266 Литература

ton D. J. (1985). Genetic polymorphisms of apolipoprotein C-III and insulin in survivors of myocardial infarction, Lancet, 2, 300303.

661. Fialkow P.J., Giblett E.R., Motulsky A.G. (1967). Measurable linkage between ocular albinism and Xg, Am. J. Hum. Genet, 19, 63-69.

662. FinkelsteinS., Walford R.L., Myers L. W., Ellison G. W. (1974). HL-A antigens and hypersensitivity to brain tissue in multiple sclerosis, Lancet, I, 736.

663. Finney D.J. (1947/9). The truncated binomial distribution, Ann. Eugen., 14, 319-328.

664. Fischer R.A. (1918). The correlation between relatives on the supposition of Mendelian inheritance, Trans. R. Soc. (Edinburgh), 52, 399-433.

665. Fischer R.A., Race R.R. (1946). Rh gene frequencies in Britain, Nature, 157, 8.

666. Flodents Y., Iselius L., Lindsten J., Wetterburg L. (1982). Evidence for a major locus as well as a multifactorial component in the regulation of human red blood cell catechol0-methyltransferase activity, Hum Hered., 32, 76-79.

667. Folstein S. K, Phillips J. A. Ill, Meyers D. A., Chase G. A., Abbott M.H., Franz M.L., Waber P. G., Kazazian H. H. Jr., Conneally P. M., Hobbs W., Tanzi R., Faryniarz A., Gibbons K., Gussella J. (1985). Huntingdon's disease: two families with differing clinical features show linkage to the G8 probe, Science, 229, 776-779.

668. Franceschetti A., Klein D. (1957). Two families with parents of different types of red-green blindness, Acta Genet. (Basel), 7, 255.

669. Fraser G.R. (1976). The cause of profound deafness in childhood, Johns Hopkins University Press, Baltimore, London.

670. Fraser G.R., Friedman A.I. (1967). The causes of blindness in с chindhood, Johns Hopkins University Press, Baltimore.

671. Fredrickson D.S., Goldstein J.L., Brown M.S. (1978). The familian hyperlipoproteinemias. In: Stanbury J. В., Wyngaarden J. В., Fredrickson D. S. (eds.), The Metabolic Basis of Inherited Disease, 4th ed., McGraw-Hill, New York, pp. 604-655.

672. Fuhrmann W. (1963). Das Syndrom der erblichen Nephropathie mit Innenohrschwerhorigkeit (Alport-Syndrom), Dtsch. Med, Wochenschr., 88, 525-532.

673. Fuhrmann W. (1974). Die formale Genetik des Menschen. In: Vogel F. (ed.), Handbuch der Allgemeinen Pathologie, Vol. IX, Erbgefüge, Springer, Berlin, Heidelberg, New York, pp. 147-259.

674. Fuhrmann W, Stahl A., Schroeder T. M. (1966). Das oro-facio-digitale Syndrom, Humangenetik, 2, 133-164.

675. Galton F. (1876). The history of twins as a critérium of the relative powers of nature, J. Anthropol. Inst.

676. Gartler S. M., Francke U. (1975). Half chroma-

tid mutations: Transmission in humans? Am. J. Hum. Genet, 27, 218-223.

677. GaulL.E. (1953). Heredity of multiple benign cystic epithelioma, Arch. Dermatol. Syph. (Chicago), 68, 517.

678. Gelb A. F., Klein E., Lieberman J. (1977). Pulmonary function in nonsmoking subjects with alpha! antitrypsin deficiency (MZ phenotype), Am. J. Med., 62, 93.

679. Gerard G., Vitrac D., Le Pendu J., Muller Α., Oriol R. (1982). Η-deficient blood groups (Bombay) of Reunion island, Amer. J. Hum. Genet, 34, 937-947.

680. Gesell Α., Thompson H. (1929). Learning and growth in identical infant twins: An experimental study by the method of co-twin control, Genet. Psychol. Monogr., 6, 5-124.

681. Gey er E. (1940). Ein Zwillingspärchen mit zwei Vätern. Arch. Rassenbiol., 34, 226-236.

682. Giblett E. R., Klebanoff S. J., Pincus S. H., Swanson J., Park B. H., McCullough J. (1971). Kell phenotypes in chronic granulomatous disease: a potential transfusion hazard, Lancet, I, 1235.

