Добавил:
Sekretar
kiopkiopkiop18@yandex.ru
t.me/Prokururor I Вовсе не секретарь, но почту проверяю
Опубликованный материал нарушает ваши авторские права? Сообщите нам.
Вуз:
Предмет:
Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_4455_Библиотеки_им_академика_М_И_Перельмана
.pdf
170 Disorders of the Auditory System
https://t.me/medicina_free
B
Figure 4–10. continued
Audiologic Recommendations
and Management
Due to the bacterial meningitis, which
was diagnosed 1 week following the
accident (see discussion in the following
medical section), it was recommended
that the patient be closely monitored
audiologically. Both the patient and his
parents were counseled to alert audiology
immediately should any changes in hearing be noted. It was also recommended
that a trial fitting with a contralateral
routing of signal (CROS) hearing aid be
considered.
Medical Recommendations
and Management
The patient remained stable until approximately 1 week following the accident. At
which time, the patient’s recovery became
complicated by a cerebrospinal fluid leak
and bacterial meningitis. Due to these
complications, the patient was taken to
the operating room by otolaryngology
and neurosurgery for a left total facial
nerve decompression and left transmastoid and transcranial repair of the temporal bone. In addition, repair of a dural
defect and placement of a lumbar drain
was required. These procedures were
completed without complication.
The patient was eventually discharged from the hospital. Prior to his
discharge, his parents were counseled
regarding the fact that it was unlikely
their son would regain any hearing function in his left ear and left facial nerve
function would be monitored for possible
recovery of or improvement. Postoperatively, the patient has demonstrated no
significant audiologic changes. To date,
the patient continues to have left-sided
facial paralysis and no residual hearing in
the left ear. Hearing sensitivity and facial
nerve function for the right side continues
to be normal.

https://t.me/medicina_free
171
C
Figure 4–10. continued

172 Disorders of the Auditory System
https://t.me/medicina_free
otheR disoRdeRs
affecting the outeR
and
middle eaRs
External Otitis
External otitis is an infection of the ear
canal and/or the pinna that is often
noticed in the summer months and is
commonly caused by swimming in water
with high counts of bacteria. It is typically caused by a bacterial invasion into
the external ear canal. The most common
bacteria is Pseudomonas. External otitis can
also be related to fungal infections, but
these are relatively rare. In viewing the
ear canal, mucopurulent matter is usually
observed along with redness and swelling of the tissue involved. The patient will
complain of severe discomfort, especially
when the pinna is moved. Audiologically,
there is seldom much of an effect on hearing unless swelling closes the ear canal.
Treatment of the patient includes cleaning
of the affected area and topical application
of antibacterial agents. These antibacterial agents are sometimes combined with
corticosteroids, which may provide better
treatment than antibacterial applications
alone (Castillo & Roland, 2007).
block the ear canal. The presence of exostoses in the ear canal may make it difficult to visualize the tympanic membrane.
If large enough to close off the ear canal,
these growths can cause conductive hearing loss. The ear canal, especially if cerumen is present, can be blocked by pushing
cerumen against the exostoses when ear
inserts are used. Therefore, care must be
exercised when using ear insert receivers
with patients with exostoses. Management of these patients usually involves
surgical removal of the exostoses (Zender,
Marzo, & Leonetti, 2007).
Osteomas are also bony growths and
are often confused with exostoses. These,
however, are usually singular in number
and are located more laterally in the canal.
They are a true neoplasm that is a mix of
bone and fibrous tissue, and they tend
to occur more often in children than in
adults. Audiologically, the same concerns
exist for osteomas as those mentioned for
exostoses. Osteomas occasionally can be
removed in the otolaryngologist’s office,
but often a brief general anesthetic is
required (Zender et al., 2007).
Tympanic Membrane
Perforation
Exostoses and Osteomas
Exostoses are bony growths occurring in
the medial (bony) portion of the ear canal
(see Appendix 4H). These multiple bony
growths seem to be related to cold water
exposure and are often seen in swimmers.
