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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_4540_Библиотеки_им_академика_М_И_Перельмана.pdf

178
CHAP TER6 Theear
Common presentations
• Abnormal sounds (tinnitus)
• Dizziness
• Fullness/ discharge
• Injuries
• Itching
• Loss of hearing/ deafness
• Pain (otalgia)
• Swellings.
Common problems and theircauses
Abnormal sounds (tinnitus)
Common
• Wax impaction
• Glueear
• Infections (otitismedia)
• Perfor ated eardrum
• Noise induced
• Presbycusis
• TMJ disorders
• Ototoxicdrugs
• Many patients have underlying depression and anxiety.
Uncommon
• Insects
• Otosclerosis
• Ménière’s disease
• Trau m a
• Acoustic neuroma
• AVMs and venoushums
• Glomus jugulare and carotid body tumours
• Patulous Eustachian tube and palatal myoclonus.
Dizziness
Common
• Otological causes:
•
Benign paroxysmal positional vertigo
•
Ménière’s disease
•
Iatrogenic (middle ear/ mastoid surgery)
•
Impac tedwax
•
Ear infection (otitismedia)
• Systemic causes:
•
Migraine
•
MS
•
Medications.

COMMON PROBLEMS AND THEIRCAUSES
Uncommon
• Otological causes:
•
Cerebellopontine angle tumours
•
Perilymphatic stulas
•
Trauma (tempor al bone fracture)
•
Vestibular neuronitis/ labyrinthitis
•
Oto syphilis
• Systemic causes:
•
Metabolic disorders (hypo/ hyperthyroidism, diabetes)
•
Vascular cause (vertebrobasilar insuciency, Eagle syndrome)
• Neurological disorders (stroke, seizures, Parkinsonism).
Fullness/ discharge
Common
• Chronic suppurative otitis media (mucoid)
• Furunculosis or local abscess (purulent)
• Trauma (bloody/ CSF)
• Eczema (watery)
• Acute otitis media (bloody)
• Eustachian tube dysfunction
• Wax.
Uncommon
• Nasopharyngeal malignancy (bloody)
• Cholesteatoma (foul smelling)
• Ménière's disease.
Injuries
Common
• Blunt/ bruising
• Lacerations.
Uncommon
• Bites (animal/ human)
• Avulsions/ tears
• Barotrauma.
179
Itching
Common
• Otitis externa
• Contact dermatitis.
Uncommon
• Cholesteatoma.

180
CHAP TER6 Theear
Loss ofhearing/ deafness
Common
• Acute otitismedia
• Acoustic trauma/ trauma
• Eustachian tube dysfunction
• Wax impaction
• Presbycusis
• Idiopathic.
Uncommon
• Autoimmune, Ménière’s,MS
• Infections (herpes, mumps, cytomegalovirus, toxoplasmosis,
syphilis)
• Foreignbody
• Otosclerosis
• Drugs
• Metabolic (diabetes/ thyroid)
• Tumour.
Pain (otalgia)
Common
• Acute otitis externa
• Otitis media with eusion
• Referredpain
• Furuncles (infected hair follicles)
• Perichondritis.
Uncommon
• Malignant otitis externa
• Barotrauma
• Herpeszoster
• Tumours
• Mastoiditis.
Referredpain
• Tonsillitis and upper respiratory tract disease
• Eustachian tube dysfunction
• Dental pathology
• Disorders of theTMJ
• Parotid disease
• The oropharynx
• The lar ynx and pharyn x
• Cervical spondylosis
• Malignancy of any of the above- listed (par ticularly the tonguebase).

