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Stress-Induced Cardiomyopathy
DOI: http://dx.doi.org/10.5772/.105584
[15]
311
https://t.me/medicina_free
Williams RB, Barefoot JC,
Schneiderman N. Psychosocial risk
factors for cardiovascular disease:
More than one culprit at work. Journal
of the American Medical Association.
2003;(16):2190-2192
[16]
Neylon A, Canniffe C, Anand S,
Kreatsoulas C, Blake GJ, Sugrue D, et
al. A global perspective on psychosocial
risk factors for cardiovascular disease.
Progress in Cardiovascular Diseases.
2013;(6):574-581
[22] Bradt J, Dileo C, Potvin N. Music for
stress and anxiety reduction in coronary
heart disease patients. Cochrane
Database of Systematic Reviews.
2013;:CD006577
[23] Karlsen HR, Matejschek F,
Saksvik-Lehouillier I, Langvik E. Anxiety
as a risk factor for cardiovascular disease
independent of depression: A narrative
review of current status and conflicting
findings. Health Psychology Open.
2021;(1):2055
[17]
Ingles J, Goldstein J, Thaxton C,
Caleshu C, Corty EW, Crowley SB, et
al. Evaluating the clinical validity of
hypertrophic cardiomyopathy genes.
Circulation. Genomic and Precision
Medicine. 2019;(2):e002460
[18]
Hohls JK, Beer K, Arolt V,
Haverkamp W, Kuhlmann SL, Martus P,
et al. Association between heart-focused
anxiety, depressive symptoms, health
behaviors and healthcare utilization in
patients with coronary heart disease.
Journal of Psychosomatic Research.
2020;:109958
[19]
Meader N, King K, Wright K,
Graham HM, Petticrew M, Power C,
et al. Multiple risk behavior
interventions: Meta-analyses of RCTs.
American Journal of Preventive
Medicine. 2017;(1):e19-e30
[20]
Jiang W, Krishnan RR,
O'Connor CM. Depression and heart
disease: Evidence of a link, and its
therapeutic implications. CNS Drugs.
2002;(2):111-127
[21]
Gathright EC, Goldstein CM,
Josephson RA, Hughes JW. Depression
increases the risk of mortality in patients
with heart failure: A meta-analysis.
Journal of Psychosomatic Research.
2017;:82-89
[24] Tully PJ, Cosh SM, Baune BT. A
review of the affects of worry and
generalized anxiety disorder upon
cardiovascular health and coronary heart
disease. Psychology, Health & Medicine.
2013;(6):627-644
[25] Pankalainen M, Kerola T,
Kampman O, Kauppi M,
Hintikka J. Pessimism and risk of death
from coronary heart disease among
middle-aged and older Finns: An elevenyear follow-up study. BMC Public Health.
2016;(1):1124
[26] Pankalainen MT, Kerola TV,
Hintikka JJ. Pessimism and the risk for
coronary heart disease among middleaged and older Finnish men and women:
A ten-year follow-up study. BMC
Cardiovascular Disorders. 2015;:113
[27] Chida Y, Steptoe A. The association
of anger and hostility with future
coronary heart disease: A meta-analytic
review of prospective evidence. Journal
of the American College of Cardiology.
2009;(11):936-946
[28] Chen H, Zhang B, Xue W, Li J,
Li Y, Fu K, et al. Anger, hostility and
risk of stroke: A meta-analysis of
cohort studies. Journal of Neurology.
2019;(4):1016-1026
[29] Biber S, Andonian C, Beckmann J,
Ewert P, Freilinger S, Nagdyman N,
Novel Pathogenesis and Treatments for Cardiovascular Disease
312
https://t.me/medicina_free
et al. Current research status on the
psychological situation of parents of
children with congenital heart disease.
Cardiovascular Diagnosis and Therapy.
2019;(Suppl 2):S369-SS76
[30] Dragano N, Siegrist J, Nyberg ST,
Lunau T, Fransson EI, Alfredsson L,
et al. Effort-Reward Imbalance at
Work and Incident Coronary Heart
Disease: A Multicohort Study of
90,164 Individuals. Epidemiology.
2017;(4):619-626
[31] Taouk Y, Spittal MJ, LaMontagne AD,
Milner AJ. Psychosocial work stressors
and risk of all-cause and coronary heart
disease mortality: A systematic review
and meta-analysis. Scandinavian Journal
of Work, Environment & Health.
2020;(1):19-31
[32] Sbarra DA, Coan JA. Divorce and
health: Good data in need of better
theory. Current Opinion in Psychology.
