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11 Anatomy oftheColon: Rectum toIleocaecal Valve
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(i) (ii)
Fig. 11.8 Ascending colon (i) 3D view of a triangular fold in ascending colon (arrows); (ii) 2D coronal view showing ascending colon (AC). Green arrow=appendix
11.3.8 Ileocaecal Valve (ICV)
It is easy to identify the ICV valve because its position is constant relative to the terminal ileum and caecum. It demarcates the caecum from the ascending colon. The appearance of an ICV var­ies from a labial type with a slit-like elongated appearance to a more bulbous polypoidal or pap­illary type (Fig.11.9a (i) and (ii)). A bulbous or papillary ICV causes a prominent polypoidal appearance with a central depression. A specic feature of the ICV is a depression or ‘pit’ orice [3] where the terminal ileum empties into the
right colon. This orice may be visualised on both 2D and 3D views as shown in Fig.11.9a (iii). An ICV on a CTC study may be open (pat­ent) or closed. Figure 11.9b demonstrates a closed IVC. If it is open, then reux of carbon dioxide may occur (Fig.11.9c (i)–(iv)). The ICV is located postero-medially where the terminal ileum enters the caecum. An ICV may be com­pletely replaced with fat (Fig.11.9d (i) and (ii)). It may have a high intensity (red) on translucent display (TD) as shown in Fig.11.9 (iii). Polyps or adenocarcinoma may occur on the surface of the ICV because it is covered by mucosa.
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a(i) a(ii)
a(iii) b(i)
J. H. Bortz
b(ii)
Fig. 11.9 Ileocaecal valve. (a) (i) 3D view shows labial ICV (open black arrow); (ii) 3D view of a bulbous ileo­caecal valve (black arrow). Appendiceal orice (circle); (iii) 3D view of ICV showing depression or ‘pit’ orice (open black arrow) where the terminal ileum empties into right colon. Closed arrow= triangular folds. (b) (i) 2D
c(i)
coronal view of a closed ICV.Terminal ileum (TI); cae­cum (C); ascending colon (AC); descending colon (DC); (ii) 2D axial view of a closed ICV (red circle). (c) (i) 3D view showing patent ICV (closed black arrow) and trian­gular folds (open black arrows);
11 Anatomy oftheColon: Rectum toIleocaecal Valve
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c(ii) c(iii)
c(iv) c(v)
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Fig. 11.9
arrow) with air in the terminal ileum (TI). Caecum (C); descending colon (DC); (iii) 2D axial supine view show­ing patent ICV (green circle); (iv) Colon- map showing
(ii) 2D coronal view of a patent ICV (white
reux of gas into small bowel (SB grey) due to patent ICV (blue arrow); (v) 2D coronal view showing small bowel valvulae conniventes (open white arrows) and gas in stomach (S).
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d(i) d(ii)
d(iii)
J. H. Bortz
Fig. 11.9
nantly fatty ICV (green, open black arrow); (iii) TD shows high intensity ICV (red, open white arrow)
(d) (i) 2D soft tissue axial view of a fatty ICV (white arrows); (ii) TD (translucent display) shows predomi-
11.3.9 Caecum
This colon segment is proximal to the ICV; its conguration and position may change as shown in Fig.11.10(i) and (ii). This occurs because 10%
of people have no peritoneal xation of the ascending colon thereby allowing for caecal mobility (Fig.11.10 (iii) and (iv)). The caecum is more capacious than the ascending colon.
11 Anatomy oftheColon: Rectum toIleocaecal Valve
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(i) (ii)
(iii) (iv)
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Fig. 11.10 Caecum (i) 3D view of ICV (closed black arrow), caecum, proximal ascending colon (AC), and appendiceal orice (open black arrow); (ii) 2D coronal view: ascending colon (AC); ileocaecal valve (ICV); cae­cum (C); descending colon (DC); (iii) Supine colon-map
11.3.10 Appendix
The vermiform appendix is part of the caecum. Its length varies from 2.5 to 33cm [11]. Its aver­age length is between 5 and 10 cm: its base is usually situated 2 cm below the ICV. Its intra­abdominal position may vary widely depending on the peritoneal fold which represents the mes­entery of the appendix [11, 12]. The convergence of the three taeniae coli in the caecum form two
showing abnormal position of caecum (C) below the TC. Rectum (R); sigmoid colon (S); descending colon (DC); transverse colon (TC); ascending colon (AC); (iv) Prone colon-map shows normal position of caecum (C) indicating mobility with postural change. Rectum (R)
prominent folds called the crow’s feet that ank the appendiceal orice and is shown on the 3D endoluminal view (Fig. 11.11a (i)) [3]. Figure11.11a (ii) and (iii) demonstrate the ori­ce of the appendix and appendiceal lumen. Figure11.11b (i)–d (iv) are a range of 2D and 3D images of the appendix in various locations in the abdomen. Figure 11.11e (i) and (ii) show the appendix in the inguinal canal.