USEF UL QU ESTI ONS AND WHAT TO LOOKFOR
Swellings
Common
• Pinna haematoma (trauma)
• Perichondritis of thepinna.
Uncommon
• Seroma/ pseudocyst of thepinna
• Erysipelas
• Gout
• Sebaceouscyst
• Herpes zoster.
Useful questions and what
tolookfor
Abnormal sounds (tinnitus)
Askabout
• Character of tinnitus:unilateral or bilateral, high- pitched or low-
pitched (roaring, buzzing), pulsatile, clicking
• Progression and frequency
• Severity:eect on sleep, dailylife
• Histor y oftr auma
• Medications
• Underlying anxiety and depression.
Lookfor
• Look for retrotympanicmasses
• Audiblebruits
• TMJ dysfunction
• Similar work- up as evaluating hearingloss.
181
Dizziness
Askabout
• Describe dizziness:rotatory vertigo (‘spinning’, ‘whirling’, or
‘turning’ of patient or surroundings), disequilibrium (‘o- balance’),
lightheadedness (sense of impending faint), physiological dizziness
(motion sickness)
• Duration:seconds to minutes (benign paroxysmal positional
vertigo (BPPV), arrhythmia), hours (Ménière’s, migraine), days
(vestibular neuritis, labyrinthitis), or constant (central)
• Associated symptoms:hearing loss, aural fullness, tinnitus,
sympathetic response (nausea, vomiting), central symptoms
(numbness, weakness, diplopia, blurred vision)

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CHAP TER6 Theear
• Contributing factors:medications (antihypertensives, ototoxic
medications, sedatives); medical history (hypertension, cardiac
arrhythmias, diabetes, vascular disease, otological disease,
neurological disease, migraines).
Lookfor
• General exam:(pulse, BP:standing and lying, carotid bruit s), with
focus on neurological, cardiovascular, and peripheral vascular
disorders
• Otoscopy:otitis media, glue ear, cholestatoma
• Neurological exam:for cranial nerve palsies, vestibulospinal
reexes (e.g. Romberg’s test, gait, past pointing test), later alizing
signs (e.g. weakness, paraesthesia)
• Eye movements:check pursuit and for nystagmus
• Specic tests:Dix– Hallpike, head- shake, stula test, caloric testing,
tuning fork, and audiometry.
Fullness/ discharge
Askabout
• Tinnitus
• Hearing disturbance
• Autophony (abnor mal hearing of one's own voice and respiratory
sounds)
• Nasal obstruction
• Otalgia
• Otorrhoea
• Rhinorrhoea
• Sore throat.
Lookfor
• Otoscopy for signs of infection, wax, discharge,etc.
• Examine the throat/ nasopharynx for disease
• Nasal endoscopy
• Ask patient to perform the Valsalva manoeuvre.
Injuries
Askabout
• When it occurred?
• Mechanism ofinjury
• Loss of consciousness/ neck,etc.
• Hearingloss
• Dizziness.
Lookfor
• Other injuries
• Facial nerve weakness
• Hearing loss (tuning forks if stable)
• Nystagmus

USEF UL QU ESTI ONS AND WHAT TO LOOKFOR
• Other neurological decits and cranial nerve palsies
• CSFleak
• Haemotympanum
• External auditory canal lacerations
• Battle’s sisgn (ecchymosis over the mastoid process).
Itching
Askabout
• Dischar ge from theear
• Tenderness orpain
• Cough
• Fever andchills
• Headache
• Runnynose
• Sneezing
• Sore throat.
Lookfor
• Crusting or akingskin
• Redness, warmth, or swelling.
Loss ofhearing/ deafness
Askabout
• Onset and duration, constant vs intermittent, progression,
unilateral or bilater al, high or low tone loss, decreased speech
intelligibility
• Contributing factors:recent infection; loud noise exposure
• Recent tr auma:barotrauma, head injury; exacerbating factors for
tinnitus (sleep, exercise, caeine, alcohol)
• Previous otological surgery, infections
• Medications
• Systemic:history of autoimmune disease, hyper tension, diabetes,
vascular disorders, neurological disease (stroke), depression
• Family history of deafness
• Associated symptoms:aural fullness, fever s, ver tigo, tinnitus,
otalgia, otorrhoea, other neurological complaints.
Lookfor
• Malformations, auricular pits, scars, oedema, mastoid tenderness,
tragal tenderness
• Otoscopy (wax, lesions, masses) and tympanic membrane (colour,
thick ness, presence of uid, perforations)
• Pneumatic otoscopy:test mobilit y of tympanic membrane with
positive and negative pressure
• Fistula test:positive pressure causes nyst agmus which rever ses
with negative pressure (perilymph stula and labyrinthitis)
• Neurological and vestibular exam,bruits
• Tuning forktests.
183

