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178
CHAP TER6 Theear
Common presentations
Abnormal sounds (tinnitus)
Dizziness
Fullness/ discharge
Injuries
Itching
Loss of hearing/ deafness
Pain (otalgia)
Swellings.
Common problems and theircauses
Abnormal sounds (tinnitus)
Common
Wax impaction
Glueear
Infections (otitismedia)
Perfor ated eardrum
Noise induced
Presbycusis
TMJ disorders
Ototoxicdrugs
Many patients have underlying depression and anxiety.
Uncommon
Insects
Otosclerosis
Ménière’s disease
Trau m a
Acoustic neuroma
AVMs and venoushums
Glomus jugulare and carotid body tumours
Patulous Eustachian tube and palatal myoclonus.
Dizziness
Common
Otological causes:
Benign paroxysmal positional vertigo
Ménière’s disease
Iatrogenic (middle ear/ mastoid surgery)
Impac tedwax
Ear infection (otitismedia)
Systemic causes:
Migraine
MS
Medications.
COMMON PROBLEMS AND THEIRCAUSES
Uncommon
Otological causes:
Cerebellopontine angle tumours
Perilymphatic stulas
Trauma (tempor al bone fracture)
Vestibular neuronitis/ labyrinthitis
Oto syphilis
Systemic causes:
Metabolic disorders (hypo/ hyperthyroidism, diabetes)
Vascular cause (vertebrobasilar insuciency, Eagle syndrome)
Neurological disorders (stroke, seizures, Parkinsonism).
Fullness/ discharge
Common
Chronic suppurative otitis media (mucoid)
Furunculosis or local abscess (purulent)
Trauma (bloody/ CSF)
Eczema (watery)
Acute otitis media (bloody)
Eustachian tube dysfunction
Wax.
Uncommon
Nasopharyngeal malignancy (bloody)
Cholesteatoma (foul smelling)
Ménière's disease.
Injuries
Common
Blunt/ bruising
Lacerations.
Uncommon
Bites (animal/ human)
Avulsions/ tears
Barotrauma.
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Itching
Common
Otitis externa
Contact dermatitis.
Uncommon
Cholesteatoma.
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CHAP TER6 Theear
Loss ofhearing/ deafness
Common
Acute otitismedia
Acoustic trauma/ trauma
Eustachian tube dysfunction
Wax impaction
Presbycusis
Idiopathic.
Uncommon
Autoimmune, Ménière’s,MS
Infections (herpes, mumps, cytomegalovirus, toxoplasmosis,
syphilis)
Foreignbody
Otosclerosis
Drugs
Metabolic (diabetes/ thyroid)
Tumour.
Pain (otalgia)
Common
Acute otitis externa
Otitis media with eusion
Referredpain
Furuncles (infected hair follicles)
Perichondritis.
Uncommon
Malignant otitis externa
Barotrauma
Herpeszoster
Tumours
Mastoiditis.
Referredpain
Tonsillitis and upper respiratory tract disease
Eustachian tube dysfunction
Dental pathology
Disorders of theTMJ
Parotid disease
The oropharynx
The lar ynx and pharyn x
Cervical spondylosis
Malignancy of any of the above- listed (par ticularly the tonguebase).
USEF UL QU ESTI ONS AND WHAT TO LOOKFOR
Swellings
Common
Pinna haematoma (trauma)
Perichondritis of thepinna.
Uncommon
Seroma/ pseudocyst of thepinna
Erysipelas
Gout
Sebaceouscyst
Herpes zoster.
Useful questions and what tolookfor
Abnormal sounds (tinnitus)
Askabout
Character of tinnitus:unilateral or bilateral, high- pitched or low-
pitched (roaring, buzzing), pulsatile, clicking
Progression and frequency
Severity:eect on sleep, dailylife
Histor y oftr auma
Medications
Underlying anxiety and depression.
