Добавил:
Sekretar
kiopkiopkiop18@yandex.ru
t.me/Prokururor I Вовсе не секретарь, но почту проверяю
Опубликованный материал нарушает ваши авторские права? Сообщите нам.
Вуз:
Предмет:
Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3798_Библиотеки_им_академика_М_И_Перельмана.pdf
X
- •Lymphedema
- •Foreword
- •Preface I
- •Preface II
- •Contents
- •Contributors
- •Clinical Presentation
- •Lymphedema Staging
- •Diagnosis
- •Therapy
- •Physical and Non-Operative Therapy
- •Operative Therapy
- •Introductory Note
- •Primary Lymphedema
- •Secondary Lymphedema
- •Complications of Lymphedema
- •Conclusions
- •References
- •Embryological Development of the Lymphatic System
- •Lymphedema
- •Lymphangioma
- •Protein-Losing Enteropathy and Intestinal Lymphangiectasia
- •Complex Vascular Malformations
- •Infectious Diseases
- •Lipedema
- •Lymphangioleiomyomatosis
- •References
- •Introduction
- •Molecular Lymphology
- •Work-up
- •Syndromes
- •Chromosomal Aneuploidies and Sporadic Syndromes
- •Conclusion
- •References
- •References
- •Anatomical
- •Functional
- •Lymph Flow Pathways
- •Skin and Subcutaneous Tissue
- •Gut Lymphatics
- •Lung Lymphatics
- •References
- •References
- •References
- •Tissue Fluid
- •Lymph
- •Physiological Observations
- •Proteins in Obstructive Lymphedema
- •Lymph Cytokines in Obstructive Lymphedema
- •References
- •Tissue Fluid Pressure and Flow
- •Pressures in the Normal Limb
- •Pressures in the Lymphedema
- •Normal Tissue Fluid Flow
- •Tissue Fluid Flow in Lymphedema
- •Lymph Pressure and Flow
- •Extrinsic Factors that Propel Lymph
- •Normal Conditions
- •Lymphedema Conditions
- •Intrinsic Factors that Propel Lymph
- •Pressures in Lymphedematous Limbs
- •Lymph Flow in Normal Limbs
- •Lymph Flow in Lymphedematous Limbs
- •General Remarks
- •References
- •Immune processes in lymphatics and nodes
- •Remarks
- •References
- •General Considerations
- •Clinical Diagnosis
- •Associated Disorders
- •When Further Investigation Is Needed
- •References
- •References
- •Conclusion
- •References
- •References
- •Consensus Documents
- •Consensus Documents in the Treatment of Lymphedema
- •International Society of Lymphology
- •International Lymphedema Framework
- •Italian
- •Latin American
- •Australian
- •American Cancer Society
- •National Lymphedema Network
- •Summary
- •Concluding Thought
- •Disclosure
- •References
- •Signs to Look for at Presentation
- •References
- •Introduction
- •Clinical Diagnosis
- •Differential Diagnosis
- •Introduction
- •Differential Diagnosis: Other Reasons for a Swollen Limb
- •Differentiating the Lymphedemas
- •Filarial Lymphedema
- •Malignant Lymphedema
- •Factitious Lymphedema
- •Primary Lymphedema
- •When a Patient Might First Present
- •Risk Factors to Consider at Presentation
- •Laboratory Diagnosis
- •Waist-to-Height Ratio
- •Streeten Test
- •Capillary Fragility Assessment
- •Assessment of Aortic Distensibility and Stiffness in Lipedema
- •Pain Perception Assessment
- •Ultrasound Examination
- •CT and MRI Examination
- •Lymphoscintigraphy and Fluorescent Microlymphography
- •Clinical Management
- •Prognosis
- •References
- •General Considerations
- •When Clinical Examination Should Be Complemented by Imaging
- •Methods to Evaluate Lymph Flow, Lymphatic Vessels, and Lymph Nodes
- •Methods of Evaluating Tissue Changes
- •References
- •Brief Historical Note
- •Materials and Methods
- •Interpretation and Comments
- •Primary Lymphedema
- •Secondary Lymphedema
- •Lymphatic Filariasis
- •Kaposi Sarcoma
- •Klippel–Trenaunay and Other Lymphangiodysplastic/Mixed Syndromes
- •The Future
- •Conclusions
- •References
- •References
- •Introduction
- •Lymphoscintigraphy and/or SPECT-CT Lymphoscintigraphy
- •Lymphoscintigraphy or SPECT-CT Lymphoscintigraphy in Relation to the Clinical Presentation of the “Simple” Lymphedematous Situations
- •In Primary Lower Limb Lymphedemas
- •In Secondary Lymphedemas
- •Lymphoscintigraphy to Demonstrate the Collateralization Pathways
- •Lymphoscintigraphy, Lymphoceles, and Lymphangiomas?
