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HALO VISION
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341
membrane. Fever and cervical lymphadenopa­thy are also common.
◆ Renal failure (chronic). Renal failure pro-
duces a urinous or ammonia breath odor. Among its widespread effects are anemia, emo­tional lability, lethargy, irritability, decreased mental acuity, coarse muscular twitching, pe­ripheral neuropathies, muscle wasting, anorex­ia, signs of GI bleeding, ecchymosis, yellow­brown or bronze skin, pruritus, anuria, and increased blood pressure.
◆ Sinusitis. Acute sinusitis causes a purulent
nasal discharge that leads to halitosis. Besides a characteristic postnasal drip, the patient may exhibit nasal congestion, sore throat, cough, malaise, headache, facial pain and tenderness, and fever.
Chronic sinusitis causes a continuous mu­copurulent discharge that leads to a musty breath odor, postnasal drip, nasal congestion, and a chronic nonproductive cough.
◆ Zenker’s diverticulum. This esophageal dis-
order causes halitosis and a bad taste in the mouth associated with regurgitation. The pa­tient may also report a chronic cough that’s most pronounced at night, hoarseness, odynophagia, neck pain, and “gurgling” sounds in the throat when he swallows liquids.
O
THER CAUSES
◆ Drugs. Drugs that can cause halitosis include
triamterene, inhaled anesthetics, and any drugs known to cause metabolic acidosis such as ni­troprusside.
HERB ALERT Some herbal medicines, such
as garlic, may cause halitosis.
S
PECIAL CONSIDERATIONS
If examination of the mouth and sinuses doesn’t reveal the cause of halitosis, prepare the patient for upper GI and chest X-rays or endoscopy.
P
EDIATRIC POINTERS
In children, halitosis commonly results from physiologic causes, such as continual mouth breathing and thumb or blanket sucking. Phenylketonuria—a metabolic disorder that af­fects infants—may produce a musty or mousy breath odor.
G
ERIATRIC POINTERS
Extensive dental caries, mouth dryness, and poor oral hygiene can cause halitosis in elderly patients.
P
ATIENT COUNSELING
To help control halitosis, encourage good oral hygiene. If halitosis is drug induced, reassure the patient that it will disappear as soon as his body completely eliminates the drug.
Halo vision
Halo vision refers to seeing rainbowlike colored rings around lights or bright objects. The rain­bowlike effect can be explained by this physical principle: As light passes through water (in the eye, through tears or the cells of various antereti­nal media), it breaks up into spectral colors.
Halo vision usually develops suddenly; its du­ration depends on the causative disorder. This symptom may occur in disorders associated with excessive tearing and corneal epithelial edema. Among these causes, the most common and sig­nificant is acute angle-closure glaucoma, which can lead to blindness. In this ophthalmic emer­gency, increased intraocular pressure (IOP) forces fluid into corneal tissues anterior to Bowman’s membrane, causing edema. Halo vision is also an early symptom of cataracts, resulting from dispersion of light by abnormal lens opacity.
Nonpathologic causes of excessive tearing associated with halo vision include poorly fitted or overworn contact lenses, emotional ex­tremes, and exposure to intense light, as in snow blindness.
H
ISTORY AND PHYSICAL EXAMINATION
First, ask the patient how long he has been see­ing halos around lights and when he usually sees them. Patients with glaucoma usually see halos in the morning, when IOP is most elevat­ed. Ask the patient if light bothers his eyes. Does he have eye pain? If so, have him describe it. Remember that halos associated with excru­ciating eye pain or a severe headache may point to acute angle-closure glaucoma, an ocular emergency. Note a history of glaucoma or cataracts.
Next, examine the patient’s eyes, noting con­junctival injection, excessive tearing, and lens changes. Examine pupil size, shape, and re­sponse to light. Then test visual acuity by per­forming an ophthalmoscopic examination.
M
EDICAL CAUSES
◆ Cataract. Halo vision may be an early symp-
tom of painless, progressive cataract formation.
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The glare of headlights may blind the patient, making nighttime driving impossible. Other fea­tures include blurred vision, impaired visual acuity, and lens opacity, all of which develop gradually.
◆ Corneal endothelial dystrophy. Typically,
halo vision is a late symptom of this disorder, which may also cause impaired visual acuity.
◆ Glaucoma. Halo vision characterizes all
types of glaucoma. Acute angle-closure glauco- ma—an ophthalmic emergency—also causes blurred vision, followed by a severe headache or excruciating pain in and around the affected eye. Examination reveals a moderately dilated fixed pupil that doesn’t respond to light, con­junctival injection, a cloudy cornea, impaired vi­sual acuity and, possibly, nausea and vomiting.
Chronic angle-closure glaucoma usually pro-
duces no symptoms until pain and blindness oc­cur in advanced disease. Sometimes, halos and blurred vision develop slowly.
In chronic open-angle glaucoma, halo vision is
a late symptom that’s accompanied by mild eye ache, peripheral vision loss, and impaired visual acuity.
S
PECIAL CONSIDERATIONS
To help minimize halo vision, remind the pa­tient not to look directly at bright lights.
P
EDIATRIC POINTERS
Halo vision in a child usually results from con­genital cataracts or glaucoma. In a young child, limited verbal ability may make halo vision diffi­cult to assess.
G
ERIATRIC POINTERS
Primary glaucoma, the most common cause of halo vision, is more common in older patients.
Headache
The most common neurologic symptom, headaches may be localized or generalized, producing mild to severe pain. About 90% of all headaches are benign and can be described as vascular, muscle-contraction, or a combination of both. (See Comparing benign headaches.) Oc­casionally, though, headaches indicate a severe neurologic disorder associated with intracranial inflammation, increased intracranial pressure (ICP), or meningeal irritation. They may also re­sult from an ocular or sinus disorder, tests, drugs, or other treatments.
Other causes of headache include fever, eye­strain, dehydration, and systemic febrile illness­es. Headaches may occur in certain metabolic disturbances—such as hypoxemia, hypercapnia, hyperglycemia, and hypoglycemia—but they aren’t a diagnostic or prominent symptom in these disorders. Some individuals get headaches after seizures or from coughing, sneezing, heavy lifting, or stooping.
H
ISTORY AND PHYSICAL EXAMINATION
If the patient reports a headache, ask him to de­scribe its characteristics and location. How of­ten does he get a headache? How long does a typical headache last? Try to identify precipitat­ing factors, such as eating certain foods or ex­posure to bright lights. Ask what helps to relieve the headache. Is the patient under stress? Has he had trouble sleeping?
Take a drug and alcohol history, and ask about head trauma within the last 4 weeks. Has the patient recently experienced nausea, vomit­ing, photophobia, or visual changes? Does he feel drowsy, confused, or dizzy? Has he recently developed seizures, or does he have a history of seizures?
Begin the physical examination by evaluat­ing the patient’s level of consciousness (LOC). Then check his vital signs. Be alert for signs of increased ICP—widened pulse pressure, brady­cardia, altered respiratory pattern, and in­creased blood pressure. Check pupil size and response to light, and note any neck stiffness. (See Differential diagnosis: Headache, pages 344 and 345.)
M
EDICAL CAUSES
◆ Anthrax, cutaneous. Along with a macular
or papular lesion that develops into a vesicle and finally a painless ulcer, this disorder may produce a headache, lymphadenopathy, fever, and malaise.
◆ Brain abscess. In this disorder, the
headache is localized to the abscess site; it usu­ally intensifies over a few days and is aggravat­ed by straining. Accompanying the headache may be nausea, vomiting, and focal or general­ized seizures. The patient’s LOC varies from drowsiness to deep stupor. Depending on the abscess site, associated signs and symptoms may include aphasia, impaired visual acuity, hemiparesis, ataxia, tremors, and personality changes. Signs of infection, such as fever and pallor, usually develop late; however, if the
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Comparing benign headaches
Of the many patients who report headaches, only about 10% have an underlying medical disor­der. The other 90% suffer from benign headaches, which may be classified as muscle-contrac­tion (tension), vascular (migraine and cluster), or a combination of both.
As you review the chart below, you’ll see that the two major types— muscle-contraction and vascular headaches — are quite different. In a combined headache, features of both appear; this type of headache may affect the patient with a severe muscle-contraction headache or a late­stage migraine. Treatment of a combined headache includes analgesics and sedatives.
