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- •Contents
- •Contributors
- •Preface
- •1. A Focused History of Surgery
- •2. Preoperative and Postoperative Management
- •3. Endoscopy and Endoscopic Intervention
- •4. Fundamentals of Laparoscopic Surgery
- •5. Laparoscopic Staging and Approaches to Cancer
- •6. Incisions, Closures, and Management of the Abdominal Wound
- •7. Hernias
- •9. Intestinal Stomas
- •10. Abdominal Abscess and Enteric Fistulae
- •11. Gastrointestinal Bleeding
- •12. Management of Abdominal Trauma
- •13. Abdominal Vascular Emergencies
- •14. Benign Esophageal Disorders
- •15. Gastroesophageal Reflux Disease and Hiatal Hernia (Including Paraesophageal)
- •16. Perspective on Benign Esophageal Disease
- •17. Cancer of the Esophagus
- •18. Surgical Procedures to Resect and Replace the Esophagus
- •19. Video-Assisted Thoracic Surgery of the Esophagus
- •20. Perspective on Malignant Esophageal Disease
- •21. Benign Gastric Disorders
- •22. Gastric Adenocarcinoma and Other Gastric Neoplasms (Except Gastrointestinal Stromal Tumors)

122 Part II Abdominal Wall
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54. Pringle JH, V. Notes on the arrest of hepatic hemorrhage due to trauma.
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55. Halstead WS. Ligature and suture material: the employment of ne silk
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56. Lucas CE, Ledgerwood AM. Prospective evaluation of hemostatic
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60. Gracias VH, Braslow B, Johnson J, et al. Abdominal compartment syndrome
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65. Culver DH, Horan TC, Gaynes RP, et al. Surgical wound infection
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elective colorectal surgery. Cochrane Database Syst Rev. 2009:CD001544.
67. Bucher P, Mermillod B, Gervaz P, Morel P. Mechanical bowel preparation for
elective colorectal surgery: a meta-analysis. Arch Surg. 2004;139:1359–1364;
discussion 65.
68. Urschel JD. Necrotizing soft tissue infections. Postgrad Med J. 1999;
75:645–649.
69. McHenry CRC, C.N. Soft tissue infection. In: Malangoni MHS, N.J.,
ed. Problems in General Surgery. Philadelphia, PA: Lippincott Williams
&Wilkins; 2002:7.
70. Anaya DA, Dellinger EP. Necrotizing soft-tissue infection: diagnosis and
management. Clin Infect Dis. 2007;44:705–710.
71. McHenry CR, Piotrowski JJ, Petrinic D, Malangoni MA. Determinants of
mortality for necrotizing soft-tissue infections. Ann Surg. 1995;221:558–563;
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72. Wall DB, Klein SR, Black S, de Virgilio C. A simple model to help distinguish necrotizing fasciitis from nonnecrotizing soft tissue infection. J Am
Coll Surg. 2000;191:227–231.
73. Struk DW, Munk PL, Lee MJ, Ho SG, Worsley DF. Imaging of soft tissue
infections. Radiol Clin North Am. 2001;39:277–303.
74. Brothers TE, Tagge DU, Stutley JE, Conway WF, Del Schutte H, Jr.,
Byrne TK. Magnetic resonance imaging dierentiates between necrotizing and non-necrotizing fasciitis of the lower extremity. J Am Coll Surg.
1998;187:416–421.
75. Hopkins KL, Li KC, Bergman G. Gadolinium-DTPA-enhanced magnetic
resonance imaging of musculoskeletal infectious processes. Skeletal Radiol.
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76. Riseman JA, Zamboni WA, Curtis A, Graham DR, Konrad HR, Ross DS.
Hyperbaric oxygen therapy for necrotizing fasciitis reduces mortality and
the need for debridements. Surgery. 1990;108:847–850.
77. Clark LA, Moon RE. Hyperbaric oxygen in the treatment of life-threatening soft-tissue infections. Respir Care Clin N Am. 1999;5:203–219.
78. Bisno AL, Stevens DL. Streptococcal infections of skin and soft tissues.
N Engl J Med. 1996;334:240–245.
79. Nurmohamed MT, Verhaeghe R, Haas S, et al. A comparative trial of a
low molecular weight heparin (enoxaparin) versus standard heparin for the
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80. Kakkar VV, Boeckl O, Boneu B, et al. Ecacy and safety of a low molecular-weight heparin and standard unfractionated heparin for prophylaxis of postoperative venous thromboembolism: European multicenter
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abdominal fascial closure: a meta-analysis. Ann Surg. 2000;231:436–442.
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prospective study of 1129 major laparotomies. Br Med J. 1982;89:1350.
83. Webster C, Neumayer L, Smout R, et al. Prognostic models of abdominal
wound dehiscence after laparotomy. J Surg Res. 2003;109:130–137.
84. Gislason H, Viste A. Closure of burst abdomen after major gastrointestinal operations–comparison of dierent surgical techniques and later
development of incisional hernia. Eur J Surg. 1999;165:958–961.
85. Pavlidis TE, Galatianos IN, Papaziogas BT, et al. Complete dehiscence of the
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86. Schimp VL, Worley C, Brunello S, et al. Vacuum-assisted closure in
the treatment of gynecologic oncology wound failures. Gynecol Oncol.
2004;92:586–591.

HERNIAS
Patrick J. Javid • Jacob A. Greenberg
• David C. Brooks
7
A hernia is de ned as an area of weakness or complete
disruption of the bromuscular tissues of the body wall.
Structures arising from the cavity contained by the body
wall can pass through, or herniate, through such a defect.
While the de nition is straightforward, the terminology is
often misrepresented. It should be clear that hernia refers to
the actual anatomic weakness or defect, and hernia contents
describe those structures that pass through the defect.
Hernias are among the oldest known a ictions of humankind, and surgical repair of the inguinal hernia is the most
common general surgery procedure performed today.
Despite the high incidence, the technical aspects of hernia
repair continue to evolve.
1
INGUINAL HERNIA
History
e word “hernia” is derived from a Latin term meaning
“arupture.” e earliest reports of abdominal wall hernias date
back to 1500 . During this early era, abdominal wall hernias
were treated with trusses or bandage dressings. e rst evidence of operative repair of a groin hernia dates back to the rst
century . e original hernia repairs involved wide operative
exposures through scrotal incisions requiring orchiectomy on
the involved side. Centuries later, around 700 , principles
of operative hernia repair evolved to emphasize mass ligation
and en bloc excision of the hernia sac, cord, and testis distal to
the external ring. e rst report of groin hernia classi cation
based on the anatomy of the defect (ie, inguinal versus femoral)
dates back to the 14th century, and the anatomical descriptions of direct and indirect types of inguinal hernia were rst
reported in 1559.
Bassini revolutionized the surgical repair of the groin hernia with his novel anatomical dissection and low recurrence
rates. He rst performed his operation in 1884, and published his initial outcomes in 1889.
follow-up of patients over a 5-year period, with just ve
recurrences in over 250 patients. is rate of recurrence was
unheard of at the time and marked a distinct turning point
2
Bassini reported 100%
in the evolution of herniorraphy. Bassini’s repair emphasizes
both the high ligation of the hernia sac in the internal ring, as
well as suture reinforcement of the posterior inguinal canal.
e operation utilizes a deep and super cial closure of the
inguinal canal. Inthe deep portion of the repair, the canal is
repaired by interrupted sutures a xing the transversalis fascia medially to the inguinal ligament laterally. is requires
an incision through the transversalis fascia. e super cial
closure is provided by the external oblique fascia.
In addition to Bassini’s contributions, the rst true
Cooper’s ligament repair, which a xes the pectineal ligament
to Poupart’s ligament and thereby repairs both inguinal
and femoral hernia defects, was introduced by Lotheissen
in 1898. McVay further popularized the Cooper’s ligament
repair with the addition of a relaxing incision to reduce the
increased wound tension.
e advances in groin hernia repair in the century following
Bassini have shared the primary goal of reducing long-term
hernia recurrence rates. To this end, e orts have been directed
at developing a repair that imparts the least tension on the
tissues that are brought together to repair the hernia defect.
Darn repairs were rst introduced in the early 20th century
to reduce wound tension by using either autologous tissue
or synthetic suture to bridge the gap between fascial tissues.
Muscle and fascial aps were attempted without consistent
success. In 1918, Handley introduced the rst use of silk as a
prosthetic darn and nylon followed several years later. However, it was found that heavy prosthetic material increased
the risk of wound infection, and the silk suture ultimately
lost its strength over time. e use of autologous or synthetic patches was also attempted in order to reduce wound
tension and improve rates of recurrence. e rst patches,
beginning in the early 20th century, consisted of silver wire
ligree sheets that were placed along the inguinal canal. Over
time, the sheets su ered from metal fatigue leading to hernia
recurrence. Reports of the wire patches eroding into adjacent inguinal structures and even the peritoneal cavity itself
caused even more concern with this technique. e modern
synthetic patch, made of a plastic mono lament polymer
(polyethylene), was introduced by Usher in 1958. Lichtenstein, who developed a sutureless hernia repair using a plastic
123

124 Part II Abdominal Wall
mesh patch placed across the inguinal oor, further popularized this technique.