This is often a bilateral condition. Exostoses become bothersome only when they
trap debris in the ear canal or become so
large that they impinge on the eardrum or
Castillo and Roland (2007) provide a
review of tympanic membrane perforations from which we draw information.
These perforations generally are related
to trauma (blasts, penetrating injuries,
head trauma, etc.) or infections, such as
otitis media. Most tympanic membrane
perforations will heal spontaneously
unless they are large or are related to
ongoing or recurring infections. They can
occur in either the pars flaccida or pars
tensa. However, if they occur in the pars

4. Outer and Middle Ear Disorders 173
https://t.me/medicina_free
flaccida, concern about cholesteatomas
would be in order.
Long-standing perforations also herald concern over possible cholesteatomas.
Small perforations generally do not yield
hearing loss; however, large tympanic
membrane perforations can result in up
to 50 dB of hearing loss, although in most
cases, the hearing loss is much less severe.
Increased volume measures during tympanometric testing are indications of tympanic membrane perforations unless the
patient has a PE tube in place. Careful otoscopic exams should always be completed
to determine if a tympanic membrane
perforation is present. Care must also be
taken during audiologic and vestibular
(caloric) exams whenever a perforation
is noted so that the tympanic membrane
perforation is not further disturbed or
contaminated. Treatment for tympanic
membrane perforations that do not spontaneously heal is otologic surgical repair
(see Appendix 4I for an example of a tympanic membrane perforation).
Tympanosclerosis
Tympanosclerosis is a condition in which
white calcified plaques of connective tissue occur at and around the circumference
of the tympanic membrane and/or the
head of the malleus (Castillo & Roland,
2007) (see Appendix 4J for an example of
tympanosclerosis). When these plaques
are only on the tympanic membrane,
the condition is called myringosclerosis, which is more common than tympanosclerosis. Both conditions are often
associated with chronic otitis media and
inflammatory processes of the middle ear.
Myringosclerosis seldom yields a measurable hearing loss; however, tympanoscle-
rosis can result in a hearing loss especially
if it involves the ossicular chain. In cases
with measurable hearing loss, the hearing
loss will be a conductive hearing loss and
the tympanograms will typically reveal
slightly reduced compliance (Castillo &
Roland, 2007).
Ossicular Chain Discontinuity
Multiple terms have been used to describe
conditions affecting the ossicular chain
where the normal articulation of the middle ear bones is absent or disrupted (e.g.,
ossicular chain discontinuity, ossicular
chain disarticulation, ossicular chain dislocation). Although this topic has been
discussed to some extent in the section of
this chapter that deals with temporal bone
trauma, it is important to highlight that
there are other etiologies that can result in
this middle ear disorder. Ossicular chain
discontinuity can be a result of various
types of congenital abnormalities, necrosis (secondary to chronic infections), and
surgical interventions, as well as trauma
(Castillo & Roland, 2007). The hearing
loss noted in cases of ossicular chain discontinuity typically is a conductive hearing loss; however, in some cases of head
injury, the associated trauma may cause
a subluxation of the ossicular chain with
injury to the cochlea, which results in a
mixed hearing loss (Castillo & Roland,
2007). Audiology plays a key role in the
diagnosis of ossicular chain discontinuity
in that near to maximum conductive loss
is often observed in patients with ossicular chain discontinuity. In addition, their
tympanograms show hypercompliance,
but no crossed acoustic reflexes even if
hearing sensitivity is good in the acoustically stimulated ear. If the ossicular chain

174 Disorders of the Auditory System
https://t.me/medicina_free
is subluxed or fibrous tissue has formed to
keep the chain somewhat intact, the hearing may be relatively good with tympanograms showing either normal or reduced
compliance, and acoustic reflexes may
be measured in some cases; however, if
reflexes are noted, they are typically elevated (Jerger & Jerger, 1981). Treatment
of ossicular chain discontinuity is otologic surgery to repair the dislocation or
disarticulation of the ossicular chain and
to resolve the conductive hearing loss
that has resulted from the disarticulated
ossicular chain. This surgical procedure
involved is called ossiculoplasty (i.e.,
repair of the ossicular chain) and is quite
successful with closure of the air-bone gap
to within 10 dB slightly less than 75% of
the time (Castillo & Roland, 2007).
summaRy
This chapter provided a brief overview of
select conditions that affect the external
and/or middle ears. Diseases of these portions of the auditory system are diverse in
etiology and can have profound effects on
auditory function. Appropriate management of these diseases requires a combination of thorough history and physical
examination, careful audiologic testing,
and often appropriate diagnostic imaging.
