Добавил:
Sekretar
kiopkiopkiop18@yandex.ru
t.me/Prokururor I Вовсе не секретарь, но почту проверяю
Опубликованный материал нарушает ваши авторские права? Сообщите нам.
Вуз:
Предмет:
Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3734_Библиотеки_им_академика_М_И_Перельмана
.pdf
Management ofType B Aortic Dissection
https://t.me/med1917
16. Nawar EW, Niska RW, Xu J.National hospital ambulatory medical care survey: 2005 emer-
gency department summary. Adv Data. 2007 Jun 29;386:1–32.
17. von Kodolitsch Y, Nienaber CA, Dieckmann C, Schwartz AG, Hofmann T, Brekenfeld C,
Nicolas V, Berger J, Meinertz T.Chest radiography for the diagnosis of acute aortic syndrome.
Am J Med. 2004 Jan 15;116(2):73–7.
18. Moore AG, Eagle KA, Bruckman D, Moon BS, Malouf JF, Fattori R, Evangelista A,
Isselbacher EM, Suzuki T, Nienaber CA, Gilon D.Choice of computed tomography, transesophageal echocardiography, magnetic resonance imaging, and aortography in acute aortic
dissection: International Registry of Acute Aortic Dissection (IRAD). Am J Cardiol. 2002 May
15;89(10):1235–8.
19. Mauban EC, Pyeritz R, Hutchison S, Ota T, Upchurch G, Nour K, Taylor B, Braverman
A, Montgomery D, Nienaber C, Isselbacher E.Diagnostic imaging for acute aortic dissection: imaging sensitivity and preference revisited. J Am Coll Cardiol. 2018 Mar 10;71(11
Supplement):A2073.
20. HIRST AE, JOHNS VJ, KIME SW.Dissecting aneurysm of the aorta: a review of 505 cases.
Medicine. 1958 Sep 1;37(3):217.
21. De Bakey ME, Beall AC Jr, Cooley DA, Crawford ES, Morris GC Jr, Garrett HE, Howell JF
Jr. Dissecting aneurysms of the aorta. Surg Clin N Am. 1966 Aug 1;46(4):1045–55.
22. Wheat MW Jr, Palmer RF, Bartley TD, Seelman RC.Treatment of dissecting aneurysms of the
aorta without surgery. J Thorac Cardiovasc Surg. 1965 Sep 1;50(3):364–73.
23. Prokop EK, Palmer RF, Wheat MW Jr. Hydrodynamic forces in dissecting aneurysms: in-vitro
studies in a tygon model and in dog aortas. Circ Res. 1970 Jul;27(1):121–7.
24. Simpson CF, Boucek RJ.The B-aminopropionitrile-fed turkey: a model for detecting potential
drug action on arterial tissue. Cardiovasc Res. 1983 Jan 1;17(1):26–32.
25. Volodos NL, Karpovich IP, Shekhanin VE, Trojan VI, Iakovenko LF.A case of distant trans-
femoral enodprosthesis of the thoracic artery using self-xing synthetic prosthesis in traumatic
aneurysm. Grudn Khir. 1988:84–6.
26. Parodi JC, Palmaz JC, Barone HD.Transfemoral intraluminal graft implantation for abdomi-
nal aortic aneurysms. Ann Vasc Surg. 1991 Nov 1;5(6):491–9.
27. Dake MD, Miller DC, Semba CP, Mitchell RS, Walker PJ, Liddell RP.Transluminal placement
of endovascular stent-grafts for the treatment of descending thoracic aortic aneurysms. N Engl
J Med. 1994 Dec 29;331(26):1729–34.
28. White RA, Miller DC, Criado FJ, Dake MD, Diethrich EB, Greenberg RK, Piccolo RS, Siami
FS. Multidisciplinary Society for Vascular Surgery Outcomes Committee. Report on the
results of thoracic endovascular aortic repair for acute, complicated, type B aortic dissection at
30 days and 1 year from a multidisciplinary subcommittee of the Society for Vascular Surgery
Outcomes Committee. J Vasc Surg. 2011 Apr 1;53(4):1082–90.