- •X-Ray Computed Tomography?
- •Positron Emission Tomography or Positron Emission Tomography Combined with X-Ray Computed Tomography?
- •Magnetic Resonance Imaging and/or Lymphangio-MRI with Injection of Contrast Enhancement?
- •Magnetic Resonance Imaging in the Diagnosis of Pathologically Positive Lymph Nodes?
- •Heavily T2-Weighted Imaging or Magnetic Resonance Lymphangiography for Lymphedemas?
- •MRI or MRL in Lymphedemas?
- •MRI and Lymphangiomatosis?
- •MRI and Lymphangiomas?
- •Lymphoscintigraphy and/or MRI?
- •Conclusions
- •References
- •Visual Lymphography and Radiological Lymphography
- •Radiological Lymphography
- •Oil Contrast Lymphography
- •References
- •Microlymphography in Healthy Individuals, in Chronic Venous Disease, and in Lymphedema (Table 23.1)
- •Measurement of Microlymphatic Pressure
- •Lymphatic Vasomotion and Lymphatic Flow Motion
- •References
- •Measurement of Fibrotic Induration
- •Measurement of Fluid Content
- •Measurement of Limb Volume and Circumference
- •Measurement of Functional Status of the Lymphatic System
- •Measurement of the Structural Status of the Lymphatic System and of the Limb
- •Measurement of the Status of the Vascular System
- •Measurement of the Subjective Parameters
- •Treatment Outcomes
- •References
- •General Overview
- •Primary and Secondary Infections
- •Primary Infections
- •Secondary Infections: Dermato-Lymphangio-Adenitis
- •Chronic Dermatolymphangioadenitis
- •Acute DLA
- •Differential Diagnosis of Lymphangitis, Erysipelas and Dermato-Lymphangio-Adenitis
- •Bacteriology of Lower Limb Skin
- •Bacterial Flora of Normal Foot and Calf Skin
- •Bacterial Flora of Normal Leg Lymph
- •Bacterial Flora of Lymphedematous Leg Lymph
- •Sensitivity of Isolates to Antibiotics
- •Prophylaxis of Recurrent DLA
- •Chronic DLA
- •Treatment of Acute DLA Attacks
- •References
- •Introduction
- •Sites of Accumulation of Lymph and Tissue Fluid in Lymphedema
- •Morphological Changes in the Lymphedematous Skin and Subcutis
- •Hydraulic Conditions in the Subcutaneous Tissue
- •Pressures
- •Pressure Gradient Across Skin and Subcutaneous Tissue
- •Conditions for Creating Centripetal Tissue Fluid Flow
- •Manual Massage
- •Indications
- •Advantages and Shortcomings
- •Manual Massage Hydraulics
- •Pneumatic Massage
- •Indications
- •Advantages and Shortcomings
- •Pneumatic Compression Hydraulics
- •Remarks for Users of Compression Devices
- •References
- •Introduction
- •Complete Decongestive Physiotherapy
- •The Use of CDP
- •Long-Term Therapy Results
- •References
- •Introduction
- •Detailed Characterization of MLD According to Dr. E. Vodder
- •Stationary Circle
- •Rotary Stroke
- •Pump Stroke
- •Scoop Technique
- •Additive Manual Techniques
- •Indication and Contraindication
- •References
- •Introduction
- •Investigations
- •References
- •Graduated Compression Garments
- •Multilayered Bandage Compression
- •Intermittent Pneumatic Compression
- •Impact of Compression Therapy upon Lymphedema Outcomes
- •References
- •References
- •Conservative Therapies for Secondary Lymph Edema
- •Contemporary Treatments
- •The Groupings of Contemporary Treatments
- •Methods
- •Pharmacogenomics and Medications Targeting the Lymphatic System
- •Low-Level Scanning and Hand-Held Laser
- •Lymphatic Drainage Massage Delivered by Partners/Carers and Mechanically
- •Mild Exercise (Tai Chi)
- •Moderate Exercise (In and Out of Water)
- •Electro-Stimulation
- •Tissue Manipulation
- •Kinesio-Taping
- •Diet (Mid-Chain Triglycerides) and Abdominal Issues
