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Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_574_Библиотеки_им_академика_М_И_Перельмана.pdf
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the position the joint assumes with joint swelling. Full flexion of the joint will be limited by the edematous dorsal skin and soft tissues(9).Ifthe PIP jointremains in amoderatelyflexed position,fibrosis,and shorteningofthe intracapsular and extracapsular structures may occur,leading to afixed contrac­tureofthe PIP joint. Pathologic tissues involved in contracture include exostosis, malunion, adhesions, and Watson’s checkrein ligaments—a thickening of Eaton’s previously described check ligaments.
Secondary to the cam effect of the metacarpal–phalangeal joint, where the intracapsular volume is maximal in extension and the capsular ligaments are lax, the accumulation of fluid in the MCP joint forces the MCP joint into extension (9).
There are avariety of etiologies that result in soft tissue contracturesand joint stiffness. Many authors have written on the importance of the various pathological structures, from thecheckrein ligaments to thecollateral ligaments (1– 3,8–11,13–19). Acareful physical exam andknowledge of thepre-existingconditioncan help distinguish between malunions or capsulo-ligamentous contractures or tendinous adhesions or muscle weakness/contractures or skin/fascial contractures. Clinically,itcan be difficult to identify which structures are predominately responsible for ajoint contrac­ture. Therefore, previously described surgical releases have emphasizedastepwise release, whichvaryfromauthor to author.
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Current Treatment Options
The current standard of carefor flexion contractures is still preventive andnon-operative management.Preventionis based on the principle of early joint motion within the safe arc of motion. Injuries requiringsurgicalrepair shouldbe performed as soon as soft tissues allow in order to achieve the earliest return of joint motion.
Once aflexion contracturehas occurred, the first modality should always be nonsurgical. Avariety of techniques have been described: serial casting, dynamic, and/or static splinting. The decision to abandonconservative treatmenthas no definitive time line. The literaturedoes not give any specifics as to when to proceedwithsurgicalrelease.Mosthand surgeons would consider surgical options after afailure to progress with three months of therapy and arange of motion of less than 458 .
Shinand Amadio suggest surgical releaseisindicated when residual joint motion is functionally disabling, there is normal to near normal joint articular surface and congruence, the joint motors are intact, and all nonoperative modalities have been correctly applied and exhausted (9). “Surgical release of the severely contracted PIP joint yields unpredictable results and should be considered only when both surgeon and patient understand its limitations” (9).
Avariety of external fixators have been developed and applied to PIP flexion contractures. External fixators have the advantageoftransmitting theextensionforce through the bone, as opposed to through the skin in splinting or casting. The few small case series in the literature have shown initial favorable results. However,they tend to have ahigh long-term recurrence of contracture and high rate of pin site infections (1,3,10).
Curtis made some of the first descriptions of the technique for acapsulectomy (8). He described this through avolar Bruner incision.In1999, Bruser and colleagues retrospectively reviewed the results of 45 fingers comparing apalmar incision to amidlateralincisionfor capsulectomy.Theyfoundthe
improvementinrange of motion of themidlateralincision group was statistically significantly better than in the volar incision group (14).
There have been many reports on the open release of PIP joint contractureswith varying results (2,8,10,11,13–18). Curtis reported that the greater the number of anatomic structures involved in the limitation of motion, the worse the postopera­tive range of motion (8). Ghidella et al. perf ormed a retrospective review of 68 PIP joints released as previously describedbyCurtis (8), whichidentified preoperative and intraoperative factorsthat were associatedwith worse outcomes. Accordingtotheir study,the ideal surgical candidate for correction of acontracted PIP joint has an exostosis (which can be removed), asimple diagnosis, is younger than 28 years, and has apreoperative maximum flexion measurement of ! 438 (2). In arecentliterature review of thetreatmentofpost­traumatic PIP flexion contractures, Hogan and Nunley found that after openrelease fin gerextension most commonly improves approximately 258 to 308 but is often accompanied by aloss of finger flexion (10). Overall, there has been anet improvement in motion, but some patients even lost motion after open release (2,10).
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Cadaveric Studies
Five fresh frozen hands were thawed and the index, long, ring,and little wereexaminedusing imag ingtoensure congruent gliding and to rule-outbonydeformity. The dorsal joint capsule of the MCP and PIP joints was located using fluoroscopy.A19-gauge needle was placed laterally into the dorsal joints under the capsule and perpendicular to the digit. A15-blade scalpel was used to incise the skin long­itudinally and asmall curved hemostat was used to bluntly dissect the soft tissue and punch into the dorsal joint through the capsule. Imaging was used to confirm the position of the hemostat in the dorsal joint. The hemostat was then pivoted at the joint entrance sweeping the tips proximally over the dorsal head,rupturing theproximal dorsal capsuleattachment. Sweeping more proximal the extensor tendon was elevated offthe proximal phalanx. Having completed the percutaneous release, tractionwas placed on theext ensor,flexor,and intrinsictendons,which werepreviouslyidentified and tagged. Each of the digits demonstrated normal congruent jointglidingthroughacomple te flexion and extension arc, withtip to palmand full extension without extensor lag.Lastly,eachofthe digits wasdissected by making a longitudinal incision andthe dorsal jointportal site was identified.The dorsal proximalcapsulewas completely divided. No extensor or joint injury was identified.
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INDICATIONS
Once aflexioncontracture hasoccurred, thepatientsare enrolledinahand rehabilitationprogram before surgery until progress in therapy has reached aplateau, usually not before three months.Patientsselectedfor surgicalrelease complainedofjoint stiffnessimpairing hand function, possessed anormal congruent articular jointsurface,and had normal motor function. Extensive previous surgery with altered anatomic landmarks maybeacontraindication to surgery.
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FIGURE 2 Fluoroscopic image of 15-blade anteriolateral stab wound into dorsal skin.
(A) (B)
(D)(C)
FIGURE 1 Percutaneous release of PIP extension contracture. ( A )Lateral fluoroscopy of previously injured PIP now with extension contracture. ( B )Fluoroscopically guided placement of 19-gauge needle into dorsal aspect of PIP joint. ( C )Lateral view of anteriolateral placement of 19-gauge needle. ( D )APview of anteriolaterally placed needle. Abbreviation:PIP, proximal interphalangeal.
Percutaneous Release of the Post-traumatic Finger Joint Contracture
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PERCUTANEOUS SURGICAL TECHNIQUE
The procedure can be performed under local, regional, or general anesthesia. Atourniquet is placed on the patient’s arm and inflated after the finger,hand, and forearm have been exsanguinated. Under fluoroscopic guidance, a19-gauge needle is placed into the PIP or MCP joint from an ante­riolateral direction at the joint line, volar to the joint capsule and extensor mechanism (proximal to the central slip of PIP joint) anddorsaltothe collateralligaments (Fig.1A–D). Ascalpel is used to incise the skin only (Fig. 2). The needle is removed and asmall hemostat snap is inserted and the skin is bluntly dissected to the dorsal joint capsule and with direct pressurethe joint capsule is penetrated under fluoro­scopic guidance (Fig. 3A,B). Using live fluoroscopy,the small curved hemostat position is confirmed in the dorsal joint under the capsule and extensor tendon (Fig. 3B). The hemo­stat is swept proximally rupturing the dorsal capsule and elevating theextensortendoncapsule andbreakingany adhesions from the dorsal aspect of the proximal phalanx or metacarpal head. The hemostat is then swept laterally between the collateral ligaments and the head of the proximal phalanx or metacarpal, breaking up any additional adhesions.
With flexion contractures, the hemostat can be swept volarly and used to break adhesions between the distal attachment of thevolar plateand thebaseofthe middlephalanx, as wellasthe proximal attachmentsofthe volarplate and checkreins to the volar head of the proximal phalanx. Care must be taken to glide the hemostat volarly close to bone avoiding theneurovascular structures.Withsevere scarring of the soft tissue, this maneuver is contraindicated becauseofthe potential for injury to theneurovascular structures.