29. Dake MD, Miller DC, Mitchell RS, Semba CP, Moore KA, Sakai T.The “rst generation”
of endovascular stent-grafts for patients with aneurysms of the descending thoracic aorta. J
Thorac Cardiovasc Surg. 1998 Nov 1;116(5):689–704.
30. Pape LA, Awais M, Woznicki EM, Suzuki T, Trimarchi S, Evangelista A, Myrmel T, Larsen M,
Harris KM, Greason K, Di Eusanio M.Presentation, diagnosis, and outcomes of acute aortic
dissection: 17-year trends from the International Registry of Acute Aortic Dissection. J Am
Coll Cardiol. 2015 Jul 28;66(4):350–8.
31. Tran TP, Khoynezhad A.Current management of type B aortic dissection. Vasc Health Risk
Manag. 2009;5:53.
32. Khoynezhad A.Antihypertensive therapy in a patient with type B and de novo type A aortic
dissection: case study. Crit Care Clin. 2007 Oct;23:29.
33. Khoynezhad A, Celis R, Hashemzadeh M, Movahed MR, Jalali Z. Current status of tho-
racic endografting and its adjunctive pharmacology. Expert Opin Pharmacother. 2009 Dec
1;10(17):2871–81.
34. Kodama K, Nishigami K, Sakamoto T, Sawamura T, Hirayama T, Misumi H, Nakao K.Tight
heart rate control reduces secondary adverse events in patients with type B acute aortic dissection. Circulation. 2008 Sep 30;118(14_suppl_1):S167–70.
397
398
https://t.me/med1917
35. Eggebrecht H, Schmermund A, Von Birgelen C, Naber CK, Bartel T, Wenzel RR, Erbel
R.Resistant hypertension in patients with chronic aortic dissection. J Hum Hypertens. 2005
Mar;19(3):227–31.
36. Feldman M, Shah M, Elefteriades JA.Medical management of acute type A aortic dissection.
Ann Thorac Cardiovasc Surg. 2009 Oct 1;15(5):286–93.
37. Eggebrecht H, Nienaber CA, Neuhäuser M, Baumgart D, Kische S, Schmermund A, Herold
U, Rehders TC, Jakob HG, Erbel R.Endovascular stent–graft placement in aortic dissection: a
meta-analysis. Eur Heart J. 2006 Feb 1;27(4):489–98.
38. Suzuki T, Mehta RH, Ince H, Nagai R, Sakomura Y, Weber F, Sumiyoshi T, Bossone E,
Trimarchi S, Cooper JV, Smith DE.Clinical proles and outcomes of acute type B aortic dissection in the current era: lessons from the International Registry of Aortic Dissection (IRAD).
Circulation. 2003 Sep 9;108(10_suppl_1):II–312.
39. Kusagawa H, Shimono T, Ishida M, Suzuki T, Yasuda F, Yuasa U, Onoda K, Yada I, Hirano T,
Takeda K, Kato N.Changes in false lumen after transluminal stent-graft placement in aortic
dissections: six years’ experience. Circulation 2005 Jun 7;111(22):2951–2957.
40. Brunkwall J, Kasprzak P, Verhoeven E, Heijmen R, Taylor P, Trialists A, etal. Endovascular
repair of acute uncomplicated aortic type B dissection promotes aortic remodelling: 1 year
results of the ADSORB trial. Eur J Vasc Endovasc Surg. 2014;48(3):285e91.
41. Nienaber CA, Rousseau H, Eggebrecht H. Randomized comparison of strategies for type B
aortic dissection: the INvestigation of STEent Grafts in Aortic Dissection (INSTEAD) trial. J
Vasc Surg. 2010 May 1;51(5):1321–2.
42. Nienaber CA, Kische S, Rousseau H, Eggebrecht H, Rehders TC, Kundt G, Glass A, Scheinert
D, Czerny M, Kleinfeldt T, Zipfel B.Endovascular repair of type B aortic dissection: long-term
results of the randomized investigation of stent grafts in aortic dissection trial. Circ Cardiovasc
Interv. 2013 Aug;6(4):407–16.