- •Placebo
- •References
- •Antibiotics
- •Conclusion
- •References
- •Introduction
- •General Considerations
- •Intermittent Pneumatic Compression
- •Compression
- •Use of Elastic Bandages
- •Special Compression Material
- •Medical Compression Stockings
- •Exercise
- •Lymphedema Severity-Adapted Forms of CDP
- •Stage I Lymphedema
- •Stages II and III Lymphedema
- •References
- •Introduction
- •Lymphedema of the Arm
- •Considerations in Manual Lymph Drainage
- •General Considerations for Compression
- •Compression Therapy in the Arms
- •References
- •Introduction
- •Physical Treatment of Lymphedema of the Face and Neck
- •Manual Lymph Drainage (Leduc Method)
- •Description of the Maneuvers
- •Protocol for Manual Treatment of Lymphedema of the Face and Neck
- •Multi-Layered Bandaging Leduc Method
- •Stimulation of Muscular Activity
- •Compression Garment
- •Education in Precautions to Apply to Avoid Exacerbation of Symptoms
- •Education in Self-Treatment
- •An Example of Self-Treatment of Head and Neck Lymphedema
- •Rehabilitation to Address Functional Impairments
- •Quality of Life
- •References
- •Introduction
- •Anatomy
- •Etiology
- •Diagnosis
- •Clinical Course
- •Treatment
- •Surgical
- •References
- •References
- •Lymphovenous Microsurgical Shunts in Lower Limbs
- •Lympho-Venous Shunts (1966–2010)
- •Pre- and Post-operative Pharmacological Treatment
- •Postoperative Physiotherapy
- •Postoperative Evaluation Criteria
- •Objective Indirect Methods for the Evaluation of the Function of the Lympho-Venous Shunt
- •Direct Methods for Evaluation of Function of Lympho-Venous Shunt
- •Factors Adversely Affecting the Patency of Lymph-Venous Shunts
- •Local
- •Distant
- •Factors Affecting Evaluation of Clinical Results
- •Results in General
- •References
- •Principles
- •Indications
- •Microsurgical Reconstructions
- •Lymphovenous Anastomosis
- •Lymph Node-to-Vein Anastomosis
- •Technique
- •Results
- •Lymph Vessel-to-Vein Anastomosis
- •Microsurgical Technique
- •Results
- •Lymphatic Grafting
- •Technique
- •Results
- •Lymph Node Transplantation
- •Technique
- •Results
- •Problems with Microvascular Lymphatic Reconstructions
- •Conclusions
- •References
- •General Considerations
- •Clinical Experience and Surgical Techniques
- •Results and Final Considerations
- •References
- •Introduction
- •Correlation With the Pathophysiology of Lymphedemas
- •Experimental Basis
- •Indications for Lymphatic Reconstruction Using Lymphatic Grafts
- •Operative Technique
- •Post-operative Procedures
- •Results
- •References
- •NodoVenal Shunt
- •Indications
- •Surgical Techniques
- •End-to-End Anastomosis
- •End-to-Side Anastomosis
- •Contraindications
- •Complications
- •References
- •Introduction
- •Secondary Lymphedema
- •Lymphedema of the Arm: Upper Extremity
- •Indication for Node Grafting
- •Operative Technique
- •Results
- •Plexopathy
- •Breast Reconstruction Combined with Lymphedema Treatment
- •Lymphedema of the Leg: Lower Extremity
- •Operative Technique
- •Results
- •Primary Lymphedema
- •Indications
- •Operative Technique
- •Results
- •Conclusion
- •References
- •Clinical Experiences (Personal)
- •Conclusion
- •References
- •References
- •Introduction
- •The Morphological Changes in Advanced Lymphedema
- •Indications for Debulking
- •Bacteriology of Skin and Deep Tissues
- •Surgical Technique
- •References
- •References
- •Clinical Experience
- •Conclusion
- •References
- •Excess Subcutaneous Adiposity and Chronic Lymphedema
- •The Outcome of Liposuction