Thejoint is then passivelyflexed andextendedunder imagingtodetermine theintraoperativegaininthe arc of motion and to confirm congruent gliding (Figs. 4A,B and 6). This percutaneous joint release can also be performed on the distal interphalangeal joint (DIP) joint in asimilar fashion since theDIP has similar anatomical structures to thePIP joint (Fig. 5A–C).
Figures1–6 show percutaneous release of both the PIP and DIP in a25-year-old male who had previously undergone a radioulnar ligamentous reconstructionwith bone anchors after adislocation injury.Hesubsequently developed an exten­sion contracture at his index finger and failed conservative management.
(A)
(B)
FIGURE3 Percutaneous dorsalcapsularreleasewith hemostat. ( A )Intraoperative view of small curved hemostatplaced percutaneously into dorsal PIP joint. ( B )Intraoperative lateral fluoroscopic view of small curved hemostat placed percutaneously into dorsal PIP joint to release dorsal capsular and extensor tendon adhesions.
(A)
(B)
FIGURE 4 ( A )Intraoperative view showing regain of full flexion at PIP joint after percutaneousrelease. ( B )Intraoperative lateral fluoroscopic view showing regain of 858 of flexion at PIP joint after percutaneous release.
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If extracapsular structures,i.e., tendonadhesions,are suspected to be an additional etiology of the contracture, they can be addressed at this time. Asingle 5-0 nylon interrupted sutureisused to close the PIP joint wound. The finger is placed in alight dressingand splinted into the corrected position.
Postoperatively,the patient is started on an immediate rehabilitation program to maintain the motion gained intra­operatively. Therapywillemploydigital wraps to control swellingand selective blocks areusedtocontrol pain. Narcoticsand NSAIDs areemployedpostoperatively, the former to control pain and the latter to reduce inflam­mation.Prior to surgery,arrangementfor postopera tive rehabilitation with askilled hand therapist is essential, as well as patient knowledge of and compliance with the post­operative program.
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RESULTS
Between 2003 and 2006, 60 patients weretreated with percuta­neous release of the MCP and PIP joints. Patients werestarted on an immediate hand therapyprogram postoperatively. The best results were observedinthe young with asingle pathological diagnosis. Those with crush injuries and scarring of multiple structures did the poorest and required multiple procedures. No complications were recorded including neuro­vascular injury,tendon disruption, or joint arthrosis.
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SUMMARY
In 1977, Harrison suggested that “adhesions between the collat­eral ligaments and the sides of the phalangeal head are amore important cause of stiffness than shortening of the collateral ligament itself” (18). Since then many papers have been written on the stiffPIP joint and implicated various pathoanatomical etiologies, most of which pertain to either the collateral ligaments or the checkrein ligaments (2,8,11,13–19). Stanley et al. described aseriesofpercutaneous releases of theaccessorycollateral ligament of the PIP joint (19 ). While their case number was small and follow-up limited, they reportedamean correction of 64%ofthe originalcontracture—si milartothe results of many open releasecasestudies (19).Theyrecognizedthat their procedurealsohad thebenefitofdividingadhesions between the collateral ligament and the phalangeal head.
We believe that intracapsular adhesions and fibrosis play a largerole in the post-traumatic MCP and PIP contracture, which is evident by the significant increase in motion postoperatively in our series. While this procedure does not address all the pathological components of aflexion contracture, the motion gained and the minimalinvasiveness of thepro cedure substantiate its use prior to subjecting the finger to the more invasive open procedures.
This technique has certain advantages over both open and external fixationprocedures.Inthe describedpercutaneous release, allofthe soft tissuestabilizersofthe jointremain intact,thus, avoiding thepotentialofjoint instability. The procedurecan be performed in amatter of minutes, enabling multiple fingers to be addressed at once and it can be used as an adjunct to other procedures such as tenolysis or fasciectomy.The healingtime is quicker than an open procedure, allowing a theoretical improved patient compliance with the postoperative rehabilitation. As opposed to externalfixators,which have approximately a40% infection rate (3), we had no incidence of postoperative infection. Patients also do not have to worry about pin loosening, external fixator care, or the prominence of the fixator, whichcan be cumbersomewith certain activities especially if dealing with multiple or non-border digits.
