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44. Misselbeck TS, Teicher EJ, Cipolle MD, et al. Hepatic angioembolization in trauma patients: indications and complications. J Trauma. 2009;67:769–773.
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47. Ciraulo DL, Luk S, Palter M, et al. Selective hepatic arterial embolization of grade IV and V blunt hepatic injuries: an extension of resuscitation in the nonoperative management of traumatic hepatic injuries. J Trauma. 1998;45:353–359.
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P
23

Pelvic Trauma

Pierre E. Bize • Yann Lachenal • Alban Denys
elvic hemorrhage is usually related to unstable pelvic fractures, which are observed in up to 25% of all polytrauma patients.1 Because pelvic
fractures are usually caused by high-energy trauma and often associated with injuries in other parts of the body, their management requires a multidisciplinary approach and triage decision between all bleeding sites. Despite the advances made in all disciplines of resuscitative medicine and surgery, the mortality rate following pelvic ring injuries ranges from 20% to 40%.
25
Pelvic trauma represents a particular physiologic condition in the trauma spectrum. Usually, the pelvic compartment represents a closed space in which tamponade occurs rapidly, limiting the extension of bleeding. But in case of pelvic ring disruption, this tamponade effect does not occur, and it has been demonstrated that a gap of only 3 cm in the pelvic symphysis increases the volume of the pelvic compartment of as much as 1.5 L.
6
Then, in case of pelvic fracture, patients can bleed up to 4 L of blood in a closed, difficult-to-reach compartment. According to the Advanced Trauma Life Support (ATLS) recommendation, the external stabilization of the pelvis should be performed in any case where there is suspicion or clinical evidence
of pelvic fracture.7 This can be achieved at the patient’s arrival in the hospital or before using dedicated pelvic belts. A chest and pelvic x-ray should be obtained in the emergency room in all trauma patients to rule out hemothorax or pneumothorax and assess the presence or absence of evident pelvic fractures. Hemodynamically stable patient should then undergo computed tomography (CT) scan for a throughout workup. Hemodynamically unstable patients should benefit from a focused assessment by sonography for trauma (FAST) to look for free peritoneal fluid. Unstable patients with free abdominal fluid should be rushed to the operating room for exploratory laparotomy. However, note that if the peritoneum is not breached, the bleeding can be completely occult at FAST. In case of hemodynamically unstable patients without any other seriously bleeding injury and with no, or not enough, free intraperitoneal fluid to explain instability, retroperitoneal
bleeding from the pelvis should be suspected.
5
Bleeding in the pelvis can occur from arteries, veins, or directly from the broken bones. The immobilization of the pelvic ring with a strap or an external fixator helps restore the tamponade effect and has been shown to restore hemodynamic stability in polytrauma patients suffering from pelvic fractures.8 However, tamponade is efficient only in case of bleeding from broken bones or lacerated veins. In patients with persistent hemodynamic instability despite pelvic ring immobilization, an arterial bleeding should be suspected. In this case, four treatment options are usually described: immediate external surgical fixation, direct surgical vessel ligation, retroperitoneal packing, and transcatheter arterial embolization (TAE).
9
There is no consensus on which of these treatment options is the most efficient in restoring hemodynamic stability, but there is a growing evidence in the literature suggesting that external fixation should be performed first and does not preclude any other subsequent adjuvant treatment.9 However, this can be ponderated when arterial lesions are evidenced on the CT scanner and when surgical procedure is complex and would delay arterial bleeding control by TAE.
Figure 23.1 describes the decision-making algorithm proposed by
Geeraerts et al.
5
There are many classifications of pelvic fractures based on the underlying traumatic mechanism or on the presence or absence of posterior ring elements instability.10 The classification of Pennal et al.11 describes three types of underlying mechanism: type 1 (anteroposterior compression), resulting in transverse opening of the pelvic ring (open book fracture) and risk of lesion of the internal iliac artery (IIA); type 2 (lateral compression), with risk of lesion of the iliac vessels and retropubic venous plexus; and type 3 (vertical shearing), with structural instability of the posterior ring elements. The classification of Burgess et al.12 adds a fourth category for mixed