscopic Hernia Repair: Avoiding Complications and Recurrence in Clinical Practice
- •13.4 Comments on Laparoscopic Hernia Repair
- •14. Closing Commentaries
- •14.1 Analysis and Prevention of Untoward Events in Laparoscopic Surgery
- •14.2 Pneumoperitoneum-Associated Alterations and Risk Factors in Laparoscopic Surgery
- •14.3 Minimally Invasive Surgery by Video-Endoscopic Techniques: New Technology Rejuvenates Proven Concept
- •Index

248
9.2 Fundoplication and Partial Fundoplication Techniques
intact
peristalsis
manometry:
Nissen procedure
impaired
peristalsis
allows only
limited mechanical
augmentation of LES:
partial Toupet
fundoplication
Fig. 9.2.3 Prevention of persistent postoperative dysphagia by using in selective cases a partial fundoplication (Toupet) when esophageal clearance is
reduced by impaired esophageal peristalsis.
Contraindications:
− Extensive cardiopulmonary and other anesthesia risk factors.
− Disorders of blood coagulation.
− Previous operations in the upper abdomen can be a contraindication to the laparoscopic approach of fundoplication.
However, if the patient is preoperatively well informed, exploratory laparoscopy prior to laparotomy can be advised to
assess the possibility of laparoscopic adhesiolysis followed
by laparoscopic fundoplication or early conversion.
Operative Risk and Informed Consent
Prior to operation the patient must be informed about the cause
of the disease and the possible corrections that can be achieved
by the operation. All patients must be informed of the possibility of conversion to conventional laparotomy. Factors that can
make a conversion likely are:
− Short esophagus after decades of reflux history and impossibility to mobilize the distal part of the esophagus into the abdomen to position the fundoplication correctly.
− Confluent active esophagitis or a history of stricture with extensive periesophageal tissue reaction in the lower mediastinum;

Special Preparations:
249
Fig. 9.2.4 Anatomy of the gastroesophageal junction showing the
phrenoesophageal membrane and its variations and insertion at the
esophagus; Anatomy of the fundus and especially fixation of the fundus
by short gastric vessels or posterior gastric artery which need to be
divided for full fundic mobilization.
Posterior
gastric artery Splenic artery
SPLEEN
Fig. 9.2.5 Different types of hiatal hernia:
a axial hernia
Special risks that the patient should be informed of before undergoing
laparoscopic antireflux surgery:
−CO2emphysema in the trunk, in the head and neck due to
mediastinal mobilization of the esophagus. (In case of head
and neck emphysema, the patient should be kept approximately 2−4 hours on the respirator postoperatively, until the
CO
emphysema dissolves. No long-term disadvantages for
2
the patient have been reported so far.)
− Pneumothorax due to pleural injury during mediastinal mobilization.
− Spleen injury due to problems with “short” gastric vessels
during mobilization of the fundus or traction on the gastrosplenic ligament.
− Esophageal wall injury due to incorrect dissection planes.
b paraesophageal hernia c combined hernia
Postoperative problems, such as:
− Temporary dysphagia for 3−4 weeks due to tissue edema at
the wrap locations.
− Gastric emptying problems due to vagus irritation.
Special Preparations
Preoperative Testing
Endoscopy, manometry, pH monitoring.

250
9.2 Fundoplication and Partial Fundoplication Techniques
Preoperative Preparation
− Foley catheter (optional).
− Nasogastric tube (optional).
− General anaesthesia:
intubation and monitoring required due to elevated intraabdominal pressure, CO
− Positioning.
In general, there are two possibilities for the organization of the
operating team. The patient can be positioned with his legs
close together on the operating table and the surgeon and his
assistant standing on each side of the patient, or the patient is
positioned with the legs spread apart, which allows the operating surgeon to stand between the legs (Fig. 9.2.6).
monitoring important.
2
Method of Access (Fig. 9.2.7)
Laparoscopic fundoplication techniques can be performed as
any other operation at the esophageal hiatus, usually using
5 trocars, because there is a need for 1 camera trocar, 2 trocars
for helping to expose the target area (liver retractor, pull-down
of stomach and gastrosplenic ligament), and 2 trocars for pickup and dissection instruments.
The camera trocar can be inserted along the midline, often a
third to half way distance between the umbilicus and the xiphoid. The trocar for the liver retractor is inserted in the right
upper quadrant at the subcostal margin. The retractor and
grasper for the gastrosplenic ligament is brought in at the respective side of the left subcostal margin. The retractor for the
right hand dissection instrument is brought in between the
midline and the left retractor trocar. It is important to make sure
that this trocar has enough room to turn and be manipulated, as
this is necessary during the operation. The position of the left
hand dissection trocar must be chosen very carefully. It depends
on the size and position of the falciform ligament. The position
must allow, on one hand, an easy gliding of the instrument to
the hiatus without hindrance by the falciform ligament, on the
other hand, it must also be far enough from the camera trocar,
so that it can be manipulated without “dueling” with the laparoscope and camera.
Fig. 9.2.6 Positioning of the patient in supine and Anti-Trendelenburg
position. Depending on the surgeon’s position, on the side or between
the legs, the legs must be positioned close together on the table or abduced, respectively.
Fig. 9.2.7 Method of access and trocar organization in the upper abdo- 컄
men. After insertion of the camera trocar (T1) the positioning of the other
trocars depends also on the anatomical situation of the patient.
T1 camera trocar
T2 main working trocar
surgeon’s right hand
T3 main working trocar
surgeon’s left hand
T4 liver retractor
T5 omentum or gastric retractor.
T3
T4
T2
T5
T1

Special Techniques
251
Fig. 9.2.8 Dissection of the gastroesophageal junction. Incision of the
peritoneum near the hiatus under careful preservation of the vagal hepatic branches and identification of the right crus and the hiatal arch. Usu-
ally the grasper will lift up the peritoneum and with scissors, using ultrasonic technique or cautery for hemostasis the dissection can be carried
out.
Creation of the “gastroesophageal window”: Once the right crus is identified, the aim of the dissection may not be the esophagus or the gastroesophageal junction, but the crus and the lower mediastinum. It is much
easier to dissect and later identify the distal esophagus in the lower mediastinum than right at the gastroesophageal junction due to the adherence of the esophageal fat pad and the tissue along the lesser curvature
of the stomach. While a grasper is pushing the right crus to the right, the
surgeon can rather easily either with the scissors or with the swab push
the loose tissue between the right crus and the esophagus from each
other. As a consequence, the intraabdominal gas will help to develop a
dissection layer in the lower mediastinum. With a disserting swab, now,
the distal esophagus can be easily pushed aside in the lower mediastinum.
Special Techniques
The Nissen Fundoplication Technique
Step-by-Step Procedure
I Technical preparation
1. Control of patient’s correct positioning.
2. Selection of ultrasonic or high frequency electrocautery instrument, correct choice of power level in relation to patient’s size.
3. Sterile preparation of the abdomen, connection of all instruments to respective sources or terminals.
II Establishment of the pneumoperitoneum
1. Skin incision at the midline and insertion of the Veress needle.
2. Safety steps with injection, aspiration as well as manometric
tests, insufflation procedurewith lowand highflow after adjustment of pressure level (children under 14: 6−8 mm Hg, adolescents and adults: 10−14 mm Hg). In patients with cardiopulmonary risk factors or small children gas flow only with 1−2 l/min.
3. Alternative: local open approach.
Fig. 9.2.9 Nissen fundoplication, fundus mobilization. The left retractor
is used to bring tension on the gastrosplenic ligament, while the right retractor is pulling the stomach towardsthe right. This allows a good view of
the gently stretched short gastric vessels. In the avascular space between
the vessels the tissue is divided. The vessels are transected between clips
or with the harmonic scalpel. This dissection is carried on along the fundic
curvature. Special care must be taken not to injure the spleen.
A similar approach is advised by using the grasper to lift up the hiatal arch
and gently push a swab right under the hiatal arch into the lower mediastinum. With gentle force, the esophagus can be pushed downwards thus
freeing the loose tissue connections between the pericardium and the distal esophagus. When this maneuver is repeated at the left crus, the distal
esophagus is almost fully mobilized.
Completed “gastroesophageal window”. This is a view through the
“gastroesophageal window” showing the anatomic landmarks that must
be dissected completely before starting with the sutures. This shows the
hiatal crura, the hiatal arch, the distal esophagus, the high pressure zone,
the vagal trunks, and the subphrenic area on the left with the upper pole
of the spleen.
III Laparoscopy
st
1. Insertion of the 1
trocar with special caution, e. g., according
to experience insertion of “Kegel” trocar under constant rotation, rather than down force pressure through the peritoneum
(caution: no guarantee for protection from damage).
2. Insertion of camera and review of intraabdominal organs and
special circumstances.