43. Gysi J, Schaffner T, Mohacsi P, Aeschbacher B, Althaus U, Carrel T.Early and late outcome of
operated and non-operated acute dissection of the descending aorta. Eur J Cardiothorac Surg.
1997 Jun 1;11(6):1163–70.
44. Roberts WC.Aortic dissection: anatomy, consequences, and causes. Am Heart J. 1981 Feb
1;101(2):195–214.
45. Evangelista A, Isselbacher EM, Bossone E, Gleason TG, Eusanio MD, Sechtem U, Ehrlich
MP, Trimarchi S, Braverman AC, Myrmel T, Harris KM. Insights from the International
Registry of Acute Aortic Dissection: a 20-year experience of collaborative clinical research.
Circulation. 2018 Apr 24;137(17):1846–60.
46. Tsai TT, Nienaber CA, Eagle KA. Acute aortic syndromes. Circulation. 2005 Dec
13;112(24):3802–13.
47. Famularo M, Meyermann K, Lombardi JV. Aneurysmal degeneration of type B aortic dis-
sections after thoracic endovascular aortic repair: a systematic review. J Vasc Surg. 2017 Sep
1;66(3):924–30.
48. Rong D, Ge Y, Liu J, Liu X, Guo W.Combined proximal descending aortic endografting plus
distal bare metal stenting (PETTICOAT technique) versus conventional proximal descending
aortic stent graft repair for complicated type B aortic dissections. Cochrane Database Syst
Rev. 2019;10
49. Bertoglio L, Rinaldi E, Melissano G, Chiesa R. The PETTICOAT concept for endovascular
treatment of type B aortic dissection. J Cardiovasc Surg. 2019 Feb;60(1):91–9.
50. Lombardi JV, Cambria RP, Nienaber CA, Chiesa R, Teebken O, Lee A, Mossop P, Bharadwaj
P. Prospective multicenter clinical trial (STABLE) on the endovascular treatment of complicated type B aortic dissection using a composite device design. J Vasc Surg. 2012 Mar
1;55(3):629–40.
51. Sobocinski J, Fabre D, Azzaoui, R, Haulon S.TEVAR alone versus the STABLE technique for
acute complicated TBAD.Endovascular Today Supplement. 2019 Nov.
A. Khoynezhad et al.
Management ofType B Aortic Dissection
https://t.me/med1917
52. Hofferberth SC, Nixon IK, Boston RC, McLachlan CS, Mossop PJ.Stent-assisted balloon-
induced intimal disruption and relamination in aortic dissection repair: the STABILISE concept. J Thorac Cardiovasc Surg. 2014 Apr 1;147(4):1240–5.
53. Faure EM, El Batti S, Rjeili MA, Julia P, Alsac JM.Mid-term outcomes of stent assisted bal-
loon induced intimal disruption and relamination in aortic dissection repair (STABILISE) in
acute type B aortic dissection. Eur J Vasc Endovasc Surg. 2018 Aug 1;56(2):209–15.
54. Patel HJ, Williams DM, Meekov M, Dasika NL, Upchurch GR Jr, Deeb GM.Long-term results
of percutaneous management of malperfusion in acute type B aortic dissection: implications
for thoracic aortic endovascular repair. J Thorac Cardiovasc Surg. 2009 Aug 1;138(2):300–8.
55. Wilkinson DA, Patel HJ, Williams DM, Dasika NL, Deeb GM.Early open and endovascular
thoracic aortic repair for complicated type B aortic dissection. Ann Thorac Surg. 2013 Jul
1;96(1):23–30.
56. Zeeshan A, Woo EY, Bavaria JE, Fairman RM, Desai ND, Pochettino A, Szeto WY.Thoracic
endovascular aortic repair for acute complicated type B aortic dissection: superiority relative
to conventional open surgical and medical therapy. J Thorac Cardiovasc Surg. 2010;140(6
Suppl):S109–15.