184
CHAP TER6 Theear
Pain (otalgia)
Askabout
• Duration of symptoms
• Discharge
• RecentURTI
• Hearingloss
• Degree of pain and radiation (malignant otitis externa pain is often
severe, post- auricular pain suggests mastoiditis)
• Previous episodes and treatment todate
• Previous ear surger y
• PMH (diabetes or other immune compromise)
• Injury to the mandible (fractured condyle).
Lookfor
• Swelling
• Discharge
• Furuncles
• Signs ofinjur y
• CN VII weakness
• Systemicupset
• Tenderness over the mastoid prominence
• Otoscopy (bulging of the external auditory canal/ tympanic
membrane may appear red, per forated, and discharging).
Swellings
Askabout
• Any known aetiological factors
• Histor y oftr auma
• Any preceding otalgia (perichondritis can arise following otitis
externa and trauma)
• Predisposing factors (e.g. immune compromise)
• Any painful (pseudocyst can present as a painless swelling).
Lookfor
• Conrm presence
• Discharge
• Otoscopy:inspection of ear drum and middle ear (otitis media,
glue ear, cholesteatoma).

EX AMIN ATION OF THEEAR
Examination oftheear
Applied anatomy
Externalear
This comprises of pinna, external auditory meatus (EAM), external auditory canal, and tympanic membrane (ear dr um). The skin on the outer
part of the E AM is self- cleansing and contains hair follicles and waxproducing ceruminous glands. These are absent on the inner part. Ears
are normally self- cleansing and use of cotton buds should be discouraged. If
wax is dislodged into the deeper part of the canal it cannot be removed
by this natural process.
Middleear
This is an air- cont aining space allowing sound transfer to the cochlea. It
contains three ossicles (malleus, incus, and stapes), two muscles (tensor tympani and st apedius), and part of the facial nerve (chorda tympani
branch). Equalization of pressures occurs via the Eustachian tube. The
mastoid air cells communicate with the middle ear space. This is closely
related to the brain (middle cranial fossa), jugular bulb (posteriorly), and
labyrinth (medially). Infections in middle ear are potentially very serious as
they can extend into surrounding structures.
Innerear
This comprises the cochlea, vestibule, and semicircular canals. The
‘membranous’ par t is lled by endolymph and is surrounded by the bony
labyr inth, which is lled with perilymph. The cochlea contains the organ
of hear ing (organ of Corti). The vestibule and semicircular canals are
involved in balance.
Nerve supply totheear
The ear is innervated by the V, IX, and X cranial nerves, and by the posterior roots of C2 and C3. Because of this, pathology at sites of similar
nerve distribution (TMJ, dental, oropharyngeal, laryngeal, and hypopharyngeal) can present with otalgia (referredpain).
Examination oftheear
Inspect the external ear r st. Remove any discharge or wax. Look for
obvious signs of abnormality:
• Size and shape of thepinna
• Extra cartilage tags/ pre- auricular sinuses orpits
• Signs oftrauma
• Skin lesions, e.g. neoplasia
• Skin condition of the pinna and externalcanal
• Infection/ inammation of the external ear canal, with discharge.
Then palpate for mastoid tenderness, tragal tenderness, and lymphadenopathy. Use an otoscope (auroscope) to inspect the exter nal auditory
canal and tympanic membrane. Gr asp the pinna and gently pull it up
and backwards. This helps str aighten the canal for inspection (in infants,
pull the pinna posteriorly). Note the condition of the canal skin and the
185