3. Insertion of all other trocars under visual control; inspection of
the upper abdominalareaand hiatalregion togetthe “feeling”
and an impression of the situs and the tissue quality before
starting with the dissection.
4. Exploration of the hiatal hernia to assess its size and compare
findings with the preoperative endoluminal endoscopy report
and esophageal function measurements. Pull on the stomach
caudally to reposition the hernia and to assess the mobility of
the distal esophagus. An inability to reduce the stomach and
the cardia into the abdomen can be an early sign that the
esophagus istoo shortand anearly conversion or evenprimary
thoracic approach is necessary.
5. Incision of the phrenoesophageal membrane with careful preservation of the hepatic vagal branches.

252
Fig. 9.2.10 Posterior crural closure or hiatoplasty. If necessary, and that
is the case in most reflux patients, the hiatus should be decreased in size
by closing posteriorly both crural pillars. After establishing the gastroe-
sophageal window, a loop can be placed around the gastroesophageal
junction to pull the latter in any direction. To have the exposure of the area
dorsal to the esophagus, the gastroesophageal junction is pulled to the
left, which allows to look from the right to both crura. A nonresorbable su-
ture (2−0 or 0) is brought in and, usually, 2 oreven 3 sutures are necessary
to close the hiatus to the appropriate size. It is very important to checkthe
hiatal size after closing it posteriorly.This can be done byputting a grasper
into the hiatus next to the esophagus. A 10-mm instrument should easily
be able to pass into the mediastinum next to the esophagus.
9.2 Fundoplication and Partial Fundoplication Techniques
Partial Fundoplication Techniques
The dissection of the gastroesophageal junction should be the
same, regardless of what kind of final fundoplication or partial
fundoplication technique is used. The popularity for one or the
other fundoplication depends often on the surgical school or
country. For example, the Toupet partial fundoplication is very
popular in French speaking countries. The most popular partial
fundoplication in open operation is the Belsey Mark IV repair
which is, in the original version, always performed through a
left thoracotomy. Only very few authors have performed so far a
thoracoscopic Belsey Mark IV repair. Other partial fundoplication techniques have all been performed laparoscopically in the
past few years.
Laparoscopic Hill Procedure (Fig. 9.2.14)
The dissection of the gastroesophageal junction at the hiatus as
well as of the crura is similar in all procedures. For the laparoscopic Hill procedure a more extensive dissection of the tissue
connections of the posterior gastric wall and the lesser curvature
is necessary to clearly identify the arcuate ligament, ventrally, to
the aorta. This can be achieved by directing the dissection from
the right crus further caudally towards the celiac trunk, until the
anterior and posterior “gastric bundle” is identified. The latter is
the perigastric tissue that attaches the proximal stomach
towards the retroperitoneum. This tissue bundle carrying small
vessels, fat, peritoneal layer and the vagus branches is grasped
and then sutured to the arcuate ligament and hiatal crura which
are also closed as described previously.By this technique, the gastric fundus is also pulled around the distal esophageal area, thus
mechanically augmenting the sphincter area.
6. Identification of the right hiatal crus and further, by continuing the dissection towards the left, identification of the anterior hiatal arch (Fig. 9.2.8).
7. Separating the right crus from the esophagus by advancing
with a swab, grasper or scissors into the lower mediastinum;
very important: crucial landmark of dissection is not the
esophageal wall, but the right crus and the hiatal arch as well
as, later in the dissection, the left crus to find the correct dissection layer to reach the low mediastinum. If one tries to use
the esophageal wall as a landmark for the dissection way
around the esophagus, a false passage can mean immediate
laceration of the esophageal wall. Therefore it is very impor-
tant to first identify and dissect the right crus, the hiatal arch
and the left crus. Once these landmarks are identified, the
proximal stomach and the distal esophagus will automatically
emerge as mobile structures even in adipose patients with a
large esophageal fat pad.
8. Identification of the vagal trunks and branches in the distal
esophagus.
9. Dissection and creation of the so-called “gastroesophageal
window” deep to the posterior esophageal wall, which allows
four important steps:
1. view of the left side to the upper pole of the spleen and the
left crus,
2. exposure of both crura to perform a posterior hiatal clo-
sure,
3. safe looping of the lower esophagus (Fig. 9.2.10),
4. a safe pull-through of the mobilized fundus (Fig. 9.2.11 +
12).
10. Mobilization of the gastric fundus and greater curvature by
dividing the short gastric vessels: This can be achieved by
single or double clipping of both the gastric and splenic sides
of the vessels, by the use of laparoscopic stapling devices or
harmonic scalpel or by bipolar coagulation (cave: thermal in-
jury of the greater curvature and gastric wall necrosis)
(Fig. 9.2.9).
