Хирургические болезни. Практикум = Surgical diseases. Practice book. Учебное пособие
.pdfe)deterministic conditionality of the main and lining cells of the gastric mucosa.
41.Features of the development of breast cancer are:
a)the same lesion of the right and left MG;
b)the most frequent localiza-
tion in the upper-outdoor quadrant; c) indicator of aggressive-
ness – the size of lesion;
d)size of lesion does not reflect the degree of aggression;
e)type of growth reflects the degree of malignancy.
42.By type of growth, breast cancer is divided into:
a)mastitis-like;
b)nodular;
c)infiltrative;
d)diffuse;
e)latent.
43.According to the histological type, breast cancer is divided into:
a)infiltrating;
b)noninfiltrating;
c)the armor;
d)separate species;
e)mixed type (Paget’s dis-
ease).
44.The nodular form of breast cancer is clinically manifested:
a)presence of palpable lesion of MG;
b)the presence of palpable dense axillary and supraclavicular lymph nodes;
c)the retraction of the skin or nipple;
d)erysipelas like skin changes;
e)edema of the hand on the side of the lesion.
45.The main mammographic signs of breast cancer are:
a)focal shadows with uneven contours and inhomogeneous density;
b)an increase in peripheral lymph nodes;
c)revealing of microcalcinates along the border of the ducts of the MG, going towards the nipple;
d)areas of darkening in the region of the foci of fibrosis;
e)shadows of connective tissue strands.
46.Possibilities of positron emission tomography in breast cancer allow us to identify:
a)the existing tumor more than 2 cm in diameter;
b)latent tumor up to 1 cm in diameter;
c)distant metastases;
d)tumor more than 5 cm in dia-
meter;
e)tissue destruction.
47.For the prognosis of breast cancer, the main immunohistochemical studies are:
a)the receptors of estrogen and progesterone;
b)a marker of the proliferation of Ki-67;
c)receptors of the human epidermal factor (HER 2);
d)MALT (mucosassociated lymphoid tissue);
e)there is no need for such a
study.
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48.The diffuse forms of breast cancer include all except:
a)purulent-destructive;
b)edematous-infiltrative;
c)gangrenous;
d)latent;
e)mastitis-like.
49.Mastitis-like breast cancer is manifested by:
a)dense palpable tumor with clear contours;
b)infiltrate without signs of fluctuation and clear contours;
c)the presence of cancer lymphadenitis;
d)local increase in tempera-
ture;
e)itching and ulceration of the nipple.
50.Features of Paget’s cancer (breast nipple cancer) are:
a)age of 50–60 years;
b)age of 35–45 years;
c)itching and change in nipple sensitivity;
d)presence of eczematous changes or erosions of the nipple;
e)appearance of late infiltrate, not associated with the nipple and without signs of fluctuation.
51.A latent form of breast cancer manifests by all except:
a)dense palpable tumor;
b)diffuse skin tickening without the presence of a palpable tumor;
c)bloody discharge from the nipple;
d)mastitis-like infiltrate with the phenomena of cancer lymphangitis;
e)enlarge axillary lymph nodes.
52.Halstead Radical mastectomy in breast cancer is:
a)removal of MG with lymph nodes and fatty tissue (lymphadenectomy);
b)removal of both pectoral muscles;
c)removal of only the pectoralis minor muscle;
d)preservation of pectoral muscles;
e)removal of the adjacent skin 5 cm from the edge of the breast.
53.Radical mastectomy in the PA for breast cancer is:
a)extended sectoral breast resection;
b)removal of breast cancer with lymphandenectomy;
c)preservation of pectoral muscles;
d)removal of only the pectoralis minor muscle;
e)removal of both pectoral muscles.
54.Madden Radial mastectomy in breast cancer is:
a)extended sectoral resection of MG;
b)removal of the breast with lymphadenectomy;
c)removal of both pectoral muscles;
d)removal of the pectoralis minor muscle;
e)preservation of both pectoral muscles.
55.Certain types of organ-pre- serving operations in breast cancer are possible with:
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a)the presence of regional metastases in the lymph nodes;
b)the size of the primary tumor up to 5 cm;
c)germination of large pectoralis and fascia;
d)the size of the primary tumor up to 1cm;
e)absence of regional metastases.
