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telangiectasias in a male hemophiliac
with human immunodeficiency virus
infection. J Am Acad Dermatol
1988;19:369.
157. Fallon T Jr, Abell E, Kingsley L, et al.
Telangiectases of the anterior chest in
homosexual men. Ann Intern Med
1986;105:679.
158. Cockerell CJ, LeBoit PE. Bacillary
angiomatosis: a newly characterized,
pseudoneoplastic, infectious,
cutaneous vascular disorder. J Am
Acad Dermatol 1990;22:501.
159. Clarke D, Martinez A, Cox RS.
Analysis of cosmetic results and
complications in patients with stage I
and II breast cancer treated by biopsy
and irradiation. Int J Radiat Oncol
Biol Phys 1983;9:1807.
160. Goldschmidt H, Sherwin WK.
Reactions to ionizing radiation. J Am
Acad Dermatol 1980;3:551.
161. Bilic M, Adams BB. Erythema ab igne
induced by a laptop computer. J Am
Acad Dermatol 2004;50:973.
162. Jagtman BA. Erythema ab igne due to
a laptop computer. Contact Dermatitis
2004;50:105.
163. Mohr MR, Scott KA, Rariser RM.
Laptop computer-induced erythema
ab igne: a case report. Cutis
2007;79:59.
164. Bachmeyer C, Bensaid P, Begon E.
Laptop computer as a modern cause
of erythema ab igne. J Eur Acad
Dermatol Venereol 2009;23:736.
165. Dover JS, Phillips TJ, Arndt KA.
Cutaneous effects and therapeutic uses
of heat with emphasis on infrared
radiation. J Am Acad Dermatol
1989;20:278.
References
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5
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C H A P T E R
Noninvasive Examination of the Patient
Before Sclerotherapy
Before sclerotherapy is performed, the examiner must obtain
a focused history and perform a meticulous physical examination including inspection and palpation (level 1). This
combination places the patient into a proper clinical classification and this, in turn, will dictate therapy. For example,
primary venous insufficiency is characterized by telangiectasias, reticular varicosities, and varicose veins without the
stigmata of chronic venous disease. The latter has characteristic hyperpigmentation, edema, ulceration, or scarring
from healed ulcers. The presence of chronic disease findings
aids the examiner to be more insistent regarding further
evaluation.
Confirmatory diagnostic testing is performed after the
history is taken and the physical examination performed. The
handheld continuous-wave Doppler historically was a routine
part of the physical examination but is slowly being replaced
by compact, portable duplex ultrasound visualization. At
present, the vascular laboratory (level 2) provides a reliable
tool for acquiring anatomic and functional information that
not only confirms the diagnosis but also formulates treatment.
Elements of the diagnostic vascular laboratory are slowly
becoming part of a routine physical examination.
examination of the venous system is becoming a more important diagnostic tool. This approach has been confirmed in the
guidelines of the American Venous Forum.
Invasive testing such as phlebography and ambulatory
venous pressure (level 3) measurements, while of historical
significance, have been relegated to a secondary role as large
amounts of data are obtained from duplex ultrasound. The
level of diagnostic testing is dictated by the severity of the
clinical condition.
2
1
A duplex
Medical History
A focused history is the beginning of every evaluation for any
patient with a suspected vascular disorder. A clear understanding and detailed description of the symptoms is the first step.
Recording of the patient’s concerns and reasons for seeking
treatment should also be done at this time since patients
concerned by pain at first interrogation may focus on esthetics
in subsequent sessions following treatment of primary sources
of reflux. Symptoms of venous insufficiency have been termed
nonspecific. However, patients with clear symptoms of venous
insufficiency typically do not ascribe these to varicose veins.
Symptoms include aching tiredness and far ranging discomfort in the legs, relieved by sitting and leg elevation. Such
discomfort increases as the day progresses with increased vein
stretching. In women symptoms are exacerbated on the first
days of a menstrual period, since elevated progesterone levels
cause increase vein swelling. Symptoms may also begin in
pregnancy. It has been said that for males with varicose veins
the lack of progesterone or estrogen results in fewer
symptoms.
