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186 CHAPTER 7: The Head and Neck
B. Palpation of the thyroid
from in front
from behind
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A. Palpation of the thyroid
FIG. 7-27 Palpation of the Thyroid Gland and Adjacent Structures. A. Palpation from behind.
B. Frontal palpation.
Inspection. With the patient seated in a good cross-light, inspect the anterior
triangles in the lower half of the neck. Have the patient swallow looking for a
mass ascending in the midline or behind the sternocleidomastoid. With obesity or a short neck, have the patient swallow with the neck tilted back while
supporting the occiput with clasped hands.
Palpation from behind. The examiner is behind the seated patient who lowers their chin relaxing the neck muscles. The thumbs are placed behind the
neck curling the ngers anteriorly so long and ring nger tips just touch over
the upper tracheal rings (Fig. 7-27A). The patient holds water in the mouth
swallowing when asked. Locate the thyroid and cricoid cartilages and tracheal rings. The lateral lobes are on either side of the trachea rising under
the ngers during a swallow. Feel for tissue overlying the tracheal rings; it is
likely a hyperplastic thyroid isthmus. Palpate systematically the lower poles
of both lateral lobes. During the exam, shift the inclination of the patient’s
head to relax the neck muscles, and have the patient swallow to test the adherence of palpated masses to the trachea. Palpate the anterior surface of each
lateral lobe through the sternocleidomastoid with the patient’s head slightly
inclined toward the side being examined to relax the muscles. Occasionally,
the thyroid is more easily felt when the neck is dorsiexed.
Palpation from the front. Place the ngers of one hand behind the neck with
the thumb on the base of the thyroid cartilage (Fig. 7-27B) pushing the trachea gently away from the midline. The ngers of the other hand palpate
the posterior aspect of the lateral lobe behind the sternocleidomastoid while
the thumb palpates the anterior surface medial to the muscle. Having the
patient swallow or depress the chin may further assist the examination. Palpate the other lateral lobe in the same manner with the tasks of the two
hands reversed.
Auscultating a goiter. Auscultate goiters for a bruit using the stethoscope’s
bell.

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Examining the Lymph Nodes: See Chapter 5, page 82.
Examining the Vascular System: See Chapter 8, page 287.
HEAD AND NECK SYMPTOMS
General Symptom
Headache. See Chapter 14, page 651.
Skull, Scalp, and Face Symptoms
Blushing and ushing. Transient dilation of supercial blood vessels of the
head, face, and neck occurs with emotional, pharmacological, or physical
stimulation. Flushing is a normal response to exercise, hot environments, and
ingestion of vasoactive substances such as alcohol or capsaicin in hot peppers.
Flushing is common in patients with rosacea or carcinoid syndrome, and in
women at the menopause. Blushing is a term usually reserved for ushing
associated with embarrassment or self-consciousness.
Face Pain. Facial pain is usually well localized, indicating the structure
involved. Uncommonly, it is a difcult diagnostic problem.
CLINICAL OCCURRENCE: Use an anatomic approach for identifying the
cause, sorting the likely causes by the structure involved. Nerves: Trigeminal
neuralgia, postherpetic neuralgia;
sinus thrombosis; Teeth: Periapical abscess, periodontitis, unerupted teeth;
Bones: Sinusitis, osteomyelitis; Joints: Temporomandibular arthritis; Salivary
Glands: Parotitis.
Blood Vessels: Temporal arteritis, cavernous
Trigeminal neuralgia (tic douloureux). See Chapter 14, page 654.
Herpes zoster. See Chapter 6, page 144. Unilateral sharp burning pain in
the distribution of one trigeminal nerve branch develops 2–3 days before
vesicles appear. Persistent pain after resolution of the skin lesions is posther-
petic neuralgia.
Acute suppurative sinusitis, orbital cellulitis. See pages 248 and 241.
Spasms of jaw muscles—trismus. See page 193.
