- •Type 1diabetes mellitus
- •Type 1diabetes mellitus
- •Diabetes mellitus
- •Diabetes insipidus
- •Type 1diabetes mellitus, inadequate control, ketosis
- •Correlates with determination of potential abnormalities of glucose tolerance
- •Impairment of carbohydrate tolerance
- •Remission of diabetes mellitus
- •Diabetes Insipidus
- •Type 2 diabetes mellitus
- •Type 1diabetes mellitus
- •Definition of the c-peptide level
- •Type 2 diabetes mellitus, diabetic angiopathy, foot ulcers
- •Diabetes insipidus
- •Type 2 diabetes mellitus, inadequate control
- •Type 2 diabetes mellitus, inadequate control, ketoacidosis
- •Type 2 Diabetes mellitus, hard form in the stage of compensation. Chronic pyelonephritis, high pressure disease
- •Type 2 Diabetes Mellitus, inadequate control, diabetic nephropathy, microalbuminuria
- •Oral glucose tolerance test (gtt) and fasting serum lipid studies
- •Determination level of the HbAlc
- •All of the above
- •All of the above
- •Options a and b
- •An a1c can be drawn quarterly in patients whose therapy has just changed or who are not meeting glycemic control. In patients who are at glycemic control, it can be drawn twice a year
- •Tight blood pressure control that includes an angiotensin-converting enzyme inhibitor
- •Type 1diabetes mellitus, diabetic background retinopathy, diabetic foot syndrome
- •Type 1 diabetes mellitus, diabetic distal symmetrical sensorimotor polyneuropathy
- •Type 1 diabetes mellitus in the stage of decompensation
- •Diabetic gastroenteropathy
- •Diabetic nephropathy
- •Diabetes insipidus
- •Ketoacidotic coma
- •Ketoacidotic coma
- •Diabetic ketoacidotic coma
- •Rapid-acting insulin in the dose of 10 u hourly I/V
- •Diabetic ketoacidosis
- •Lactic acidosis
- •Somogyi effect
- •0, 5 Iu/kg/daily
- •Diet with carbohydrate restriction
- •Diet recommendations
- •Transfer to monotherapy by short acting insulin
- •Rapid-acting insulin in the dose of 10 u hourly I/V
- •Can be done, but is not recommended by most physicians
- •Gdm is typically found in the third trimester
- •All of the above
- •Proliferative retinopathy
- •Good and protracted compensation of diabetes mellitus
- •Diet with carbohydrate restriction
- •Diet recommendations
- •Reduction in triglycerides
- •Agree with the mother that her suspicions are probably true
- •Ketoacidotic coma
- •Diffuse toxic goiter
- •Thyrotoxic crisis
- •Endemic diffuse nontoxic goiter of 3 grade
- •Graves’ disease
- •Diffuse toxic goiter
- •Thyrotoxic crisis
- •Thiamazole
- •Nodular goiter 3 grade, thyrothoxicosis
- •Diffuse toxic goiter in decompensation. Thyrotoxic crisis.