The magnitude of symptoms is not dependent on the size
of the varicosities. Telangiectasias and reticular varicosities
may cause symptoms identical to those of gross varicose veins.
Since symptoms are due to pressure of dilated veins on somatic
nerves, it is not surprising that some patients experience
burning near varices. This is thought to be ischemic neuropathy, but may be capillary pressure related. A disabling bursting
pain that develops on standing or sitting with legs dependent
and that is relieved by muscular activity is paradoxically called
venous claudication, although incorrectly.
A family history of varicose veins is common. A personal
and family history of a diagnosis of venous thrombosis should
be requested. If present, it is important to clarify the way in
which this diagnosis was confirmed (phlebography, venous
duplex, or simply clinically). The patient should be prompted
to recall specific aspects of their medical history, such as traumatic fractures, leg swelling, need for systemic anticoagulation,
illness requiring bed rest, or major surgical intervention.
CEAP classification
An international ad hoc committee of the American Venous
Forum developed the CEAP classification for chronic venous
disease in 1994 (Table 5.1; see also Chapter 2) with the goal
of stratifying clinical levels of venous insufficiency. The four
categories and descriptors selected for classification were clinical state (C), etiology (E), anatomy (A), and pathophysiology
(P). The CEAP classification has been endorsed worldwide,
despite its acknowledged deficiencies. It has been adopted as
a standard in many clinics in Europe, Asia, South America,
and the United States it is considered the only modern method
for reporting data. It’s weakness is the inability to distinguish
between levels of smaller superficial veins. The CEAP classification was revised in 2004,
4
CEAP.
It includes:
Clinical assessment:
• C1 is presence of telangiectatic and reticular veins (see
definitions in Chapter 2)
• C2 is presence of varicose veins
• C3 is presence of venous edema
• C4 is presence of skin changes (C4a for pigmentation
and/or eczema, C4b for lipodermatosclerosis and/or
atrophie blanche)
• C5 is presence of healed ulcer
• C6 of an active ulcer.
The descriptor (a) is added for asymptomatic patients and
(s) in case of symptoms.
All clinical features must be reported in the advanced CEAP;
for example a patient with telangiectasias, varicose veins,
edema, pigmentation, active ulcer and pain is classified C6s
in the basic (classical) CEAP but will be C1,2,3,4a,6,s in the
advanced (revised) CEAP. This approach carries much more
information but stratifies the patients’ samples.
3
and is now referred to as ‘advanced’

Table 5.1 CEAP classification
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CLINICAL CLASSIFICATION
C0 No visible or palpable signs of venous disease
C1 Telangiectasias or reticular veins
C2 Varicose veins – separated from reticular veins by a diameter of 3 mm as the upper limit of size of a reticular vein
C3 Edema
C4 Changes in the skin and subcutaneous tissue secondary to chronic venous disease are divided into two subclasses to better define the
differing severity of venous disease:
C4a Pigmentation and eczema
C4b Lipodermatosclerosis and atrophie blanche
C5 Healed venous ulcer
C6 Active venous ulcer
Each clinical class is further characterized by a subscript for the presence of symptoms (S, symptomatic) or their absence (A, asymptomatic). Symptoms
include aching, pain, tightness, skin irritation, heaviness, and muscle cramps, as well as other complaints attributable to venous dysfunction.
ETIOLOGIC CLASSIFICATION
Ec Congenital
Ep Primary
Es Secondary (postthrombotic)
En No venous etiology identified
ANATOMIC CLASSIFICATION
As Superficial veins
Ap Perforator veins
Ad Deep veins
An No venous location identified
PATHOPHYSIOLOGIC CLASSIFICATION
Basic CEAP
Pr Reflux
Po Obstruction
Pro Reflux and obstruction
Pn No venous pathophysiology identifiable
Advanced CEAP
Same as Basic with the addition that any of 18 named venous segments (below) can be utilized as locators for venous pathology.
All items listed in C should be repeated.