Pain with chewing—masseter claudication. Ischemia of the masseter and/
or temporalis muscles is induced by chewing, especially tough meats, and
relieved by rest. Patients alter their diet to avoid the pain. Giant cell arteritis
should be suspected.
TMJ pain. Symptoms include pain, felt in the ear or temple, clicking, and
occasionally locking. Trauma causes joint injury and crepitation. See page
240.
Numb chin syndrome. Invasion of the mental or inferior alveolar nerve
causes chin numbness. Patients complain of persistent chin numbness with-
out other symptoms or signs. If a thorough oral exam does not identify a local
cause of nerve injury, a search for neoplastic disease is indicated.

188 CHAPTER 7: The Head and Neck
D. Bulging T. membrane
serous otitis
membrane
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Ear Symptoms
Tinnitus. Ringing in the ears, tinnitus, is often sufciently distressing for a
patient to seek care. Unilateral tinnitus may be the rst symptom of an acoustic neuroma.
CLINICAL OCCURRENCE: Outer Ear: Cerumen, foreign body or polyp in
the external meatus;
Inner Ear: Meniere disease, syphilis, fevers, labyrinth suppuration, basilar
skull fracture, acoustic neuroma, trauma;
glycoside antibiotics.
Temporary altered hearing. Eustachian tube dysfunction causes mild intermittent pain, ear fullness, and altered hearing. Patients hear a popping sound
with swallowing or yawning. The eardrum may be retracted (Fig. 7-28B).
Middle Ear: Inammation, otosclerosis, polychondritis;
Drugs: Quinine, salicylates, amino-
Earache.
The middle ear arises from the rst and second pharyngeal pouches.
Pain is caused by inammation of ear structures or is referred from pharyngeal sites, including the thyroid. Although the cause of acute ear pain is usu-
ally readily identied, chronic earache may offer a considerable diagnostic
challenge.
CLINICAL OCCURRENCE: Auricle: Trauma, hematoma, frostbite, burn, epi-
thelioma, perichondritis, gout, eczema, impetigo, insect bites, carcinoma,
herpes zoster;
Meatus: External otitis, malignant external otitis, carbuncle,
meatitis, eczema, hard cerumen, foreign body, injury, epithelioma, carcinoma, insect invasion, herpes zoster, trigeminal neuralgia (CN-V3); Middle
malleus meniscus
A. Normal tympanic
membrane
B. Retracted T.
membrane
E. Air bubbles in
C. Serous otitis with
meniscus
F. Perforated T.
FIG. 7-28 Lesions of the TM. A. Normal: The normal TM is slanted downward and forward; its surface glistens and
contains a brilli ant triangle, t he light reflex, w ith its apex at t he center, or umbo, an d its base at th e annuals. The ha ndle of the
malleu s makes an impres sion on the disk from the umb o upward and for ward. B. The retracted eardrum: The light reflex
is bent, and the malleus stands out in sharper relief than normally. C. Serous middle ear fluid: Hairline menisci curve
from the handle of the malleus to the annulus. D. Bulging drumhead: The curves in the membrane obscure the normal
landmarks of the malleus and distort the light reflex. E. Serous fluid mixed with air: Bubbles may be seen through the
drumhead. F. Perforations of the membrane appear as oval holes with a dark shadow behind.

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Ear: Acute otitis media, acute mastoiditis, cholesteatoma, malignant disease;
Referred Pain through CNs-V, IX, and X and the second and third cervical
nerves:
Unerupted lower third molar, carious teeth, TMJ arthritis, tonsillitis, carcinoma or sarcoma of pharynx, ulcer of epiglottis or larynx, cervical
lymphadenitis, subacute thyroiditis, trigeminal neuralgia.
Dizziness and vertigo. See page 245.
Eye Symptoms
Double vision—diplopia. Perception of two visual images results from
refraction abnormalities or, less commonly, nonconjugative gaze. Deter-
mine the symptom pattern (e.g., vertical or horizontal), precipitating activities, visual axis where diplopia occurs, and head position or gaze giving
relief. If the patient reports monocular diplopia or diplopia when one eye is
covered, the cause is nearly always refractive. Binocular diplopia: see Chapter
14, page 653.
Dry eyes. See Keratoconjunctivitis sicca, page 241.
Blurred vision.
failure of accommodation, i.e., altering lens shape for near and far vision,
or light scattering by cornea, lens, or vitreous opacities. History is the key
to identifying the etiology. Eye pain suggests inammation (keratitis, iritis,
and uveitis) or acute angle closure glaucoma. Abnormality of the oils in
the tear lm is a frequent cause of visual aberration. Use of topical and
systemic drugs, especially anticholinergics, dilate the pupil and decreases
accommodation. Unilateral vision loss may also be described as “blurred
vision,” meaning vision is less distinct than normal without binocular
sight. The pinhole test (page 180) is used to determine if the blurred vision
is refractive.
Eye pain. Eye pain is caused by inammation, infection, trauma, and
increased intraocular pressure. Inspect the lids, conjunctivae, and sclera for
lesions. Careful examination of the cornea, anterior chamber, iris, and retina
are mandatory. Always assess visual acuity in each eye. Optimal examination
requires an ophthalmologist.
CLINICAL OCCURRENCE: Degenerative/Idiopathic: Cluster headache;
Infectious: Infective keratitis (herpes simplex, zoster, and others), sinusitis
(ethmoid, frontal, sphenoid);
zion, interstitial keratitis, iritis, iridocyclitis, episcleritis, scleritis, band keratopathy, optic neuritis; Mechanical/Traumatic: Foreign body, corneal abrasion,
entropion, glaucoma, eye strain.
Vision loss. Injury or impairment to any portion of the visual pathways
causes vision loss. Acute vision loss is a medical emergency (see page 243).
Chronic progressive vision loss is common with diseases of the cornea, lens,
or retina. Standard tests of visual acuity will quantitate the impairment and
formal visual eld testing is required. Ophthalmology referral is indicated.
Inability to sharply focus light on the retina is caused by
Inammatory/Immune: Hordeolum (sty), chala-
Nose Symptoms
Loss of smell—anosmia. See page 218.

190 CHAPTER 7: The Head and Neck
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Abnormal smell or taste—dysgeusia. This is a common complaint in patients
who have loss of smell (anosmia). If it is paroxysmal and associated with
behavioral symptoms, it suggests complex partial seizures.
Lip, Mouth, Tongue, Teeth, and Pharynx Symptoms
Sore throat. See syndromes, pharyngitis, page 250.
Tongue or mouth soreness. Pain or tenderness in the tongue or mouth is
evaluated by inspection and palpation.
CLINICAL OCCURRENCE: No Lesions: Tobacco smoking, early glossitis
from all causes, menopausal symptom, heavy metal poisoning; Deep Lesions:
Calculus in duct of submaxillary or sublingual gland, foreign body, myositis
of lingual muscles, trichinosis, periostitis of hyoid bone, neoplasm of lingual
muscles; Localized Supercial Lesions: Tongue biting, trauma to lingual frenulum, dental ulcer, injury while under anesthesia, foreign body (e.g., sh bone),
epithelioma or carcinoma, ranula, tuberculous ulcer, herpes, Vincent stomatitis, leukoplakia, thrush; Generalized Disease: Irradiation, pellagra, riboavin
deciency, scurvy, pernicious anemia, atrophic glossitis, leukemia, exanthematous disorders, collagen diseases, pemphigus, cicatricial pemphigoid, lichen
planus, heavy-metal poisoning, phenytoin, uremia, cancer chemotherapy,
drug sensitivity, systemic fungal infections, e.g., histoplasmosis.
Irradiation injury. Therapeutic irradiation for head and neck malignancy
causes temporary or permanent loss of saliva production. Within 2–4 weeks
of the beginning of treatment, and lasting 6 or more weeks, patients experience increasing dryness and generalized soreness of the mouth and throat.
Difcult or painful swallowing—dysphagia and odynophagia. Swallowing
disorders are dysphagias. With oropharyngeal dysphagia, the patient describes
difculty initiating a swallow or choking and coughing with swallowing.
With esophageal dysphagia, the patient experiences a sense of obstruction at
a denite level when uid or a food bolus is swallowed. Neurogenic dys-
phagia is accompanied by regurgitation through the nose. Some dysphagias
cause localized pain (odynophagia); others are painless. Deglutition involves
muscles in the oropharynx and esophagus. Pain from the oropharynx is
accurately localized, but esophageal pain is dispersed in the thoracic sixdermatome band, presenting as chest pain (see Chapter 8, page 292 and
Chapter 9, page 411).
CLINICAL OCCURRENCE: Oropharynx—Painful Intrinsic Lesions: Glossitis,
tonsillitis, stomatitis, pharyngitis, laryngitis, lingual ulcer, carcinoma, pemphigus, erythema multiforme, Ludwig angina, mumps, bee sting on the tongue,
angioedema, candidiasis, Plummer–Vinson syndrome; Painful Local Extrinsic
Lesions: Cervical adenitis, subacute thyroiditis, carotid arteritis, infected thy-
roglossal cysts or sinuses, pharyngeal cysts or sinuses, carotid body tumor,
spur in cervical spine, pericarditis; Painful Systemic Conditions: Rabies, tetanus;
Painless Intrinsic Lesions: Cleft palate, neck exion from cervical osteoporosis,
xerostomia in Sjögren syndrome, magnesium deciency; Painless Neurogenic
Lesions: CN-IX or CN-X damage, globus hystericus, postdiphtheritic paralysis,
bulbar paralysis, West Nile virus, myasthenia gravis, amyotrophic lateral sclerosis, Wilson disease, syphilis, parkinsonism, botulism, poisoning (lead, alcohol,

Head and Neck Symptoms 191
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uoride); Esophagus—Painful Intrinsic Lesions: (see Chapter 8 pages 292–293 and
Chapter 9 page 411) Foreign body, carcinoma, esophagitis, diverticulum, hia-
tal hernia; Painless Intrinsic Lesions: Achalasia, congenital stricture, stricture,
scleroderma, dermatomyositis; Sjögren syndrome, amyloidosis, thyrotoxicosis; Painless Extrinsic Lesions: Aortic aneurysm, aberrant right subclavian artery
(page 252), vertebral spurs, enlarged left atrium.
Larynx Symptoms
Hoarseness. See page 232.
Salivary Gland Symptoms
Dry mouth—xerostomia. See page 255.
Neck Symptoms
Neck pain. Neck pain is often readily diagnosed by a careful history, neck
palpation, and oropharyngeal exam. Posttraumatic and postural cervical
strain are most common. Palpate each anatomic structure systematically. Pain
increasing with specic movements helps to localize the pain’s source.
CLINICAL OCCURRENCE: Neck Pain Increased by Swallowing—Pharynx:
Pharyngitis, Ludwig angina, inamed thyroglossal duct or cyst; Tonsils:
Tonsillitis, neoplasm; Tongue: Ulcers, neoplasm; Larynx: Laryngitis, neoplasm,
ulcer, foreign body; Esophagus: Inamed diverticulum, esophagitis; Thyroid:
Suppurative or subacute thyroiditis, hemorrhage; Carotid artery: Carotodynia,
carotid body tumor; Salivary glands: Mumps, suppurative parotitis; Neck
Pain Increased by Chewing—Mandible: Fracture, osteomyelitis, periodonti-
tis; Salivary Glands: Mumps, suppurative parotitis; Neck Pain Increased by
Head Movements—Sternocleidomastoid: Torticollis, hematoma; Neck Muscles:
Viral myalgia, muscle tension; Cervical Spine: Herniated intervertebral disk,
spinal arthritis, meningitis, meningismus, craniovertebral junction abnormalities; Neck Pain Increased by Shoulder Movement—Superior Thoracic Aperture:
Cervical rib, scalenus anticus syndrome, costoclavicular syndrome;
Pain Not Increased by Movement—Skin and Subcutaneous Tissues: Furuncle,
carbuncle, erysipelas; Lymph Nodes: Acute adenitis. Deep Veins: Septic thrombophlebitis of the internal jugular vein (Lemmiere syndrome); Branchial Cleft
Remnants: Inamed pharyngeal cyst; Salivary Glands: Duct calculus; Subclavian
Artery: aneurysm; Nervous System: Poliomyelitis, West Nile virus, herpes zos-
ter, epidural abscess, spinal cord neoplasm; Spinal Vertebrae: Herniated inter-
vertebral disk, metastatic carcinoma; Referred Pain: Pancoast syndrome,
angina pectoris, and other conditions in the six-dermatome band.