2017;:91-95
by social isolation and loneliness. Redox
Biology. 2020;:101585
[37] Golaszewski NM, LaCroix AZ,
Godino JG, Allison MA, Manson JE,
King JJ, et al. Evaluation of social
isolation, loneliness, and cardiovascular
disease among older women in
the US. JAMA Network Open.
2022;(2):e2146461
[38] Kim ES, Chen Y, Nakamura JS,
Ryff CD, VanderWeele TJ. Sense of
purpose in life and subsequent physical,
behavioral, and psychosocial health:
An outcome-wide approach. American
Journal of Health Promotion.
2022;(1):137-147
[39] Jaschinski C, Knetsch V, Parzer P,
Meyr J, Schroeder B, Fonseca E, et al.
Psychosocial impact of congenital heart
diseases on patients and their families:
A parent's perspective. World Journal of
Pediatric Congenit ive and Heart Surgery.
2022;(1):9-15
[33] Dhindsa DS, Khambhati J,
Schultz WM, Tahhan AS, Quyyumi AA.
Marital status and outcomes in patients
with cardiovascular disease. Trends
in Cardiovascular Medicine.
2020;(4):215-220
[34] Grauman A, Viberg Johansson J,
Falahee M, Veldwijk J. Public perceptions
of myocardial infarction: Do illness
perceptions predict preferences for
health check results. Preventive Medical
Reports. 2022;:101683
[35] Kilby CJ, Sherman KA,
Wuthrich VM. A scoping review of
stress beliefs: Literature integration,
measurement issues, and theoretical
concerns. Annals of Behavioral Medicine.
2020;(8):595-610
[36] Li H, Xia N. The role of oxidative
stress in cardiovascular disease caused
[40] Kovacs AH, Bellinger DC.
Neurocognitive and psychosocial
outcomes in adult congenital heart
disease: A lifespan approach. Heart.
2021;(2):159-167
[41] Osei AD, Mirbolouk M,
Orimoloye OA, Dzaye O, Uddin SMI,
Benjamin EJ, et al. Association between
E-cigarette use and cardiovascular
disease among never and current
combustible-cigarette smokers.
The American Journal of Medicine.
2019;(8):949-54 e2
[42] Sylvia LG, Faulkner M, Rakhilin M,
Amado S, Gold AK, Albury EA, et al.
An online intervention for increasing
physical activity in individuals
with mood disorders at risk for
cardiovascular disease: Design
considerations. Journal of Affective
Disorders. 2021;:102-109
Stress-Induced Cardiomyopathy
DOI: http://dx.doi.org/10.5772/.105584
[43]
313
https://t.me/medicina_free
Damasio AR. Emotion in the
perspective of an integrated nervous
system. Brain Research. Brain Research
Reviews. 1998;(2-3):83-86
[44]
Sirois BC, Burg MM. Negative
emotion and coronary heart disease:
A review. Behaviour Modification.
2003;(1):83-102
[45]
Tuck NL, Adams KS, Pressman SD,
Consedine NS. Greater ability to express
positive emotion is associated with
lower projected cardiovascular disease
risk. Journal of Behavioral Medicine.
2017;(6):855-863
[46]
AbuRuz ME. Anxiety and
depression predicted quality of life
among patients with heart failure.
Journal of Multidisciplinary Healthcare.
2018;:367-373
[47]
Meyer FA, von Kanel R, Saner H,
Schmid JP, Stauber S. Positive affect
moderates the effect of negative affect
on cardiovascular disease-related
hospitalizations and all-cause mortality
after cardiac rehabilitation. European
Journal of Preventive Cardiology.
2015;(10):1247-1253
[48]
Marshall CR, Hardy CJD, Allen M,
Russell LL, Clark CN, Bond RL,
et al. Cardiac responses to viewing
facial emotion differentiate
frontotemporal dementias. Annals
of Clinical Translational Neurology.
2018;(6):687-696
[49]
Knapp M, Tu X, Wu R. Vascular
endothelial dysfunction, a major
mediator in diabetic cardiomyopathy.
Acta Pharmacological Sinica.
2019;(1):1-8
[50]
Hassan M, York KM, Li H, Li Q ,
Gong Y, Langaee TY, et al. Association
of beta1-adrenergic receptor
genetic polymorphism with mental
stress-induced myocardial ischemia
in patients with coronary artery
disease. Archives of Internal Medicine.
2008;(7):763-770
[51] Chen ZY, Jing D, Bath KG, Ieraci A,
Khan T, Siao CJ, et al. Genetic variant
BDNF (Val66Met) polymorphism
alters anxiety-related behavior. Science.
