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186 CHAPTER 7: The Head and Neck
B. Palpation of the thyroid
from in front
from behind
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A. Palpation of the thyroid
FIG. 7-27 Palpation of the Thyroid Gland and Adjacent Structures. A. Palpation from behind.
B. Frontal palpation.
Inspection. With the patient seated in a good cross-light, inspect the anterior triangles in the lower half of the neck. Have the patient swallow looking for a mass ascending in the midline or behind the sternocleidomastoid. With obe­sity or a short neck, have the patient swallow with the neck tilted back while supporting the occiput with clasped hands.
Palpation from behind. The examiner is behind the seated patient who low­ers their chin relaxing the neck muscles. The thumbs are placed behind the neck curling the ngers anteriorly so long and ring nger tips just touch over the upper tracheal rings (Fig. 7-27A). The patient holds water in the mouth swallowing when asked. Locate the thyroid and cricoid cartilages and tra­cheal rings. The lateral lobes are on either side of the trachea rising under the ngers during a swallow. Feel for tissue overlying the tracheal rings; it is likely a hyperplastic thyroid isthmus. Palpate systematically the lower poles of both lateral lobes. During the exam, shift the inclination of the patient’s head to relax the neck muscles, and have the patient swallow to test the ad­herence of palpated masses to the trachea. Palpate the anterior surface of each lateral lobe through the sternocleidomastoid with the patient’s head slightly inclined toward the side being examined to relax the muscles. Occasionally, the thyroid is more easily felt when the neck is dorsiexed.
Palpation from the front. Place the ngers of one hand behind the neck with the thumb on the base of the thyroid cartilage (Fig. 7-27B) pushing the tra­chea gently away from the midline. The ngers of the other hand palpate the posterior aspect of the lateral lobe behind the sternocleidomastoid while the thumb palpates the anterior surface medial to the muscle. Having the patient swallow or depress the chin may further assist the examination. Pal­pate the other lateral lobe in the same manner with the tasks of the two hands reversed.
Auscultating a goiter. Auscultate goiters for a bruit using the stethoscope’s bell.
Head and Neck Symptoms 187
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Examining the Lymph Nodes: See Chapter 5, page 82.
Examining the Vascular System: See Chapter 8, page 287.
HEAD AND NECK SYMPTOMS
General Symptom
Headache. See Chapter 14, page 651.
Skull, Scalp, and Face Symptoms
Blushing and ushing. Transient dilation of supercial blood vessels of the head, face, and neck occurs with emotional, pharmacological, or physical stimulation. Flushing is a normal response to exercise, hot environments, and ingestion of vasoactive substances such as alcohol or capsaicin in hot peppers. Flushing is common in patients with rosacea or carcinoid syndrome, and in women at the menopause. Blushing is a term usually reserved for ushing associated with embarrassment or self-consciousness.
Face Pain. Facial pain is usually well localized, indicating the structure involved. Uncommonly, it is a difcult diagnostic problem.
CLINICAL OCCURRENCE: Use an anatomic approach for identifying the
cause, sorting the likely causes by the structure involved. Nerves: Trigeminal neuralgia, postherpetic neuralgia; sinus thrombosis; Teeth: Periapical abscess, periodontitis, unerupted teeth;
Bones: Sinusitis, osteomyelitis; Joints: Temporomandibular arthritis; Salivary Glands: Parotitis.
Blood Vessels: Temporal arteritis, cavernous
Trigeminal neuralgia (tic douloureux). See Chapter 14, page 654.
Herpes zoster. See Chapter 6, page 144. Unilateral sharp burning pain in
the distribution of one trigeminal nerve branch develops 2–3 days before vesicles appear. Persistent pain after resolution of the skin lesions is posther- petic neuralgia.
Acute suppurative sinusitis, orbital cellulitis. See pages 248 and 241.
Spasms of jaw muscles—trismus. See page 193.
