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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_113_библиотеки_им_акад_М_И_Перельмана
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Index220
https://t.me/med1917
fibroblast growth factor (FGF) 20
fibroblasts
Formitopsis niagra
fractured tooth fragment
full pulpotomy
22
32
177
109
g
geneencoding plasmids 14
gingival crevicular fluid (GCF)
gingivectomy
GRADE
growth/differentiation factor 15 (GDF15)
growth factors (GF) 22
120
53–54
52
26
h
haemostasis 91–92
hard tissue formation 93
hardtissue healing
HCSC see hydraulic calcium silicate
cement (HCSC)
healing capability of, dental pulp
immuneinflammatory response, in dentine
pulp complex repair 27–28
immunological response, in dental
pulp
molecular and cellular regulatory processes
bioactive molecules
cellular responses 27
healthrelated quality of life (HRQOL) 201
Hertwig’s epithelial root sheath (HERS) 5, 193
heterotopic mineralization
homeobox genes 12
human dental pulp 100
hybridization process 11
hydraulic calcium silicate cement (HCSC) 93,
112, 113, 124, 154, 185
biocompatible and noncytotoxic material 94
formulations
light cured 117
powder and liquid capsules 116–117
powder and liquid hand mixed 116
premixed 117
hydroxyapatitelike crystal formation 94
hyperplastic tissue 68
hypoxia 7
hypoxiainduced angiogenesis 8
hypoxiainduced dentinogenesis 8
33
28–32
26–27
68
hypoxiainducible transcription factor 1alpha
(HIF1alpha) 7
i
Ibuprofen 211
immune cells
immunomodulatory functions
immunomodulatory therapies
immunosensory leukocytes
immunosurveillance leukocytes
induced regulatory T (iTreg) cells
inflamed dental pulp
inflamed pulp tissues 121
inflammatory cytokines 12
inflammatory infiltrates 66
inflammatory mediators
inflammatory molecules 29
interleukin 6 (IL6) 12
interleukin 8 (IL8) 12
interleukin 1 beta (IL1beta)
International Association of Dental
International Caries Consensus
intraoral periapical radiographs
intrapulpal cartilagelike tissue 68
intrapulpal hard tissue formations 89
intrapulpal mineralization 68
irreversible pulpitis
23
114
33
Traumatology (IADT)
Collaboration
61, 75
48, 87, 99
23
32
28
29
30
7, 12
112, 155
51
l
laser Doppler 51
lipoproteins 136
lipoteichoic acid (LTA) 11, 136
local anaesthesia
LTA see lipoteichoic acid (LTA)
lymphatic circulation
lymphatic drainage
lymphatic vessels 7
177
8
8
m
macrophages 28
magnification systems 114
Maillard reaction 65
matrix extracellular phosphoglycoprotein
(MEPE) 2, 23
matrix metalloproteinases (MMP) 22, 52
t.me/Dr_Mouayyad_AlbtousH
Index 221
https://t.me/med1917
McGill questionnaire (MPQ) 203
microbial contamination
microbial ecosystem
microorganisms
mineral trioxide aggregate (MTA)
166, 188
mitogen activated protein (MAP) kinase
molecular immune processes
myeloidderived suppressor cells (MDSC)
62
113
65, 68
4, 31, 110,
28
29
30
n
Nacetylcysteine (NAC) 32
nanofibrous scaffolds 144
natural killer (NK) cells
natural scaffolds 34
Nestin 2
neurogenic inflammation process 9, 10
neurokinin A 24
neutrophil extracellular traps (NET)
neutrophils 29
noncavitated enameldentine lesion 64–65
nonhealing tissue 161
nonodontogenic pain
nonphysiological stimulus 50
nonspontaneous pain 50
nonsteroidal antiinflammatory drugs
(NSAID)
nuclear factorkappa B pathway (NFKb) 28, 137
nucleotideoligomerization binding domain
(NOD) receptors
numeric rating scale (NRS) 203
211
30, 136
30
50
22
o
occlusal cavitated carious lesion 65–66
odontoblastderived chemokines 30
odontoblastic lineage 143
odontoblastlike cells
odontoblast pseudostratified layer 2
odontoblasts 21–22, 147
older pulp tissue
oral health impact profile (OHIP) 208
oral healthrelated quality of life
(OHRQoL) 172, 208
oral pathogens 23
orthodontic therapy 191
osteodentine phenotype 12
osteopontin (OPN) 23
22
6
p
paediatric dentistry 92
pain intensity
PAMP lipopolysaccharide (LPS)
partial or total radiographic obliteration
partial pulpotomy
pathogenassociated molecular patterns
patientreported outcome (PRO)
patternrecognition receptors (PRR)
peptidoglycan
periapical index (PAI) criteria
periapical radiolucency 74
periodontal ligament stem cells (PDLSC) 27
piezo channels 22
placenta GF (PlGF) 26
plasma rich in growth factors (PRGF)
plateletderived GF (PDGF) 26
plateletrich fibrin (PRF) 33, 196, 211
plateletrich plasma (PRP) 33, 196
polyglycolic acid (PGA) scaffolds
polylactic acid (PLA) 35
polylactic acid (PLLA) scaffolds 35
pragmatic subdivision, extensive carious lesions
caries profunda 1
caries profunda 2 62–63
caries profunda 3 63
proinflammatory cytokines 22, 23
proinflammatory mediators 31
ProRoot MTA
pulpal diagnosis
clinical to, molecular diagnostic tests
diagnoses and terminologies 48
diagnostic process
accuracy of, sensibility tests 50–51
pain history 49–50
pulpitis classification 49
pulp sensibility/sensibility testing 50
radiographic examination 51
thermal and electric tests 51
preoperative diagnosis vs. direct
pulpal disease, diagnostics for 32–33
pulpal homeostasis 23
pulpal inflammation 12
204
(PCO)
167
109
(PAMP)
observation 51–52
22, 136
136
62
94, 116
137
201
22, 136
205
196
35
52–55
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pulpal inflammatory response 30
pulp bleeding
pulp canal calcification
pulp capping
pulp capping material
pulpectomy
pulp fibroblasts
pulpinduced regeneration
pulp inflammation
pulpitis
pulp necrosis
pulp nerve tissue 204
pulp revitalization (PR) 202
as regenerative endodontic therapy
OHIP14 questionnaire
outcomes and measures
pulp sensibility 50
pulp tissue 7, 10
pulp vitality
dental pulp development
apical papilla, role of
and cell signalling 2–5
tooth development 1, 2
and inflammation 12
internal calcifications
pulp innervation 9–11
pulp repair mechanisms 12–14
pulp response, to restorative processes 11
pulp vascularization and clinical
regeneration potential of, dental pulp 6
stem cell reserve 6
96
4, 14
112
22
47, 202
185
implications
167–170
92–94
13
59
212
209–212
5
6
6–9
r
RCT see root canal treatments (RCT)
reactionary dentine 25
formation 26
secretion
reactive oxygen species (ROS) 30
refrigerant spray 50
regenerative endodontics
biological goals, of pulp
ETE
23
regeneration
autologous platelet products 196
classification 195
plateletbased techniques 196
186–187
historical development
mechanical goals, of pulp regeneration
186–187
revitalization
biological result
clinical outcome
clinical protocol
contraindications
followup
indications
of mature teeth
outcome measures 191–192
regenerative endodontic therapies (RET) 201
regenerative procedures
regulatory immune cells 30
repairmatrix proteins mechanisms 143–144
resinbased composite (RBC) restoration 120
resinreinforced glass ionomer cement (RMGI) 98
restorative material
revascularization procedures 5
reversible pulpitis 100
revitalization procedures 5
root canal treatments (RCT)
root carious lesions
191
189
133, 201
187–188
193–194
192–193
189–191
189
193
4
114
59, 68, 109, 112,
67
s
selective caries excavation 90, 91
sensibility testing 50
sensory nerves
sentinel immune cells 23
simple descriptor scale (SDS) 203
small integrinbinding ligand Nlinked
sodium hypochlorite (NaOCl) 27, 92, 113, 190
haemostasis 162
softtissue healing 33
sonic hedgehog (SHH) pathways 4, 20
stem cells from human exfoliated deciduous
stem cells of the apical papilla (SCAP) 27