57. Zhu Y, Wang B, Meng Q, Liu J, Zhai S, He J. Long-term efcacy of endovascular vs open
surgical repair for complicated type-B aortic dissection: a single-center retrospective study and
meta-analysis. Braz J Med Biol Res. 2016;49(6).
58. van Bogerijen GH, Patel HJ, Williams DM, Yang B, Dasika NL, Eliason JL, Deeb
GM.Propensity adjusted analysis of open and endovascular thoracic aortic repair for chronic
type B dissection: a twenty-year evaluation. Ann Thorac Surg. 2015 Apr 1;99(4):1260–6.
59. Lou X, Chen EP, Duwayri YM, Veeraswamy RK, Jordan WD Jr, Zehner CA, Leshnower
BG.The impact of thoracic endovascular aortic repair on long-term survival in type B aortic
dissection. Ann Thorac Surg. 2018 Jan 1;105(1):31–8.
60. Fattori R, Cao P, De Rango P, Czerny M, Evangelista A, Nienaber C, Rousseau H, Schepens
M.Interdisciplinary expert consensus document on management of type B aortic dissection. J
Am Coll Cardiol. 2013 Apr 23;61(16):1661–78.
399
Subacute andChronic Type AAortic
https://t.me/med1917
Dissection
LarsG.Svensson
Current anatomic descriptions of aortic dissection categorize dissection based on
where the original intimal tear is located (DeBakey classication) or whether the
ascending aorta is involved (Stanford classication). Following a study of aortic
dissection of 690 patients surgically treated by E Stanley Crawford etal. [
acute and chronic aortic dissections were dened as dissections operated on between
two and six weeks and beyond six weeks after onset of symptoms, respectively [1–
5]. Further, when dealing with subacute and chronic aortic dissection, it is worth-
while to recall classes of aortic dissection, particularly for intramural hematomas
[6]. This chapter examines how course of treatment for aortic dissection is determined based on class, comorbidities, and time interval from dissection.
Our previous study [6] categorizing aortic dissection into subclasses was based
on the appearance of the aortic dissection, apart from the well-known denitions of
extent from DeBakey and Stanford (Fig.1) [4, 5, 7]. Class I tears are associated with
classic aortic dissection and two classical lumens for extent DeBakey I or II (recall
Stanford A includes the ascending aorta, Stanford B does not). Class II tears are
intramural hematomas. While some 15% are classied as such by computed tomography (CT) or magnetic resonance imaging (MRI), in actuality at postmortem evaluation, only 5% have no tear. We rarely see tears that do not involve an intimal tear;
usually, if carefully looked for, even tears of 1–2mm in diameter can be found or in
the descending aorta with retrograde dissection or, more rarely, from the abdominal
aorta. Of note, some institutions, particularly with older patients, treat intramural
Class II aortic dissection conservatively and then perform surgery in the chronic
phase, if needed. Class III tears involve limited areas of dissection with exposure of
the tunica media or middle layer but without extensive dissection of the intimal ap
that separates the false from the true lumen or undermining of the tunica intima, or
1], sub-
L. G. Svensson (*)
Department of Thoracic and Cardiovascular Surgery, Heart, Vascular, and Thoracic Institute,
Cleveland Clinic, Cleveland, OH, USA
e-mail: svenssl@ccf.org
J. S. Coselli et al. (eds.), Aortic Dissection and Acute Aortic Syndromes,
https://doi.org/10.1007/978-3-030-66668-2_28
401© Springer Nature Switzerland AG 2021
402
Proximal
Ascending,
Arch and
Descending
Descending
Proximal
Distal
abc
https://t.me/med1917
L. G. Svensson
Ascending
DeBakey I and II
Stanford A
Stanford B
Arch and
Descending
Descending
penetrating ulcer
latrogenic
DeBakey IIIa and IIIb
Stanford B
Fig. 1 (a) Proximal, DeBakey I and II, Stanford A. (b) Proximal, Stanford B. (c) Distal, DeBakey
IIIa and IIIb, Stanford B.Reprinted from Svensson LG.Limited intimal aorta tears: Royalty torn
asunder, and a nation was created, J Am Coll Cardiol. 2019;71:2786–2789. Copyright © 2019,
with permission from Elsevier [7]
inner layer (Fig.2) [7]. In earlier studies [6, 7], we described the difculty in identifying limited Class III aortic dissection by imaging, noting that angiography of the