56.Preoperative chemotherapy for breast cancer is called:
a)adjuvant;
b)neoadjuvant;
c)preventive;
d)adical;
e)latent.
57. Neoadjuvant chemotherapy for breast cancer aims at:
a)elimination of an existing tumor;
b)regression of the primary tumor to increase its resectability;
c)prevention of possible metastasis;
d)destruction of existing regional micrometastases;
e)destruction of green metas-
tases.
58.The main indicators for the appointment of adjuvant chemotherapy for breast cancer are:
a)ostoperative effect on the undeveloped tumor;
b)evention of metastasis in the I–II stages of the disease;
c)revention of metastasis in the II–III stages of the disease;
d)alliative effect at stage I of the disease;
e)revention of infection of a postoperative wound.
59.The main indications for preoperative radiotherapy for breast cancer are all except:
a)decrease in the number of relapses after organ-preserving operations;
b)increase the ablasticity of the planned operation;
c)increasing the resectability of the tumor more than 5 cm in diameter;
d)increase the resectability of the tumor more than 2 cm in diameter;
e)blocking the function of the ovaries.
60.The main indications for postoperative radiotherapy for breast cancer are:
a)decrease in the number of relapses after organ-saving operations;
b)increased resectability of the tumor more than 5 cm in diameter;
c)increasing the ablastity of the operation;
d)treatment of identified regional metastases;
e)prevention of distant metas-
tases.
61.After surgical treatment of breast cancer in the period of premenopause, the following are used:
a)progestins;
b)aromatase inhibitors;
c)antiestrogens;
d)surgical or radial methods;
e)analogues of gonadotropinreleasing hormone.
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Answers on the topic “Diseases of the mammary glands”
Question № |
Correct answer |
Question № |
Correct answer |
Question № |
Correct answer |
1 |
b; c; d; e |
22 |
а; c; d; e |
43 |
а; b; d; e |
2 |
c; d |
23 |
d; e |
44 |
а; b; c; e |
3 |
b; c |
24 |
d |
45 |
а; b; c |
4 |
а; e |
25 |
d; e |
46 |
b; c |
5 |
b; d |
26 |
а; c; d |
47 |
а; b; c |
6 |
а; d; e |
27 |
а; b; e |
48 |
а; c; d |
7 |
d |
28 |
а; b; c; d |
49 |
b; c; d |
8 |
а; e |
29 |
e |
50 |
а; c; d; e |
9 |
c |
30 |
а; b; d; e |
51 |
а; b; c; d |
10 |
а; b; c; e |
31 |
b; c; d |
52 |
а; b |
11 |
c; d |
32 |
b; d; e |
53 |
b; d |
12 |
b; c; d |
33 |
a; b; d |
54 |
b; e |
13 |
b; c; e |
34 |
e |
55 |
d; e |
14 |
e |
35 |
a; e |
56 |
b |
15 |
а; b; c; e |
36 |
а; b; c; d |
57 |
b; d |
16 |
а; d |
37 |
b; e |
58 |
c |
17 |
а; b; c; d; e |
38 |
b; c; d; e |
59 |
а; d; e |
18 |
d |
39 |
d; e |
60 |
а; d |
19 |
c; e |
40 |
а; c |
61 |
d; e |
20 |
d |
41 |
а; b; c; e |
|
|
21 |
а |
42 |
b; d |
|
|
Control test on Chapter 7 “Purulent diseases of the lungs and pleura”
1.To the acquired purulent-de- structive diseases of the bronchopulmonary system and the pleura belong all below except:
a)bronchoectatic disease;
b)acute and chronic lung abscess;
c)acute purulent pleuritis;
d)gangrene of the lungs;
e)Mounier-Kuhn’s syndrome (tracheobronchomegaly).
2.Bacterial lung destruction (BLD) includes the following processes, except:
a)acute and chronic pleural empyema;
b)gangrene of the lungs;
c)pulmonary sequestration;
d)acute and chronic lung ab-
scess;
e)Mounier-Kuhn’s syndrome (tracheobronchomegaly).
3.External manifestations of respiratory failure in purulent diseases of the lungs and pleura include:
a)sitting position with the upper shoulder girdle fixation;
b)acrocyanosis of the face;
c)thickening of the terminal phalanges of the fingers as “drumsticks”;
d)bulging of the cervical veins;
e)wideningoftheintercostalspaces.