- •How to Perform Liposuction for Lymphedema
- •Surgical Technique
- •Postoperative Care
- •Controlled Compression Therapy
- •Volume Measurements
- •When to Use Liposuction to Treat Lymphedema
- •Summary
- •Key Points
- •References
- •Extratruncular Lymphatic Malformation Lesions
- •Truncular Lymphatic Malformation Lesions
- •Clinical Evaluation
- •Clinical Management
- •Conservative (Physical) Therapy
- •Surgical Therapy: Reconstructive Surgery
- •Surgical Therapy: Ablative/Excisional Surgery
- •Liposuction: Circumferential Suction-Assisted Lipectomy
- •Prospect: Primary Lymphedema as Lymphatic Malformation
- •Conclusion
- •References
- •References
- •Diagnosis
- •Management
- •General Considerations
- •References
- •Medical Therapies for Chylorrhea
- •References
- •Introduction
- •Drainage Procedures
- •Image-Guided Approaches
- •Open Surgical Approaches
- •Treatment of Cutaneous Chylorrhea and Chylorrhagia
- •Treatment of Chylothorax
- •Treatment of Chylous Ascites
- •Summary
- •References
- •References
- •Morphology
- •Life Cycle
- •Pathology
- •Gross Pathology
- •Changes Attributed to Filariae
- •Changes Ascribed to Bacterial Infections
- •Immunology
- •References
- •Manifestations

37 Genital Lymphedema
309
Etiology
The etiology of genital lymphedema, except after surgery and irradiation, is usually
postinflammatory and is caused by prior infection by microbes in the perineal and
anal regions. The microbes entering the lymphatics are cocci in approximately 60%
and bacilli in 40%. The most common are Staphylococcus epidermidis and
S. aureus, Enterococcus, and Micrococcus. In the bacilli population Pseudomonas,
Acinetobacter, Proteus, Enterococcus, Enterobacter, and Corynebacteria dominate.
Why only a few individuals develop infections is unclear; however, genetic predisposition is suspected.
The bacterial inflammatory process is followed by obliteration of afferent lymphatics and fibrosis of the inguinal lymph nodes. This is why, in some cases of genital lymphedema, lymphedema of the lower limbs also is seen.
Diagnosis
Diagnosis of genital lymphedema is based on:
(a) Physical examination revealing edema of the penis, scrotum or labia, disfigure-
ment of these parts, erythema and increased temperature, and, not infrequently,
oozing of lymph from epidermal blisters.
(b) Lymphoscintigraphy with intradermal injection of T99-Nanocoll.
Differential diagnosis between postinflammatory, chyloperitoneum, and chylu-
ria with retroperitoneal backflow is based on:
(a) Histochemistry of oozing fluid: postinflammatory (presence of lymphocytes
and dendritic cells with few granulocytes); chyloperitoneum (high concentration of lymphocytes and large macrophages).
(b) Chemistry of the fluid: presence of lipids determines intestinal origin of fluid
(chyloperitoneum).
(c) Lymphoscintigraphy: postinflammatory (lack of absorption and flow of radio-
isotope from the skin of the swollen parts toward the inguinal area, chyloperitoneum (isotope accumulates in dilated lymphatics spreading toward the thigh,
pelvis, and retroperitoneal space, chyluria (dilated genital lymphatic with isotope flow to the retroperitoneum and kidney pelvis).
Clinical Course
Genital lymphedema develops relatively quickly because of the ready accumulation
of tissue fluid in loose subcutaneous tissue. The main complications are: (a) rapid
increase in mass to several kilograms, (b) lymph oozing from epidermal blisters,
and (c) recurrent attacks of acute inflammation followed by chronic inflammation.

