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HALO VISION
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341
membrane. Fever and cervical lymphadenopathy are also common.
◆ Renal failure (chronic). Renal failure pro-
duces a urinous or ammonia breath odor.
Among its widespread effects are anemia, emotional lability, lethargy, irritability, decreased
mental acuity, coarse muscular twitching, peripheral neuropathies, muscle wasting, anorexia, signs of GI bleeding, ecchymosis, yellowbrown or bronze skin, pruritus, anuria, and
increased blood pressure.
◆ Sinusitis. Acute sinusitis causes a purulent
nasal discharge that leads to halitosis. Besides a
characteristic postnasal drip, the patient may
exhibit nasal congestion, sore throat, cough,
malaise, headache, facial pain and tenderness,
and fever.
Chronic sinusitis causes a continuous mucopurulent discharge that leads to a musty
breath odor, postnasal drip, nasal congestion,
and a chronic nonproductive cough.
◆ Zenker’s diverticulum. This esophageal dis-
order causes halitosis and a bad taste in the
mouth associated with regurgitation. The patient may also report a chronic cough that’s
most pronounced at night, hoarseness,
odynophagia, neck pain, and “gurgling” sounds
in the throat when he swallows liquids.
O
THER CAUSES
◆ Drugs. Drugs that can cause halitosis include
triamterene, inhaled anesthetics, and any drugs
known to cause metabolic acidosis such as nitroprusside.
HERB ALERT Some herbal medicines, such
as garlic, may cause halitosis.
S
PECIAL CONSIDERATIONS
If examination of the mouth and sinuses doesn’t
reveal the cause of halitosis, prepare the patient
for upper GI and chest X-rays or endoscopy.
P
EDIATRIC POINTERS
In children, halitosis commonly results from
physiologic causes, such as continual mouth
breathing and thumb or blanket sucking.
Phenylketonuria—a metabolic disorder that affects infants—may produce a musty or mousy
breath odor.
G
ERIATRIC POINTERS
Extensive dental caries, mouth dryness, and
poor oral hygiene can cause halitosis in elderly
patients.
P
ATIENT COUNSELING
To help control halitosis, encourage good oral
hygiene. If halitosis is drug induced, reassure
the patient that it will disappear as soon as his
body completely eliminates the drug.
Halo vision
Halo vision refers to seeing rainbowlike colored
rings around lights or bright objects. The rainbowlike effect can be explained by this physical
principle: As light passes through water (in the
eye, through tears or the cells of various anteretinal media), it breaks up into spectral colors.
Halo vision usually develops suddenly; its duration depends on the causative disorder. This
symptom may occur in disorders associated with
excessive tearing and corneal epithelial edema.
Among these causes, the most common and significant is acute angle-closure glaucoma, which
can lead to blindness. In this ophthalmic emergency, increased intraocular pressure (IOP) forces
fluid into corneal tissues anterior to Bowman’s
membrane, causing edema. Halo vision is also
an early symptom of cataracts, resulting from
dispersion of light by abnormal lens opacity.
Nonpathologic causes of excessive tearing
associated with halo vision include poorly fitted
or overworn contact lenses, emotional extremes, and exposure to intense light, as in
snow blindness.
H
ISTORY AND PHYSICAL
EXAMINATION
First, ask the patient how long he has been seeing halos around lights and when he usually
sees them. Patients with glaucoma usually see
halos in the morning, when IOP is most elevated. Ask the patient if light bothers his eyes.
Does he have eye pain? If so, have him describe
it. Remember that halos associated with excruciating eye pain or a severe headache may point
to acute angle-closure glaucoma, an ocular
emergency. Note a history of glaucoma or
cataracts.
Next, examine the patient’s eyes, noting conjunctival injection, excessive tearing, and lens
changes. Examine pupil size, shape, and response to light. Then test visual acuity by performing an ophthalmoscopic examination.
M
EDICAL CAUSES
◆ Cataract. Halo vision may be an early symp-
tom of painless, progressive cataract formation.

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The glare of headlights may blind the patient,
making nighttime driving impossible. Other features include blurred vision, impaired visual
acuity, and lens opacity, all of which develop
gradually.
◆ Corneal endothelial dystrophy. Typically,
halo vision is a late symptom of this disorder,
which may also cause impaired visual acuity.
