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Chapter 85

Finger and Metacarpal Fratures

Oliver Kloeters and John Y.S. Kim

Phalangeal Fractures

Introduction

The bones of the fingers are the most commonly fractured bones in the human body, accounting for roughly 10% of all fractures. Half of all finger fractures occur in the proximal and middle phalanges, and the other half in the distal phalanx. Most phalangeal fractures can be treated nonoperatively with splinting alone. Fractures involving the articular surfaces and those with accompanying tendon or soft tissue injury are more complicated and usually require operative intervention. This chapter will focus on the commonly encountered fractures.

Relevant Anatomy

The fingers are comprised of three bones, the proximal, middle and distal phalanges. Each phalanx is further subdivided into a shaft, neck and head. The bones articulate at the proximal interphalangeal (PIP) and the distal interphalangeal (DIP) joints. The proximal phalanx articulates with the metacarpal bone at the metacarpophalangeal (MP) joint. On the palmar surface, the tendon of flexor digitorum superficialis (FDS) inserts on the volar aspect of the middle phalanx, and the flexor digitorum profundus (FDP) tendon inserts on the volar aspect of the distal phalanx. FDS flexes the PIP joint and FDP flexes primarily the DIP joint. When evaluating a finger for possible tendon injury, it is important to distinguish between these two tendons. Since FDP is the sole flexor of the distal phalanx, the PIP joint has to be immobilized in extension in order to accurately assess FDP function. Evaluation of FDS function requires immobilization of the DIP joints of the other fingers, since the tendons arise from the same muscle belly and can flex the digits simultaneously. Each extensor tendon inserts on the middle and distal phalanges, via the central and lateral bands, respectively. A more detailed discussion of flexor and extensor anatomy can be found in the chapters on flexor and extensor tendon repair.

Clinical Presentation

Presentation of a finger fracture will vary depending on the mechanism and location of the injury. Fracture signs include swelling, hematoma, ecchymosis, deformity, tenderness/pain, decreased range of motion (ROM) and accompanying soft tissue injury. In addition, the possibility of tendon avulsion, collateral ligament damage, or volar plate rupture should be determined. Assessment and documentation of any neurovascular injury must be carried out prior to any therapeutic or diagnostic manipulation, such as stress view radiographs.

Rotation deformity is an often-neglected problem and should be addressed by at the time of fracture fixation, as an untreated rotational deformity can impair ROM

Practical Plastic Surgery, edited by Zol B. Kryger and Mark Sisco. ©2007 Landes Bioscience.

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and functional outcome. A useful clinical test is to ask the patient to flex his fingers while observing the position of the nails and comparing them to the other hand. As a rule, all abnormal findings in the injured hand should be compared to the other hand as individual malformation or previous injuries may lead to an inaccurate diagnosis.

Fracture Patterns

 

Phalangeal fractures can be classified according to the following: (1) open or

 

closed; (2) displaced or nondisplaced; (3) the finger and phalanx involved; (4) frac-

 

ture pattern: oblique, transverse, spiral, or comminuted; (5) any associated soft tis-

 

sue or neurovascular injuries.

 

Imaging

 

Negative plain radiographs in three views (posterior-anterior, oblique and lat-

 

eral) are ample for ruling out most phalangeal fractures. If necessary, additional

 

stress views can be obtained in which the injured phalanx is manually stressed. Since

 

this maneuver is usually painful, a digital block with lidocaine can be helpful. CT

 

and MRI are not indicated for the diagnosis of a phalangeal fracture.

 

Transverse fractures of the proximal phalanx are more likely to result in volar

 

displacement of the distal fragment due to traction on the proximal bone by intrin-

 

sic and extensor tendons. Middle phalanx fractures can result in distal fragment

 

displacement either volarly or dorsally, depending on whether the fracture is proxi-

 

mal or distal to the FDS insertion. Fractures of the distal phalanx are usually related

 

to crush injury involving the nail bed.

 

85

 

Treatment Principles

 

The principal goals of treatment are complete functional restoration and full

 

ROM of the injured finger. This is achieved through precise anatomical alignment

 

(open or closed), and early mobilization. Full immobilization (e.g., splinting) should

 

not exceed 3 weeks as the extensor and flexor tendon systems as well as the capsules

 

will begin to irreversibly contract and stiffen. In general, uninvolved joints should

 

be excluded from splinting (e.g., avoid splitting the PIP and MP joints in the setting

 

of a distal phalanx fracture). A meticulous physical examination is key to detecting

 

rotation deformities or poor anatomical reduction. Postreduction splinting plays an

 

important role in the treatment of almost all fractures, and various finger splints are

 

widely available in most acute care settings. Open fractures or extensive soft tissue

 

injury should be treated with antibiotics and tetanus prophylaxis.