Lookfor
Look for retrotympanicmasses
Audiblebruits
TMJ dysfunction
Similar work- up as evaluating hearingloss.
181
Dizziness
Askabout
Describe dizziness:rotatory vertigo (‘spinning’, ‘whirling’, or
‘turning’ of patient or surroundings), disequilibrium (‘o- balance’), lightheadedness (sense of impending faint), physiological dizziness (motion sickness)
Duration:seconds to minutes (benign paroxysmal positional
vertigo (BPPV), arrhythmia), hours (Ménière’s, migraine), days (vestibular neuritis, labyrinthitis), or constant (central)
Associated symptoms:hearing loss, aural fullness, tinnitus,
sympathetic response (nausea, vomiting), central symptoms (numbness, weakness, diplopia, blurred vision)
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CHAP TER6 Theear
Contributing factors:medications (antihypertensives, ototoxic
medications, sedatives); medical history (hypertension, cardiac arrhythmias, diabetes, vascular disease, otological disease, neurological disease, migraines).
Lookfor
General exam:(pulse, BP:standing and lying, carotid bruit s), with
focus on neurological, cardiovascular, and peripheral vascular disorders
Otoscopy:otitis media, glue ear, cholestatoma
Neurological exam:for cranial nerve palsies, vestibulospinal
reexes (e.g. Romberg’s test, gait, past pointing test), later alizing signs (e.g. weakness, paraesthesia)
Eye movements:check pursuit and for nystagmus
Specic tests:Dix– Hallpike, head- shake, stula test, caloric testing,
tuning fork, and audiometry.
Fullness/ discharge
Askabout
Tinnitus
Hearing disturbance
Autophony (abnor mal hearing of one's own voice and respiratory
sounds)
Nasal obstruction
Otalgia
Otorrhoea
Rhinorrhoea
Sore throat.
Lookfor
Otoscopy for signs of infection, wax, discharge,etc.
Examine the throat/ nasopharynx for disease
Nasal endoscopy
Ask patient to perform the Valsalva manoeuvre.
Injuries
Askabout
When it occurred?
Mechanism ofinjury
Loss of consciousness/ neck,etc.
Hearingloss
Dizziness.
Lookfor
Other injuries
Facial nerve weakness
Hearing loss (tuning forks if stable)
Nystagmus
USEF UL QU ESTI ONS AND WHAT TO LOOKFOR
Other neurological decits and cranial nerve palsies
CSFleak
Haemotympanum
External auditory canal lacerations
Battle’s sisgn (ecchymosis over the mastoid process).
Itching
Askabout
Dischar ge from theear
Tenderness orpain
Cough
Fever andchills
Headache
Runnynose
Sneezing
Sore throat.
Lookfor
Crusting or akingskin
Redness, warmth, or swelling.
Loss ofhearing/ deafness
Askabout
Onset and duration, constant vs intermittent, progression,
unilateral or bilater al, high or low tone loss, decreased speech intelligibility
Contributing factors:recent infection; loud noise exposure
Recent tr auma:barotrauma, head injury; exacerbating factors for
tinnitus (sleep, exercise, caeine, alcohol)
Previous otological surgery, infections
Medications
Systemic:history of autoimmune disease, hyper tension, diabetes,
vascular disorders, neurological disease (stroke), depression
Family history of deafness
Associated symptoms:aural fullness, fever s, ver tigo, tinnitus,
otalgia, otorrhoea, other neurological complaints.
Lookfor
Malformations, auricular pits, scars, oedema, mastoid tenderness,
tragal tenderness
Otoscopy (wax, lesions, masses) and tympanic membrane (colour,
thick ness, presence of uid, perforations)
Pneumatic otoscopy:test mobilit y of tympanic membrane with
positive and negative pressure
Fistula test:positive pressure causes nyst agmus which rever ses
with negative pressure (perilymph stula and labyrinthitis)
Neurological and vestibular exam,bruits
Tuning forktests.