◆ Glaucoma. Halo vision characterizes all
types of glaucoma. Acute angle-closure glauco-
ma—an ophthalmic emergency—also causes
blurred vision, followed by a severe headache
or excruciating pain in and around the affected
eye. Examination reveals a moderately dilated
fixed pupil that doesn’t respond to light, conjunctival injection, a cloudy cornea, impaired visual acuity and, possibly, nausea and vomiting.
Chronic angle-closure glaucoma usually pro-
duces no symptoms until pain and blindness occur in advanced disease. Sometimes, halos and
blurred vision develop slowly.
In chronic open-angle glaucoma, halo vision is
a late symptom that’s accompanied by mild eye
ache, peripheral vision loss, and impaired visual
acuity.
S
PECIAL CONSIDERATIONS
To help minimize halo vision, remind the patient not to look directly at bright lights.
P
EDIATRIC POINTERS
Halo vision in a child usually results from congenital cataracts or glaucoma. In a young child,
limited verbal ability may make halo vision difficult to assess.
G
ERIATRIC POINTERS
Primary glaucoma, the most common cause of
halo vision, is more common in older patients.
Headache
The most common neurologic symptom,
headaches may be localized or generalized,
producing mild to severe pain. About 90% of all
headaches are benign and can be described as
vascular, muscle-contraction, or a combination
of both. (See Comparing benign headaches.) Occasionally, though, headaches indicate a severe
neurologic disorder associated with intracranial
inflammation, increased intracranial pressure
(ICP), or meningeal irritation. They may also result from an ocular or sinus disorder, tests,
drugs, or other treatments.
Other causes of headache include fever, eyestrain, dehydration, and systemic febrile illnesses. Headaches may occur in certain metabolic
disturbances—such as hypoxemia, hypercapnia,
hyperglycemia, and hypoglycemia—but they
aren’t a diagnostic or prominent symptom in
these disorders. Some individuals get
headaches after seizures or from coughing,
sneezing, heavy lifting, or stooping.
H
ISTORY AND PHYSICAL
EXAMINATION
If the patient reports a headache, ask him to describe its characteristics and location. How often does he get a headache? How long does a
typical headache last? Try to identify precipitating factors, such as eating certain foods or exposure to bright lights. Ask what helps to relieve
the headache. Is the patient under stress? Has
he had trouble sleeping?
Take a drug and alcohol history, and ask
about head trauma within the last 4 weeks. Has
the patient recently experienced nausea, vomiting, photophobia, or visual changes? Does he
feel drowsy, confused, or dizzy? Has he recently
developed seizures, or does he have a history of
seizures?
Begin the physical examination by evaluating the patient’s level of consciousness (LOC).
Then check his vital signs. Be alert for signs of
increased ICP—widened pulse pressure, bradycardia, altered respiratory pattern, and increased blood pressure. Check pupil size and
response to light, and note any neck stiffness.
(See Differential diagnosis: Headache, pages 344
and 345.)
M
EDICAL CAUSES
◆ Anthrax, cutaneous. Along with a macular
or papular lesion that develops into a vesicle
and finally a painless ulcer, this disorder may
produce a headache, lymphadenopathy, fever,
and malaise.
◆ Brain abscess. In this disorder, the
headache is localized to the abscess site; it usually intensifies over a few days and is aggravated by straining. Accompanying the headache
may be nausea, vomiting, and focal or generalized seizures. The patient’s LOC varies from
drowsiness to deep stupor. Depending on the
abscess site, associated signs and symptoms
may include aphasia, impaired visual acuity,
hemiparesis, ataxia, tremors, and personality
changes. Signs of infection, such as fever and
pallor, usually develop late; however, if the

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Comparing benign headaches
Of the many patients who report headaches, only about 10% have an underlying medical disorder. The other 90% suffer from benign headaches, which may be classified as muscle-contraction (tension), vascular (migraine and cluster), or a combination of both.
As you review the chart below, you’ll see that the two major types— muscle-contraction and
vascular headaches — are quite different. In a combined headache, features of both appear; this
type of headache may affect the patient with a severe muscle-contraction headache or a latestage migraine. Treatment of a combined headache includes analgesics and sedatives.
Characteristics
Incidence
Precipitating
factors
Intensity and
duration
Associated signs
and symptoms
Muscle-contraction
headaches
◆ Most common type, ac-
counting for 80% of all
headaches
◆ Stress, anxiety, tension,
improper posture, and body
alignment
◆ Prolonged muscle con-
traction without structural
damage
◆ Eye, ear, and paranasal si-
nus disorders that produce
reflex muscle contractions
◆ Produce an aching tight-
ness or a band of pain
around the head, especially
in the neck and in occipital
and temporal areas
◆ Occur frequently and usu-
ally last for several hours
◆ Tense neck and facial
muscles
Vascular headaches
◆ More common in women and those
with a family history of migraines
◆ Onset after puberty
◆ Hormone fluctuations
◆ Alcohol
◆ Emotional upset
◆ Too little or too much sleep
◆ Foods, such as chocolate, cheese,
monosodium glutamate, and cured
meats; caffeine withdrawal
◆ Weather changes, such as shifts in
barometric pressure
◆ May begin with an awareness of an
impending migraine or a 5- to 15minute prodrome of neurologic
deficits, such as visual disturbances,
dizziness, unsteady gait, or tingling
of the face, lips, or hands
◆ Produce severe, constant, throb-
bing pain that is typically unilateral
and may be incapacitating
◆ Last for 4 to 6 hours
◆ Anorexia, nausea, and vomiting
◆ Occasionally, photophobia, sensi-
tivity to loud noises, weakness, and
fatigue
◆ Depending on the type (cluster
headache or classic, common, or
hemiplegic migraine), possibly chills,
depression, eye pain, ptosis, tearing,
rhinorrhea, diaphoresis, and facial
flushing
Alleviating
factors
◆ Mild analgesics, muscle
relaxants, or other drugs
during an attack
◆ Measures to reduce stress,
such as biofeedback, relaxation techniques, and counseling; posture correction to
prevent attacks
◆ Methysergide and propranolol to
prevent vascular headache
◆ Ergot alkaloids or serotonin-
receptor drugs at the first sign of a
migraine
◆ Rest in a quiet, darkened room
◆ Elimination of irritating foods
from diet

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Differential diagnosis: Headache
History of present illness
Focused physical examination: Neurologic and musculoskeletal systems;
head, eyes, ears, nose, and throat; neck; mental health; lymph nodes
Sinusitis
Signs and
symptoms
◆ Dull periorbital
headache
◆ Unilateral or bi-
lateral frontal or
maxillary sinus
pain that’s increased by palpation or bending
over
◆ Fever
◆ Malaise
◆ Nasal turbinate
edema
◆ Sore throat
◆ Nasal discharge
Diagnosis: Physical examination,
transillumination,
sinus X-ray
Treatment: Medication (decongestants, analgesics,
antibiotics)
Follow-up: None
unless signs and
symptoms
worsen or recur
Brain
abscess
Signs and
symptoms
◆ Localized head-
ache that
increases over a
few days
◆ Possible nausea
and vomiting
◆ Focal or gen-
eralized seizures
◆ Drowsiness
Subdural
hematoma
Signs and
symptoms
◆ Decreased level
of consciousness
(LOC)
◆ Acute drowsi-
ness, confusion,
or agitation
◆ Pounding head-
ache
◆ Giddiness
◆ Personality
changes
◆ Dizziness
◆ Confusion
Encephalitis
Signs and
symptoms
◆ Severe,
generalized
headache
◆ Deteriorating
LOC within 48
hours of initial
headache
◆ Fever
◆ Nuchal rigidity
◆ Irritability
◆ Seizures
◆ Nausea and
vomiting
◆ Photophobia
Diagnosis: Possible history of head trauma, lumbar puncture, imaging studies (computed
tomography scan, magnetic resonance imaging, arteriography)
Treatment: Medication (antibiotics if indicated; analgesics, anticonvulsants, osmotic diuretics);
surgery if appropriate; chemotherapy or radiation therapy if malignancy is present
Follow-up: Referral to neurologist or neurosurgeon
abscess remains encapsulated, these signs may
not appear.
◆ Brain tumor. Initially, a tumor causes a lo-
calized headache near the tumor site; as the tumor grows, the headache eventually becomes
generalized. The pain is usually intermittent,
deep seated, and dull and is most intense in the
morning. It’s aggravated by coughing, stooping,
Valsalva’s maneuver, and changes in head
position, and it’s relieved by sitting and rest.

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Epidural
hemorrhage
Signs and
symptoms
◆ Progressively
severe headache
◆ Unilateral
seizures
◆ Decreased LOC
◆ Hemiparesis or
hemiplegia
◆ High-grade
fever
Cerebral
aneurysm
(ruptured)
Signs and
symptoms
◆ Sudden severe,
possibly unilateral
headache
◆ Possible nausea
and vomiting
◆ Change in LOC
◆ Vision changes
Intracranial
hemorrhage
Signs and
symptoms
◆ Severe gen-
eralized headache
◆ Rapid, steady
decrease in LOC
◆ Hemiparesis or
hemiplegia
◆ Aphasia
◆ Dizziness
◆ Nausea and
vomiting
◆ Irregular res-
pirations
◆ Positive Babin-
ski’s reflex
Brain tumor
Signs and
symptoms
◆ Localized or
generalized headache
◆ Intermittent
deep pain that’s
more intense in
the morning and
increases with
Valsalva’s
maneuver
◆ Personality
changes
◆ Changes in LOC
Associated signs and symptoms include personality changes, altered LOC, motor and sensory
dysfunction, and eventually signs of increased
ICP, such as vomiting, increased systolic blood
pressure, and widened pulse pressure.
◆ Cerebral aneurysm (ruptured). Cerebral
aneurysm is a life-threatening disorder that’s
characterized by a sudden excruciating
headache, which may be unilateral and usually
peaks within minutes of the rupture. The patient

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may lose consciousness immediately or display
a variably altered LOC. Depending on the severity and location of the bleeding, he may also exhibit nausea and vomiting; signs and symptoms
of meningeal irritation, such as nuchal rigidity
and blurred vision; hemiparesis; and other features.
◆ Ebola virus. A sudden headache commonly
occurs on the 5th day of this deadly illness. Additionally, the patient has a history of malaise,
myalgia, high fever, diarrhea, abdominal pain,
dehydration, and lethargy. A maculopapular
rash develops between the 5th and 7th days of
the illness. Other possible findings include pleuritic chest pain; a dry, hacking cough; pronounced pharyngitis; hematemesis; melena;
and bleeding from the nose, gums, and vagina.
Death usually occurs in the 2nd week of the illness, preceded by massive blood loss and
shock.
◆ Encephalitis. A severe, generalized
headache is characteristic with this disorder.
Within 48 hours, the patient’s LOC typically deteriorates—perhaps from lethargy to coma. Associated signs and symptoms include fever,
nuchal rigidity, irritability, seizures, nausea and
vomiting, photophobia, cranial nerve palsies
such as ptosis, and focal neurologic deficits,
such as hemiparesis and hemiplegia.
◆ Epidural hemorrhage (acute). Head trauma
and a sudden, brief loss of consciousness usually precede this hemorrhage, which causes a
progressively severe headache that’s accompanied by nausea and vomiting, bladder distention, confusion, and then a rapid decrease in
LOC. Other signs and symptoms include unilateral seizures, hemiparesis, hemiplegia, high
fever, decreased pulse rate and bounding pulse,
widened pulse pressure, increased blood pressure, a positive Babinski’s reflex, and decerebrate posture.
If the patient slips into a coma, his respirations deepen and become stertorous, then shallow and irregular, and eventually cease. Pupil
dilation may occur on the same side as the hemorrhage.
◆ Glaucoma, acute angle-closure. This type
of glaucoma is an ophthalmic emergency that
may cause an excruciating headache as well as
acute eye pain, blurred vision, halo vision, nausea, and vomiting. Assessment reveals conjunctival injection, a cloudy cornea, and a moderately dilated, fixed pupil.
◆ Hantavirus pulmonary syndrome. Noncar-
diogenic pulmonary edema distinguishes this
viral disease, which was first reported in the
United States in 1993. Common reasons for
seeking treatment include flulike signs and
symptoms—headache, myalgia, fever, nausea,
vomiting, and a cough—followed by respiratory
distress. Fever, hypoxia, and (in some patients)
serious hypotension typify the hospital course.
