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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3643_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Preface
- •Abbreviations
- •Contents
- •1.1.2.4 The Carotid Artery
- •1.1.2.5 The Internal Jugular Vein
- •1.1.2.6 The Nerves of the Neck
- •1: The Cerebral Circulation
- •1.1 Clinical and Surgical Anatomy
- •1.1.1 Anterior Triangle of the Neck
- •1.1.2 Posterior Triangle of the Neck
- •1.1.2.2 The Cervical Fascia and Its Layers
- •1.1.2.3 The Carotid Sheath
- •1.1.2.7 The Segments of the Carotid Artery
- •References
- •2: Cerebral Vascular Territories and the Major Neurovascular Syndromes
- •2.1 The Arterial Supply of the Brain
- •2.2 The Collateral Circulation
- •2.2.1.2 Persistence of Vestigial Arteries/Persistent Carotid-Vertebrobasilar Anastomoses
- •2.3 The Target Tissues Vascularized
- •References
- •3: Stroke Subtypes
- •References
- •4: Surgical Approaches for Cerebrovascular Revascularization
- •4.1 Surgical Approach to the Principal Target Arteries
- •4.1.1 Exposure of the Carotid Bifurcation
- •4.1.2 Exposure of the Vertebral Artery: The Segments V0 and V1
- •4.1.3 Exposure of the Subclavian Artery
- •4.4 Concomitant or More Extensive Arterial Exposure
- •4.6 Approaches for Harvesting of Venous Grafts
- •References
- •5: Diagnostic Approach to Cerebrovascular Disease: Ultrasound
- •References
- •6: Endovascular Approach: From Diagnosis to Therapy
- •References
- •7: Diagnostic Approach to Cerebrovascular Disease: CT and MRI
- •7.1 Introduction
- •7.2 Carotid Atherosclerotic Vascular Disease (CAVD): Diagnostic Imaging
- •7.3 Conclusions and Future
- •References
- •8: Pharmacological Measures for the Treatment and Prevention of Stroke: The Choice of Initial Therapy
- •8.1 Acute Ischemic Stroke
- •8.2.2.1 Cervical (Carotid and Vertebral) Atherosclerosis
- •Antithrombotic Treatment
- •Antihypertensive Treatment
- •8.2.3 Intracranial Large Artery Stenosis
- •8.2.4 Cerebral Small Vessel Disease
- •References
- •9: Anesthesia for Carotid Surgery and Stenting: Neuromonitoring and Perioperative Care
- •9.1 General Preoperative Evaluation for Carotid Endarterectomy
- •9.2 Choice of Anesthesia
- •9.2.1 General Anesthesia
- •9.2.2 Locoregional Anesthesia
- •9.2.2.1 Cervical Plexus Block
- •9.2.2.2 Cervical Epidural Anesthesia
- •9.2.3 Conversion from Local/Regional to General Anesthesia
- •9.3 Neurologic Monitoring
- •9.6 Perioperative Complication
- •References
- •10: Carotid Angioplasty and Stenting
- •10.1 Introduction
- •10.2 Method
- •10.4 Our Personal Experience
- •10.4.1 Inclusion and Exclusion Criteria
- •10.4.3 Early Complications
- •10.4.4 Late Complications
- •10.4.5 Other Uses of Angioplasty and Stenting in the Carotid Territory
- •Bibliography
- •11: Carotid Endarterectomy
- •11.1 Surgical Technique
- •11.2 Conclusive Remarks
- •References
- •12: Vertebral Artery Revascularization
- •References
- •13: Extensive Cerebrovascular Arterial Revascularization
- •13.1 Simultaneous Bilateral Carotid Endarterectomy
- •13.2 Synchronous Carotid and Vertebral Artery Revascularization
- •13.2.1 CEA + VA Reimplantation
- •13.3 Occlusive Disease of the BCT
- •13.5 Aortic Arch Syndrome
- •13.6 Revascularization of the ECA
- •13.7 ICA Thrombectomy
- •13.8.1 CEA + CCA-to-SCA Bypass + Bypass on V3
- •13.9 Particular Situations
- •13.10 Conclusive Remarks
- •References
- •14: Cervico-cerebral Arteries Dissection
- •14.1 Cervical Artery Dissection
- •14.1.1 Epidemiology, Pathophysiology, and Risk Factors for Cervical Artery Dissection
- •14.1.3 Acute Treatment and Secondary Prevention in Patients with CAD
- •14.2 Intracranial Artery Dissection
- •14.2.1 Epidemiology, Pathophysiology, and Risk Factors for Intracranial Artery Dissection
- •14.2.2 Clinical Symptoms
- •14.2.3 Treatment of IAD
- •14.3 Carotid Artery Dissection
- •14.3.1 Common Carotid Artery Dissection
- •14.3.2 Extracranial Internal Carotid Artery Dissection
- •14.4 Vertebral Artery Dissection
- •References
- •15: Extracranial Carotid and Vertebral Artery Aneurysm
- •References
- •16: Asymptomatic Carotid and Vertebral Artery Stenosis
- •References
- •17: Lessons from Experimental-Induced Atherosclerosis: Valuable for the Precision Medicine of Tomorrow
- •17.1 Introduction
- •17.2.2.1 Cytokines
- •17.2.2.2 Chemokines
- •17.3.3 Role of NADPH Oxidase Complex
- •17.4 Nanotechnology-Based Therapies: A New Prospect for Diagnosis and Treatment of Atherosclerosis
- •17.4.1 Designing “Smart” Nanocarriers
- •17.4.2 Nanoparticles Designed to Diagnose Atherosclerosis
- •17.4.8 Nanoparticles Designed to Modulate LDL and HDL Levels
- •17.4.12 Clinical Use of Nanoparticles for Diagnosis and Therapy of Atherosclerosis
- •References
- •18: Choice of the Proper Therapeutic Measure in the Individual Patient and Prevention of Stroke