11. Dissection of the left hiatal crus.
12. Division of posterior gastric artery usually coming from the
splenic artery in patients in whom this vessel limits the mobil-
ity of the posterior wall of the gastric fundus.
13. Partial closure of the hiatus posteriorly by approximating the
left and right crus using non resorbable sutures (0-metric)
(Fig. 9.2.10).
14. Transoral introduction of a 45−60 French bougie for calibration to have a minimum diameter around which a wrap is sutured. Once the bougie is in place in the gastric lumen, a posterior and anterior fundic flap should be pulled around the location of the distal esophageal sphincter without any tension.
The pulled-through posterior flap to the right side should stay
in that position when the grasper is removed. The latter is a
good sign for a tensionfree pull-through.
15. The final stage of the Nissen fundoplication is performed with
the bougie in place using 1−3 stitches with non-absorbable
suture material to complete a floppy and short wrap
(Fig. 9.2.3). The looseness of the wrap should be checked
after the suture is finished by introducing a grasper comfortably between wrap and esophagus containing the bougie.
16. Removal of the trocars, if possible under visual control.

Complications
253
Fig. 9.2.11+12 Nissen fundoplication. The posterior aspect of the fundus
is picked up with the grasper exactly at the position which should be used
to have a symmetric wrap around the lower esophageal sphincter. The
gastroesophageal junction is pulled ventrally and the grasper will pull the
flap of the fundus dorsally and from the left to the right side. There, the
flap is advanced further and picked up by a second grasper for the pull-
Laparoscopic Toupet Procedure (Fig. 9.2.3)
For the laparoscopic Toupet procedure, dissection of the
gastroesophageal junction, the hiatal crura as well as the proximal fundus is necessary as described above. The fundic flap is
pulled through the gastroesophageal window from the left to
the right side. Then the fundus is fixed to the right diaphragmatic crus with 2−3 sutures. In addition, the fundus is then su-
tured to the distal esophagus, again 3 sutures on the right side
of the esophagus and 3 sutures on the left side of the esophagus.
Care is taken not to damage the vagal trunks.
Anterior 180° Hemifundoplication (Fig. 9.2.15)
Several procedures of anterior partial fundoplication are
known, such as the Dor fundoplication, Lind fundoplication and
Watson fundoplication. The Dor technique is usually used for
coverage of a myotomy for achalasia at the anterior aspect of the
distal esophagus and proximal stomach. In the Watson technique the fundus is attached over a few centimeters to the left
anterior aspect of the distal esophagus covering approximately
90° of the circumference. Depending on the author, these partial
anterior fundoplications cover between 90 and 180° of the anterior aspect of the esophagus. In patients with reflux disease and
an impaired esophageal peristalsis an anterior 180° hemifundoplication has been very effective in both preventing postopera-
tive persistent dysphagia and reflux. Only a limited fundic mo-
bilization is necessary to perform an anterior partial fundopli-
cation (Fig. 9.2.17). The mobilization of the fundus must involve
a limited portion, to the level of the upper pole of the spleen.
After finishing the posterior hiatoplasty, the anterior flap of the
through to the appropriate extent. It is very important for an optimal
postoperative function that the correct part of the proximal stomach is
pulled through the posterior gastroesophageal “window”. An alternative
method is to push through to the right side. In this case the grasper is
moved behind the esophagus from the left, carrying the posterior aspect
of the fundus and pushing it over to the right side.
fundus is pulled over to the right crus. The upper border of the
fundic flap is sutured to the distal esophagus (high pressure
zone area) with 3 non-resorbable sutures. Attaching the anterior fundic flap with one stitch to the left side of the esophagus,
one to the middle and one to the right side of the esophageal
wall, the hemifundoplication is completed. An additional one or
two stitches are used to fix the fundic flap to the right crus in
order to assure the abdominal maintenance of the wrap.
Complications
Intraoperative Complications
Injury to the esophagus, cardia and stomach
Therapy: If there is an intraoperative suspicion of a wall laceration, a test with methylene blue solution should be performed.
Full thickness interrupted suture should be used to closed the
defect and it should be covered with the fundus during fundoplication. Drainage of the abdomen is the surgeon’s choice.