Other signs and symptoms include a rising respiratory rate (28 breaths/minute or more) and
an increased heart rate (120 beats/minute or
more).
◆ Hypertension. This disorder may cause a
slightly throbbing occipital headache on awakening that decreases in severity during the day.
However, if the patient’s diastolic blood pressure exceeds 120 mm Hg, the headache remains
constant. Associated signs and symptoms include an atrial gallop, restlessness, confusion,
nausea and vomiting, blurred vision, seizures,
and altered LOC.
◆ Influenza. A severe generalized or frontal
headache usually begins suddenly with the flu.
Accompanying signs and symptoms may last for
3 to 5 days and include stabbing retro-orbital
pain, weakness, diffuse myalgia, fever, chills,
coughing, rhinorrhea and, occasionally, hoarseness.
◆ Influenza type A H1N1 virus (swine flu).
Influenza type A H1N1, or swine flu, is a respiratory disease of pigs caused by type A influenza
virus. Swine flu viruses cause high levels of illness and low death rates in pigs. Swine flu
viruses normally don’t infect humans; however,
sporadic human infections with swine flu have
occurred. Most commonly, these cases occur in
persons with direct exposure to pigs. The virus
has changed slightly and is known as H1N1 flu.
Recent outbreaks of H1N1 flu have shown that
the virus can be transmitted from person to person, causing transmission across the globe. The
H1N1 flu is similar to influenza, and causes illness and in some cases death. The symptoms of
swine flu include headache, nonproductive
cough, fatigue, myalgia, chills, fever, and vomiting. The use of antiviral drugs is recommended
to treat H1N1 flu.
◆ Intracerebral hemorrhage. In some pa-
tients, this hemorrhage produces a severe generalized headache. Other signs and symptoms
vary with the size and location of the hemorrhage. A large hemorrhage may produce a
rapid, steady decrease in LOC, perhaps resulting
in a coma. Other common findings include
hemiplegia, hemiparesis, abnormal pupil size
and response, aphasia, dizziness, nausea,

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vomiting, seizures, decreased sensation, irregular respirations, positive Babinski’s reflex,
decorticate or decerebrate posture, and increased blood pressure.
◆ Listeriosis. If this infection spreads to the
nervous system, it may cause meningitis, whose
signs and symptoms include headache, nuchal
rigidity, fever, and change in LOC. Earlier signs
and symptoms of listeriosis include fever, myalgia, abdominal pain, nausea, vomiting, and diarrhea.
GENDER CUE Listeriosis during pregnancy
may lead to premature delivery, infection of
the neonate, or stillbirth.
◆ Meningitis. This disorder is marked by the
sudden onset of a severe, constant, generalized
headache that worsens with movement. Fever
and chills are other early signs. As meningitis
progresses, it also causes nuchal rigidity, positive Kernig’s and Brudzinski’s signs, hyperreflexia, altered LOC, seizures, ocular palsies, facial
weakness, hearing loss, vomiting and, possibly,
opisthotonos and papilledema.
◆ Plague. The pneumonic form of this lethal
bacterial infection causes a sudden onset of
headache, chills, fever, and myalgia. Pulmonary
findings include a productive cough, chest pain,
tachypnea, dyspnea, hemoptysis, respiratory
distress, and cardiopulmonary insufficiency.
◆ Postconcussion syndrome. A generalized
or localized headache may develop 1 to 30 days
after head trauma and last for 2 to 3 weeks.
This characteristic symptom may be described
as an aching, pounding, pressing, stabbing, or
throbbing pain. The patient’s neurologic examination is normal, but he may experience giddiness or dizziness, blurred vision, fatigue, insomnia, inability to concentrate, and noise and
alcohol intolerance.
◆ Q fever. Signs and symptoms of this disease
include severe headaches, fever, chills, malaise,
chest pain, nausea, vomiting, and diarrhea. The
fever may last for up to 2 weeks, and in severe
cases, the patient may develop hepatitis or
pneumonia.
◆ Severe acute respiratory syndrome
(SARS). SARS is an acute infectious disease
caused by a coronavirus. Although most cases
have been reported in Asia (China, Vietnam,
Singapore, Thailand), cases have cropped up in
Europe and North America. After an incubation
period of 2 to 7 days, the illness generally begins with a fever (usually greater than 100.4° F
[38° C]). Other symptoms include headache,
malaise, a nonproductive cough, and dyspnea.