ascending aorta and root from multiple angles shows the bulge. Subsequently, the
Stanford group [7, 8] indicated that 4.8% of patients in their series had this Class III
dissection. Aortic dissections in these patients are often missed and present in the
subacute phase, for example, because of pericardial effusion or progression of dissection diagnosed in retrospect or at the time of frank aortic rupture. Class IV tears
are penetrating ulcers that appear to have a different etiology, often associated with
calcication of the aortic wall, frequently with infection. While they may be seen in
the ascending aorta, more often, class IV tears are seen in the lesser curve of the
aortic arch, proximal descending thoracic aorta, or opposite the visceral arteries. Of
note, the natural history is often much more lethal than expected. Based on postmortem examinations done by the author many years ago, the plane of dissection is
often between the tunica media and tunica adventitia, and hence, rupture occurs or
patients present with large pleural effusions more often. Class V tears are iatrogenic
dissections, typically seen after an attempted catheterization of the coronary ostia or
after transcatheter aortic valve replacement.
IV V
Subacute andChronic Type AAortic Dissection
https://t.me/med1917
403
I
Fig. 2 Classes of local aortic dissection. Class 1, classic dissection with ap between true and
false aneurysm and clot in false lumen; Class 2, intramural hematoma; Class 3, limited intimal tear
with eccentric bulge at tear site; Class 4, penetrating atherosclerotic ulcer with surrounding hematoma, usually subadventitial; Class 5, latrogenic or traumatic dissection illustrated by coronary
catheter causing dissection. Reprinted from Svensson LG.Limited intimal aorta tears: Royalty
torn asunder, and a nation was created, J Am Coll Cardiol. 2019;71:2786–2789. Copyright © 2019,
with permission from Elsevier [7]
II III
Because not all patients with acute or chronic dissections are symptomatic, such
as patients with Marfan syndrome, and the interval from dissection may be unknown,
the question then becomes, do all subacute dissections or chronic dissections require
surgical intervention? E Stanley Crawford, MD, believed that all Type A Class I
aortic dissections, even if not dilated, should be treated surgically unless other serious comorbidities precluded surgery. We have followed that advice, particularly as
404
https://t.me/med1917
L. G. Svensson
it is not unusual to nd healed tears with superimposed new acute dissections, particularly for Class III tears.
As more patients with Class II intramural hematomas are treated conservatively,
including elderly patients or patients with diabetes, and many may resolve with the
appearance of two lumens, when should these patients undergo surgery? Our general advice has been to operate if there are complications, like stroke perhaps related
to platelet/thrombi or if the size exceeds the cross-sectional area to height ratio,
exceeding a ratio of 10 [9, 10].
With modern imaging, it has become increasingly rare to see patients with Type
A chronic Class I tears who have not had surgery. Nevertheless, in a series of 151
patients with Marfan syndrome, one patient was surgery-free for 27years after a
Type A dissection, a distinct outlier [11].
In emergency and urgent Type A aortic dissections, the priority is to save the
patient’s life and deal with a denitive procedure and the consequences of the aortic
dissection later, if needed. Clearly, for emergency and urgent operations, acute dissection outcomes are not as positive. Our mortality rate of 595 patients with acute dissection was 8.1% and stroke risk was 7.6% following ascending/hemi-arch repairs; for
total arch replacements, mortality and stroke were 9.7% and 8.4%, respectively, as
reported at the 2018 American Association for Thoracic Surgery meeting on whether
mortality could be reduced to less than 5% [12]. This was supported by a more detailed
analysis [13] of our data, although the risks are higher in an analysis of STS data [14].