(A)
(B)
(C)
FIGURE 5 Percutaneous release of DIP joint extension contracture by same methoddescribed forPIP joint. ( A )Fluoroscopically guided placementof19-gauge needle into dorsal aspect of DIPjoint. ( B )Lateral view of anteriolateral placement of 19-gauge needle into DIP. ( C )Percutaneous placement of small curved hemostat into dorsal DIP capsule.
Percutaneous Release of the Post-traumatic Finger Joint Contracture
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SUMMATION POINTS
Indications
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Soft-tissue contractures of MP,PIP,and DIP joints
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Normal articular anatomy
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Joints that fail conservative treatment including therapy and splinting
Outcomes
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Excellent range of motion with less soft-tissue scarring
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Low complication rate
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Short surgical time allowing multiple digits to be treated
Complications
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Failure to achieve adequate release
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Tendon injury (theoretical)
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REFERENCES
1. SegalmanK.Surgical management of the stiffPIP joint. In: Proceedings of the 61st Annual Meeting of the American Society for Surgery of the Hand. Wa shington, DC: Omnipress, 2006.
2. Ghidella SD, Segalman KA, Murphey MS. Long-term resultsof surgical management of proximal interphalangealjoint contrac­ture.JHand Surg [Am] 2002; 27(5):799–805.
3. Houshian S, Gynning B, Schroder HA. Chronic flexion contracture of proximal interphalangeal joint treated with the compass hinge external fixator.Aconsecutive series of 27 cases. JHand Surg [Br] 2002; 27(4):356–8.
4. Gutow AP,Slade JFI, MahoneyJD. Phalangeal injuries. In: Thomas E, Trumble MD, eds. Hand Surgery Update 3. Rosemont, IL: American Society for Surgery of the Hand, 2003:3–28 (chap. 1).
5. Slade JFI, Choi J, Panjabi M, Wolfe S. The influenceofjoint position on fracturetype and soft tissue injuries of proximal interphalan­geal joint injuries.Orthop Tr ans 1997; 21(1):349.
6. Slade JFI, Choi J, Wo lfe S. Acadaveric model of the unstable fracture-dislocation of the proximal interphalangealjoint. Orthop Trans 1997; 21(1):120.
7. Slade JI, Baxamusa T, Wolfe S. External fixation of proximal interphalangeal joint fracturedislocations. In: Raskin KB, ed. Atlas of the Hand Clinics, March 2000, (5)1:1–29.
8. Curtis RM. Capsulectomy of the interphalangeal joints of the fingers. JBone Joint Surg 1954; 36-A(6):1219–32.
9. Shin A, Amadio P. Stifffinger joints. In: Green P, HotchkissWolfe, eds. Green’s Operative Hand Surgery.Philadelphia,PA: Elsevier, 2005:417–38 (chap. 11).
10. Hogan CJ, Nunley JA. Posttraumatic proximal interphalangeal joint flexion contractures. JAmAcad Orthop Surg 2006; 14(9):524–33.
11.Watson HK, Light TR,Johnson TR .Checkrein resection for flexion contractureofthe middle joint. JHand Surg [Am] 1979; 4(1):67–71.
12. Hume MC, Gellman H, McKellop H, Brumfield RH, Jr.Functional range of motion of the joints of the hand. JHand Surg [Am] 1990; 15(2):240–3.
13. Abbiati G, Delaria G, Saporiti E, Petrolati M, Tr emolada C. The treatment of chronic flexion contractures of the proximal inter­phalangeal joint. JHand Surg [Br] 1995; 20(3):385–9.
14. Bruser P, Poss T, Larkin G. Results of proximal interphalangeal joint release for flexion contractures: midlateral versus palmar incision. JHand Surg [Am] 1999; 24(2):288–94.
15. Curtis RM. Management of the stiffproximal interphalangeal joint. Hand 1969; 1:32–7.
16. Diao E, Eaton RG. Total collateral ligament excision for contrac­turesofthe proximal interphalangeal joint. JHand Surg[Am] 1993; 18(3):395–402.