SARS may produce only mild symptoms, or it
may progress to pneumonia and, in some cases,
even respiratory failure and death.
◆ Sinusitis (acute). This disorder is usually
marked by a dull periorbital headache that’s
usually aggravated by bending over or touching
the face and is relieved by sinus drainage.
Fever, sinus tenderness, nasal turbinate edema,
sore throat, malaise, cough, and nasal discharge
may accompany the headache.
◆ Smallpox (variola major). Initial signs and
symptoms of this virus include a severe
headache, backache, abdominal pain, high
fever, malaise, prostration, and a maculopapular rash on the mucosa of the mouth, pharynx,
face, and forearms and then on the trunk and
legs. The rash becomes vesicular, then pustular.
After 8 or 9 days, the pustules form a crust,
which later separates from the skin, leaving a
pitted scar. Death may result from encephalitis,
extensive bleeding, or secondary infection.
◆ Subarachnoid hemorrhage. This hemor-
rhage commonly produces a sudden, violent
headache along with nuchal rigidity, nausea and
vomiting, seizures, dizziness, ipsilateral pupil
dilation, and altered LOC that may rapidly
progress to coma. The patient also exhibits positive Kernig’s and Brudzinski’s signs, photophobia, blurred vision and, possibly, a fever. Focal
signs and symptoms (such as hemiparesis,
hemiplegia, sensory or vision disturbances, and
aphasia) and signs of elevated ICP (such as
bradycardia and increased blood pressure) may
also occur.
◆ Subdural hematoma. Typically associated
with head trauma, both acute and chronic subdural hematomas may cause headache and decreased LOC. An acute subdural hematoma also
produces drowsiness, confusion, and agitation
that may progress to coma. Later findings include signs of increased ICP and focal neurologic deficits such as hemiparesis.
A chronic subdural hematoma produces a
dull, pounding headache that fluctuates in
severity and is located over the hematoma.
Weeks or months after the initial head trauma,
the patient may experience giddiness, personality changes, confusion, seizures, and progressively worsening LOC. Late signs may include
unilateral pupil dilation, sluggish pupil reaction
to light, and ptosis.
◆ Temporal arteritis. A throbbing unilateral
headache in the temporal or frontotemporal region may be accompanied by vision loss, hearing loss, confusion, and fever. The temporal

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arteries are tender, swollen, nodular, and sometimes erythematous.
◆ Tularemia. Signs and symptoms following
inhalation of the bacterium Francisella tularensis
include abrupt onset of headache, fever, chills,
generalized myalgia, a nonproductive cough,
dyspnea, pleuritic chest pain, and empyema.
◆ Typhus. In typhus, initial symptoms of
headache, myalgia, arthralgia, and malaise are
followed by an abrupt onset of chills, fever, nausea, and vomiting. A maculopapular rash may
also occur.
◆ West Nile encephalitis. This brain infection
is caused by West Nile virus, a mosquito-borne
flavivirus commonly found in Africa, West Asia,
the Middle East and, rarely, in North America.
Most patients have mild signs and symptoms,
including fever, headache, body aches, rash,
and swollen lymph glands. More severe infection is marked by high fever, headache, neck
stiffness, stupor, disorientation, coma, tremors,
and paralysis.
O
THER CAUSES
◆ Diagnostic tests. A lumbar puncture or
myelogram may produce a throbbing frontal
headache that worsens on standing.
◆ Drugs. A wide variety of drugs can cause
headaches. For example, indomethacin produces headaches—usually in the morning—in
many patients. Vasodilators and drugs with a
vasodilating effect, such as nitrates, typically
cause a throbbing headache. Headaches may
also follow withdrawal from vasopressors, such
as caffeine, ergotamine, and sympathomimetics.
HERB ALERT Herbal remedies, such as
St. John’s wort, ginseng, and ephedra (ma
huang), can cause various adverse reactions, including headaches. (Note: The FDA has banned
the sale of dietary supplements containing
ephedra because they pose an unreasonable risk
of injury or illness.)
◆ Traction. Cervical traction with pins com-
monly causes a headache, which may be generalized or localized to pin insertion sites.