Characteristics
Incidence
Precipitating factors
Intensity and duration
Associated signs and symptoms
Muscle-contraction headaches
◆ Most common type, ac-
counting for 80% of all headaches
◆ Stress, anxiety, tension,
improper posture, and body alignment
◆ Prolonged muscle con-
traction without structural damage
◆ Eye, ear, and paranasal si-
nus disorders that produce reflex muscle contractions
◆ Produce an aching tight-
ness or a band of pain around the head, especially in the neck and in occipital and temporal areas
◆ Occur frequently and usu-
ally last for several hours
◆ Tense neck and facial
muscles
Vascular headaches
◆ More common in women and those
with a family history of migraines
◆ Onset after puberty
◆ Hormone fluctuations
◆ Alcohol
◆ Emotional upset
◆ Too little or too much sleep
◆ Foods, such as chocolate, cheese,
monosodium glutamate, and cured meats; caffeine withdrawal
◆ Weather changes, such as shifts in
barometric pressure
◆ May begin with an awareness of an
impending migraine or a 5- to 15­minute prodrome of neurologic deficits, such as visual disturbances, dizziness, unsteady gait, or tingling of the face, lips, or hands
◆ Produce severe, constant, throb-
bing pain that is typically unilateral and may be incapacitating
◆ Last for 4 to 6 hours
◆ Anorexia, nausea, and vomiting
◆ Occasionally, photophobia, sensi-
tivity to loud noises, weakness, and fatigue
◆ Depending on the type (cluster
headache or classic, common, or hemiplegic migraine), possibly chills, depression, eye pain, ptosis, tearing, rhinorrhea, diaphoresis, and facial flushing
Alleviating factors
◆ Mild analgesics, muscle
relaxants, or other drugs during an attack
◆ Measures to reduce stress,
such as biofeedback, relax­ation techniques, and coun­seling; posture correction to prevent attacks
◆ Methysergide and propranolol to
prevent vascular headache
◆ Ergot alkaloids or serotonin-
receptor drugs at the first sign of a migraine
◆ Rest in a quiet, darkened room
◆ Elimination of irritating foods
from diet
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Differential diagnosis: Headache
History of present illness
Focused physical examination: Neurologic and musculoskeletal systems;
head, eyes, ears, nose, and throat; neck; mental health; lymph nodes
Sinusitis
Signs and symptoms
◆ Dull periorbital
headache
◆ Unilateral or bi-
lateral frontal or maxillary sinus pain that’s in­creased by pal­pation or bending over
◆ Fever
◆ Malaise
◆ Nasal turbinate
edema
◆ Sore throat
◆ Nasal discharge
Diagnosis: Phys­ical examination, transillumination, sinus X-ray Treatment: Med­ication (decon­gestants, anal­gesics, antibiotics) Follow-up: None unless signs and symptoms worsen or recur
Brain
abscess
Signs and symptoms
◆ Localized head-
ache that increases over a few days
◆ Possible nausea
and vomiting
◆ Focal or gen-
eralized seizures
◆ Drowsiness
Subdural
hematoma
Signs and symptoms
◆ Decreased level
of consciousness (LOC)
◆ Acute drowsi-
ness, confusion, or agitation
◆ Pounding head-
ache
◆ Giddiness
◆ Personality
changes
◆ Dizziness
◆ Confusion
Encephalitis
Signs and symptoms
◆ Severe,
generalized headache
◆ Deteriorating
LOC within 48 hours of initial headache
◆ Fever
◆ Nuchal rigidity
◆ Irritability
◆ Seizures
◆ Nausea and
vomiting
◆ Photophobia
Diagnosis: Possible history of head trauma, lumbar puncture, imaging studies (computed tomography scan, magnetic resonance imaging, arteriography) Treatment: Medication (antibiotics if indicated; analgesics, anticonvulsants, osmotic diuretics); surgery if appropriate; chemotherapy or radiation therapy if malignancy is present Follow-up: Referral to neurologist or neurosurgeon
abscess remains encapsulated, these signs may not appear.
