
- •Lesson 1 Methods for examination of a patient in skin diseases. Deontology in the practice of dermatologist and venereologist.
- •Application;
- •Palpation;
- •Lesson 2 Anatomy, histology and physiology of the normal skin. Histomorphological changes in the skin.
- •Nucleus;
- •Nucleus;
- •Nucleus;
- •Melatonin;
- •Langerhans cells;
- •Lesson 3 Morphology of primary and secondary skin lesions.
- •Ballooning degeneration;
- •Papule;
- •Erythema;
- •Lichenification;
- •Purpura;
- •Parakeratosis;
- •Purpura;
- •Lesson 4 Psoriasis. Lichen ruber planus. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Lesson 5 Scabies. Pediculosis. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •33 % Sulphur ointment;
- •33 % Sulphur ointment;
- •Vesicle;
- •Koebner's phenomenon;
- •Tubercle;
- •What are the routes of infection with scabies?
- •By droplet transmission;
- •Lesson 6 Diseases due to virus infection. Etiology. Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •Tubercle;
- •Corticosteroid ointments;
- •Macula;
- •Aniline dyes;
- •Lesson 7 Pyodermia. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Streptococcal impetigo;
- •Sycosis vulgaris;
- •Sycosis vulgaris;
- •Deep pustule not connected with appendages of the skin;
- •Pediculi pubis;
- •Lesson 8 Keratomycoses. Candidiasis. Clinical features. Diagnostics. Treatment. Prevention. Classification of mycoses. Laboratory diagnostics of mycoses
- •Candidiasis;
- •Psoriasis
- •Lichen ruber planus.
- •Sycosis vulgaris;
- •Candidiasis;
- •Antibiotics of a wide spectrum of action;
- •Staphylococcus;
- •Corynobacteria minutissimum;
- •Streptococcus.
- •Candidiasis;
- •Lesson 9 Dermatomycoses. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Candidiasis;
- •Pityrosporum orbiculare.
- •None of the above.
- •Lesson 10 Collagenoses. Lupus erythematosus. Sclerodermia. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment. Prevention.
- •Tubercles.
- •Erythema;
- •Atrophy;
- •Wickham's striae;
- •Koebner's phenomenon;
- •Erythema;
- •Lesson 11 Neurodermatoses. Atopic dermatitis. Prurigo. Urticaria. Etiology. Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •Eczema.
- •Lesson 12 Dermatitis. Toxicodermia. Etiology. Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •All listed above.
- •To prescribe antihistaminics;
- •To prescribe antihistaminics;
- •Wilkinson's ointment;
- •Erythema;
- •Erosions;
- •Lesson 13 Eczema. Etiology, Pathogenesis. Classification. Clinical features. Diagnostics. Treatment. Prevention.
- •Vesicles;
- •Antibiotics.
- •All listed above.
- •Powders;
- •Pastes;
- •Erosion;
- •Ulcers.
- •Pastes;
- •Lesson 14 Bullous dermatoses. Erytema exudativum multiforme. Etiology. Pathogenesis. Clinical features. Diagnostics. Treatment.
- •Erosion;
- •Lichenification.
- •Solutions of aniline dyes;
- •Lotions;
- •Corticosteroid ointments;
- •Genitals;
- •Herpes simplex;
- •Herpes zoster;
- •Allergic dermatitis;
- •Lesson 18
- •Sulphonamides;
- •Antibiotics;
- •Epidermis;
- •Hypodermis;
- •Dermis.
- •Lesson 19 Primary period of syphilis.
- •Sulphonamides;
- •Antibiotics;
- •Ulcer. Lesson 20 Secondary period of syphilis.
- •Leucoderma;
- •Bullae;
- •Roseola;
- •Roseola;
- •Lichen ruber planus;
- •Psoriasis;
- •Lichen ruber planus.
- •Hard chancre;
- •Lesson 21 Tertiary period of syphilis. Congenital syphilis.
- •Conjunctivitis;
- •1 Year;
- •Tubercles;
- •Dense consistency;
- •Tubercles;
- •Vesicles.
- •Lesson 22 Laboratory diagnostics of venereal diseases. Treatment of syphilis.
- •All above-listed.
- •All above-listed.
- •All above-listed.
- •Candidiasis;
- •Lesson 23 Gonorrhoeal and non-gonorrhoeal urethritis in males. Treatment and prevention.
- •Sulphonamides.
- •Antibiotics;
- •All above-listed.
- •All above-listed.
- •Antibiotics;
What are characteristic signs of neurodermitis?
Itch;
Erythema;
Papulae;
Vesicles;
Crusts;
Scales;
Lichenification.
Atopic dermatitis quite often is combined with such diseases, except:
Hay fever;
Bronchial asthma;
Ulcerous illness;
Hives.
Atopic dermatitis can appear such lesions, except:
Ulcer;
Erythema;
Papula;
Erosion;
Pustule;
Scale;
Crust;
What are the most effective medical and preventive measures in atopic dermatitis:
Climatotherapy;
Hypoallergic diet;
Antibiotics;
Sulphonamides;
Vitamins.
In what layer of the skin primary morphological elements can appear in the hives?
Horny;
Prickle-cell layer;
Basal;
Papillary layer of the dermis;
Reticular;
Hypodermis.
Which primary morphological elements of rash are characteristic for hives?