In the search for a technical means to reduce recurrence,
emphasis was also placed on a meticulous dissection that
would avoid placement of a prosthetic mesh. e most
popular version was the Shouldice technique, initially introduced in 1958, and in essence a modication of the Bassini
operation. is technique involves meticulous dissection of
the entire inguinal oor and closure of the inguinal canal in
four layers. e transversalis fascial layer itself is closed in two
layers, as opposed to the single layer of interrupted suture
advocated by Bassini. While the operation can be technically
challenging to the beginner, it has been associated with excellent long-term outcomes and low recurrence rates.
Today, laparoscopic techniques have been validated as safe
and eective in the treatment of groin hernias and have become
commonplace. e laparoscopic approaches were initially
developed in the early 1990s as laparoscopic techniques diffused throughout other specialties of general surgery.
Epidemiology
Seventy-ve percent of all abdominal wall hernias are found
in the groin, making it the most common location for an
abdominal wall hernia. Of all groin hernias, 95% are hernias
of the inguinal canal with the remainder being femoral hernia
defects. Inguinal hernias are nine times more common in men
than in women. Although femoral hernias are found more
often in women, the inguinal hernia is still the most common
3
hernia in women.
e overall lifetime risk of developing a
groin hernia is approximately 15% in males and less than 5%
in females. ere is clearly an association between age and
hernia diagnosis. After an initial peak in the infant, groin hernias become more prevalent with advancing age. In the same
way, the complications of hernias (incarceration, strangulation, and bowel obstruction) are found more commonly at
the extremes of age.
Currently in this country, approximately 700,000 operations
for inguinal hernia repair are performed annually.
4
Anatomic Classication
A thorough classication system has been developed to assist in
the proper diagnosis and management of the inguinal hernia.
All hernias can be broadly classied as congenital or acquired,
and it is thought that the vast majority of inguinal hernias
are congenital in nature. Acquired groin hernias develop after
surgical incision and manipulation of the involved abdominal wall tissues. Given the paucity of primary groin incisions
utilized in modern general surgery, acquired hernias of the
inguinal or femoral region are rare.
Inguinal hernias are further divided by anatomical location
into direct and indirect types. is dierentiation is based
on the location of the actual hernia defect in relation to the
inferior epigastric vessels. e inferior epigastric vessels are
continuous with the superior epigastric vessels that originate
from the internal mammary artery cephalad and ultimately
course caudally into the common femoral artery and vein.
ese vascular structures make up the lateral axis of Hesselbach’s triangle, which includes the lateral border of the rectus
sheath as its medial border and the inguinal (Poupart’s) ligament itself as the inferior border. Hernias that develop lateral
to the inferior epigastric vessels are termed indirect inguinal
hernias, and those that develop medial to the vessels are direct
inguinal hernias. In this way, direct hernia defects are found
within Hesselbach’s triangle. Hernias of the femoral type are
located caudal or inferior to the inguinal ligament in a medial
position.
e indirect inguinal hernia develops at the site of the
internal ring, or the location where the spermatic cord in
men and the round ligament in women enters the abdomen.
While they may present at any age, indirect inguinal hernias are thought to be congenital in etiology. e accepted
hypothesis is that these hernias arise from the incomplete
or defective obliteration of the processus vaginalis during
the fetal period. e processus is the peritoneal layer that
covers the testicle or ovary as it passes through the inguinal
canal and into the scrotum in men or the broad ligament in
women. e internal ring closes, and the processus vaginalis
becomes obliterated following the migration of the testicle
into the inguinal canal. e failure of this closure provides an
environment for the indirect inguinal hernia to develop. In
this way, the remnant layer of peritoneum forms a sac at the
internal ring through which intra-abdominal contents may
herniate, thereby resulting in a clinically detectable inguinal hernia. Anatomically, the internal ring is lateral to the
external ring and the remainder of the inguinal canal, and
this explains the lateral relationship of the indirect inguinal
hernia to the inferior epigastric vessels. It is noteworthy that
indirect inguinal hernia develops more frequently on the
right, where descent of the gonads occurs later during fetal
development.
Direct inguinal hernias, in contrast, are found medial to
the inferior epigastric artery and vein, and within Hesselbach’s
triangle. ese hernias are acquired and only rarely found in
the youngest age groups. ey are thought to develop from
an acquired weakness in the bromuscular structures of the
inguinal oor, so that the abdominal wall in this region can
no longer adequately contain the intra-abdominal contents.
e exact relationship between direct inguinal hernias and
heavy lifting or straining remains unclear, and some studies
suggest that the incidence of direct hernia is no greater in
people in professions that routinely involve heavy manual
5
labor.
While femoral hernias account for less than 10% of all groin
hernias, their presentation can be more acute in nature. In fact, it
is estimated that up to 40% of femoral hernias present as emer-
3
gencies with hernia incarceration or strangulation.