4.The main indicators of the external respiration function in pa-
144
tients with purulent diseases of the lungs and pleura are:
a)vital capacity of the lungs;
b)minute volume of respiration;
c)maximum ventilation of the
lungs;
d)Tifno-votchal’s test;
e)increase in the volume of “deadspace”.
5.The main pathologies of the pulmonary function in purulent diseases of the lungs and pleura are:
a)general hypoventilation;
b)development of the “shunting” syndrome in the damaged parts of the lung);
c)imbalance between ventilation and perfusion;
d)Tifno-Votchal’s test;
e)impairment of alveolar and blood gases diffusion.
6.Radioisotope lung scintigraphy in case of purulent diseases of the lungs and pleura makes it possible for the doctors:
a)to estimate the amount of fluid in the pleural cavity;
b)to detect neoplasms of the tracheobronchial tree;
c)to assess the state of the lungs and mediastinum in relation to the surrounding tissues;
d)to assess the state of ventilation and perfusion of various parts of the lung;
e)to verify the structure of the bronchi or bronchiectasis.
7.Bronchography in patients with purulent diseases of the lungs and pleura cnobles the doctors:
a)to assess the condition of the lungs and mediastinum in relation to the surrounding tissues;
b)to assess the state of ventilation and perfusion of various lung parts;
c)to estimate the amount of fluid in the pleural cavity;
d)to verify the structure of the bronchi or bronchiectasis;
e)to assess the vital capacity of the lungs.
8.According to the form of enlargement bronchiectases are divided into:
a)serpentine;
b)cylindrical;
c)sac-like;
d)cyst-like;
e)mixed.
9.Bronchiectasic disease has the following stages, except:
a)latent;
b)reversible changes;
c)irreversible changes;
d)hemodynamic disorders;
e)deformation of the chest.
10.According to the development mechanism acute lung abscesses are divided into:
a)hematogenous-embolic;
b)aspiration;
c)post-traumatic;
d)lymphogenous;
e)post-pneumonic.
11.The predisposing factors of the acute lung abscess development include everything below, except:
a)chronic nonspecific lung diseases;
b)diseases accompanied by local and general immunoresistance decrease;
c)diseases accompanied by hemocoagulation disorders;
145
d)processes accompanied by cough and epiglottis reflexes pathology;
e)diseases, accompanied by the thyroid hormone synthesis disorder.
12.The main mechanisms of the acute lung abscess pathogenesis are all below, except:
a)translocation of microflora to the lower parts of the tracheobronchial tree;
b)hypoventilation and microatelectasation of the bronchial-al- veolar structures;
c)disorders of microcirculation with vascular thrombosis;
d)osteomalacia of the tracheal rings from the outside;
e)necrosis of bronchial-alveo- lar structures.
13.The most typical localizations of acute lung abscess are the following segments:
a)1st;
b)2nd;
c)3rd;
d)4th;
e)5th.
14.The following periods are distinguished during the acute lung abscess:
a)latent;
b)asymptomatic;
c)formation of the abscess;
d)breakthrough of the abscess into the bronchus;
e)restitution.
15.The period of acute lung abscess formation is characterized by all signs below, except:
a)hectic temperature;
b)pain in the chest during breathing;
c)dry cough;
d)abundant discharge of purulent sputum;
e)severe general condition.
16.The period of acute abscess rupture into the bronchi is characterized by:
a)deterioration of the general condition;
b)sudden onset of cough with abundant purulent sputum (“full mouth”);
c)improvement in general condition;
d)tendency of the body temperature to normalization;
e)disappearance of dyspnoea.
17.Acute lung abscess can be drained:
a)into pericardial cavity;
b)into pleural cavity;
c)into abdominal cavity;
d)into bronchus cavity;
e)hematogenously.
18.The period of acute lung abscess development is radiologically manifested by:
a)a “basket” symptom;
b)multiple foci of pulmonary destruction;
c)hydrothorax;
d)parietal pneumothorax;
e)a rounded homogeneous cavity surrounded by perifocal infiltration.
19.The radiological symptom of a “basket” (an abscess cavity containing a liquid layer with a gas bubble above it) is caused by:
a)fibro-inflammatory fusions around;
b)changes in the walls of the abscess;
146
c)perifocal infiltration;
d)inadequate bronchial drainage;
e)pneumohydrothorax.