Macula;
Erosion;
Excoriation;
Tubercle;
Wheal;
Bulla;
Nodule.
What zones may be distinguished in localized neurodermitis?
Zone with hyperpigmentation;
Zone with lichenification;
Zone with follicular hyperkeratosis;
Zone with isolated papules;
Zone with verrucosus proliferating.
The followings diseases are distinguished in the group of neurodermatoses, except:
Eczema;
Skin pruritus;
Hives;
Neurodermitis ;
Prurigo;
Lyell’s syndrome;
Atopic dermatitis.
Which morphological elements of rash can’t appear in patients with neurodermatoses?
Tubercle;
Ulcer;
Macula;
Wheal;
Bulla;
Papula;
Lichenification.
Which morphological elements are typical for prurigo:
Bulla;
Erosion;
Crust;
Papula;
Macula;
Tubercle;
Ulcer.
Select the form of preparation, which is recommended for treatment of patients with atopic dermatitis:
Varnish;
Plaster;
Powder;
Aerosol;
Cream;
Ointment.
Select foodstuffs that are not recommended for patient with atopic dermatitis:
Chocolate;
Eggs;
Milk;
Kefir;
Cottage cheese;
Apples;
Boiled meat;
Smoked sausage
Select diseases in which often we can examine white dermographism:
Hives;
Skin pruritus;
Idiopathic eczema;
Psoriasis;
Atopic dermatitis;
Toxicodermia.
Which are the most effective medical measures in hives:
Hypoallergic diet;
Ointment with corticosteroid;
Vitamins;
Antihistaminics.
Which from the following lesions is the most typical for the clinical picture of atopic dermatitis?
Wheal;
Vesicle;
Erythema;
Lichenification;
Nodule.
What lesions are characteristic for hives?
Papula;
Lichenification;
Macula;
Wheal;
Excoriation.
What external medicine is the most effective for treatment of atopic dermatitis?
Sol. acidi borici 2%;
Lorinden C;
Ung. acidi salicylici 2%;
Lotions with decoction of bur-marigold;
Shake lotions.
What are/is untypical localization of lesions in atopic dermatitis of adults?
Face;
Buttocks;
Neck;
Flexor surfaces of the cubital fossae;
Upper part of the chest.
What is the leading theory of etiopathogenesis of atopic dermatitis?
Neurogenic ;
Hereditary;
Viral;
Metabolic ;
Hormonal.
What foodstuffs should be eliminate from a ration of patients with atopic dermatitis?
Ketchup;
Apples;
Smoked sausage;
Chocolate;
Mustard;
Kefir.
In what age beginning of atopic dermatitis is improbable:
5 months;
5 years;
30 years;
50 years.
What is a frequent change in laboratory tests of patients with atopic dermatitis?
Increase of level Іg E antibodies in serum;
Detection of acantholytic cells;
Eosinophilia in blood;
Increase of C reactive albumen.
Chronic hives need to be differentiated from:
Scabies;
All allergic dermatitis;
Toxicodermia;
Eczema.
Exacerbations and remissions of atopic dermatitis depends on:
Seasons;
Dietetical mistakes;
Stress load;
Insolations.
What sings can we find out in patients with atopic dermatitis?
Polished nails;
Itch;
White dermographism;
Seasonal exacerbation.
Which from the following lesions is the most typical for the clinical picture of skin pruritus;
Papulae;
Wheals ;
Erosions;
Vesicles;
Excoriation.
Which from the following lesions is the most typical for the clinical picture of prurigo of adults:
Papula;
Nodule;
Wheals;
Lichenification;
Pustule.
What are initiating agents of exacerbation of strophulus in children?
Nervous factors;
Ecological factors;
Artificial feeding;
Home dust;
Medicines;
Food allergy.
The skin pruritus as concomitant symptom can be at such diseases:
Diabetes mellitus;
Helminth invasion;
Diseases of the liver;
Herpes zoster.
The most typical location of localized neurodermitis is:
Scalp;
Palms;
Soles;
Back surface of neck.
What histomorphological changes cause a clinical picture at patients with atopic dermatitis?
Spongiosis;
Acanthosis;
Parakeratosis;
Hyperkeratosis;
Papillomatosis;
Acantholysis;
Granulosis.
Which zones are selected in patients with localized neurodermitis?
Lichenification;
Vesicles;
Isolated papules;
Atrophy.
Individuals with atopic dermatitis are prone to develop:
Bacterial infections;
Viral infections;
Fungal infections;
Allergic rhinitis;
All of the above.
Urticaria can be caused by:
Penicillin;
Insect bite;
Infection;
Cheese;
All of the above.
People who have atopic dermatitis also may have:
Asthma;
Allergies;
Acne;
1) and 2).
Which group of people is more likely to develop atopic dermatitis?
Infants and young children;
Teenagers;
Adults 20 to 49;
Older adults.
One characteristic of atopic dermatitis is:
It affects the face more than the rest of the body;
It can leave pockmarks on the skin;
It cycles through periods of flares and remissions;
It is worse in autumn.
Which is a symptom of atopic dermatitis?
Bullae on the palms of hands and soles of feet;
Itchy, inflamed skin;
Scaly patches of skin on the scalp;
Coin-shaped patches of irritated skin on the arms and lower legs.
The skin of a person with atopic dermatitis is more susceptible to:
Skin infections;
Warts
Molluscum contagiosum;
Herpes simplex;
All of the above.