In this way,
femoral hernias may also present with bowel obstruction. e
empty space through which a femoral hernia forms is medial to
the femoral vessels and nerve in the femoral canal and adjacent
to the major femoral lymphatics. e inguinal ligament forms

Chapter 7 Hernias 125
the cephalad border of the empty space. However, while the
empty space is inferior to the ligament, the herniated contents
may present superior tothe ligament, thereby making an accurate diagnosis dicult.
Femoral hernias are much more common in females than
in males, although inguinal hernias are still the most common hernia in women. e predilection for femoral hernias
in women may be secondary to less bulky groin musculature
or weakness in the pelvic oor tissues from previous childbirth. It has been shown that previous inguinal hernia repair
may be a risk factor for the subsequent development of a
femoral hernia.
3
Anatomy of the Groin
e boundaries of the inguinal canal must be understood to
comprehend the principles of hernia repair. In the inguinal
canal, the anterior boundary is the external oblique aponeurosis; the posterior boundary is composed of the transversalis
fascia with some contribution from the aponeurosis of the
transversus abdominis muscle; the inferior border is imparted
by the inguinal and lacunar ligaments; and the superior
boundary is formed by the arching bers of the internal
oblique musculature.
e internal (or deep) inguinal ring is formed by a normal
defect in the transversalis fascia through which the spermatic
cord in men and the round ligament in women pass into
the abdomen from the extraperitoneal plane. e external
(orsupercial) ring is inferior and medial to the internal ring
and represents an opening of the aponeurosis of the external oblique. e spermatic cord passes from the peritoneum
through the internal ring and then caudally into the external
ring before entering the scrotum in males.
From supercial to deep, the surgeon rst encounters
Scarpa’s fascia after incising the skin and subcutaneous
tissue. Deep to Scarpa’s layer is the external oblique aponeurosis, which must be incised and spread to identify the cord
structures. e inguinal ligament represents the inferior
extension of the external oblique aponeurosis, and extends
from the anterior superior iliac spine to the pubic tubercle.
e medial extension of the external oblique aponeurosis
forms the anterior rectus sheath. e iliohypogastric and
ilioinguinal nerves, which provide sensation to the skin,
penis, and the upper medial thigh, lie deep to the external oblique aponeurosis in the groin region. e internal
oblique aponeurosis is more prominent cephalad in the
inguinal canal, and its bers form the superior border of the
canal itself. e cremaster muscle, which envelops thecord
structures, originates from the internal oblique musculature.
e transversus abdominis muscle and its fascia represent
thetrue oor of the inguinal canal. Deep to the oor isthe
preperitoneal space, which houses the inferior epigastric
artery and vein, the genitofemoral and lateral femoral cutaneous nerves, and the vas deferens, which traverses this
space to join the remaining cord structures at the internal
inguinal ring.
Etiology
e indirect inguinal hernia, the most common form of
groin hernia across all ages and both genders, is thought to be
congenital in etiology. e processus vaginalis is the pocket
of peritoneum that forms around the testicle as it descends
through the internal ring and along the inguinal canal into
the scrotum during the 28th week of gestation. e primary
etiology behind the indirect inguinal hernia is believed to be
a patent processus vaginalis, which in essence represents a
hernia sac. In this way, the hernia defect is the internal ring
itself, and the sac is preformed but never closes at the end of
gestation. Once intra-abdominal contents nd their way into
the sac, an indirect inguinal hernia is formed.
It is likely, however, that every person with a patent
processus vaginalis does not develop an inguinal hernia during
his or her lifetime. us, other predisposing factors must aid
in indirect inguinal hernia formation. It is commonly thought
that repeated increases in intra-abdominal pressure contribute
to hernia formation; hence, inguinal hernias are commonly
associated with pregnancy, chronic obstructive pulmonary
disease, abdominal ascites, patients who undergo peritoneal
dialysis, laborers who repeatedly ex the abdominal wall
musculature, and individuals who strain from constipation.
Itis also thought that collagen formation and structure deteriorates with age, and thus hernia formation is more common
in the older individual.
Several inborn errors of metabolism can lead to hernia
formation. Specically, conditions such as Ehlers–Danlos syndrome, Marfan’s syndrome, Hunter’s syndrome, and Hurler’s
syndrome can predispose to defects in collagen formation.
ere is evidence that cigarette smoking is associated with
connective tissue disruption, and hernia formation is more
common in the chronic smoker.