The denitive operations in these two time-related (subacute or chronic) subtypes should thus be used for dilated aortas without aortic dissection. Hence, for
larger roots with trileaet valves, an aortic valve reimplantation and replacement of
the root and ascending aorta should be used if needed [15]. In our current series of
over 1000 reimplantations, we have done this operation frequently for subacute or
chronic dissection with no deaths and 97% overall freedom from reoperation at
10years [15]. For patients with bicuspid valves, the procedure is chosen as appropriate for each individual case. For example, procedures can involve reimplantation,
the inclusion technique type of remodeling, simple tricuspid valve repair, or ascending arch with separate tube graft replacement [16]. In some patients, the root can be
left alone and only the ascending aorta and arch replaced [17]. How the arch and
greater vessels are dealt with is a matter of getting the most durable repair, depending also if connective tissue disorder is present in the patient. An ascending aorta
tube graft is all that is needed for many classic DeBakey Type 2 extent dissections.
For most patients, a total arch with an elephant trunk procedure [18], even if prophylactic for a non dilated descending aorta [19], is the procedure of choice for
more extensive dissections (DeBakey Type 1). If the patient has a connective tissue
disorder, especially if young, the best option is an elephant trunk procedure distally.
In this case, separate tube grafts for the innominate artery or separate right subclavian and right carotid artery with another tube graft to the left carotid artery, with or
without grafting of the left subclavian artery are key depending on the distal elephant trunk anastomosis site.
For brain protection, key methods are cooling to a nasopharyngeal temperature
below 20°C, CO
eld ooding at 10 L/min, right subclavian artery perfusion,
2
Subacute andChronic Type AAortic Dissection
https://t.me/med1917
405
centrifugal pump with white cell ltration, steep Trendelenburg position, and meticulous de-airing of CO2 from the arch by antegrade perfusion from the right subclavian artery. In our recent prospective, randomized trial of total arch replacements,
there was no difference between antegrade and retrograde brain perfusion [20]; if
circulatory arrest time is expected to be less than 30minutes, neither is used based
on our ndings that risk of stroke increases after 40minutes [21]. In the abovementioned prospective, randomized trial of total arch replacements, 30-day mortality was 0.8% and stroke was also 0.8%, despite 39% being reoperations and 61%
having elephant trunk procedures [20].
The management of patients with chronic Type A aortic dissections after previous repairs should be similar to those who have not had surgery for subacute or
chronic dissection, with the exception of chest entry and perhaps valve reimplantation. Hence, the priority is denitive repair of the ascending aorta and arch. While
some programs have chosen to treat a dilated descending thoracic aorta that is dissected by dealing with the enlarged descending segment rst, our preference has
been to do a total arch with elephant trunk procedure and then a second-stage
descending aortic repair [18]. The root is treated as needed.
For chest re-entry, our preference is to place the patient on cardiopulmonary
bypass using a side graft on the right subclavian artery and cannulating the right
femoral vein with a venous cannula threaded into the right atrium with transesophageal echocardiography [22]. The patient is opened on pump and given lidocaine and
atropine to delay brillation when cooling. For severe aortic valve regurgitation, a
percutaneous retrograde cardioplegic cannula is placed by anesthesia to allow for
arresting the heart until antegrade cardioplegia can be given.
In summary, because the management of subacute and chronic aortic dissection
is typically completed by elective surgery, a denitive aortic operation should be
performed as often as possible so that there is no need for another operation via
median sternotomy [23].
Author Disclosures
The author declares no conicts of interest.
Funding
The study was funded in part by the Delos M. Cosgrove, MD, Chair for Heart
Disease Research.
References
1. Svensson LG, Crawford ES, Hess KR, Coselli JS, Sa HJ.Dissection of the aorta and dissecting
aortic aneurysms: improving early and long-term surgical results. Circulation. 1990;82(suppl
IV):IV-24–IV-48.
2. Crawford ES, Svensson LG, Coselli JS, Sa HJ, Hess KR. Aortic dissection and dissecting
aortic aneurysms. Ann Surg. 1988;208:254–73.