A clear understanding of these conditions
and the implementation of appropriate
management procedures (whether audiological or medical) can lead to improved
auditory function and quality of life for
those patients who present with external
and/or middle ear disorders.
Acknowledgments. The authors grate-
fully acknowledge the contributions of
Matthew Bush, MD, PhD, Associate Professor, Department of OtolaryngologyHead and Neck Surgery, University of
Kentucky, to this chapter.
RefeRences
Abramson, M., & Huang, C. C. (1977). Local-
ization of collagenase in human middle ear
cholesteatoma. Laryngoscope, 87(5 Pt. 1),
771–791.
Aithal, V., Aithal, S., & Pulotu, L. (1995). Otitis
media with effusion in children: An audiological case series study. Papua and New
Guinea Medical Journal, 38(2), 79–94.
Amar, M. S., Wishahi, H. F., & Zakhary, M.
M. (1996). Clinical and biochemical studies
of bone destruction in cholesteatoma. Jour-
nal of Laryngology and Otology, 110(6), 534–
539.
Bennett, M., Warren, F., & Haynes, D. (2006).
Indications and technique in mastoidectomy. Otolaryngologic Clinics of North Amer-
ica, 39(6), 1095–1113.
Bluestone, C. (1998). Anatomy and physiology
of the Eustachian tube. In C. Cummings, J.
M. Fredrickson, L. A. Harker, C. J. Krause,
M. A. Richardson, & D. E. Schuller (Eds.),
Otolaryngology: Head and neck surgery (Vol. 3,
pp. 3003–3025). St. Louis, MO: Mosby.
Bluestone, C. D. (2004). Studies in otitis media:
Children’s Hospital of Pittsburgh — University of Pittsburgh progress report — 2004.
Laryngoscope, 114(11 Pt. 3, Suppl. 105), 1–26.
Bluestone, C. D., & Klein, J. O. (2003). Otitis
media and Eustachian tube dysfunction. In
C. D. Bluestone, S. E. Stool, C. M. Alper, E.
M. Arjmand, C. I. Casselbrant, J. E. Dohar,
& R. F. Yellon (Eds.), Pediatric otolaryngology
(4th ed., Vol. 1, pp. 474–685). New York, NY:
W. B. Saunders.
Bluestone, C. D., Stephenson, J. S., & Martin,
L. M. (1992). Ten-year review of otitis media
pathogens. Pediatric Infectious Disease Jour-
nal, 11(Suppl. 8), S7–S11.

4. Outer and Middle Ear Disorders 175
https://t.me/medicina_free
Borsanyi, S. J. (1962). Glomus jugulare tumors.
Laryngoscope, 72, 1336–1345.
Brodie, H. A., & Thompson, T. C. (1997). Man-
agement of complications from 820 temporal bone fractures. American Journal of Otol-
ogy, 18(2), 188–197.
Brown, J. S. (1985). Glomus jugulare tumors
revisited: A ten-year statistical follow-up of
231 cases. Laryngoscope, 95(3), 284–288.
Canalis, R. F. (1990). Valsalva’s contribution to
otology. American Journal of Otolaryngology,
11(6), 420–427.
Canalis, R. F., & Lambert, P. R. (2000). Chronic
otitis media and cholesteatoma. In R. F.
Canalis & P. R. Lambert (Eds.), The ear:
Comprehensive otology (pp. 409–431). Philadelphia, PA: Lippincott Williams & Wilkins.