310 W.L. Olszewski
Treatment
Conservative: (a) in each case administration of either long-term penicillin 1,200,000
IU, intramurally, every 3 weeks or amoxicillin + clavulanic acid 2 g for 3 days orally
every 3 weeks, cleansing with antibacterial soap, (b) manual massage of the swollen
parts toward the groin, (c) wearing a suspensory to support and compress the scrotum and penis.
Surgical
Postinflammatory: (a) removal of excess of skin and subcutaneous tissue after intraoperative elimination of the edema fluid from the penis, scrotum or labia toward the
groin (Fig. 37.3)
inguinal nodes, (c) in advanced cases, excision of the swollen skin and subcutis of
the hypogastrium.
Chyloperitoneum: (a) laparotomy followed by (b) ligation and severing of retroperitoneal lymphatic bundles along the iliac veins (Fig. 37.4), (c) in advanced and
recurrent cases, excision of half the length of the small bowel that produces stagnant
lymph leaking into the peritoneal cavity, (d) postoperative injection of bleomycin or
3% aetoxisclerol into scrotal or labial blisters. In advanced edema of the genitals,
plastic surgery is performed in the postinflammatory group.
Chyluria with genital edema: (a) retroperitoneal laparoscopic or open denudation of the kidney(s) and ligation of the dilated lymphatics running toward kidney
pelvis (Fig. 37.4).
Technical hints: (a) scrotum: create a small anterior flap, leave as little skin as possible just to cover the denuded testes, remove afferent lymphatics with swollen tissues
in the hypogastrium, use diathermy and avoid ligatures (foci of future abscesses),
1-8
, (b) excision of fibrotic afferent lymphatics running toward the
Fig. 37.3 Postoperative view
of a reduced scrotum and
exposure of the hidden penis.
Small scrotal anterior flap.
Circumcision of the penis.
Elongation of the penis will
require further plastic surgery

37 Genital Lymphedema
Fig. 37.4 Lymphoscintigram
depicting iliac lymphatic
bundles running along large
pelvic veins. Lines crossing
the bundles indicate where
ligation and transection
should take place. Arrows
show sites of retroperitoneal
lymphatics draining the
kidneys and extravasated
isotope in cases of chyluria
311
retain the urinary catheter until the wound has healed up, (b) penis: perform circumcision followed by longitudinal incision on the dorsum or lateral aspects and remove
excess tissue, leave some more skin to allow future erection. Secondary plastic surgery of the penis is usually necessary. The application of a split-thickness skin graft
involving the penile shaft promotes adequate skin coverage, with a penile girth compatible with sexual intercourse and little alteration in sensitivity. The zigzag suture of
the graft on the ventral surface of the penis is aimed at avoiding scar contracture and
subsequent ventral curvature of the penis. (c) Labia: remove as much tissue as possible, recurrence of edema is frequent.
Postoperative therapy: (a) antibiotics: long-term penicillin (bicillin) 1,200,000
IU, intramurally every seventh day for 1–2 months, followed by one injection every
3 weeks for 1 year or alternatively amoxicillin + clavulanic acid at a dosage of 1 g
daily for 3 months and then 2 g for 3 days every 3 weeks, (b) manual massage of the
remnant swollen tissues.
Postoperative complications: (a) slow recurrence of edema, (b) recurrence of
local inflammatory episodes, (c) recurrence of lymph oozing, requiring injection of
sclerosants, and (d) hidden penis requiring elongation.
References
1. Dandapat MC, Mohapatro SK, Patro SK. Elephantiasis of the penis and scrotum. A review of
350 cases. Am J Surg. 1985;149:686-690.
2. McDougal WS. Lymphedema of the external genitalia. J Urol. 2003;170:711-716.

312 W.L. Olszewski
3. Modolin M, Mitre AI, da Silva JC, et al. Surgical treatment of lymphedema of the penis and
scrotum. Clinics (Sao Paulo). 2006;61:289-294.
4. Halperin TJ, Slavin SA, Olumi AF, Borud LJ. Surgical management of scrotal lymphedema
using local flaps. Ann Plast Surg. 2007;59:67-72.
5. Milanović R, Stanec S, Stanec Z, Zic R, Rudman F, Kopljar M. Lymphedema of the penis and
scrotum: surgical treatment and reconstruction. Acta Med Croat. 2007;61:211-213.