683. Goldberg A.S. (1978). Pitfalls in the resolution of I. Q. inheritance, pp. 195-222, In: Morton N. E., Chung C. S. (eds.), Genetic epidemiology, Academic Press, New York etc.

684. Goldstein J.L., Brown M.S. (1977). Atherosclerosis: thelow-density lipoprotein receptor hypothesis, Metabolism, 26, 1257-1275.

685. Goldstein J.L., Brown M.S. (1979). LDL receptor defectin familial hypercholesterolemia, Med. Clin. North Am., 66, 335-362.

686. Goldstein J.L., HazzardW.R., Schrott H. G., Bierman E. L., Motulsky A. G. (1973). Hyperlipidemia in coronary heart disease. II. Genetic analysis of lipid levels in 176 families and delineation of a new inherited disorder, J. Clin Invest, 54, 1544-1568.

687. Grebe H. (1959). Erblicher Zwergwuchs, Ergeb. Inn. Med. Kinderheilkd., 12, 343-427.

688. Greiner J., Schleiermacher E., Smith T., Lenhard V., Vogel F. (1978). The HLA system and leprosy in Thailand, Hum. Genet., 42, 201-213.

688a. Greiner J., Kruger J., Palden L., Jung E. G., Vogel F. (1983). A linkage study of acrokeratoelastoidosis, Possible mapping to chromosome 2, Hum. Genet, 63, 222-227.

689. Grosse-Wilde H., Bertrams J., Schuppten W., Netzel B., Kuppelt W., Kuwert E.K. (1977). HLA-D typing in 111 multiple sclerosis patients: Distribution of four HLA-D alleles, Immunogenetics, 4, 481-488.

690. Gruneberg H. (1952). Quasi-continuous variations in the mouse, Symp. Genet, 3, 215-227.

691. Gruneberg H. (1952). Genetical studies in the skeleton of the mouse. IV. Quasi-continuous variations, J. Genet, 51, 95-114.

692. Girzeschik K.-H. (1973). Utilization of mosaic cellhypbrids for genetic studies in man, Hum. Genet, 19, 1-40.

693. Gusella J. F., Tanzi R. E., Anderson Μ. Α., Hobbs W., Gibbons K., Raschtchian R., Gilli-

Литература 267

am Т.С., Wallace M. R., Wexler N. S., Conneally P. M. (1984). DNA markers for nervous system diseases, Science, 225, 1320-1326.

694. Gusella J. F., Wexler N. S., Conneally P.M., Naylor S. L., Anderson M. Α., Tanzi R. E., Watkins P. С., Ottina С., Wallace M. R., Sakaguchi A. Y., Young А. В., Shoulson I., BonillaE., Martin J.B. (1983). A polymorphic DNA marker genetically linked to Huntington's disease, Nature, 306, 234-238.

695. HaaseF.H. (1938/39). Die Übersterblichkeit der Knaben als Folge recessiver geschlechtsgebundener Anlagen, Z. Menschl. Vererbungs-Konstitutionslehre, 22, 105-126.

696. Hadorn E. (1955). Developmental genetics and lethal factors, John Wiley and Sons, London, New York.

697. Haldane J.B.S. (1941). The relative importance of principal and modifying genes in determining some human diseases, J. Genet, 41, 149-157.

698. Haldane J.B.S., Smith C. A.B. (1947/9). A simple exact test for birth order effect, Ann. Eugen., 14, 116-122.

699. Haldane J.B.S., Smith C.A.B. (1947). A new estimate of the linkagebetween the genes for color blindness and haemophilia in man, Ann. Eugen., 14, 10-31.

700. Hall J. G., Dorst J. P., Taybi H., Scott C. I., Langer L. О., McKusick V. A. (1969). Two probable cases of homozygosity for the achondroplasia gene, Birth Defects, 4, 24-34.

701. Hornby R.I. (1981). Hereditary aspects of coronary artery disease, Am. Heart. J., 101, 639-649.

702. Harris H. (1970). Cell fusion, Clarendon, Oxford.

702a. Harris H. (1948). A sex-limiting modifying gene in diaphyseal aclasis (multiple exostoses), Ann. Eugen, 14, 165-170.

703. Harris H., Smith C.A.B. (1948). The sib-sib age of onset correlation among individuals suffering from a hereditary syndrome produced by more than one gene, Ann. Eugen, 14, 309-318.