 

Nonsurgical Treatment

 

Treatment of finger fractures without any form of surgical fixation consists pri-

 

marily of splinting. The aim of splinting is to immobilize the fractured segment and

 

surrounding joints without interfering with the motion of the uninvolved joints.

 

Stack splints are used to immobilize distal phalanx fractures while allowing full ROM

 

of both the PIP and MP joints.

 

Extraarticular, nondisplaced, stable phalangeal fractures are treated with closed re-

 

duction, splinting and early rehabilitation. Closed reduction is performed using axial

 

traction with concomitant alignment of the bone. For most shaft fractures of the middle

 

or proximal phalanges, “buddy splinting” to the adjacent finger for 1-2 weeks is suffi-

 

cient. In addition, radial or ulnar gutter splints are useful as they leave the remaining

 

digits uninvolved. Postreduction films (2 views) should be obtained after closed reduc-

 

tion and following one week to ensure continued fracture reduction.

 

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Surgical Treatment

Surgical fixation and reduction are indicated for any of the following:

Open fractures

Unstable fractures

Dislocated or intraarticular fractures

Irreducible fractures

Proximal Phalanx Fractures

Different types of shaft fractures, such as transverse, oblique or spiral are in general considered to be unstable and require surgical fixation using two longitudinal K-wires. Intraarticular base fractures of the proximal phalanx are divided into three types: vertical shear fractures, compression fractures, and collateral ligament avulsions. The first two types may have impaction or rotated fragments. If this is the case, fixation is required. Stable and nondisplaced avulsion fractures are treated with splinting alone, using a buddy strap to the adjacent digit. If displacement is evident on radiographs, anatomic reduction is mandatory in order to prevent post-traumatic arthritis or persistent instability. Reduced vertical shear fractures should be stabilized using a percutaneous K-wire or an interfragmentary screw under image magnification. Fractures that have undergone bone-to-bone fixation can usually be mobilized the following day with a buddy strap to the adjacent finger. Ligament-to-bone repairs usually take longer to heal and should not be mobilized for 4 weeks. Compression fractures usually require open reduction with internal fixation (ORIF), especially if there is in an intraarticular

85step off of greater than 2 mm or visible angulation. Intrafragmentary screw fixation, mini-T, or minicondylar plates or used for internal fixation. The addition of a cancellous bone graft is useful to avoid over-compression and incongruity of the fragments.

Middle Phalanx Fractures

Middle phalanx fractures usually do better than proximal phalanx fractures since the DIP joint is functionally less important than the PIP or MP joints. Malunion, or post-traumatic arthrosis of the DIP joint is less severe than in the PIP or MP joints and can be treated without difficulty by arthrodesis. The vast majority of middle phalanx fractures are stable and well aligned. These can be treated with a buddy strap to the adjacent digit or gutter splints. Radiographs should be performed every 5 days for about 2 weeks to detect displacement. In the event of an unstable middle phalangeal shaft fracture, K-wire fixation is sufficient. Open fractures with extensive soft tissue damage may require ORIF.

If a shaft fracture is stable but malaligned after reduction, an extension block cast should be considered as it provides stabilization of the reduction and reduces the risk for PIP joint stiffness. The extension block should be maintained for 4 weeks followed by 2 weeks in a buddy strap. Rotational abnormalities should be ruled out by noting the alignment of the nail beds under active flexion of the fingers towards the palm and comparing them to the healthy hand. Malalignment of a fracture can occur in patients presenting weeks after the initial trauma, with a callus already present or soft tissue interposition. Often, these can no longer be reduced successfully in a closed fashion. Therefore, an ORIF is required, usually accompanied by an osteotomy along with tenolysis and capsulotomy.