2006;(5796):140-143
[52] Hajjari P, Mattsson S, McIntyre KM,
McKinley PS, Shapiro PA, Gorenstein EE,
et al. The effect of hostility reduction
on autonomic control of the heart and
vasculature: A Randomized Controlled
Trial. Psychosomatic Medicine.
2016;(4):481-491
[53] Paine NJ, Watkins LL, Blumenthal JA,
Kuhn CM, Sherwood A. Association
of depressive and anxiety symptoms
with 24-hour urinary catecholamines in
individuals with untreated high blood
pressure. Psychosomatic Medicine.
2015;(2):136-144
[54] Liu T. Effect of negative emotion
evocation on autonomic function activity
of hearts in healthy population: An
empirical study. Academic Journal of
Second Military Medical University.
2011;:1204-1207
[55] Liu MY, Yang Y, Zhang LJ, Pu LH,
He DF, Liu JY, et al. Potential predictors
for mental stress-induced myocardial
ischemia in patients with coronary
artery disease. Chinese Medical Journal.
2019;(12):1390-1399
[56] Hammadah M, Sullivan S, Pearce B,
Al Mheid I, Wilmot K, Ramadan R, et al.
Inflammatory response to mental stress
and mental stress induced myocardial
ischemia. Brain, Behavior, and Immunity.
2018;:90-97
[57] Jiang W, Velazquez EJ, Samad Z,
Kuchibhatla M, Martsberger C, Rogers J,
Novel Pathogenesis and Treatments for Cardiovascular Disease
314
https://t.me/medicina_free
et al. Responses of mental stress-induced
myocardial ischemia to escitalopram
treatment: Background, design, and
method for the Responses of Mental
Stress Induced Myocardial Ischemia to
Escitalopram Treatment trial. American
Heart Journal. 2012;(1):20-26
[58] Benjamin MM, Bossarte R, Guha A,
Shah M, Patel B. Depression and anxiety
in patients with heart disease and/
or cancer based on the National
Health Interview Survey. Proceedings
(Baylor University Medical Center).
2020;(1):11-16
[59] Bremner JD, Campanella C, Khan Z,
Fani N, Kasher N, Evans S, et al. Brain
mechanisms of stress and depression
in coronary artery disease. Journal of
Psychiatric Research. 2019;:76-88
[60] Shah A, Chen C, Campanella C,
Kasher N, Evans S, Reiff C, et al. Brain
correlates of stress-induced peripheral
vasoconstriction in patients
with cardiovascular disease.
Psychophysiology. 2019;(2):e13291
[61] Nadir MA, Witham MD,
Szwejkowski BR, Struthers AD. Metaanalysis of B-type natriuretic
peptide's ability to identify stress
induced myocardial ischemia. The
American Journal of Cardiology.
2011;(5):662-667
[62] Akinboboye O, Krantz DS, Kop WJ,
Schwartz SD, Levine J, Del Negro A,
et al. Comparison of mental stressinduced myocardial ischemia in coronary
artery disease patients with versus
without left ventricular dysfunction.
The American Journal of Cardiology.
2005;(3):322-326
[63] Hammadah M, Alkhoder A,
Al Mheid I, Wilmot K, Isakadze N,
Abdulhadi N, et al. Hemodynamic,
catecholamine, vasomotor and vascular
responses: Determinants of myocardial
ischemia during mental stress.
International Journal of Cardiology.
2017;:47-53
[64] Broadley AJ, Korszun A, Abdelaal E,
Moskvina V, Jones CJ, Nash GB, et al.
Inhibition of cortisol production with
metyrapone prevents mental stressinduced endothelial dysfunction and
baroreflex impairment. Journal of
the American College of Cardiology.
2005;(2):344-350
[65] Seldenrijk A, Hamer M, Lahiri A,
Penninx BW, Steptoe A. Psychological
distress, cortisol stress response and
subclinical coronary calcification.
Psychoneuroendocrinology.
2012;(1):48-55
[66] Bozzini S, Gambelli P, Boiocchi C,
Schirinzi S, Falcone R, Buzzi P, et al.
Coronary artery disease and depression:
Possible role of brain-derived
neurotrophic factor and serotonin
transporter gene polymorphisms.
International Journal of Molecular
Medicine. 2009;(6):813-818
[67] Busselman RE, Hamer SA. Chagas
disease ecology in the united states:
Recent advances in understanding
trypanosoma cruzi transmission among
triatomines, wildlife, and domestic
animals and a quantitative synthesis of
vector-host interactions. Annual Review
of Animal Biosciences. 2022;:325-348
[68] Chagas disease in Latin America:
An epidemiological update based on
2010 estimates. Weekly Epidemiological
Record. 2015;(6):33-43
[69] Rassi A Jr, Rassi A, Marin-Neto JA.