286
VA
CEA
CEA
b
Fig. 13.5 (continued)
SCA
CCA
Ao
Fig. 13.6 Bypass for occluded BCT. Anatomic reconstruction for the occlusion of the BCT: bypass from the ascending aorta to the right SCA and
right CCA. Whenever the bifurcation appears diseased, the carotid limb is anastomosed to the right carotid bifurcation after CEA
H. Muresian

287
SCA
BP
VCCA
IJV
ASM
SCA
CCA
CCA
PTFE
graft
a
b
Fig. 13.7 CEA + revascularization
of the SCA. Panel ( a ) completion
angiogram illustrating the bypass
from the CCA to the occluded SCA
with PTFE graft. Note also the
retrograde fi lling of SCA branches.
Particular attention is focused on the
purging (air, thrombi) as embolic
material may pass either into the
distal CCA and ICA or into the
VA. Panel ( b ) alternative surgical
solution by using an autologous
venous graft ( G ). Note the position
of the bypass posterior to the
IJV. The ASM was cut
13 Extensive Cerebrovascular Arterial Revascularization

288
ICA
CCA
SC
PTFE
graft
ECA
Fig. 13.8 Carotid bifurcation-to-SCA bypass. The technique
includes CEA and bypass from the endarterectomized carotid to
the occluded SCA. The superior part of the PTFE graft serves as an
enlargement patch for the carotid bifurcation. The graft is tunneled
underneath the SCM
H. Muresian

289
CCA
G
ICA
ICA
CCA
G
ICA
ICA
Fig. 13.9 Extraanatomic carotid-to-carotid bypass. The fi gure illus-
trates the case of an old female patient, with severe atherosclerotic
lesions in all neck vessels, and having the only fair donor vessel, the
right CCA. The left CCA was occluded but the bifurcation remained
patent. Both carotid bifurcations had atherosclerotic lesions, requiring
the SBCE. A prelaryngeal PTFE graft ( G ) was used to revascularize the
left carotid system
13 Extensive Cerebrovascular Arterial Revascularization