◆ Brain tumor. Initially, a tumor causes a lo-
calized headache near the tumor site; as the tu­mor grows, the headache eventually becomes
generalized. The pain is usually intermittent, deep seated, and dull and is most intense in the morning. It’s aggravated by coughing, stooping, Valsalva’s maneuver, and changes in head position, and it’s relieved by sitting and rest.
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Epidural
hemorrhage
Signs and symptoms
◆ Progressively
severe headache
◆ Unilateral
seizures
◆ Decreased LOC
◆ Hemiparesis or
hemiplegia
◆ High-grade
fever
Cerebral
aneurysm
(ruptured)
Signs and symptoms
◆ Sudden severe,
possibly unilateral headache
◆ Possible nausea
and vomiting
◆ Change in LOC
◆ Vision changes
Intracranial
hemorrhage
Signs and symptoms
◆ Severe gen-
eralized headache
◆ Rapid, steady
decrease in LOC
◆ Hemiparesis or
hemiplegia
◆ Aphasia
◆ Dizziness
◆ Nausea and
vomiting
◆ Irregular res-
pirations
◆ Positive Babin-
ski’s reflex
Brain tumor
Signs and symptoms
◆ Localized or
generalized head­ache
◆ Intermittent
deep pain that’s more intense in the morning and increases with Valsalva’s maneuver
◆ Personality
changes
◆ Changes in LOC
Associated signs and symptoms include person­ality changes, altered LOC, motor and sensory dysfunction, and eventually signs of increased ICP, such as vomiting, increased systolic blood pressure, and widened pulse pressure.
◆ Cerebral aneurysm (ruptured). Cerebral
aneurysm is a life-threatening disorder that’s characterized by a sudden excruciating headache, which may be unilateral and usually peaks within minutes of the rupture. The patient
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may lose consciousness immediately or display a variably altered LOC. Depending on the sever­ity and location of the bleeding, he may also ex­hibit nausea and vomiting; signs and symptoms of meningeal irritation, such as nuchal rigidity and blurred vision; hemiparesis; and other fea­tures.
◆ Ebola virus. A sudden headache commonly
occurs on the 5th day of this deadly illness. Ad­ditionally, the patient has a history of malaise, myalgia, high fever, diarrhea, abdominal pain, dehydration, and lethargy. A maculopapular rash develops between the 5th and 7th days of the illness. Other possible findings include pleu­ritic chest pain; a dry, hacking cough; pro­nounced pharyngitis; hematemesis; melena; and bleeding from the nose, gums, and vagina. Death usually occurs in the 2nd week of the ill­ness, preceded by massive blood loss and shock.
◆ Encephalitis. A severe, generalized
headache is characteristic with this disorder. Within 48 hours, the patient’s LOC typically de­teriorates—perhaps from lethargy to coma. As­sociated signs and symptoms include fever, nuchal rigidity, irritability, seizures, nausea and vomiting, photophobia, cranial nerve palsies such as ptosis, and focal neurologic deficits, such as hemiparesis and hemiplegia.
◆ Epidural hemorrhage (acute). Head trauma
and a sudden, brief loss of consciousness usual­ly precede this hemorrhage, which causes a progressively severe headache that’s accompa­nied by nausea and vomiting, bladder disten­tion, confusion, and then a rapid decrease in LOC. Other signs and symptoms include unilat­eral seizures, hemiparesis, hemiplegia, high fever, decreased pulse rate and bounding pulse, widened pulse pressure, increased blood pres­sure, a positive Babinski’s reflex, and decere­brate posture.
If the patient slips into a coma, his respira­tions deepen and become stertorous, then shal­low and irregular, and eventually cease. Pupil dilation may occur on the same side as the he­morrhage.
◆ Glaucoma, acute angle-closure. This type
of glaucoma is an ophthalmic emergency that may cause an excruciating headache as well as acute eye pain, blurred vision, halo vision, nau­sea, and vomiting. Assessment reveals conjunc­tival injection, a cloudy cornea, and a moder­ately dilated, fixed pupil.
◆ Hantavirus pulmonary syndrome. Noncar-
diogenic pulmonary edema distinguishes this
viral disease, which was first reported in the United States in 1993. Common reasons for seeking treatment include flulike signs and symptoms—headache, myalgia, fever, nausea, vomiting, and a cough—followed by respiratory distress. Fever, hypoxia, and (in some patients) serious hypotension typify the hospital course. Other signs and symptoms include a rising res­piratory rate (28 breaths/minute or more) and an increased heart rate (120 beats/minute or more).