Prevention: During the dissection of the gastroesophageal junction it is important to focus on identification of the right crus,
the hiatal arch and then the lower mediastinum, rather than the
esophageal wall. Once both right and left crus and the hiatal
arch are dissected free, the esophagus usually is free and can be
palpated with the laparoscopic instruments without any problem. The dissection of the posterior aspect of the esophagus
above the aorta is facilitated by dissection with blunt instruments.

254
9.2 Fundoplication and Partial Fundoplication Techniques
Fig. 9.2.13 Completed Nissen Rossetti Fundoplication.
Spleen injury
Therapy: Bleeding from small decapsulations or lacerations can
be stopped with electrocautery, compression or laser coagulation. The surgeon should decide quickly whether the bleeding
has a high probability of getting under control by laparoscopic
means. If there is any doubt, immediate conversion is obligatory.
Prevention: Usually spleen injuries occur during the mobilization of the fundus. Only a very careful dissection technique
using gentle blunt dissection, clip applications, and if possible a
harmonic scalpel help prevent splenic injury.
Liver injury
Therapy: Compression, laser coagulation, electrocautery, local
hemostyptics, fibrin sealant or suture.
Prevention: Liver injuries occur when the retractor is not
handled in the proper way or when the liver tissue is very
fragile. Only gentle manipulation and frequent control of the
correct usage of the retractor can prevent liver laceration.
Fig. 9.2.14 Laparoscopic Hill procedure.
Pneumothorax
Therapy: A chest tube can be placed intraoperatively if there are
respiratory problems.
Prevention: Careful dissection without damage.
Postoperative Complications:
Dysphagia
Temporary dysphagia in the first postoperative weeks is not a
complication, but a rather normal finding in patients after Nissen fundoplication. Patients should be informed about this
problem prior to surgery. A real complication is persistent dysphagia continuing for months after surgery.
Therapy: flexible endoscopic bouginage should be the first step.
In severe cases with persistent dysphagia even after 3 months
and bouginage treatment, a relaparoscopy can be considered by
a well-experienced surgeon and correction of the wrap might
be possible. If there is any doubt about the anatomy or limited
exposure because of adhesions, a conversion is necessary.
CO2emphysema
Therapy: CO2emphysema is not a major problem when it is re-
stricted to the trunk. If it is moving up into the neck and head, a
conversion must be considered before skin damage occurs, e.g.,
at the eyelids:
− after reduction of intraoperative pressure to 8 mm Hg,
− postoperative respirator therapy for 2−3 hours until the CO
emphysema dissolves.
Prevention: Low intraabdominal pressure reduces CO
insuffla-
2
tion. If in old patients the tissue seems to be very fragile and in
cases where an extensive dissection of the lower mediastinum
is necessary, CO
emphysema of the head and neck is likely. In
2
these cases, the mobilization of the fundus can be performed
before dissecting the lower mediastinum thus reducing the
operating time, once the CO
emphysema starts developing.
2
Recurrent or persistent reflux
Persistent reflux is usually due to a misplaced wrap around the
proximal stomach. It can also occur in the early postoperative
weeks because of early disruption of the wrap.
Therapy: relaparoscopy by a well-experienced laparoscopic surgeon and redo.
2
Slipped Nissen
Currently, this phenomenon occurs rarely, since the wrap is
usually correctly anchored to the esophagus. Another method of
reducing the incidence of this complication is an only limited
dissection of the gastroesophageal junction at the lesser curvature which allows only a small space for the passage of the
wrap. As a consequence, the wrap, when placed correctly, cannot move.

Fig. 9.2.15 Anterior 180° hemifundoplication.
255
Denervation symptoms
This complication is not very frequent. Usually, gastric emptying problems are clinically prominent. It can be also associated
with temporary diarrhea and abdominal discomfort.
Therapy: Prokinetic medication, if not successful surgical reintervention. Postoperative care: A nasogastric tube should be left
in the stomach for 2 days for removal of air and fluid from the
gastric lumen to prevent gastric dilation and early disruption of
the wrap. It is a high probability that the patient will have early
postoperative dysphagia because of a tissue edema at the
gastroesophageal junction. Again, the patient needs to be informed about this problem prior to surgery. In the postoperative
follow-up after 3 months or half a year an upper GI endoscopy
should be performed to document persistance or healing of
esophagitis.
Re-do fundoplications
These operations are technically very demanding, especially if
an extensive dissection has been performed during the first
operation. However, adhesions after gastrofundophrenicopexy
or other pexy procedures can be very limited, so explorative laparoscopy to check out the situation at the hiatus after previous
operation can be reasonable. However the level of changeover
to conversion must be very low. If there are severe adhesions
and especially if the gastroesophageal junction is embedded
into scar tissue, differentiation between the esophageal wall,
vagus nerves, crura and scar tissue cannot be possible. For this
kind of dissection the surgeon needs all tactile capacities of the
human fingers. Therefore an early conversion or a primary plan
to do a laparotomy is necessary.