Chagas disease. Lancet. 2010;(9723):
1388-1402
[70] Gascon J, Bern C, Pinazo MJ. Chagas
disease in Spain, the United States and
Stress-Induced Cardiomyopathy
DOI: http://dx.doi.org/10.5772/.105584
other non-endemic countries. Acta
315
https://t.me/medicina_free
Tropica. 2010;(1-2):22-27
[71]
Abras A, Ballart C, FernandezArevalo A, Pinazo MJ, Gascon J,
Munoz C, et al. Worldwide control and
management of chagas disease in
a new era of globalization: A close
look at congenital trypanosoma cruzi
infection. Clinical Microbiology Reviews.
2022;(2):e0015221
[72]
Ramirez JC, Acevedo GR,
Torres C, Parrado R, De La Barra A,
Villarroel S, et al. Genetic polymorphism
of Trypanosoma cruzi bloodstream
populations in adult chronic
indeterminate Chagas disease patients
from the E1224 clinical trial. The Journal
of Antimicrobial Chemotherapy.
2022;(3):578-584
[73]
Kun H, Moore A, Mascola L,
Steurer F, Lawrence G, Kubak B, et al.
Chagas disease in transplant recipients
investigation T. Transmission
of Trypanosoma cruzi by heart
transplantation. Clinical Infectious
Diseases. 2009;(11):1534-1540
[74]
Prata A. Clinical and epidemiological
aspects of Chagas disease. The Lancet
Infectious Diseases. 2001;(2):92-100
[75]
Duthie MS, Kahn M, Zakayan A,
White M, Kahn SJ. Parasite-induced
chronic inflammation is not exacerbated
by immunotherapy before or during
Trypanosoma cruzi Infection.
Clinical and Vaccine Immunology.
2007;(8):1005-1012
[76]
De Bona E, Lidani KCF, Bavia L,
Omidian Z, Gremski LH, Sandri TL,
et al. Autoimmunity in chronic chagas
disease: A road of multiple pathways
to cardiomyopathy? Frontiers in
Immunology. 2018;:1842
[77]
Daltro RT, Leony LM, Freitas NEM,
Silva AAO, Santos EF, Del-Rei RP, et al.
Cross-reactivity using chimeric
Trypanosoma cruzi antigens: Diagnostic
performance in settings where chagas
disease and American cutaneous or
visceral leishmaniasis are coendemic.
Journal of Clinical Microbiology.
2019;(8)
[78] Ferreira CP, Cariste LM, Santos
Virgilio FD, Moraschi BF, Monteiro CB,
Vieira Machado AM, et al. LFA-1 mediates
cytotoxicity and tissue migration of
specific CD8(+) T cells after heterologous
prime-boost vaccination against
trypanosoma cruzi infection. Frontiers in
Immunology. 2017;:1291
[79] Laucella SA, Mazliah DP,
Bertocchi G, Alvarez MG, Cooley G,
Viotti R, et al. Changes in Trypanosoma
cruzi-specific immune responses
after treatment: Surrogate markers of
treatment efficacy. Clinical Infectious
Diseases. 2009;(11):1675-1684
[80] Wen JJ, Yachelini PC, Sembaj A,
Manzur RE, Garg NJ. Increased oxidative
stress is correlated with mitochondrial
dysfunction in chagasic patients.
Free Radical Biology & Medicine.
2006;(2):270-276
[81] Cuervo H, Guerrero NA, Carbajosa S,
Beschin A, De Baetselier P, Girones N,
et al. Myeloid-derived suppressor cells
infiltrate the heart in acute Trypanosoma
cruzi infection. Journal of Immunology.
2011;(5):2656-2665
[82] Bonney KM, Luthringer DJ, Kim SA,
Garg NJ, Engman DM. Pathology and
pathogenesis of chagas heart disease.
Annual Review of Pathology.
2019;:421-447
[83] Batista CM, Kessler RL,
Eger I, Soares MJ. Trypanosoma cruzi
Intracellular amastigotes isolated
by nitrogen decompression are
capable of endocytosis and Cargo
Novel Pathogenesis and Treatments for Cardiovascular Disease
316
https://t.me/medicina_free
storage in reservosomes. PLoS One.
2015;(6):e0130165
[84] Wen JJ, Garg N. Oxidative
modification of mitochondrial
respiratory complexes in response to the
stress of Trypanosoma cruzi infection.
Free Radical Biology & Medicine.
2004;(12):2072-2081
[85] Wen JJ, Bhatia V, Popov VL, Garg NJ.
Phenyl-alpha-tert-butyl nitrone reverses
mitochondrial decay in acute Chagas'
disease. The American Journal of
Pathology. 2006;(6):1953-1964
[86] Wen JJ, Garg NJ. Mitochondrial
generation of reactive oxygen species is
enhanced at the Q(o) site of the complex
III in the myocardium of Trypanosoma
cruzi-infected mice: Beneficial effects of
an antioxidant. Journal of Bioenergetics
and Biomembranes. 2008;(6):587-598
[87] Gupta S, Bhatia V, Wen JJ, Wu Y,
Huang MH, Garg NJ. Trypanosoma
cruzi infection disturbs mitochondrial
membrane potential and ROS
production rate in cardiomyocytes.
Free Radical Biology & Medicine.
2009;(10):1414-1421
[88] Wen JJ, Garg NJ. Mitochondrial
complex III defects contribute to
inefficient respiration and ATP synthesis
in the myocardium of Trypanosoma
cruzi-infected mice. Antioxidants &
Redox Signaling. 2010;(1):27-37
[89] Wen JJ, Gupta S, Guan Z, Dhiman M,
Condon D, Lui C, et al. Phenyl-alphatert-butyl-nitrone and benzonidazole
treatment controlled the mitochondrial
oxidative stress and evolution of
cardiomyopathy in chronic chagasic
rats. Journal of the American College of
Cardiology. 2010;(22):2499-2508
element pathway by mitochondrial
development of cardiomyopathy and
left ventricular dysfunction in chagas
disease. Antioxidants & Redox Signaling.
2017;(9):550-566
[91] Wen JJ, Garg NJ. Manganese
superoxide dismutase deficiency
exacerbates the mitochondrial ROS
production and oxidative damage in
Chagas disease. PLoS Neglected Tropical
Diseases. 2018;(7):e0006687
[92] Wen JJ, Yin YW, Garg NJ. PARP1
depletion improves mitochondrial
and heart function in Chagas disease:
Effects on POLG dependent mtDNA
maintenance. PLoS Pathogens.
2018;(5):e1007065
[93] Wen JJ, Vyatkina G,
Garg N. Oxidative damage during
chagasic cardiomyopathy development:
Role of mitochondrial oxidant release
and inefficient antioxidant defense.
Free Radical Biology & Medicine.
2004;(11):1821-1833
[94] Wen JJ, Dhiman M, Whorton EB,
Garg NJ. Tissue-specific oxidative
imbalance and mitochondrial
dysfunction during Trypanosoma
cruzi infection in mice. Microbes and
Infection. 2008;(10-11):1201-1209
[95] Wen JJ, Garg NJ. Proteome
expression and carbonylation changes
during Trypanosoma cruzi infection
and Chagas disease in rats. Molecular &
Cellular Proteomics. 2012;(4):010918
[96] Wen JJ, Nagajyothi F, Machado FS,
Weiss LM, Scherer PE, Tanowitz HB,
et al. Markers of oxidative stress in
adipose tissue during Trypanosoma
cruzi infection. Parasitology Research.
2014;(9):3159-3165
[90] Wen JJ, Porter C, Garg NJ. Inhibition
of NFE2L2-antioxidant response
[97] Wen JJ, Wan X, Thacker J,
Garg NJ. Chemotherapeutic efficacy
Stress-Induced Cardiomyopathy
DOI: http://dx.doi.org/10.5772/.105584
of phosphodiesterase inhibitors in
317
https://t.me/medicina_free
chagasic cardiomyopathy. JACC Basic
Translational Science. 2016;(4):235-250
[98]
Tanowitz HBWJ-J, Machado
F-S, Desruisseaux M-S, Robello C,
Garg NJ. Trypanosoma cruzi and Chagas
disease: Innate immunity, ROS, and
cardiovascular system. In: Vascular
Responses to Pathogens, Waltham. 2016
[99]
Organization WH. Deaths by cause,
age, sex, by country and by region,
2000-2016. 2018. Geneva: Global Health
Estimates 2016; 2018. [Accessed: 23
October 2019]
[100]
Collaborators GBDRF. Global,
regional, and national comparative
risk assessment of 84 behavioural,
environmental and occupational, and
metabolic risks or clusters of risks for
195 countries and territories, 1990-2017:
A systematic analysis for the Global
Burden of Disease Study 2017. Lancet.