Pain with chewing—masseter claudication. Ischemia of the masseter and/ or temporalis muscles is induced by chewing, especially tough meats, and relieved by rest. Patients alter their diet to avoid the pain. Giant cell arteritis should be suspected.
TMJ pain. Symptoms include pain, felt in the ear or temple, clicking, and occasionally locking. Trauma causes joint injury and crepitation. See page
240.
Numb chin syndrome. Invasion of the mental or inferior alveolar nerve
causes chin numbness. Patients complain of persistent chin numbness with-
out other symptoms or signs. If a thorough oral exam does not identify a local cause of nerve injury, a search for neoplastic disease is indicated.
188 CHAPTER 7: The Head and Neck
D. Bulging T. membrane
serous otitis
membrane
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Ear Symptoms
Tinnitus. Ringing in the ears, tinnitus, is often sufciently distressing for a patient to seek care. Unilateral tinnitus may be the rst symptom of an acous­tic neuroma.
CLINICAL OCCURRENCE: Outer Ear: Cerumen, foreign body or polyp in
the external meatus;
Inner Ear: Meniere disease, syphilis, fevers, labyrinth suppuration, basilar
skull fracture, acoustic neuroma, trauma; glycoside antibiotics.
Temporary altered hearing. Eustachian tube dysfunction causes mild inter­mittent pain, ear fullness, and altered hearing. Patients hear a popping sound with swallowing or yawning. The eardrum may be retracted (Fig. 7-28B).
Middle Ear: Inammation, otosclerosis, polychondritis;
Drugs: Quinine, salicylates, amino-
Earache.
The middle ear arises from the rst and second pharyngeal pouches. Pain is caused by inammation of ear structures or is referred from pharyn­geal sites, including the thyroid. Although the cause of acute ear pain is usu-
ally readily identied, chronic earache may offer a considerable diagnostic challenge.
CLINICAL OCCURRENCE: Auricle: Trauma, hematoma, frostbite, burn, epi-
thelioma, perichondritis, gout, eczema, impetigo, insect bites, carcinoma, herpes zoster;
Meatus: External otitis, malignant external otitis, carbuncle,
meatitis, eczema, hard cerumen, foreign body, injury, epithelioma, carci­noma, insect invasion, herpes zoster, trigeminal neuralgia (CN-V3); Middle
malleus meniscus
A. Normal tympanic
membrane
B. Retracted T.
membrane
E. Air bubbles in
C. Serous otitis with
meniscus
F. Perforated T.
FIG. 7-28 Lesions of the TM. A. Normal: The normal TM is slanted downward and forward; its surface glistens and
contains a brilli ant triangle, t he light reflex, w ith its apex at t he center, or umbo, an d its base at th e annuals. The ha ndle of the malleu s makes an impres sion on the disk from the umb o upward and for ward. B. The retracted eardrum: The light reflex is bent, and the malleus stands out in sharper relief than normally. C. Serous middle ear fluid: Hairline menisci curve from the handle of the malleus to the annulus. D. Bulging drumhead: The curves in the membrane obscure the normal landmarks of the malleus and distort the light reflex. E. Serous fluid mixed with air: Bubbles may be seen through the drumhead. F. Perforations of the membrane appear as oval holes with a dark shadow behind.
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Ear: Acute otitis media, acute mastoiditis, cholesteatoma, malignant disease; Referred Pain through CNs-V, IX, and X and the second and third cervical
nerves:
Unerupted lower third molar, carious teeth, TMJ arthritis, tonsilli­tis, carcinoma or sarcoma of pharynx, ulcer of epiglottis or larynx, cervical lymphadenitis, subacute thyroiditis, trigeminal neuralgia.
Dizziness and vertigo. See page 245.
Eye Symptoms
Double vision—diplopia. Perception of two visual images results from
refraction abnormalities or, less commonly, nonconjugative gaze. Deter-
mine the symptom pattern (e.g., vertical or horizontal), precipitating activi­ties, visual axis where diplopia occurs, and head position or gaze giving relief. If the patient reports monocular diplopia or diplopia when one eye is covered, the cause is nearly always refractive. Binocular diplopia: see Chapter 14, page 653.
Dry eyes. See Keratoconjunctivitis sicca, page 241.
Blurred vision.
failure of accommodation, i.e., altering lens shape for near and far vision, or light scattering by cornea, lens, or vitreous opacities. History is the key
to identifying the etiology. Eye pain suggests inammation (keratitis, iritis, and uveitis) or acute angle closure glaucoma. Abnormality of the oils in the tear lm is a frequent cause of visual aberration. Use of topical and systemic drugs, especially anticholinergics, dilate the pupil and decreases accommodation. Unilateral vision loss may also be described as “blurred vision,” meaning vision is less distinct than normal without binocular sight. The pinhole test (page 180) is used to determine if the blurred vision is refractive.
Eye pain. Eye pain is caused by inammation, infection, trauma, and
increased intraocular pressure. Inspect the lids, conjunctivae, and sclera for
lesions. Careful examination of the cornea, anterior chamber, iris, and retina are mandatory. Always assess visual acuity in each eye. Optimal examination requires an ophthalmologist.
CLINICAL OCCURRENCE: Degenerative/Idiopathic: Cluster headache;
Infectious: Infective keratitis (herpes simplex, zoster, and others), sinusitis
(ethmoid, frontal, sphenoid); zion, interstitial keratitis, iritis, iridocyclitis, episcleritis, scleritis, band kera­topathy, optic neuritis; Mechanical/Traumatic: Foreign body, corneal abrasion, entropion, glaucoma, eye strain.
Vision loss. Injury or impairment to any portion of the visual pathways
causes vision loss. Acute vision loss is a medical emergency (see page 243).
Chronic progressive vision loss is common with diseases of the cornea, lens, or retina. Standard tests of visual acuity will quantitate the impairment and formal visual eld testing is required. Ophthalmology referral is indicated.
Inability to sharply focus light on the retina is caused by
Inammatory/Immune: Hordeolum (sty), chala-
Nose Symptoms
Loss of smell—anosmia. See page 218.
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Abnormal smell or taste—dysgeusia. This is a common complaint in patients who have loss of smell (anosmia). If it is paroxysmal and associated with behavioral symptoms, it suggests complex partial seizures.
Lip, Mouth, Tongue, Teeth, and Pharynx Symptoms
Sore throat. See syndromes, pharyngitis, page 250.
Tongue or mouth soreness. Pain or tenderness in the tongue or mouth is
evaluated by inspection and palpation.
CLINICAL OCCURRENCE: No Lesions: Tobacco smoking, early glossitis
from all causes, menopausal symptom, heavy metal poisoning; Deep Lesions: Calculus in duct of submaxillary or sublingual gland, foreign body, myositis of lingual muscles, trichinosis, periostitis of hyoid bone, neoplasm of lingual muscles; Localized Supercial Lesions: Tongue biting, trauma to lingual frenu­lum, dental ulcer, injury while under anesthesia, foreign body (e.g., sh bone), epithelioma or carcinoma, ranula, tuberculous ulcer, herpes, Vincent stoma­titis, leukoplakia, thrush; Generalized Disease: Irradiation, pellagra, riboavin deciency, scurvy, pernicious anemia, atrophic glossitis, leukemia, exanthem­atous disorders, collagen diseases, pemphigus, cicatricial pemphigoid, lichen planus, heavy-metal poisoning, phenytoin, uremia, cancer chemotherapy, drug sensitivity, systemic fungal infections, e.g., histoplasmosis.
Irradiation injury. Therapeutic irradiation for head and neck malignancy causes temporary or permanent loss of saliva production. Within 2–4 weeks of the beginning of treatment, and lasting 6 or more weeks, patients experi­ence increasing dryness and generalized soreness of the mouth and throat.
Difcult or painful swallowing—dysphagia and odynophagia. Swallowing disorders are dysphagias. With oropharyngeal dysphagia, the patient describes difculty initiating a swallow or choking and coughing with swallowing. With esophageal dysphagia, the patient experiences a sense of obstruction at a denite level when uid or a food bolus is swallowed. Neurogenic dys- phagia is accompanied by regurgitation through the nose. Some dysphagias cause localized pain (odynophagia); others are painless. Deglutition involves muscles in the oropharynx and esophagus. Pain from the oropharynx is accurately localized, but esophageal pain is dispersed in the thoracic six­dermatome band, presenting as chest pain (see Chapter 8, page 292 and Chapter 9, page 411).
CLINICAL OCCURRENCE: Oropharynx—Painful Intrinsic Lesions: Glossitis,
tonsillitis, stomatitis, pharyngitis, laryngitis, lingual ulcer, carcinoma, pemphi­gus, erythema multiforme, Ludwig angina, mumps, bee sting on the tongue, angioedema, candidiasis, Plummer–Vinson syndrome; Painful Local Extrinsic
Lesions: Cervical adenitis, subacute thyroiditis, carotid arteritis, infected thy-
roglossal cysts or sinuses, pharyngeal cysts or sinuses, carotid body tumor, spur in cervical spine, pericarditis; Painful Systemic Conditions: Rabies, tetanus;
Painless Intrinsic Lesions: Cleft palate, neck exion from cervical osteoporosis,
xerostomia in Sjögren syndrome, magnesium deciency; Painless Neurogenic
Lesions: CN-IX or CN-X damage, globus hystericus, postdiphtheritic paralysis,
bulbar paralysis, West Nile virus, myasthenia gravis, amyotrophic lateral scle­rosis, Wilson disease, syphilis, parkinsonism, botulism, poisoning (lead, alcohol,
Head and Neck Symptoms 191
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uoride); Esophagus—Painful Intrinsic Lesions: (see Chapter 8 pages 292–293 and Chapter 9 page 411) Foreign body, carcinoma, esophagitis, diverticulum, hia-
tal hernia; Painless Intrinsic Lesions: Achalasia, congenital stricture, stricture, scleroderma, dermatomyositis; Sjögren syndrome, amyloidosis, thyrotoxico­sis; Painless Extrinsic Lesions: Aortic aneurysm, aberrant right subclavian artery (page 252), vertebral spurs, enlarged left atrium.
Larynx Symptoms
Hoarseness. See page 232.
Salivary Gland Symptoms
Dry mouth—xerostomia. See page 255.
Neck Symptoms
Neck pain. Neck pain is often readily diagnosed by a careful history, neck palpation, and oropharyngeal exam. Posttraumatic and postural cervical strain are most common. Palpate each anatomic structure systematically. Pain increasing with specic movements helps to localize the pain’s source.
CLINICAL OCCURRENCE: Neck Pain Increased by Swallowing—Pharynx:
Pharyngitis, Ludwig angina, inamed thyroglossal duct or cyst; Tonsils: Tonsillitis, neoplasm; Tongue: Ulcers, neoplasm; Larynx: Laryngitis, neoplasm, ulcer, foreign body; Esophagus: Inamed diverticulum, esophagitis; Thyroid: Suppurative or subacute thyroiditis, hemorrhage; Carotid artery: Carotodynia, carotid body tumor; Salivary glands: Mumps, suppurative parotitis; Neck
Pain Increased by Chewing—Mandible: Fracture, osteomyelitis, periodonti-
tis; Salivary Glands: Mumps, suppurative parotitis; Neck Pain Increased by
Head Movements—Sternocleidomastoid: Torticollis, hematoma; Neck Muscles:
Viral myalgia, muscle tension; Cervical Spine: Herniated intervertebral disk, spinal arthritis, meningitis, meningismus, craniovertebral junction abnormal­ities; Neck Pain Increased by Shoulder Movement—Superior Thoracic Aperture: Cervical rib, scalenus anticus syndrome, costoclavicular syndrome;
Pain Not Increased by Movement—Skin and Subcutaneous Tissues: Furuncle,
carbuncle, erysipelas; Lymph Nodes: Acute adenitis. Deep Veins: Septic throm­bophlebitis of the internal jugular vein (Lemmiere syndrome); Branchial Cleft
Remnants: Inamed pharyngeal cyst; Salivary Glands: Duct calculus; Subclavian Artery: aneurysm; Nervous System: Poliomyelitis, West Nile virus, herpes zos-
ter, epidural abscess, spinal cord neoplasm; Spinal Vertebrae: Herniated inter- vertebral disk, metastatic carcinoma; Referred Pain: Pancoast syndrome, angina pectoris, and other conditions in the six-dermatome band.