◆ Hypertension. This disorder may cause a
slightly throbbing occipital headache on awak­ening that decreases in severity during the day. However, if the patient’s diastolic blood pres­sure exceeds 120 mm Hg, the headache remains constant. Associated signs and symptoms in­clude an atrial gallop, restlessness, confusion, nausea and vomiting, blurred vision, seizures, and altered LOC.
◆ Influenza. A severe generalized or frontal
headache usually begins suddenly with the flu. Accompanying signs and symptoms may last for 3 to 5 days and include stabbing retro-orbital pain, weakness, diffuse myalgia, fever, chills, coughing, rhinorrhea and, occasionally, hoarse­ness.
◆ Influenza type A H1N1 virus (swine flu).
Influenza type A H1N1, or swine flu, is a respira­tory disease of pigs caused by type A influenza virus. Swine flu viruses cause high levels of ill­ness and low death rates in pigs. Swine flu viruses normally don’t infect humans; however, sporadic human infections with swine flu have occurred. Most commonly, these cases occur in persons with direct exposure to pigs. The virus has changed slightly and is known as H1N1 flu. Recent outbreaks of H1N1 flu have shown that the virus can be transmitted from person to per­son, causing transmission across the globe. The H1N1 flu is similar to influenza, and causes ill­ness and in some cases death. The symptoms of swine flu include headache, nonproductive cough, fatigue, myalgia, chills, fever, and vomit­ing. The use of antiviral drugs is recommended to treat H1N1 flu.
◆ Intracerebral hemorrhage. In some pa-
tients, this hemorrhage produces a severe gen­eralized headache. Other signs and symptoms vary with the size and location of the hemor­rhage. A large hemorrhage may produce a rapid, steady decrease in LOC, perhaps resulting in a coma. Other common findings include hemiplegia, hemiparesis, abnormal pupil size and response, aphasia, dizziness, nausea,
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vomiting, seizures, decreased sensation, irregu­lar respirations, positive Babinski’s reflex, decorticate or decerebrate posture, and in­creased blood pressure.
◆ Listeriosis. If this infection spreads to the
nervous system, it may cause meningitis, whose signs and symptoms include headache, nuchal rigidity, fever, and change in LOC. Earlier signs and symptoms of listeriosis include fever, myal­gia, abdominal pain, nausea, vomiting, and di­arrhea.
GENDER CUE Listeriosis during pregnancy
may lead to premature delivery, infection of
the neonate, or stillbirth.
◆ Meningitis. This disorder is marked by the
sudden onset of a severe, constant, generalized headache that worsens with movement. Fever and chills are other early signs. As meningitis progresses, it also causes nuchal rigidity, posi­tive Kernig’s and Brudzinski’s signs, hyperreflex­ia, altered LOC, seizures, ocular palsies, facial weakness, hearing loss, vomiting and, possibly, opisthotonos and papilledema.
◆ Plague. The pneumonic form of this lethal
bacterial infection causes a sudden onset of headache, chills, fever, and myalgia. Pulmonary findings include a productive cough, chest pain, tachypnea, dyspnea, hemoptysis, respiratory distress, and cardiopulmonary insufficiency.
◆ Postconcussion syndrome. A generalized
or localized headache may develop 1 to 30 days after head trauma and last for 2 to 3 weeks. This characteristic symptom may be described as an aching, pounding, pressing, stabbing, or throbbing pain. The patient’s neurologic exami­nation is normal, but he may experience giddi­ness or dizziness, blurred vision, fatigue, in­somnia, inability to concentrate, and noise and alcohol intolerance.
◆ Q fever. Signs and symptoms of this disease
include severe headaches, fever, chills, malaise, chest pain, nausea, vomiting, and diarrhea. The fever may last for up to 2 weeks, and in severe cases, the patient may develop hepatitis or pneumonia.
◆ Severe acute respiratory syndrome
(SARS). SARS is an acute infectious disease caused by a coronavirus. Although most cases have been reported in Asia (China, Vietnam, Singapore, Thailand), cases have cropped up in Europe and North America. After an incubation period of 2 to 7 days, the illness generally be­gins with a fever (usually greater than 100.4° F [38° C]). Other symptoms include headache, malaise, a nonproductive cough, and dyspnea.