704. HatzoldO. (1966). Die Sexualproportion der Geborenen und ihre Schwankungen als präkonzeptionelles Wahrscheinlichkeitsproblem, Dtsch. Akad. f Bevölkerungswissenschaft, Reihe B., Vol. 5.

705. Hauge M., Harvald B., Fischer M., GottliebJensen K., Juel: nielsen N., Raebild I., Shapiro R., Videbech T. (1968). The Danish twin register, Aota Genet. Med. Gemellol (Roma), 18, 315-332.

706. Havel R. J. (1982). Familial dysbetalipoproteinemia, Med. Clin. North Am., 66, 441-454.

707. Haverkamp-Begemann N., Lookeren-Campagne van A (1952). Homozygous from of Feiger Huet's nuclear anomaly in man, Acta Haematol (Basel), 7, 295-303.

708. Haws D. V., McKusick V. A. (1963). Farabee's brachydactylous kindred revisited, Johns Hopkins Med. J., 113, 20-30.

709. Hegemon J.P., Mash A.J., Spivey B.E. (1974). Genetic analysis of human visual parameters in populations with varying incidence of strabism, Am. J. Hum. Genet., 26, 549-562.

710. Helmbold W. (1959). Über den möglichen Aufbau des Rh-Genkomplexes, Blut, 5, 141-148.

711. Helmbold W., Prokop 0. (1958). Die Bestimmung der ABO-Genfrequenzen mittels der maximum-likelihood-Methode und anderer Verfahren anhand forensischer Blutgruppenbestimmungen in Berlin, Blut, 4, 190-201.

712. Hershon K., Brunzell J., Albers J.J., Haas L., Motulsky A. (1981). Hyper-apo-B-lipoproteinemia with variable lipid phenotype (familial combined hyperlipidernia). Arterosclerosis, l, 380a.

712a. Hirszfeld L. (1928). Konstitutionsserologie und Blutgruppenforschung, Springer, Berlin.

713. Hitman G. Α., Jowett N. I., Williams L. G., Humphries S., Winter R. M., Galton D. J. (1984). Polymorphism in the 5'-flamking region of the insulin gene and non-insulindependent diabetes, Clin. Sei., 66, 383-388.

714. Hitzig W. H., Seger R. A. (1983). Chronic granulanatous disease, a heterogenous syndrome, Hum. Genet., 64, 207-215.

715. Holmes В., Quie P.G., Windhorst D. В., Good R. А. (1966). Fatal granulomatous disease of childhood: an inborn abnormality of phagocytic function, Lancet, I, 1225.

716. Howell-Evans W., McConnel R.B., Clarke C.A., SheppardP.M. (1958). Carcinoma of the oesophagus with keratosis palmaris and plantaris (tylosis), Q.J. Med. (new series), 27, 413-429.

717. HrubecZ., Robinette C.D. (1984). The study of human twins in medical research, N. Engl. J., Med., 310, 435-441.

718. Hully S.B., Rosenman R.H., Bawol R.D., Brand R. J. (1980). Epidemiology as a guide to clinical decisions. The association between triglycéride and coronary heart disease, N. Engl. J. Med., 302, 1383-1389.

719. Humphries S.E., Kessling A.M., Horsthemke В., Donald J. Α., Seed M., Jowett N.. Holm M., Galton D.J., Wynn V., Williamson R. (1985). A common DNA polymorphism of the low-density lipoprotein (LDL) receptor gene and its use in diagnosis, Lancet, 1, 10031005.

720. Hülfen M. (1974). Chiasma distribution at diakinesis in the normal human male, Hereditas, 76, 55-78.

721. Husén T. (1959). Psychological twin research, Almquist and Wiksell, Stockholm.

722. Illingworth D. R., Sexton G. J. (1984). Hypocholesterolemic effects of mevinolin in patients with heterozygous familial hyperholesterolemia, J. Clin. Invest., 74, 1972-1978.

723. Inkeles S., Eisenberg D. (1981). Hyperlipidernia and coronary atherosclerosis: a review, Medicine, 60, 110-123.

724. IseliusL., LalouelJ.M. (1982). Complex segregation analysis of hyperalphalipoproteinemia, Metabolism, 31, 521-523.

268 Литература

725. Jackson J. l·'., Currier R. D., Terasaki P. L, M or ton N. E. (1977). Spinocerebellar ataxia and HLA linkage. Risk prediction by HLA typing, N. Engl. J. Med., 296, 1138-1141.

