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Ординатура / Хирургия / Библиотека им академика М.И. Перельмана / Книга_4366_Библиотеки_им_академика_М_И_Перельмана

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and periosteal fibers
Chapter  Advanced Rhinoplasty Anatomy 25
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INTERNAL NASAL ANATOMY: SEPTUM AND TURBINATES
e central supporting system of the nose is the septal cartilage, which articu­lates posteriorly with the perpendicular plate of the ethmoid superiorly and the vomer inferiorly. e tongue-and-groove articulation between the quadrangular cartilage and the maxillary and palatine crest deserves special mention.
,,
e vomer itself rests on the maxillary-palatine crests.
Perichondrial layers
Septal cartilage
Maxillary/
palatine crest
Crossed perichondrial
Periosteal layers
e perichondrium of the cartilage is only partially contiguous with the peri­osteum of the crests. Other bers pass through the articulation to join the contralateral perichondrium. is crossed conguration makes a contiguous submucoperichondrial dissection dicult at the osteocartilaginous junctions.
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is same anatomic conguration also allows some movement between the crest and the septum, and it is this instability that explains the frequent posttraumatic ndings of a displaced quadrangular septal cartilage from the groove of the crest. e anterior septum articulates caudally with the anterior nasal spine.
Middle
turbinate
Superior turbinate
Inferior turbinate
Parasagittal view of lateral nasal wall
e inferior turbinates are a key functional component in nasal airway breathing, because their anterior heads occupy a signicant portion of the nasal passage. ey are composed of dense lamellar bone originating from the medial maxil­lae and are covered with erectile mucosal tissue. is tissue is under autonomic control, and chronic inammation can lead to brous deposition and chronic hypertrophy of the turbinate so tissues and/or bone.
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Clinical Applications
Autologous septal cartilage is a valuable commodity, with many indicated uses in plastic surgery. In fact, it is the preferred material for numerous gras in modern rhinoplasty. When harvesting these gras, an intact L-strut must be maintained for support. e width of the dorsal and caudal aspects of the L-strut will depend on the quality of the septal cartilage. With thick, strong cartilage, a minimum of  to  mm may be enough to ensure long-term stability. More commonly, the L-strut should be approximately  mm wide.
Aesthetic shaping of the septum may be performed to sculpt the nasal dorsum, help adjust the projection and rotation of the nasal tip, and improve the alar­columellar relationship.
e septum may play a less important role as a primary cause of nasal airway obstruction. In fact, not all deviated septa need correction, because it is common to have an asymptomatic septal deviation. When deviation occurs anteriorly and inferiorly (that is, in the area of the internal nasal valve), it is more likely to be a source of obstruction as a result of the smaller cross-sectional area of the airway in this region. Portions of the septum causing airway obstruction should be re­positioned to the midline or removed. However, cartilage preservation should always be prioritized.
When harvesting septal cartilage and/or removing posterior septal deviations, it is important to perform a gentle sidewise fracture of the bony part of the per­pendicular plate of the ethmoid, which is in continuity with the cribriform plate. ese fractured bony fragments should be easy to remove. Otherwise, any re­maining bony or so tissue attachments should be completely detached. is avoids injury to the cribriform plate and the resulting cerebrospinal uid rhi­norrhea.
Enlarged turbinates may cause and/or contribute to airway compromise in some patients. Numerous treatment options have been proposed in the literature to re­duce the mass of the turbinates and therefore improve the passage of air through the internal valve. In general, more limited approaches to turbinoplasty should be performed, because complete turbinectomy may put the patient at increased risk for developing atrophic rhinitis postoperatively.
e dorsal septum has a T geometry in cross-section. Techniques such as com­ponent dorsal hump reduction, spreader gras, and aps for both functional and aesthetic reasons aim to preserve or recreate this anatomy.
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KEY POINTS
Excellent rhinoplasty results can only be obtained if the surgeon has a thorough knowledge of nasal anatomy and a grasp of the surgical relevance of alteration of the anatomic structures.
e type, texture, and sebaceous content of the skin must be carefully analyzed, because it will inuence the approach for modifying the framework and there­fore the nal result.
An active depressor septi nasi muscle can be identied on preoperative clini­cal analysis, and its modication intraoperatively can enhance the tip-lip re­lationship.
When the open approach is used, alar base excisions that extend more than mm superior to the alar groove and defatting the nasal tip should be avoided to prevent vascular compromise of the nasal tip.
e brous attachments of the lower lateral cartilages to the septal angle, up­per lateral cartilages, piriform aperture, caudal septum, and premaxilla provide support and determine the position of the tip.
REFERENCES
1. Hewell TS, Tardy ME. Nasal tip renement: reliable approaches and sculpture techniques. Facial
Plast Surg :, .
2. McCollough EG, Mangat D. Systematic approach to correction of nasal tip in rhinoplasty. Arch
Otolaryngol :-, .
3. Bernstein L. A basic technique for surgery of the nasal lobule. Otolaryngol Clin North Am :-
, .
4. Dingman RO, Natvig P. e infracartilaginous incision for rhinoplasty. Plast Reconstr Surg :-
, .
5. Janeke JB, Wright WK. Studies on the support of the nasal tip. Arch Otolaryngol :-, .
6. Beekhuis GJ. Nasal septoplasty. Otolaryngol Clin North Am :-, .
7. Toriumi DM, Mueller RA, Grosch T, et al. Vascular anatomy of the nose and the external rhino-
plasty approach. Arch Otolaryngol Head Neck Surg :-, .
8. Wu WT. e Oriental nose: an anatomical basis for surgery. Ann Acad Med Singapore :-,
.
9. Cakir B, Oreroğlu AR, Doğan T, et al. A complete subperichondrial dissection technique for rhino-
plasty with management of the nasal ligaments. Aesthet Surg J :-, .
10. Rohrich RJ, Huynh B, Muzaar AR, Adams WP Jr, Robinson JB Jr. Importance of the depressor septi
nasi muscle in rhinoplasty: anatomic study and clinical application. Plast Reconstr Surg :­; discussion -, .
11. Daniel RK, Glasz T, Molnar G, et al. e lower nasal base: an anatomical study. Aesthet Surg J
:-, .
12. Rohrich RJ, Gunter JP, Friedman RM. Nasal tip blood supply: an anatomic study validating the
safety of the transcolumellar incision in rhinoplasty. Plast Reconstr Surg :-; discussion -, .
13. Sheen JH, Sheen AP. Aesthetic Rhinoplasty, ed . St Louis: Quality Medical Publishing, .
14. Ford CN, Battaglia DG, Gentry LR. Preservation of periosteal attachment in lateral osteotomy. Ann
Plast Surg :-, .
15. Tardy ME, Denney JC. Micro-osteotomies in rhinoplasty. Facial Plast Surg :-, .
Chapter  Advanced Rhinoplasty Anatomy 29
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16. Hilger JA. e internal lateral osteotomy in rhinoplasty. Arch Otolaryngol :-, .
17. Constantian MB. e incompetent external nasal valve: pathophysiology and treatment in primary
and secondary rhinoplasty. Plast Reconstr Surg :-; discussion -, .
18. Constantian MB, Clardy RB. e relative importance of septal and nasal valvular surgery in cor-
recting airway obstruction in primary and secondary rhinoplasty. Plast Reconstr Surg :-; discussion -, .
19. Sheen JH. Spreader gra: a method of reconstructing the roof of the middle nasal vault following
rhinoplasty. Plast Reconstr Surg :-, .
20. Gunter JP, Rohrich RJ. e external approach for secondary rhinoplasty. Plast Reconstr Surg :-
, .
21. Peck GC. e onlay gra for nasal tip projection. Plast Reconstr Surg :-, .
22. Horton CE. Achieving more nasal tip projection by use of small autogenous vomer or septal car-
tilage gras. Plast Reconstr Surg :, .
23. Adams WP Jr, Rohrich RJ, Hollier LH, et al. Anatomic basis and clinical implications for nasal tip
support in open versus closed rhinoplasty. Plast Reconstr Surg :-; discussion -, .
24. Lee MR, Malafa M, Roostaeian J, Unger JG, Geissler P, Rohrich RJ. So tissue composition of the
columella and clinical relevancy in rhinoplasty. Plast Reconstr Surg (in press).
25. Pollock RA, Rohrich RJ. Inferior turbinate surgery: an adjunct to the successful treatment of nasal
obstruction in  patients. Plast Reconstr Surg :-, .
26. Gunter JP, Rohrich RJ. Management of the deviated nose: the importance of the septal reconstruc-
tion. Clin Plast Surg :-, .
27. Howard BK, Rohrich RJ. Understanding the nasal airway: principles and practice. Plast Reconstr
Surg :-, .
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3
Nasal Physiology
Michael R. Lee  Rod J. Rohrich  Jamil Ahmad
A thorough understanding of nasal physiology is essential to provide opti-
mal results in rhinoplasty. Although achieving aesthetic improvement is oen the focus of surgery, nasal function is of equal and, occasionally, primary impor­tance. Surgeons should understand the basic functions of the nose and its inter­nal structures. An intimate relationship exists between the anatomy of the nose and its associated functions. An understanding of normal physiology and the aberrations that occur in disease states enable the surgeon to provide a compre­hensive approach to treatment. When this understanding guides surgical plan­ning and execution, existing deformities are corrected and untoward functional consequences can be avoided.
ere is an intimate relationship between the anatomy of the nose and its asso­ciated physiology. An understanding of this relationship provides the basis for both nasal analysis and treatment planning.
Approximately half of the total airway resistance occurs in the nasal cavity, un­derscoring its signicant role in the regulation of airow through the respiratory tract. However, nasal function extends beyond the role of the nose as a simple conduit. Inspired air is ltered, humidied, and warmed in preparation for con­tinued passage toward the nasopharynx. e nose also contributes to such func­tions as smell and phonation.