Distal Phalanx Fractures

Distal phalangeal fractures are the most common finger fracture. Kaplan’s classification divides them into three types: Type A, or longitudinal; Type B, or comminuted;

Finger and Metacarpal Fratures

515

and Type C or transverse. Type B fractures typically result from a crush injury. This often leads to an exquisitely painful subungual hematoma. Puncture of the nail with a heated paper clip works well to release the trapped blood. Up to 2 weeks of splinting with a Stax splint is sufficient for most cases. Transverse or longitudinal shaft fractures (Type A and C) are mostly stable with minimal displacement. The finger should be splinted (usually 3 to 4 weeks) until evidence of bony union is seen on radiographs. Open and unstable shaft fractures require K-wire fixation after alignment. A detached nail should be replaced and if necessary, secured with suture fixation. The nail helps protect the underlying matrix, prevents scar formation between the nail matrix and eponychium, and guides the direction of growth of the new nail.

Fractures of the base of the distal phalanx are usually unstable and tend to angulate in a dorsal fashion due to the traction of the extensor tendons. Closed fractures are treated by splinting (e.g., using Alumafoam) of the finger in extension for at least 4 weeks. Open fractures of the base are considered to be unstable and tend to have a rotational deformity. Retrograde K-wire fixation is a straight forward method of stabilization.

Condylar Fractures

Meticulous anatomic reduction is essential in condylar fractures, as malalignment can lead to a flexion contracture. A closed reduction will generally not provide an adequate result, requiring ORIF for reconstruction. Unicondylar fractures are usually unstable and often demonstrate a rotation deformity due to traction of the collateral ligament. If closed reduction is attempted by axial traction, a reduction clamp should be used to maintain the repositioned condyle and a 0.028 inch K-wire percutaneously inserted

through the uninjured condyle. Intraoperative films in all three dimensions should be 85 obtained to insure precise alignment of the small but functionally important fragment.

Metacarpal Fractures

Introduction

A fracture of the metacarpal is often termed a “boxer’s fracture” alluding to the etiology, which is usually direct or indirect trauma. Metacarpal fractures account for nearly 40% of all hand fractures. About a quarter of these are located in the neck of the 5th metacarpal bone.

The four metacarpals are tubular bones, each with an intrinsic arch. The 2nd and 3rd carpometacarpal (CMC) joints have almost no range of motion, whereas the 4th and 5th CMC joints provide 15˚ and 25˚ of relative motion, respectively.

Injuries of the metacarpal bone are clinically divided into fractures of the metacarpal base, the metacarpal shaft or neck and the metacarpal head.

Imaging

For most fractures, three-view conventional radiographs are sufficient. Severely comminuted or intraarticular fractures may also require stress views. CT scanning can be helpful for evaluation of questionable intraarticular involvement, since this can be difficult to determine on plain radiographs. The purpose of a CT scan in this setting is to assess the alignment or displacement of the fracture, and to aid in planning the operative approach.

Clinical Findings

Signs and symptoms include pain, hematoma, swelling, reduced motion and crepitus. These findings are common in metacarpal shaft fractures, while fractures of the metacarpal base and neck may have a subtle presentation.

 

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Fractures of the shaft tend to be shortened and rotated. Rotation deformities must be

 

 

addressed; if untreated they pose a risk for a weakened grip and incomplete fist closure

 

 

due to overlapping fingers. Fractures of the metacarpal base may be impacted since they

 

 

are often caused by an axial load, making the diagnosis easy to miss. Fractures of the

 

 

metacarpal head often present with nothing more than localized swelling and pain. Crepi-

 

 

tus can be heard but should not be elicited since it has no additional diagnostic benefit.

 

 

Treatment

 

 

 

Most metacarpal fractures are adequately treated with closed reduction followed

 

 

by immobilization in a forearm-based splint. two-view radiographs should be taken in

 

 

the follow-up period in order to confirm that the reduction has been maintained.

 

 

Early mobilization of wrist and fingers is absolutely critical to achieve optimal long-term

 

 

functional results. Apex and dorsal angulation are often seen in shaft fractures. There

 

 

is, however, a certain degree of angulation that is acceptable (up to 10˚ for the second

 

 

and third, up to 20˚ for the fourth, and up to 30˚ for the fifth metacarpal).

 

 

The following is a useful list of the indications for surgical treatment of a meta-

 

 

carpal fracture:

 

 

 

1. Open fractures

 

 

 

2. Unstable and extensively displaced fractures requiring stabilization

 

 

3. Nonreducible fractures

 

 

 

4. Multiple fractures

 

 

 

5. Old fractures that need to be reduced (>1 week post-trauma)

 

 

6. Failure of nonsurgical treatment

 

85

 

 

7. Fractures with extensive soft tissue injury

 

 

 

 

Metacarpal Base Fractures

Most base fractures are impacted and are treated by splinting alone and early mobilization. Base fractures of the 5th metacarpal often require surgical treatment if displaced greater than 2 mm. Fractures involving the CMC joint are unstable and have a strong tendency to destabilize after a successful reduction due to traction forces of the surrounding ligaments. Therefore, CMC fractures often need open reduction with pin fixation. Postoperative splinting should be maintained for 2-3 weeks followed by mobilization. The pins should be removed after 6-8 weeks.

Metacarpal Shaft Fractures

In general, metacarpal shaft fractures are treated with reduction and splinting alone. The MP and PIP joint of the affected metacarpal bone are flexed to a 90˚ angle. Reduction is achieved with upward directed traction on the middle phalanx and downward resistance at the level of the proximal metacarpal shaft. Either splinting (extending to the distal palmar crease) or percutaneous pins are employed to maintain the reduction result.

First Metacarpal Fractures

Introduction

Thumb fractures are usually the result of occupational or sporting-related trauma. The thumb consists of a proximal and distal phalanx and a metacarpal bone. The first CMC joint is the articulation of the trapezium and the metacarpal base and provides a wide range of motion. It is stabilized by a number of ligaments, most importantly the anterior and posterior oblique ligaments, the intermetacarpal ligaments, and the dorsal radial ligament. The ROM of the MP joint of the thumb is extremely variable. It has relatively little intrinsic stability, and the stabilizing ligaments are prone to in-

Finger and Metacarpal Fratures

517

jury. The ulnar and radial collateral ligaments stabilize the MP joint on its sides, and the volar plate stabilizes it on the palmar surface.

Fracture Patterns

Metacarpal fractures of the thumb are divided into base, shaft and head fractures. Typical signs of fracture are swelling, decreased range of motion, pain (most notably in base fractures) and occasional crepitus. Shaft fractures are rare due to the stable cortical bone structure and tend to result in apex-dorsal or apex-radial angulation. The most common fractures are of the metacarpal base, and these can be intraarticular (e.g., Bennet fracture) or extraarticular. Base fractures are usually due to a gently flexed metacarpal during an axial force. Adequate imaging of the CMC joint is essential in diagnosing a metacarpal base fracture: a true lateral view of the joint with the palm flat on the plate and wrist and hand pronated 15-35˚ is obtained. The imaging beam is aimed at the CMC joint from a 15˚ oblique angle from distal to proximal.

Extra-Articular

Extraarticular fractures are usually oblique or transverse. Closed reduction with a thumb spica cast for 4 weeks excluding the distal phalanx are usually sufficient. Reduction is obtained with axial traction, palmar directed pressure at the level of the fracture, and pronation of the distal fragment. Percutaneous pin fixation is indicated in cases of an unstable fracture postreduction (more likely in oblique fractures), open fractures, and fractures with angulation greater than 30˚ with shortening.

External or internal fixation is required for extensively comminuted fracture types. 85

Intra-Articular

The Bennett fracture is an intraarticular fracture of the metacarpal base with a volar-ulnar fragment of variable size. The fragment is often kept in anatomic position through the anterior oblique ligament. Restoring full anatomic congruity is mandatory to avoid post-traumatic arthritis. If articular incongruity is less than 2 mm postreduction, the fracture can be stabilized with percutaneous pin fixation. Open or unstable fractures, irreducible fractures, or those with an incongruity greater than 2 mm postreduction are indications for ORIF.

Pearls and Pitfalls

Fractures of the metacarpals and phalanges may be associated with ligamentous injuries (collateral ligaments or carpometacarpal ligaments), and therefore a careful examination for dislocations and joint instability must also be performed. Stiffness is a prime complication of a phalangeal injury. Early mobilization is key to optimal outcome (within 3-4 weeks of injury if possible). Differences in bony healing in the adult and pediatric population should also be recognized. Since children heal faster, they can be splinted for shorter duration compared to adults (with minimally displaced metacarpal fractures for example).

Suggested Reading

1.Blair WF, ed. Techniques in Hand Surgery. Baltimore, Maryland, USA: Williams and Wilkins, 179-284.

2.Stern PJ. Fractures of the metacarpals and phalanges. In: Green DP, Hotchkiss R, Pederson WC, eds. Green’s Operative Hand Surgery. 4th ed. New York: Churchill Livingstone, 1999.

3.Weiss AP, Hastings H. Distal unicondylar fractures of the proximal phalanx. J Hand Surg 1993; 18A:594.

4.Gonzales MH, Hall RF. Intramedullary fixation of metacarpal and proximal phalangeal fractures of the hand. Clin Orthop 1996; 327:47.

Chapter 86

Brachial Plexus Injuries

Mark Sisco and John Y.S. Kim

Introduction

Injuries to the brachial plexus can result in significant long-term functional impairment. Recent advances in microneurosurgery have allowed for the reconstruction of many of these injuries. This chapter will focus on specific goals and general approaches toward adult brachial plexus reconstruction.

Anatomy

The brachial plexus is formed from the branches of the C5 to T1 spinal nerves (Fig. 86.1). The spinal nerves are comprised of dorsal and ventral roots. The ventral roots carry motor fibers. The dorsal roots are made up of sensory fibers that originate in the dorsal root ganglion in or just distal to the intervertebral foramen. Each root has a root sleeve comprised of dura and arachnoid that forms the nerve sheath at the level of the intervertebral foramen.

Typically, the upper trunk derives from the C5-C6 nerve roots, the middle trunk from the C7 nerve root, and the lower trunk from C7 and T1 nerve roots. Each trunk divides into an anterior and posterior division. The anterior divisions of the upper and middle trunks comprise the lateral cord while the anterior division of the lower trunk forms the medial cord. The posterior divisions of all three roots form the posterior cord. These cords will then differentiate into terminal branches.

Epidemiology

Adult brachial plexus injuries usually result from significant trauma to the upper limb. High-speed motor-vehicle collisions, especially those involving motorcycles, are the most common mechanism of injury. Other forms of blunt trauma, including industrial accidents, falls, and pedestrian-motor-vehicle accidents, are typical causes. Penetrating trauma is a less common cause of injury and may be classified into sharp injuries and blast-effect injuries. Young men constitute the largest demographic group affected.

Mechanisms of Injury

There are five main mechanisms that lead to brachial plexus injury: (1) crush injury, caused by blunt extrinsic force; (2) traction injury, usually caused by stretching of the nerves; (3) compression injury from intrinsic force (pressure from adjacent tissues or hematoma); (4) sharp penetrating injury; and (5) blast injury (typically from gunshot wounds).

Initial Assessment

Since the mechanism of brachial plexus injury often involves significant blunt or penetrating trauma, other injuries are often present. Initial management must

Practical Plastic Surgery, edited by Zol B. Kryger and Mark Sisco. ©2007 Landes Bioscience.

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Figure 86.1. The brachial plexus.

86

address life-threatening injuries. As such, the diagnosis of brachial plexus injuries is often delayed until overall stabilization has occurred and mental status has improved.

History and Physical

The mechanism of injury must be accurately determined since prognosis is intimately related to etiology. Manual muscle testing should include observation of muscle atrophy and tone. The British Medical Research Council grading system for muscle function is often helpful for standardized documentation of strength. Passive range of motion should be assessed to check for contractures, followed by active range of motion testing. Median, radial, and ulnar nerve function can be assessed by testing finger and wrist motion. The musculocutaneous nerve is responsible for elbow flexion. Abduction of the shoulder is dependent on the axillary nerve. The posterior cord can be assessed by checking finger and wrist extension, elbow extension, and shoulder abduction. The medial and lateral cords can be assessed via the medial and lateral pectoral nerves, which innervate the sternal and clavicular heads of the pectoralis major, respectively. The sensory examination must be thorough, and should include testing for Tinel’s sign at the supraclavicular fossa and the infraclavicular coracoid process.

Radiographic Evaluation

Plain film radiographic evaluation of the cervical spine and ipsilateral shoulder and scapula can delineate contributing bony injury or consequent bony deformity. Penetrating trauma in the setting of abnormal pulses suggests a hematoma-related

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plexopathy; an arteriogram may be helpful in this case. On CT myelogram, nerve root avulsion is suggested by demonstration of a meningocele, which results from tearing of the nerve root sleeve.

Injury Patterns

Determining the level of injury is critical to planning operative exposure, determining treatment modality, and assessing prognosis.