Clinical Manifestations
e groin hernia can present in a variety of ways, from the
asymptomatic hernia to frank peritonitis in a strangulated
hernia. Many hernias are found on routine physical examination or on a focused examination for an unrelated complaint.
ese groin hernias are usually fully reducible and chronic
in nature. Such hernias are still referred for repair since they
invariably develop symptoms, and asymptomatic hernias still
have an inherent risk of incarceration and strangulation.
e most common presenting symptomatology for a
groin hernia is a dull feeling of discomfort or heaviness in the
groin region that is exacerbated by straining the abdominal
musculature, lifting heavy objects, or defecating. ese maneuvers worsen the feeling of discomfort by increasing the intra abdominal pressure and forcing the hernia contents through the
hernia defect. Pain develops as a tight ring of fascia outlining
the hernia defect compresses intra-abdominal structures with a
visceral neuronal supply. With a reducible hernia, thefeeling of
discomfort resolves as the pressure is released when the patient
stops straining the abdominalmuscles. epain is often worse

126 Part II Abdominal Wall
at the end of the day, and patients in physically active professions may experience the pain more often than those who lead
a sedentary lifestyle.
Overwhelming or focal pain from a groin hernia is unusual
and should raise the suspicion of hernia incarceration or
strangulation. An incarcerated hernia occurs when the hernia
contents are trapped in the hernia defect so that the contents
cannot be reduced back into the abdominal cavity. e tight
circumferential pressure applied by the hernia defect serves to
impede the venous outow from the hernia contents, resulting in congestion, edema, and tissue ischemia. Ultimately, the
arterial inow to the hernia contents is compromised as well,
resulting in tissue loss and necrosis, termed strangulation of
the hernia.
All types of groin hernias are at risk for incarceration and
strangulation, although the femoral hernia seems to be predisposed to this complication. Incarceration and strangulation
of a groin hernia may present as a bowel obstruction when the
tight hernia defect constricts the lumen of the viscus. Hence,
all patients presenting with bowel obstruction require a thorough physical examination of the groin region for inguinal
and femoral hernias. If there is no bowel in the hernia sac, an
incarcerated groin hernia may alternatively present as a hard,
painful mass that is tender to palpation.
e physical examination diers between an incarcerated
hernia and a strangulated hernia. e incarcerated hernia may
be mildly tender due to venous congestion from the tight
defect. e strangulated hernia will be tender and warm and
may have surrounding skin erythema secondary to the inammatory reaction from the ischemic bowel. e patient with
the strangulated hernia may have a fever, hypotension from
early bacteremia, and a leukocytosis. e incarcerated hernia
requires operation on an urgent basis within 6–12 hours of
presentation. If the operation is delayed for any reason, serial
physical examinations are mandated to follow any change in
the hernia site indicating the onset of tissue loss. e strangulated hernia clearly requires emergent operation immediately
following diagnosis.
It may also be dicult to dierentiate fat from bowel
contents in the hernia sac. It is important to recognize that
incarcerated omental fat alone can produce signicant pain
and tenderness on physical examination.
Pregnancy and Groin Hernia
Not surprisingly, groin hernias during pregnancy may become
symptomatic. is is related to the increased intra-abdominal
pressure from the growing fetus and enlarging uterus. e
symptomatic groin discomfort may become positional later in
pregnancy as the uterus shifts location with movement. While
the risk of complications of groin hernias still exists during
pregnancy, the enlarging uterus may in theory protect against
incarceration by physically blocking the intra-abdominal contents from the inlet of the defect.
In general, elective repair of groin hernias during pregnancy is not recommended, even if they become increasingly
symptomatic. Emergent repair of the incarcerated or strangulated hernia is undertaken as needed.
Physical Examination
As with any hernia, the groin hernia should be properly examined with the patient in the standing position. is allows the
hernia contents to ll the hernia sac and make the hernia
obvious on physical examination. Some hernias, however,
may be easily identiable in the supine position. It should
be noted that the exact anatomical classication of the inguinal hernia (ie, indirect vs direct) is impossible to accurately
predict based on physical examination alone.
In the male patient, using the second or third nger, the
examiner should invaginate the scrotum near the external
ring and direct the nger medial toward the pubic tubercle. e examiner’s nger will thus lie on the spermatic
cord with the tip of the nger within the external ring.
e patient is then asked to cough or perform a Valsalva
maneuver. A true inguinal hernia will be felt as a silklike
sensation against the gloved nger of the examiner. is is
the infamous “silk glove” sign.
e female patient does not have the long and stretched
spermatic cord to follow with the examiner’s nger during the
physical examination. Instead, two ngers can be placed along
the inguinal canal, and the patient is asked to cough or strain.
If present, the examiner should feel the sensation of the hernia
sac against the gloved nger. Particular attention in the female
patient should be paid to the location of the sensation; femoral hernia sacs will present medial and just inferior to the lower
border of the inguinal ligament.