726. Johannsen W. (1909). Elemente der exakten Erblichkeitslehre, G. Fischer (3rd ed., 1926), Jena.

727. Johnson B. C., Epstein F. H., Kjelsberg M. O. (1965). Distribution and familial studies of blood pressure and serum cholesterol levels in a total community - Tecumseh, Michigan, J. Chron. Dis., 18, 147.

728. Jörgensen G. (1974). Erbfaktoren bie häufigen Krankheiten. In: Vogel F. (ed.), Handbuch der allgemeinen Pathologie, Vol. IX, Erbgefuge, Springer, Berlin, Heidelberg, New York, pp. 581 665.

729. Kaelin A. (1955). Statistische Prüf-und Schätzverfahren für die relative haufigkeit von Merkmalsträgern bei einem der Auslese unterworfenen Merkmal mit Anwendung auf das Retinagliom, Arch. Julius Klaus Stiftung Vererbungsforsch., 30, 263-485.

729a. Kampe O., Larhammer D., Wiwan K., Scheming L., Claesson L.,Gustafsson K., Paabo S., Hyldig - Nielsen J. J., Rask L., Peterson P. A. (1983). Molecular analysis of MHC antigens. In: Möller E., Möller G. (eds.), Genetics of the immune response, Plenum, 001 York, pp. 61 -79.

730. Кадет Α., Rhoads G. С., Zeegan P. D., Nichaman M. Z. (1971). Coronary heart disease among men of Japanese ancestry in Hawaii: the Honolulu heart study, Isr. J. Med., Sei., 7, 1573.

731. KallmannF.J. (1938). The genetics of schizophrenia, Augustin, New York.

732. KallmannF.J. (1946). The genetic theory of schizophrenia. An analysis of 691 schizophrénie twin index families, Am. J. Psychiatry, 103, 3.

733. Kan Y. W., Dozy A. M. (1978). Antenatal diagnosis of sicklecell anaemia by DNA analysis of amnioticfluid cells, Lancet, II, 910-912.

734. Kanneil W.B. (1976). Some lessons in cardiovascular epidemiolog from Framingham, Am. J. Cardiol, 37, 268-282.

735. KeatsB.J.B., Morton N. E., Rao D.C., Williams W. (1979). A sourse book for linkage in man, Johns Hopkins Univ. Press, Baltimore.

736. Keats J. В., Morton N. E., Rao D. C. (1981). Reduction of physical assigment to a standard lod table: Chromosome 1, Hum. Genet., 56, 353 359.

737. Kelley W.N., Greene M.L., Rosenbloom P.M., Henderson J.F., Seeqmiller J.E. (1969). Hypoxanthine-guanine phosphoribosyltranspherase deficiency in guut, Ann. Intern. Med., 70, 155.

IIS. Kiekebusch-Müller B.D., Arnold H., Mayr W. R. (1980). Ein Algorithmus zur Berechnung der HLA-Haplotypfrequenzen (an algorhithm for calculating HLA haplotype frequencies), Anthrop. Anzeiger, 38, 1-10.

739. Kissmeyer-Nielsen F. (ed.) (1975). Histocom-

patibility testing 1975, Munkagaard, Copenhagen.

740. Klein J. (1974). Genetic polymorphism of the histocompatibility 2 loci of the mouse, Ann. Rev. Genet., 8, 63-77.

741. Klein J. (1975). Biology of the mouse histocompatibility-2 complex, Springer, Berlin, Heidelberg, New York.

742. Kloimwieder R. (1942). Die Intelligenz in ihren Beziehungen zur Vererbung, Umwelt Übung, Z. Menschl. Vererbungs-Konstitutionslehre, 25, 582-617.

743. Knowler W. C., Pettit D.J., Vasquez B., Rotwein P. S. Andreone T. L., Permutt M. A. (1984). Polymorphism in the У flanking region of the human insulin gene, J. Clin. Invest., 74, 2129-2135.