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184
CHAP TER6 Theear
Pain (otalgia)
Askabout
Duration of symptoms
Discharge
RecentURTI
Hearingloss
Degree of pain and radiation (malignant otitis externa pain is often
severe, post- auricular pain suggests mastoiditis)
Previous episodes and treatment todate
Previous ear surger y
PMH (diabetes or other immune compromise)
Injury to the mandible (fractured condyle).
Lookfor
Swelling
Discharge
Furuncles
Signs ofinjur y
CN VII weakness
Systemicupset
Tenderness over the mastoid prominence
Otoscopy (bulging of the external auditory canal/ tympanic
membrane may appear red, per forated, and discharging).
Swellings
Askabout
Any known aetiological factors
Histor y oftr auma
Any preceding otalgia (perichondritis can arise following otitis
externa and trauma)
Predisposing factors (e.g. immune compromise)
Any painful (pseudocyst can present as a painless swelling).
Lookfor
Conrm presence
Discharge
Otoscopy:inspection of ear drum and middle ear (otitis media,
glue ear, cholesteatoma).
EX AMIN ATION OF THEEAR
Examination oftheear
Applied anatomy
Externalear
This comprises of pinna, external auditory meatus (EAM), external audi­tory canal, and tympanic membrane (ear dr um). The skin on the outer part of the E AM is self- cleansing and contains hair follicles and wax­producing ceruminous glands. These are absent on the inner part. Ears are normally self- cleansing and use of cotton buds should be discouraged. If wax is dislodged into the deeper part of the canal it cannot be removed by this natural process.
Middleear
This is an air- cont aining space allowing sound transfer to the cochlea. It contains three ossicles (malleus, incus, and stapes), two muscles (ten­sor tympani and st apedius), and part of the facial nerve (chorda tympani branch). Equalization of pressures occurs via the Eustachian tube. The mastoid air cells communicate with the middle ear space. This is closely related to the brain (middle cranial fossa), jugular bulb (posteriorly), and labyrinth (medially). Infections in middle ear are potentially very serious as they can extend into surrounding structures.
Innerear
This comprises the cochlea, vestibule, and semicircular canals. The ‘membranous’ par t is lled by endolymph and is surrounded by the bony labyr inth, which is lled with perilymph. The cochlea contains the organ of hear ing (organ of Corti). The vestibule and semicircular canals are involved in balance.
Nerve supply totheear
The ear is innervated by the V, IX, and X cranial nerves, and by the pos­terior roots of C2 and C3. Because of this, pathology at sites of similar
nerve distribution (TMJ, dental, oropharyngeal, laryngeal, and hypopharyn­geal) can present with otalgia (referredpain).
Examination oftheear
Inspect the external ear r st. Remove any discharge or wax. Look for obvious signs of abnormality:
Size and shape of thepinna
Extra cartilage tags/ pre- auricular sinuses orpits
Signs oftrauma
Skin lesions, e.g. neoplasia
Skin condition of the pinna and externalcanal
Infection/ inammation of the external ear canal, with discharge.
Then palpate for mastoid tenderness, tragal tenderness, and lymphade­nopathy. Use an otoscope (auroscope) to inspect the exter nal auditory canal and tympanic membrane. Gr asp the pinna and gently pull it up and backwards. This helps str aighten the canal for inspection (in infants, pull the pinna posteriorly). Note the condition of the canal skin and the
185
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CHAP TER6 Theear
presence of wax, foreign tissue, or discharge. Inspect the tympanic mem­brane for signs of injury, perforation, or discharge. The mobility of the eardrum can be evaluated using a pneumatic speculum, which attaches to the otoscope. Check facial nerve function.
Special manoeuvres include the following:
Dix– Hallpike manoeuvre
This is a positional test for BPPV. The patient sits upright with the legs extended. Their head is rotated by approximately 45 degrees. The clini­cian then helps the patient to quickly lie down backwards with the head held in approximately 20 degrees of extension. The patient’s eyes are obser ved for nystagmus. If rot ational nystagmus occurs then the test is considered positive for benign positional vertigo.