SARS may produce only mild symptoms, or it may progress to pneumonia and, in some cases, even respiratory failure and death.
◆ Sinusitis (acute). This disorder is usually
marked by a dull periorbital headache that’s usually aggravated by bending over or touching the face and is relieved by sinus drainage. Fever, sinus tenderness, nasal turbinate edema, sore throat, malaise, cough, and nasal discharge may accompany the headache.
◆ Smallpox (variola major). Initial signs and
symptoms of this virus include a severe headache, backache, abdominal pain, high fever, malaise, prostration, and a maculopapu­lar rash on the mucosa of the mouth, pharynx, face, and forearms and then on the trunk and legs. The rash becomes vesicular, then pustular. After 8 or 9 days, the pustules form a crust, which later separates from the skin, leaving a pitted scar. Death may result from encephalitis, extensive bleeding, or secondary infection.
◆ Subarachnoid hemorrhage. This hemor-
rhage commonly produces a sudden, violent headache along with nuchal rigidity, nausea and vomiting, seizures, dizziness, ipsilateral pupil dilation, and altered LOC that may rapidly progress to coma. The patient also exhibits pos­itive Kernig’s and Brudzinski’s signs, photopho­bia, blurred vision and, possibly, a fever. Focal signs and symptoms (such as hemiparesis, hemiplegia, sensory or vision disturbances, and aphasia) and signs of elevated ICP (such as bradycardia and increased blood pressure) may also occur.
◆ Subdural hematoma. Typically associated
with head trauma, both acute and chronic sub­dural hematomas may cause headache and de­creased LOC. An acute subdural hematoma also produces drowsiness, confusion, and agitation that may progress to coma. Later findings in­clude signs of increased ICP and focal neuro­logic deficits such as hemiparesis.
A chronic subdural hematoma produces a
dull, pounding headache that fluctuates in severity and is located over the hematoma. Weeks or months after the initial head trauma, the patient may experience giddiness, personal­ity changes, confusion, seizures, and progres­sively worsening LOC. Late signs may include unilateral pupil dilation, sluggish pupil reaction to light, and ptosis.
◆ Temporal arteritis. A throbbing unilateral
headache in the temporal or frontotemporal re­gion may be accompanied by vision loss, hear­ing loss, confusion, and fever. The temporal
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arteries are tender, swollen, nodular, and some­times erythematous.
◆ Tularemia. Signs and symptoms following
inhalation of the bacterium Francisella tularensis include abrupt onset of headache, fever, chills, generalized myalgia, a nonproductive cough, dyspnea, pleuritic chest pain, and empyema.
◆ Typhus. In typhus, initial symptoms of
headache, myalgia, arthralgia, and malaise are followed by an abrupt onset of chills, fever, nau­sea, and vomiting. A maculopapular rash may also occur.
◆ West Nile encephalitis. This brain infection
is caused by West Nile virus, a mosquito-borne flavivirus commonly found in Africa, West Asia, the Middle East and, rarely, in North America. Most patients have mild signs and symptoms, including fever, headache, body aches, rash, and swollen lymph glands. More severe infec­tion is marked by high fever, headache, neck stiffness, stupor, disorientation, coma, tremors, and paralysis.
O
THER CAUSES
◆ Diagnostic tests. A lumbar puncture or
myelogram may produce a throbbing frontal headache that worsens on standing.
◆ Drugs. A wide variety of drugs can cause
headaches. For example, indomethacin pro­duces headaches—usually in the morning—in many patients. Vasodilators and drugs with a vasodilating effect, such as nitrates, typically cause a throbbing headache. Headaches may also follow withdrawal from vasopressors, such as caffeine, ergotamine, and sympathomimet­ics.
HERB ALERT Herbal remedies, such as
St. John’s wort, ginseng, and ephedra (ma huang), can cause various adverse reactions, in­cluding headaches. (Note: The FDA has banned the sale of dietary supplements containing ephedra because they pose an unreasonable risk of injury or illness.)