VENOUS SEGMENTS
Superficial veins
1 Telangiectasias/reticular veins
2 GSV above knee
3 GSV below knee
4 SSV
5 Nonsaphenous veins
Deep veins
6 IVC
7 Common iliac vein
8 Internal iliac vein
9 External iliac vein
10 Pelvic: gonadal, broad ligament veins, other
11 Common femoral vein
12 Deep femoral vein
13 Femoral vein
14 Popliteal vein
15 Crural: anterior tibial, posterior tibial, fibular veins (all paired)
16 Muscular: gastrocnemial, soleal veins, other
Perforating veins
17 Thigh
18 Calf
Medical History
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Noninvasive Examination of the Patient Before Sclerotherapy
90
• Etiology is reported by descriptor E: Ec is congenital
(usually present at birth), Ep is primary (degenerative,
typically varicose veins), Es is secondary (like in
post-thrombotic syndrome), En is for no venous
etiology identified.
• Anatomy is described by the A: As for superficial
(venous network involvement), Ap for perforators, Ad
for deep, An when no venous location has been
identified. Involved venous segments are recorded
with a number (see Table 5.1).
• Pathophysiology is reported by the P descriptor: Pr for
reflux, Po for obstruction, Pro for association of both
and Pn if pathophysiology has not been recognized.
Here again, involved venous segments must be
recorded by numbers (see Table 5.1).
• Date of patient’s evaluation
• Level of examination: L1 (level 1) is clinical plus
continuous wave Doppler, L2 is noninvasive (duplex
ultrasound, plethysmography), L3 includes complex
imaging such as computed tomography (CT) and
magnetic resonance imaging (MRI) and invasive
investigations such as venograms, intravenous
ultrasound and blood pressure measurement.
Diagnostic approach
The first step in evaluating a patient with venous disease is to
establish his or her clinical class, which rapidly progresses
from cosmetic to chronic venous insufficiency. The next is to
correlate the symptoms, which place the limb being examined
into one of the classes shown in Table 5.1. The patient’s clinical class will dictate the need for further evaluation. In patients
with telangiectasias (class 1 or 2), the evaluation can be
limited to a physical examination and evaluation of the
superficial venous system with a handheld continuous-wave
Doppler. Imaging of 83 limbs with clinical evidence of only
telangiectatic vessels demonstrated that nearly 25% had insufficiency of the great saphenous vein (GSV) or small saphenous
veins (SSV), which was not apparent on physical examina-
5
tion.
Patients with symptomatic class 2 varicosities and class
4, 5, and 6 skin changes require a duplex venous reflux examination because surgical intervention is indicated.
trant cases may require more extensive imaging studies to
detect venous occlusive disease. Physiologic testing can be
relegated to documentation rather than to diagnosis, and
phlebography should be performed only when venous reconstruction is contemplated.
Prior treatment
The physician should discuss prior treatment for venous
disease. However, he or she must realize that although proper
ligation, with or without stripping of the main saphenous
trunks, implies that reflux through the saphenofemoral junction (SFJ) and saphenopopliteal junction (SPJ) has been prevented, this is not always the case. Some have proposed that
in up to 27% of patients there is a duplication of the GSV;
thus, the removal of the GSV may be followed by the development of varicosity in the remaining GSV, although this is not
universally accepted.
In 20% to 40% the SSV has a variable termination
is not in the popliteal vein at or above the popliteal fossa.
Therefore, the actual SPJ must be correctly diagnosed otherwise it will lead to an apparent rapid recurrence with varicose
changes occurring in the remaining segment of the SSV and
its tributaries. Finally, in a number of patients, a recurrence of
varicose veins in the upper thigh may be the result of incomplete ligation and division of the other tributaries arising at
the level of the SFJ or failure to accomplish the ligation flush
with the femoral vein. In fact, in a review of 341 extremities
that underwent repeat operations for varicose veins, Lofgren
6–10
Recalci-
14–17
11–13
that
et al17 found that 61% had inadequate ligation. These facts
make it imperative that, even in the patient with a history of
ligation, division, and stripping, an examination for reflux
through the SFJ and SPJ be performed.
The physician should consider responses to and complications from all treatment modalities (sclerotherapy, laser, etc.).
Certain complications, such as ischemic ulceration caused by
injection into an arteriovenous malformation, can be avoided
more easily if their prior diagnosis is made. Given the predilection for these to occur in a particular anatomic distribution(s),
the physician might avoid treating that area or use greater
caution in the previously affected region. A history of prior
hyperpigmentation, blushing, or poor response to a particular
sclerosing agent may support a variety of changes in treatment
protocol, such as altering the sclerosant concentration, increasing the strength or duration of compression, and paying
greater attention to post-treatment thrombectomy.
Symptoms
It is not well known that presence and severity of symptoms
has no correlation with the size or severity of varicose veins
present. Symptoms usually attributable to varicose veins
include feelings of heaviness, tiredness, aching, burning,
throbbing, itching, and cramping in the legs (Box 5.1). These
symptoms are generally worse with prolonged sitting or
standing and are improved with leg elevation or walking. A
premenstrual exacerbation of symptoms is also common.
Patients typically find relief with the use of compression in
the form of either support hose or an elastic bandage if
they are compliant. Compliance can be a challenge. Weight
loss or the commencement of a regular program of lower
extremity exercise may also lead to a diminution in the severity of varicose vein symptoms. Clearly, these symptoms are
not specific, as they may also be indicative of a variety of
rheumatologic or orthopedic problems. However, their relationship to lower extremity movement and compression is
usually helpful in establishing a venous origin for the symptoms. Significant symptoms suggestive of chronic venous
disease should prompt further evaluation for valvular insufficiency and calf muscle pump dysfunction. If a venous etiology is suspected but all examinations are negative, repeat
examination during a symptomatic period is warranted and
often fruitful.
The recent development of an extremely painful area on
the lower leg associated with an overlying area of erythema
and warmth may be indicative of lipodermatosclerosis, which
may be associated with insufficiency of underlying perforator
veins or reflux from a proximal point. Examination for underlying perforator vein reflux should be performed. Lipodermatosclerosis may precede ulceration and has been shown to be
improved by stiff compression and certain pharmacologic
interventions.
18
Rarely, patients with a history of iliofemoral thrombophlebitis who describe ‘bursting’ pain with walking may be suffering from ‘venous claudication’. In these patients, an evaluation
for persistent hemodynamically significant obstruction, possibly treatable with venous bypass surgery, is appropriate.
Box 5.1
Symptoms attributable to varicose veins
• Aching
• Heaviness, tiredness
• Pain (throbbing, burning, sharp, tingling)
• Itching
• Cramping
19

Complications of varicose vein disease
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Complications such as ulceration and hemorrhage should be
discussed with the patient because this provides additional
insight into both the severity and the probable locations of
abnormality within the venous system. A history of ulceration
of the medial aspect of the lower leg should prompt further
examination of the GSV trunk,
6
whereas involvement of the
lateral aspect of the lower leg suggests an abnormality in the
SSV, in addition to the deep and perforating vein systems.
A history of hemorrhage from telangiectasias in a particular
area suggests further examination for underlying incompetent
perforators and is an indication to treat all suspicious
telangiectasias.
20
Purpose of Venous Evaluation
More extensive evaluation can provide essential information
regarding both venous anatomy and function. Abnormalities
of the superficial, perforating, or deep systems can be diagnosed, and the exact sites of valvular insufficiency within the
various systems (and therefore the sites where treatment must
be directed) can be determined. The hemodynamic significance of each of these abnormalities may be defined, and the
effect of correcting each site of reflux may be assessed. Insufficiency at a particular site within the venous system may be
found to have no importance in the patient’s pathologic
venous hypertension or symptoms. This site may or may not
be incorporated into the treatment plan to minimize the
number of treatments. With the examination techniques discussed in the following sections, the result of sclerotherapy
may be documented in a more accurate and sensitive manner
than with simple observation and palpation. Treatment
success may be enhanced significantly with the goal being
restoration of normal venous flow. If the presence of deep
venous thrombosis (DVT) and/or deep venous valvular insufficiency are detected, these are absolute and relative contraindications to sclerotherapy. Once properly diagnosed, serious
complications in these non-candidates for sclerotherapy can
be avoided.
Physical Examination
The best way to approach examination of the venous system
before sclerotherapy is to be methodical. Although the exact
method is a matter of personal preference, a systematic
approach is advisable.
Using skills of clinical practice, the practitioner can obtain
a degree of information regarding overall venous outflow
from the leg, the sites of valvular insufficiency, the presence
of primary versus secondary varicose veins, and the presence
of DVT.
The screening physical examination consists of careful
observation of the legs. Any patient with the following conditions should be examined more fully: large varicose veins;
bulges in the thigh, calf, or the inguinal region representative
of incompetent perforating veins (IPVs) or a saphena varix;
signs of superficial venous hypertension, such as an accumulation of telangiectasias in the ankle region (corona phlebectatica); or any finding suggestive of venous dermatitis
(pigmentation, induration, eczema). This includes patients
with obvious cutaneous signs of venous disease, such as
venous ulceration, atrophie blanche, or lipodermatosclerosis.
An obvious but often forgotten point is the necessity of observing the entire leg and not confining the examination simply
to the area that the patient feels is abnormal. The importance
of this is demonstrated in Figure 5.1. This patient came for
treatment of an obviously dilated anterior thigh vein, but
further inspection revealed a saphena varix, with incompetence at the level of the SFJ; thus defining the first step in her
treatment. Similarly, patients often seek treatment of specific
clusters of telangiectasia and do not notice the underlying
reticular veins that should be treated before or at the same
time (see Chapter 12).
Finally, because the veins of the leg empty into the pelvic
and abdominal veins, inspection of the abdomen is very
21
Physical Examination
A
Figure 5.1 A, This patient sought treatment because of an obviously enlarged vein in her thigh. B, Further inspection revealed a saphena varix (arrow)
indicative of saphenofemoral junction insufficiency.
B
(Courtesy Anton Butie, MD)
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5
important, since dilation of veins on the abdominal wall or
across the pubic region suggests an old iliofemoral thrombus
or, rarely, a developmental anomaly of the venous system.
22
23
Dilated veins along the medial or posterior aspect of the proximal thigh or buttocks most often arise from varicosities
involving the pudendal or other pelvic vessels. These can be
associated with vulvar varices that may remain symptomatic
after the completion of the pregnancy during which they
formed. The enlarged veins in the thigh or buttocks may also
be quite symptomatic and respond well to treatment.
Cough test
One hand is placed gently over the GSV or SFJ and the patient
is asked to cough or perform a Valsalva maneuver (Fig. 5.3).
Simply palpating an impulse over the vein being examined
may be indicative of insufficiency of the valve at the SFJ and
below to the level of the palpating hand. This test, however,
is not applicable to the examination of the SSV and SPJ (see
following section).
ver is generally more diagnostic. One study found a low sensitivity of 0.59 and low specificity of 0.67 with this test.
14
Palpation of a thrill during this maneu-
25
Clinical testing
Historically, tests of venous function have been part of the
physical examination of venous insufficiency. These tests have
been slowly abandoned due to lack of specificity and sensitivity. As the ‘stethoscope’ of the venous examination, the
continuous-wave Doppler examination has replaced most of
these tests, while confirmatory duplex testing has supplanted
all. With that being said, an educated physician who treats
venous insufficiency must have knowledge of these tests and
their physiologic background.
Knowledge of the Trendelenburg test (or BrodieTrendelenburg test) is important for understanding venous
physiology and is of historical significance.
Noninvasive Examination of the Patient Before Sclerotherapy
Trendelenburg test
For the Trendelenburg test, a tourniquet may be placed around
the patient’s proximal thigh while the patient is standing. The
patient then assumes the supine position with the affected leg
elevated 45 degrees. The tourniquet is removed and the time
required for the leg veins to empty, which is indicative of the
adequacy of venous drainage, is recorded.