744. Koller S. (1940). Methodik der menschlichen Erbforschung II. Die Erbstatistik in der Familie. In: Just G., Bauer K. H., Hanhart E., Lange J. (eds.), Handbuch der Erbbiologie des Menschen, Vol. II, Methodik, Genetik der Gesamtperson, Springer, Berlin, pp. 261284.

745. Kravitz K., Skolnick M., Edwards C., Cartwright G., Amos B., Carmelli D., Baty B. (1978). Pedigree analysis of the linkage between HLA and hemochromatosis. In: Morton N. E., Chung C. S. (eds.), Genetic epidemiology, Academic Press, New York, pp. 241-246.

746. Krüger J. (1973). Zur Unterscheidung zwischen multifaktoriellen Erbgang mit Schwellenwerteffekt und einfachen diallem Erbgang, Hum. Genet., 17, 181-252.

747. Krüger J., Propping P. (1976). Rückgang der Zwillingsgeburten in Deutschland, Dtsch. Med. Wochenschr., 101, 475-480.

748. Kruger J., Vogel F. (1975). Population genetics of unequal cros sing over, J. Mol. Evol, 4, 201—247

749. Kueppers F. (1975). сц-Antitrypsin. In: Becker P. E. (ed.), Humangenetik, ein kurzes Handbuch, Vol. 1/3, Thieme, Stuttgart, pp. 35-49.

750. Kunkel L. M., Tantravahi U., Eisenhard M., Latt S. (1978). Regional localization on the human X of DNA segments cloned from flow sorted chromosomes, Nucleic Acid Res., 10, 1557-1578.

751. Kurachi K., Chandra T., Degen S. J., White T. T., Marchioro T.L., Woo S. L. С., Davie E. W., (1981). Cloning and sequence of cDNA coding for alpha-antitrypsin, Proc. Natl. Acad. Sei. USA, 78, 6826-6830.

752. Lalouel J. M., Morton N. E., MacLean C. J., Jackson J. (1977). Recurrence risk in complex inheritance with special regard to pyloric stenosis, J. Med. Genet., 14, 408-414.

753. Landsteiner K., Wiener A.S. (1940). An agglutinable factor in human blood recognized by immune sera for rhesus blood, Proc. Soc. Exp. Biol. Med., 43, 223.

754. Lange К., Boehnke M. (1982). How many polymorphic genes will it take to span the

Литература 269

human genome? Amer. J. Hum. Genet., 34, 842-845.

754a. Lange K., Page B. M., Eiston R. С. (1975). Age trends in human hiasma frequencies and recombination fractions. I. Chiasma frequencies, Am. J. Hum. Genet, 27, 410-418.

755. Laurell C.-B., Eriksson S. (1963). The electrophoretic a_t- globulin pattern of serum 04antitrypsin deficiency, Scand. J. Clin. Lab. Invest., 15, 132.

756. Lemser H. (1938). Zur Erb- und Rassenpathologie des Diabetes mellitus, Arch. Rassenbiol., 32, 481.

757. Lenz F. (1923). Ubersterblichkeit der Knaben im Lichte der Erbhchkcitslehre, Arch. Hyg., 93, 126-150.

757a. Lenmark A. (1985). Molecular biology of type 1 (insulin-dependent) diabetes mellitus, Diabetes, 28, 195-203.

758. Lenz W. (1959). Ursachen des gesteigerten Wachstums der heutigen Jugend. In: Akzeleration und Ernährung Fettlösliche Wirkstoffe, Vol. 4.

759. Lenz W. (1961). Zur Genetik der Incontinentia pigmenti, Ann. Paediatr. (Basel), 196, 141.

760. Lenz W. (1975). Half chromatid mutations may explain incontinentia pigmenti in males, Am. J. Hum. Genet, 27, 690-691.

761. Lenz W. (1973). Vererbung und Umwelt bie der Entstehung von Missbildungen, Uumanbiologie, 121, 132-145.

762. Levine P., Stetson R. E. (1939). An unusual case of intragroup agglutination, J. Am. Med. Assoc., 113, 126-127.

763. Levy R. I. (1981). Declining mortality in coronary heart disease, Arteriosclerosis, 1, 312-325.

764. Lewis E. B. (1951). Pseudoallelism and gene evolution, Cold Spring Harbor Symp. Quant. Biol., 16, 159-174.

765. Lieberman R., Humphrey W., (1972). Association of the H-2 type with genetic control of immune responsiveness to (y2a) allotypes in the mouse, J. Exp. Med., 136, 1222-1230.

