- •Type 1diabetes mellitus
- •Type 1diabetes mellitus
- •Diabetes mellitus
- •Diabetes insipidus
- •Type 1diabetes mellitus, inadequate control, ketosis
- •Correlates with determination of potential abnormalities of glucose tolerance
- •Impairment of carbohydrate tolerance
- •Remission of diabetes mellitus
- •Diabetes Insipidus
- •Type 2 diabetes mellitus
- •Type 1diabetes mellitus
- •Definition of the c-peptide level
- •Type 2 diabetes mellitus, diabetic angiopathy, foot ulcers
- •Diabetes insipidus
- •Type 2 diabetes mellitus, inadequate control
- •Type 2 diabetes mellitus, inadequate control, ketoacidosis
- •Type 2 Diabetes mellitus, hard form in the stage of compensation. Chronic pyelonephritis, high pressure disease
- •Type 2 Diabetes Mellitus, inadequate control, diabetic nephropathy, microalbuminuria
- •Oral glucose tolerance test (gtt) and fasting serum lipid studies
- •Determination level of the HbAlc
- •All of the above
- •All of the above
- •Options a and b
- •An a1c can be drawn quarterly in patients whose therapy has just changed or who are not meeting glycemic control. In patients who are at glycemic control, it can be drawn twice a year
- •Tight blood pressure control that includes an angiotensin-converting enzyme inhibitor
- •Type 1diabetes mellitus, diabetic background retinopathy, diabetic foot syndrome
- •Type 1 diabetes mellitus, diabetic distal symmetrical sensorimotor polyneuropathy
- •Type 1 diabetes mellitus in the stage of decompensation
- •Diabetic gastroenteropathy
- •Diabetic nephropathy
- •Diabetes insipidus
- •Ketoacidotic coma
- •Ketoacidotic coma
- •Diabetic ketoacidotic coma
- •Rapid-acting insulin in the dose of 10 u hourly I/V
- •Diabetic ketoacidosis
- •Lactic acidosis
- •Somogyi effect
- •0, 5 Iu/kg/daily
- •Diet with carbohydrate restriction
- •Diet recommendations
- •Transfer to monotherapy by short acting insulin
- •Rapid-acting insulin in the dose of 10 u hourly I/V
- •Can be done, but is not recommended by most physicians
- •Gdm is typically found in the third trimester
- •All of the above
- •Proliferative retinopathy
- •Good and protracted compensation of diabetes mellitus
- •Diet with carbohydrate restriction
- •Diet recommendations
- •Reduction in triglycerides
- •Agree with the mother that her suspicions are probably true
- •Ketoacidotic coma
- •Diffuse toxic goiter
- •Thyrotoxic crisis
- •Endemic diffuse nontoxic goiter of 3 grade
- •Graves’ disease
- •Diffuse toxic goiter
- •Thyrotoxic crisis
- •Thiamazole
- •Nodular goiter 3 grade, thyrothoxicosis
- •Diffuse toxic goiter in decompensation. Thyrotoxic crisis.
