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Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3627_Библиотеки_им_академика_М_И_Перельмана

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RadCases.thieme.com RadCases Interventional Radiology
Imaging Findings
A
C D
(A, B) Selected images from a contrast-enhanced CT scan of the abdomen and pelvis. (A) Arterial phase, shows a large retroperitoneal hematoma. There is
active extravasation of contrast (arrow). (B) Delayed image, shows pooling of the extravasated contrast within the retroperitoneum. (C) Selected image of a digital subtraction angiogram (DSA) of the right iliac artery. The image shows active, massive extravasation of contrast. (D) Selected image from a DSA
immediately after placement of a stent graft. No further extravasation is identied.
Dierential Diagnosis
Iliac artery perforation with active bleeding: The images on the CT scan are diagnostic. The injury likely occurred after a high puncture during cardiac catheterization. The bleeding most probably started after the patient resumed her anticoagulants.
Spontaneous bleed: This is an unlikely option as there is history of a previous cardiac catheterization.
Pearls and Pitfalls
üUltrasound guidance is useful to prevent puncture-
related complications.
üCTA is the imaging modality of choice to evaluate these
cases.
üIf CTA is positive, it should be reported immediately. üImmediate angiographic evaluation is essential and may
be therapeutic.
B
üAccurate stent graft placement may be life-saving.
üMake sure all images are read properly; missing one of
Essential Facts
these cases can be a fatal mistake.
• Vascular access site–related complications are common. Range is between 1 and 9%.
• Punctures above the inguinal ligament are complicated with pelvic and retroperitoneal bleeding.
• Punctures below the inguinal ligament distribute into the thigh compartments.
• Triple-phase CT and CT angiography (CTA) are excellent diagnostic methods to detect acute internal bleeding.
• Active bleeding detected on a CTA is a medical emergency; it needs to be assessed immediately.
Case 32
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A
Clinical Presentation
A 42-year-old man with a history of alcoholism presents with upper gastrointestinal bleeding.
B
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Imaging Findings
A B
(A, B) Selected images from a contrast-enhanced CT scan of the abdomen. (A) A delayed axial image showing absence of the splenic vein and large gastric
varices. (B) A coronal reconstruction showing similar ndings. (C) Selected spot lm obtained during a transhepatic variceal embolization. The occlusion balloon is placed within the left gastric vein. The presence of large gastric varices is conrmed with a balloon-occluded retrograde venogram.
Dierential Diagnosis
Sinistral portal hypertension: Splenic vein occlusion and isolated gastric varices. Images show large gastric varices but no esophageal varices.
Cirrhosis with gastric varices: This is a possibility because the patient has a history of ethanol abuse. However, most patients with post-sinusoidal portal hypertension usually have esophageal varices.
Essential Facts
• Isolated gastric varices are not common.
• The presence of gastric varices without esophageal varices should signal the possibility of splenic vein occlusion.
• Splenic vein occlusion may be secondary to pancreatitis or tumors.
• A common clinical presentation of splenic vein occlusion is bleeding from gastric varices.
Pearls and Pitfalls
üThe presence of gastric varices without esophageal
varices is strongly associated with splenic vein occlusion.
üTranscatheter embolization of bleeding gastric varices
is eective.
üEndovascular approach to gastric varices for
embolization may be antegrade, via the left gastric vein, or retrograde, via the adrenal vein (balloon-occluded retrograde transvenous obliteration [BRTO]).
üSplenic vein recanalization and stent placement is
another endovascular option to solve this problem.
üA fatal complication of BRTO is a fatal pulmonary
embolism resulting from accidental embolization of the therapeutic embolic agents to the lungs.
C
Case 33
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A
C
Clinical Presentation
A 62-year-old man with a history of alcohol abuse presents with grade 3 encephalopathy.
B
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Imaging Findings
A
(A, B) Selected axial images from a contrast-enhanced MRI of the abdomen. The images show a small, irregular liver consistent with a cirrhotic liver. The
spleen is enlarged as well. There is a small amount of ascites. The portal vein is patent. There is a large recanalized paraumbilical vein, which forms a large shunt into the lower areas of the abdomen. The shunt arises from the left portal vein. (C) A coronal view at the supercial anterior abdominal wall. The large shunt is clearly demonstrated.
Dierential Diagnosis
Recanalized paraumbilical vein forming a large portosystemic shunt: The images are diagnostic. The
classic entry point in the left portal vein is nicely demonstrated.
Vascular venous malformation: A possibility. However, with the history of alcohol abuse and other signs of
portal hypertension, this possibility loses strength.