Carhart, R. (1950). Clinical application of bone
conduction audiometry. Archives of Otolar-
yngology, 51(6), 798–808.
Carrasco, V., & Rosenman, J. (1993). Radiation
therapy of glomus jugulare tumors. Laryn-
goscope, 103(11 Pt. 2, Suppl. 60), 23–27.
Castillo, M. P., & Roland, P. S. (2007). Disorders
of the auditory system. In R. J. Roeser, M.
Valente, & H. Hosford-Dunn (Eds.), Audi-
ology: Diagnosis (2nd ed., pp. 77–99). New
York, NY: Thieme.
Cawthorne, T. (1955). Otosclerosis. Journal of
Laryngology and Otology, 69(7), 437–456.
da Costa, C. F., & Polanski, J. F. (2015). Wegener
granulomatosis: Otologic manifestation as
first symptom. International Archives of Oto-
rhinolaryngology, 19(3), 266–268.
Dahiya, R., Keller, J. D., Litofsky, N. S., Ban-
key, P. E., Bonassar, L. J., & Megerian, C. A.
(1999). Temporal bone fractures: Otic capsule sparing versus otic capsule violating
clinical and radiographic considerations.
Journal of Trauma, 47(6), 1079–1083.
Danishyar, A., & Ashurst, J. V. (Updated 2019,
July 30). Acute otitis media. StatPearls [Inter-
net]. Treasure Island, FL: StatPearls Publishing. Retrieved from https://www.ncbi
.nlm.nih.gov/books/NBK470332/
Declau, F., Cremers, C., & Van de Heyning, P.
(1999). Diagnosis and management strategies in congenital atresia of the external
auditory canal. Study Group on Otological
Malformations and Hearing Impairment.
British Journal of Audiology, 33(5), 313–327.
De la Cruz, A., & Chandrasekhar, S. S. (1994).
Congenital malformation of the temporal bone. In D. E. Brackmann, S. Shelton, & M. A. Arriaga (Eds.), Otologic sur-
gery (pp.
Saunders.
Derkay, C. S., Bluestone, C. D., Thompson, A.
E., & Kardatske, D. (1989). Otitis media in
the pediatric intensive care unit: A prospective study. Otolaryngology-Head and Neck
Surgery, 100(4), 292–299.
Dhooge, I. J. M. (2003). Risk factors for the
development of otitis media. Current
Allergy and Asthma Reports, 3(4), 321–325.
Dobberpuhl, M. R., Maxwell, S., Feddock, J.,
St. Clair, W., & Bush, M. L. (2016).Treatment
outcomes for single modality management
of glomus jugulare tumors with stereotactic
radiosurgery. Otology & Neurotology, 37(9),
1406–1410.
Doherty, J. K., & Linthicum, F. H., Jr. (2004).
Spiral ligament and stria vascularis changes
in cochlear otosclerosis: Effect on hearing
level. Otology & Neurotology, 25(4), 457–464.
Erickson, D., Kudva, Y. C., Ebersold, M. J.,
Thompson, G. B., Grant, C. S., van Heerden,
J. A., & Young, W. F., Jr. (2001). Benign
paragangliomas: Clinical presentation and
treatment outcomes in 236 patients. Jour-
nal of Clinical Endocrinology and Metabolism,
86(11), 5210–5216.
Fayad, J. N., Keles, B., & Brackmann, D. E.
(2010). Jugular foramen tumors: Clinical
characteristics and treatment outcomes.
Otology & Neurotology, 31(2), 299–305.
Fowler, C. G., & Shanks, J. E. (2002). Tympa-
nometry. In J. Katz (Ed.), Handbook of clinical
audiology (5th ed., pp. 175–204), Philadelphia, PA: Lippincott Williams & Wilkins.
Gates, G. A., Avery, C. A., Prihoda, T. J., &
Cooper, J. C., Jr. (1987). Effectiveness of
adenoidectomy and tympanostomy tubes
in the treatment of chronic otitis media with
effusion. New England Journal of Medicine,
317(23), 1444–1451.