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a(i) a(ii)
a(iii) b(i)
J. H. Bortz
Fig. 11.11 Appendix. (a) (i) 3D view of appendiceal ori- ce (open black arrow) and crow’s feet (closed black arrows); (ii) 3D view of orice of appendix (open black
arrow); (iii) 3D view of appendiceal lumen (open black arrows). (b) (i) Air in appendix (open white arrow) on 2D coronal view;
11 Anatomy oftheColon: Rectum toIleocaecal Valve
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b(ii) c(i)
c(ii) d(i)
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Fig. 11.11
dix (open white arrow). (c) (i) 2D coronal view showing malrotated caecum (C), air-lled appendix (open white arrow), and ileocaecal valve (ICV); (ii) 2D coronal view
(ii) 2D sagittal view showing air in the appen-
showing air in terminal ileum (open red arrow) and air in appendix (closed white arrow). (d) (i) 2D axial showing barium lled appendix (open red arrow);
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J. H. Bortz
d(ii)
d(iv) e(i)
d(iii)
Fig. 11.11
lled with air (open white arrow); (iii) 2D sagittal view showing appendix (open red arrow) adjacent to spine; (iv)
(ii) 2D axial showing retrocaecal appendix
2D coronal view showing sub-hepatic appendix (yellow arrow). (e) (i) 2D coronal view showing appendix (closed red arrow) in inguinal canal;
11 Anatomy oftheColon: Rectum toIleocaecal Valve
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e(ii)
Fig. 11.11
(closed red arrow) in inguinal canal
(e) (ii) 2D sagittal view showing appendix
11.4 Malrotation oftheBowel
Malrotation is a failure during the development of normal rotation of any part of the intestinal tract. Congenital malrotation of the midgut often presents clinically in the rst month of life; more commonly in the rst post-natal week where the newborn presents with bilious vomiting [13]. This would be a medical emergency as the cause may be due to malrotation of the midgut with vol­vulus. If an early diagnosis is not made, this could result in complications, such as ischaemia of the small bowel loops, and subsequent death. Most patients born with malrotation will be asymptomatic with a normal clinical history [14]. Malrotation in such patients is an incidental nd­ing when they undergo a screening CTC exami-
nation after the age of 50years. Malrotation does not occur in isolation in this abnormality. With it comes malxation of the mesentery, which results in abnormal mobility of portions of the bowel [15]. Figure11.12a (i)–b are examples of such a pathology.
At CTC when patients with bowel malrotation are shown the images, they are often very sur­prised as they were unaware and asymptomatic with a normal clinical history. They usually do not entertain the possible need for surgical inter­vention . Some authorities advocate surgical cor­rection (Ladd’s procedure) for all patients with malrotation, regardless of age [16]. Failure to correct the abnormality may result in an intus­susception or volvulus, in the future. This would then become a surgical emergency to correct the underlying abnormality.
11.4.1 Mobility ofColon Segments
The sigmoid colon and transverse colon are intra­peritoneal structures and may be mobile depend­ing on how loosely the mesentery is attached to them [3]. In view of such mobility, it often appears as if polyps move with postural change (supine to prone) during a CTC study. A mobile lesion on CTC should not be assumed to be stool. The ascending colon, descending colon, and rec­tum are retroperitoneal in position and do not usually change position. As shown in Fig. 11.1 above, the caecum usually lies in the right iliac fossa. However, in approximately 10% of the population the caecum and ascending colon are incompletely xed which allows for a wide range of mobility. Although displacement of the cae­cum and ascending colon does not cause symptoms, the onset of appendicitis may be dif­cult to diagnose clinically, especially if the dis­placed colon lies in the left upper quadrant of the abdomen or is sub-hepatic in position. Figure11.13 (i) and (ii) demonstrate mobility of the caecum.
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J. H. Bortz
a(i)
b
a(ii)
Fig. 11.12 Malrotation of the bowel. (a) (i) Supine colon-map of a malrotated caecum.(C). Rectum (R); descending colon (DC); transverse colon (TC). Note the
gap in the ascending colon (technical); (ii) 2D coronal view of a malrotated cecum (C). (b) Supine colon-map showing sub-hepatic caecum (C). Rectum (R)