- •Nodular goiter
- •Autoimmune (Hashimotos) thyroiditis, hypothyroidism
- •All methods
- •Subacute thyroiditis
- •Syndrome of Van – Vik – Ross – Geness
- •Myxedema coma
- •Nodular euthyreoid goiter
- •Aspirational biopsy
- •Antibiotics
- •Salicylates and corticosteroids
- •Surgery
- •Primary hypoparathyroidism
- •Postoperative hypoparathyroidism
- •Primary hyperparathyroidism
- •Hypoparathyroidism
- •Post-operative hypoparathyroidism
- •Primary hyperparathyroidism
- •Primary hyperparathyroidism, renal form
- •Pseudohypoparathyroidism
- •Primary hyperparathyroidism
- •Primary hyperparathyroidism
- •Hypotension
- •Addison’s disease
- •Intestinal colic
- •In the diagnostic approach to determine hypercortisolism
- •Cushing’s syndrome. Steroid’s diabetes
- •All of the above
- •Conn’s syndrome
- •Phaeochromocytoma
- •All of the above
- •Acute adrenal insufficiency
- •Cushing’s syndrome
- •Salt-wasting congenital adrenal hyperplasia (cah) from mineralocorticoid deficiency
- •Measure plasma vasopressin before and after an infusion of hypertonic saline
- •Patau syndrome
- •Down's syndrome
- •Follicle Stimulating Hormone (fsh)
- •Diabetes mellitus
- •Steroid diabetes
- •Hypothyrosis
- •Diabetes mellitus
- •Diabetes insipidus
- •Diabetes insipidus
- •Pituitary tumor
- •Obesity class III
- •Pubertal-juvenile dispituitarism
- •Alimentary obesity
- •Hypothalamic obesity
- •Adipose-genital dystrophy
- •Arterial hypertension
- •Hypothalamic obesity
- •Hypothalamic obesity, class II
- •Cushing disease, obesity, class II
- •Hypothalamic obesity, class II
- •Alimentary constitutive obesity, class II
- •Shereshevsky-Turner syndrome
- •Secondary sexual characters development
- •Increasing of the thyroxin and triiodothyronine level
- •Diabetes mellitus
- •Melatonin
- •Adrenal gland
- •1,5 Mmol/l
- •Alkaptonuria
- •Down's syndrome
Alimentary obesity
Pubertal-juvenile dispituitarism
Hypothalamic obesity
Cushing disease
Cushing syndrome
Case 124. A patient Z., 19 years old, complains of overweight, increased appetite, headache, weakness, fatigue. He had frequent quinsy before. A growth of weight has begun from the age of 14 years, especially it progressed at last year. The patient doesn't limit herself in carbohydrates and doesn't follow any diet. Her mother is obese. Patient's height is 167 cm, body mass is 90 kg, body mass index – 32,2 kg/m2. Adipose cellular disposal is equable. What is the diagnosis?
Adipose-genital dystrophy
Alimentary obesity, class II
Pubertal-juvenile dispituitarism
Hypothalamic obesity, class II
Cushing disease, class II
Case 125. A patient F., 54 year old complains of headache, increased sweating, periodical palpitation, elevation of blood pressure, severe weight gain (30 kg in 2 years). The patient considers himself to be ill for 3 years. He connects his disease with having craniocerebral trauma. Body mass index - 42 kg/m2. There are numerous pink thin stria on the hips and abdomen, the skin is highly wet. Blood pressure is 180/110 mmHg on the left arm and 160/100mmHg - on the right arm. What is the clinical diagnosis?
Hypothalamic obesity, class III
Cushing disease, obesity, class II
Alimentary constitutive obesity, class II
Obesity class III
Arterial hypertension
Case 126. A patient W., 16 years old, has overweight. He was born in asphyxia at premature delivery with body mass 2800 g, length 48 cm. His weight gain has begun at the age of 10. Now his height is 172 cm, body mass is 87 kg. Female stature, gynecomastia are present. Secondary sexual charachters are bad developed - hair growth at the face is absent, pubic hair is lean. What is the diagnosis?
Pubcrtal-juvenilc dispituitarism
Alimentary obesity
Hypothalamic obesity
Gushing disease
Adipose-genital dystrophy
Case 127. A 28 years old patient complains of general weakness, which increases to the evening, frequent headache, thirst. She is ill for 3 years and influenza is supposed to be the cause. Her height is 168 cm, body mass is 88 kg, body mass index – 31,4 kg/m2. Adipose disposal is dysplastic, prevalent on trunk, upper body. The face is round, red. The skin is dry. There are deep-red stria on the skin of the abdomen and hips. What is the diagnosis?
Alimentary obesity, class I
Pubertal-juvenile dispituitarism
Hypothalamic obesity, class II
Cushing disease, obesity class I
Cushing syndrome, obesity class I
Case 128. A patient I., 45 year old complains of headache, increased sweating, periodical palpitation, elevation of blood pressure, severe weight gain (28 kg in 1,5 years). The patient considers himself to be ill for 2 years. He connects his disease with having craniocerebral trauma. Body mass index - 38 kg/m2. There are numerous pink thin stria on the hips and abdomen. Blood pressure is 190/110 mmHg. What is the clinical diagnosis?