Arterioportal stula: This is another option; however, this is usually related to trauma in childhood, and this patient
has no such history. The hepatic or mesenteric arteries
are usually prominent in case of stulas.
Essential Facts
• Spontaneous portosystemic shunts form in people who
have cirrhosis with signicant portal hypertension.
• Examples include the following: Esophageal varices drain into the azygos system; gastrosplenic shunt arises from
the short gastric veins, connecting with the splenic vein,
and then drain into the left renal vein.
• The recanalized paraumbilical vein is another example.
These shunts typically arise from the left portal vein, have a tortuous course, form the “caput medusa” in the umbilical region, and usually drain into the left iliac veins.
B
Pearls and Pitfalls
üAngiographic evaluation and management of these
shunts is possible.
üEncephalopathy improves after successful embolization
of these shunts.
üIt is important to describe the nding in detail because
it is clinically important.
üMost clinicians only read the conclusion of the imaging
reports. If these ndings are not described in the nal comments, the clinician may miss the cause of
encephalopathy.
üThese shunts need to be addressed because if they
bleed, bleeding may have a fatal outcome.
C
Case 34
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BA
Clinical Presentation
A patient with liver cirrhosis presents with upper gastrointestinal bleeding. Patient had a history of a transjugular intrahepatic portosystemic shunt procedure 2 years prior to this admission.
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RadCases.thieme.com RadCases Interventional Radiology
Imaging Findings
A
(A) A spot lm from a selective splenic arteriogram. The angiographic catheter was placed in the mid-splenic artery. The splenic artery is patent and the spleen parenchyma is opacied. (B) A spot lm in the venous phase of a splenic arteriogram. The image shows a patent splenic vein, patent main portal vein, and a patent transjugular intrahepatic portosystemic shunt.
Dierential Diagnosis
Patent transjugular intrahepatic portosystemic shunt (TIPS): There are no gastric or esophageal varices
opacied. The shunt is patent, and there is no reux to
the mesenteric vein.
There is really no good dierential diagnosis: The TIPS
is patent, no varices are seen, and the splenic artery is
normal.
Essential Facts
• If a patient has a TIPS and the patient has an acute
gastrointestinal (GI) bleed, the rst thought is TIPS
failure.
TIPS created with VIATORR (W. L. Gore & Associates,
Flagsta, AZ) stent grafts have a signicantly improved
patency.
• Patients with cirrhosis may bleed from gastric ulcers or duodenal ulcers.
• Patients with acute GI bleed and a patent TIPS need a diagnostic work-up.
Pearls and Pitfalls
üDoppler ultrasound is the imaging method of choice to
evaluate a TIPS.
üClinical correlation is of great importance.
üVariceal bleeding is unusual if the patient has a patent
and functional TIPS.
üOther sources of bleeding need to be identied,
including ulcers, tears, or even portal gastropathy.
B
Case 35
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BA
Clinical Presentation
A 62-year-old man with a history of abdominal trauma presents with melena.
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Imaging Findings
A
(A) A spot lm showing the arterial phase of selective superior mesenteric artery injection. The superior mesenteric artery branches are normal. (B) A spot
lm during the venous phase of the same injection. The superior mesenteric vein branches are patent. There is occlusion of the main trunk of the portal vein. Small irregular venous collaterals are identied around the hepatic hilum and the stomach.
Dierential Diagnosis
Chronic portal vein thrombosis: The superior mesenteric arterial branches are patent and normal. The venous phase shows occlusion of the main trunk of the portal vein.
Acute portal vein thrombosis: This is not a good option
because there is no opacication of the main portal vein
or its branches. The main trunk of the portal vein is usually visible in acute thrombosis.
Pearls and Pitfalls
üInjection of 30 mg of intra-arterial papaverine
immediately before intra-arterial contrast injection improves imaging of the mesenteric veins (pharmacoangiography).
üPapaverine injection requires technical skill. Iodinated
contrast cannot be injected immediately after papaverine injection. Papaverine will crystallize and may cause local acute occlusion. The angiographic
catheter needs to be ushed with 10 to 15 mL of saline
immediately before contrast is injected.
B
Essential Facts
• Chronic portal vein thrombosis can be diagnosed by CT, MRI, or angiography.
• The main advantage of angiography is that it provides a map for therapy planning.
• Portal vein recanalization and stent placement are excellent therapeutic options.
• Surgical correction with mesoportal shunt is feasible in good surgical candidates.
Case 36
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A
Clinical Presentation
A 75-year-old man with psoriasis under treatment with methotrexate. Transjugular liver biopsy was performed to evaluate abnormal liver function tests. Presents to the emergency department 8 days after biopsy with sudden, severe abdominal pain.
B