While the physical examination does not dier in the
infant, it can be more challenging to elicit the hernia impulse
given the compressed groin anatomy of the young child. It is
well known that a groin hernia can be more readily diagnosed
in the infant who is actively crying and hence increasing the
intra-abdominal pressure through exion of the abdominal
wall musculature.
e examination for the femoral hernia in both genders
involves palpation of the femoral canal just below the inguinal ligament in the upper thigh. In this way, the most easily
palpable landmark is the femoral artery, which is located lateral in the canal. Medial to the femoral artery is the femoral
vein, and the femoral empty space is just medial to the vein.
is area can be located easily, palpated with two ngers, and
then examined closely while the patient coughs or strains. In
general, a focused groin hernia examination should involve
the investigation for both inguinal and femoral hernias in
both genders.
Treatment
e treatment of all hernias, regardless of their location or
type, is surgical repair. Elective repair is performed to alleviate symptoms and to prevent the signicant complications

Chapter 7 Hernias 127
of hernias, such as incarceration or strangulation. While the
limited data available on the natural history of groin hernias
show that these complications are rare, the complications are
associated with a high rate of morbidity and mortality when
they occur. At the same time, the risks of elective groin hernia
repair, even in the patient with a complicated medical history,
are exceedingly low. Outcomes of surgical repair are generally
excellent with minimal morbidity and relatively rapid return
to baseline health.
e major risk with delayed surgical repair is the risk
of incarceration and/or strangulation. It is not possible to
reliably identify those hernias that are at an increased risk for
these complications. It is known that the risk of incarceration
of a hernia is greatest soon after the hernia manifests itself.
is is likely due to the fact that at the early stage of the
hernia, the defect is small and ts tightly around the hernia sac; therefore, any contents that ll the sac may quickly
become trapped within the hernia. Over time, the hernia
defect stretches due to the tissue that enters and leaves the sac
with changes in intra-abdominal pressure. After 6 months,
the risk of hernia incarceration decreases from 5% per year
to 1–2% per year. In general, the larger the palpable defect
on physical examination, the lower the risk of incarceration.
Clearly, all risks of tissue loss aside, elective hernia repair is
still preferred to emergent repair.
Anesthesia
Groin hernia repair can be performed using a variety of
anesthesia options, including general, regional (such as spinal
or epidural), and local anesthesia.
require general anesthesia in order to provide the complete muscle relaxation needed to achieve insuation of the preperitoneal
or peritoneal space.
Open groin hernia repairs are most often performed
using either regional or local anesthesia. Local anesthesia
with controlled intravenous sedation, referred to as monitored anesthesia care, is often preferred in the repair of the
reducible inguinal hernia. Its advantages include the ease of
induction and awakening, the short postanesthesia recovery
period, and the fact that its intensity can easily be titrated up
or down based on patient comfort levels intraoperatively. e
only major disadvantage to this approach is in patients who
experience considerable pain during repairs of large groin
hernias.
In groin hernia repair, local anesthesia can be administered as a direct inltration of the tissues to be incised or as
a local nerve block of the ilioinguinal and iliohypogastric
nerves. e latter is associated with improved local pain control, but may be dicult to achieve. e local nerve block
also spares the soft tissue of edema from diuse inltration
of local anesthesia.
Spinal or continuous epidural anesthesia allows the surgeon greater freedom to maneuver within the operative eld
since the anesthetized region is larger than in local anesthesia. However, these modes of anesthesia carry their own
6
Laparoscopic repairs usually
infrequent risks such as urinary retention, prolonged anesthetic eect, hypotension, and spinal headache. ey may
also be associated with longer in-hospital recovery times on
the day of surgery.
A randomized trial of local, regional, and general
anesthesia in 616 adult patients undergoing open inguinal
hernia repair in 10 hospitals found that local anesthesia was
7
superior in the early postoperative period.
Compared to
those who received regional or general anesthesia, patients
who received local anesthesia had less postoperative pain
and nausea, shorter time spent in the hospital, and fewer
unplanned overnight admissions (3% vs 14% and 22%,
respectively).
Operative Techniques
Successful surgical repair of a hernia depends on a tensionfree closure of the hernia defect to attain the lowest possible
recurrence rate. Previous eorts to simply identify the defect
and suture it closed resulted in unacceptably high recurrence
rates of up to 15%. Modern techniques have improved upon
this recurrence rate by placement of mesh over the hernia
defect, or in the case of laparoscopic repair, behind the hernia defect. One exception to this rule is the classic Shouldice
repair, which uses meticulous dissection and closure without
mesh placement to obtain a consistently low recurrence rate.
Another benet of the tension-free closure is that it has been
shown to cause the patient signicantly less pain and discomfort in the short-term postoperative period.
Figure 7-1 illustrates the essential steps to the modern open
inguinal hernia repair. All of the open anterior herniorraphy
techniques begin with a transversely oriented, slightly curvilinear skin incision of approximately 6–8 cm positioned one
to two ngerbreadths above the inguinal ligament. Dissection
is carried down through the subcutaneous and Scarpa’s layers.
e external oblique aponeurosis is identied and cleaned
so that the external ring is identied inferomedially. Being
careful to avoid injury to the iliohypogastric and ilioinguinal
nerves, the aponeurosis is incised sharply and opened along
its length through the external ring with ne scissors. e
nerves underlying the external oblique fascia are then identied and isolated for protection. e soft tissue is cleared o
the posterior surface of the external oblique aponeurosis on
both sides and the spermatic cord is mobilized. Using a combination of blunt and sharp dissection, the cremaster muscle
bers enveloping the cord are separated from the cord structures and the cord itself is isolated. At this point, it is possible
to accurately dene the anatomy of the hernia. An indirect
hernia will present with a sac attached to the cord in an anteromedial position extending superiorly through the internal
ring. A direct inguinal hernia will present as a weakness in the
oor of the canal posterior to the cord. A pantaloon defect
will present as both a direct and an indirect defect in the same
inguinal canal.
e specics of the common modern techniques for hernia
repair will be discussed further.

128 Part II Abdominal Wall
A
C
B
D
E
F
FIGURE 7-1 Adult hernia incision and dissection. A. Transverse incision. B. Curved skin crease incision. C. e aponeurosis of the
external oblique is incised along the direction of its bers. D. e inguinal canal is exposed and the spermatic cord mobilized. E. e
spermatic cord has been skeletonized, and the internal ring and posterior wall of the canal (the transversalis fascia) have been dened.
F. A medium-sized sac has been dissected free of the cord elements.

Chapter 7 Hernias 129
G
I
FIGURE 7-1 Continued—G. e sac has been invaginated. H. A long or complete sac is being dissected free close to the internal ring. I.
e sac has been transected.
THE SHOULDICE TECHNIQUE
e Shouldice technique is commonly used for open repair
of inguinal hernias and is the most popular pure tissue hernia
repair. It is in essence the modern evolution of the Bassini
repair performed in a multilayered fashion. Both operations
use a tightening of the internal ring and closure of the transversalis fascia to the inguinal ligament as their primary tenets
of hernia repair.
8
Figure 7-2 illustrates the basic steps in the Shouldice repair.
After suitable exposure and isolation of the cord, a pair of
scissors is passed posterior to the transversalis fascia beginning
at the medial pillar of the internal ring and extending inferomedially to the pubic tubercle. In this way, the transversalis
fascia is separated from the preperitoneal fat plane. Care must
be taken at this stage to preserve the inferior epigastric vessels
that reside in the preperitoneal space. e transversalis fascia
is then opened with scissors along the entire inguinal oor
H
from internal ring to pubic tubercle, and the posterior surface of the transversalis is cleaned of its preperitoneal attachments. As the rst layer of the repair, the free edge of the
lower transversalis ap is sutured in a continuous, imbricated
fashion behind the upper ap to the posterior surface of the
upper transversalis fascia and the lateral component of the
posterior rectus sheath. is running suture layer is started
medially at the pubic tubercle and carried up to and through
the internal ring, thereby tightening the transversalis fascia
around the cord at its entrance to the inguinal canal. e
rst layer is not tied but continued in a running fashion from
lateral to medial as a second layer closing the upper transversalis ap to the base of the lower edge as well as the inguinal
ligament. is second layer progresses medially to the pubic
tubercle where it is tied to the original tail that started the rst
layer. e third layer of continuous suture starts at the tightened internal ring and brings together the conjoined tendon
(the internal oblique and transversus abdominis aponeuroses)

A
B
C
E
D
F
G
FIGURE 7-2 e Shouldice operation. A. e transversalis fascia is being incised. B. e upper and lower aps of the transversalis fascia have
been dissected free and elevated to expose the extraperitoneal fat and the inferior epigastric vessels. C. e rst layer of the Shouldice operation.
D. e second layer. E. e third layer. F. e fourth layer. G. e external oblique aponeurosis has been repaired anterior to the spermatic cord.