Head- shake nystagmus
Move the head in the horizont al plane for 20– 30 seconds, then suddenly stop and evaluate for nystagmus.
Fistula test and pneumatic otoscopy
Positive pressure causes nystagmus which reverses with negative pres­sure (perilymph stula and labyrinthitis). Test mobility of tympanic mem­brane with pressure changes.
Tuning forktests
Rinnetest
Use a 512 Hz tuning fork to compare air conduction (AC) and bone con­duction (BC). Strike the tuning fork then place it within 1cm of the EAM (AC) and then immediately place on the mastoid(BC).
Normal hearing
AC should be greater than BC and so the patient should be able to hear the tuning fork next to the pinna after they can no longer hear it when held against the mastoid.
Abnormal hearing
If they are not able to hear the tuning fork after the mastoid, it means
that BC is greater than AC. Something is inhibiting the passage of
sound from the ear canal, through the middle ear apparatus and into
the cochlea (i.e. there is a conductive hearingloss).
In sensorineural hearing loss both BC and AC are equally diminished.
Patients with sensorineur al hearing loss can usually hear better on
the mastoid process than air, but indicate the sound has stopped
much earlier than conductive loss patients.
Webertest
Strike the tuning fork and place it in the centre of the forehead. The per­ceived sound should normally be heard centrally.
Remember that nasophar yngeal pathology can present with unilateral ear symptoms. Always look at the throat and nasopharynx, especially in adults presenting with unilateral glue ear — this can be a presenting symptom of a nasopharyngeal tumour.
INVESTIGATIONS
Investigations
Laborator ytests
FBC may show a raised WCC (neutrophils).
Electrolytes:ototoxicity
Glucose:screen for diabetes (associated with hearingloss)
Coagulation and immunological prole:coagulopathies can be
associated with hearing disorders
ESR
Treponemal studies:Lyme titres/ TPHA/ VDRL/ FTA- ABS depending
on where youwor k
Lipid prole:atherosclerotic disease can be associated with sudden
senorineural hearingloss
Fluid analysis (beta- 2 tr ansferr in in suspected CSF leakage)
Microbiology for any discharge.
Plainlms
Mastoid X- rays:will show opacity of the air cells. These and other special views of the temporal bone (Schuller’s, Stenvere’s, Towne’s views) have now largely been replaced by CT andMRI.
CT/ MRI oftemporalbones
These look for potential complications of suppurative ear disease, cho ­lesteatoma, mastoiditis, temporal bone fracture, congenital disorder or neoplasm (especially acoustic neuroma).
MRI is often required for suspected cerebellopontine angle tumours,
acoustic neuromas, meningiomas, and petrous apex lesions.
Audiometrictests
Audiometr y tests are carr ied out in soundproofed rooms using preci­sion equipment. Tests may be subjective (pure tone audiograms, speech audiometry), or objective (impedance audiometry, evoked response audiometry).
These tests are essential to dene auditory function and to quantify
the thresholds of AC/ BC hearing for both ears. It distinguishes conduc­tive loss from sensorineural hearing loss; cochlear ver sus neural dysfunc­tion, and malingering (pseudohypacusis).
Dierent audiologicaltests:
Pure tone audiometr y:tests threshold of AC andBC.
Tympanometry:check s middle ear pressure and impedance (indirect
measure of Eustachian tube function).
Auditory brainstem response (ABR):recording of the activity of the
eighth ner ve and central auditor y pathway response to an auditor y signal.
Otoacoustic emissions (OAEs):tests objective sounds in external
auditory canal emitted from outer hair cells (cochlearecho).
AB R and OAE are objective tests and do not need the patient’s
cooperation and hence used identify malingerers and for medico­legal purposes.
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