20.Sputum, appearing after the drainage of an acute abscess into the bronchi:
a)forms 3 layers upon sedimen-
tation;
b)homogeneous in structure;
c)foam-purulent;
d)brown and homogeneous;
e)forms 2 layers upon sedimentation.
21.Treatment of acute lung abscess includes everything below, except for:
a)adequate drainage of the purulent focus into the tracheobronchial tree;
b)pleural adhesiolysis;
c)adequate antibiotic therapy;
d)increased immunoresistance;
f)disintoxication therapy.
22.To ensure adequate drainage of acute lung abscess, everything below is applied, except:
a)postural drainage;
b)endotracheal sanation;
c)Mohnaldi’s percutaneous microdrainage;
d)pleurodesis and decortication of the lung;
e)videothoracoabsessoscopy.
23.Indications for surgical treatment of acute lung abscess are:
a)inefficiency of drainage with the help of minimally invasive techniques;
b)complications;
c)chronic abscess;
d)gangrene of the lung;
e)lack of dynamics in the puru-
lent cavity healing during 4 weeks.
24.The operation of choice in case of acute lung abscess is:
a)pneumonectomy;
b)pleural adhesiolysis;
c)decortication of the lung;
d)lobe, bilobectomy with the removal of the pyogenic capsule and the destruction cavity;
e)thoracomioplasty of the purulent cavity.
25.Optimal routes of antibiotics administration in case of acute lung abscess are as follows:
a)intramuscular;
b)intravenous;
c)oral;
d)selective intra-arterial;
e)selective intralesional (performed by means of Mohnaldi’s percutaneous drainage or sanation bronchoscopy).
26.Pyopneumothorax with acute lung abscess develops due to:
a)drainage into the large bron-
chus;
b)communication between the second purulent cavity, connected with the bronchus;
c)development of a dense con- nective-tissue pyogenic shell;
d)drainage into the pleural cavity;
e)inadequate blood transfusion therapy.
27.The optimal outcome of acute abscess is:
a)chronic abscess;
b)pneumothorax;
c)localized pneumosclerosis;
d)chronic pleural empyema;
e)amyloidosis.
28.Postural drainage in the treatment of acute lung abscess includes:
147
a)percutaneous microdrainage of the abscess cavity;
b)creation of a specific system for vacuum-drainage of the purulent cavity;
c)placing the patient in a position for natural adequate drainage from the mild destruction cavity;
d)creating a system for flowwashing drainage;
e)varieties of sanative bronchoscopy.
29.The operation of choice for the lung gangrene is:
a)bilobectomy;
b)pleural adhesiolysis;
c)lung decortication;
d)pneumonectomy;
e)thoracoabsessocopy.
30.Acute empyema of the pleura is called:
a)limited purulent destructive process of the lung tissue;
b)acute inflammation of the pleura layers with purulent exudates;
c)widespread destruction of the lung tissue;
d)limited purulent-destructive disease of the lung tissue;
e)transsudate in the pleural cavity.
31.The main microflora causing the development of acute pleural empyema is:
a)clostridial anaerobes;
b)adenovirus;
c)nonclostridial anaerobes;
d)gram-positive and gramnegative aerobes;
e)fungal flora.
32.Primary acute pleural empyema is a complication of:
a)pus-destructive diseases of the pleural cavity;
b)chest injuries;
c)thoracic interventions;
d)acute lung abscess;
e)transsudate suppuration.
33.The main routs of pleural cavity infection are:
a)aspiration;
b)contact;
c)perforating;
d)hematogenous-metastatic;
e)obturating.
34.The main types of the pleural cavity contact infection are:
a)perforating diseases;
b)parapneumonic foci;
c)metapneumonic diseases;
d)secondary piometastatic foci;
e)suppuration of a thoracic wound.
35.In terms of prevalence, acute pleural empyema is divided into:
a)bilateral;
b)putrefactive;
c)total;
d)polysegmentary;
e)limited.
36.Among the localizations of limited pleural empyema are all below, except:
a)apical;
b)parietal;
c)paramediastinal;
d)metapneumonic;
e)interlobal.