Neck
Carotodynia. Constant or throbbing pain in the anterior lateral neck intensi-
es with swallowing. It may radiate to the mandible or ear. Symptoms frequently follow viral pharyngitis with fever. Some patients have profound
lassitude. Several relapses may occur within a few months. The carotid bulb
is exquisitely tender and may seem enlarged with exaggerated pulsations;
the common carotid may be tender as well. Carotid compression causes
radiating pain along external carotid branches to the jaw, ear, and temple (Fay
sign). One or both common carotid arteries is/are involved. The pharynx and
larynx are normal or have slight hyperemia and edema. Carotid artery tenderness is diagnostic.

192 CHAPTER 7: The Head and Neck
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Neck fullness. Goiters cause a sense of constriction or fullness in the neck.
See page 256.
HEAD AND NECK SIGNS
Scalp, Face, Skull, and Jaw Signs
Scalp wounds. The scalp is extremely vascular so scalp wounds bleed profusely. Gaping wounds have penetrated the galea aponeurotica and may contain an open skill fracture.
Fluctuant scalp mass—hematoma, abscess, fracture. Blood or pus accumu-
lating in the skin or subcutaneously form a discreet soft mass sliding readily
over the skull. Blood or pus under an adult aponeurosis forms a boggy, uctuant mass covering the entire scalp. A uctuant mass bounded by the skull
suture lines indicates subperiosteal blood or pus or a depressed fracture. A
subperiosteal hematoma usually has a soft plastic center and rm edges, feeling much like a depressed fracture.
Scalp cellulitis. The scalp is tender, soft, and boggy. Infection expands rapidly causing edema of the eyelids and pinnae. Regional lymph nodes are
swollen and tender.
Sebaceous cyst (wen). Arising from the skin, single or multiple cysts slide
easily over the skull. Each is rm, nontender, and often hemispheric. Infected
cysts bleed easily and may be mistaken for squamous cell carcinoma.
Scalp mass—lipoma. A smooth, soft, mobile, discreet subcutaneous mass,
the nger slides easily around its edges. When beneath the pericranium,
movement is limited, but palpation detects the smooth, rounded border.
Parotid enlargement. See page 234.
Preauricular abscess. A suppurating preauricular lymph node produces an
abscess that may ulcerate. The swelling is localized, tender, and sometimes
warm. The source of infection is in the side of the face, pinna, anterior wall
of the external acoustic meatus, anterior third of the scalp, eyebrows, or
eyelids.
Masseter muscle hypertrophy. Spontaneous hypertrophy of one or both
masseter muscles produces facial swelling mimicking parotid gland swelling.
If the entire mass hardens when the patient clenches his teeth, it is muscular.
Cheek erythema. Erythema, scaling, pustules, and tenderness in a malar
distribution occur with sunburn, cellulitis, rosacea, seborrheic dermatitis, discoid or systemic lupus erythematosus (SLE), or acne vulgaris.
Forehead wrinkles. Absence of normal transverse furrowing with upward
gaze is a sign of hyperthyroidism. Deep wrinkling, with longitudinal furrowing and prominence of intervening tissue is the bulldog skin of pachydermatosis. Unilateral loss of wrinkling results from paralysis of muscles innervated
by the facial nerve (CN-VII).

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Enlarged adult skull—Paget disease. See Chapter 13, page 590. In addition to
bone pain, the patient complains of hats becoming too small. The calvarium is
large compared with the facial bones. A bruit is sometimes heard in the skull.
Mastoid pain and tenderness—mastoiditis. See page 244.
Skull masses—neoplasms. Osteomas, frequent in the skull’s outer table,
produce a hard, sessile bony eminence. Pericranial sarcoma, metastatic carcinoma, lymphoma, leukemia, or multiple myeloma can present as a hard or
soft cranial bone mass.
Trismus. Trismus is tight jaw closure from spasm of the masticatory muscles.
Trismus, common with tetanus, has many other more common causes.
CLINICAL OCCURRENCE: Local Disorders: Impacted third molar, TMJ
arthritis, malignant external otitis, lymphadenitis, trigeminal neuralgia,
scleroderma, dermatomyositis; Disorders with Widespread Muscle Spasm:
Trichinosis, rabies, tetany, tetanus, strychnine poisoning, typhoid fever, cholera, septicemia; Cerebral Disorders: Encephalitis, epilepsy (transient), catalepsy, hysteria, malingering.
Inability to close the jaw—TMJ dislocation.
biconcave surface, it easily partially subluxes or completely dislocates. The
jaw won’t close after a yawn or receipt of an upward blow on the chin with
the mouth wide open. The mandible protrudes, the lower teeth overriding
the uppers. There is a depression or pit anterior to the tragus that is more
obvious when bilateral. In unilateral dislocation, the pretragal depression
occurs only on the affected side. No movement of the mandibular head is felt
when palpating through the external acoustic meatus on the affected side.
External Ear Signs
Earlobe crease. A visible crease extending at least one-third of the distance
from tragus to posterior pinna is associated with a higher rate of cardiac
events in hospital admissions with suspected coronary heart disease.
Earlobe nodule: gouty tophus. In long-standing gout, sodium urate crystals
accumulate in the helix and antihelix, the olecranon bursa, tendon sheaths,
and aponeuroses of the extremities. The nodules are painless, hard, and irreg-
ular. They may open discharging chalky contents.
Darwin tubercle. This is a harmless developmental eminence in the upper
third of the posterior helix that must be distinguished from acquired nodules,
such as tophi.
Other nodules. Nontender nodules may be basal cell carcinomas, rheumatoid
nodules, or leprosy. Cartilage calcication is a rare complication of Addison
disease.
Hematoma. Trauma or a hemostatic defect results in blood accumulating
between the cartilage and the perichondrium as a tender, blue, doughy mass,
usually without spontaneous pain. Prompt incision and drainage avoid suppuration or cauliower ear.
Because the TMJ is a shallow

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Recurring inammation—relapsing polychondritis. There is inammation
and degeneration of cartilage especially of the pinna, nasal septum, laryngeal cartilages, tracheal and bronchial rings; joint cartilages may be affected.
The ear is painful, swollen, and reddened, except over the lobule. Hoarseness
indicates laryngeal involvement and blindness results from involvement of
the sclerae, and tinnitus and deafness from middle ear involvement. Rarely,
aortic or mitral valve ring degeneration produces valvular regurgitation or
aortic aneurysm.
Dermoid cyst. A favorite site is just behind the pinna. It is soft and slightly
uctuant.
External Acoustic Meatus Signs
Cerumen impaction. The wax of Native Americans and East Asians is often
dry and akey, and more yellow than amber. Excessive wax production or
a narrow meatus leads to impacted cerumen and partial or complete canal
obstruction. Complete obstruction causes partial deafness; tinnitus or dizziness may occur. Partial obstructions can suddenly become complete when
water enters the meatus during bathing or swimming. The obstructing wax is
easily seen in the external meatus.