Neck
Carotodynia. Constant or throbbing pain in the anterior lateral neck intensi-
es with swallowing. It may radiate to the mandible or ear. Symptoms fre­quently follow viral pharyngitis with fever. Some patients have profound lassitude. Several relapses may occur within a few months. The carotid bulb is exquisitely tender and may seem enlarged with exaggerated pulsations; the common carotid may be tender as well. Carotid compression causes radiating pain along external carotid branches to the jaw, ear, and temple (Fay sign). One or both common carotid arteries is/are involved. The pharynx and larynx are normal or have slight hyperemia and edema. Carotid artery ten­derness is diagnostic.
192 CHAPTER 7: The Head and Neck
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Neck fullness. Goiters cause a sense of constriction or fullness in the neck. See page 256.
HEAD AND NECK SIGNS
Scalp, Face, Skull, and Jaw Signs
Scalp wounds. The scalp is extremely vascular so scalp wounds bleed pro­fusely. Gaping wounds have penetrated the galea aponeurotica and may con­tain an open skill fracture.
Fluctuant scalp mass—hematoma, abscess, fracture. Blood or pus accumu-
lating in the skin or subcutaneously form a discreet soft mass sliding readily over the skull. Blood or pus under an adult aponeurosis forms a boggy, uc­tuant mass covering the entire scalp. A uctuant mass bounded by the skull
suture lines indicates subperiosteal blood or pus or a depressed fracture. A subperiosteal hematoma usually has a soft plastic center and rm edges, feel­ing much like a depressed fracture.
Scalp cellulitis. The scalp is tender, soft, and boggy. Infection expands rap­idly causing edema of the eyelids and pinnae. Regional lymph nodes are swollen and tender.
Sebaceous cyst (wen). Arising from the skin, single or multiple cysts slide easily over the skull. Each is rm, nontender, and often hemispheric. Infected cysts bleed easily and may be mistaken for squamous cell carcinoma.
Scalp mass—lipoma. A smooth, soft, mobile, discreet subcutaneous mass, the nger slides easily around its edges. When beneath the pericranium, movement is limited, but palpation detects the smooth, rounded border.
Parotid enlargement. See page 234.
Preauricular abscess. A suppurating preauricular lymph node produces an
abscess that may ulcerate. The swelling is localized, tender, and sometimes
warm. The source of infection is in the side of the face, pinna, anterior wall of the external acoustic meatus, anterior third of the scalp, eyebrows, or eyelids.
Masseter muscle hypertrophy. Spontaneous hypertrophy of one or both masseter muscles produces facial swelling mimicking parotid gland swelling. If the entire mass hardens when the patient clenches his teeth, it is muscular.
Cheek erythema. Erythema, scaling, pustules, and tenderness in a malar distribution occur with sunburn, cellulitis, rosacea, seborrheic dermatitis, dis­coid or systemic lupus erythematosus (SLE), or acne vulgaris.