9.3 Comments on Nissen Fundoplication
J. H. Peters, T. R. DeMeester
The surgical treatment of gastroesophageal reflux disease is expanding rapidly, largely due to the advent of laparoscopic techniques. Drs. Pier, Götz, and Fuchs have, in general, superbly outlined the indications, patient selection and techniques of antireflux surgery as practiced in their respective departments. Two
areas deserve specific comment; the indications for antireflux
surgery and the technique of fundoplication.
Drs. Pier and Götz (chapter 9.1) suggest that surgical therapy be
restricted to patients “refractory to long term non-operative
treatment or severe esophagitis (grades III and IV).” We would
like to outline a more cause and effect specific approach. The
non-operative management of gastroesophageal reflux disease
is improving rapidly. Most candidates for operation can, and
should, be controlled with proton pump inhibitors prior to an-
tireflux surgery. Indeed, one should be cautious in operating on
a patient who has no response to medical treatment. Further,
limiting antireflux surgery to patients with severe esophagitis
denies a large population of patients who are excellent candi-
dates for laparoscopic fundoplication the opportunity for cure
of their disease with a single intervention.
For these reasons we have moved away from the concept of
failure of nonoperative therapy as an indication for antireflux
surgery. Rather, patients with symptoms of reflux disease are
thoroughly evaluated with respect to their risk of persistent and
progressive disease. Patients presenting for the first time with
symptoms suggestive of gastroesophageal reflux may be given
initial therapy with H2 blockers. Failure of H2 blockers to control the symptoms, or immediate return of symptoms after
stopping treatment suggests either that the diagnosis is incorrect, or that the patient has relatively severe disease. Endoscopic examination at this stage of the patient’s evaluation provides the opportunity for early identification of complications
including Barrett’s esophagus. Twenty four hour esophageal pH
monitoring should be pursued to confirm the diagnosis.
Esophageal manometry and/or ambulatory esophageal bile
monitoring should also be done to identify features predictive
of severe disease such as a mechanically deficient lower
esophageal sphincter and duodenogastroesophageal reflux. In
the absence of risk factors predictive of severe disease and
esophageal mucosal injury, the patient’s symptoms may be controlled with proton pump inhibitors. Factors that predict a poor
response to medical therapy, frequent relapses, and the
development of complications include; supine reflux, erosive
esophagitis or a columnar lined esophagus at initial presentation, bile in the refluxate, and a structurally defective sphincter.
Patients who have these risk factors should be given the option
of surgery as a primary therapy with the expectation of long
term control of symptoms and complications.
Although many internists and surgeons are reluctant to advise
operation in the absence of demonstrable esophagitis, we

256
9.3 Comments on Nissen Fundoplication
believe that one should not be deterred from considering antireflux surgery in a symptomatic patient provided the disease
process has been objectively documented by 24 hour pH monitoring. This is particularly true in patients who have become dependent upon therapy with proton pump inhibitors. Indeed, investigations of the natural history of GERD in the absence of
esophagitis have demonstrated return of symptoms in the ma-
jority of patients following cessation of nonoperative therapy.
The second area of comment relates to evolving concepts in the
selection and technique of antireflux procedures. Antireflux
surgery appears deceptively simple. Achieving a ninety plus
percent success rate is not as easy as it seems. Successful out-
come is highly dependent upon the meticulous performance of
the appropriate antireflux procedure in each patient. We would
agree with the selection of the various antireflux procedures as
outlined by Dr. Fuchs (chapter 9.2). Our experience suggests
that in approximately 80−90 percent of patients referred for
operative treatment a transabdominal Nissen fundoplication is
the most suitable procedure. The remaining 10−20 percent of
patients are best treated with an antireflux procedure tailored
to their underlying anatomic and functional abnormalities. This
approach results in excellent long-term relief of reflux symp-
toms across the whole spectrum of disease.
In addition, we believe that the advent of the laparoscopic ap-
proach provides an ideal opportunity for standardization of the
technique of Nissen fundoplication. The essential elements nec-
essary for the performance of a transabdominal fundoplication
are common to both the laparoscopic and open procedures and
include the following:
1. Crural dissection, identification and preservation of both
vagi including the hepatic branch of the anterior vagus.
