Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3879_Библиотеки_им_академика_М_И_Перельмана

Constructive pericarditis, which has been described as “Pericardial adhesion”, “Chronic pericardial adhesion”, has also been named as callous, calcified or ossi­fied pericardium, “Concertio cordis cum pericarditis”, as a result of knowledge, experience and observations on this subject. This clinical syndrome is also known as “Pick’s disease” []. In  Lower described the clinical effect of the constrictive pericardium on the diastole of the heart in detail []. Based on the autopsy findings of a -year-old woman with pericardial adhesion in , Lower stated that the pericardium, which should have been thin/translucent, was thickened, opaque and hardened (callous), which would limit the movements of the heart. J. Mayow, in , described a pericardial adhesion as “as if the heart was surrounded by cartilage and stuck to the front”, and reported that this condition prevented blood return to the ventricle []. T. Bonet, in , said that pericardial adhesion was the cause of palpitation and used the term (cordis tremor) []. R. De Vieussens mentioned the effect of pericardial adhesions on cardiac functions in two cases in  and , and stated that the adhesions were of inflammatory origin, not congenital []. A. von Haller described pericardial calcification in  based on autopsy findings []. G. B. Morgagni described pericardial adhesions and calcifications in  cases in  and gave information about the physiopathology and clinic of constructive pericarditis []. In , R. T. H. Laénec detected calcification between the pericardial leaves in the autopsy of a -year-old patient with exertional dyspnea but no orthopnea, with cyanotic lips []. N. Chevers explained diastolic dysfunction in constructive pericar­ditis and its clinical picture for the first time in  []. Wilks explained constructive pericarditis in detail in  []. A. Kusmaul reported in  that venous filling in the inspiratory increased in constrictive pericarditis (Kusmaul’s sign) []. J. M. Charcat reported constructive pericarditis due to rheumatoid arthritis in  []. F. Pick mentioned “pseudo cirrhosis” (Pick’s disease) resulting from right heart fail­ure in  []. G. Daniel and S. Puder drew attention to the relationship between hemopericardium and constrictive pericarditis in  []. T. H. Sellors in  [] and P. While in  indicated tuberculosis as the primary cause of constructive pericarditis []. W.G. Bigelow et al. in  [], H. B. Schumaker Jr. and Rose [] in , and Fitzpatrick et al. [] in  reported that radical pericardiectomy was the only method to prevent recurrence of pericardial constructions. C.A. Bush et al. reported that constrictive pericarditis can disrupt hemodynamics even without adherence to the epicardium [] E. Weil predicted the excision of the thickened fibrous pericardium in constructive pericarditis in , and E. Delorme also showed on cadavers that pericardial decortication could be applied in  [, ]. E. Rehn performed experimental pericardiectomy in , performed pericardiectomy in  cases of constrictive pericarditis in humans in , and reported that this was the treatment method of choice []. It was predicted by C. S. Beck that this type of intervention could be performed in  [] and helped in the development of the pericardiectomy technique from  []. C. S. Beck reported in  as a result of his experimental studies that the removal of the thickened pericardium provided hemodynamic improvement [].