◆ Traction. Cervical traction with pins com-
monly causes a headache, which may be gener­alized or localized to pin insertion sites.
S
PECIAL CONSIDERATIONS
Continue to monitor the patient’s vital signs and LOC. Watch for any change in the headache’s severity or location. To help ease the headache, administer an analgesic, darken the patient’s room, and minimize other stimuli. Explain the rationale of these interventions to the patient.
Prepare the patient for diagnostic tests, such as skull X-rays, computed tomography scan, lumbar puncture, or cerebral arteriography.
P
EDIATRIC POINTERS
If a child is too young to describe his symptom, suspect a headache if you see him banging or holding his head. In an infant, a shrill cry or bulging fontanels may indicate increased ICP and headache. In a school-age child, ask the parents about the child’s recent scholastic per­formance and about any problems at home that may produce a tension headache.
Twice as many young boys have migraine headaches as girls. In children older than age 3, headache is the most common symptom of a brain tumor.
Hearing loss
Affecting nearly 16 million Americans, hearing loss may be temporary or permanent and partial or complete. This common symptom may involve reception of low-, middle-, or high-frequency tones. If the hearing loss doesn’t affect speech fre­quencies, the patient may be unaware of it.
Normally, sound waves enter the external au­ditory canal and travel to the middle ear’s tym­panic membrane and ossicles (incus, malleus, and stapes) and then into the inner ear’s cochlea. The cochlear division of the eighth cra­nial (auditory) nerve carries the sound impulse to the brain. This type of sound transmission, called air conduction, is normally better than bone conduction—sound transmission through bone to the inner ear.
Hearing loss can be classified as conductive, sensorineural, mixed, or functional. Conductive hearing loss results from external or middle ear disorders that block sound transmission. This type of hearing loss usually responds to medical or surgical intervention (or in some cases, both). Sensorineural hearing loss results from disorders of the inner ear or of the eighth cranial nerve. Mixed hearing loss combines aspects of conduc­tive and sensorineural hearing loss. Functional hearing loss results from psychological factors rather than identifiable organic damage.
Hearing loss may also result from trauma, in­fection, allergy, tumors, certain systemic and hereditary disorders, and the effects of ototoxic drugs and treatments. In most cases, though, it results from presbycusis, a type of sensorineural hearing loss that usually affects people older
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349
than age 50. Other physiologic causes of hear­ing loss include cerumen (earwax) impaction; barotitis media (unequal pressure on the eardrum) associated with descent in an airplane or elevator, diving, or close proximity to an ex­plosion; and chronic exposure to noise over 90 decibels, which can occur on the job, with cer­tain hobbies, or from listening to live or record­ed music.
H
ISTORY AND PHYSICAL
EXAMINATION
If the patient reports hearing loss, ask him to describe it fully. Is it unilateral or bilateral? Con­tinuous or intermittent? Ask about a family his­tory of hearing loss. Then obtain the patient’s medical history, noting chronic ear infections, ear surgery, and ear or head trauma. Has the patient recently had an upper respiratory tract infection? After taking a drug history, have the patient describe his occupation and work envi­ronment.
Next, explore associated signs and symptoms. Does the patient have ear pain? If so, is it unilat­eral or bilateral? Continuous or intermittent? Ask the patient if he has noticed discharge from one or both ears. If so, have him describe its color and consistency, and note when it began. Does he hear ringing, buzzing, hissing, or other noises in one or both ears? If so, are the noises con­stant or intermittent? Does he experience any dizziness? If so, when did he first notice it?
Begin the physical examination by inspecting the external ear for inflammation, boils, foreign bodies, and discharge. Then apply pressure to the tragus and mastoid to elicit tenderness. If you detect tenderness or external ear abnormal­ities, ask the physician whether an otoscopic examination should be done. (See Using an oto- scope correctly, page 255.) During the otoscopic examination, note any color change, perfora­tion, bulging, or retraction of the tympanic membrane, which normally looks like a shiny, pearl gray cone.
Next, evaluate the patient’s hearing acuity, using the ticking watch and whispered voice tests. Then perform the Weber and Rinne tests to obtain a preliminary evaluation of the type and degree of hearing loss. (See Differentiating conductive from sensorineural hearing loss, page
350.)