130

Chapter 7 Hernias 131
medially with the inguinal ligament laterally. is layer is run
down to the pubic tubercle, and returns to the internal ring as
the fourth layer including the anterior rectus sheath medially
with the posterior aspect of the external oblique aponeurosis
laterally. e cord can now be relaxed gently on the new
inguinal oor, and the external oblique aponeurosis is closed
in one to two additional continuous layers extending down to
the external ring to reapproximate this structure. e original
descriptions of the operation by Shouldice used continuous
stainless steel wire suture for all four layers of repair, although
surgeons commonly use permanent synthetic suture today.
e Shouldice Hospital reports excellent long-term outcomes from their operation with recurrence rates less than 1%
9,10
in selected patients.
ese results have not been achieved
with any other pure tissue technique. e operation is well
tolerated by most patients using local anesthesia only. From
the multiple, overlapping, continuous suture lines, Shouldice
proponents argue that any tension brought about in this type
of closure is dispersed throughout the entire inguinal canal.
e dissection is complicated, however, and requires excellent
surgical technique and anatomic awareness. Moreover, other
surgeons utilizing the Shouldice method have not achieved
recurrence rates this low. us, the low rate of recurrence
associated with the Shouldice technique likely depends on
the level of surgical expertise and the patient selection. In
one report of 183 inguinal hernia repairs using the Shouldice technique under local anesthesia, the recurrence rates
for beginners versus more experienced surgeons were 9.4%
versus 2.5%, respectively.
11
A recent meta-analysis conducted by the Cochrane
Collaboration compared the Shouldice technique with other
12
open techniques for inguinal hernia repair.
e analysis
incorporated results from 16 dierent randomized or quasirandomized studies and compared 2566 hernias repaired via
the Shouldice technique with 1121 hernias repaired with mesh
and 1608 hernias repaired with other nonmesh techniques.
e recurrence rate for the Shouldice repair was signicantly
higher than mesh repair (odds ratio 3.8), but signicantly
lower than nonmesh repair (Odds Ratio 0.62). ere were
no signicant dierences between the groups with respect
to complications, length of stay, or chronic pain following
12
herniorraphy.
us, the Shouldice technique is associated
with a higher recurrence rate than mesh repairs, but appears
to be the repair of choice in situations where mesh cannot be
implanted.
THE COOPER LIGAMENT REPAIR
e Cooper ligament repair is the only technique that denitively repairs both the inguinal and femoral hernia defects in
the groin. e operation is often named after Chester McVay,
who popularized the operation in the 1940s and introduced
the concept of the relaxing incision to decrease the tension
from the repair. e repair is also a primary tissue repair in
that no mesh is utilized.
e Cooper ligament repair begins similar to the Shouldice
procedure, and exposure and isolation of the cord is performed.
e transversalis fascia is then opened and cleaned posteriorly.
At this time, Cooper’s ligament is identied and dissected free
of its brous and fatty attachments. e defects are repaired
by using interrupted suture to ax the upper border of the
transversalis fascia to Cooper’s ligament beginning medially at
the pubic tubercle and continuing until the femoral sheath is
reached. At this point, the femoral canal is closed by carefully
suturing Cooper’s ligament to the femoral sheath. e repair
is continued with interrupted sutures between the transversalis fascia and the iliopubic tract laterally until the entrance
point of the cord is reached. In this way, the closure creates a
new, and tighter, internal inguinal ring around the cord.
e Cooper ligament repair requires a relaxing incision
because this pure tissue repair is associated with signicant tension in closing all three groin hernia defects. After
the transversalis fascia has been mobilized, and prior to the
closure of the fascia to Cooper’s ligament, a 2–4 cm vertical
incision is made at the lateral border of the anterior rectus
sheath beginning at the pubic tubercle and extending superiorly. e relaxing incision can be left open since the rectus
muscle should protect against any herniation; alternatively,
some surgeons argue for placement of a mesh over the relaxing
incision since hernia formation can occur at this site.
e Cooper ligament repair is an outstanding technique
for a femoral hernia and is associated with excellent long-term
results in experienced hands. Disadvantages of the repair
include a longer operating time, a more extensive dissection,
the potential for vascular injury and thromboembolic
complications from the femoral vessels, and a longer postoperative recovery phase.
Prosthetic Repairs
Polypropylene mesh is the most common prosthetic used
today in mesh repairs of the inguinal hernia. e two most
common prosthetic repairs are the Lichtenstein
and patch” repair as described by Gilbert
by Rutkow and Robbins.
15
e type of mesh to be used during prosthetic inguinal
hernia repairs deserves a brief discussion. e most common
and preferred mesh for groin hernia repair is a polypropylene
woven mesh marketed under a variety of names. Polypropylene
is preferred because it allows for a brotic reaction to occur
between the inguinal oor and the posterior surface of the
mesh, thereby forming scar and strengthening the closure of
the hernia defect. is brotic reaction is not seen to the same
extent with other varieties of prosthetic, namely expanded
polytetrauoroethylene (PTFE) mesh. PTFE is often used for
repair of ventral or incision hernias in which the brotic reaction with the underlying serosal surface of the bowel is best
avoided.
ere are limited prospective, randomized data comparing
the recurrence rate of open prosthetic repairs versus open
nonprosthetic repairs. An attempted meta-analysis concluded
that mesh repair was associated with fewer overall recurrences,
although the authors report that formal analysis was limited
13
and the “plug
14
and popularized
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