3. Svensson LG, Crawford ES.Aortic dissection and aortic aneurysm surgery, II: clinical observa-
tions, experimental investigations, and statistical analyses. Curr Probl Surg. 1992;29:917–22.
406
https://t.me/med1917
4. Svensson LG, Adams DH, Bonow RO, Kouchoukos NT, Miller DC, O'Gara PT, Shahian
DM, Schaff HV, Akins CW, Bavaria JE, Blackstone EH, David TE, Desai ND, Dewey TM,
D'Agostino RS, Gleason TG, Harrington KB, Kodali S, Kapadia S, Leon MB, Lima B, Lytle
BW, Mack MJ, Reardon M, Reece TB, Reiss GR, Roselli EE, Smith CR, Thourani VH, Tuzcu
EM, Webb J, Williams MR.Aortic valve and ascending aorta guidelines for management and
quality measures. Ann Thorac Surg. 2013;95(6 Suppl):S1–66.
5. Hiratzka LF, Bakris GL, Beckman JA, Bersin RM, Carr VF, Casey DE Jr, Eagle KA, Hermann
LK, Isselbacher EM, Kazerooni EA, Kouchoukos NT, Lytle BW, Milewicz DM, Reich DL,
Sen S, Shinn JA, Svensson LG, Williams DM, American College of Cardiology Foundation;
American Heart Association Task Force on Practice Guidelines; American Association for
Thoracic Surgery; American College of Radiology; American Stroke Association; Society of
Cardiovascular Anesthesiologists; Society for Cardiovascular Angiography and Interventions;
Society of Interventional Radiology; Society of Thoracic Surgeons; Society for Vascular
Medicine. 2010 ACCF/AHA/AATS/ACR/ASA/SCA/SCAI/SIR/STS/SVM guidelines for the
diagnosis and management of patients with thoracic aortic disease: executive summary. A
report of the American College of Cardiology Foundation/American Heart Association Task
Force on Practice Guidelines, American Association for Thoracic Surgery, American College of
Radiology, American Stroke Association, Society of Cardiovascular Anesthesiologists, Society
for Cardiovascular Angiography and Interventions, Society of Interventional Radiology,
Society of Thoracic Surgeons, and Society for Vascular Medicine. Catheter Cardiovasc Interv.
2010;76:E43–86.
6. Svensson LG, Labib SB, Eisenhauer AC, Butterly JR.Intimal tear without hematoma: an
important variant of aortic dissection that can elude current imaging techniques. Circulation.
1999;99:1331–6.
7. Svensson LG.Limited intimal aorta tears: Royalty torn asunder, and a nation was created. J
Am Coll Cardiol. 2019;71:2786–9.
8. Chin AS, Willemink MJ, Kino A, Hinostroza V, Sailer AM, Fischbein MP, Mitchell RS, Berry
GJ, Miller DC, Fleischmann D.Acute limited intimal tears of the thoracic aorta. J Am Coll
Cardiol. 2018;71:2773–85.
9. Svensson LG, Kim K-H, Lytle BW, Cosgrove DM.Relationship of aortic cross-sectional area
to height ratio and the risk of aortic dissection in patients with bicuspid aortic valves. J Thorac
Cardiovasc Surg. 2003;126:892–3.
10. Masri A, Kalahasti V, Svensson LG, Roselli EE, Johnston D, Hammer D, Schoenhagen P,
Grifn BP, Desai MY.Aortic cross-sectional area/height ratio and outcomes in patients with a
trileaet aortic valve and a dilated aorta. Circulation. 2016;134:1724–37.
11. Svensson LG, Crawford ES, Coselli JS, Sa HJ, Hess KR.Impact of cardiovascular operation
on survival in the Marfan patient. Circulation. 1989;80(suppl I):I-233–I-242.
12. Svensson LG. How to repair an uncomplicated Type A dissection with <5% mortality.
Presented at the American Association for Thoracic Surgery 98th Annual Meeting. April 29–
May 1, 2018; San Diego, CA.