6. Garaffa G, Christopher N, Ralph DJ. The management of genital lymphedema. BJU Int.
2008;102:480-484.
7. Zacharakis E, Dudderidge T, Zacharakis E, Ioannidis E. Surgical repair of idiopathic scrotal
elephantiasis. South Med J. 2008;101:208-210.
8. Zugor V, Horch RE, Labanaris AP, Schreiber M, Schott GE. Penoscrotal elephantiasis: diag-
nostics and treatment options. Urologe A. 2008;47:472-476.

Chapter 38
Psychological Aspect: Compliance
and Quality of Life
Cheryl L. Morgan
An estimated 99 million Americans live with chronic illness. The majority have not
received effective treatment or optimal disease control.1 Health services research
indicates that it is the design of the care system, not the specialty of the physician or
allied health professional, that is the primary determinant of the quality of chronic
care.2 Collaborative relationships with health care providers can help patients and
families acquire effective medical, preventive, and health maintenance interventions.3 Moreover, collaborative care between health care providers can help improve
outcomes and adherence to self-care programs. Patient adjustment to requirements
to maintain their health is improved when reinforced by each professional involved
in his or her plan of care. They even develop adaptations to improve their own abilities to maintain the results achieved by treatment when accountability is emphasized by all members of their health care team.
The term compliance has mostly been superseded by the term adherence, a similar concept, but one that has fewer negative connotations regarding the physician
and patient relationship. Use of the term compliance has been strongly criticized
because it was thought to convey a negative image of the relationship between
patient and physician, in which the role of the physician was to issue the instructions
and the patient’s role was to follow the doctor’s orders. Noncompliance, therefore,
could be interpreted as patient incompetence with being unable to follow instructions or as deliberate, self-sabotaging behavior.
The term adherence was introduced in an attempt to recognize a patient’s right to
choose, to participate actively in his or her plan of care, and to remove the concept
of blame. It recognizes the need for patients, physicians, and allied health care professionals to work together to reach agreement and improve outcomes.6 How we
deal with this presents a major challenge for medicine, particularly in the management of chronic illnesses, such as lymphedema.
4
5
C.L. Morgan
Department of Rehabilitation Medicine,
Therapy Concepts Inc., Leawood, KS, USA
B.-B. Lee et al. (eds.), Lymphedema,
DOI 10.1007/978-0-85729-567-5_38, © Springer-Verlag London Limited 2011
313

314 C.L. Morgan
The issue of one’s quality of life for those living with chronic illness is important
to consider when evaluating a patient’s ability to manage lymphedema during treatment and thereafter. When possible, a comprehensive assessment would include not
only a physical evaluation, but also would consider the psychological, emotional,
and social concerns the patient may encounter.
Lymphedema patients are living an interrupted life replete with stages of emotional experiences and adaptive behavioral responses that correspond or collide with
the individual’s unique coping mechanisms. These emotional stages can range from
feeling a violation of physical intactness to loss of autonomy. In some cases, lymphedema patients are dually threatened with death if they are suffering with cancer
or other comorbidities complicating their recovery. Loss of activity, social isolation,
and fear of stigmatization can threaten social identity and self worth.
7
Adaptive behavioral responses can include resignation, disengagement or a
refusal to help themselves. Some patients present with denial, avoidance, repressed
anger or depression. These are largely unconscious responses that stop imagination
or emotions from threatening a patient’s sense of self. All represent defense mechanisms that can impair results of treatment if not addressed or are dismissed as
noncompliance.
8
Conscious coping mechanisms act as reality adjustors for the real world and are
generally considered a more constructive overt behavior.9 Stress research models
have identified three fundamental types of conscious coping. Information focused
coping is demonstrated by patients who seek instruments of emotional help.
Emotional focused coping is observed in patients who vent emotions, denial, escape
avoidance or acceptance of responsibility. Patients who utilize problem-oriented
coping mechanisms will exercise active coping techniques, plan, and identify prac-
tical approaches to address stress.
10
Consistent knowledge base and training of physicians and allied health professionals, paired with standardized information or resources, provide patients with
confidence in the treatment approach and its demands. This is particularly important
with protocols as demanding of time and resources as lymphedema management.