766. Lilly F. (1966). The inheritance of susceptibility to the Gross leukemia virus in mice, Genetics, 53, 529-539.

767. Lundsgaard R. (1944). Leber's disease. A généalogie, genetic and clinical study of 101 cases of retrobulbar optic neuritis in 20 Danish families, Acta Ophthalmol. /Suppl/ (Kbh.), 30.

768. Luxemburger H. (1935). Untersuchungen an schizophrenen Zwillingen und ihren Geschwistern zur Prüfung der Realität von Manifestationsschwankungen, Z. Gesamte Neurol. Psychiat, 154, 351-394.

769. Luxenburger H. (1940). Zwillingsforschung als Methode der Erbforschung beim Menschen. In: Just G., Bauer K. H., Hanhart E., Lange J. (eds.), Handbuch der Erbbiologie des Menschen, Vol. II, Methodik, Genetik der Gesamtperson, Springer, Berlin, pp. 213-248.

770. Maartmenn-Moe K., Magnus P., Golden W., Berg K. (1981). Genetics of the low density

lipoprotein receptor: III. Evidence for multiple normal alleles at the low density lipoprotein receptor locus, Clin. Genet., 20, 113-129.

771. Maartmann-Moe K., Magnus P., Borresen A.-L., Berg K. (1981). Low density lipoprotein receptor activity in cultured fibroblasts from subjects with or without ischémie heart disease (in the ab sence of familial hypercholesterolemia), Clin. Genet, 20, 337-346.

772. Madiener M. (1928). Eine Bluterfamilie, Arch. Rassenbiol., 20, 390-394.

773. Mann J. D., Cahan A., Gelb A., Fischer N., Hamper J., Tipett P., Sanger R., Race R.R. (1962). A sex-linked blood group, Lancet, I, 8.

774. Maroteaux P. (1974). Les maladies esseuses de l'enfant, Flammarion, Paris.

775. Marsh D.G., Meyers D.Α., Bias W.B. (1981). The epidemiology and genetics of atopic allergy, New Engl. J. Med., 305, 1551-1559.

776. Marsh W.L. (1978). Chronic granulomatous disease, Kx antigen and the Kell blood groups. In: Brewer G. J. (ed.), Progress in clinical and biological research. The red cell, Vol. 21, Liss, New York, pp. 493-507.

777. Marsh W.L., Oyen R., Nichols M. K, Allen F. H . (1975). Chronic granulomatous dosease and the Kell blood groups, Br. J. Haematol., 29, 247.

778. Massart С., Bussen M., Deschamps I., Hors J., LalouelJ.-M., Dausset J., Cohen D. (1985). A systematic study of HLA class II-ß DNA restriction fragments in insulin-dependent diabetes mellitus, Proc. Natl. Acad. Sei. USA, 82, 3335-3339.

779. Matthiuz P. L., Ihde D., Piazza A., Ceppelini R., Bodmer W.F. (1970). New Approaches to the population genetics and segregation analysis of the HL-A system. In: Terasaki P. (ed.), Histocompatibility Testing, Munksgaard, Kopenhagen, pp. 193-205.

780. Maynard-Smith S., Penrose L. S., Smith С. А. В. (1961). Mathematical tables for research workers in human genetics, J and A Churchill, London.

781. McArthur N. (1953). Statistics in twin birth in Italy, 1949 and 1950, Ann. Eugen, 17, 249.

782. UcClintock B. (1944). The relation of homozygous deficiencies to mutations and allelic series in Maize, Genetics, 29, 478-502.

783. McDevitt H. O. et al. (1972). Genetic control of the immune response, mapping of the Ir-1 locus, J. Exp. Med., 135, 1259-1278.

784. McKusick V.A. (1984). The human gene map 15 november 1983. The morbid anatomy of the human genome, Clin. Genet, 25, 89-123.

785. McMichael A., McDewitt H. (1977). The association between the HLA system and disease, Prog. Med. Genet. (New Series), 2, 39100.

786. Menzel H.-J., Kladetzky R.-G., Assmann G. (1983). Apolipoprotein E polymorphism and coronary artery disease, Arteriosclerosis, 3, 310-315.

787. MétnekiJ., Czeizel A. (1980). Contraceptive