Ear discharge—otorrhea. Ear discharge has many causes, the type suggesting
the diagnosis: Yellow Discharge: Melting cerumen; Serous Discharge: Eczema,
early ruptured acute otitis media; Bloody Discharge: External canal trauma or
longitudinal temporal bone fracture with TM and external canal laceration;
Purulent Discharge: Chronic external otitis, perforating acute suppurative oti-
tis media, chronic suppurative or tuberculous otitis media with or without
cholesteatoma.
External otitis. See page 243.
Dermatitis. Seborrheic dermatitis commonly causes scaling and pruritus of
the choana and meatus. Medicated eardrops can cause contact dermatitis.
Carcinoma. Either squamous cell or basal cell carcinoma can involve the
meatal epithelium. Pain and discharge are presenting symptoms, with deafness and facial paralysis occurring in advanced disease.
Foreign body. Children often place objects in their ears. A purulent discharge
from the canal or an earache may be the rst indication.
Polyps. A bulbous, reddened, pedunculated mass arising from the canal wall
or middle ear is associated with a foul purulent discharge. Moving it with
forceps may reveal the origin.
Exostoses and chondromas. Exostoses form nodules in the osseous canal
near the TM. They rarely produce obstruction, although the TM may be partially obscured. A single bony osteoma may occur. Rarely, chondromas arise
from the cartilaginous canal, usually without obstruction.

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Furuncle. A red, tender prominence with or without a pustule forms in the
cartilaginous canal producing extreme pain.
Vesicles. Pain in the ear with vesicles in the canal and facial weakness is
caused by herpes zoster of CN-VII (Ramsey–Hunt Syndrome), Chapter 14,
Facial Weakness and Paralysis, pages 665–666.
TM Signs
Retracted TM. See Otitis Media with Effusion, page 244.
Red or bulging TM. See Acute Suppurative Otitis Media, page 244.
Vesicles on the TM. Mycoplasma pneumoniae infection causes severe ear pain
and an inamed TM often with hemorrhagic vesicles (bullous myringitis).
Perforated TM. A healed suppurative middle ear infection eroded through
the TM leaving an oval hole through which the middle ear cavity is seen.
Chronic perforations are asymptomatic other than mildly decreased auditory
acuity.
Hearing Signs
Lateralizing Weber test—ipsilateral conductive hearing loss or contralateral neurosensory loss. When neurosensory hearing is intact bilaterally,
sound lateralizes to the side of conductive loss that has lost the masking effect
of background noise. Neurosensory loss on one side results in a louder sound
on the opposite side. Therefore, lateralization of sound to the right ear means
conductive loss on the right or perceptive loss on the left.
Bone conduction greater than air conduction (Rinne-negative test)—
conductive hearing loss. When amplication of sound by the TM and ossi-
cles is impaired, direct transmission of vibrations to the cochlea through bone
appears louder than sound transmitted through air. Conductive hearing loss
results from auditory canal obstruction, TM damage, middle ear uid, and
destruction or ankylosis of the ossicles.
Balance and position sense signs. See Chapter 14, page 671.
Eye Lid Signs
Lacrimation, tearing. Although strictly speaking an overproduction of tears,
lacrimation usually refers to any condition resulting in tears. Epiphora means
an overow of tears from any cause.
CLINICAL OCCURRENCE: Increased Secretion: Weeping from emotion, for-
eign body irritation, corneal ulcer, conjunctivitis, coryza, measles, hay fever,
poisoning (iodide, bromide, arsenic);
cicatrix, eyelid edema, lacrimal calculus, dacryocystitis; Puncta Separation
from the Globe: Facial paralysis, aging, chronic marginal blepharitis, ectropion,
proptosis.
Widened palpebral ssures. The ssures are widened by lid retraction (con-
traction of Mueller muscle) or protrusion of the globe. Normally, with eyes
Lacrimal Duct Obstruction: Congenital,
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