- •Nodular goiter
- •Autoimmune (Hashimotos) thyroiditis, hypothyroidism
- •All methods
- •Subacute thyroiditis
- •Syndrome of Van – Vik – Ross – Geness
- •Myxedema coma
- •Nodular euthyreoid goiter
- •Aspirational biopsy
- •Antibiotics
- •Salicylates and corticosteroids
- •Surgery
- •Primary hypoparathyroidism
- •Postoperative hypoparathyroidism
- •Primary hyperparathyroidism
- •Hypoparathyroidism
- •Post-operative hypoparathyroidism
- •Primary hyperparathyroidism
- •Primary hyperparathyroidism, renal form
- •Pseudohypoparathyroidism
- •Primary hyperparathyroidism
- •Primary hyperparathyroidism
- •Hypotension
- •Addison’s disease
- •Intestinal colic
- •In the diagnostic approach to determine hypercortisolism
- •Cushing’s syndrome. Steroid’s diabetes
- •All of the above
- •Conn’s syndrome
- •Phaeochromocytoma
- •All of the above
- •Acute adrenal insufficiency
- •Cushing’s syndrome
- •Salt-wasting congenital adrenal hyperplasia (cah) from mineralocorticoid deficiency
- •Measure plasma vasopressin before and after an infusion of hypertonic saline
- •Patau syndrome
- •Down's syndrome
- •Follicle Stimulating Hormone (fsh)
- •Diabetes mellitus
- •Steroid diabetes
- •Hypothyrosis
- •Diabetes mellitus
- •Diabetes insipidus
- •Diabetes insipidus
- •Pituitary tumor
- •Obesity class III
- •Pubertal-juvenile dispituitarism
- •Alimentary obesity
- •Hypothalamic obesity
- •Adipose-genital dystrophy
- •Arterial hypertension
- •Hypothalamic obesity
- •Hypothalamic obesity, class II
- •Cushing disease, obesity, class II
- •Hypothalamic obesity, class II
- •Alimentary constitutive obesity, class II
- •Shereshevsky-Turner syndrome
- •Secondary sexual characters development
- •Increasing of the thyroxin and triiodothyronine level
- •Diabetes mellitus
- •Melatonin
- •Adrenal gland
- •1,5 Mmol/l
- •Alkaptonuria
- •Down's syndrome
Infectioustoxic shock
Hepatic coma
Diabetic ketoacidotic coma
Hyperosmolar coma
Case N 39. The unconscious woman of 25 years old is delivered to hospital. She is suffering from Diabetes Mellitus for the past 8 years. In the morning the patient could not wake. Objectively: The skin is dry, the turgor is reduced, eyeballs are mild. Kussmaul's respiration, the odor of acetone from the mouth. The pulse - 130 beats /minutes, the blood pressure - 100/65 mmHg. The liver is +2 cm. Lab studies: the glucose of blood - 25,5 mmol/L. The emergency treatment will consist of introduction:
A. Insulin of prolonged acting 10 U hourly i/v
40% glucose solution i/v
Reopolyglukin up to 1,0 L i/v
0,9% NaCl solution i/v
Rapid-acting insulin in the dose of 10 u hourly I/V
Case N 40. Patient X., 67 years old. Duration of type 2 Diabetes Mellitus during 9 years. Patient keeps to the prescribed diet, receives regular treatment with oral drugs, and several times in the few last years was treated by sulfonylureas with maximal therapeutic doses. What complication should you consider?
A. Hyperglycaemia
B. Hypoglycaemia
C. Diabetic ketoacidosis
D. Hyperosmolality
E. Lactic acidosis
Case N 41. Patient M., 67 years old. Duration of type 2 Diabetes Mellitus during 19 years. He treats by Metformin in a daily doses 1500 mg. In anamnesis – cardiovascular disease and chronic bronchitis. He has coma in investigation of pneumonia. Objectively: the skin is wet. Kussmaul's respiration. The pulse - 110 beats /minutes, the blood pressure - 110/70 mmHg. Lab studies: the glucose of blood - 12,5 mmol/L. The ketonemia and acetonuria are absent. Establish the correct diagnosis:
Hyperglycaemia
Hypoglycaemia
Diabetic ketoacidosis
Hyperosmolality
Lactic acidosis
Case N 42. Patient E., 72 years old, is suffering from type 2 diabetes mellitus during 23 years. She treats of biguanides (daily doses - 2 gram). On the second week of pneumonia she is hospitalized to the emergency department. Objectively: somnolence, the skin is wet. Kussmaul's respiration. Muscle tones are normal. Lab studies: the glucose of blood - 13,6 mmol/L. The ketonemia and acetonuria are absent. The emergency treatment will consist of introduction:
40% glucose solution i/v
50-100 ml 1 % methylene-blue solution and trisamine i/v
0,45% NaCl solution i/v
Rapid-acting insulin in the dose of 10 IU hourly i/v
2,5 % sodium bicarbonate solution 1-2 l/day i/v
Case N 43. The patient T., 50 years old, body height of 165 cm, weight of 78 kg, suffers from type 2 diabetes mellitus during 10 years. In connection with the decompensation of carbohydrate metabolism she is transferred to insulin, the daily dose - 60 IU. For last 5 months her weight has increased for 5 kg, night hypoglycemias are observed during this period. The fasting level of glucose of the capillary blood is 15,5 mmol/L. The cause of the decompensation of diabetes mellitus is:
