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Файл:Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_3643_Библиотеки_им_академика_М_И_Перельмана.pdf
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- •Preface
- •Abbreviations
- •Contents
- •1.1.2.4 The Carotid Artery
- •1.1.2.5 The Internal Jugular Vein
- •1.1.2.6 The Nerves of the Neck
- •1: The Cerebral Circulation
- •1.1 Clinical and Surgical Anatomy
- •1.1.1 Anterior Triangle of the Neck
- •1.1.2 Posterior Triangle of the Neck
- •1.1.2.2 The Cervical Fascia and Its Layers
- •1.1.2.3 The Carotid Sheath
- •1.1.2.7 The Segments of the Carotid Artery
- •References
- •2: Cerebral Vascular Territories and the Major Neurovascular Syndromes
- •2.1 The Arterial Supply of the Brain
- •2.2 The Collateral Circulation
- •2.2.1.2 Persistence of Vestigial Arteries/Persistent Carotid-Vertebrobasilar Anastomoses
- •2.3 The Target Tissues Vascularized
- •References
- •3: Stroke Subtypes
- •References
- •4: Surgical Approaches for Cerebrovascular Revascularization
- •4.1 Surgical Approach to the Principal Target Arteries
- •4.1.1 Exposure of the Carotid Bifurcation
- •4.1.2 Exposure of the Vertebral Artery: The Segments V0 and V1
- •4.1.3 Exposure of the Subclavian Artery
- •4.4 Concomitant or More Extensive Arterial Exposure
- •4.6 Approaches for Harvesting of Venous Grafts
- •References
- •5: Diagnostic Approach to Cerebrovascular Disease: Ultrasound
- •References
- •6: Endovascular Approach: From Diagnosis to Therapy
- •References
- •7: Diagnostic Approach to Cerebrovascular Disease: CT and MRI
- •7.1 Introduction
- •7.2 Carotid Atherosclerotic Vascular Disease (CAVD): Diagnostic Imaging
- •7.3 Conclusions and Future
- •References
- •8: Pharmacological Measures for the Treatment and Prevention of Stroke: The Choice of Initial Therapy
- •8.1 Acute Ischemic Stroke
- •8.2.2.1 Cervical (Carotid and Vertebral) Atherosclerosis
- •Antithrombotic Treatment
- •Antihypertensive Treatment
- •8.2.3 Intracranial Large Artery Stenosis
- •8.2.4 Cerebral Small Vessel Disease
- •References
- •9: Anesthesia for Carotid Surgery and Stenting: Neuromonitoring and Perioperative Care
- •9.1 General Preoperative Evaluation for Carotid Endarterectomy
- •9.2 Choice of Anesthesia
- •9.2.1 General Anesthesia
- •9.2.2 Locoregional Anesthesia
- •9.2.2.1 Cervical Plexus Block
- •9.2.2.2 Cervical Epidural Anesthesia
- •9.2.3 Conversion from Local/Regional to General Anesthesia
- •9.3 Neurologic Monitoring
- •9.6 Perioperative Complication
- •References
- •10: Carotid Angioplasty and Stenting
- •10.1 Introduction
- •10.2 Method
- •10.4 Our Personal Experience
- •10.4.1 Inclusion and Exclusion Criteria
- •10.4.3 Early Complications
- •10.4.4 Late Complications
- •10.4.5 Other Uses of Angioplasty and Stenting in the Carotid Territory
- •Bibliography
- •11: Carotid Endarterectomy
- •11.1 Surgical Technique
- •11.2 Conclusive Remarks
- •References
- •12: Vertebral Artery Revascularization
- •References
- •13: Extensive Cerebrovascular Arterial Revascularization
- •13.1 Simultaneous Bilateral Carotid Endarterectomy
- •13.2 Synchronous Carotid and Vertebral Artery Revascularization
- •13.2.1 CEA + VA Reimplantation
- •13.3 Occlusive Disease of the BCT
- •13.5 Aortic Arch Syndrome
- •13.6 Revascularization of the ECA
- •13.7 ICA Thrombectomy
- •13.8.1 CEA + CCA-to-SCA Bypass + Bypass on V3
- •13.9 Particular Situations
- •13.10 Conclusive Remarks
- •References
- •14: Cervico-cerebral Arteries Dissection
- •14.1 Cervical Artery Dissection
- •14.1.1 Epidemiology, Pathophysiology, and Risk Factors for Cervical Artery Dissection
- •14.1.3 Acute Treatment and Secondary Prevention in Patients with CAD
- •14.2 Intracranial Artery Dissection
- •14.2.1 Epidemiology, Pathophysiology, and Risk Factors for Intracranial Artery Dissection
- •14.2.2 Clinical Symptoms
- •14.2.3 Treatment of IAD
- •14.3 Carotid Artery Dissection
- •14.3.1 Common Carotid Artery Dissection
- •14.3.2 Extracranial Internal Carotid Artery Dissection
- •14.4 Vertebral Artery Dissection
- •References
- •15: Extracranial Carotid and Vertebral Artery Aneurysm
- •References
- •16: Asymptomatic Carotid and Vertebral Artery Stenosis
- •References
- •17: Lessons from Experimental-Induced Atherosclerosis: Valuable for the Precision Medicine of Tomorrow
- •17.1 Introduction
- •17.2.2.1 Cytokines
- •17.2.2.2 Chemokines
- •17.3.3 Role of NADPH Oxidase Complex
- •17.4 Nanotechnology-Based Therapies: A New Prospect for Diagnosis and Treatment of Atherosclerosis
- •17.4.1 Designing “Smart” Nanocarriers
- •17.4.2 Nanoparticles Designed to Diagnose Atherosclerosis
- •17.4.8 Nanoparticles Designed to Modulate LDL and HDL Levels
- •17.4.12 Clinical Use of Nanoparticles for Diagnosis and Therapy of Atherosclerosis
- •References
- •18: Choice of the Proper Therapeutic Measure in the Individual Patient and Prevention of Stroke

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Fig. 7.13 CTA is the only current angiographic method that allows
such high-resolution images of contrast-fi lled vessels, plaque, and surrounding soft tissues
Fig. 7.12 When the residual lumen is elongated in the axial plane, the stenosis at its narrowest point can be measured with much more accuracy
than on angiograms
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175
Fig. 7.14 In the case of very coarse wall calcifi cations, properly wide window level settings can reduce beam-hardening artifacts
Fig. 7.15 Axial sections are
obtained by using a postprocessing reconstruction
technique, such as MIP and/or
SSD reconstruction (MIP is more
reliable than SSD)
7 Diagnostic Approach to Cerebrovascular Disease: CT and MRI

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Fig. 7.16 In very severe stenosis, the decrease in distal ICA diameter
precludes an accurate measurement of the degree of stenosis by the
NASCET method and usually leads to a classifi cation of cases with
ICA diameter lower than that of the external carotid artery as near
occlusion (in image ACI, ACE - Internal and External Carotid Arteries,
PRE-OCL - Preocclusion)
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177
Fig. 7.17 MPR: in cases of very short stenosis, the assessment of the stenosis on axial images can become diffi cult, in which cases MIP recon-
structions can help
Fig. 7.18 In cases of tortuous vessels, make interpretation of images diffi cult; MIP reconstructions can help
7 Diagnostic Approach to Cerebrovascular Disease: CT and MRI

178
Fig. 7.19 MIP: calcifi cations are easily detected on MIP and can artifi cially shrink the diameter of the artery, but this might theoretically be
avoided with multiple view angles
Fig. 7.20 MIP: the readers attempts to select a windowing producing the best available edge detection for each vessel
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Fig. 7.21 The advantage of SSD resides in the possibility of moving, rotating, and adjusting for tilt
7 Diagnostic Approach to Cerebrovascular Disease: CT and MRI

180
Fig. 7.22 Even in the absence of calcifi cation, SSD frequently underestimated the degree of stenosis (indicated by arrow in image), probably due
to the arbitrary selection of the lower threshold
Fig. 7.23 SSD, MPR, and MIP reconstructions: accuracy is reduced on SSD reconstruction if extensive calcifi cation is present, but this limitation
can be avoided when MPR and MIP reconstructions are used
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Fig. 7.24 When using an automated 3D CTA analysis program, several misregistrations of the enhanced carotid artery lumen were noticed
Fig. 7.25 The major disadvantage of the technique is represented by the need to post-process data to remove calcifi cations, veins, and bone struc-
tures from the images
7 Diagnostic Approach to Cerebrovascular Disease: CT and MRI

182
Fig. 7.26 Stents in the carotid artery may cause severe streak artifacts
Fig. 7.27 Obese patients are suboptimally imaged
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Fig. 7.28 Motion artifacts (as shown by arrow in image)
7 Diagnostic Approach to Cerebrovascular Disease: CT and MRI
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