17. Gould JS, Nicholson BG. Capsulectomyofthe metacarpophalan­geal and proximal interphalangeal joints. JHand Surg [Am] 1979; 4(5):482–6.
18. Harrison DH. The stiffproximal interphalangeal joint. Hand 1977; 9(2):102–8.
19. Stanley JK, Jones WA ,Lynch MC. Percutaneous accessory collateral ligament release in the treatment of proximal inter­phalangeal joint flexion contracture. JHand Surg [Br] 1986; 11(3):360–3.
FIGURE 6 Intraoperative fluoroscopy and photo showing 908 of flexion at the PIP and 608 at the DIP joints with flexor tendon tensioning after percutaneous release of both joints.
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Part IV:Minimally Invasive Procedures of the Carpus

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Percutaneous Scaphoid Fixation via aDorsal Technique
Joseph F. Slade III
Hand and Upper Extremity Service, Department of Orthopedics and Rehabilitation, Yale University School of Medicine,New Haven, Connecticut, U.S.A.
Greg Merrell
Department of Orthopedics, Brown University School of Medicine, Providence, Rhode Island, U.S.A.
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INDICATIONS
This technique is appropriate for any acute scaphoid fracture in thewaistorproximal pole. Many angular or translational displacements canbecorrected andrigidly fixed percuta­neously. Fracturesofthe distal pole may be more appropriately treated conservatively or with volar fixation to maximize screwpurchaseinthe distal fragment. We rec­ommend starting with nondisplaced fracturestoestablish the skill set needed for moredifficult displaced fractures.
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CHOICE AND POSITION OF IMPLANT
An increasing variety of compression screws are available for fixation of scaphoid fractures. Toby found resistance to cyclic loading was proportional to the radius of the screw to the fourth power ( r
4
)(1).Further,hefoundthe cannulatedAcutrak (Acumed, Beaverton, Oregon, U.S.A.) screw was the strongest headless compression screw, giving thehighest number of cycles to failure. The Herbert-Whipple(Zimmer, Wa rsaw, Indiana, U.S.A.), and AO (Synthes Corp., We st Chester,Penn­sylvania,U.S.A.) lagscrewsfailedcatastrophicallywith a resulting “windshield wiper” effect under conditions of volar comminution. The Acutrak screw underwent gradual separ­ation via plastic deformation rather than catastrophic failure. Trumble described the clinical importance of central axis screw placement (2).
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TECHNIQUE
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ASimple New Targeting Guide
We now use an external cross Kirschner (K) wire targeting guide. This simple technique permits external sighting of the central axis by percutaneously placed perpendicular K-wires. This system also decreases radiation exposure as imaging is used primarily to set up the initial targeting system. This guide requires the placement of two K-wires in the distal scaphoid in the same axial plane, one perpendicular to the scaphoid, and one offset in a90 8 arc. First awireisdriven dorsal to volar in the Posteroanterior (PA) plane of the distal scaphoid while ulnar deviating the wrist to extend the scaphoid (Fig. 1). Asecond wireisplaced in the lateral scaphoid-radial to ulnar (Fig. 2). These wires cross at the distal scaphoid central axis and form a crosshair target for guidewire placement (Fig. 3). With the wrist extended and ulna deviated, PA imaging of the dorsal scaphoid
wire if correctly perpendicular to the bone axis will appear as a single dark point. The lateral radial wire is introduced also perpendicular to the distal scaphoid and driven toward and across the dorsal wire as it appears on image as asingle point. Alateral fluoroscopic image will confirm that the lateral wire has been placed in the mid axis of the distal scaphoid.