2018;(10159):1923-1994
[101]
Services. USDoHaH. How tobacco
smoke causes disease: The biology and
behavioral basis for smoking-attributable
disease: A report of the Surgeon General.
Atlanta (GA): Centers for Disease
Control and Prevention; 2010 [Accessed:
2010]
[102]
Csordas A, Bernhard D. The biology
behind the atherothrombotic effects
of cigarette smoke. Nature Reviews.
Cardiology. 2013;(4):219-230
[103]
Arteriosclerosis, Thrombosis, and
Vascular Biology 2015;35(1):1
[104]
Services. USDoHaH. The Health
Consequences of Involuntary Exposure
to Tobacco Smoke: A Report of the
Surgeon General. Atlanta (GA): Centers
for Disease Control and Prevention; 2006
[105]
Otsuka R, Watanabe H, Hirata K,
Tokai K, Muro T, Yoshiyama M, et al.
Acute effects of passive smoking on the
coronary circulation in healthy young
adults. Journal of the American Medical
Association. 2001;(4):436-441
[106] Hackshaw A, Morris JK, Boniface S,
Tang JL, Milenkovic D. Low cigarette
consumption and risk of coronary heart
disease and stroke: Meta-analysis of 141
cohort studies in 55 study reports. BMJ.
2018;:j5855
[107] Prevention. Cardiovascular
Disease: A Report of the Surgeon
General. Atlanta (GA): United States
Department of Health and Human
Services; 1983
[108] McInnes GT. Hypertension
and coronary artery disease: Cause
and effect. Journal of Hypertension.
Supplement. 1995;(2):S49-S56
[109] Rossi M, Negri E, La Vecchia C,
Campos H. Smoking habits and the
risk of non-fatal acute myocardial
infarction in Costa Rica. European
Journal of Cardiovascular Prevention and
Rehabilitation. 2011;(3):467-474
[110] Olasky SJ, Levy D, Moran A. Second
hand smoke and cardiovascular disease
in Low and Middle Income Countries:
A case for action. Global Heart.
2012;(2):151-60 e5
[111] Prevention CfDCa. The Health
Consequences of Smoking: 50 years
of Progress. A Report of the Surgeon
General. Atlanta (GA): United States
Department of Health and Human
Services; 2014
[112] Lv X, Sun J, Bi Y, Xu M, Lu J,
Zhao L, et al. Risk of all-cause mortality
and cardiovascular disease associated
with secondhand smoke exposure: A
systematic review and meta-analysis.
International Journal of Cardiology.
2015;:106-115
Novel Pathogenesis and Treatments for Cardiovascular Disease
318
https://t.me/medicina_free
[113] Metsios GS, Flouris AD, Angioi M,
Koutedakis Y. Passive smoking and the
development of cardiovascular disease in
children: A systematic review. Cardiology
Research and Practice. 2010;
[114] West HW, Juonala M, Gall SL,
Kahonen M, Laitinen T, Taittonen L,
et al. Exposure to parental smoking in
childhood is associated with increased
risk of carotid atherosclerotic plaque
in adulthood: The Cardiovascular Risk
in Young Finns Study. Circulation.
2015;(14):1239-1246
[115] Weitkunat R, Lee PN, Baker G,
Sponsiello-Wang Z, Gonzalez-Zuloeta
Ladd AM, Ludicke F. A novel approach
to assess the population health impact
of introducing a Modified Risk Tobacco
Product. Regulatory Toxicology and
Pharmacology. 2015;(1):87-93
[116] Max WB, Sung HY, Lightwood J,
Wang Y, Yao T. Modelling the impact
of a new tobacco product: Review of
Philip Morris International's Population
Health Impact Model as applied to the
IQOS heated tobacco product. Tobacco
Control. 2018;(Suppl. 1):s82-ss6
[117] Organization WH. Electronic
nicotine delivery systems and electronic
non-nicotine delivery systems (ENDS/
ENNDS) (report to the seventh session
of the Conference of the Parties to
the WHO Framework Convention on
Tobacco Control (Delhi, India, 7-12
November 2016)) Geneva: World Health
Organization; 2016 (FCTC/COP/7/11;
https://wwwwhoint/fctc/cop/cop7/
Documentation-Main-documents/en/,
accessed 25 October 2019). 2019
[118] Alzahrani T, Pena I,
Temesgen N, Glantz SA. Association
between electronic cigarette use and
myocardial infarction. American
Journal of Preventive Medicine.
2018;(4):455-461
[119] Bhatta DN, Glantz SA. Electronic
cigarette use and myocardial infarction
among adults in the US Population
Assessment of Tobacco and Health.
