
- •Lesson 1 Methods for examination of a patient in skin diseases. Deontology in the practice of dermatologist and venereologist.
- •Application;
- •Palpation;
- •Lesson 2 Anatomy, histology and physiology of the normal skin. Histomorphological changes in the skin.
- •Nucleus;
- •Nucleus;
- •Nucleus;
- •Melatonin;
- •Langerhans cells;
- •Lesson 3 Morphology of primary and secondary skin lesions.
- •Ballooning degeneration;
- •Papule;
- •Erythema;
- •Lichenification;
- •Purpura;
- •Parakeratosis;
- •Purpura;
- •Lesson 4 Psoriasis. Lichen ruber planus. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Lesson 5 Scabies. Pediculosis. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •33 % Sulphur ointment;
- •33 % Sulphur ointment;
- •Vesicle;
- •Koebner's phenomenon;
- •Tubercle;
- •What are the routes of infection with scabies?
- •By droplet transmission;
- •Lesson 6 Diseases due to virus infection. Etiology. Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •Tubercle;
- •Corticosteroid ointments;
- •Macula;
- •Aniline dyes;
- •Lesson 7 Pyodermia. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Streptococcal impetigo;
- •Sycosis vulgaris;
- •Sycosis vulgaris;
- •Deep pustule not connected with appendages of the skin;
- •Pediculi pubis;
- •Lesson 8 Keratomycoses. Candidiasis. Clinical features. Diagnostics. Treatment. Prevention. Classification of mycoses. Laboratory diagnostics of mycoses
- •Candidiasis;
- •Psoriasis
- •Lichen ruber planus.
- •Sycosis vulgaris;
- •Candidiasis;
- •Antibiotics of a wide spectrum of action;
- •Staphylococcus;
- •Corynobacteria minutissimum;
- •Streptococcus.
- •Candidiasis;
- •Lesson 9 Dermatomycoses. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Candidiasis;
- •Pityrosporum orbiculare.
- •None of the above.
- •Lesson 10 Collagenoses. Lupus erythematosus. Sclerodermia. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Tubercles.
- •Erythema;
- •Atrophy;
- •Wickham's striae;
- •Koebner's phenomenon;
- •Erythema;
- •Lesson 11 Neurodermatoses. Atopic dermatitis. Prurigo. Urticaria. Etiology. Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •Eczema.
- •Lesson 12 Dermatitis. Toxicodermia. Etiology. Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •All listed above.
- •To prescribe antihistaminics;
- •To prescribe antihistaminics;
- •Wilkinson's ointment;
- •Erythema;
- •Erosions;
- •Lesson 13 Eczema. Etiology, Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •Vesicles;
- •Antibiotics.
- •All listed above.
- •Powders;
- •Pastes;
- •Erosion;
- •Ulcers.
- •Pastes;
- •Lesson 14 Bullous dermatoses. Erytema exudativum multiforme. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment.
- •Erosion;
- •Lichenification.
- •Solutions of aniline dyes;
- •Lotions;
- •Corticosteroid ointments;
- •Genitals;
- •Herpes simplex;
- •Herpes zoster;
- •Allergic dermatitis;
- •Lesson 18
- •Sulphonamides;
- •Antibiotics;
- •Epidermis;
- •Hypodermis;
- •Dermis.
- •Lesson 19 Primary period of syphilis.
- •Sulphonamides;
- •Antibiotics;
- •Ulcer. Lesson 20 Secondary period of syphilis.
- •Leucoderma;
- •Bullae;
- •Roseola;
- •Roseola;
- •Lichen ruber planus;
- •Psoriasis;
- •Lichen ruber planus.
- •Hard chancre;
- •Lesson 21 Tertiary period of syphilis. Congenital syphilis.
- •Conjunctivitis;
- •1 Year;
- •Tubercles;
- •Dense consistency;
- •Tubercles;
- •Vesicles.
- •Lesson 22 Laboratory diagnostics of venereal diseases. Treatment of syphilis.
- •All above-listed.
- •All above-listed.
- •All above-listed.
- •Candidiasis;
- •Lesson 23 Gonorrhoeal and non-gonorrhoeal urethritis in males. Treatment and prevention.
- •Sulphonamides.
- •Antibiotics;
- •All above-listed.
- •All above-listed.
- •Antibiotics;
Purpura;
Hemorrhages;
Roseola;
Erythema;
Leucoderma.
Which from the enumerate elements are the primary exudative lesions?
Pustule;
Vesicle;
Tubercle;
Bulla;
Papule.
A nodule differs from a tubercle:
In depth of placing;
By a color;
By the character of secretions;
By size;
By reverse development.
Scales can appear at such histopathological changes in an epidermis:
Parakeratosis;
Granulosis;
Hyperkeratosis;
Spongiosis;
Acanthosis.
Which from the enumerate elements are the primary non-inflammatory maculae?
Purpura;
Roseola;
Erythema;
Telangiectasias;
Leucoderma.
A wheal - it is:
Primary infiltrative lesions;
Primary exudative lesions;
Primary non-inflammatory infiltrative lesions;
Secondary lesions;
Primary lesions.
Which of the followings lesions terminates atrophy of skin?
Inflammatory spot;
Papule;
Tubercle;
Vesicle;
Nodule.
Monomorphism – it is when on a skin of a patient observed simultaneously:
Primary lesions of one size;
Primary lesions of one kind;
Only primary lesions;
Primary and secondary lesions;
Secondary lesions of one size.
True polymorphism – it is when on a skin of a patient observed simultaneously:
Primary lesions of different sizes;
Primary lesions of different kinds;
Primary and secondary lesions;
Secondary lesions of different sizes;
Secondary lesions of different kinds.
Vesicles can appear at such histopathological changes in an epidermis:
Acantholysis;
Spongiosis;
Acanthosis;
Ballooning degeneration;
Vacuolar degeneration.
When vesicles disappear they can leave such secondary lesions:
Erosions;
Ulcers;
Scars;
Atrophy;
Lichenification.
A bulla may be situated:
Intraepidermal;
Subcorneal;
Subepidermal;
Into a papillary layer;
Under subdermal layer.
When bulla disappear it can leave such secondary lesions:
Erosion;
Ulcer;
Scar;
Purulent crust;
Serous crust.
Spongiosis – it is:
Enlargement of papillary layer;
Intercellular edema of papillary layer;
Intercellular edema of prickle-cell layer;
Intracellular edema of prickle-cell layer;
Destruction of intercellular connections in prickle-cell layer.
Papillomatosis – it is:
Enlargement of papillary layer;
Intercellular edema of papillary layer;
Intercellular edema of prickle-cell layer;
Intracellular edema of prickle-cell layer;
Destruction of intercellular connections in prickle-cell layer.
The followings elements of rash have a cavity:
Papule;
Bulla;
Pustule;
Wheal;
Vesicle.
False polymorphism – it is when on a skin of a patient observed simultaneously:
Primary lesions of different sizes;
Primary lesions of different kinds;
Primary and secondary lesions;
Secondary lesions of different sizes;
Secondary lesions of different kinds.