2. Circumferential dissection of the esophagus.
3. Crural closure.
4. Fundic mobilization by division of short gastric vessels.
5. Creation of a short, loose fundoplication by wrapping the
anterior and posterior wall of the fundus around the lower
esophagus.
Although, considerable argument continues regarding the significance of technical aspects such as the orientation of the fundoplication and the need for fundic mobilization and crural clo-
sure, there is growing evidence of the importance of these tech-
nical features. Several authors have compared the incidence of
dysphagia following laparoscopic Nissen fundoplication with
short gastric vessels’ division, to dysphagia following the
Rosetti/Hell modification of Nissen’s (no division of short gastric
vessels) procedure. Evaluation of these data would support the
contention that complete fundic mobilization by division of
vessels is important to allow construction of a tension free fundoplication.
Despite the fact that optimal results following open antireflux
surgery have included routine crural closure, the need for crural
closure in every patient continues to be debated. Although no
prospective randomized studies exist, data are emerging to reinforce the need for crural closure in all patients. Watson and
Jamieson have reported upon a 6.7% incidence of acute para-
esophageal herniation following laparoscopic fundoplication.
Herniation occurred at a mean of 1 month after operation, and
occurred in patients in whom crural closure was omitted. Of the
17 patients developing the complication only 3 had a hiatal re-
pair and then only with a single stitch. Ten of the 17 patients required re-operation.
Our early results stimulated us to look more closely at how we
constructed the fundoplication with the laparoscopic approach.
Despite calibration with a 60F bougie, laparoscopic fundoplication may result in a fundoplication that is tighter than that constructed during an open procedure. One of the reasons may be
the manner in which the wrap is constructed. Ideally, the anterior and posterior fundic wall should be folded around the distal
esophagus to meet in the right lateral aspect of the esophagus
rather than at the anterior or 12 o’clock position. Rudolph Nissen described this as a “Witzel” of the distal esophagus into the
stomach. This can be difficult to achieve laparoscopically unless
attention is given to complete fundic mobilization and the careful grasping of the posterior fundus to pass behind the
esophagus. This is best accomplished by placing a Babcock
clamp through the lower left port (surgeon’s right) and grasping
a suitable portion of the posterior fundus. It is then passed behind the esophagus from left to right and grasped with the surgeon’s left hand. We prefer this approach to the more common
method of passing an instrument from right to left, as it assures
passage of the posterior fundus and not the anterior one, during
creation of the fundoplication. The anterior wall of the fundus is
then folded over the esophagus above the gastroesophageal fat
pad. Both posterior and anterior fundic lips are manipulated to
allow the fundus to envelope the esophagus without twisting.
With adequate dissection the right portion of the fundoplication should rest comfortably in place without the tendency to
retract back toward the spleen. A 60F bougie is then carefully
passed under direct vision to properly size the fundoplication.
The relative roles of medical versus surgical therapy for gastroesophageal reflux disease are changing, as both patients and
their physicians recognize that surgery provides the only
known means of altering the natural history of the disease. At
present, it seems that laparoscopic Nissen fundoplication will
become the standard of care in the surgical treatment of
patients with gastroesophageal reflux disease. Although laparoscopic fundoplication results in control of typical symptoms in
over 90% of patients, it should be recognized that achieving optimal results requires careful patient selection and attention to
meticulous technique. The lessons of history have allowed improvements in antireflux surgery to the point that excellent results can be achieved in the majority of patients. It is hoped that
in this era of minimally invasive surgery, mistakes of the past
will not be repeated by those who are unfamiliar with the
pathophysiology of the disease or the history of antireflux
surgery.
Bibliography
Hunter JG, Swanstrom L, Waring JP. Patterns of dysphagia following laparo-
scopic antireflux surgery. Ann. Surg. 1996; 224:51−57.
Jamieson GG, Duranceau A. What is a Nissen fundoplication. Surg. Gynecol.
Obstet. 1984; 159:591:593.
Kauer WKH, Peters JH, DeMeester TR, Heimbucher J, Ireland AP, Bremner CG.
A tailored approach to antireflux surgery. J. Thoracic Cardiovasc. Surg.
1995; 110:141−147.
Klinkenberg-Knoll EC, Festen HPM, Jansen JBMJ, Lamers CBHW, Nelis F, Snel
P, Luckers A, Dekkers CPM, Havu N, Meuwissen SGM. Longterm treatment
with omeprozole for refractory reflux esophagitis; efficacy and safety.
Ann. Int. Med. 1994; 121:161−167.