Forehead wrinkles. Absence of normal transverse furrowing with upward gaze is a sign of hyperthyroidism. Deep wrinkling, with longitudinal furrow­ing and prominence of intervening tissue is the bulldog skin of pachydermato­sis. Unilateral loss of wrinkling results from paralysis of muscles innervated by the facial nerve (CN-VII).
Head and Neck Signs 193
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Enlarged adult skull—Paget disease. See Chapter 13, page 590. In addition to bone pain, the patient complains of hats becoming too small. The calvarium is large compared with the facial bones. A bruit is sometimes heard in the skull.
Mastoid pain and tenderness—mastoiditis. See page 244.
Skull masses—neoplasms. Osteomas, frequent in the skull’s outer table,
produce a hard, sessile bony eminence. Pericranial sarcoma, metastatic car­cinoma, lymphoma, leukemia, or multiple myeloma can present as a hard or soft cranial bone mass.
Trismus. Trismus is tight jaw closure from spasm of the masticatory muscles.
Trismus, common with tetanus, has many other more common causes.
CLINICAL OCCURRENCE: Local Disorders: Impacted third molar, TMJ
arthritis, malignant external otitis, lymphadenitis, trigeminal neuralgia, scleroderma, dermatomyositis; Disorders with Widespread Muscle Spasm: Trichinosis, rabies, tetany, tetanus, strychnine poisoning, typhoid fever, chol­era, septicemia; Cerebral Disorders: Encephalitis, epilepsy (transient), cata­lepsy, hysteria, malingering.
Inability to close the jaw—TMJ dislocation.
biconcave surface, it easily partially subluxes or completely dislocates. The
jaw won’t close after a yawn or receipt of an upward blow on the chin with the mouth wide open. The mandible protrudes, the lower teeth overriding the uppers. There is a depression or pit anterior to the tragus that is more obvious when bilateral. In unilateral dislocation, the pretragal depression occurs only on the affected side. No movement of the mandibular head is felt when palpating through the external acoustic meatus on the affected side.
External Ear Signs
Earlobe crease. A visible crease extending at least one-third of the distance from tragus to posterior pinna is associated with a higher rate of cardiac events in hospital admissions with suspected coronary heart disease.
Earlobe nodule: gouty tophus. In long-standing gout, sodium urate crystals
accumulate in the helix and antihelix, the olecranon bursa, tendon sheaths, and aponeuroses of the extremities. The nodules are painless, hard, and irreg-
ular. They may open discharging chalky contents.
Darwin tubercle. This is a harmless developmental eminence in the upper third of the posterior helix that must be distinguished from acquired nodules, such as tophi.
Other nodules. Nontender nodules may be basal cell carcinomas, rheumatoid nodules, or leprosy. Cartilage calcication is a rare complication of Addison disease.
Hematoma. Trauma or a hemostatic defect results in blood accumulating between the cartilage and the perichondrium as a tender, blue, doughy mass, usually without spontaneous pain. Prompt incision and drainage avoid sup­puration or cauliower ear.
Because the TMJ is a shallow
194 CHAPTER 7: The Head and Neck
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Recurring inammation—relapsing polychondritis. There is inammation
and degeneration of cartilage especially of the pinna, nasal septum, laryn­geal cartilages, tracheal and bronchial rings; joint cartilages may be affected.
The ear is painful, swollen, and reddened, except over the lobule. Hoarseness indicates laryngeal involvement and blindness results from involvement of the sclerae, and tinnitus and deafness from middle ear involvement. Rarely, aortic or mitral valve ring degeneration produces valvular regurgitation or aortic aneurysm.
Dermoid cyst. A favorite site is just behind the pinna. It is soft and slightly uctuant.
External Acoustic Meatus Signs
Cerumen impaction. The wax of Native Americans and East Asians is often dry and akey, and more yellow than amber. Excessive wax production or a narrow meatus leads to impacted cerumen and partial or complete canal obstruction. Complete obstruction causes partial deafness; tinnitus or dizzi­ness may occur. Partial obstructions can suddenly become complete when water enters the meatus during bathing or swimming. The obstructing wax is easily seen in the external meatus.