When compared with the contralateral leg, the method just
described may demonstrate a degree of venous obstructive
disease. Another approach is to elevate the leg while the
patient is supine and to observe the height of the heel in relation to the level of the heart that is required for the prominent
veins to collapse (Fig. 5.2). Unfortunately, neither procedure
is sufficiently sensitive or accurate, or able to differentiate
acute from chronic obstruction, which means neither of them
is much assistance in current medical practice. This emphasizes the important role of duplex ultrasound in modern
evaluation of the superficial venous system. One study found
that pneumatic tourniquets only occluded 27% of saphenous
24
trunks.
described below, can provide information on the competence
of the venous valves.
Several other physical examination maneuvers,
Percussion/Schwartz test
One hand is placed over the SFJ or SPJ while the other hand
is used to tap very lightly on a distal segment of the GSV or
SSV (Fig. 5.4). The production of an impulse in this manner
implies insufficiency of the valves in the segment between the
two hands. Confirmation of valvular insufficiency can be
achieved by tapping proximally while palpating distally. This
test can also be used to detect whether an enlarged tributary
is in direct connection with the GSV or SSV by palpating over
the main trunk and tapping lightly on the dilated tributary, or
vice versa. The presence of a direct connection results in a
palpable impulse being transmitted from the percussing to the
palpating hand. As might be expected, these tests are far from
infallible. In a study of 105 limbs, Chan et al
these clinical examination techniques correctly identified SFJ
incompetence in only 82% of limbs. False negatives were
believed to be caused primarily by previous groin surgery with
resultant scarring and by obesity. However, false positives
were the result of variations in venous anatomy, such as a
dilated tributary emptying into the common femoral vein
(CFV) adjacent to the GSV or the absence of valves in an otherwise normal CFV and external iliac vein (seen in 5% to 30%
of patients).
0.59 with a high specificity of 0.92.
27,28
Another study showed a low sensitivity of
25
A further source of error
with the cough and/or percussion test is simply a misinterpretation of the muscle contraction that occurs with coughing as
a reflux impulse.
26
found that
Figure 5.2 Venous outflow may also be assessed by elevating the leg until
the superficial veins collapse and then measuring the distance (X) from the
heart to the heel and comparing this measurement with the other leg.
92
X
Figure 5.3 Cough test. The saphenofemoral junction (SFJ) is palpated
while the patient coughs. Palpation of an impulse is indicative of SFJ
insufficiency.

Figure 5.4 Percussion test. The saphenopopliteal junction (SPJ) is palpated
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while the small saphenous vein is gently percussed. Palpation of an impulse
is indicative of SPJ insufficiency.
Brodie-Trendelenburg test
The Brodie-Trendelenburg test traditionally involves the
manual obstruction of the proximal end of the GSV (or SSV)
while the patient lies supine with the leg elevated, after stroking the vein in a cephalad direction to empty it of blood.
The patient then assumes the standing position, and the leg
is observed for 30 seconds (Fig. 5.5). In a ‘nil’ test there is slow
filling of the veins from below, and the release of the compression does not result in rapid filling from above, indicating
competence of valves in deep and perforating veins and at the
SFJ (Fig. 5.6A). Rapid filling of the GSV or more distal tributaries that occurs only after release of the compression constitutes a ‘positive’ test, indicating the presence of an insufficient
valve at the SFJ (Fig. 5.6B). In the ‘double-positive’ test, some
distension of the veins occurs within the initial 30 seconds
while the compression is maintained, as well as additional
filling once the compression is released (Fig. 5.6C). This is
taken as evidence of incompetent deep and perforating veins
as well as reflux through the SFJ. A ‘negative’ test occurs when
the veins fill within the initial 30 seconds with no increased
filling after the compression is released, implying only deep
and perforating valvular insufficiency (Fig. 5.6D). The reverse
may not be true; that is, filling in longer than 30 seconds does
not imply competence of perforating veins. In a study of 901
extremities, Sherman
33
found that 95% had a nil Trendelenburg test, but surgical exploration later showed incompetent
perforators in 90% of these patients. Another study showed a
high sensitivity of 0.91 with a low specificity of 0.15.