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Technique for Displaced Fractures
After the “external cross K-wire scaphoid guide” is in place, aPAimage is obtained andthe fracture site is identified. Since the distal scaphoid fracture fragment is usually flexed exposing thedorsalintramedullarycanal of thescaphoid, aK-wire can be introduced into the fracture site and driven through the distal scaphoid intramedullary canal. The “external cross K-wire scaphoid guide” provides direction as the wire is driven from dorsal to volar.The proximal fragment at this time is irrelevant, and will later be reduced. The wire is withdrawn volarly until the trailing edge of the wire is at the fracture site. Now a0.062 K-wire joystick is placed in the proximal fragment, dorsal to volar.The wrist is placed in aneutral position and imaged, as the two dorsal joysticks (one in the distal fragment and one in the proximal scaphoid fragment) are manipulated until fracture alignment is obtained (Fig. 4). The lateral view of the concave scaphoid surface serves as the key reference for fracture reduction. Fracturereduction is secured by driving the volar wireretrograde across the fracture site.
Note if adorsal intercalated segment instability deformity is present due to the extreme scaphoid flexion at the fracture site, fracture reduction can be achieved by hyper flexion of the wrist until the lunate is in aneutral position and awire is driven through the distal radius into the lunate securing it provisionally in aneutral position. Or the wire can be placed dorsal directly into the lunate in aneutral position. As long as an intact scapholunate interosseous (SLIO) ligament exist the reduction force is transferred from the lunate to the scaphoid (Fig. 5).
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Technique for Nondisplaced Fractures
Once reductionand provisional fixation of thedisplaced fracture has occurred or in the case of anondisplaced fracture proceed as follows. With the wrist partially flexed amini­fluoroscopic unit is used to locate the tip of the proximal scaphoid pole, the starting point for the central axis scaphoid guidewire.Drive the central axis wire towardthe thumb base,
correcting its directionwith the use of the external K-wire targeting guides (the dorsal wire providesradial–ulnar guidance and the lateral K-wire provides dorsal–volar orien­tation). Acorrectly placed central axis scaphoid wire will hit the crossing wires in the distal scaphoid, the location of the
central axis. The wire is driven volarly past this intersection, through the trapezium, and exits at the thumb base in asafe zone without neurovascular structures.The wire is advanced volarly until the trailing edge crosses the radiocarpal joint and the wrist can be safely extended. In the case of adisplaced
FIGURE 1 First 0.06200targeting wire placed dorsal to volar in the distal scaphoid with the wrist in ulnar deviation to extend the scaphoid.
FIGURE 2 Second 0.06200targeting wire placed radial to ulnar in the mid-lateral position of the distal scaphoid.
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fracture thereare nowtwo wires down thelengthofthe scaphoid, one was used to capture the initial reduction and the second placed down the long axis (Fig. 6). The wire used to capturethe reduction also acts as an anti-rotation construct during scaphoid reaming and screw placement. For nondis­placed fracturesasecond wire is rarely needed.Imaging confirms the position of the wireand the scaphoid fracture reduction. If satisfactory,the next step is an arthroscopic inspection of the joint.
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ARTHROSCOPY
The goal of arthroscopy is to identify and treat ligament injuries, and directly inspect the quality of the reduction. The elbow is flexed and the wrist is positioned upright in aspring-scale
driven traction tower.Twelve pounds of traction is distributed between four finger traps to reduce the possibility of atraction injury.Ifthe finger traps slip off, apply mastisol or steristrips circumferentially at the baseofthe finger trap. The arm is exsanguinated. Afluoroscopy unit is placed horizontal to the floor and perpendicular to the wrist as the radiocarpal and midcarpal joint are identified with imaging. 19-gauge needles are introduced into the wrist joint identifying the radiocarpal and midcarpal portals. This maneuver minimizes iatrogenic injury to the joint.
The skin is incisedand ablunt hemostat is used to separate thesofttissueand enterthe wristjoint.Ablunt trocar is placed at the radial midcarpal portal and asmall jointangledarthroscopeisinserted. A19-gaugeneedleis inserted to establish outflow.Aprobe is introduced at the
FIGURE 3 Pronated,ulnarly deviatedview of the central axis with the targeting system guide wires in place.
FIGURE 4 Displaced scaphoid fracture with 0.06200joysticks in place prior to reduction, fluoroscopic image of
fracture reduction using joysticks, capturing the reduction with the central axis wire.
Percutaneous Scaphoid Fixation via aDorsal Technique
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ulnar midcarpalportaland subsequentlyatthethird and fourth portal to assess the competency of the carpal ligaments by directly stressing their attachments to detect partial and complete tears.