stepwise excavation 76–77
Strindberg criteria 205
subodontoblast cells 25
substance P (SP) 10, 24
symptomatic irreversible pulpitis, treatment
21, 24
glycoproteins (SIBLINGS) 23
teeth (SHED) 27
110–111
t.me/Dr_Mouayyad_AlbtousH
Index 223
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t
TDI see traumatic dental injuries (TDI)
tertiary dentine
tertiary dentinogenesis process
1,1,1,2tetrafluoroethane
Theracal
tolllike receptors (TLR)
tooth discolouration
tooth mobility
tooth morphogenesis
tooth sensitivity
transforming GF (TGF)
transient receptor potential (TRP) channels
traumatically exposed pulp treatment 111–112
traumatic dental injuries (TDI) 137, 154
CCF with, luxation and complex
injuries
clinical protocols
direct pulp capping
followup protocol 177–178
pulpotomy 177
complications, from VPT
discolouration
pulp canal calcification 167–170
resorption 171
factors affecting VPT outcomes
achieving haemostasis
material choice, for CCF 165–166
mobility 160–161
partial pulpotomy 167
pulp exposure size
restorations 160
root development, stage of 164–165
time or patient limited options 166
treatment delay 161–163
pulp condition, diagnosing 155–156
traumatised teeth, management 156
trauma vs. caries management 172–174
treatment choice 156–159
treatment modalities, for vital pulp
treatment (VPT)
achieving haemostasis
final restoration placement 126
pulp capping material, placement
of 124–125
caries pathology 64–68
6, 21, 25
25–27
50
117, 118
22, 136
170
160
21
211
26
22
174–177
177
170, 172
166
163–164
clinical protocol
armamentarium 118
assessment and anaesthesia
caries removal and pulp exposure
preVPT preparatory procedures
pulp tissue removal (pulpotomy)
clinicianreported outcomes
contraindications
direct pulp capping
epidemiology
evidencebased recommendations
exposing pulp 60
exposure 84–85
future of 77–78
indications 109–112
monitoring 74–75
pragmatic subdivision, extensive carious
lesions
procedure 113–118
pulpotomy outcomes 126–127
traumatic and iatrogenic pulp exposures
trigeminal ganglion level 10
tubular dentine 2
phenotype 12
tubular structure
tumour necrosis factoralpha (TNFalpha) 7, 28
tunnel defects 26, 31
112–113
86–87
76–77
60–63
26
118–120
121
120–121
121–123
72–74
75–76
85–86
u
untreated carious lesions 66
v
vasculature 24
vasoactive intestinal polypeptide (VIP) 24
vasoconstrictors 191
verbal descriptor scale (VDS)
verbal pain scale (VPS) 203
verbal rating scales (VRS) 203
VIP see vasoactive intestinal polypeptide (VIP)
visual analogue scale (VAS) 203
vital pulp treatment (VPT) 1, 47, 84, 201
dental pulp 20–25
future 33–35
healing capability 25–28
history 139–141
immunological response, in dental pulp 28–32
203
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vital pulp treatment (VPT) (cont’d)
material selection
biology of, tissue response
gold standard and limitations
native pulp conditions
opportunities and nextgeneration
VPT
144–148
pulp response to, traumatic/carious
injuries
136–138
outcomes
composite outcomes
continued root development
206
CRO
definitive restoration 207
hard tissue barrier, formation of 208
histopathological outcomes 207
143–144
136
208–209
141–143
205
202–204
pain
PRO
207
radiographic assessment, timing of
sensibility testing, timing
sensibility/vitality, of pulp
stakeholder considerations
success
202
207
survival
timing, pain measurement
pulpal disease diagnostics
VPT see vital pulp treatment (VPT)
205
204–205
209
204
32–33
w
Well root PT 117
white spot lesion 64
winglessrelated integration site (Wnt)
205
20
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https://t.me/med1917
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