Ear discharge—otorrhea. Ear discharge has many causes, the type suggesting the diagnosis: Yellow Discharge: Melting cerumen; Serous Discharge: Eczema, early ruptured acute otitis media; Bloody Discharge: External canal trauma or longitudinal temporal bone fracture with TM and external canal laceration;
Purulent Discharge: Chronic external otitis, perforating acute suppurative oti-
tis media, chronic suppurative or tuberculous otitis media with or without cholesteatoma.
External otitis. See page 243.
Dermatitis. Seborrheic dermatitis commonly causes scaling and pruritus of
the choana and meatus. Medicated eardrops can cause contact dermatitis.
Carcinoma. Either squamous cell or basal cell carcinoma can involve the meatal epithelium. Pain and discharge are presenting symptoms, with deaf­ness and facial paralysis occurring in advanced disease.
Foreign body. Children often place objects in their ears. A purulent discharge from the canal or an earache may be the rst indication.
Polyps. A bulbous, reddened, pedunculated mass arising from the canal wall or middle ear is associated with a foul purulent discharge. Moving it with forceps may reveal the origin.
Exostoses and chondromas. Exostoses form nodules in the osseous canal near the TM. They rarely produce obstruction, although the TM may be par­tially obscured. A single bony osteoma may occur. Rarely, chondromas arise from the cartilaginous canal, usually without obstruction.
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Furuncle. A red, tender prominence with or without a pustule forms in the cartilaginous canal producing extreme pain.
Vesicles. Pain in the ear with vesicles in the canal and facial weakness is caused by herpes zoster of CN-VII (Ramsey–Hunt Syndrome), Chapter 14, Facial Weakness and Paralysis, pages 665–666.
TM Signs
Retracted TM. See Otitis Media with Effusion, page 244.
Red or bulging TM. See Acute Suppurative Otitis Media, page 244.
Vesicles on the TM. Mycoplasma pneumoniae infection causes severe ear pain
and an inamed TM often with hemorrhagic vesicles (bullous myringitis).
Perforated TM. A healed suppurative middle ear infection eroded through the TM leaving an oval hole through which the middle ear cavity is seen. Chronic perforations are asymptomatic other than mildly decreased auditory acuity.
Hearing Signs
Lateralizing Weber test—ipsilateral conductive hearing loss or contra­lateral neurosensory loss. When neurosensory hearing is intact bilaterally,
sound lateralizes to the side of conductive loss that has lost the masking effect of background noise. Neurosensory loss on one side results in a louder sound on the opposite side. Therefore, lateralization of sound to the right ear means conductive loss on the right or perceptive loss on the left.
Bone conduction greater than air conduction (Rinne-negative test)— conductive hearing loss. When amplication of sound by the TM and ossi-
cles is impaired, direct transmission of vibrations to the cochlea through bone appears louder than sound transmitted through air. Conductive hearing loss results from auditory canal obstruction, TM damage, middle ear uid, and destruction or ankylosis of the ossicles.
Balance and position sense signs. See Chapter 14, page 671.
Eye Lid Signs
Lacrimation, tearing. Although strictly speaking an overproduction of tears, lacrimation usually refers to any condition resulting in tears. Epiphora means an overow of tears from any cause.
CLINICAL OCCURRENCE: Increased Secretion: Weeping from emotion, for-
eign body irritation, corneal ulcer, conjunctivitis, coryza, measles, hay fever, poisoning (iodide, bromide, arsenic); cicatrix, eyelid edema, lacrimal calculus, dacryocystitis; Puncta Separation
from the Globe: Facial paralysis, aging, chronic marginal blepharitis, ectropion,
proptosis.
Widened palpebral ssures. The ssures are widened by lid retraction (con-
traction of Mueller muscle) or protrusion of the globe. Normally, with eyes
Lacrimal Duct Obstruction: Congenital,