S
PECIAL CONSIDERATIONS
Continue to monitor the patient’s vital signs
and LOC. Watch for any change in the
headache’s severity or location. To help ease
the headache, administer an analgesic, darken
the patient’s room, and minimize other stimuli.
Explain the rationale of these interventions to
the patient.
Prepare the patient for diagnostic tests, such
as skull X-rays, computed tomography scan,
lumbar puncture, or cerebral arteriography.
P
EDIATRIC POINTERS
If a child is too young to describe his symptom,
suspect a headache if you see him banging or
holding his head. In an infant, a shrill cry or
bulging fontanels may indicate increased ICP
and headache. In a school-age child, ask the
parents about the child’s recent scholastic performance and about any problems at home that
may produce a tension headache.
Twice as many young boys have migraine
headaches as girls. In children older than age 3,
headache is the most common symptom of a
brain tumor.
Hearing loss
Affecting nearly 16 million Americans, hearing
loss may be temporary or permanent and partial
or complete. This common symptom may involve
reception of low-, middle-, or high-frequency
tones. If the hearing loss doesn’t affect speech frequencies, the patient may be unaware of it.
Normally, sound waves enter the external auditory canal and travel to the middle ear’s tympanic membrane and ossicles (incus, malleus,
and stapes) and then into the inner ear’s
cochlea. The cochlear division of the eighth cranial (auditory) nerve carries the sound impulse
to the brain. This type of sound transmission,
called air conduction, is normally better than
bone conduction—sound transmission through
bone to the inner ear.
Hearing loss can be classified as conductive,
sensorineural, mixed, or functional. Conductive
hearing loss results from external or middle ear
disorders that block sound transmission. This
type of hearing loss usually responds to medical
or surgical intervention (or in some cases, both).
Sensorineural hearing loss results from disorders
of the inner ear or of the eighth cranial nerve.
Mixed hearing loss combines aspects of conductive and sensorineural hearing loss. Functional
hearing loss results from psychological factors
rather than identifiable organic damage.
Hearing loss may also result from trauma, infection, allergy, tumors, certain systemic and
hereditary disorders, and the effects of ototoxic
drugs and treatments. In most cases, though, it
results from presbycusis, a type of sensorineural
hearing loss that usually affects people older

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349
than age 50. Other physiologic causes of hearing loss include cerumen (earwax) impaction;
barotitis media (unequal pressure on the
eardrum) associated with descent in an airplane
or elevator, diving, or close proximity to an explosion; and chronic exposure to noise over 90
decibels, which can occur on the job, with certain hobbies, or from listening to live or recorded music.
H
ISTORY AND PHYSICAL
EXAMINATION
If the patient reports hearing loss, ask him to
describe it fully. Is it unilateral or bilateral? Continuous or intermittent? Ask about a family history of hearing loss. Then obtain the patient’s
medical history, noting chronic ear infections,
ear surgery, and ear or head trauma. Has the
patient recently had an upper respiratory tract
infection? After taking a drug history, have the
patient describe his occupation and work environment.
Next, explore associated signs and symptoms.
Does the patient have ear pain? If so, is it unilateral or bilateral? Continuous or intermittent? Ask
the patient if he has noticed discharge from one
or both ears. If so, have him describe its color
and consistency, and note when it began. Does
he hear ringing, buzzing, hissing, or other noises
in one or both ears? If so, are the noises constant or intermittent? Does he experience any
dizziness? If so, when did he first notice it?
Begin the physical examination by inspecting
the external ear for inflammation, boils, foreign
bodies, and discharge. Then apply pressure to
the tragus and mastoid to elicit tenderness. If
you detect tenderness or external ear abnormalities, ask the physician whether an otoscopic
examination should be done. (See Using an oto-
scope correctly, page 255.) During the otoscopic
examination, note any color change, perforation, bulging, or retraction of the tympanic
membrane, which normally looks like a shiny,
pearl gray cone.
Next, evaluate the patient’s hearing acuity,
using the ticking watch and whispered voice
tests. Then perform the Weber and Rinne tests
to obtain a preliminary evaluation of the type
and degree of hearing loss. (See Differentiating
conductive from sensorineural hearing loss, page
350.)