290
Fig. 13.10 Bypasses from the ascending aorta. Numerous variations
and types of bypasses are performed, for the vascularization of the neck
vessels. All these variants can be easily applied in cases with aneurysms
of the aortic arch or as debranching procedures. Panel ( a ) a quadrifur-
cated graft (with the additional two branches anastomosed ad hoc) was
inserted on the ascending aorta. The branches reach the SCA on both
sides and the carotid arteries on both sides too. Note that the bifurcations
were endarterectomized (excised plaques shown in the left extremity),
and synthetic patches were inserted. Right lower Panel ( d ) demonstrates
the incisions for the combined approach: sternotomy and bilateral double parallel incisions. We prefer cervical parallel incisions instead of a
single longitudinal incision. Panel ( b ) bypass from the ascending aorta
to the left neck vessels: ICA and SCA. Note that even in this case, the
carotid limb is used as an enlargement patch. Panel ( c ) a similar recon-
struction on the right side, in case of obstruction of the BCT. Panel ( d )
completion angiogram in a patient with a quadrifurcated graft
SCA
SCA
CEA
CEA
CEA
SCA
Ao
a
b
H. Muresian

291
SCA
CCA
SCA
SCA
ICA ICA
c
d
Fig. 13.10 (continued)
13 Extensive Cerebrovascular Arterial Revascularization

292
CCA
ECA
ICA
CCA
ECA
ICA
CCA
ECA
ICA
Fig. 13.12 Revascularization of the ECA. Severe stenotic lesions at
the origin of the ECA in patients with bilateral occlusion of the ICA are
treated either by endovascular procedures (see Chap.
7 ) or by surgery.
Intraoperative aspect of the right carotid bifurcation. The ICA is
occluded (note that the ICA is declamped and no back bleeding is present). After performing the endarterectomy of the origin of the ECA, the
patch is inserted and oriented toward the ECA
Ao
BCT
Ao
BCT G
Fig. 13.11 The BCT as a donor vessel. Whenever the quality of the
ascending aorta limits the use of this vessel for bypass, or when the
patient cannot tolerate the lateral clamping of the ascending aorta, the
BCT may offer a good alternative. Preoperative diagnostic interrogation
must rule out dilatations of the ascending aorta or the presence of heavy
and extensive calcifi cations, but in some cases, this may come as an
intraoperative surprise, in spite of all the precautions previously taken.
This is also valid for coronary bypass grafting when the proximal graft
anastomosis may be performed on the BCT instead of the ascending
aorta
H. Muresian

293
Fig. 13.13 Thrombectomy of
the ICA (1). Recently thrombosed ICAs can be restored by
thrombectomy and subsequent
patching. Panel ( a ) occlusive
stenosis at the origin of the left
ICA (note also the intraplaque
hemorrhage). The end of the
thrombus appears at the origin of
the ICA. Panel ( b ) the ICA is
dethrombosed with a Fogarty
catheter. The plaque is removed
and the bifurcation is closed with
a patch. Panel ( c ) macroscopic
aspect of the excised carotid
plaque and thrombus from the
ICA. Retrograde fl ow from the
intracranial segments of the ICA
prevents distal embolization of
the thrombotic material
CCA
ECA
ICA
ICA
a
b
13 Extensive Cerebrovascular Arterial Revascularization

294
ICA
ICA
CCA
CCA
ICA
ICA thrombus
Fig. 13.14 Thrombectomy of the ICA (2). Thrombosis of the ICA
appears as an intraoperative surprise, in a patient referred for CEA, and
known to have a pre-occlusive stenosis. In the time interval from diag-
nosis to surgery, the ICA is occluded. The ICA was treated as above.
The plaque was removed by using the eversion technique
CCA
ICA thrombus
ICA
c
Fig. 13.13 (continued)
H. Muresian

295
CCA CCA CCA
ECA
ICA
ECA
ICA
ECA
ICA
Fig. 13.15 Bifurcated patch. Intraoperative aspect of the right carotid
bifurcation. The ICA appears hypoplastic (compare the diameter with
that of the ECA). Patency of the ICA was probed with a malleable catheter. With the bifurcation opened, it appears evident that the origin of
the ECA is larger, and the normal patch was inserted toward the origin
of the ECA. An additional tail of the patch is used to enlarge the origin
of the ICA. Should the ICA occlude with time, the ECA will remain
patent
13 Extensive Cerebrovascular Arterial Revascularization
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