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e nasal airway is responsible for 50% of overall airway resistance, underscor­ing its importance in nasal airow
Nasal physiology is inuenced by the external and internal structures of the nose. Nasal structures can be conceptually divided into the bony and cartilaginous framework and the nasal mucosa and so tissues. ese two components work in concert to provide optimal function of the nose and nasal cavity. Surgeons should be aware of the interplay between these two components, because both are crucial to properly addressing nasal dysfunction.
Mucosal disease is infrequently cured with surgical manipulation alone and of­ten requires medical management. Improvement in mucosal disease may occur if the condition is caused by underlying anatomic deformities that are addressed during surgery. With normal mucosal behavior and isolated anatomic deformi­ties, surgery should result in an improvement in nasal airow.
Nasal airway obstruction may have a structural and/or functional cause, and accurate diagnosis will guide appropriate treatment planning.
NASAL FUNCTION
e primary function of the nose is to provide a passageway for external air to be transported to the pulmonary system for blood oxygenation. Despite the rela­tively short length of the nasal cavity compared with the length of the remain­ing airway, approximately half of total airway resistance occurs within the nose. Airow patterns through the nose and upper airway are well understood, since they follow basic physical laws.
In addition to respiration, the nose and nasal cavity perform an additional six functions: particle ltration, air humidication, temperature modication, ol­faction, phonation, and chemoreception.
Respiration
External air is transported into and through the nasal cavity by a generated pres­sure dierential. e negative intranasal pressure generated during inspiration allows air movement and is inuenced by the velocity of airow and resistance
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along the nasal cavity. Initiation of inspiration is associated with nostril enlarge­ment to accommodate the inux of external air into the nose and through the external nasal valve. Inspired air continues through the nasal cavity in a para­bolic curve pattern following this pressure gradient. Most of the air continues over the inferior turbinate, passing through the middle meatus; the greatest re­sistance occurs at the internal nasal valve.
,
Superior turbinate
Middle turbinate
Internal nasal valve
Inferior turbinate
Most of the air continues over the inferior turbinate, passing through the middle meatus; the greatest resistance occurs at the internal nasal valve.
Approximation of the caudal upper lateral cartilage and nasal septum creates an angle at the internal nasal valve. is anatomic location is frequently susceptible to collapse from negative pressure generated at the beginning of inspiration. Ex­piration has the converse eect as the internal nasal valve is enlarged and nostril size becomes smaller.
Airow may be described as laminar or turbulent. Laminar ow is organized and predictable, following a straight pattern. Such a ow pattern exhibits high eciency, with centrally rapidly moving particles anked by more stagnant pe­ripheral particle movement. Conversely, turbulent ow is less predictable and follows a more random pattern. Such ow is the result of disrupted laminar ow and requires an increased pressure gradient to correct. Turbulent ow is com­posed of whorls and eddies that allow increased particle contact with mucosal surfaces. Because the nasal cavity is not a tubular structure, nasal airow is not truly laminar in nature. Instead, the parabolic curve pattern of airow is another mechanism that contributes to turbulent airow and increases resistance to air­ow. At low pressures less than cm of water, airow approximates laminar ow.
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us quiet respiration tends to create a more laminar ow pattern, whereas more labored inspiration is associated with turbulent ow patterns.
Nasal airow is best described using several basic physical laws. Air is introduced through both nostrils and directed into the nasal vestibule. e following for­mula relates nasal airow with resistance at the level of the external nasal valve:
1/Total resistance 5 /Le nostril resistance 1 /Right nostril resistance
Inspired air may meet resistance at the external nasal valve before reaching the internal nasal valve. Obstruction or dysfunction of either nostril leads to an overall increase in nasal airway resistance. Airow follows the path of least re­sistance. For example, air may be shunted in the case of an anterior septal perfo­ration. By following the path of least resistance, a perforation allows the airow from the more resistant side to redirect to the less resistant side, thus lowering total resistance.
Ohm’s Law
Ohm’s law has been used to describe uid ow and states that ow is directly proportional to the dierence in pressure (dP) and inversely proportional to the resistance:
Flow 5 dP/Resistance
Ohm’s law applied to nasal physiology stresses the importance of structural integ­rity and patency to accommodate airow pressure gradients. e cartilaginous infrastructure must be sucient to withstand the negative pressures generated during inspiration, or collapse will exacerbate obstructive symptoms during in­spiration. Anatomic deformities that lead to increased resistance will decrease airow. us increased pressure is required to generate airow. is increased pressure may result in greater deformation of nasal structures leading to yet greater obstructive symptoms.
Bernoulli’s Principle
Bernoulli’s principle establishes that airow is equal at opposing ends of a tube when the diameter is equivalent. A decrease in diameter leads to a lower pres­sure and increased velocity. An example of this eect can be seen at the level of the nasal valves. If the internal nasal valve is restricted, velocity of airow is increased, generating greater negative pressure. Greater negative pressure po­tentiates further internal valve collapse and may worsen obstructive symptoms.