69–84). Philadelphia, PA: W. B.

176 Disorders of the Auditory System
https://t.me/medicina_free
Ghorayeb, B. Y., & Yeakley, J. W. (1992). Tempo-
ral bone fractures: Longitudinal or oblique?
The case for oblique temporal bone fractures. Laryngoscope, 102(2), 129–134.
Goldenberg, D., Karam, M., Danino, J., Flax-
Goldenberg, R., & Joachims, H. (1998). Temporal bone fracture following blunt trauma
caused by a flying fish. Journal of Laryngol-
ogy and Otology, 112(10), 959–961.
Goossens, H., Ferech, M., Vander Stichele,
R., & Elseviers, M. (2005). Outpatient antibiotic use in Europe and association with
resistance: A cross-national database study.
Lancet, 365(9459), 579–587.
Guild, S. R. (1941). A hitherto unrecognized
structure, the glomus jugularis, in man.
Anatomical Record, 79(Suppl. 2), 28.
Gulya, A. J. (1993). The glomus tumor and its
biology. Laryngoscope, 103(11 Pt. 2, Suppl.
60), 7–15.
Hannley, M. T. (1993). Audiologic character-
istics of the patient with otosclerosis. Oto-
laryngologic Clinics of North America, 26(3),
373–387.
Henderson, F. W., Collier, A. M., Sanyal, M. A.,
Watkins, J. M., Fairclough, D. L., Clyde, W.
A., Jr., & Denny, F. W. (1982). A longitudinal study of respiratory viruses and bacteria in the etiology of acute otitis media with
effusion. New England Journal of Medicine,
306(23), 1377–1383.
Herbella, F. A., Mudo, M., Delmonti, C., Braga,
F. M., & Del Grande, J. C. (2001). “Raccoon
eyes” (periorbital haematoma) as a sign of
skull base fracture. Injury, 32(10), 745–747.
Hobson, J. C., Roper, A. J., Andrew, R., Rothera,
M. P., Hill, P., & Green, K. M. (2010). Complications of bone-anchored hearing aid
implantation. Journal of Laryngology and
Otology, 124(2), 132–136.
Holmquist, J. (1969). Eustachian tube function
assessed with tympanometry. A new testing procedure in ears with intact tympanic
membrane. Acta Otolaryngologica, 68(6),
501–508.
Horn, K. L., & Hankinson, H. (1994). Tumors
of the jugular foramen. In R. K. Jackler &
D. E. Brackmann (Eds.), Neurotology (pp.
1059–1068). St. Louis, MO: Mosby.
House, J. W. (1997). Otosclerosis. In G. B.
Hughes & M. L. Pensak (Eds.), Clinical otol-
ogy (2nd ed., pp. 241–249). New York, NY:
Thieme.
Ivy, R. G. (1975). Tympanometric curves and
otosclerosis. Journal of Speech and Hearing
Research, 18(3), 554–558.
Jackson, C. G., Glasscock, M. E., III, & Harris, P.
E. (1982). Glomus tumors. Diagnosis, classification, and management of large lesions.
Archives of Otolaryngology, 108(7), 401–410.
Jacobson, J. T., & Mahoney, T. M. (1977).
Admittance tympanometry in otosclerotic
ears. Journal of the American Audiology Soci-
ety, 3(2), 91–98.
Jahrsdoerfer, R. A. (1978). Congenital atresia of
the ear. Laryngoscope, 88(9 Pt. 3, Suppl. 13),
1–48.
Jahrsdoerfer, R. A., Yeakley, J. W., Aguilar, E.
A., Cole, R. R., & Gray, L. C. (1992). Grading system for the selection of patients with
congenital aural atresia. American Journal of
Otology, 13(1), 6–12.
Jerger, S., & Jerger, J. (1981). Auditory disorders:
A manual for clinical evaluation. Boston, MA:
Little Brown.