13. Rosinski BF, Idrees JJ, Roselli EE, Germano E, Pasadyn SR, Lowry AM, Blackstone
EH, Johnston DR, Soltesz EG, Navia JL, Desai MY, Mick SL, Bakaeen FG, Svensson
LG. Cannulation strategies in acute type A dissection repair: A systematic axillary artery
approach. J Thorac Cardiovasc Surg. 2019;158:647–659.e5.
14. Ghoreishi M, Sundt TM, Cameron DE, Holmes SD, Roselli EE, Pasrija C, Gammie JS, Patel
HJ, Bavaria JE, Svensson LG, Taylor BS.Factors associated with acute stroke after type A aortic dissection repair: an analysis of the Society of Thoracic Surgeons National Adult Cardiac
Surgery Database. J Thorac Cardiovasc Surg. 2020 Jun;159(6):2143–2154.e3.
15. Svensson LG. Root reimplantation with leaet repair. Semin Thorac Cardiovasc Surg.
2019;31:153–4.
16. Zeeshan A, Idrees JJ, Johnston DR, Rajeswaran J, Roselli EE, Soltesz EG, Gillinov AM,
Grifn B, Grimm R, Hammer DF, Pettersson GB, Blackstone EH, Sabik JF 3rd, Svensson
L. G. Svensson
Subacute andChronic Type AAortic Dissection
https://t.me/med1917
LG.Durability of aortic valve cusp repair with and without annular support. Ann Thorac Surg.
2018;105:739–48.
17. Svensson LG, Al Kindi AH, Vivacqua A, Pettersson GB, Gillinov AM, Mihaljevic T, Roselli
EE, Sabik JF 3rd, Grifn B, Hammer DF, Rodriguez L, Williams SJ, Blackstone EH, Lytle
BW.Long-term durability of bicuspid aortic valve repair. Ann Thorac Surg 2014;97:1539–1547;
discussion 1548.
18. Svensson LG, Rushing GD, Valenzuela ES, Rafael AE, Batizy LH, Blackstone EH, Roselli EE,
Gillinov AM, Sabik JF 3rd, Lytle BW.Modications, classication, and outcomes of elephanttrunk procedures. Ann Thorac Surg. 2013;96:548–58.
19. Idrees JJ, Roselli EE, Wojnarski CM, Feng K, Aftab M, Johnston DR, Soltesz EG, Sabik JF
3rd, Svensson LG.Prophylactic stage 1 elephant trunk for moderately dilated descending aorta
in patients with predominantly proximal disease. J Thorac Cardiovasc Surg. 2015;150:1150–5.
20. Svensson LG, Blackstone EH, Apperson-Hansen C, Ruggieri PM, Ainkaran P, Naugle RI,
Lima B, Roselli EE, Cooper M, Somogyi D, Tuzcu EM, Kapadia S, Clair DG, Sabik JF 3rd,
Lytle BW.Implications from neurologic assessment of brain protection for total arch replacement from a randomized trial. J Thorac Cardiovasc Surg. 2015;150:1140–7.e11.
21. Svensson LG, Crawford ES, Hess KR, Coselli JS, Raskin S, Shenaq SA, Sa HJ.Deep hypo-
thermia with circulatory arrest. Determinants of stroke and early mortality in 656 patients. J
Thorac Cardiovasc Surg 1993;106:19–28; discussion 28–31.
22. Svensson LG, Blackstone EH, Rajeswaran J, Sabik JF 3rd, Lytle BW, Gonzalez-Stawinski G,
Varvitsiotis P, Banbury MK, McCarthy PM, Pettersson GB, Cosgrove DM.Does the arterial
cannulation site for circulatory arrest inuence stroke risk? Ann Thorac Surg. 2004;78:1274–84.
discussion 1274-1284
23. Svensson LG. Are we there yet? Emerging milestones in aortic dissection care. J Am Heart
Assoc. 2019;8:e012402.
407
Соседние файлы в папке Библиотека им академика М.И. Перельмана