Preganglionic Injuries

Root avulsion refers to injury of the nerve root within the intervertebral foramen proximal to the sensory root ganglion. The spinal nerves are attached to the transverse processes in the lower cervical spine. As progressive traction is placed on the nerve root, these attachments are the first to break. The nerve root sleeve is then pulled through the intervertebral foramen and may tear, causing a meningocele. Finally, the nerve root itself avulses. Horner syndrome consists of ipsilateral meiosis, upper eyelid ptosis, and facial anhydrosis; it implies disruption to of the sympathetic fibers traveling through C8-T1 and suggests root avulsion at these levels. Winging of the scapula or weakness of the rhomboid suggests C5 or C6 root injury, respectively, since the long thoracic and dorsal scapular nerves arise from the nerve roots. Since no proximal stump is available for reconstruction, root avulsions may require neurotization.

Postganglionic Injuries

Postganglionic injuries, or ruptures, may have regenerative capacity and can of-

86ten be grafted. Supraclavicular injuries involve the spinal nerves, trunks, or divisions. They manifest most commonly as Erb’s palsy (C5-C6 injury) or total plexus injury. Erb’s palsy includes loss of deltoid, supraspinatus, infraspinatus, biceps, and brachialis function and presents with internal rotation, shoulder adduction, and elbow extension. Total plexus injury presents with a flail arm. Infraclavicular injuries involve the cords and their distal branches. They often present with focal weaknesses in shoulder abduction and flexion with or without deficits in elbow, wrist, or hand strength.

Preganglionic lesions can be differentiated from postganglionic lesions by weakness of the rhomboids, serratus, and supraspinatus in combination with an electrophysiologic profile of normal sensory conduction velocities with absent somatosensory evoked potentials. It may also be possible to see meningoceles on CT myelogram or MRI. However, preand postganglionic injuries may coexist and accurate diagnosis may not be possible until surgical exploration.

Goals of Reconstruction

Terzis and Papakonstantinou suggest the following priorities in reconstructing target muscles:

1.Return function to the supraspinatus and deltoid muscles to stabilize the shoulder and prevent severe neck pain.

2.Restore the biceps via the musculocutaneous nerve, allowing the hand to be brought to the face. Neurotization of the ipsilateral latissimus will also assist biceps function.

3.Attempt to reestablish triceps function.

4.Reconstruct the median nerve.

Brachial Plexus Injuries

521

Surgical Strategies

Low velocity penetrating injuries should be explored immediately and repaired primarily or with nerve graft. Injuries related to gunshot wounds typically result from blast effect rather than by direct nerve injury; as such, spontaneous recovery may be possible. Surgery for closed injuries may be performed 3-6 months after injury, after any spontaneous recovery has been assessed and before irreversible muscle atrophy has occurred.

Neurolysis

The removal of scar tissue surrounding intact nerve fibers, known as neurolysis, may help nerve regeneration.

Nerve Repair and Grafting

When nerve continuity is interrupted distal to the sensory root ganglion, nerve repair or grafting may be beneficial. Primary repair may be attempted for simple nerve lacerations. Nerve grafts, often using the sural, antebrachial cutaneous, or radial sensory nerve, are used to reconstruct traction and crush injuries.

Neurotization

Preganglionic lesions may be treated with neurotization, in which functional nerves are harvested and transferred to distal nerve root stumps. Intraplexus donors, because of their large number of axons, provide the best motor donors for neurotization. Extraplexus motor donors, such as intercostal, phrenic, accessory, and medial pectoral nerves, may be used for multiple-root avulsions. The contralateral C7 nerve root,

combined with a nerve graft, may be also be used. Recently success has been reported 86 using branches of the ulnar nerve to neurotize the musculocutaneous nerve.

Nerve Root Repair and Reimplantation

There have been several recent reports of nerve root repair in which a nerve graft is inserted into the spinal cord and attached to the avulsed distal root. Some have also attempted to reimplant the avulsed root directly into the spinal cord. It is too early to know whether these methods will prove to be successful.

Secondary Procedures

Muscle and tendon transfers may be necessary to augment function. Concomitant release of muscle contractures that develop may be helpful in restoring balanced external rotation and abduction of the upper extremity. In refractory cases, functional free muscle transfer with the gracilis may be necessary to restore distal function.

Postoperative Care

After 2-4 weeks of splinting to prevent traction on the repair, an aggressive regimen of physical therapy, occupational therapy, and electric stimulation should be initiated. Induction exercises, which stimulate the donor nerves used during neurotization should be begun when recipient motor function begins to return. Patient expectations and compliance are paramount in achieving a successful long-term outcome. Secondary reconstruction with muscle transfers and contracture releases may be necessary. Patients in whom primary nerve reconstruction is performed should be counseled on the staged nature of reconstruction.

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