37.By the nature of the exudate, acute pleural empyema is divided into:
148
a)purulent;
b)putrefactive;
c)serous;
d)hemorrhagic;
e)fungal.
38.By the type of complications, acute empyema is divided into processes that occur:
a)without lung destruction;
b)with lung destruction;
c)with pyopneumothorax;
d)with the diaphragm involve-
ment;
e)with pneumonia.
39.In the development of acute pleural empyema there are the following stages:
a)the stage of fibrous pleurisy;
b)the latent stage;
c)the stage of fibrin organization into connective tissue structures (moorings);
d)the reparative stage;
e)the pleurobronchothoracic complications.
40.The stage of fibrin and adhesions formation in acute pleural empyema is characterized by all below symptoms, except:
a)purulent exudate;
b)adhesion of both pleural layers with the fibrosis development;
c)hyperemia and pleura leukocyte infiltration;
d)separation of purulent exudate from the unchanged pleural cavity with spikes;
e)formation of scar tissue.
41.The main complications of acute pleural empyema are all below except:
a)chest soft tissue phlegmons;
b)pyopneumothorax;
c)syndrome of healthy lung tissue bypass;
d)compartment syndrome of the abdominal cavity;
e)dislocation of the mediastinum to a healthy side.
42.Clinical stage of acute pleural empyema is characterized by:
a)strong stitching pain in the chest, worse when inhaled;
b)frequent, shallow breathing;
c)reflex cough;
d)decrease in body temperature against the background of skin pallor;
e)febrile fever with chills and increased sweating.
43.The main methods of acute pleural empyema instrumental diagnosis are:
a)MRI;
b)polypositional radiography;
c)pleural puncture with bacteriological examination;
d)fine needle aspiration biopsy with cytological examination;
e)videothoracopy.
44.The basis of acute pleural empyema treatment is all below, except:
a)drainage of the pleural cavity with active aspiration;
b)postural drainage;
c)rational antibiotic therapy;
d)Mounaldi’s microdrainage;
e)pleurectomy.
45.Indications for acute pleural empyema surgical treatment are:
a)amyloidosis of internal organs;
b)active pleural fistula;
149
c)large sequestration in the empyema cavity;
d)multi-chamber empyema;
e)inadequate postural drainage.
46.In case of pleural empyema surgical treatment preference is given to:
a)wide lateral thoracotomy;
b)videothoracoscopic sanation;
c)mini-thoracotomy;
d)puncture draining interventions under ultrasound and radiographic control;
e)robot-assisted interventions.
47.The aims of videothorascopic sanation of the pleural cavity in case of acute pleural empyema are all below, except:
a)removal of fibrous overlays, moorings and adhesions;
b)removal of sequestrants and blood clots;
c)extrapleural thoracoplasty;
d)rooting of the affected lung;
e)open thoracostomy formation.
48.The manifestation of the clinical pyopneumothorax in acute pleural empyema depends on:
a)pulmonary edema;
b)the size of the lung purulent cavity;
c)the valve mechanism;
d)the diameter of the involved bronchus;
e)the character of empyema (limited or common).
49.The clinically acute form of pyopneumothorax with acute pleural empyema is manifested by all below symptoms, except:
a)sudden severe chest pain with the signs of shock;
b)dyspnea at rest;
c)forced position of the patient with support on the upper shoulder girdle;
d)absence of pain syndrome;
e)increase in the body temperature up to 39 °C accompanied with chills.
50.Valve pyopneurmothorax is accompanied by the following disorders, except:
a)flotation of the mediastinum;
b)development of the healthy lung areas bypass syndrome;
c)Kurnan’s syndrome (increased pressure in the pulmonary vessels caused by general hypoxemia);
d)compartment syndrome in the abdominal cavity;
e)paradox breathing.
51.The main diagnostic methods to reveal pyopneumothorax are:
a)celiacography;
b)diagnostic puncture;
c)fistulopleurobronchography;
d)videothoracoscopy;
e)MRI.
52.In complex treatment of pyopneumothorax, all below methods are used, except:
a)sanitation of the empyema cavity;
b)drainage of the pleural cavity;
c)restoration of airway patency;
d)elimination of respiratory hypoxemia;
e)lobectomy with circular resection of the adjacent bronchi.
53.Causes contributing to the transition of acute pleural empyema to its chronic form are all below, except:
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