Journal of the American Heart
Association. 2019;(12):e012317
[120] Qasim H, Karim ZA, Rivera JO,
Khasawneh FT, Alshbool FZ. Impact of
electronic cigarettes on the cardiovascular
system. Journal of the American Heart
Association. 2017;(9):1-14
[121] Ikonomidis I, Vlastos D, Kourea K,
Kostelli G, Varoudi M, Pavlidis G, et al.
Electronic cigarette smoking increases
arterial stiffness and oxidative stress to a
lesser extent than a single conventional
cigarette: An Acute and Chronic Study.
Circulation. 2018;(3):303-306
[122] Biondi-Zoccai G, Sciarretta S,
Bullen C, Nocella C, Violi F, Loffredo L,
et al. Acute effects of heat-not-burn,
electronic vaping, and traditional tobacco
combustion cigarettes: The Sapienza
University of Rome-Vascular Assessment
of Proatherosclerotic Effects of Smoking
( SUR - VAPES ) 2 Randomized
Trial. Journal of the American Heart
Association. 2019;(6):e010455
[123] Wang JB, Olgin JE, Nah G,
Vittinghoff E, Cataldo JK, Pletcher MJ,
et al. Cigarette and e-cigarette dual use
and risk of cardiopulmonary symptoms
in the Health eHeart Study. PLoS One.
2018;(7):e0198681
[124] National Institutes of Health
NCIaGWHO. United States National
Cancer Institute and World Health
Organization. The economics of
tobacco and tobacco control (National
Cancer Institute Tobacco Control
Monograph No. 21; NIH Publication No.
16-CA-8029A). Bethesda, MD: United
States Department of Health and Human
Services, National Institutes of Health,
National Cancer Institute; and Geneva:
Stress-Induced Cardiomyopathy
DOI: http://dx.doi.org/10.5772/.105584
World Health Organization; 2016.
319
https://t.me/medicina_free
Available from: https://cancercontrol.
cancer.gov/brp/tcrb/monographs/21/
[Accessed: 25 October 2019]
[125]
Lippi G, Favaloro EJ, Meschi T,
Mattiuzzi C, Borghi L, Cervellin G.
E-cigarettes and cardiovascular risk:
Beyond science and mysticism. Seminars
in Thrombosis and Hemostasis.
2014;(1):60-65
[126]
Grana R, Benowitz N,
Glantz SA. E-cigarettes: A
scientific review. Circulation.
2014;(19):1972-1986
[127]
Buchanan ND,
Grimmer JA, Tanwar V, Schwieterman N,
Mohler PJ, Wold LE. Cardiovascular
risk of electronic cigarettes: A
review of preclinical and clinical
studies. Cardiovascular Research.
2020;(1):40-50
[128]
Majid S, Keith RJ, Fetterman JL,
Weisbrod RM, Nystoriak J, Wilson T, et
al. Lipid profiles in users of combustible
and electronic cigarettes. Vascular
Medicine. 2021;(5):483-488
[129]
Moheimani RS, Bhetraratana M,
Yin F, Peters KM, Gornbein J, Araujo JA,
et al. Increased cardiac sympathetic
activity and oxidative stress in habitual
electronic cigarette users: Implications
for cardiovascular risk. JAMA
Cardiology. 2017;(3):278-284
[130]
El-Mahdy MA, Ewees MG, Eid MS,
Mahgoup EM, Khaleel SA, Zweier JL.
Electronic cigarette exposure causes
vascular endothelial dysfunction due
to NADPH oxidase activation and
eNOS uncoupling. American Journal
of Physiology. Heart and Circulatory
Physiology. 2022;(4):H549-HH67
[131]
Li J, Huynh L, Cornwell WD,
Tang MS, Simborio H, Huang J,
et al. Electronic cigarettes induce
mitochondrial DNA Damage and Trigger
TLR9 (Toll-Like Receptor 9)-mediated
atherosclerosis. Arteriosclerosis,
Thrombosis, and Vascular Biology.
2021;(2):839-853
[132] Espinoza-Derout J, Shao XM,
Lao CJ, Hasan KM, Rivera JC,
Jordan MC, et al. Electronic cigarette use
and the risk of cardiovascular diseases.
Frontier in Cardiovascular Medicine.
2022;:879726
[133] Lee WH, Ong SG, Zhou Y,
Tian L, Bae HR, Baker N, et al. Modeling
cardiovascular risks of E-Cigarettes
with human-induced pluripotent stem
cell-derived endothelial cells. Journal
of the American College of Cardiology.