First identify the scapholunate ligament sulcus. Injuries to the scapholunateligamentdetectedare gradedusingthe Geissler grading system (3). Grade IandII ligament injuries are debrided. Grade III injuries are debrided and pinned for six weeks. Grade IV ligament injuries require openrepair of the dorsal SLIO ligament with bone anchors and carpal pinning. The need for the addition of adorsalcapsulodesis tether is determined by the quality of the acute repair after scaphoid fixation. Tea r s ofthetriangular fibrocartilage complexare classified usingthe Palmerclassificationandtreated accor­dingly (4).
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SCAPHOID LENGTH
The screw length should be 4 mm less than the scaphoid length. This permits 2mm ofclearanceat each end of the scaphoid, thus minimizing the risk of prominent hardware. The most common complication ofpercutaneousscaphoid fixation,is implantation of ascrewwhich is too long (5).
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FIXATION
Remove the extremity from the arthroscopy traction tower, flex the wrist and advance the wire retrograde until it is equally exposed on both ends. This prevents the wire from becoming dislodged during reaming. It is crucial that the wrist maintains aflexedposition topreventthe wire from bending. Dorsal placement is recommended for fractures of the proximal pole
FIGURE 5 Wire to control dorsal intercalated segment instability deformityof the lunate.
A
D
C
B
FIGURE 6 ( A )Wiretocontrollunateposition,(B )percutaneoussnapto assist with reduction, (C )wireusedtocapturereductionandnowserves as derotation wire, and ( D )centralaxis wire.
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and volar implantation is used for distal pole fractures. Frac­tures of the waist may be fixed from either adorsal or volar approach. Vo lar implantation often requires reaming through part of thetrapezium, sincethisisthe central axis.Blunt dissection along the guide wire exposes atract to the dorsal wrist capsule and the scaphoid base.
The scaphoid is reamed 2mmshort of opposite cortex with acannulated hand drill. Newer self-drilling screws have reduced the needfor extensive reaming, butthe scaphoid shouldstillbereamedpastthe fracture site to prevent gapping. It is critical to use fluoroscopy to check the position and depth during reaming.The scaphoid should never be reamedtothe opposite bone cortex (over-drilling). This reducesfracturecompression and increases theriskof motion at thefracturesite.AstandardAcutrak screw is advanced underfluoroscopic guidance down thecentral scaphoid axis to within 1to2mm of the opposite cortex. If the screw is advanced to the distal cortex, attempts to advance the screw further will force the fracture fragments to gap and separate.
With unstable or displaced fractures, acounter force is applied with the dorsal K-wire holding pressure against the proximal fragment to prevent gapping. Unstable fractures may not achieve rigid fixation with screw implantation alone. Other temporaryfixationmay be required,toachieve arigid
construct, until healinghas occurred. Thedistalscaphoid pole acts as along lever arm to the proximal scaphoid pole and proximal carpal row during wrist motion. Proximal pole fractureshave only afew threads crossing the fracture line. Wrist motion results in continuous rocking at the fracturesite. The forces concentrated here are significant and can result in reductionofcompression andloosening of fixation. These forces canbebalanced by theplacementofa0.062inch K-wire or headless compressionscrew from thescaphoid into thecapitate(Fig. 7).These instrumentstemporarily blocksmidcarpalmotionand reduce forces acting on the scaphoid fracture site. Another mechanical block is a0.062 inch K-wire placed between the II or III web-space into the capitate and the lunate. After healing has been confirmed with computed tomography (CT) scan,these mechanical blocks are removed percutaneously.
Severe comminution may make rigid fixation impossible. In thesecases,align the scaphoid fracturefragments with multiple K-wiresdownthe central axis,and stabilize the capitolunate jointwith K-wires. After,one month, these provisional fixation wiresare removed. Dorsal percutaneous bone grafting of thescaphoidand rigidfixationofthe scaphoid with aheadlesscompression screwcan then be accomplished.
FIGURE 7 Increasing fracture stability by mechanicalblock of scaphoid lever arm.
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