The Brodie-Trendelenburg test thus can be an important
method of localizing the most proximal site of reflux in most
dilated superficial veins by obstructing the GSV, SSV, or
whichever vein is suspected of refluxing into a more distal
vein. The physician can also place the examining finger over
palpable fascial defects in the leg while the patient is supine
and then release the obstructions one by one after the patient
is standing. This allows the sites of insufficient perforators, or
‘points of control’ (considered so crucial in Fegan’s technique
of sclerotherapy), to be defined, because the superficial veins
distal to the insufficient perforator fill rapidly once the
obstructing fingers are removed (see Chapter 9).
22,29–32
25
34,35
With this technique, described well in many papers,
the practitioner first marks on the leg the sites of all dilated
varicosities. The patient then assumes the supine position with
the leg elevated to approximately 60 degrees to empty the
veins. After at least 20 seconds, or when the distended veins
are flattened, the leg is gently and rapidly palpated to detect
any defects in the fascia. With experience, these can be detected
easily as places that allow the entrance of the examining finger
without the use of any pressure. Fascial defects can be caused
by many abnormalities other than perforating veins, thus the
practitioner continues the examination by compressing the
individual fascial defects with his or her fingers and then
having the patient stand. The fingers are then released one by
one, starting with the most distal defect, and rapid filling of
more distal varicosities is noted (Fig. 5.7). Those defects that
cause distal filling when released are assumed to correspond
to sites of IPVs. In the presence of a dilated GSV or SSV, these
points of reflux first must be controlled with either digital
compression or a tourniquet to evaluate the lower volume
reflux through the perforators. For the evaluation to be helpful,
compression of the defects must first cause sustained flattening of the varicosities when the patient initially stands. If the
veins fill before any of the fingers are released, the test must
be restarted and other sites compressed until the sites responsible for the reflux are located. This examination is associated
with a 50% to 70% accuracy
36–39
compared with findings at
surgical exploration. Repeated examination at different times
and improvement of edema allows the detection of increased
numbers of perforators.
Bracey Variation
A clever variation of the Brodie-Trendelenburg technique was
proposed by Bracey
cm-wide, rubber tourniquet and two rubber rings covered
with latex, with inside diameters of 7 cm and 8.2 cm. The
smaller ring is used between the ankle and knee and may also
be used for the thigh if the patient is thin. If not, the larger
ring is used for the thigh. With the patient standing, the small
ring is rolled over the foot to just above the ankle, and the
rubber tourniquet is then placed below the ring to obstruct
any upward flow of blood through the superficial veins. The
small ring is then slowly rolled upward, emptying the superficial veins as it moves. As soon as it passes an IPV, the blood
enters the superficial vein that connects with it, causing a dilation of the vein. The exit site of the perforating vein may then
be marked. This reflux of blood can be accentuated by asking
the patient to repetitively dorsiflex the foot. When the ring
reaches the knee, the tourniquet is moved up to the knee, just
below the ring. Either the smaller or larger ring is then used
similarly to examine the thigh.
40
in 1958 (Fig. 5.8). He used a flat, 3.8-
Perthes’ test
The Perthes’ test
ing between venous valvular insufficiency in the deep, perforator, and superficial systems and screening for DVT (Table 5.2).
To localize the site of valvular disease, the physician places a
tourniquet around the proximal thigh with the patient standing. When the patient ambulates, a decrease in the distension
of varicose veins suggests a primary process without underlying deep venous disease because the calf muscle pump effectively removes blood from the leg and empties the varicose
veins. Secondary varicose veins do not change caliber (if there
is patency of the deep venous system) because of the inability
to empty blood out of the veins as a result of impairment of
the calf muscle pump. In the setting of a concurrent DVT, they
may increase in size. If there is significant chronic or acute
obstructive disease in the iliofemoral segment, the patient
may note pain (venous claudication)
obstruction to outflow through both the deep and superficial
22,32,41
has several uses, including distinguish-
42–44
as a result of the
21,34–37
Physical Examination
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