186
CHAP TER6 Theear
presence of wax, foreign tissue, or discharge. Inspect the tympanic membrane for signs of injury, perforation, or discharge. The mobility of the
eardrum can be evaluated using a pneumatic speculum, which attaches
to the otoscope. Check facial nerve function.
Special manoeuvres include the following:
Dix– Hallpike manoeuvre
This is a positional test for BPPV. The patient sits upright with the legs
extended. Their head is rotated by approximately 45 degrees. The clinician then helps the patient to quickly lie down backwards with the head
held in approximately 20 degrees of extension. The patient’s eyes are
obser ved for nystagmus. If rot ational nystagmus occurs then the test is
considered positive for benign positional vertigo.
Head- shake nystagmus
Move the head in the horizont al plane for 20– 30 seconds, then suddenly
stop and evaluate for nystagmus.
Fistula test and pneumatic otoscopy
Positive pressure causes nystagmus which reverses with negative pressure (perilymph stula and labyrinthitis). Test mobility of tympanic membrane with pressure changes.
Tuning forktests
Rinnetest
Use a 512 Hz tuning fork to compare air conduction (AC) and bone conduction (BC). Strike the tuning fork then place it within 1cm of the EAM
(AC) and then immediately place on the mastoid(BC).
Normal hearing
AC should be greater than BC and so the patient should be able to hear
the tuning fork next to the pinna after they can no longer hear it when
held against the mastoid.
Abnormal hearing
• If they are not able to hear the tuning fork after the mastoid, it means
that BC is greater than AC. Something is inhibiting the passage of
sound from the ear canal, through the middle ear apparatus and into
the cochlea (i.e. there is a conductive hearingloss).
• In sensorineural hearing loss both BC and AC are equally diminished.
Patients with sensorineur al hearing loss can usually hear better on
the mastoid process than air, but indicate the sound has stopped
much earlier than conductive loss patients.
Webertest
Strike the tuning fork and place it in the centre of the forehead. The perceived sound should normally be heard centrally.
Remember that nasophar yngeal pathology can present with unilateral ear
symptoms. Always look at the throat and nasopharynx, especially in adults
presenting with unilateral glue ear — this can be a presenting symptom of a
nasopharyngeal tumour.

INVESTIGATIONS
Investigations
Laborator ytests
• FBC may show a raised WCC (neutrophils).
• Electrolytes:ototoxicity
• Glucose:screen for diabetes (associated with hearingloss)
• Coagulation and immunological prole:coagulopathies can be
associated with hearing disorders
• ESR
• Treponemal studies:Lyme titres/ TPHA/ VDRL/ FTA- ABS depending
on where youwor k
• Lipid prole:atherosclerotic disease can be associated with sudden
senorineural hearingloss
• Fluid analysis (beta- 2 tr ansferr in in suspected CSF leakage)
• Microbiology for any discharge.
Plainlms
Mastoid X- rays:will show opacity of the air cells. These and other special
views of the temporal bone (Schuller’s, Stenvere’s, Towne’s views) have
now largely been replaced by CT andMRI.
CT/ MRI oftemporalbones
These look for potential complications of suppurative ear disease, cho lesteatoma, mastoiditis, temporal bone fracture, congenital disorder or
neoplasm (especially acoustic neuroma).
MRI is often required for suspected cerebellopontine angle tumours,
acoustic neuromas, meningiomas, and petrous apex lesions.
Audiometrictests
Audiometr y tests are carr ied out in soundproofed rooms using precision equipment. Tests may be subjective (pure tone audiograms, speech
audiometry), or objective (impedance audiometry, evoked response
audiometry).
These tests are essential to dene auditory function and to quantify
the thresholds of AC/ BC hearing for both ears. It distinguishes conductive loss from sensorineural hearing loss; cochlear ver sus neural dysfunction, and malingering (pseudohypacusis).
Dierent audiologicaltests:
• Pure tone audiometr y:tests threshold of AC andBC.
• Tympanometry:check s middle ear pressure and impedance (indirect
measure of Eustachian tube function).
• Auditory brainstem response (ABR):recording of the activity of the
eighth ner ve and central auditor y pathway response to an auditor y
signal.
• Otoacoustic emissions (OAEs):tests objective sounds in external
auditory canal emitted from outer hair cells (cochlearecho).
• AB R and OAE are objective tests and do not need the patient’s
cooperation and hence used identify malingerers and for medicolegal purposes.
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