Jones, L. L., Hassanien A., Cook, D. G., Brit-
ton, J., & Leonardi-Bee, J. (2012). Parental
smoking and the risk of middle ear disease
in children: A systematic review and metaanalysis. Archives of Pediatric and Adolescent
Medicine, 166(1), 18–27.
Karas, D. E., & Kwartler, J. A. (1993). Glomus
tumors: A fifty-year historical perspective.
American Journal of Otology, 14(5), 495–500.
Kaur, R., Morris, M., & Pichichero, M. E.
(2017). Epidemiology of acute otitis media
in the postpneumococcal conjugate vaccine
era. Pediatrics, 140(3), e20170181. https://
doi.org/10.1542/peds.2017-0181
Kessel, J. (1878). Uber das Mobilisieren des
Steigbugels durch Ausschneiden des
Trommelfelles, Hammers und Ambosses
bei Undurchgangigkeit der tube. Archiv fur
Ohrenheilkunde, 13, 69–72.
Lambert, P. R. (1998). Congenital aural atresia.
In B. J. Bailey (Ed.), Head and neck surgery;
Otolaryngology (2nd ed., pp. 1997–2009).
Philadelphia, PA: Lippincott-Raven.

4. Outer and Middle Ear Disorders 177
https://t.me/medicina_free
Lempert, J. (1938). Improvement of hearing
in cases of otosclerosis: A new, one stage
surgical technic. Archives of Otolaryngology-
Head and Neck Surgery, 28(1), 42–97.
Leo, G., Piacentini, E., Incorvaia, C., & Con-
sonni, D. (2007). Sinusitis and Eustachian
tube dysfunction in children. Pediatric Al-
lergy and Immunology, 18(Suppl. 18), 35–39.
Leveque, H., Bialostozky, F., Blanchard, C. L.,
& Suter, C. M. (1979). Tympanometry in the
evaluation of vascular lesions of the middle
ear and tinnitus of vascular origin. Laryngo-
scope, 89(8), 1197–1218.
Levin, G., Fabian, P., & Stahle, J. (1988). Inci-
dence of otosclerosis. American Journal of
Otology, 9(4), 299–301.
McCoul, E. D., Weinreich, H. M., Mulder, H.,
Man, L. X., Schulz, K., & Shin, J. J. (2019).
Health care utilization and prescribing patterns for adult Eustachian tube dysfunction. Otolaryngology-Head and Neck Surgery,
160(6), 1071–1080.
McGuirt, W. F., Jr., & Stool, S. E. (1992). Tem-
poral bone fractures in children: A review
with emphasis on long-term sequelae. Clin-
ical Pediatrics, 31(1), 12–18.
McKenna, M., & de Venecia, R. (2007). Oto-
sclerosis. In G. Hughes & M. Pensak (Eds.),
Clinical otology (Vol. 3, pp. 258–271). New
York, NY: Thieme.
Metz, O. (1946). The acoustic impedance mea-
sured on normal and pathological ears.
Acta Otolaryngologica, 33(Suppl. 63), 29–48.
Moffat, D. A., & Hardy, D. G. (1989). Surgi-
cal management of large glomus jugulare tumours: Infra- and trans-temporal
approach. Journal of Laryngology and Otol-
ogy, 103(12), 1167–1180.
Morgan, W. E., Coker, N. J., & Jenkins, H. A.
(1994). Histopathology of temporal bone
fractures: Implications for cochlear implantation. Laryngoscope, 104(4), 426–432.
Müller, J. (1838). Ueber den feineren Bau und die
formen der krankhaften Geschwulste. Berlin,
Germany: G. Reimer.
Musiek, F. E., & Baran, J. A. (2020). The audi-
tory system: Anatomy, physiology, and clinical
correlates (2nd ed.). San Diego, CA: Plural
Publishing.
Nelson, M., Roger, G., Koltai, P. J., Garabedian,
E. N., Triglia, J. M., Roman, S., . . . Hammel, J. P. (2002). Congenital cholesteatoma:
Classification, management, and outcome.
Archives of Otolaryngology-Head and Neck
Surgery, 128(7), 810–814.