M
EDICAL CAUSES
◆ Acoustic neuroma. This eighth cranial
nerve tumor causes unilateral, progressive, sen-
sorineural hearing loss. The patient may also
develop tinnitus, vertigo, and—with cranial
nerve compression—facial paralysis.
◆ Adenoid hypertrophy. Eustachian tube dys-
function gradually causes conductive hearing
loss accompanied by intermittent ear discharge.
The patient also tends to breathe through his
mouth and may complain of a sensation of ear
fullness.
◆ Allergies. Conductive hearing loss may re-
sult when an allergy produces eustachian tube
and middle ear congestion. Other features include ear pain or a feeling of fullness, nasal
congestion, and conjunctivitis.
◆ Aural polyps. If a polyp occludes the exter-
nal auditory canal, partial hearing loss may occur. The polyp typically bleeds easily and is covered by a purulent discharge.
◆ Cholesteatoma. Gradual hearing loss is
characteristic in this disorder and may be accompanied by vertigo and, at times, facial paralysis. Examination reveals eardrum perforation,
pearly white balls in the ear canal and, possibly,
a discharge.
◆ Cyst. Ear canal obstruction by a sebaceous or
dermoid cyst causes progressive conductive
hearing loss. On inspection, the cyst looks like a
soft mass.
◆ External ear canal tumor (malignant).
Progressive conductive hearing loss is characteristic and is accompanied by deep, boring ear
pain; a purulent discharge; and eventually facial
paralysis. Examination may detect the granular,
bleeding tumor.
◆ Furuncle. Reversible conductive hearing loss
may occur when one of these painful, hard nodules forms in the ear. The patient may report a
sense of fullness in the ear and pain on palpation of the tragus or auricle. Rupture relieves
the pain and produces a purulent, necrotic discharge.
◆ Glomus jugulare tumor. Initially, this be-
nign tumor causes mild, unilateral conductive
hearing loss that becomes progressively more
severe. The patient may report tinnitus that
sounds like his heartbeat. Associated signs
and symptoms include gradual congestion in
the affected ear, throbbing or pulsating discomfort, bloody otorrhea, facial nerve paralysis, and vertigo. Although the tympanic membrane is normal, a reddened mass appears
behind it.
◆ Glomus tympanum tumor. This cancerous
middle ear tumor causes slowly progressive
hearing loss and throbbing or pulsating tinnitus.

350 HEARING LOSS
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EXAMINATION TIP
Differentiating conductive from
sensorineural hearing loss
The Weber and Rinne tests can help determine whether the patient’s hearing loss is conductive or
sensorineural. The Weber test evaluates bone conduction; the Rinne test, bone and air conduction. Using a 512-Hz tuning fork, perform these preliminary tests as described below.
Weber test
Place the base of a vibrating tuning fork firmly against the
midline of the patient’s skull. Ask him if he hears the tone
equally well in both ears. If he does, the Weber test is
graded midline—a normal finding. In an abnormal Weber
test (graded right or left), sound is louder either in the impaired ear, suggesting a conductive hearing loss in that ear,
or in the normal ear, suggesting a sensorineural loss in the
opposite ear.
Rinne test
Hold the base of a vibrating tuning fork against the patient’s
mastoid process to test bone conduction (BC). Then quickly
move the vibrating fork in front of his ear canal to test air
conduction (AC). Ask him to tell you which location has the
louder or longer sound. Repeat the procedure for the other
ear. In a positive Rinne test, the AC sound lasts longer or is
louder than the BC sound—a normal finding. In a negative
test, the opposite is true: the BC sound lasts as long as or
longer than the AC sound. In sensorineural loss, the AC sound
lasts longer than the BC sound, but the BC sound is louder.
After performing both tests, correlate the results with oth-
er assessment data.
Implications of results
Conductive hearing loss produces:
◆ abnormal Weber test result
◆ negative Rinne test result
◆ improved hearing in noisy areas
◆ normal ability to discriminate sounds
◆ difficulty hearing when chewing
◆ a quiet speaking voice.
Sensorineural hearing loss produces:
◆ positive Rinne test
◆ poor hearing in noisy areas
◆ difficulty hearing high-frequency sounds
◆ complaints that others mumble or shout
◆ tinnitus
◆ loud speaking voice.
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