Reliable direction helps prevents patients from pursuing unproven and often costly
treatments. Obtaining incorrect or contradictory information promotes frustration
and distrust and can lead to patients rejecting medical attention or non-adherence.
11
Communication of expectations of therapeutic intervention involving family or
caregivers in the selection of means of accountability fosters adherence with even
strict protocols. Establishing reasonable outcomes and providing simple methods of
measuring progress encourage patients and improve consistency with self care.12
Instructions should be practical and individualized. Successful treatment is probable
when a continuum of support and training is available to patients and active and
sustained follow-up is available.
13
Access to resources, treatment, literature, and supplies continues to encumber
patients with lymphedema. Physician-directed care that is provided by experienced
therapists, will enhance a patient’s experience and provide superior results.
2
Support
systems that provide additional resources are an important part of a successful program.4 Without standardized educational requirements for health professionals or

38 Psychological Aspect: Compliance and Quality of Life
315
treatment protocols, lymphedema patients are often left on their own to learn about
their condition and find treatment they can afford to pursue.
Improved communication between physicians and allied health professionals is
increasing as awareness of the options for these patients increases. Once consensus
on diagnosis, staging, and treatment of lymphedema can be reached, measureable
outcomes can support the standardization of professional medical education, and
improve reimbursement for treatment and supplies.
14
Effective medical management of lymphedema is more likely to occur when collaborative care is consistently provided, and takes into consideration the patient’s individual coping skills and variables that affect their quality of life and access to optimal
treatment. Respecting each patient’s autonomy, drawing out any concerns or ambivalence about pursuing treatment, and allowing the patient to develop and/or own the
treatment plan greatly improve the odds of achieving positive clinical outcomes.15 The
growing population of chronically ill patients, such as patients with lymphedema, will
require the development of successful systems and collaborative care.
16
References
1. Rothman A, Wagner E, et al. Chronic illness management: What is the role of primary care?
Ann Intern Med. 2003;138(3):256-261.
2. Casalino LP. Disease management and organization of physician practice. JAMA. 2005;
293(4):485-488.
3. Von Korff M, Gruman J, et al. Collaborative management of chronic illness. Ann Intern Med.
1997;127(12):1097-1102.
4. Cretin S, Shortell SM, Keeler EB. An evaluation of collaborative interventions to improve
chronic illness care: framework and study design. Eval Rev. 2004;28(1):28-51.
5. Haynes RB, Sackett DL, Taylor DW. Compliance in Healthcare. Baltimore: Johns’ Hopkins
University Press; 1979.
6. World Health Organization. Adherence to Long-Term Therapies: Evidence in Action. Geneva:
World Health Organization; 2003.
7. Redman BK. The ethics of self management for chronic illness. Nurs Ethics. 2005;12(4):
360-369.
8. Cramer P. The Development of Defence Mechanisms: Theory, Research, and Assessment. New
York: Springer; 1991.
9. Faller H, Bülzebruck H, Drings P, Lang H. Coping, distress, and survival among patients with
lung cancer. Arch Gen Psychiatry. 1999;56(8):756-762.
10. Lazarus RS, Folkman S. Stress, Appraisal, and Coping. New York: Springer; 1984.
11. Stille CJ, Jerant A, et al. Coordinating care across diseases, settings and clinicians. Ann Intern
Med. 2005;142(8):700-708.
12. Zatzick D, Roy-Byrne P, et al. A randomized effectiveness trial of stepped collaborative care
for acutely injured trauma survivors. Arch Gen Psychiatry. 2004;61(5):498-506.
13. Russell E, Wagner EH, Schaefer J, et al. Development and validation of patient assessment of
chronic illness care (PACIC). Med Care. 2005;43(5):436-444.
14. Tretbar LL, Lee BB, Morgan CL, et al. Lymphedema; Diagnosis and Treatment. New York:
Springer; 2007.
15. Butterworth S. Influencing patient adherence to treatment guidelines. J Manag Care Pharm.
2008;14(suppl S-b):S21-S25.
16. Wagner EH. Chronic disease care. BMJ. 2004;328(7433):177-178.


Part IX
Surgical Treatment:
Reconstructive Surgery

Соседние файлы в папке Библиотека им академика М.И. Перельмана