Nicol, J. W., & Johnstone, A. J. (1994). Tempo-
ral bone fractures in children: A review of
34 cases. Journal of Accident and Emergency
Medicine, 11(4), 218–222.
Oldring, D., & Fisch, U. (1979). Glomus tumors
of the temporal region: Surgical therapy.
American Journal of Otology, 1(1), 7–18.
O’Leary, M. J., Shelton, C., Giddings, N. A.,
Kwartler, J., & Brackmann, D. E. (1991). Glomus tympanicum tumors: A clinical perspective. Laryngoscope, 101(10), 1038–1043.
Olszewska, E., Wagner, M., Bernal-Sprekelsen,
M., Ebmeyer, J., Dazert, S., Hildmann, H., &
Sudhoff, H. (2004). Etiopathogenesis of cholesteatoma. European Archives of Oto-rhino-
laryngology, 261(1), 6–24.
O’Neill, P., Roberts, T., & Bradley Stevenson,
C. (2006). Otitis media in children (acute).
Clinical Evidence, 15, 500–510.
Palva, T., Ramsay, H., & Böhling, T. (1996).
Prussak’s space revisited. American Journal
of Otology, 17(4), 512–520.
Paradise, J. L., Rockette, H. E., Colborn, D. K.,
Bernard, B. S., Smith, C. G., Kurs-Lasky, M.,
& Janosky, J. E. (1997). Otitis media in 2253
Pittsburgh-area infants: Prevalence and risk
factors during the first two years of life.
Pediatrics, 99(3), 318–333.
Patel, S. J., Sekhar, L. N., Cass, S. P., & Hirsch, B.
E. (1994). Combined approaches for resection of extensive glomus jugulare tumors.
review of 12 cases. Journal of Neurosurgery,
A
80(6), 1026–1038.
Penido Nde, O., Borin, A., Iha, L. C., Sug-
uri, V. M., Onishi, E., Fukuda, Y., & Cruz,
O. L. (2005). Intracranial complications of
otitis media: 15 years of experience in 33
patients. Otolaryngology-Head and Neck Sur-
gery, 132(1), 37–42.
Pensak, M. L., & Jackler, R. K. (1997). Removal
of jugular foramen tumors: The fallopian
bridge technique. Otolaryngology-Head and
Neck Surgery, 117(6), 586–591.

178 Disorders of the Auditory System
https://t.me/medicina_free
Politzer, A. (1894). Veber primare Erkankung
der Knochernen Labyrinth Kapsel. Ohren-
heilkunde, 25, 309–312.
Probst, R. (2007). Audiological evaluation of
patients with otosclerosis. Advances in Oto-
rhino-laryngology, 65, 119–126.
Proctor, B. (1967). Embryology and anatomy of
the Eustachian tube. Archives of Otolaryngol-
ogy, 86(5), 503–514.
Rane, R. L., Yut, J. P., & Berger, K. W. (1978).
Negative needle deflection of the acoustic
reflex in otosclerotics. Journal of the Ameri-
can Audiology Society, 3(6), 241–244.
Rosenfeld, R. M., Shin, J. J., Schwartz, S. R.,
Coggins, R., Gagnon, L., Hackell, J. M.,
. . . Corrigan, M. D. (2016). Clinical practice guideline: Otitis media with effusion
(Update). Otolaryngology-Head and Neck
Surgery, 154(Suppl. 1), S1–S41.
Rosenwasser, H. (1945). Carotid body tumor
of the middle ear and mastoid. Archives of
Otolaryngology, 41, 64–67.
Rovers, M. M., Glasziou, P., Appelman, C. L.,
Burke, P., McCormick, D. P., Damoiseaux,
R. A., . . . Hoes, A. W. (2006). Antibiotics for
acute otitis media: A meta-analysis with
individual patient data. Lancet, 368(9545),
1429–1435.
Rovers, M. M., Schilder, A. G., Zielhuis, G. A.,
& Rosenfeld, R. M. (2004). Otitis media.