Nissen R. Gastropexy as the lone procedure in the surgical repair of hiatus
hernia. Am. J. Surg. 1956; 92:389−392.
Peters JH, Heimbucher J, Kauer WH, Incarbone R, Bremner CG, DeMeester
TR. Clinical and physiologic comparison of laparoscopic and open Nissen
fundoplication. J. Am. Coll. Surg. 1995; 180:385−393.
Watson DI, Jamieson GG, Devitt PG, Mitchell PC, Game PA. Paraesophageal
hiatus hernia; an important complication of laparoscopic Nissen fundoplication. Br. J. Surg. 1995; 82:521−523.

9.4 Gastropexy in Paraesophageal Hiatus Hernia Repair
F. Köc kerling
257
Goals and Methods
Paraesophageal hiatus hernias account for about 10% of all diaphragmatic hernias. This type of hernia is characterized by displacement of part of the fundus or of the entire stomach into the
mediastinal space next to the esophagus in the absence of axial
dislocation of the cardia (upside-down stomach).
Complications must be expected often after the disorder has
been asymptomatic for a long period of time. In addition to
chronic anemia, occult bleeding, and impaired motility, lifethreatening complications such as perforation, strangulation,
gangrene, obstruction, or pulmonary problems may occur in
27% of patients. Due to the high morbidity and mortality as-
sociated with these complications, operative repair is indicated
whenever a paraesophageal hiatus hernia is diagnosed.
Gastropexy with closure of the esophageal hiatus has proven to be
a safe and effective surgical procedure. Generally, the part of the
stomach that has herniated into the chest is easily reduced, be-
cause adhesions are rare. These circumstances represent a
favorable situation for laparoscopic gastropexy. After replacing
the stomach into the abdomen, the surgeon sutures the fundus
to the inferior aspect of the diaphragm and repairs the diaphragmatic esophageal hiatus. This procedure requires that the
surgeon be sufficiently accomplished in the laparoscopic
manipulation of hollow organs and in laparoscopic suturing and
knot-tying techniques.
Note: Preoperative diagnostic studies must confirm the absence
of a composite hiatus hernia, since cephalad expansion of a primarily axial sliding hernia can result in paraesophageal displacement of parts of the fundus of the stomach. In such cases,
gastropexy must be combined with a Nissen fundoplication.
patient must be made aware of possible problems of laparotomy. If prosthetic material is to be used to close the defect
of the enlarged esophageal hiatus, the problems of implanting a
nonabsorbable mesh should be discussed. The patient should
also be informed of the general level of experience with laparoscopic gastric surgery at a given institution.
Special Preparations
As in all procedures in the region of the lower esophageal
sphincter, a large nasogastric tube is placed to identify the
esophagus. An indwelling urinary catheter is indicated, as in
any procedure involving the establishment of a pneumoperitoneum with a Veress needle and the percutaneous placement
of the laparoscope/camera trocar.
Anesthesia
General anesthesia.
Patient Position; Position of the
Operating Team and Equipment
(Figs. 9.4.1 and 9.4.2).
Trocar Placement
(Fig. 9.4.3).
Indications
Gastropexy is indicated in the presence of a confirmed paraesophageal hiatus hernia (which may remain asymptomatic)
with only partial displacement of the stomach into the chest.
Contraindications
− Paraesophageal hernias with complications (bleeding,
necrosis of the stomach wall, ulceration, or perforation).
− Displacement of the entire stomach into the chest (upsidedown stomach).
− Presence of general or anesthetic laparoscopic risk factors.
Surgical Risks and Patient Information
Complications of gastropexy and repair of the esophageal hiatus
in general must be discussed with the patient, who must also be
informed of the specific laparoscopic complications involved
with establishing the pneumoperitoneum and placing the tro-
cars. The patient must also be aware that if laparoscopic reposi-
tioning of the stomach is not feasible or intraoperative compli-
cations such as bleeding or perforation of a hollow viscus occur,
the surgeon must convert to an open procedure. Therefore the
Complications
Intraoperative Complications
− Arterial or venous bleeding from placement of the Veress
needle or laparoscope/camera trocar.
Fig. 9.4.1 Gastropexy. Positioning.
The patient is placed supine with the left arm tucked to his or her side and
the right arm extended to facilitate intravenous access. After establishing
the pneumoperitoneum and inserting the laparoscope/camera trocar,
move the patient into a 20−30° reverse Trendelenburg position so that
the greater omentum, transverse colon, and small intestine fall into the
lower abdomen.
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