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Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_4450_Библиотеки_им_академика_М_И_Перельмана

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Fig. 2.60Irregularity of the bony pyramid.
The bony dorsum or the lateral bony wall is irregular. Irregularities do not always cause symptoms and might only be noticed on palpation. In some cases, they are clearly visible, and tender on palpation. Irregularities can be made more evident by stretching the overlying skin. They are usually caused by traumatic fractures, but may also be due to incomplete or asymmetrical hump removal
with insufficient smoothing of the dorsum. Furthermore, they may result from inadequate repositioning of the bony walls after osteotomies.
Lateral Step (▶ Fig. 2.61)
Fig. 2.61Lateral step. On the lateral wall of the bony pyramid, a ridge is visible and
palpable.
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On the lateral bony wall, a ridge or groove is visible and/or palpable. This deformity is mostly caused by a high lateral osteotomy and (too much) infraction of the bone.
Defect of the Bony Pyramid (▶ Fig. 2.62)
Fig. 2.62Defect of the bony pyramid. A defect of the lateral wall or dorsum of the bony
pyramid is visible and/or palpable.
A defect of the bony pyramid may occur after severe trauma with multiple fractures and dislocation or necrosis of bony fragments.
Open Roof (▶ Fig. 2.63)
Fig. 2.63Open roof. A defect of the bony dorsum. This abnormality is usually caused by
a hump resection without proper reconstruction of the dorsum.
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The bony dorsum is defective. This is most commonly due to a hump resection without adequate closure of the resulting defect of the bony pyramid. An open roof is usually diagnosed by palpation. Large defects are sometimes visible too. Various neuralgic symptoms may occur. We then speak of an “open roof syndrome” (see
Open Roof Syndrome).
Asymmetry of the Cartilaginous Pyramid (▶ Fig. 2.64)
Fig. 2.64Asymmetry of cartilaginous pyramid.
The lateral walls (i.e., triangular cartilages) are unequal in position and length. The cartilaginous septum is usually deformed, too; it is either dislocated or defective. Both function and nasal aesthetics are impaired. This asymmetry results mostly from trauma or impaired nasal growth.
Dorsal Step (see ▶ Fig. 2.55 and ▶ Fig. 2.56)
A dorsal step is due to disruption of the junction between the triangular cartilages and the nasal bones. The attachment of the cranial margin of the triangular cartilage to the undersurface of the caudal border of the nasal bone is disrupted. A depression at the upper part of the triangular cartilage is visible and palpable.
Atrophy of the Cartilaginous Pyramid (▶ Fig. 2.65)
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Fig. 2.65Atrophy of the cartilaginous pyramid.
The triangular cartilages are atrophied and partially missing, having been replaced by scar tissue. The lateral soft or hinge area may be scarred, atrophied, and retracted. The lateral wall of the valve is weakened and collapsible, which may cause impairment of inspiratory breathing. This pathology is usually the result of (repeated) trauma, a dorsal hematoma, and/or infection.
2.2.5Lobule
Low, Wide Lobule (▶ Fig. 2.66)
Fig. 2.66Low and wide lobule.
The lobule is low and wide at all levels (tip, nares, and base). The tip is broad and underprojected or depressed. The alae are convex, the columella short, the nostrils more
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or less round. The vestibules are wide and low. The tip index is large. A low and wide lobule is normal among black people and, to a lesser degree, Asians. In Caucasians, it is only seen in newborns and infants. The condition may also be caused by retardation of nasal growth after trauma or a septal abscess at a young age (see also ▶ Childhood Septal
Abscess and ▶ Fig. 1.118a-h).
High, Narrow Lobule (▶ Fig. 2.67)
Fig. 2.67High and narrow lobule.
The lobule is prominent and narrow, the tip narrow and pointed, the alae long and stretched, and the columella long. The nostrils are narrow and the vestibules high and narrow. The tip index is small. A high, narrow lobule is common in Caucasians, especially in combination with dolichocephaly (see also ▶ Fig. 2.11 and ▶ Fig. 2.12).
Tip
Broad Tip (▶ Fig. 2.68)
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