Lancet, 363(9407), 465–473.
Sadé, J., Babiacki, A., & Pinkus, G. (1983). The
metaplastic and congenital origin of cholesteatoma. Acta Otolaryngologica, 96(1–2),
119–129.
Sadler-Kimes, D., Siegel, M. I., & Todhunter,
J. S. (1989). Age-related morphologic dif
ferences in the components of the Eustachian tube/middle ear system. Annals of
Otology, Rhinology, and Laryngology, 98(11),
854–858.
Saringer, W., Khayal, H., Ertl, A., Schoeggl, A.,
& Kitz, K. (2001). Efficiency of gamma knife
radiosurgery in the treatment of glomus
jugulare tumors. Minimally Invasive Neuro-
surgery, 44(3), 141–146.
Schuknecht, H. F. (1989). Congenital aural
atresia. Laryngoscope, 99(9), 908–917.
-
Shapiro, R. S. (1979). Temporal bone fractures
in children. Otolaryngology-Head and Neck
Surgery, 87(3), 323–329.
Shea, J. J., Jr. (1958). Fenestration of the oval
window. Annals of Otology, Rhinology, and
Laryngology, 67(4), 932–951.
Sheehan, J., Kondziolka, D., Flickinger, J., &
Lunsford, L. D. (2005). Gamma knife surgery for glomus jugulare tumors: An intermediate report on efficacy and safety. Jour-
nal of Neurosurgery, 102(Suppl.), 241–246.
Spector, G. J., Ciralsky, R. H., & Ogura, J. H.
(1975). Glomus tumours in the head and
neck: III. Analysis of clinical manifestations.
Annals of Otology, Rhinology, and Laryngology, 84(1 Pt. 1), 73–79.
Spector, G. J., Maisel, R. H., & Ogura, J. H.
(1974). Glomus jugulare tumors: II. A clinicopathologic analysis of the effects of radiotherapy. Annals of Otology, Rhinology, and
Laryngology, 83(1), 26–32.
Sutherland, J. E., & Campbell, K. (1990).
Immittance audiometry. Primary Care, 17(2),
233–247.
Tos, M. (2000). A new pathogenesis of meso-
tympanic (congenital) cholesteatoma. Laryn-
goscope, 110(11), 1890–1897.
Tos, M., & Bak-Pedersen, K. (1972). The patho-
genesis of chronic secretory otitis media.
Archives of Otolaryngology, 95(6), 511–521.
Touma, J. B., & Touma, B. J. (2006). Atlas of otos-
copy. San Diego, CA: Plural Publishing.
Toynbee, J. (1841). Pathological and surgi-
cal observations on the diseases of the ear.
Medico-Chirurgical Transactions, 24, 190–211.
Ulrich, K. (1926). Verletzungen des Gehoror-
gans bei schadelbasisfrakturen (eine histologische und klinissche Studie). Acta Oto-
laryngologica, 6(Suppl.), 1–150.
Williams, W. T., Ghorayeb, B. Y., & Yeakley,
J. W. (1992). Pediatric temporal bone fractures. Laryngoscope, 102(6), 600–603.
Woods, C. I., Strasnick, B., & Jackson, C. G.
(1993). Surgery for glomus tumors: The
Otology Group experience. Laryngoscope,
103(11 Pt. 2, Suppl. 60), 65–70.
Zender, C. A., Marzo, S. J., & Leonetti, J. P.
(2007). Diseases of the external auditory

4. Outer and Middle Ear Disorders 179
https://t.me/medicina_free
canal. In G. B. Hughes & M. L. Pensak (Eds.),
Clinical otology (3rd ed., pp. 211–222). New
York, NY: Thieme.
Zielhuis, G. A., Rach, G. H., van den Bosch,
A., & van den Broek, P. (1990). The prevalence of otitis media with effusion: A critical
review of the literature. Clinical Otolaryngol-
ogy and Allied Sciences, 15(3), 283–288.
Соседние файлы в папке Библиотека им академика М.И. Перельмана