2019;(21):2722-2737
[134] Williams FN, Herndon DN,
Suman OE, Lee JO, Norbury WB,
Branski LK, et al. Changes in cardiac
physiology after severe burn injury.
Journal of Burn Care & Research.
2011;(2):269-274
[135] Hoesel LM, Niederbichler AD,
Schaefer J, Ipaktchi KR, Gao H,
Rittirsch D, et al. C5a-blockade improves
burn-induced cardiac dysfunction.
Journal of Immunology. 2007;(12):
7902-7910
[136] Jeschke MG, Chinkes DL,
Finnerty CC, Kulp G, Suman OE,
Norbury WB, et al. Pathophysiologic
response to severe burn injury. Annals of
Surgery. 2008;(3):387-401
[137] Davies JL. The endocrine response.
In: 1982, editor. Physiological Responses
to Burning Injury. Orlando: Academic
Press
[138] Crum RL, Dominic W,
Hansbrough JF, Shackford SR, Brown MR.
Cardiovascular and neurohumoral
Novel Pathogenesis and Treatments for Cardiovascular Disease
320
https://t.me/medicina_free
responses following burn injury. Archives
of Surgery. 1990;(8):1065-1069
[139] Mak GZ, Hardy AR, Meyer RA,
Kagan RJ. Reversible cardiomyopathy
after severe burn injury. Journal of Burn
Care & Research. 2006;(4):482-486
[140] Zabala LM, Parray T. Cardiac arrest
because of unrecognized delayed dilated
cardiomyopathy in a child with severe
burn injury. Paediatric Anaesthesia.
2006;(3):358-359
[141] Chen TJ, Shen BH, Yeh FL, Lin JT,
Ma H, Huang CH, et al. Delayed dilated
cardiomyopathy for major burn injuries.
Burns. 2003;(4):343-348
[142] Abu-Sittah GS, Sarhane KA,
Dibo SA, Ibrahim A. Cardiovascular
dysfunction in burns: Review of the
literature. Annals of Burns and Fire
Disasters. 2012;(1):26-37
[143] Adams HR, Baxter CR, Izenberg SD.
Decreased contractility and compliance
of the left ventricle as complications of
thermal trauma. American Heart Journal.
1984;(6):1477-1487
[144] Adams HR, Baxter CR, Parker JL.
Contractile function of heart muscle
from burned guinea pigs. Circulatory
Shock. 1982;(1):63-73
[145] Suzuki KOT, Takasu N, et al.
Changes in left ventricular preload
and contractility following severe
bunrs in the dog. Heart and Vessels.
1986;:147-153
[146] Martyn JWR, Burke IF. Right
ventricular function and pulmonary
hemodynamics during dopamine
infusion in burned patients. Chest.
1986;:357-360
[147] Ganrot KJS, Rothman U.
Transcapillary passage of plasma proteins
in experimental burns. Acta Physiologica
Scandinavica. 1974;:497-501
[148] Reiss E, Pearson E, Artz CP. The
metabolic response to burns. The
Journal of Clinical Investigation.
1956;(1):62-77
[149] Wilmore DW, Aulick LH. Metabolic
changes in burned patients. The
Surgical Clinics of North America.
1978;(6):1173-1187
[150] Wolf SE, Rose JK, Desai MH,
Mileski JP, Barrow RE, Herndon DN.
Mortality determinants in massive
pediatric burns. An analysis of 103
children with > or = 80% TBSA burns
(> or = 70% full-thickness). Annals of
Surgery. 1997;(5):554-565
[151] Wilmore DW, Long JM, Mason
AD Jr, Skreen RW, Pruitt BA Jr.
Catecholamines: Mediator of the
hypermetabolic response to thermal
injury. Annals of Surgery.
1974;(4):653-669
[152] Goodall M, Stone C, Haynes BW
Jr. Urinary output of adrenaline and
noradrenaline in severe thermal burns.
Annals of Surgery. 1957;(4):479-487
[153] Mohammadi F, Ramachandran J,
Woodman R, Muller K, John L,
Chen J, et al. Impact of cardiac
dysfunction on morbidity and mortality
in liver transplant candidates. Clinical
Transplantation. 2022:e14682
[154] Jeschke MG, Gauglitz GG,
Kulp GA, Finnerty CC, Williams FN,
Kraft R, et al. Long-term persistance
of the pathophysiologic response
to severe burn injury. PLoS One.
2011;(7):e21245
[155] Raab W. Key position of
catecholamines in functional and
degenerative cardiovascular pathology.
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