M
EDICAL CAUSES
◆ Acoustic neuroma. This eighth cranial
nerve tumor causes unilateral, progressive, sen-
sorineural hearing loss. The patient may also develop tinnitus, vertigo, and—with cranial nerve compression—facial paralysis.
◆ Adenoid hypertrophy. Eustachian tube dys-
function gradually causes conductive hearing loss accompanied by intermittent ear discharge. The patient also tends to breathe through his mouth and may complain of a sensation of ear fullness.
◆ Allergies. Conductive hearing loss may re-
sult when an allergy produces eustachian tube and middle ear congestion. Other features in­clude ear pain or a feeling of fullness, nasal congestion, and conjunctivitis.
◆ Aural polyps. If a polyp occludes the exter-
nal auditory canal, partial hearing loss may oc­cur. The polyp typically bleeds easily and is cov­ered by a purulent discharge.
◆ Cholesteatoma. Gradual hearing loss is
characteristic in this disorder and may be ac­companied by vertigo and, at times, facial paral­ysis. Examination reveals eardrum perforation, pearly white balls in the ear canal and, possibly, a discharge.
◆ Cyst. Ear canal obstruction by a sebaceous or
dermoid cyst causes progressive conductive hearing loss. On inspection, the cyst looks like a soft mass.
◆ External ear canal tumor (malignant).
Progressive conductive hearing loss is charac­teristic and is accompanied by deep, boring ear pain; a purulent discharge; and eventually facial paralysis. Examination may detect the granular, bleeding tumor.
◆ Furuncle. Reversible conductive hearing loss
may occur when one of these painful, hard nod­ules forms in the ear. The patient may report a sense of fullness in the ear and pain on palpa­tion of the tragus or auricle. Rupture relieves the pain and produces a purulent, necrotic dis­charge.
◆ Glomus jugulare tumor. Initially, this be-
nign tumor causes mild, unilateral conductive hearing loss that becomes progressively more severe. The patient may report tinnitus that sounds like his heartbeat. Associated signs and symptoms include gradual congestion in the affected ear, throbbing or pulsating dis­comfort, bloody otorrhea, facial nerve paraly­sis, and vertigo. Although the tympanic mem­brane is normal, a reddened mass appears behind it.
◆ Glomus tympanum tumor. This cancerous
middle ear tumor causes slowly progressive hearing loss and throbbing or pulsating tinnitus.
350 HEARING LOSS
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EXAMINATION TIP
Differentiating conductive from sensorineural hearing loss
The Weber and Rinne tests can help determine whether the patient’s hearing loss is conductive or sensorineural. The Weber test evaluates bone conduction; the Rinne test, bone and air conduc­tion. Using a 512-Hz tuning fork, perform these preliminary tests as described below.
Weber test
Place the base of a vibrating tuning fork firmly against the midline of the patient’s skull. Ask him if he hears the tone equally well in both ears. If he does, the Weber test is graded midline—a normal finding. In an abnormal Weber test (graded right or left), sound is louder either in the im­paired ear, suggesting a conductive hearing loss in that ear, or in the normal ear, suggesting a sensorineural loss in the opposite ear.
Rinne test
Hold the base of a vibrating tuning fork against the patient’s mastoid process to test bone conduction (BC). Then quickly move the vibrating fork in front of his ear canal to test air conduction (AC). Ask him to tell you which location has the louder or longer sound. Repeat the procedure for the other ear. In a positive Rinne test, the AC sound lasts longer or is louder than the BC sound—a normal finding. In a negative test, the opposite is true: the BC sound lasts as long as or longer than the AC sound. In sensorineural loss, the AC sound lasts longer than the BC sound, but the BC sound is louder.
After performing both tests, correlate the results with oth-
er assessment data.
Implications of results
Conductive hearing loss produces:
◆ abnormal Weber test result
◆ negative Rinne test result
◆ improved hearing in noisy areas
◆ normal ability to discriminate sounds
◆ difficulty hearing when chewing
◆ a quiet speaking voice.
Sensorineural hearing loss produces:
◆ positive Rinne test
◆ poor hearing in noisy areas
◆ difficulty hearing high-frequency sounds
◆ complaints that others mumble or shout
◆ tinnitus
◆ loud speaking voice.